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Books on the topic 'Thrombu'

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Published: 14 March 2023

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1

Maragoudakis, Michael E., and Nikos E. Tsopanoglou, eds. Thrombin. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-0-387-09637-7.

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2

Berliner, Lawrence J., ed. Thrombin. Boston, MA: Springer US, 1992. http://dx.doi.org/10.1007/978-1-4615-3296-5.

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3

Tew, David. Throsby revisited. Wymondhanm, Melton Mowbray, Leicestershire: Witmehá Press, 1989.

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4

Heart throbs. London: Octopus Books, 1985.

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5

Heart throbs. London: Xpresso, 1991.

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6

J, Berliner Lawrence, ed. Thrombin: Structure and function. New York: Plenum Press, 1992.

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7

Wang, Lemin. The Origin and Onset of Thrombus Disease. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-7344-1.

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8

A, Greer I., ed. Thrombo-embolic disease in obstetrics and gynaecology. London: Baillière Tindall, 1997.

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9

Summers, Andy. Throb. London: Sidgwick & Jackson, 1986.

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10

Berliner, Lawrence J. Thrombin: Structure and Function. Boston, MA: Springer US, 1992.

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11

Stief, Thomas. Thrombin: Function and pathophysiology. Hauppauge, N.Y: Nova Science Publisher's, 2011.

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12

Throsby, Jonathan. Jonathan Throsby: Recent paintings. London: Fabian Carlsson Gallery, 1988.

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13

A, Walz Daniel, Fenton John W, Shuman Marc A, New York Academy of Sciences., and Conference on Bioregulatory Functions of Thrombin (1986 : New York, N.Y.), eds. Bioregulatory functions of thrombin. New York, N.Y: New York Academy of Sciences, 1986.

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14

Thrills throbs murmurs. New Delhi: Literti Group, 2006.

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15

Elton, Ben. Chart Throb. London: Transworld, 2008.

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16

Chart throb. London: Bantam, 2006.

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17

Bishop, Carly. Heart throb. Richmond: Silhouette Books, 1995.

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18

Heart-Throb. Richmond: Mills & Boon, 2004.

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19

Kamalakēs, Sp Th. Patriarchika tagmata aristeias tou Alexandrinou Thronou. Athēna: Edkoseis Angelakē, 2010.

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20

Claeson, Goran, Michael F. Scully, Vijay V. Kakkar, and John Deadman, eds. The Design of Synthetic Inhibitors of Thrombin. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4899-2418-6.

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21

Giddings, J. C. Thrombin, thrombomodulin and the control of hemostasis. Austin: R.G. Landes, 1994.

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22

Lee, Miranda. Heart-throb for hire. Richmond: Mills & Boon, 1993.

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23

Sheares, Karen, and Joanna Pepke-Zaba. Venous thrombosis and pulmonary embolism. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0101.

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Venous thromboembolism (VTE) is a condition in which thrombus forms in a vein, commonly in the deep veins of the leg, termed deep-vein thrombosis (DVT). The thrombus may dislodge from the site of origin and be carried into the pulmonary vasculature, causing a pulmonary embolism (PE). Deep vein thrombosis and pulmonary embolism share similar predisposing factors; however, mortality is greater in those who present with PE than in those who present with DVT. Thrombi may form in other parts of the vasculature.
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24

Cardiovascular Thrombus. Elsevier, 2018. http://dx.doi.org/10.1016/c2016-0-03582-9.

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25

Thrombus and stroke. New York: Informa Healthcare, 2008.

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26

Wakhloo, Ajay K., Matthew J. Gounis, Baruch B. Lieber, Robert A. Mericle, and Italo Linfante, eds. Thrombus and Stroke. CRC Press, 2008. http://dx.doi.org/10.3109/9781420020830.

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27

Linfante, Italo, Ajay K. Wakhloo, Matthew J. Gounis, Baruch B. Lieber, and Robert A. Mericle. Thrombus and Stroke. Taylor & Francis Group, 2008.

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28

Linfante, Italo, Ajay K. Wakhloo, Matthew J. Gounis, Baruch B. Lieber, and Robert A. Mericle. Thrombus and Stroke. Taylor & Francis Group, 2008.

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29

Thrombo And Other Plays. Performing Books, 2012.

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30

Patenaude, Jean-Victor, Sylvie Desmarais, and Université de Montréal Faculté de médecine. Les maladies thrombo-emboliques veineuses. Presses universitaires de Montréal, 1998.

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31

Hirsh, Jack, and Robert J. Ginsberg. Management Guidelines in Venous Thrombo. 2nd ed. BC Decker Inc., 1998.

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32

Benyounes, Nadia, Mauro Pepi, Roberta Esposito, Carmen Ginghina, and Ariel Cohen. Cardiac masses and potential sources of emboli. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0051.

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Cardiac masses are abnormal structures within or immediately adjacent to the heart. They have to be distinguished from variants of normal cardiac structures, postoperative changes, and ultrasound artefacts. These abnormal masses may be localized in the left or right heart cavities, with different clinical manifestations according to their localization. Among the abnormal cardiac masses (thrombus, vegetations, tumours), tumours are not discussed in this chapter. Echocardiography is the main but not the only imaging technique for the evaluation of cardiac masses, and is largely available. Hence, it is indicated in patients with a systemic embolic event, searching for a cardiac source embolism. When transthoracic echocardiography is negative, transoesophageal echocardiography is indicated, in cryptogenic ischaemic events (no cause found). Right heart masses are mainly responsible for pulmonary embolisms, but may be the cause of a systemic embolus, via the atrial septum. Right heart thrombi rarely form in situ, and are hence more often venous thrombi entrapped in the right heart on their way to the pulmonary arteries. Echocardiography is mandatory in the setting of pulmonary embolism.
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33

K, Das S., and V. K. Kapoor. to Z of Venous Thrombo-Embolism. Taylor & Francis Group, 2021.

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34

K, Das S., and V. K. Kapoor. to Z of Venous Thrombo-Embolism. Taylor & Francis Group, 2021.

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35

Heart Throbs. Vantage Pr, 2004.

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36

Publishing, RH Value. Heart Throbs. Random House Value Publishing, 1988.

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37

Floria, Marge. Heart Throbs. Balboa Press, 2017.

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38

Floria, Marge. Heart Throbs. Balboa Press, 2017.

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39

(Editor), Ajay K. Wakhloo, Matthew J. Gounis (Editor), Baruch B. Lieber (Editor), Robert A. Mericle (Editor), and Italo Linfante (Editor), eds. Thrombus and Stroke (Neurological Disease and Therapy). Informa Healthcare, 2008.

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40

Álvarez, Alberto Pozo. Origin of Thrombi in the Left Atrium. Independently Published, 2020.

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41

Keeland, Vi, and Molly Glenmore Sebastian York. Throb. Audible Studios on Brilliance Audio, 2016.

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42

Keeland, Vi. Throb. C. Scott Publishing Corp, 2019.

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43

Keeland, Vi. Throb. C. Scott Publishing Corp., 2015.

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44

Keeland, Vi. Throb. CreateSpace Independent Publishing Platform, 2015.

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45

Keeland, Vi. Throb. C. Scott Publishing Corp., 2015.

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46

Keeland, Vi. Throb. Diversion Publishing Corp., 2015.

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47

Throb. Inkblot Pubns, 1988.

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48

Morman, Vanessa. Throb. Ladies of Caliber Publishing, 2009.

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49

Sinnaeve, Peter, and Frans Van de Werf. Fibrinolytic, antithrombotic, and antiplatelet drugs in acute coronary syndromes. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0044.

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Antithrombotic therapy is a major cornerstone in the treatment for acute coronary syndromes, as thrombus formation upon a plaque rupture or an erosion plays a pivotal role in non-ST-segment elevation as well as ST-segment elevation acute coronary syndromes. Both acute and long-term oral antiplatelet therapies, targeting specific platelet activation pathways, have demonstrated significant short- and long-term benefits. The use of anticoagulants is currently largely confined to the acute setting, except in patients with a clear indication for long-term treatment, including atrial fibrillation or the presence of intraventricular thrombi. Despite the benefit of primary percutaneous coronary intervention in ST-segment elevation myocardial infarction, fibrinolysis continues to play an important role throughout the world as well. In this chapter, the fibrinolytic, antiplatelet, and anticoagulant agents used in the management of acute coronary syndrome patients are discussed.
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50

Sinnaeve, Peter, and Frans Van de Werf. Fibrinolytic, antithrombotic, and antiplatelet drugs in acute coronary syndromes. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0044_update_001.

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Antithrombotic therapy is a major cornerstone in the treatment for acute coronary syndromes, as thrombus formation upon a plaque rupture or an erosion plays a pivotal role in non-ST-segment elevation as well as ST-segment elevation acute coronary syndromes. Both acute and long-term oral antiplatelet therapies, targeting specific platelet activation pathways, have demonstrated significant short- and long-term benefits. The use of anticoagulants is currently largely confined to the acute setting, except in patients with a clear indication for long-term treatment, including atrial fibrillation or the presence of intraventricular thrombi. Despite the benefit of primary percutaneous coronary intervention in ST-segment elevation myocardial infarction, fibrinolysis continues to play an important role throughout the world as well. In this chapter, the fibrinolytic, antiplatelet, and anticoagulant agents used in the management of acute coronary syndrome patients are discussed.
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