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Books on the topic 'Thrombophlebitis'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 August 2024

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1

Medicine, Society of Hospital. Preventing hospital-acquired venous thromboembolism: A guide for effective quality improvement. Rockville, MD: Agency for Healthcare Research and Quality, U.S. Dept. of Health and Human Services, 2008.

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Medicine, Society of Hospital. Preventing hospital-acquired venous thromboembolism: A guide for effective quality improvement. Rockville, MD: Agency for Healthcare Research and Quality, U.S. Dept. of Health and Human Services, 2008.

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3

Medicine, Society of Hospital. Preventing hospital-acquired venous thromboembolism: A guide for effective quality improvement. Rockville, MD: Agency for Healthcare Research and Quality, U.S. Dept. of Health and Human Services, 2008.

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4

Fulkerson, William J., and Herbert A. Saltzman. Venous thromboembolism. Philadelphia: Saunders, 1995.

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5

Great Britain. Dept. of Health., ed. Report of the independent expert working group on the prevention of venous thromboemlism in hospitalised patients. London: The Dept., 2005.

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6

Ogston, Derek. Venous thrombosis: Causation and prediction. Chichester: Wiley, 1987.

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7

Malone, P. Colm. The aetiology of deep venous thrombosis: A critical, historical and epistemological survey. Dordrecht: Springer, 2008.

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8

National Library of Medicine (U.S.), ed. Prevention of venous thrombosis and pulmonary embolism: January 1984 through January 1986, 250 citations in English. [Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health], 1986.

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9

Hirsh, Jack. Venous thromboembolism: Guide to management. Mississauga, Ont: Du Pont Pharmaceuticals, 1986.

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10

WHO Research into Global Hazards of Travel (Wright). WHO Research into Global Hazards of Travel (Wright) Project: Final report of phase I. Geneva: World Health Organization, 2007.

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11

Russell, Hull, ed. Venous thromboembolism: Natural history, diagnosis, and management. Boca Raton, Fla: CRC Press, 1987.

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12

Volovik, Mihail, I. Dolgov, and Natal'ya Muravina. Thermal imaging screening and diagnostics. Diseases of the circulatory system. Varicose veins of the lower extremities. Phlebitis. Thrombophlebitis. ru: INFRA-M Academic Publishing LLC., 2020. http://dx.doi.org/10.12737/1159602.

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13

I, Schafer Andrew, ed. Molecular mechanisms of hypercoagulable states. New York: Chapman & Hall, 1997.

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14

Patenaude, Jean-Victor. Les maladies thrombo-emboliques veineuses: Module d'auto-apprentissage : les thrombophlébites superficielles et profondes, les embolies pulmonaires. 2nd ed. Montréal: Presses de l'Université de Montréal, 1998.

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15

Greer, I. A. Venous thrombosis in women: Pregnancy, the contraceptive pill, and hormone replacement therapy / I.A. Greer. Boca Raton: Parthenon Pub., 2003.

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16

Saha, Sudip. Septic Thrombophlebitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0021.

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Septic (suppurative) thrombophlebitis is venous thrombosis in the setting of bacteremia. There is usually a degree of perivascular inflammation seen on histology. Septic thrombophlebitis occurs most commonly with intravenous catheters. However, most cases of infection related to intravenous catheters are not complicated by septic thrombophlebitis. Catheter-related septic thrombophlebitis includes erythema, tenderness, and/or drainage at the site of an intravenous catheter. Jugular vein septic thrombophlebitis, also known as Lemierre’s syndrome, is a subset of septic thrombophlebitis. This condition can affect otherwise young, healthy adults and is often preceded by pharyngitis with tonsillar and peritonsillar involvement, dental infections, or infectious mononucleosis. Presentation of jugular vein septic thrombophlebitis includes high fevers, rigors, respiratory distress, ulceration or erythema of the oropharynx, and tenderness and swelling of the neck. Primary treatment of thrombophlebitis includes removal of infected materials, intravenous antibiotics, and possible anticoagulation.

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17

Stein, Matthew A. Thrombophlebitis (Mondor Disease). Edited by Christoph I. Lee, Constance D. Lehman, and Lawrence W. Bassett. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190270261.003.0052.

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This chapter, appearing in the section on nipple, skin, and lymph nodes, reviews the key imaging and clinical features, imaging protocols and pitfalls, differential diagnosis, and clinical recommendations of superficial thrombophlebitis of the breast and anterior chest wall (Mondor disease). The condition is quite rare; incidence rates in the literature are reported as being less than 0.1%. Mondor disease is often temporally associated with a history of recent breast surgery, core needle biopsy, inflammatory process, or episode of trauma. Topics in this chapter include discussions of the incidence of Mondor disease, its presumptive pathophysiology, typical and atypical clinical/imaging presentations, and management considerations.

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18

Shirato, Kunio. Venous Thromboembolism: Prevention and Treatment. Springer London, Limited, 2006.

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19

Piazza, Gregory, Benjamin Hohlfelder, and Samuel Z. Goldhaber. Handbook for Venous Thromboembolism. Springer, 2015.

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20

Piazza, Gregory, Benjamin Hohlfelder, and Samuel Z. Goldhaber. Handbook for Venous Thromboembolism. Springer London, Limited, 2015.

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21

Comerota, Anthony J. Practical Phlebology: Deep Vein Thrombosis. Taylor & Francis Group, 2013.

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22

Comerota, Anthony J. Practical Phlebology: Deep Vein Thrombosis. Taylor & Francis Group, 2014.

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23

Comerota, Anthony J. Practical Phlebology: Deep Vein Thrombosis. Taylor & Francis Group, 2013.

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24

Venous thrombosis in heart-disease. [S.l: s.n., 1985.

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25

Wood, Kenneth E. Venous Thromboembolism in Critical Care. Elsevier - Health Sciences Division, 2012.

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26

The Surgeon General's call to action to prevent deep vein thrombosis and pulmonary embolism. [Rockville, MD]: U.S. Public Health Service, [Office of the Surgeon General], 2008.

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27

Ogston, Derek. Venous Thrombosis: Causation and Prediction (Wiley Medical Publications). John Wiley & Sons, 1988.

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28

Deep Vein Thrombosis. Taylor & Francis Group, 2013.

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29

Diagnosis and treatment of deep venous thrombosis and pulmonary embolism. Rockville, Md.]: Agency for Healthcare Research and Quality, 2003.

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30

Laurie G. Jacobs MD FACP ACSF. Thromboembolic Disease and Antithrombotic Agents in the Elderly: An Issue of Cardiology Clinics. Saunders, 2008.

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31

Shirato, K. Venous Thromboembolism: Prevention and Treatment. Springer, 2004.

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32

Agutter, Paul S., and P. Colm Malone. Aetiology of Deep Venous Thrombosis: A Critical, Historical and Epistemological Survey. Springer, 2010.

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33

Agutter, Paul S., and P. Colm Malone. The Aetiology of Deep Venous Thrombosis. Springer, 2008.

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34

Doumouchtsis, Stergios K., S. Arulkumaran, Stergios K. Doumouchtsis, and Austin Ugwumadu. Sepsis in pregnancy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199651382.003.0007.

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This chapter discusses sepsis in pregnancy, and outlines the causes, risk factors, symptoms, investigations, and management of maternal sepsis and postpartum infections such as endometritis, septic pelvic thrombophlebitis, urinary tract infections, mastitis and breast abscess, and wound and episiotomy infection.

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35

James, Andra H. 100 Q&A About Deep Vein Thrombosis and Pulmonary Embolism (100 Questions & Answers about . . .) (100 Questions & Answers). Jones and Bartlett Publishers, Inc., 2007.

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36

Khorana, Alok A. Cancer-associated Thrombosis: New Findings in Translational Science, Prevention, and Treatment. Informa Healthcare, 2007.

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37

Deep vein thrombosis and pulmonary embolism. Chichester, West Sussex: J. Wiley, 2009.

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38

Venous Thromboembolism: An Evidence-Based Atlas. Futura Publishing Company, 1996.

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39

Hull, Russell. Venous Thromboembolism. Wiley & Sons, Incorporated, John, 1995.

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40

Segal, Jodi B. Vaginal Birth After Cesarean (Vbac) (Evidence Report/Technology Assessment). Dept. of Health and Human Services Ncy for He, 2003.

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41

Sharnoff, J. G. Prevention of Venous Thrombosis and Pulmonary Embolism. Springer London, Limited, 2012.

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42

Sharnoff, J. G. Prevention of Venous Thrombosis and Pulmonary Embolism. Springer, 2011.

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43

News, PM Medical Health. 21st Century Complete Medical Guide to Thrombophlebitis, Deep Vein Thrombosis (DVT), and Pulmonary Embolism, Authoritative Government Documents, Clinical ... Information for Patients and Physicians. Progressive Management, 2004.

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44

Venous Thromboembolism. New York: Marcel Dekker, Inc., 2003.

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45

Patenaude, Jean-Victor, Sylvie Desmarais, and Université de Montréal Faculté de médecine. Les maladies thrombo-emboliques veineuses. Presses universitaires de Montréal, 1998.

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46

Yurdakul, Sebahattin, Emire Seyahi, and Hasan Yazici. Behçet’s syndrome. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0135.

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Behçet's syndrome is a systemic inflammatory panvasculitis (affecting all sizes of vessels) of unknown aetiology. It is in vogue to include it among the systemic autoinflammatory conditions. Behçet's syndrome is more frequent along the ancient 'Silk Route' across Asia than it is in Western countries. The usual onset is the second or third decade, equally affecting either gender. However, young patients and male patients have more severe disease. Almost all patients have recurrent oral ulceration. Scar-forming genital ulcers, a variety of skin lesions including acneiform, erythema nodosum-like lesions, arthritis, potentially blinding panuveitis, thrombophlebitis, gastrointestinal disease, central nervous system (CNS) involvement, and life-threatening bleeding pulmonary artery aneurysms are seen. The pathergy phenomenon is a heightened tissue inflammatory response. The strongest genetic association is with HLA B51. There are immunological aberrations but not prominent enough to call it an autoimmune disease. Similarly, Behçet's syndrome does not fit easily into the broad concept of autoinflammatory diseases. The histopathology is also non-specific and the diagnosis is mainly clinical. Differentiation from Crohn's disease is very difficult. In more than one-half of the patients the disease burns out in time, thus only symptomatic therapy is indicated in some patients. However, eye involvement, pulmonary vascular disease, thrombophilic complications, CNS involvement, and gastrointestinal disease need prompt recognition and treatment. Brief courses of glucocorticosteroids along with immunosuppressives including the newer biologicals, interferon, and colchicine are commonly used. However, controlled clinical trials are not available for some of these medications especially when thrombophilia, CNS, and gastrointestinal disease are present.

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