Relevant bibliographies by topics / Thermoregulatory responses

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  2. Dissertations / Theses
  3. Books
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Academic literature on the topic 'Thermoregulatory responses'

Author: Grafiati
Published: 4 June 2021
Last updated: 6 February 2022

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Journal articles on the topic "Thermoregulatory responses"

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Robinson, B. J., R. H. Johnson, D. G. Lambie, and E. A. Whiteside. "Thermoregulatory Responses in Alcoholism." Australian Alcohol/Drug Review 4, no. 2 (July 1985): 157–59. http://dx.doi.org/10.1080/09595238580000231.

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Lovegrove, Barry G. "Seasonal thermoregulatory responses in mammals." Journal of Comparative Physiology B 175, no. 4 (March 8, 2005): 231–47. http://dx.doi.org/10.1007/s00360-005-0477-1.

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Gordon, N., H. Russell, P. Krüger, and J. Cilliers. "Thermoregulatory Responses to Weight Training." International Journal of Sports Medicine 06, no. 03 (June 1985): 145–50. http://dx.doi.org/10.1055/s-2008-1025828.

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Brown-Brandl, T. M., J. A. Nienaber, R. A. Eigenberg, G. L. Hahn, and H. Freetly. "Thermoregulatory responses of feeder cattle." Journal of Thermal Biology 28, no. 2 (February 2003): 149–57. http://dx.doi.org/10.1016/s0306-4565(02)00052-9.

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Shalaby, T. H., M. K. Yousef, and R. K. Dupré. "Thermoregulatory responses of diabetic rats." Comparative Biochemistry and Physiology Part A: Physiology 94, no. 1 (January 1989): 153–57. http://dx.doi.org/10.1016/0300-9629(89)90800-1.

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Castellani, John W. "Cold thermoregulatory responses following exertional fatigue." Frontiers in Bioscience S2, no. 3 (2010): 854–65. http://dx.doi.org/10.2741/s106.

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YOUSEF, Mohamed K., Sueko SAGAWA, and Keizo SHIRAKI. "Thermoregulatory Responses of the Elderly Population." Journal of UOEH 8, no. 2 (1986): 219–27. http://dx.doi.org/10.7888/juoeh.8.219.

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Morante, S. M., and J. R. Brotherhood. "Thermoregulatory responses during competitive singles tennis." British Journal of Sports Medicine 42, no. 9 (May 7, 2008): 736–41. http://dx.doi.org/10.1136/bjsm.2007.037002.

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Adair, Eleanor R., and David R. Black. "Thermoregulatory responses to RF energy absorption." Bioelectromagnetics 24, S6 (2003): S17—S38. http://dx.doi.org/10.1002/bem.10133.

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Passias, T. C., G. S. Meneilly, and I. B. Mekjavic. "Effect of hypoglycemia on thermoregulatory responses." Journal of Applied Physiology 80, no. 3 (March 1, 1996): 1021–32. http://dx.doi.org/10.1152/jappl.1996.80.3.1021.

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The effects of hypoglycemia on sweating, skin blood perfusion, and shivering responses were investigated in 10 healthy male volunteers. They exercised on an underwater cycle ergometer while immersed to the neck in 28 degrees C water for 20 min at 50% of their maximal work rate. The exercise-induced elevation in esophageal temperature (T(es)) initiated the sweating response (Esw) and increased skin blood perfusion (SkBP) as measured at the forehead. In the 99-min postexercise immersion period, the values of T es relative to resting level (delta T(es)) at which Esw abated, SkBP reached preexercise values, and shivering commenced were defined as the delta T(es) thresholds for cessation of sweating, passive vasodilation, and onset of shivering, respectively. Two trials were conducted 1 wk apart. The subject was hypoglycemic in one trial and euglycemic in the other (plasma glucose was maintained at 2.8 and 5 mM, respectively) with the use of the hyperinsulinemic (insulin infusion rate = 60 mU.m-2.min-1) glucose-clamp technique. Oxygen uptake, Esw, T(es), mean skin temperature, heat flux from the skin, and SkBP were recorded at minute intervals. Although heat flux and SkBP attained significantly higher end-exercise levels during euglycemia, the responses were similar during the postexercise cooling period. Hypoglycemia did not affect the Esw response during the exercise and cooling periods. Whereas the exercise delta T(es) response was unaffected by hypoglycemia, the decrease in T(es) was greater (P < or = 0.005) during the hypoglycemic than during the euglycemic condition. Hypoglycemia did not alter the delta T(es) threshold for cessation of sweating and passive vasodilation but reduced (P < or = 0.001) the delta T(es) threshold for onset of shivering (from -0.09 +/- 0.07 degrees C in the euglycemic condition to -0.65 +/- 0.12 degrees C in the hypoglycemic condition). The present results indicate that hypoglycemia (2.8 mM) does not affect the delta T(es) threshold for cessation of thermoregulatory sweating or the threshold for passive vasodilation during recovery from exercise-induced moderate heat stress but that it decreases the core temperature threshold for shivering during cold exposure.
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Dissertations / Theses on the topic "Thermoregulatory responses"

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Meir, Rudi A., and mikewood@deakin edu au. "The Effect of jersey type on thermoregulatory responses during exercise in a warm humid environment." Deakin University. School of Education, 1992. http://tux.lib.deakin.edu.au./adt-VDU/public/adt-VDU20050915.132750.

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The thermoregulatory responses of subjects wearing two different forms of rugby league jersey, one with plastic sponsorship recognition and numbering (trial Gl) and one without (trial G2), and a lightweight alternative (trial G3), were compared with a trial without any form of upper body garment (trial GO). Ten male volunteers, mean age 20.9 (±2.3) years, height 179.8 (±4.7) cm, weight 80.2 (±8.9) kg, and body surface area 1.99 (±0.13) m2, participated in this study. Subjects had a mean maximal oxygen uptake capacity of 56.0 (±6.3) ml.kg.min-1 and a sum of 8 skinfolds of 80.6 (±23.8) mm. Subjects were exercised at approximately 50% of maximal oxygen uptake in a warm humid environment for 50 minutes. Mean ambient temperature was 27.6°C (±0.32) with a relative humidity of 64.7% (±1.44). Measurements of core and skin (7 sites) temperature, heart rate, oxygen uptake, plasma volume, peak lactate concentration, and pre- and post-trial body weight, hematocrit and garment weight were recorded. The statistical results showed that all subjects experienced significant (p ≤.0001) decreases in body weight representing a percentage decrease ranging from 1.2-1.3%. No significant difference was found between trials with respect to body weight change. No significant effect of garment type was found on pre- and post-trial hematocrit, plasma volume changes or peak blood lactic acid concentration. However, mean peak lactate was highest for trial Gl (5.6 mmol.L-1 ±2.2) and lowest for trial G3 (4.6 mmol.L-1 ±1.27). Post-trial core temperature was significantly (p≤ .0001) higher than the resting value; no significant difference was found between trials. The mean absolute increase for all experimental trials was 0.9°C. A significant (p≤.005) difference between mean total (7 sites) skin temperature was found with a post-hoc test revealing that trials Gl and G2 were significantly higher than trial GO; no significant difference was found when comparing trial G3 with trial GO or when comparing the garments between each other. Mean skin temperature under the garment (4 sites) was found to be significantly (p≤.05) higher for all trials involving a garment when compared with mean skin temperature outside (3 sites) the garment; no significant difference was found between trials. Mean oxygen uptake was significantly different between trials (p≤.005), with trial Gl and G3 found to be significantly lower than trial GO; no difference was found when comparing the garments with each other. Post-trial garment weights were significantly (p≤.001) heavier than pre-trial and were significantly (p≤.0001) different when compared with each other. There was no significant effect on heart rate, haematocrit, plasma volume changes, peak blood lactic acid concentration, or core temperature due to garment type. However, differences in skin temperature suggest that the garment used in trial G3 may have a benefit. Further research should consider the impact of increased exercise intensity and/or environmental temperature and humidity on the measured parameters while wearing the garments described in this study.
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Bottoms, Lindsay. "Thermoregulatory Responses during Upper Body Exercise, Thermal Stress, Training and Heat Acclimation." Thesis, Coventry University, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.487373.

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The area of thermoregulation during upper body exercise has received limited research in able bodied individuals. The series of studies presented in this thesis investigated the effect of exercise intensity, duration and environmental temperature on thermoregulatory responses, including calf volume and blood flow, during upper body exercise and recovery. In order to manipulate these variables and observe the adaptive thermoregulatory responses, upper body training and heat acclimation were also performed. Chapter 4 examined the effect of exercise intensity (45, 60, 75· and 90% peak power; Wpeak) on thermoregulatory responses during 5 min of upper body exercise. The results of this study suggest a redistribution of blood from the relatively'inactive lower body during arm exercise of intensities up to 60%Wpeak after which point calf volume did not 'I significantly decrease further. The calf volume decrease is possibly a result of vasoconstriction reducing blood pooling in the leg. Chapter 5 examined the effects of exercise duration (15,30, arid 45 min at 60%Wpeak) on thermoregulatory responses during and after upper body exercise. During upper body exercise at 60% Wpeak calf volume decreased up to 15 min with no further decrease thereafter. In all trials calf blood flow was greater at the cessation of exercise compared to rest suggesting hyperaemia occurred at the end of exercise. Chapter ~ examined the effects of exercise (30 min, 60%Wpeak) in different environmental temperatures (21, 27 ~nd 33°C) on thermoregulatory responses. Calf skin blood flow from Laser Doppler measurements increased in all trials with a concomitant decrease in calf volume. The decrease in the calf volume..reported therefore reflected a greater and more substantial muscle vasoconstriction compared to increased skin blood flow. There was a greater decrease in calf volume during the 27°C trial which appears to be, a result of a lower skin blood flow response compared to exercising in 33°C. Chapter 7 examined the effect of 8 weeks of upper body training on thermoregulatory responses during upper body exercise. Upper body training reduced aural temperature and heat storage at a given power output as a result of increased whole body sweating and heat flow. Upper body training produced a smaller calf volume change after training at the same absolute exercise intensity demonstrating less leg vasoconstriction which was possibly as a result of a reduced response to sympathetic nervous activity or the fact that exercise was performed overall at a lower intensity post training (47% vs. 60%Wpeak). Chapter 8 examined the effect cSf exercising (30 min, 60%Wpeak) evrpry day in the h~at for 7 . '. days on'thermoregulatory responses to upper body exercise in the heat. There was reduced core temperature during exercise. The calf volume decrease was significantly greater during exercise in the heat following heat acclimation which may be a .result of increased vasoconstriction compensating, for an increase in skin blood flow to reduce venous pooling and therefore to maintain both stroke volume and blood flow to the skin. The lower body appears to have an important role in both cardiovascular stability as well as thermoregulation during upper body exercise.. It is proposed that this is achieved by reducing venous pooling in the calf thus increasing the availability of blood for maintaining stroke volume and increasing skin blood flow during exercise. The responses at the calf during exercise can be adapted through heat acclimation and upper body training.
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Griggs, Katharine E. "Thermoregulatory responses of athletes with a spinal cord injury during rest and exercise." Thesis, Loughborough University, 2017. https://dspace.lboro.ac.uk/2134/24903.

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Following on from Rio de Janeiro 2016, the Tokyo 2020 Paralympic Games will present a unique challenge for athletes, needing to prepare and adapt to the potential challenging environmental conditions of 20-27°C and ~73% relative humidity. It is well known that during exercise in hot and/or humid climates, able-bodied athletes experience an increase in thermal strain and a reduction in performance compared to cooler/drier conditions. Yet these conditions prove even more problematic for athletes, who as a consequence of their impairment have a dysfunctional thermoregulatory system, such as athletes with a spinal cord injury (SCI). To date, the thermoregulatory responses of athletes with an SCI have been an under-studied area of research. To gain a greater understanding of how heat balance is altered in individuals with an SCI and the thermoregulatory consequences as a result, studies need to first be conducted at rest, removing the additional metabolic heat production from exercise. Although a large majority of athletes with an SCI compete indoors in wheelchair court sports (e.g. wheelchair basketball and rugby), exercising even in these climate-controlled environments has been shown to place these athletes under considerable thermal strain. In light of this, it is remarkable that existing research on the thermoregulatory responses of athletes with an SCI during exercise is scarce, especially studies encompassing real-world sporting environments. Athletes with high level lesions (tetraplegia, TP) are a particularly under-studied population group shown to have a greater thermoregulatory impairment than individuals with low level lesions (paraplegia, PA) during continuous exercise. Thus the aim of this thesis was to investigate the thermoregulatory responses of athletes with an SCI at rest and during real-world sporting scenarios, with specific focus on athletes with TP. Study 1 aimed to determine how evaporative heat loss is altered, as a result of an SCI, compared to the able-bodied (AB), and the effect lesion level has on this response. The results provide evidence that in individuals with TP, even at rest, evaporative heat loss is not large enough to balance the heat load, when evaporation is the primary source of heat dissipation. Even though in individuals with PA Tgi increased by a smaller magnitude and they possessed a greater sweating capacity than individuals with TP, at ambient temperatures above Tsk latent heat loss is insufficient to attain heat balance, compared to the AB. To investigate the thermoregulatory responses of athletes with an SCI during real-world sporting scenarios Study 2 examined athletes with TP compared to athletes with PA during 60 min of intermittent sprint wheelchair exercise on a wheelchair ergometer. The study was conducted in conditions representative of an indoor playing environment for wheelchair rugby and basketball (~21°C, 40% relative humidity). Results demonstrated that, despite similar external work, athletes with TP were under greater thermal strain than athletes with PA. Study 3 s novel approach investigated both physiological responses and activity profiles of wheelchair rugby players during competitive match play. Despite players with TP covering 17% less distance and pushing on average 10% slower, they were under a greater amount of thermal strain than players with non-spinal related physical impairments (NON-SCI). Furthermore, this study demonstrated that players with TP that had a larger body mass, larger lean mass, covered a greater relative distance and/or were a higher point player had a greater end Tgi. These data provide an insight for coaches and support staff regarding which players may need greater attention in regards to cooling strategies or breaks in play. The effectiveness of cooling practices currently employed by athletes with TP has not been previously investigated. Study 4 determined the effectiveness of pre-cooling, using an ice vest alone and in combination with water sprays between quarters, at attenuating thermal strain in athletes with TP. Using the activity profile data from Study 3, an intermittent sprint protocol, conducted on a wheelchair ergometer, was used to represent a wheelchair rugby match. The combination of cooling methods lowered Tgi and Tsk to a greater extent than pre-cooling only, despite neither cooling condition having a positive or negative effect on performance. Unexpectedly, the pre-cooling only condition lowered Tgi, compared to no cooling, throughout the subsequent exercise protocol, even though the reduction in Tsk was not long lasting. This thesis provides comprehensive evidence that athletes with TP experience heightened thermal strain during both rest and real-world sporting scenarios compared to the AB, athletes with PA, and within the sport of wheelchair rugby. Athletes with TP should employ practices, such as appropriate cooling methods or alter playing tactics to reduce thermal strain and the likelihood of attaining a heat related injury.
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Dervis, Sheila. "The Independent Influence of Large Differences in Adiposity on Thermoregulatory Responses during Exercise." Thesis, Université d'Ottawa / University of Ottawa, 2014. http://hdl.handle.net/10393/31216.

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Currently no previous study has isolated the independent influence of body fat (BF) on thermoregulatory responses from the confounding biophysical factors of body mass and metabolic heat production (Hprod). Therefore, seven lean (L, BF:10.7 ± 4.1%) and seven non-lean (NL, BF:32.2 ± 6.4%) males matched for total body mass (TBM, L: 87.8 ± 8.5 kg, NL: 89.4 ± 7.8 kg; P= 0.73), cycled for 60 min in a 28.2 ±0.2˚C and 27 ± 10% RH room at i) a Hprod of 546 W; and ii) a Hprod of 7.5 W·kg lean body mass (LBM). Rectal (Tre) and esophageal (Tes) temperatures, and local sweat rate (LSR) were measured continuously; while whole body sweat loss (WBSL) was measured from 0-60 mins. At 546 W, changes in Tre (L: 0.74 ± 0.16ºC, NL: 0.83 ± 0.14ºC), mean local sweat rate (MLSR) based on an average of upper-back and forearm local sweat rates (L: 0.65 ± 0.25, NL: 0.59±0.12 mgcm-2min-1) and WBSL (L: 568 ± 28 mL, NL: 567 ± 29 mL) were similar (P>0.58). At 7.5 W·kg LBM, the L group had greater changes in Tre (L: 0.87 ± 0.16ºC, NL: 0.55 ± 0.11ºC), MLSR (L: 0.83 ±0 .38, NL: 0.41 ± 0.13 mgcm-2min-1) and WBSL (L: 638 ± 19 mL, NL: 399 ± 17 mL) (P<0.05). In conclusion, i) body fat does not independently alter thermoregulatory responses during exercise; ii) core temperature comparisons between groups differing in BF should be performed using a Hprod normalized for TBM, not LBM.
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Saiphon, Kongkum. "The effect of the circadian and menstrual cycles on cardiovascular and thermoregulatory responses to exercise." Thesis, Liverpool John Moores University, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572050.

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Many physiological variables exhibit circadian rhythmicity. The circadian rhythm in core temperature is a well-established and it has been extensively studied during both passive and exercise heat exposure. In females, a circamensal rhythm in core temperature is also present and well established. However, there is little knowledge about whether there is an interaction effect between time of day and phase of menstrual cycle on core temperature and thermoregulatory and cardiovascular responses during and following exercise. The studies in the present thesis were designed to investigate such an interaction effect on the effector responses of the thermoregulation and cardiovascular systems during the exercise and post-exercise periods. The first experiment was designed to examine the time of day effect on thermoregulatory and cardiovascular responses during and following exercise in female subjects. Eight healthy participants completed 30-min exercise at 65%Y02peak at 07:00 and 19:00hr. Core temperature was significantly higher by 0.3, 0.4 and 0.3DC at rest (P=O.OOI), during the exercise (P=O.OOI) and post-exercise (P=0.008) periods in the evening compared to the morning. The second experiment was designed to examine the phases of menstrual cycle on thermoregulatory and cardiovascular responses during and following exercise. Ten healthy participants completed two exercise protocols (65%Y02peak) during the late follicular and luteal phases of the menstrual cycle (day 10-12 and day 20-22 after the onset of menstruation, respectively). Core temperature tended to be higher in the luteal phase compared with the late follicular phase (0.2DC) both at rest (p=O.064) and during exercise (p=0.062), whereas the heat-loss mechanisms were unaffected by menstrual cycle phase. In addition, resting stroke volume and cardiac output was greater in the late follicular phase compared to the luteal phase. The third experiment was designed to explore the interaction effect between time of day and phase of the menstrual cycle on thermoregulatory and cardiovascular responses during the exercise and the post-exercise periods. Ten healthy participants completed four exercise protocol (65% Y02peak); two exercise protocols in the morning of the late follicular and luteal phases, and two exercise protocols in the evening of the late follicular and luteal phases. Core temperature was higher in the evening of both phases of the menstrual cycle during exercise (p=O.OOI) and the post-exercise periods (p=O.003). There was an interaction effect between times of day and phase of the menstrual cycle on mean skin temperature during the exercise (p=0.048) and the post-exercise periods (p=0.006), a lower mean skin temperature in the evening compared with the morning during the late follicular phase and higher in the evening than in the morning in the luteal phase. However, there was no interaction of times of day and phase of menstrual cycle for other thermoregulatory and cardiovascular parameters measured. The results in the thesis indicate that temperature regulation is set around higher values in the evening and late luteal phase of the cycle, but that these changes are likely independent of each other. Future work, should more systematically investigate these responses, collecting data at more times of day and phases of the cycle.
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Mawhinney, C. "The influence of cold-water immersion on limb blood flow and thermoregulatory responses to exercise." Thesis, Liverpool John Moores University, 2016. http://researchonline.ljmu.ac.uk/4709/.

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The accumulated stresses of training and competition may temporarily cause impairments in an athlete’s physiological and muscular function, leading to suboptimal performance levels. Cold-water immersion (CWI) has become a widely used post-exercise recovery method to accelerate the recovery process by purportedly reducing the symptoms associated with exercise-induced muscle damage (EIMD). However, the underlying physiological mechanisms, which mediate the effects of CWI, are not well understood. Therefore, the aim of this thesis was to investigate the influence of cold-water immersion (CWI) on limb blood flow and thermoregulatory responses following different modes of exercise. In study 1 (Chapter 4), the reliability of Doppler ultrasound in the assessment of superficial femoral artery blood flow (FABF) was examined under resting conditions. A Doppler ultrasound scan of the superficial femoral artery was measured on eight recreationally active male participants; twice on the same day separated by 5-min (within-day), and on a separate day (between-days). The coefficient of variation (CV) for mean blood flow (MBF) was ~16 % and ~20 % for within and between-days, respectively. A relatively small standard error of measurement (SEM) was found both within day, 13.30 mL·min-1 (95% CI, -14.79 to 38.40 mL·min-1) and between-day, 17.75 mL·min-1 (95% CI, -40.12 to 30.88 mL·min-1) for MBF differences. These findings suggest duplex Doppler ultrasound is a reliable method to collect measurements of FABF under resting conditions. The purpose of study 2 and 3 was to determine the influence of different degrees of water immersion cooling on FABF and cutaneous blood flow (CBF) and thermoregulatory responses after endurance (Chapter 5) and resistance (Chapter 6) exercise, respectively. Participants completed a prescribed endurance of resistance exercise protocol prior to immersion into 8 ºC (cold) or 22 ºC (cool) water to the iliac crest or rested non-immersion (CON) in a randomized order. Limb blood flow and thermoregulatory responses were measured before and up to 30-min after immersion. In both studies, thigh skin temperature (Tskthigh) (P < 0.001) and muscle temperature (Tmuscle) (P < 0.01) were lowest in the 8 ºC trial compared with 22 ºC and control trials. However, femoral artery conductance (FVC) was similar after immersion in both cooling conditions and was reduced (~50-55 %) compared with the CON condition 30-min after immersion (P < 0.01). Similarly, there was a greater thigh (P < 0.01) and calf (P < 0.05) cutaneous vasoconstriction during and after immersion in both cooling conditions relative to CON with no differences noted between 8 and 22 ºC immersion. Together, these findings suggest that colder water temperatures may be more effective in the treatment of EIMD and injury after both endurance and resistance exercise, respectively, due to greater reductions in Tmuscle and not limb blood flow per se. The aim of study 4 (Chapter 7) was to compare the influence of CWI and whole body cryotherapy (WBC) on FABF and CBF and thermoregulatory responses after endurance exercise. On separate days, participants completed a continuous cycle ergometer protocol before being immersed semi-reclined into 8 ºC water to the iliac crest for 10 min (CWI), or exposed to 2.5 min (30 s -60 ºC, 2 min -110 ºC) WBC in a specialized cryotherapy chamber, in a randomized order. Limb blood flow and thermoregulatory responses were measured before and up to 40-min after immersion Reductions in Tskthigh (P < 0.001) and Tmuscle (P < 0.001) were larger in CWI during recovery. Similarly, decreases in FVC were greater (~45-50 %) in the CWI condition throughout the recovery period (P < 0.05). There was also a greater skin vasoconstriction observed in CWI at the thigh (P < 0.001) and calf (P < 0.001) throughout the post-cooling recovery period. These results demonstrate that CWI may be a better recovery strategy compared with WBC due greater reductions in both Tmuscle and limb blood flow. This thesis provides a novel insight into the influence of different degrees of water immersion cooling, as well as WBC, on limb blood flow and thermoregulatory responses after different modes of exercise. These findings provide practical application for athletes and an important insight into the possible mechanisms responsible for CWI in alleviating inflammation in sport and athletic contexts.
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Smoljanic, Jovana. "The Independent Influence of Aerobic Fitness and Running Economy on Thermoregulatory Responses During Treadmill Running." Thesis, Université d'Ottawa / University of Ottawa, 2014. http://hdl.handle.net/10393/31589.

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The independent influence of maximum oxygen consumption (VO2max) and running economy (RE) on thermoregulatory responses during treadmill exercise have not been isolated due to the complex interactions between VO2max, RE, body mass, body surface area (BSA), and metabolic heat production (Hprod). The purpose of the thesis is to determine whether large differences in VO2max and/or running economy independently alter thermoregulatory responses during running in a neutral environment. Seven aerobically unfit (LO-FIT: ~ 40 mlO2·kg-1·min-1) and sevn aerobically fit (HI-FIT: ~ 60 mlO2·kg-1·min-1) males, matched for body mass and BSA ran at 1) a fixed metabolic heat production of 640 W (FHP trial) and 2) 60%VO2max (REL trial). Also, seven high RE (HI-ECO: ~ 185 mlO2·kg-1·km-1) and seven low RE (LO-ECO: ~ 220 mlO2·kg-1·km-1) males, matched for body mass, BSA and VO2max (~ 60 mlO2·kg-1·min-1) ran at a 1) fixed Hprod of 640 W (FHP trial) and 2) fixed running speed of 10.5 km·h-1 (FRS trial). All trials were performed in a thermoneutral environment. The data was analyzed using a two-way mixed ANOVA, with the significance level set at an alpha of 0.05 for all comparisons. It was hypothesized that thermoregulatory responses (i.e., core temperature and sweating), during exercise will not be independently altered by VO2max, but will be altered by any differences in heat production and running economy. The FHP trial resulted in similar changes in esophageal temperature (∆Tes), changes in rectal temperature (∆Tre), and WBSL between the HI-FIT and LO-FIT groups, despite vastly different %VO2max. Whereas the REL trial resulted in greater ΔTeso, ΔTre, and WBSL in the HI-FIT group, in parallel with their greater Hprod. In groups greatly differing in RE, the FHP trial elicited similar ∆Tes, ∆Tre, and WBSL; however the HI-ECO group had to run faster to achieve the same heat production as their LO-ECO counterparts. Moreover, a FRS of 10.5 kmh-1 produced a greater Hprod, ∆Tes, ∆Tre, and WBSL in the LO-ECO group. In conclusion, thermoregulatory responses are determined by Hprod and RE, not VO2max, when differences in mass and BSA are eliminated between groups. Thus, these findings support the initially stated hypotheses.
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8

Goulet, Éric. "Effect of glycerol hyperhydration before exercise in trained triathletes on endurance performance and cardiovascular and thermoregulatory responses." Sherbrooke : Université de Sherbrooke, 2001.

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9

Goulet, Éric. "Effect of glycerol hyperhydration before exercise in trained triathletes on endurance performance and cardiovascular and thermoregulatory responses." Mémoire, Université de Sherbrooke, 2001. http://savoirs.usherbrooke.ca/handle/11143/742.

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This experimental project determined the effect of glycerol hyperhydration in 4 trained male triathletes on endurance performance and cardiovascular and thermoregulatory responses. For this purpose, the subjects received, using a randomized, double-blind and crossover protocol, either a glycerol (1.2 g glycerol/kg/bodyweight (BW) with 18 ml/kg/BW of aspartame-flavored juice plus 8 ml/kg/BW of distilled water) or a placebo solution (aspartame-flavored juice and water only) over an 80 min period, 40 min before exercise, then performed 2 h of cycling at 65% of maximal oxygen consumption (VO[subscript 2] max), which was immediately followed by an endurance performance test to exhaustion. All trials were conducted at 25[degrees Celsius], 38-42% relative humidity (RH). During exercise, subjects consumed a 6% sports drink (SD) solution at a rate of 166 ml every 20 min, up to min 100. Preliminary results suggest that glycerol hyperhydration could attenuate dehydration better than water hyperhydration during a cycling exercise at 65% VO[subscript 2] max in a thermoneutral climate, which could improve core temperature, but not HR. With respect to the endurance performance test, the comparison of the mean of each group reveals that glycerol hyperhydration did not increase time to exhaustion."--Résumé abrégé par UMI.
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10

Rollins, Evvi-Lynn. "The role of the cerebral cortex in the thermoregulatory responses to acute, moderate hypoxemia in young male rats." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/mq24695.pdf.

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Books on the topic "Thermoregulatory responses"

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Morgan, Karin. Short-term thermoregulatory responses of horses to brief changes in ambient temperature. Uppsala, Sweden: Sveriges lantbruksuniversitet, Institutionenför lantbruksteknik, 1996.

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Cunliffe, Melora. Menstrual status and thermoregulatory responses of active adolescents during exercise in a cold environment. St. Catharines, Ont: Brock University, Department of Physical Education and Kinesiology, 2002.

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Levesque, Danielle L. Seasonal changes in behavioural and thermoregulatory responses to hypoxia in the Eastern Chipmunk (Tamias striatus). St. Catharines, Ont: Brock University, Dept. of Biological Sciences, 2008.

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B, Jeffcott L., and Clarke A. F, eds. Thermoregulatory responses during competitive exercise in the performance horse. Newmarket, Suffolk: Equine Veterinary Journal Ltd., 1995.

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Thermoregulatory responses during competitive exercise in the performance horse. [Newmarket]: Equine Veterinary Journal, 1996.

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B, Jeffcott Leo, and Clarke A. F, eds. Thermoregulatory responses during competitive exercise in the performance horse. [Newmarket]: Equine Veterinary Journal, 1995.

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Heat, exercise and adrenergic blockade: Cardiovascular and thermoregulatory responses of six young men. 1989.

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Heat, exercise and adrenergic blockade: Cardiovascular and thermoregulatory responses of six young men. 1988.

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Falk, Bareket, and Raffy Dotan. Temperature regulation. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199232482.003.0023.

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This chapter outlines the physical and physiological changes that occur during growth and maturation and the possible effects these changes can have on the nature and effectiveness of thermoregulation. The physiological responses to heat stress are discussed in terms of metabolic, circulatory, hormonal, and sweating responses, changes in body temperature, and in terms of heat tolerance. Also discussed is hydration status, which can affect thermoregulatory effectiveness in the heat. The physiological response to cold stress is considered in terms of the metabolic and circulatory responses and their possible influence on the effectiveness of thermoregulation. The discussion does not outline the thermoregulatory response per se, but rather emphasizes the differences in that response between children and adults. Finally, child–adult differences in the acclimatization- and training-induced adaptations to thermal stress are discussed.
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Bassi, Gabriele, and Roberto Fumagalli. Pathophysiology and management of fever. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0352.

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Core body temperature is strictly regulated by autonomic and behavioural compensatory adaptations and an increase may represent a physiological stereotypical controlled response to septic and inflammatory conditions, or an uncontrolled drop in the hypothalamic thermoregulatory threshold. Fever has been demonstrated to be a potential mechanism of intrinsic resistance against infectious disease playing a pivotal role in the human evolution. High temperature may be detrimental during oxygen delivery-dependent conditions and in a neurological population. Despite this evidence, a definitive conclusion, between the association of fever and the outcome in critically-ill patients, is still lacking. The decision-making strategy in the context of fever management in critical care must be supported by single case assessment. This chapter summarizes the main physiological mechanisms of temperature control that physicians should consider when dealing with fever or deliberate hypothermia and analyses the main evidence in the role of fever in the critically ill in order to help bedside clinical strategy.
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Book chapters on the topic "Thermoregulatory responses"

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Dawson, William R., and Timothy P. O’Connor. "Energetic Features of Avian Thermoregulatory Responses." In Avian Energetics and Nutritional Ecology, 85–124. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-0425-8_4.

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Jessen, Claus. "Interaction of Various Body Temperatures in Control of Thermoregulatory Responses." In Temperature Regulation in Humans and Other Mammals, 87–98. Berlin, Heidelberg: Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-642-59461-8_12.

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Blatteis, C. M., A. L. Ungar, and R. B. Howell. "Thermoregulatory Responses of Rabbits to Combined Heat Exposure, Pyrogen and Dehydration." In Temperature Regulation, 71–74. Basel: Birkhäuser Basel, 1994. http://dx.doi.org/10.1007/978-3-0348-8491-4_12.

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Gordon, Christopher J. "Response of the Thermoregulatory System to Toxic Chemicals." In Theory and Applications of Heat Transfer in Humans, 529–52. Chichester, UK: John Wiley & Sons Ltd, 2018. http://dx.doi.org/10.1002/9781119127420.ch25.

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Schroeder, M., M. Ozaki, A. Kurz, D. I. Sessler, R. Lenhardt, A. Moayeri, K. M. Noyes, E. Rotheneder, and M. Kurz. "Epidural and Spinal Anesthesia Alter Thermoregulatory Response Thresholds." In Thermal Balance in Health and Disease, 305–10. Basel: Birkhäuser Basel, 1994. http://dx.doi.org/10.1007/978-3-0348-7429-8_41.

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"Thermoregulatory effector responses." In Temperature Regulation in Laboratory Rodents, 73–108. Cambridge University Press, 1993. http://dx.doi.org/10.1017/cbo9780511565595.005.

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"Acute Toxic Thermoregulatory Responses." In Temperature and Toxicology, 51–105. CRC Press, 2005. http://dx.doi.org/10.1201/9781420037906.ch3.

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W. Potter, Adam, David P. Looney, Xiaojiang Xu, William R. Santee, and Shankar Srinivasan. "Modeling Thermoregulatory Responses to Cold Environments." In Autonomic Nervous System Monitoring - Heart Rate Variability. IntechOpen, 2020. http://dx.doi.org/10.5772/intechopen.81238.

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Cooper, Angela L., and Nancy J. Rothwell. "Central control of metabolic and thermoregulatory responses to injury." In Brain Control of Responses to Trauma, 260–94. Cambridge University Press, 1994. http://dx.doi.org/10.1017/cbo9780511752698.011.

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Borges, Laylson da Silva, Geandro Carvalho Castro, João Lopes Anastácio Filho, Isak Samir de Sousa Lima, Flávio Carvalho de Aquino, Marcelo Richelly Alves de Oliveira, Amauri Felipe Evangelista, Wéverton José Lima Fonseca, and Fernanda Samara Barbosa Rocha. "THERMOREGULATORY RESPONSES OF GOATS REARED IN THE BRAZILIAN SEMIARID REGION." In Investigação Científica e Técnica em Ciência Animal 2, 62–68. Atena Editora, 2019. http://dx.doi.org/10.22533/at.ed.2631912099.

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Conference papers on the topic "Thermoregulatory responses"

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Boregowda, S., S. Tiwari, and S. Chaturvedi. "Investigation of transient human thermoregulatory responses to different environmental conditions." In 31st Thermophysics Conference. Reston, Virigina: American Institute of Aeronautics and Astronautics, 1996. http://dx.doi.org/10.2514/6.1996-1829.

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Zhang, Xianghui, and Jun Li. "Effects of Clothing Ventilative Designs on Thermoregulatory Responses during Exercise." In International Conference on Biomedical Engineering and Computer Science (ICBECS 2010). IEEE, 2010. http://dx.doi.org/10.1109/icbecs.2010.5462337.

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Shrivastava, Devashish, Lance DelaBarre, Timothy Hanson, and J. Thomas Vaughan. "Improved MR Thermometry to Measure Brain Temperatures." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192017.

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An MR thermometry technique with sub-degree celsius accuracy is needed to measure in vivo temperatures vs. time in porcine brains at ultra-high fields. Porcine models are used to study thermoregulatory temperature response of the ultra-high field radiofrequency (RF) heating. The porcine hot critical temperature limit is comparable to and lower than that of humans. Also, porcine thermoregulatory mechanisms are similar to humans. Thus, conservative porcine thermoregulatory temperature responses can help develop new RF safety thresholds for ultra-high field human MRI. Sub-degree C temperature accuracy is needed since RF safety guidelines limit the maximum in vivo head temperature change due to RF heating to 1 °C over the core body temperature. Three-dimensional temperature maps over time are required since non-uniform RF power deposition at ultra-high fields and blood flow produce non-uniform in vivo temperatures with local hot spots. Thermogenic hazards are related to in vivo temperatures and temperature-time history — and not to the typically measured whole head average specific absorption rate.
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Berglund, Larry G., Richard R. Gonzalez, Yuval Heled, and Daniel S. Moran. "Simulated Human Thermoregulatory Responses to Events of a Cold Wet Sea Rescue." In International Conference On Environmental Systems. 400 Commonwealth Drive, Warrendale, PA, United States: SAE International, 2003. http://dx.doi.org/10.4271/2003-01-2508.

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Carluccio, G., and C. M. Collins. "Safety evaluation of algorithms for local excitation with a transmit array considering thermoregulatory responses." In 2017 International Conference on Electromagnetics in Advanced Applications (ICEAA). IEEE, 2017. http://dx.doi.org/10.1109/iceaa.2017.8065673.

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Farrington, Robert B., John P. Rugh, Desikan Bharathan, and Rick Burke. "Use of a Thermal Manikin to Evaluate Human Thermoregulatory Responses in Transient, Non-Uniform, Thermal Environments." In International Conference On Environmental Systems. 400 Commonwealth Drive, Warrendale, PA, United States: SAE International, 2004. http://dx.doi.org/10.4271/2004-01-2345.

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MacLean, Heidi. "Historical changes in thermoregulatory traits of alpine butterflies: Environmental variability limits adaptive responses to recent climate change." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.105880.

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Angela R Green and Hongwei Xin. "Effects of Stocking Density and Group Size on Thermoregulatory Responses of Laying Hens under Heat Challenging Conditions." In Livestock Environment VIII, 31 August - 4 September 2008, Iguassu Falls, Brazil. St. Joseph, MI: American Society of Agricultural and Biological Engineers, 2008. http://dx.doi.org/10.13031/2013.25554.

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Frederico M C Vieira, Iran J O Silva, Késia O Silva-Miranda, Aérica C Nazareno, and Priscila N Faria. "Thermoregulatory responses of day-old chickens submitted to simulated transport condition: effect of thermal environment and box placement." In 2012 IX International Livestock Environment Symposium (ILES IX). St. Joseph, MI: American Society of Agricultural and Biological Engineers, 2012. http://dx.doi.org/10.13031/2013.41604.

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Frederico M C Vieira, Iran J O Silva, Késia O Silva-Miranda, Aérica C Nazareno, Juliano R Camargo, and Afrânio M C Vieira. "Thermoregulatory responses of day-old chickens submitted to simulated transport condition: effect of exposure time under different thermal ranges." In 2012 IX International Livestock Environment Symposium (ILES IX). St. Joseph, MI: American Society of Agricultural and Biological Engineers, 2012. http://dx.doi.org/10.13031/2013.41608.

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Reports on the topic "Thermoregulatory responses"

1

Endrusick, Thomas L., and Richard R. Gonzalez. Effects of Wearing Impermeable and Permeable Protective Clothing on Thermoregulatory Responses While Sedentary. Fort Belvoir, VA: Defense Technical Information Center, November 2000. http://dx.doi.org/10.21236/ada384152.

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Castellani, John W., Andrew J. Young, Michael N. Sawka, Pang N. Shek, and Ingrid K. Brenner. Thermoregulatory and Immune Responses During Cold Exposure: Effects of Repeated Cold Exposure and Acute Exercise. Fort Belvoir, VA: Defense Technical Information Center, March 2000. http://dx.doi.org/10.21236/ada375860.

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Heaps, Cristine L., and Stefan H. Constable. The Metabolic and Thermoregulatory Responses of Rhesus Monkeys to Combined Exercise and Environmental Heat Load. Fort Belvoir, VA: Defense Technical Information Center, August 1993. http://dx.doi.org/10.21236/ada269756.

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