Academic literature on the topic 'Torb'

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Journal articles on the topic "Torb"

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Bordi, Christophe, Chantal Iobbi-Nivol, Vincent Méjean, and Jean-Claude Patte. "Effects of ISSo2 Insertions in Structural and Regulatory Genes of the Trimethylamine Oxide Reductase of Shewanella oneidensis." Journal of Bacteriology 185, no. 6 (March 15, 2003): 2042–45. http://dx.doi.org/10.1128/jb.185.6.2042-2045.2003.

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ABSTRACT We have isolated three Shewanella oneidensis mutants specifically impaired in trimethylamine oxide (TMAO) respiration. The mutations arose from insertions of an ISSo2 element into torA, torR, and torS, encoding, respectively, the TMAO reductase TorA, the response regulator TorR, and the sensor TorS. Although TorA is not the sole enzyme reducing TMAO in S. oneidensis, growth analysis showed that it is the main respiratory TMAO reductase. Use of a plasmid-borne torE′-lacZ fusion confirmed that the TorS-TorR phosphorelay mediates TMAO induction of the torECAD operon.
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Hölper, Julia E., Barbara G. Klupp, G. W. Gant Luxton, Kati Franzke, and Thomas C. Mettenleiter. "Function of Torsin AAA+ ATPases in Pseudorabies Virus Nuclear Egress." Cells 9, no. 3 (March 17, 2020): 738. http://dx.doi.org/10.3390/cells9030738.

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Newly assembled herpesvirus nucleocapsids traverse the intact nuclear envelope by a vesicle-mediated nucleo-cytoplasmic transport for final virion maturation in the cytoplasm. For this, they bud at the inner nuclear membrane resulting in primary enveloped particles in the perinuclear space (PNS) followed by fusion of the primary envelope with the outer nuclear membrane (ONM). While the conserved viral nuclear egress complex orchestrates the first steps, effectors of fusion of the primary virion envelope with the ONM are still mostly enigmatic but might include cellular proteins like SUN2 or ESCRT-III components. Here, we analyzed the influence of the only known AAA+ ATPases located in the endoplasmic reticulum and the PNS, the Torsins (Tor), on nuclear egress of the alphaherpesvirus pseudorabies virus. For this overexpression of wild type and mutant proteins as well as CRISPR/Cas9 genome editing was applied. Neither single overexpression nor gene knockout (KO) of TorA or TorB had a significant impact. However, TorA/B double KO cells showed decreased viral titers at early time points of infection and an accumulation of primary virions in the PNS pointing to a delay in capsid release during nuclear egress.
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YAMAMOTO, ISAMU, MASATO HOHMURA, and MAKOTO ISHIMOTO. "A novel gene, torB, for trimethylamine N-oxide reductase in Escherichia coli." Journal of General and Applied Microbiology 35, no. 2 (1989): 95–105. http://dx.doi.org/10.2323/jgam.35.95.

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Bordi, Christophe, Mireille Ansaldi, Stéphanie Gon, Cécile Jourlin-Castelli, Chantal Iobbi-Nivol, and Vincent Méjean. "Genes Regulated by TorR, the Trimethylamine Oxide Response Regulator of Shewanella oneidensis." Journal of Bacteriology 186, no. 14 (July 15, 2004): 4502–9. http://dx.doi.org/10.1128/jb.186.14.4502-4509.2004.

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ABSTRACT The torECAD operon encoding the trimethylamine oxide (TMAO) respiratory system of Shewanella oneidensis is positively controlled by the TorS/TorR two-component system when TMAO is available. Activation of the tor operon occurs upon binding of the phosphorylated response regulator TorR to a single operator site containing the direct repeat nucleotide sequence TTCATAN4TTCATA. Here we show that the replacement of any nucleotide of one TTCATA hexamer prevented TorR binding in vitro, meaning that TorR specifically interacts with this DNA target. Identical direct repeat sequences were found in the promoter regions of torR and of the new gene torF (SO4694), and they allowed TorR binding to both promoters. Real-time PCR experiments revealed that torR is negatively autoregulated, whereas torF is strongly induced by TorR in response to TMAO. Transcription start site location and footprinting analysis indicate that the operator site at torR overlaps the promoter −10 box, whereas the operator site at torF is centered at −74 bp from the start site, in agreement with the opposite role of TorR in the regulation of the two genes. Since torF and torECAD are positively coregulated by TorR, we propose that the TorF protein plays a role related to TMAO respiration.
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Sturgill, Thomas W., Adiel Cohen, Melanie Diefenbacher, Mark Trautwein, Dietmar E. Martin, and Michael N. Hall. "TOR1 and TOR2 Have Distinct Locations in Live Cells." Eukaryotic Cell 7, no. 10 (August 22, 2008): 1819–30. http://dx.doi.org/10.1128/ec.00088-08.

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ABSTRACT TOR is a structurally and functionally conserved Ser/Thr kinase found in two multiprotein complexes that regulate many cellular processes to control cell growth. Although extensively studied, the localization of TOR is still ambiguous, possibly because endogenous TOR in live cells has not been examined. Here, we examined the localization of green fluorescent protein (GFP) tagged, endogenous TOR1 and TOR2 in live S. cerevisiae cells. A DNA cassette encoding three copies of green fluorescent protein (3XGFP) was inserted in the TOR1 gene (at codon D330) or the TOR2 gene (at codon N321). The TORs were tagged internally because TOR1 or TOR2 tagged at the N or C terminus was not functional. The TOR1 D330-3XGFP strain was not hypersensitive to rapamycin, was not cold sensitive, and was not resistant to manganese toxicity caused by the loss of Pmr1, all indications that TOR1-3XGFP was expressed and functional. TOR2-3XGFP was functional, as TOR2 is an essential gene and TOR2 N321-3XGFP haploid cells were viable. Thus, TOR1 and TOR2 retain function after the insertion of 748 amino acids in a variable region of their noncatalytic domain. The localization patterns of TOR1-3XGFP and TOR2-3XGFP were documented by imaging of live cells. TOR1-3XGFP was diffusely cytoplasmic and concentrated near the vacuolar membrane. The TOR2-3XGFP signal was cytoplasmic but predominately in dots at the plasma membrane. Thus, TOR1 and TOR2 have distinct localization patterns, consistent with the regulation of cellular processes as part of two different complexes.
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Turner, Elizabeth M., Rebecca S. H. Brown, Ethan Laudermilch, Pei-Ling Tsai, and Christian Schlieker. "The Torsin Activator LULL1 Is Required for Efficient Growth of Herpes Simplex Virus 1." Journal of Virology 89, no. 16 (June 3, 2015): 8444–52. http://dx.doi.org/10.1128/jvi.01143-15.

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ABSTRACTTorsinA is a membrane-tethered AAA+ ATPase implicated in nuclear envelope dynamics as well as the nuclear egress of herpes simplex virus 1 (HSV-1). The activity of TorsinA and the related ATPase TorsinB strictly depends on LAP1 and LULL1, type II transmembrane proteins that are integral parts of the Torsin/cofactor AAA ring, forming a composite, membrane-spanning assembly. Here, we use CRISPR/Cas9-mediated genome engineering to create single- and double knockout (KO) cell lines of TorA and TorB as well as their activators, LAP1 and LULL1, to investigate the effect on HSV-1 production. Consistent with LULL1 being the more potent Torsin activator, a LULL1 KO reduces HSV-1 growth by one order of magnitude, while the deletion of other components of the Torsin system in combination causes subtle defects. Notably, LULL1 deficiency leads to a 10-fold decrease in the number of viral genomes per host cell without affecting viral protein production, allowing us to tentatively assign LULL1 to an unexpected role that precedes HSV-1 nuclear egress.IMPORTANCEIn this study, we conduct the first comprehensive genetic and phenotypic analysis of the Torsin/cofactor system in the context of HSV-1 infection, establishing LULL1 as the most important component of the Torsin system with respect to viral production.
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Li, Ning, Junping Jiang, Fulu Sun, and Mingrui Ye. "Study on the Influence of Suspension Parameters on Longitudinal Impact Comfort." Security and Communication Networks 2022 (May 2, 2022): 1–12. http://dx.doi.org/10.1155/2022/7749029.

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Ride comfort criteria are a key challenge for vehicle dynamic design and optimization. Currently optional parameter is the vertical impact, and longitudinal impact is neglected. With further requirements for future comfortability, effects of longitudinal impact should be investigated in detail. A longitudinal impact model is firstly proposed to evaluate the ride comfort factors based on the dynamic theory and commercial ADAMS® software. Predictions revealed that the hard points of the suspension and the stiffness of rubber bushing (SORB) are the primary factors. A novelty finding is that travel of rubber bushing (TORB) in the linear region is the most important parameter for ride comfort optimization and suspension factor is the weakest, and experimental validation is performed with better agreements.
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Yanagida, Mitsuhiro, Nobuyasu Ikai, Mizuki Shimanuki, and Kenichi Sajiki. "Nutrient limitations alter cell division control and chromosome segregation through growth-related kinases and phosphatases." Philosophical Transactions of the Royal Society B: Biological Sciences 366, no. 1584 (December 27, 2011): 3508–20. http://dx.doi.org/10.1098/rstb.2011.0124.

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In dividing fission yeast Schizosaccharomyces pombe cells, the balance between Wee1 kinase and Cdc25 phosphatase which control the cyclin-dependent kinase (CDK) at the G2–M transition determines the rod-shaped cell length. Under nitrogen source starvation or glucose limitation, however, cell size determination is considerably modulated, and cell size shortening occurs for wild-type cells. For several mutants of kinases or phosphatases, including CDK, target of rapamycin complex (TORC) 1 and 2, stress-responsive mitogen-activated protein kinase (MAPK) Sty1/Spc1, MAPK kinase Wis1, calcium- and calmodulin-dependent protein kinase kinase-like Ssp1, and type 2A and 2A-related phosphatases inhibitor Sds23, this cell shortening does not normally occur. In tor1 and ssp1 mutants, cell elongation is observed. Sds23 that binds to and inhibits 2A and 2A-related phosphatases is synergistic with Ssp1 in the cell size determination and survival under low glucose and nitrogen source. Tor2 (TORC1) is required for growth, whereas Tor1 (TORC2) is needed for determining division size according to different nutrient conditions. Surprisingly, in growth-diminished tor2 mutant or rapamycin-treated cells, the requirement of separase/Cut1-securin/Cut2 essential for chromosome segregation is greatly alleviated. By contrast, defects of tor1 with secruin/ cut2 or overproduction of Cut1 are additive. While Tor1 and Tor2 are opposite in their apparent functions, both may actually coordinate cell division with growth in response to the changes in nutrients.
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Ansaldi, Mireille, Gwénola Simon, Michèle Lepelletier, and Vincent Méjean. "The TorR High-Affinity Binding Site Plays a Key Role in Both torR Autoregulation and torCADOperon Expression in Escherichia coli." Journal of Bacteriology 182, no. 4 (February 15, 2000): 961–66. http://dx.doi.org/10.1128/jb.182.4.961-966.2000.

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ABSTRACT In the presence of trimethylamine N-oxide (TMAO), the TorS-TorR two-component regulatory system induces thetorCAD operon, which encodes the TMAO respiratory system ofEscherichia coli. The sensor protein TorS detects TMAO and transphosphorylates the response regulator TorR which, in turn, activates transcription of torCAD. The torRgene and the torCAD operon are divergently transcribed, and the short torR-torC intergenic region contains four direct repeats (the tor boxes) which proved to be TorR binding sites. The tor box 1-box 2 region covers thetorR transcription start site and constitutes a TorR high-affinity binding site, whereas box 3 and box 4 correspond to low-affinity binding sites. By using torR-lacZ operon fusions in different genetic backgrounds, we showed that thetorR gene is negatively autoregulated. Surprisingly, TorR autoregulation is TMAO independent and still occurs in atorS mutant. In addition, this negative regulation involves only the TorR high-affinity binding site. Together, these data suggest that phosphorylated as well as unphosphorylated TorR binds the box 1-box 2 region in vivo, thus preventing RNA polymerase from binding to the torR promoter whatever the growth conditions. By changing the spacing between box 2 and box 3, we demonstrated that the DNA motifs of the high- and low-affinity binding sites must be close to each other and located on the same side of the DNA helix to allow induction of the torCAD operon. Thus, prior TorR binding to the box 1-box 2 region seems to allow cooperative binding of phosphorylated TorR to box 3 and box 4.
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Helliwell, S. B., P. Wagner, J. Kunz, M. Deuter-Reinhard, R. Henriquez, and M. N. Hall. "TOR1 and TOR2 are structurally and functionally similar but not identical phosphatidylinositol kinase homologues in yeast." Molecular Biology of the Cell 5, no. 1 (January 1994): 105–18. http://dx.doi.org/10.1091/mbc.5.1.105.

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The Saccharomyces cerevisiae genes TOR1 and TOR2 were originally identified by mutations that confer resistance to the immunosuppressant rapamycin. TOR2 was previously shown to encode an essential 282-kDa phosphatidylinositol kinase (PI kinase) homologue. The TOR1 gene product is also a large (281 kDa) PI kinase homologue, with 67% identity to TOR2. TOR1 is not essential, but a TOR1 TOR2 double disruption uniquely confers a cell cycle (G1) arrest as does exposure to rapamycin; disruption of TOR2 alone is lethal but does not cause a cell cycle arrest. TOR1-TOR2 and TOR2-TOR1 hybrids indicate that carboxy-terminal domains of TOR1 and TOR2 containing a lipid kinase sequence motif are interchangeable and therefore functionally equivalent; the other portions of TOR1 and TOR2 are not interchangeable. The TOR1-1 and TOR2-1 mutations, which confer rapamycin resistance, alter the same potential protein kinase C site in the respective protein's lipid kinase domain. Thus, TOR1 and TOR2 are likely similar but not identical, rapamycin-sensitive PI kinases possibly regulated by phosphorylation. TOR1 and TOR2 may be components of a novel signal transduction pathway controlling progression through G1.
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Dissertations / Theses on the topic "Torb"

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Andersson, Martin. "Torkning integrerat med kraftvärmeverk : Påverkan av energibalanser i torksystem vid integrering till kraftvärmeverk." Thesis, Karlstads universitet, Institutionen för ingenjörs- och kemivetenskaper, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:kau:diva-46998.

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I Sveg finns ett pneumatiskt torksystem. Systemet har sedan 1989 torkat torv till briketter men har på senare tid torkat både trä och torv för produktion av bränsle i form av pellets och briketter. Idag består anläggningen av två symmetriska torklinjer. I torklinjerna används en värmepumpkrets för återvinning av råvaruånga. I värmepumpkretsen används en kompressor driven av inköpt elenergi. Planerna är att integrera en av linjernas nuvarande torkprocess till ett kraftvärmeverk genom installation av ångpanna med bränslet torv och således producera el istället för att köpa el. Integrationen till kraftvärmeverket ger torklinjens värmepumpkrets ett nytt utseende. Kompressorn tas bort i utvald torklinje och ersätts av två lågtrycksturbiner. Det nya utseendet av värmepumpkretsen kommer innebära ett samspel mellan torkning och kraftvärmeverk. I värmepumpkretsen används olika ångflöden beroende av vilket råvarumaterial som torkas. Därför beräknades och användes olika ångflöden beroende av olika fukthalter och inmatningar i torksystemet för att se hur el- och fjärrvärmeproduktionen i kraftvärmeverket påverkades. Samtidigt jämfördes nuvarande torksystem med torksystem integrerat med kraftvärmeverk ur ett energiperspektiv genom förändring av el, fjärrvärmeproduktion och biobränsle. Fjärrvärmeproduktionen i kraftvärmeverket ökade vid ångflödena 31,0 ton/h och 24,0 ton/h ”till kompressor” men på grund av större beräkningsosäkerhet bortsågs dessa. Detta gav en varierad sammanlagd fjärrvärmeproduktion av systemen mellan 23,0-23,8 MW respektive 22,0-22,5 MW vid torkning av torv respektive trä (bortseende av torvscenario 1 vid fukthalten 65 %). En fjärrvärmeproduktion i den storleken, jämfört med Svegs fjärrvärmebehov, kan sägas vara för stort. En stor problematik blir därför vad all spillvärme ska användas till, framförallt på sommaren. Vid integrering till kraftvärmeverk minskade ”bränsle” medan ”fjärrvärme” och ”el” ökade. Beroende av vilket råvarumaterial som torkades gav en integrering ett större resultat för ”el” men mindre ”fjärrvärme” och ”bränsle” vid torkning av trä. Därför rekommenderas torkning av trä eftersom större resultat av ”el” minskar inköpt elenergi samt mindre ”fjärrvärme” resulterar till mindre spillvärme. Vid beräkningarna användes Simulink, ett verktyg för grafisk lösning av differentialekvationer, som ingår i programvaran MATLAB från Mathworks. I Simulink formerades olika ekvationer i statiska beräkningsmodeller för beräkning av energibalanserna i nuvarande torksystem och torksystem integrerat med kraftvärmeverk. Energibalanserna i nuvarande torksystem undersökte svårplacerade effektförluster samt genom olika energibalanskontroller kontrollerade och bedömde trovärdigheten i beräkningsmodellen. Resultaten för energibalanskontrollerna kallades ”beräkningsosäkerhet” vilket var en av flera parametrar som framlade bevis för ett variabelberoende, känsligt, komplext och svårtydande torksystem där övriga effektförluster var svåra att placera. Ett mer korrekt resultat av nuvarande system kan därför möjligtvis uppnås vid användande av en dynamisk istället för statisk modell samt användande enbart av mätdata istället för dimensionerande värden och således göra effektförlusterna lättare att placera.
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Genest, Olivier. "Les chaperons dédiés à la biogénèse des molybdoenzymes : étude du couple chaperon TorD - molybdoenzyme TorA chez Escherichia coli." Aix-Marseille 2, 2008. http://theses.univ-amu.fr.lama.univ-amu.fr/2008AIX22087.pdf.

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Les molybdoenzymes sont des métalloprotéines dont le site actif est constitué d’un cofacteur à molybdène. Ces molybdoenzymes sont retrouvées chez tous les êtres vivants, des bactéries à l’homme. Leur biogenèse est un processus complexe qui nécessite la présence de protéines chaperons spécifiques. Au cours de ma thèse, j’ai étudié le rôle de la protéine chaperon TorD dans la biogenèse de la molybdoenzyme TorA chez Escherichia coli. TorA est l’enzyme terminale périplasmique de la chaîne respiratoire triméthylamine oxyde (TMAO) réductase. J’ai montré que le chaperon spécifique TorD, localisé dans le cytoplasme, est essentiel à la protection de la forme non mature de TorA (apoTorA) lors d’un stress thermique ou d’une carence en cofacteur à molybdène. En effet, l’absence de TorD dans ces conditions entraîne la dégradation complète de l’apoprotéine. J’ai également montré que la séquence signal Tat de TorA, qui permet l’export de la protéine vers le périplasme est hypersensible à la dégradation par les protéases. Cette séquence signal pourrait être une voie d’entrée pour les protéases qui ensuite dégraderaient l’ensemble de l’apoenzyme. TorD en interagissant avec la séquence signal de TorA empêche cette première dégradation et permet donc la protection de l’apoenzyme. TorD se lie également à la partie globulaire d’poTorA. Par cette interaction, TorD permet une maturation optimale de l’apoenzyme. Les acides aminés de TorD impliqués dans cette interaction ont été déterminés après mutagenèse aléatoire. Ils sont localisés dans la cinquième hélice de TorD. J’ai également montré que TorD présente un rôle de plate-forme sur laquelle se lie le précurseur du cofacteur à molybdène et l’enzyme MobA permettant la synthèse de la forme mature du cofacteur. Après catalyse, cette forme mature du cofacteur qui se lie à TorD peut être délivrée à l’apoenzyme TorA. Ainsi, TorD connecte tous les éléments nécessaires à la maturation de TorA : d’une part il interagit avec le cofacteur à molybdène et d’autre part avec l’apoenzyme. Nous proposons donc que TorD interagisse à proximité du site actif de TorA pour y délivrer directement le cofacteur à molybdène
T-ALL is a lymphoid neoplasia that accounts for 10-15% of pediatric ALL and 25% of adult ALL. Alarmingly, and despite indisputable success achieved in treatments its incidence is increasing and its prognostic remains pejorative. Survival rate outcome depend notably on a better understanding in pathogenic mechanisms. In this context, the thesis work has been the following: 1) Based on the observation that rare chromosomal SJ keep on recombining in cis using V(D)J recombination, we hypothesized that episomal SJ (ESJ) still remain reactives and can undergo genomic reintegration. We show that mechanistically, ESJ efficiently rearrange in trans and that the cRSS, the sequences targeted in oncogenic chromosomal translocations, are good ESJ integration sites. Moreover, we demonstrate the presence of ESJ reintegration events in vivo and estimate their frequency to ~1/104-6. In conclusion, ESJ reintegration is a potential mechanism of oncogenic deregulation. 2) Conventional and illegitimate V(D)J recombination events (e. G. Translocations) are ordered during lymphocyte development. Based on our knowledge on chromosomal translocation mechanisms, we determine the kinetics of a subset of oncogenic activations acquired during the transformation process in a T-ALL patient’s leukemic cells. Moreover, we identified up to 10 independent oncogenic events in this patient, illustrating the multi-hit characteristic of T-ALL. Finally, the oncogenic event’s functional impact suggests that cMyc play an important role in the particularly aggressive features of the T-ALL developed by this patient
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Varol, Oğuz. "Kriterien für Tor1[alpha] (E, F)=0 [Tor 1 alpha (E, F)=0] für (DF)- und Frécheträume." [S.l. : s.n.], 2002. http://deposit.ddb.de/cgi-bin/dokserv?idn=969223277.

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Moenikes, Ansgar. "Tora ohne Mose : zur Vorgeschichte der Mose-Tora /." Berlin : Philo, 2004. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=013004084&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.

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Schnapp, Jonathan. "Torn away /." Online version of thesis, 2008. http://hdl.handle.net/1850/6248.

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Buhl, Nils E. "Tora! Tora! Tora!: The attack on Pearl Harbor from a prospect theoretic, culture-centric perspective." Connect to online resource, 2007. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:1446084.

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Pettersson, Ina. "Sergels torg och Medborgarplatsen - En jämförande studie av torg." Thesis, Blekinge Tekniska Högskola, Institutionen för fysisk planering, 2018. http://urn.kb.se/resolve?urn=urn:nbn:se:bth-16560.

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Kandidatarbetet undersöker torg och orsakerna till dess olika användningar ur ett socio-rumsligt tillvägagångssätt, det vill säga förhållandet mellan människan och den fysiska miljön. I detta arbete kommer de två valda torgen, Medborgarplatsen och Sergels torg undersökas och analyseras för att finna komponenter som kan vara en del av förklaringen till dess olika användning. Genom arbetet kommer det tas fram olika begrepp och delar som anses vara viktiga för hur ett torg fungerar och påverkar de som väljer att besöka platsen. I slutsatsen har det samanställts viktiga komponenter. Komponenter som tas upp är bland annat utformingen, platsens tillgänglighet, om torget bjuder in till aktivitetet eller hindrar andra aktiviteter och hur torget uppfattas av dess användare. Dessa utgör tillsammans en betydande roll för hur platserna kommer användas av dess besökare. Vidare diskteras effekterna av torgens komponenter och dess potentiella effekter på de människor som väljer att besöka platserna.
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Jarlegård, Hanna, and Bastien Lacombe. "Trygga torg : En studie av torg ur ett trygghetsperspektiv." Thesis, KTH, Urbana och regionala studier, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-252783.

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Trygghetsarbete ärnågot som både statliga myndigheter och kommuner intresserar sig för. Kommunernämner gärna i sina översiktsplaner att trygghetsfrämjande åtgärder ska vidtasoch att områden ska utvecklas med hänsyn till tryggheten. Hur dettatrygghetsarbete tar sig form i den fysiska miljön är mindre tydligt och syftetmed denna studie har således varit att ta fram aspekter som går att arbeta medför att främja trygghet. Dessa aspekter har sedan använts för att analyseratorg i Stockholm stad, Nacka kommun och Vaxholm stad för att se hur väl torgensvarar mot dessa aspekter. Aspekterna som tagits fram med hjälp av enlitteraturstudie och den fallstudie som har utförts har sedan legat till grundför att ta fram tips för hur trygga torg kan utformas.Litteraturstudien ledde till att fem aspekter av stor betydelse för trygghetentogs fram: närhet till andra människor, synlighet och överblickbarhet,belysning, grönska och skötsel av allmän plats. Inget av de torg som omfattadesav fallstudien uppnådde samtliga trygghetsaspekter, men vissa uppfyllde fler änandra. Utöver de slutsatser som har dragits kring vilka aspekter som är trygghetsfrämjandeså ledde studien till slutsatser kring vilka torg som uppfyller vilka aspekter.Slutligen ledde studien fram till konkreta förbättringsförslag för trygghetenpå de torg som fallstudien omfattade och handfasta tips för hur torg kanutformas med hänsyn till trygghet
Working with perceived safety is something that both government agencies and municipalities are interested in. Municipalities gladly mention in their general plans that perceived safety actions is something that should be prioritised and that new areas should be developed with regard to the perceived safety. Exactly how these perceived safety actions takes shape in the physical environment is less clear and the purpose has thus been to identify aspects that are possible to work with and that promotes the perceived safety. The identified aspects have then been used to analyse squares in Stockholm stad, Nacka kommun and Vaxholm stad to see how well these places correspond to the these aspects. Tips on how squares that are perceived as safe was then formulated based on the case study and the aspects that was identified in the literature study.The literature study led to the identification of five aspects with great significance to the perceived safety: closeness to other people, visibility and overview, lightning, greenery and maintenance of public space. None of the observed squares fulfill all the aspects, but some fulfill more than others. Other than the conclusions that were drawn concerning which aspects that are promoting for the perceived safety, the study also led to conclusions regarding which squares that fulfill which aspects. Finally the study led to improvement-proposals for perceived safety on the squares of the case study and concrete tips on how squares can be designed with regard to perceived safety.
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Freedman, Margot. "Paternalistic tort law." Diss., Online access via UMI:, 2005. http://wwwlib.umi.com/dissertations/fullcit/1425588.

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Goudkamp, James. "Tort law defences." Thesis, University of Oxford, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.539957.

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Books on the topic "Torb"

1

Viadiu, Francesc. Entre el torb i la Gestapo. Barcelona: Llar del Llibre, 1985.

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Viadiu, Francesc. Entre el torb i la Getapo. Barcelona: R. Dalmau, 2000.

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Viadiu, Francesc. Entre el torb i la Gestapo. Barcelona: R. Dalmau, 2000.

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Rozin, M. Tora vo tʹme tolkovaniĭ i tolkovanii︠a︡ v svete tory. Ierusalim: [s.n.], 2000.

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Mata, Humberto. Toro-toro. Caracas, Venezuela: Alfadil Ediciones, 1991.

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Morpurgo, Michael. Toro! Toro! London: Collins, 2002.

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Quiroga, Juan Cristóbal. Toro toro. Bolivia]: Expediciones Bolivia, 2005.

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Mata, Humberto. Toro-toro. Caracas, Venezuela: Alfadil Ediciones, 1991.

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Stille, Mark. Tora! Tora! Tora!: Pearl Harbor 1941. Oxford: Osprey, 2011.

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Stille, Mark. Tora! Tora! Tora!: Pearl Harbor 1941. Oxford: Osprey, 2011.

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Book chapters on the topic "Torb"

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Bährle-Rapp, Marina. "Torf." In Springer Lexikon Kosmetik und Körperpflege, 560–61. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-71095-0_10611.

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Neufert, Peter, and Ludwig Neff. "Tore." In Gekonnt Planen Richtig Bauen, 107. Wiesbaden: Vieweg+Teubner Verlag, 1997. http://dx.doi.org/10.1007/978-3-322-96920-0_44.

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Gracia-Bondía, José M., Joseph C. Várilly, and Héctor Figueroa. "Tori." In Elements of Noncommutative Geometry, 519–56. Boston, MA: Birkhäuser Boston, 2001. http://dx.doi.org/10.1007/978-1-4612-0005-5_12.

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Schwartz, Maurice. "Tors." In Encyclopedia of Earth Sciences Series, 1771–73. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-93806-6_327.

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Bump, Daniel. "Tori." In Lie Groups, 101–8. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-8024-2_15.

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Bump, Daniel. "Tori." In Lie Groups, 86–93. New York, NY: Springer New York, 2004. http://dx.doi.org/10.1007/978-1-4757-4094-3_15.

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Gooch, Jan W. "Torr." In Encyclopedic Dictionary of Polymers, 755. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_11961.

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Schwartz, Maurice. "Tors." In Encyclopedia of Earth Sciences Series, 1–3. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-48657-4_327-2.

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Mustoe, George, Paolo A. Pirazzoli, Rhodes W. Fairbridge, Terry R. Healy, Edward B. Hands, B. W. Flemming, Duncan M., et al. "Tors." In Encyclopedia of Coastal Science, 1001–2. Dordrecht: Springer Netherlands, 2005. http://dx.doi.org/10.1007/1-4020-3880-1_327.

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Foster, Charles, and Jonathan Herring. "Tort." In The Law as a Moral Agent, 23–36. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-71334-8_3.

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Conference papers on the topic "Torb"

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Steiner, Neil, Aaron Wood, Hamid Shojaei, Jacob Couch, Peter Athanas, and Matthew French. "Torc." In the 19th ACM/SIGDA international symposium. New York, New York, USA: ACM Press, 2011. http://dx.doi.org/10.1145/1950413.1950425.

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Lee, Jaeyeon, Mike Sinclair, Mar Gonzalez-Franco, Eyal Ofek, and Christian Holz. "TORC." In CHI '19: CHI Conference on Human Factors in Computing Systems. New York, NY, USA: ACM, 2019. http://dx.doi.org/10.1145/3290605.3300301.

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Smith, John, and Kazuhiro Jo. ""Toru"." In CHI PLAY '14: The annual symposium on Computer-Human Interaction in Play. New York, NY, USA: ACM, 2014. http://dx.doi.org/10.1145/2658537.2662973.

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Makmur, Makmur, Yadi Mulyadi, Hasanuddin Hasanuddin, Muhlis Hadrawi, Nila Kalsum, Ade Sahroni, and Lucas Wattimena. "Tomb Architecture." In 9th Asbam International Conference (Archeology, History, & Culture In The Nature of Malay) (ASBAM 2021). Paris, France: Atlantis Press, 2022. http://dx.doi.org/10.2991/assehr.k.220408.014.

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De, P., Y. Sun, N. Dey, and B. Leyland-Jones. "P3-18-03: In Vitro Potency of mTOR Kinase (TOR1/TOR2) Inhibitor, INK128 in ER+ and HER2 Overexpressing Breast Cancer Cells." In Abstracts: Thirty-Fourth Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 6‐10, 2011; San Antonio, TX. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/0008-5472.sabcs11-p3-18-03.

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Shih, Yuan-Kang, Shin-Shin Kao, Lih-Hsing Hsu, Theodore E. Simos, and George Psihoyios. "Deformed Honeycomb Tori." In INTERNATIONAL ELECTRONIC CONFERENCE ON COMPUTER SCIENCE. AIP, 2008. http://dx.doi.org/10.1063/1.3037088.

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Lee, Jaeyeon, Mike Sinclair, Mar Gonzalez-Franco, Eyal Ofek, and Christian Holz. "Demonstration of TORC." In UIST '19: The 32nd Annual ACM Symposium on User Interface Software and Technology. New York, NY, USA: ACM, 2019. http://dx.doi.org/10.1145/3332167.3356898.

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Truckenbrod, Joan. "Torn touch, 1997." In ACM SIGGRAPH 98 Electronic art and animation catalog. New York, New York, USA: ACM Press, 1998. http://dx.doi.org/10.1145/281388.281775.

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Taylor, Paul. "Tort/professional negligence." In IEE Colloquium on `Principles of Law for Engineers and Managers'. IEE, 1996. http://dx.doi.org/10.1049/ic:19961425.

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Hilal, Allaa R., Amal El Nahas, and Ahmed Bashandy. "RT-TORA: A TORA modification for real-time interactive applications." In 2008 Canadian Conference on Electrical and Computer Engineering - CCECE. IEEE, 2008. http://dx.doi.org/10.1109/ccece.2008.4564772.

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Reports on the topic "Torb"

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Barton, Adam. The Secret Sauce of Development Professionals: Tools for Assessing TOR Potential to Source Scalable Learning Interventions. Research on Improving Systems of Education (RISE), March 2023. http://dx.doi.org/10.35489/bsg-rise-ri_2023/054.

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Abstract:
Terms of reference (TORs) play an outsized role in driving scalable educational programming. These procurement documents shape, constrain, and signal programme priorities and possibilities. Successful funders and implementers across the globe hold rich processual knowledge about this documentation, which they use to draft and assess TORs. This project explores such best-practice knowledge around TOR review, seeking to support the design and implementation of educational programmes that can improve learning at scale in developing contexts.
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Frost, R. L. TORT certification package. Office of Scientific and Technical Information (OSTI), October 1993. http://dx.doi.org/10.2172/10107073.

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Galasso, Alberto, and Hong Luo. Tort Reform and Innovation. Cambridge, MA: National Bureau of Economic Research, October 2016. http://dx.doi.org/10.3386/w22712.

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Ehlen, Judy. Classification of Dartmoor Tors. Fort Belvoir, VA: Defense Technical Information Center, September 1992. http://dx.doi.org/10.21236/ada257191.

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Rhoades, W. A., and D. B. Simpson. The TORT three-dimensional discrete ordinates neutron/photon transport code (TORT version 3). Office of Scientific and Technical Information (OSTI), October 1997. http://dx.doi.org/10.2172/582265.

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Birdsey, R. A., and D. Jiménez. The Forests of Toro Negro. New Orleans, LA: U.S. Department of Agriculture, Forest Service, Southern Forest Experiment Station, 1985. http://dx.doi.org/10.2737/so-rp-222.

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Clawson, Patrick. Iran: Torn by Domestic Disputes. Fort Belvoir, VA: Defense Technical Information Center, July 1997. http://dx.doi.org/10.21236/ada385964.

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Longacre, Ronald S. A Hole Torn by QCD Fields. Office of Scientific and Technical Information (OSTI), November 2018. http://dx.doi.org/10.2172/1481407.

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Jansen, Rob, Paul Syverson, and Nicholas J. Hopper. Throttling Tor Bandwidth Parasites. Fort Belvoir, VA: Defense Technical Information Center, September 2011. http://dx.doi.org/10.21236/ada559183.

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R. B. White, F. W. Perkins, X. Garbet, C. Bourdelle, V. Basiuk, and L. G. Eriksson. Rotation and particle loss in Tore Supra. Office of Scientific and Technical Information (OSTI), June 2000. http://dx.doi.org/10.2172/756909.

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