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Books on the topic 'Tissu vasculaire'

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Author: Grafiati

Published: 7 September 2022

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1

Zhao, Feng, and Kam W. Leong, eds. Vascular Tissue Engineering. New York, NY: Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-1708-3.

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2

Walpoth, Beat, Helga Bergmeister, Gary Bowlin, Deling Kong, Joris Rotmans, and Peter Zilla, eds. Tissue-Engineered Vascular Grafts. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-71530-8.

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3

service), ScienceDirect (Online, ed. Tissue-specific vascular endothelial signals and vector targeting. Amsterdam: Elsevier, 2009.

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4

Scarpella, Enrico. Genetic control fo vascular tissue development in rice. [Leiden]: E. Scarpella, 2003.

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5

Advanced School on "Biomechanics of Soft Tissue" (2001 Udine, Italy). Biomechanics of soft tissue in cardiovascular systems. Wien: Springer, 2003.

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6

Fu, Bingmei M., and Neil T. Wright, eds. Molecular, Cellular, and Tissue Engineering of the Vascular System. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-96445-4.

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7

Modeling tumor vasculature: Molecular, cellular, and tissue level aspects and implications. New York: Springer, 2012.

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8

Laey, J. J. De. Vascular tumors and malformations of the ocular fundus. Dordrecht: Kluwer Academic Publishers, 1990.

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9

Luis, Requena, ed. Pathology of vascular skin lesions: Clinicopathological correlations. Totowa, N.J: Humana Press, 2003.

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10

M, Hanssens, and Belgian Ophthalmological Society, eds. Vascular tumours and malformations of the ocular fundus. Dordrecht: Kluwer Academic Publishers, 1990.

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11

Birthmarks of medical significance. Philadelphia, Pa: Saunders, 2010.

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12

H, Müller Gottfried, and Ehrenfeld M, eds. Microvascular tissure transfer in the head and neck region. Stuttgart: Georg Thieme Verlag, 1993.

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13

Tissue Engineering of Vascular Prosthetic Grafts (Tissue Engineering Intelligence Unit). Landes Bioscience, 1999.

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14

P, Zilla P., and Greisler Howard P, eds. Tissue engineering of prosthetic vascular grafts. Austin: R.G. Landes Co., 1999.

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15

Zilla, Peter, Gary L. Bowlin, Helga Bergmeister, Deling Kong, Beat H. Walpoth, and Joris I. Rotmans. Tissue-Engineered Vascular Grafts. Springer, 2020.

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16

Zilla, Peter, Beat Walpoth, Helga Bergmeister, Gary Bowlin, Deling Kong, and Joris Rotmans. Tissue-Engineered Vascular Grafts. Springer, 2020.

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17

H, Campbell Julie, and Campbell Gordon R, eds. Vascular smooth muscle in culture. Boca Raton, Fla: CRC Press, 1987.

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18

Johri, Brij Mohan. Experimental Embryology of Vascular Plants. Springer, 2011.

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19

Tissue-Specific Vascular Endothelial Signals and Vector Targeting. Elsevier Science & Technology Books, 2010.

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20

Spittell, Peter C. Vascular Diseases. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0728.

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Peripheral vascular diseases, such as aneurysmal disease, intermittent claudication, acute arterial occlusion, and thromboangiitis, are prevalent in current medical practice. Vasospastic disorders, another class of vascular disease, are characterized by episodic color changes of the skin resulting from intermittent spasm of the small arteries and arterioles of the skin and digits. Vasospastic disorders are important because they frequently are a clue to another underlying disorder, such as arterial occlusive disease, connective tissue disorders, neurologic disorders, or endocrine disease. Characteristic clinical features, accurate diagnostic techniques, and improved treatment of peripheral vascular disease further emphasize the need for increased awareness of this group of disorders.

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21

Fu, Bingmei M., and Neil T. Wright. Molecular, Cellular, and Tissue Engineering of the Vascular System. Springer, 2018.

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22

Molecular, Cellular, and Tissue Engineering of the Vascular System. Springer, 2018.

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23

Soutar, David S. Microvascual Surgery & Free Tissue Transfer. Edward Arnold, 1993.

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24

J, Bunt T., ed. Vascular graft infections. Mount Kisco, NY: Futura Pub., 1994.

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25

S, Soutar David, ed. Microvascular surgery and free tissue transfer. Boston: Little, Brown, 1993.

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26

G, Mikos Antonios, and Johnson Peter C, eds. Advances in tissue engineering angiogenesis. New Rochelle, NY: Mary Ann Liebert, Inc., 2010.

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27

Microvascular surgery and free tissue transfer. London: E. Arnold, 1993.

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28

Tissue-Specific Vascular Endothelial Signals and Vector Targeting, Part A. Elsevier, 2009. http://dx.doi.org/10.1016/c2009-0-01783-8.

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29

Tissue-Specific Vascular Endothelial Signals and Vector Targeting, Part B. Elsevier, 2010. http://dx.doi.org/10.1016/c2009-0-01868-6.

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30

Herrington, William G., Aron Chakera, and Christopher A. O’Callaghan. Renal vascular disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0171.

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Renal vascular disease typically occurs with progressive narrowing of the main renal artery or smaller arterial vessels. Often, both patterns of disease coexist and result in ‘ischaemic nephropathy’ with damage to renal tissue. Much less commonly, inflammatory vasculitis can affect small or medium vessels. Ninety per cent of renal vascular disease is caused by atherosclerosis. Patients with renal vascular disease have an increased risk of cardiovascular death from associated cerebrovascular and coronary heart disease. Less than 10% of renal vascular disease is caused by fibromuscular dysplasia. The cause is unknown, but smoking is a risk factor. The disease is often bilateral and multifocal. It tends to affect the mid-portion of the renal artery, while atherosclerosis tends to occur at points of stress, especially at the junction of renal arteries with the aorta. This chapter reviews the diagnosis and management of renal vascular disease.

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31

Jackson, Trachette L. Modeling Tumor Vasculature: Molecular, Cellular, and Tissue Level Aspects and Implications. Springer, 2014.

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32

Covic, Adrian, Mugurel Apetrii, Luminita Voroneanu, and David J. Goldsmith. Vascular calcification. Edited by David J. Goldsmith. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0120_update_001.

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Vascular calcification (VC) is a common feature of patients with advanced CKD and it could be, at least in part, the cause of increased cardiovascular mortality in these patients. From a morphologic point of view, there are at least two types of pathologic calcium phosphate deposition in the arterial wall—namely, intima calcification (mostly associated with atherosclerotic plaques) and media calcification (associated with stiffening of the vasculature, resulting in significantly adverse cardiovascular outcomes). Although VC was viewed initially as a passive phenomenon, it appears to be a cell-mediated, dynamic, and actively regulated process that closely resembles the formation of normal bone tissue, as discovered recently. VC seems to be the result of the dysregulation of the equilibrium between promoters and inhibitors. The determinants are mostly represented by altered calcium and phosphorus metabolism, secondary hyperparathyroidism, vitamin D excess, high fibroblast growth factor 23, and high levels of indoxyl sulphate or leptin; meanwhile, the inhibitors are vitamin K, fetuin A, matrix G1a protein, osteoprotegerin, and pyrophosphate. A number of non-invasive imaging techniques are available to investigate cardiac and vascular calcification: plain X-rays, to identify macroscopic calcifications of the aorta and peripheral arteries; two-dimensional ultrasound for investigating the calcification of carotid arteries, femoral arteries, and aorta; echocardiography, for assessment of valvular calcification; and, of course, computed tomography technologies, which constitute the gold standard for quantification of coronary artery and aorta calcification. All these methods have a series of advantages and limitations. The treatment/ prevention of VC is currently mostly around calcium-mineral bone disease interventions, and unproven. There are interesting hypotheses around vitamin K, Magnesium, sodium thiosulphate and other potential agents.

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33

Yang, Zhihong, and Xiu-Fen Ming. Adventitia and perivascular adipose tissue—the integral unit in vascular disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0020.

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Obesity and obesity-associated metabolic disorders are highly associated with cardiovascular disease. Abnormal ectopic deposition and accumulation of adipose tissue in organs, including perivascular space (perivascular adipose tissue, PVAT) in obesity are emerging to contribute to vascular disease development through pathological paracrine and/or endocrine secretion of cytokines, namely adipokines, which are vasoactive factors including vascular relaxing and contracting factors, smooth muscle growth promoting and inhibiting factors, and pro- and anti-inflammatory factors. In obesity, production of these factors from PVAT is altered and in imbalance which favours vascular contraction, pathological remodelling, and inflammation. In cross-talk with the endothelium, the functional changes of adventitia and PVAT are detrimental and importantly contribute to the acceleration of vascular atherosclerosis and complications associated with obesity and metabolic disorders

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34

Mandley, David John. Laser activated tissue glues for use in laser assisted vascular anastomosis. 1995.

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35

Rivera, Francisco J., and Ludwig Aigner, eds. The Vascular Niche in Tissue Repair: A Therapeutic Target for Regeneration. Frontiers Media SA, 2018. http://dx.doi.org/10.3389/978-2-88945-410-5.

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36

Edith, Grishman, ed. The Kidney in collagen-vascular diseases. New York: Raven Press, 1993.

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37

BIOPATOLOGIA VASCULAR E SANGUÍNEA. VOL 1 (1974-1984): -. Lisbon, Portugal: Lisboa: Publicações Ciência e Vida, 2005.

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38

Brey, Eric M. Vascularization: Regenerative Medicine and Tissue Engineering. Taylor & Francis Group, 2017.

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39

Vascularization: Regenerative Medicine and Tissue Engineering. Taylor & Francis Group, 2014.

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40

(Editor), Stephen C. Cowin, and Jay D. Humphrey (Editor), eds. Cardiovascular Soft Tissue Mechanics. Springer, 2002.

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41

Gordon, J. L. Vascular Endothelium: Interactions With Circulating Cells (Research Monographs in Cell and Tissue Physiology). Elsevier Publishing Company, 1991.

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42

Magnedans, J. Morphogenesis of Primary Vascular Tissue and Regeneration (Agricultural University Wageningen Papers, 88/4). Center Agricultural Pub & Document, 1988.

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43

Angiogenesis. New Rochelle, NY: Mary Ann Liebert, 2010.

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44

G, Mikos Antonios, and Johnson Peter C, eds. Angiogenesis. New Rochelle, NY: Mary Ann Liebert, 2010.

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45

Narouze, Samer N. Cervical Sympathetic Block: Ultrasound. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199908004.003.0028.

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To improve the safety of the stellate ganglion block (SGB), the techniques for SGB have evolved over time from the standard blind technique to fluoroscopy and more recently to ultrasound-guided technique. Ultrasound-guided SGB may also improve the safety of the procedure by direct visualization of vascular structures and soft-tissue structures. Accordingly, the risk of vascular and soft-tissue injury may be minimized. Ultrasound guidance will allow direct monitoring of the spread of the injectate and hence may minimize complications such as recurrent laryngeal nerve (RLN) palsy and intrathecal, epidural, or intravascular spread.

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46

Job, Harenberg, and Heidelberger Akademie der Wissenschaften, eds. New trends in haemostasis: Coagulation proteins, endothelium, and tissue factors. Berlin: Springer-Verlag, 1990.

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47

Bhumbra, Rej S., Panagiotis D. Gikas, Sammy Hanna, Jakub Jagiello, and Stephen R. Cannon. Malignant bone tumours. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.002006.

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48

Bhumbra, Rej S., Panagiotis D. Gikas, Sammy Hanna, Jakub Jagiello, and Stephen R. Cannon. Malignant tumours of soft tissues. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.002004.

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♦ Malignant vascular tumours of soft tissue♦ Synovial sarcoma♦ Malignant peripheral nerve sheath tumours♦ Rhabdomyosarcoma♦ Leiomyosarcoma♦ Epithelioid sarcoma♦ Clear cell sarcoma♦ Malignant fibrous histiocytoma♦ Chordoma.

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49

Saremi, Farhood. Perfusion Imaging in Clinical Practice: A Multimodality Approach to Tissue Perfusion Analysis. LWW, 2015.

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50

Selim, Magdy. Neuroprotection for General, Orthopedic, Peripheral Vascular, and ENT Surgery. Edited by David L. Reich, Stephan Mayer, and Suzan Uysal. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190280253.003.0022.

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Unlike stroke after cardiac and carotid surgery, stroke after general; orthopedic; peripheral vascular; and ear, nose, and throat surgery has not been investigated extensively. The incidence, predisposing factors, and etiological mechanisms of stroke in patients undergoing these procedures are reviewed. Recommendations to prevent, recognize, and treat stroke following these surgical procedures are provided to minimize postoperative stroke risk and its associated morbidity and disability. Although these recommendations can help to decrease the incidence of perioperative stroke, there is an unmet need to find novel and effective neuroprotective strategies that can be used pre- or intraoperatively to minimize the effects of stroke on brain tissue and resulting disability. Future studies should evaluate the potential usefulness of neuroprotective therapies or interventions, including various anesthetic agents that can be used prophylactically in the perioperative setting.

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