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1

Instructor's guide/test bank for microbiology: An introduction. 7th ed. San Francisco, CA: Benjamin Cummings, 2001.

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2

V, Kilgore M., Mikall A. T, and United States. National Aeronautics and Space Administration., eds. Microbiological methods for the water recovery systems test. [Washington, DC: National Aeronautics and Space Administration, 1990.

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3

D, Tatara J., and George C. Marshall Space Flight Center., eds. Analytical control test plan and microbiological methods for the water recovery test. Marshall Space Flight Center, Ala: National Aeronautics and Space Administration, Marshall Space Flight Center, 1994.

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4

L, Batzing Barry, ed. Instructor's manual with test bank for Batzing's microbiology: An introduction. Australia: Brooks/Cole, Thomson Learning, 2002.

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5

United States. National Aeronautics and Space Administration., ed. Microbiological test results using three urine pretreatment regimes with 316L stainless steel. [Washington, DC: National Aeronautics and Space Administration, 1993.

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6

Peter, James B. Use and interpretation of tests in medical microbiology. 3rd ed. Santa Monica, CA: Specialty Laboratories, 1992.

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7

Peter, James B. Use and interpretation of tests in medical microbiology. 2nd ed. Santa Monica, CA: Specialty Laboratories, 1990.

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8

Sprague, John B. Biological test method: Toxicity test using luminescent bacteria (photobacterium phosphoreum). [Ottawa]: Environment Canada, 1992.

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9

L, Williams Kevin, ed. Endotoxins: Pyrogens, LAL testing and depyrogenation. 3rd ed. New York: Informa Healthcare, 2007.

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10

Assessment and Remediation of Contaminated Sediments Program (U.S.) and U.S. Fish and Wildlife Service, eds. Hazard ranking of contaminated sediments based on chemical analysis, laboratory toxicity tests, and benthic community structure: Method of prioritizing sites for remedial action. Chicago, Ill. (77 West Jackson, Chicago 60604): Great Lakes National Program Office, U.S. Environmental Protection Agency, 1994.

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11

Murray, Patrick R. Pocket guide to clinical microbiology. 2nd ed. Washington, D.C: ASM Press, 1998.

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12

ASM pocket guide to clinical microbiology. Washington, D.C: ASM Press, 1996.

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13

Alcamo, I. Edward. Schaum's outline of theory and problems of microbiology. New York: McGraw-Hill, 1998.

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14

Sprague, John B. Biological test method: Fertilization assay using echinoids (sea urchins and sand dollars). [Ottawa]: Environment Canada, 1992.

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15

Clontz, Lucia. Microbial limit and bioburden tests: Validation approaches and global requirements. Buffalo Grove, IL: Interpharm Press, 1998.

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16

Clontz, Lucia. Microbial limit and bioburden tests: Validation approaches and global requirements. 2nd ed. Boca Raton: Taylor & Francis, 2009.

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17

Clontz, Lucia. Microbial limit and bioburden tests: Validation approaches and global requirements. Buffalo Grove, IL: Interpharm Press, 1998.

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18

Antibiotiques et antibiogrammes. Montréal: Décarie, 1994.

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19

Langer, Martin, and Edoardo Carretto. Diagnosis and management of atypical pneumonia. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0118.

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‘Atypical pneumonia’ is an old, but successful term, which covers respiratory infections caused by different micro-organisms, causing similar clinical symptoms, and characterized by similar antimicrobial sensitivity/resistance. Out of specific epidemic contexts, Legionella pneumophila, Mycoplasma pneumoniae, and Chlamydophila pneumonia are the micro-organisms involved, L. pneumophila being by far the most frequently involved in severe community-acquired ‘atypical’ pneumonia. It is important to suspect ‘atypical’ pneumonia on the basis of clinical presentation and the correlated extrapulmonary symptoms. Knowledge of symptoms leads to suspicion and, consequently, to timely and adequate empiric treatment and proper diagnostic work-up. Standard microbiological diagnosis is based on urinary antigen test for L. pneumophila and on serology for the other pathogens. A culture should be performed if legionellosis is suspected. NATs techniques could be the future diagnostic tests. Close collaboration with the microbiologist will improve the diagnosis.
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20

Brock: Biology of Microorganisms: Instructor's Manual & Test Item File. Prentice Hall, 2000.

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21

Use and Interpretation of Tests in Medical Microbiology. Specialty Laboratories, 1992.

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22

Biological test method. Ottawa: Environment Canada, 1992.

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23

Microbiological Deterioration of Organic Materials: Its Prevention and Methods of Test; NBS Miscellaneous Publication 188. Creative Media Partners, LLC, 2021.

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24

Williams, Kevin L. Endotoxins: Pyrogens, LAL Testing and Depyrogenation. Taylor & Francis Group, 2007.

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25

Williams, Kevin L. Endotoxins: Pyrogens, LAL Testing and Depyrogenation. Taylor & Francis Group, 2007.

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26

Institution, British Standards. Microbiology of food and animal feeding stuffs: Preparation of test samples, initial suspension and decimal dilutions for microbiological examination. BSI, 2003.

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27

Institution, British Standards, and International Organization for Standardization, eds. Microbiology of food and animal feeding stuffs: Preparation of test samples, initial suspension and decimal dilutions for microbiological examination. BSI, 2003.

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28

Abdelattif, Engy, and Anthony J. Freemont. Assessment of synovial joint fluid. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0072.

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Assessment of synovial joint fluid is a simple, cheap, and relatively non-invasive test that can be applied to the whole spectrum of joint diseases, provided there is sufficient fluid within the joint to aspirate. The chapter outlines the key steps in undertaking microscopic analysis of synovial fluid in order to maximize the diagnostic and prognostic return, while placing the features seen within the context of some of the most important joint diseases. The chapter also examines the changing face of microbiological examination of synovial fluid to diagnose joint infection as a primary event and also the increasingly important problem of infection secondary to joint replacement surgery.
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29

Abdelattif, Engy, Anthony J. Freemont, and DC Mangham. Assessment of synovial joint fluid. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0072_update_002.

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Assessment of synovial joint fluid is a simple, cheap, and relatively non-invasive test that can be applied to the whole spectrum of joint diseases, provided there is sufficient fluid within the joint to aspirate. The chapter outlines the key steps in undertaking microscopic analysis of synovial fluid in order to maximize the diagnostic and prognostic return, while placing the features seen within the context of some of the most important joint diseases. The chapter also examines the changing face of microbiological examination of synovial fluid to diagnose joint infection as a primary event and also the increasingly important problem of infection secondary to joint replacement surgery.
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30

Murray, Patrick R., and Yvonne R. Shea. Pocket Guide to Clinical Microbiology. Wiley & Sons, Limited, John, 2014.

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31

National Aeronautics and Space Administration (NASA) Staff. Microbiological Test Results of the Environmental Control and Life Support Systems Vapors Compression Distillation Subsystem Recycle Tank Components Following Various Pretreatment Protocols. Independently Published, 2018.

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32

Pocket Guide to Clinical Microbiology. 3rd ed. ASM Press, 2004.

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33

Schaum's outline of theory and problems of microbiology . McGraw-hill, 1998.

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34

Biological test method: Fertilization assay using echinoids (sea urchins and sand dollars). Ottawa, Ont., Canada: Environmental Protection, Conservation and Protection, Environment Canada, 1992.

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35

Clontz, Lucia. Microbial Limit and Bioburden Tests: Validation Approaches and Global Requirements. Taylor & Francis Group, 2008.

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36

Clontz, Lucia. Microbial Limit and Bioburden Tests: Validation Approaches and Global Requirements. CRC, 1997.

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37

Clontz, Lucia. Microbial Limit and Bioburden Tests: Validation Approaches and Global Requirements,Second Edition. Taylor & Francis Group, 2008.

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38

Clontz, Lucia. Microbial Limit and Bioburden Tests: Validation Approaches and Global Requirements,Second Edition. Taylor & Francis Group, 2008.

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39

Clontz, Lucia. Microbial Limit and Bioburden Tests: Validation Approaches and Global Requirements,Second Edition. 2nd ed. CRC, 2008.

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40

COM (94) 539 Final, Brussels, 06.12.1994: 11 - External Relations: Proposal for a Council Decision Laying Down the Rules for the Microbiological Test by ... (COM (94) 539 Final, Brussels, 06.12.1994). European Communities / Union (EUR-OP/OOPEC/OPOCE), 1994.

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41

COM (94) 560 Final, Brussels, 06.12.1994: 11 - External Relations: Proposal for a Council Decision Laying Down the Rules for the Microbiological Test by ... (COM (94) 560 Final, Brussels, 06.12.1994). European Communities / Union (EUR-OP/OOPEC/OPOCE), 1994.

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42

White, P. Lewis, and Rosemary A. Barnes. Molecular diagnosis of fungal disease. Edited by Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum, and Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0043.

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Molecular techniques to aid in the diagnosis of fungal disease have been in use for over two decades. However, for polymerase chain reaction (PCR) to gain widespread acceptance outside of specialist centres, methodology must be standardized and in line with general microbiological molecular diagnostics assays, yet for infections other than fungal disease. Apart from Aspergillus PCR, standardized methodology is lacking. It is also essential to identify the optimal role for an assay. Whether this is to confirm a specific disease in symptomatic patients or to exclude disease and prevent the unnecessary use of antifungals will, in part, be determined by prevalence, but will also, along with the disease manifestation, dictate specimen choice and subsequent methodological procedure. This chapter will focus on disease processes determining optimal sample types, before concentrating on the clinical validation of molecular tests for the diagnosis of the main causes of invasive fungal disease, concluding with recent developments. The clinical utility of molecular approaches and potential future benefits that can address emerging issues, such as azole resistance, will also be discussed.
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43

ANSI/AAMI/ISO 11737-2:2019; Sterilization of medical devices—Microbiological methods—Part 2: Tests of sterility performed in the definition, validation and maintenance of a sterilization process. AAMI, 2019. http://dx.doi.org/10.2345/9781570207341.

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44

Abdulkader, Rita, and Richard A. Watts. Mycobacterial diseases. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0103.

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The main diseases caused by mycobacterial infection are tuberculosis (TB) and leprosy. Despite a fall in the prevalence of these diseases over the last decade, they are still significant causes of morbidity and mortality worldwide. Atypical mycobacterial infections are encountered less frequently. Immigration patterns, the frequency of human immunodeficiency infection, and the increased numbers of patients on immunosuppressive treatments render mycobacterial infections relevant not only to physicians in the developing world where they traditionally occurred but also in the developed world. Skeletal TB occurs in 1–3% of cases of TB infection, and is more frequently encountered in the immunocompromised. A high index of suspicion is required, diagnosis relies on a combination of clinical features and radiological, histological, and microbiological tests. Multidrug regimens are required for treatment with surgery in selected cases. Leprosy is caused by M. leprae infection. The disease is still a leading cause of disability worldwide. Diagnosis is usually clinical. The course of the disease is indolent but may be interrupted by acute inflammatory reactions, which contribute to nerve damage and disability. Treatment aims at eliminating the mycobacteria using multidrug regimens, and management of complications including leprosy reactions and long-term nerve damage. Atypical mycobacterial infections affecting bone and joints are uncommon; they usually follow direct inoculation of the pathogen. Haematogenous dissemination is encountered in immunocompromised patients. These microorganisms are not usually susceptible to the same drug regimens used in the treatment of tuberculosis.
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