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Academic literature on the topic 'Tau Effect'

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Published: 9 March 2023
Last updated: 10 March 2023

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Journal articles on the topic "Tau Effect"

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Kawabe, Takahiro, Kayo Miura, and Yuki Yamada. "Audiovisual tau effect." Acta Psychologica 128, no. 2 (June 2008): 249–54. http://dx.doi.org/10.1016/j.actpsy.2008.01.004.

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Barthelemy, Nicolas R., Haiyan Liu, Randall Bateman, and Brendan P. Lucey. "P4-066: EFFECT OF SLEEP ON CSF TAU AND P-TAU." Alzheimer's & Dementia 15 (July 2019): P1298. http://dx.doi.org/10.1016/j.jalz.2019.06.3726.

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Barthelemy, Nicolas R., Haiyan Liu, Randall Bateman, and Brendan P. Lucey. "F2-08-03: EFFECT OF SLEEP ON CSF TAU AND P-TAU." Alzheimer's & Dementia 15 (July 2019): P529—P530. http://dx.doi.org/10.1016/j.jalz.2019.06.4446.

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Das, Rashmi, Abhishek Ankur Balmik, and Subashchandrabose Chinnathambi. "Effect of Melatonin on Tau aggregation and Tau-mediated cell surface morphology." International Journal of Biological Macromolecules 152 (June 2020): 30–39. http://dx.doi.org/10.1016/j.ijbiomac.2020.01.296.

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Kawabe, Takahiro, Nobu Shirai, Yuji Wada, Kayo Miura, So Kanazawa, and Masami K. Yamaguchi. "The Audiovisual Tau Effect in Infancy." PLoS ONE 5, no. 3 (March 3, 2010): e9503. http://dx.doi.org/10.1371/journal.pone.0009503.

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Gil, Laura, Concetta Federico, Fernando Pinedo, Francesca Bruno, Ana B. Rebolledo, Juan J. Montoya, Isabel M. Olazabal, Isidre Ferrer, and Salvatore Saccone. "Aging dependent effect of nuclear tau." Brain Research 1677 (December 2017): 129–37. http://dx.doi.org/10.1016/j.brainres.2017.09.030.

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Liu, Guanghao, Ramasamy Thangavel, Jacob Rysted, Yohan Kim, Meghan B. Francis, Eric Adams, Zhihong Lin, et al. "Loss of tau and Fyn reduces compensatory effects of MAP2 for tau and reveals a Fyn‐independent effect of tau on calcium." Journal of Neuroscience Research 97, no. 11 (August 26, 2019): 1393–413. http://dx.doi.org/10.1002/jnr.24517.

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Ward, Sarah M., Diana S. Himmelstein, Jody K. Lancia, and Lester I. Binder. "Tau oligomers and tau toxicity in neurodegenerative disease." Biochemical Society Transactions 40, no. 4 (July 20, 2012): 667–71. http://dx.doi.org/10.1042/bst20120134.

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AD (Alzheimer's disease) is a progressive neurodegenerative disorder characterized by the extracellular accumulation of amyloid β-peptide and the intracellular accumulation of tau. Although there is much evidence linking tau to neurodegeneration, the precise mechanism of tau-mediated neurotoxicity remains elusive. The presence of tau-positive pre-tangle neurons lacking neurofibrillary tangles has been reported in AD brain tissue. In order to study this non-fibrillar tau, we generated a novel monoclonal antibody, named TOC1 (tau oligomeric complex 1), which selectively labels tau dimers and oligomers, but does not label filaments. Time-course analysis and antibody labelling indicates that oligomers appear as an early event in AD pathogenesis. Using a squid axoplasm assay, we have demonstrated that aggregated tau inhibits anterograde FAT (fast axonal transport), whereas monomeric tau has no effect. This inhibition requires a small stretch of N-terminal amino acids termed the PAD (phosphatase-activation domain). Using a PAD-specific antibody, TNT1 (tau N-terminal 1), we demonstrate that PAD exposure is increased in diseased neurons and this leads to an increase in FAT inhibition. Antibody co-labelling with the early-AD marker AT8 indicates that, similar to TOC1, TNT1 expression represents an early event in AD pathogenesis. Finally, the effects of the molecular chaperone Hsp70 (heat-shock protein 70) were also investigated within the squid axoplasm assay. We illustrate that Hsp70 preferentially binds to tau oligomers over filaments and prevents anterograde FAT inhibition observed with a mixture of both forms of aggregated tau. Together, these findings support the hypothesis that tau oligomers are the toxic form of tau in neurodegenerative disease.
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Khan, Md Ishak, Kathleen Gilpin, Fuad Hasan, Khandakar Abu Hasan Al Mahmud, and Ashfaq Adnan. "Effect of Strain Rate on Single Tau, Dimerized Tau and Tau-Microtubule Interface: A Molecular Dynamics Simulation Study." Biomolecules 11, no. 9 (September 4, 2021): 1308. http://dx.doi.org/10.3390/biom11091308.

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Microtubule-associated protein (MAP) tau is a cross-linking molecule that provides structural stability to axonal microtubules (MT). It is considered a potential biomarker for Alzheimer’s disease (AD), dementia, and other neurological disorders. It is also a signature protein for Traumatic Brain Injury (TBI) assessment. In the case of TBI, extreme dynamic mechanical energies can be felt by the axonal cytoskeletal members. As such, fundamental understandings of the responses of single tau protein, polymerized tau protein, and tau-microtubule interfaces under high-rate mechanical forces are important. This study attempts to determine the high-strain rate mechanical behavior of single tau, dimerized tau, and tau-MT interface using molecular dynamics (MD) simulation. The results show that a single tau protein is a highly stretchable soft polymer. During deformation, first, it significantly unfolds against van der Waals and electrostatic bonds. Then it stretches against strong covalent bonds. We found that tau acts as a viscoelastic material, and its stiffness increases with the strain rate. The unfolding stiffness can be ~50–500 MPa, while pure stretching stiffness can be >2 GPa. The dimerized tau model exhibits similar behavior under similar strain rates, and tau sliding from another tau is not observed until it is stretched to >7 times of original length, depending on the strain rate. The tau-MT interface simulations show that very high strain and strain rates are required to separate tau from MT suggesting Tau-MT bonding is stronger than MT subunit bonding between themselves. The dimerized tau-MT interface simulations suggest that tau-tau bonding is stronger than tau-MT bonding. In summary, this study focuses on the structural response of individual cytoskeletal components, namely microtubule (MT) and tau protein. Furthermore, we consider not only the individual response of a component, but also their interaction with each other (such as tau with tau or tau with MT). This study will eventually pave the way to build a bottom-up multiscale brain model and analyze TBI more comprehensively.
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Tchounwou, Christine, Bretton Fletcher, Chaeyeon Song, Phillip A. Kohl, Peter J. Chung, Herb P. Miller, Youli Li, et al. "The Effect of Site-Specific Acetylation Based Tau Mutations on Tau-Microtubule Associations." Biophysical Journal 116, no. 3 (February 2019): 255a. http://dx.doi.org/10.1016/j.bpj.2018.11.1391.

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Dissertations / Theses on the topic "Tau Effect"

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Subramaniyan, Parimalam Subhathirai. "Study of Tau Protein's Effect on Microtubule-Kinesin Molecular System and Development of Tau Detection Microfluidic Device." 京都大学 (Kyoto University), 2016. http://hdl.handle.net/2433/216189.

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Kestoras, Dimitra. "Investigating the effect of pathological tau on ROS production and cell death in two in vitro models of tau pathology." Thesis, University of Cambridge, 2015. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.709236.

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Pearce, Janice. "The trafficking of apolipoprotein E and its effect upon tau phosphorylation." Thesis, King's College London (University of London), 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.325158.

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Utton, Michelle Anne. "The effect of phosphorylation on the function of the microtubula-associated protein tau." Thesis, King's College London (University of London), 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.338785.

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Poots, Rosemary Elizabeth. "The effect of supplementary dietary protein on rumen fermintation, energy metabolism and N-tau-Methylhistidine excretion in lactating dairy cows." Thesis, Queen's University Belfast, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.334600.

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Xiao, Chaowu. "Effect of interferon-[tau] and steroid hormones on the expression of genes involved in the prostaglandin synthesis in bovine endometrial cells." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ42296.pdf.

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CERMISONI, ROBERTA. "L'effetto Tau di Vittorio Benussi come modello di studio del presente fenomenico: nuove prospettive di ricerca per l'esame della percezione del tempo e della personalità." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2013. http://hdl.handle.net/10281/43699.

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This thesis investigates the concept of psychological time and, in particular, the concept of psychological present. Starting by the studies of the main authors who dealt with these issues, I delved into the relationship that space and time have within the present. The tau effect, a perceptual phenomenon discovered by Vittorio Benussi in 1907, is of particular interest for this field. The tau effect indeed shows that the duration of temporal intervals influences the perception of spatial distances in the sense of “equation” (e.g. briefer temporal intervals corresponded to perceptual shorter spatial distances). Even though this effect has been extensively investigated in the last decades, a conclusive theory doesn’t exist. Five experiments were hence performed to better understand this effect. The main result is that the tau effect could be found only when the Standard Stimulus (i.e. the Stimulus that does not vary during the whole experiment, Ss) is presented as first. Furthermore, results proved that this effect emerged only within the temporal limits of the psychological present (i.e. 2‐3 sec. on average). Since, according to the authors examined in this work, one of the features of present is its flexibility depending on individual differences, the possible correlations between the tau effect and personality traits have been also investigated. Results proved that the way space and time are intertwined is linked to Extroversion and Emotional Stability, as defined by the Big Five model. These results are in line with the literature on time perception. To explain the results about the order of presentation of Ss, an alternative explanation based on the general theories developed by Benussi is discussed. According to this alternative explanation, in our experiments the tau effect occurs only when Ss is presented as first because its temporal interval used is perceived as “indifferent” or “indeterminate”, that is not “long‐” or “short‐lived”. This condition would allow participants to judge only the spatial distances without being influenced by the corresponding temporal intervals of the first stimulus presented.
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Degerman, Gunnarsson Malin. "Biomarkers as Monitors of Drug Effect, Diagnostic Tools and Predictors of Deterioration Rate in Alzheimer’s Disease." Doctoral thesis, Uppsala universitet, Institutionen för folkhälso- och vårdvetenskap, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-196965.

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Decreased amyloid-ß42 (Aß42), increased total tau (t-tau) and phosphorylated tau (p-tau) in cerebrospinal fluid (CSF) reflect histopathological core changes in the most common dementia disorder, Alzheimer’s disease (AD). They discriminate AD from healthy controls and predict conversion to AD with a relatively high accuracy. Memantine, an uncompetitive NMDA-receptor antagonist, is indicated for symptomatic treatment of AD. The first aim of this thesis was to investigate effects of memantine on CSF concentrations of Aβ42, tau and p-tau. Secondly, the aim was to explore the relation between these CSF biomarkers and retention of the amyloid biomarker Pittsburgh compound B using positron emission tomography (PIB PET), regional glucose metabolism measured with 18Fluoro-2-deoxy-d-glucose (FDG) PET and neuropsychological test performance. The third aim was to investigate their possible utility as predictors of future rate of AD dementia deterioration. All patients in the studies were recruited from the Memory Clinic, Uppsala University Hospital. In study I CSF p-tau concentrations in 11 AD patients were reduced after twelve months treatment with memantine, indicating that this compound may affect a key pathological process in AD. Results from study II showed that the concentrations of CSF Aß42 are lower in PIB+ patients than in PIB- patients, and that the PIB retention was stable during 12 months. In study III 10 patients with the diagnoses AD (6 PIB+/4 PIB-) and 8 subjects (1 PIB+/7 PIB-) with frontotemporal dementia were included. PIB+ patients had lower psychomotor speed measured by performance on the Trail Making Test A and impaired visual episodic memory compared to the PIB- patients. The initial clinical diagnoses were changed in 33% of the patients (6/18) during follow-up. Study IV is the first-ever report of an association between high CSF tau and dying in severe dementia. These 196 AD patients were followed up to nine years after baseline lumbar puncture. Moreover, CSF t-tau concentrations above median was associated with an increased risk of rapid cognitive decline (OR 3.31 (95% CI 1.53-7.16), independently of baseline functional stage. Thus, a clear association between high levels of CSF t-tau and p-tau and a more aggressive course of the disease was shown.
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Bruch, Daniel Mathias Julius [Verfasser], Günter [Akademischer Betreuer] [Gutachter] Höglinger, Thomas [Gutachter] Misgeld, and Stefan [Gutachter] Lichtenthaler. "New insights into factors affecting the pathogenesis of Progressive Supranuclear Palsy: Tau splicing and the effect of protein kinase RNA-like endoplasmic reticulum kinase (PERK) dysfunction / Daniel Mathias Julius Bruch ; Gutachter: Günter Höglinger, Thomas Misgeld, Stefan Lichtenthaler ; Betreuer: Günter Höglinger." München : Universitätsbibliothek der TU München, 2017. http://d-nb.info/1135385327/34.

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El, Khoury Noura. "Effet du diabète sur la pathologie de la protéine Tau «in vivo»." Thesis, Université Laval, 2013. http://www.theses.ulaval.ca/2013/30032/30032.pdf.

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La maladie d’Alzheimer (MA) représente aujourd’hui la forme de démence la plus commune pour laquelle il n’existe toujours pas de traitement curatif. Dans le cerveau, elle se caractérise par la présence de deux agrégats protéiques majeurs : les plaques amyloïdes qui résultent de l’accumulation extracellulaire d’un peptide nommé peptide amyloïde, et les enchevêtrements neurofibrillaires correspondant à l’agrégation intra-neuronale d’une protéine nommée Tau qui se trouve dans un état anormalement hyperphosphorylé. La pathologie Tau est importante puisque son étendue corrèle avec le degré du déficit cognitif retrouvé dans la MA. Seule une petite proportion des cas de la MA est causée par des mutations génétiques. En revanche, l’étiologie de la majorité des cas (~99%), qui est d’origine sporadique et à apparition tardive, semble être multifactorielle, avec des facteurs externes pouvant interagir avec des susceptibilités biologiques ou génétiques afin d’accélérer la manifestation de la maladie. Au cours de la dernière décennie, des données cliniques et précliniques émergentes suggèrent que le diabète sucré et la dysfonction de l’insuline qui l’accompagne pourrait représenter l’un de ces facteurs. En plus de son rôle métabolique, l’insuline a été rapportée pour avoir un rôle neurotrophique et régulateur dans le cerveau humain. Plus particulièrement, des études in vitro ont montré que l’insuline est capable de moduler la phosphorylation de Tau dans les cellules neuronales. Cette hypothèse a été par la suite renforcée par les observations d’hyperphosphorylation de Tau dans les cerveaux de souris montrant des anomalies au niveau de la signalisation de l’insuline. Malgré toutes ces données, on connaît très peu concernant l’impact du diabète sur la pathologie Tau in vivo. Le but global de ce projet de doctorat a été donc de clarifier l’impact du diabète sur la pathogenèse de la protéine Tau, dans deux modèles génétiques de diabète de type 1 (DT1) et diabète de type 2 (DT2), qui sont les souris NOD (non-obese-diabetic) et les souris ob/ob, respectivement. Nos résultats montrent que le DT1 entraine une hyperphosphorylation progressive de Tau qui commence à être détectée même en absence de toute dérégulation dans le métabolisme du glucose. De plus, cette hyperphosphorylation est plus prononcée en présence des caractéristiques du DT1 (hyperglycémie et glycosurie) et encore plus amplifiée en présence de l’hypothermie. D’une manière intéressante, nos résultats suggèrent que l’hyperphosphorylation de Tau chez ces souris corrèle avec une dérégulation de PP2A (protein phosphatase 2A), l’une des phosphatases les plus importantes de Tau in vivo. Quant au DT2, nos résultats montrent une hyperphosphorylation de Tau chez les souris ob/ob à 4 et 26 semaines, au niveau de plusieurs sites spécifiques. De plus, ces souris développent une hypothermie modérée, mais le rétablissement de la normothermie ne restaure pas les niveaux de phosphorylation de Tau, ce qui suggère que cette hyperphosphorylation serait plutôt la conséquence des composantes du DT2, et non pas de l’hypothermie qui en résulte. D’une manière intéressante, nos résultats ne montrent pas de dérégulation au niveau des protéines impliquées dans la voie de signalisation de l’insuline, suggérant par conséquent que, d’autres facteurs, probablement associés à l’obésité, pourraient contribuer à l’hyperphosphorylation de Tau dans le DT2. La compréhension des mécanismes qui sous-tendent la corrélation entre la dysfonction de l’insuline et la pathologie Tau aidera par la suite à trouver de nouvelles cibles thérapeutiques visant à contrôler la progression de la maladie.
Alzheimer’s disease (AD) is the leading form of dementia. There is actually no cure for AD, but even a treatment that would slow down the progression of the disease by 5 or 10 years will have a tremendous socio-economic impact for Canada. The neuropathological hallmarks of Alzheimer's disease include senile plaques of -amyloid (A) peptides (a cleavage product of the amyloid precursor protein, or APP), and neurofibrillary tangles (NFT) of hyperphosphorylated Tau protein assembled in paired helical filaments (PHF). NFT pathology is important since it correlates with the degree of cognitive impairment in AD. Only a small proportion of AD is due to genetic variants, the large majority of cases (~99%) is late onset and sporadic in origin. The cause of sporadic AD is likely to be multifactorial, with external factors interacting with biological or genetic susceptibilities to accelerate the manifestation of the disease. Diabetes mellitus (DM) might be such factor, as there is extensive data from epidemiological studies suggesting that DM is associated with an increased relative risk for AD. Type 1 diabetes (T1DM) and type 2 diabetes (T2DM) are known to affect multiple cognitive functions in patients. However, the consequences of both type of diabetes on AD pathology are not well understood. The challenge is therefore to better understand the mechanisms of AD pathology and how they are affected by factors such as diabetes. The overall goal of this project is therefore to clarify the impacts that diabetes have on Tau protein pathogenesis, in two well-characterized mouse models of T1DM and T2DM: NOD (non-obese diabetic) and ob/ob mice, respectively. Our data suggest that spontaneous T1DM provokes a progressive Tau hyperphosphorylation that begin to be detectable in adult mice even during the non-diabetic stage, where there is no apparent deregulation of glucose metabolism. We further show that Tau phosphorylation is greatly exacerbated in the presence of principal T1DM features, notably hyperglycemia and glycosuria, and further amplified by hypothermia. Finally, we demonstrate that Tau hyperphosphorylation during T1DM is likely attributable to a deregulation in PP2A (protein phosphatase 2A), the major Tau phosphatase in vivo. Furthermore, we show that ob/ob male mice aged 4 and 26 weeks present Tau hyperphosphorylation at specific sites, but also have mild hypothermia. However, restoring normothermia did not rescue Tau hyperphosphorylation to control levels. These data indicate that Tau hyperphosphorylation accompanies major features of T2DM. Interestingly, we did not observe any deregulation in the proteins implicated in the insulin-signaling pathway, suggesting that other obesity-associated factors, contribute to Tau phosphorylation in ob/ob mice. In turn, this understanding will help the development of treatments or life-style strategies destined to check the advance of the disease.
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Books on the topic "Tau Effect"

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Claudio, De Simone, Arrigoni Martelli Edoardo 1930-, and Sigma-tau (Organization), eds. Stress, immunity, and ageing: A role for acetyl-L-carnitine : proceedings of the workshop held in Pomezia, Rome, Italy, organised by Sigma Tau, 13 September 1988. Amsterdam: Excerpta Medica, 1989.

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J, Wald Nicholas, Froggatt Peter, and Great Britain. Independent Scientific Committee on Smoking and Health., eds. Nicotine, smoking, and the low tar programme. Oxford: Oxford University Press, 1989.

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Tai feng dui wo guo feng dian kai fa de ying xiang yu dui ce. Beijing Shi: Qi xiang chu ban she, 2010.

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United States. Dept. of Health and Human Services., United States. Public Health Service., United States. Agency for Toxic Substances and Disease Registry., and Research Triangle Institute, eds. Toxicological profile for wood creosote, coal tar creosote, coal tar, coal tar pitch, and coal tar pitch volatiles. [Atlanta, Ga.]: The Dept., Public Health Service, The Agency, 1996.

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Corporation, Syracuse Research, ed. Toxicological profile for wood creosote, coal tar creosote, coal tar, coal tar pitch, and coal tar pitch volatiles. [Atlanta, GA]: U.S. Department of Health and Human Services, Public Health Service, Agency for Toxic Substances and Disease Registry, 2002.

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Ulmer, Nancy Sue. Effect of chlorine on chromium speciation in tap water. Cincinnati, OH: U.S. Environmental Protection Agency, Water Engineering Research Laboratory, 1986.

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Ulmer, Nancy Sue. Effect of chlorine on chromium speciation in tap water. Cincinnati, OH: U.S. Environmental Protection Agency, Water Engineering Research Laboratory, 1986.

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Dan, Rostenkowski. Distributional effect of Chairman Rostenkowski's capital gains proposal. [Washington, D.C: Joint Committee on Taxation, 1989.

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Chao yin bo, tai er, ni. Taibei Shi: Jian kang shi jie za zhi she, 1994.

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United States. Congress. Joint Committee on Taxation., ed. Estimated revenue effects of extending expiring provisions permanently: Provisions with positive or no revenue effects. [Washington, D.C: Joint Committee on Taxation, 1990.

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Book chapters on the topic "Tau Effect"

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Maji, P., and S. Sahoo. "Effect of Non-universal $$Z^{\prime}$$ Boson on $$B\to {K}^{*}{\tau }^{+}{\tau }^{-}$$ Decay." In Springer Proceedings in Physics, 875–79. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-33-4408-2_123.

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Palmer, Heather, and Ajoy Basak. "Effect of phosphorylation on tau aggregation using model peptides and Circular Dichroism studies." In Advances in Experimental Medicine and Biology, 259–61. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-0-387-73657-0_117.

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Schechter, R., and K. E. Miller. "Effects of Insulin on Tau and Neurofilament." In Advances in Neurobiology, 679–95. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-6787-9_28.

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Kurakawa, Yukihide. "Climate Policy in Power Sector: Feed-in Tariff and Carbon Pricing." In Economics, Law, and Institutions in Asia Pacific, 79–95. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-6964-7_5.

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Abstract The purpose of this chapter is to investigate the effects of some mainstream policy schemes in the power sector on the reduction of CO2 emissions. The first part of this chapter is the analysis on the effects of promoting generation (fuel) efficiency of fossil-fuel power generation, specifically assuming more efficient coal-fired power plants that recently indicates increased presence in the Japanese power sector. Improvement in generation efficiency of fossil-fuel power plants is expected to reduce emissions of carbon dioxide mainly from a technological aspect. However, overall effects on carbon reduction in the whole industry would be ambiguous since it also depends on market structure. The increased efficiency in generation leads to an improvement in cost conditions of fossil-fuel power producers relative to their rivals. It enables them to expand their generation and market share. Analyzing the Cournot oligopoly model, it is shown that an improvement in fossil-fuel power generations produces two effects: the ‘saving effect’ and the ‘rebound effect’. The total CO2 emission in the whole industry decrease if the former effect exceeds the other, and vice versa. In addition, it is indicated that a rise in the generation efficiency would increase a difficulty of implementing carbon tax. In the second part of this chapter, I study the combination of feed-in tariff and carbon tax; that would be worthy to investigate since they could possibly complement each other. FIT policy could be financed by the revenue of carbon tax, and a reduction in electricity supply by the carbon tax would be lessen by supporting renewable power generations under FIT. It is demonstrated that FIT had the combined effects: it fosters a competitive environment in addition to indirectly reduces CO2 emissions. The result indicates that the combination of these policies would produce potential welfare gains.
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Zaidi, F., V. Ansari, H. Haider, M. Sajjad Athar, and S. K. Singh. "$$\nu _\tau /\bar{\nu }_\tau $$-$$^{40}$$Ar DIS Cross Sections with Perturbative and Nonperturbative Effects." In Springer Proceedings in Physics, 541–45. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-2354-8_99.

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Sugino, Makoto. "The Economic Effects of Equalizing the Effective Carbon Rate of Sectors: An Input-Output Analysis." In Economics, Law, and Institutions in Asia Pacific, 197–215. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-6964-7_11.

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Abstract The 2 °C target of the Paris Agreement has stimulated the implementation of carbon reducing policies such as carbon taxes and emission trading schemes, which explicitly applies a price on carbon emitting fuels. However, OECD (2016) reports that the effective carbon rate must be at least 30 Euros per ton of CO2. The effective carbon rate includes the implicit carbon price, e.g. energy taxes, along with the explicit carbon price. Previous studies have focused on the effects of explicit carbon prices. In this chapter, we will focus on the effective carbon rate and estimate the effects of carbon policies that increase the effective carbon rate to the 30 Euro threshold. We find that the short-term effect of a carbon tax that raises the effective carbon rate for all industries above 30 Euros will not only effect energy intensive industries, but also downstream industries that already have high effective carbon rates. Furthermore, we find that the carbon tax implemented in 2012 increase the average effective carbon rate, but increases the difference between taxed emitters and non-taxed emitters. Thus, tax exemption for energy intensive industries sacrifices economic efficiency.
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He, Rongqiao. "Effects of Formaldehyde on Protein (Tau) Aggregation and Cytotoxicity." In Formaldehyde and Cognition, 121–42. Dordrecht: Springer Netherlands, 2017. http://dx.doi.org/10.1007/978-94-024-1177-5_7.

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Miller, Sharon A., Janusz Z. Beer, Nelson T. Lao, and Barbara Z. Zmudzka. "Production and Persistence of UV-Induced Tan." In Biologic Effects of Light 2001, 113–26. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0937-0_10.

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Young, Antony R. "How Much Photoprotection Does a Tan Afford?" In Biologic Effects of Light 2001, 103–12. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0937-0_9.

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Yanamandra, Kiran, Marc I. Diamond, and David M. Holtzman. "Active and Passive Immunotherapy Against Tau: Effects and Potential Mechanisms." In Methods in Pharmacology and Toxicology, 121–38. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3560-4_9.

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Conference papers on the topic "Tau Effect"

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Sturm, Deborah, Alejandra Alonso, Christopher Corbo, Isaac Osores, and Cynthia Murillo. "Quantitative analysis of the effect of phosphorylated tau on cellular microfilament networks." In SPIE Medical Imaging, edited by John B. Weaver and Robert C. Molthen. SPIE, 2013. http://dx.doi.org/10.1117/12.2007056.

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Kordenat, K., and J. Leasure. "PROTECTIVE EFFECT OF CARNITINE (ST-261, SIGMA-TAU) IN ACUTE MYOCARDIAL INFARCTION IN DOGS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643012.

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Limitation of infarct size (IS), using ST-261, was evaluated in a group (I) of eight dogs, during acute MI. Another group (II) served as the control group. The protocol for both groups was the same except that each dog in the treated group was ST-261 as a single bolus (25 mg/kg, in 20ml normal saline), before inducing an occluding thrombus in the mid-LAD, using a closed-chest model, under x-ray visualization. Percentages of total (gms) myocardium at jeopardy (TMJW) and myocardial necrosis (TMNW), delineated by fluoroscein and TTC dyes, respectively, were calculated and compared to the total ventricular myocardial weight (TVMU), by computer technique for both groups at 3 Hrs post-occlusion of the LAD. Mean serum total CPK (CPK-t) and isozymes (mb-band) were measured before and up to 3 Hrs post-occlusion, as were various hemodynamic and mean precordial (21 lead) ST-segment and T-wave amplitudes. There was 14% less TMJU (p<0.05) and 41% less TMNW (p<0.01) in Group I compared to Group II. The mean % of CPK-mb/CPK-t decreased in I and increased in II over the 3 Hrs of observation. Mean HR decreased (p<0.01) in I compared to II at 3 Hrs postocclusion. The sum of the mean T-wave amplitudes from the precordial electrode sites was less in I at 3 Hrs. It is felt that ST-261 had a protective effect on the myocardium during acute myocardial infarction.
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Faleyev, D. G., K. K. Boguspaev, E. G. Faleyev, and J. J. Myrzagaliev. "The effect of biohumus on the growth of seedlings of Scorzonera tau-saghyz Lipsch. et Bosse in the laboratory." In Fifth International Conference of CIS IHSS on Humic Innovative Technologies «Humic substances and living systems». CLUB PRINT ltd., 2019. http://dx.doi.org/10.36291/hit.2019.faleyev.060.

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Fingerhut, Joelle. "Impact of Within-Case Variability on Tau-U and Regression-Based Effect Size Measures for Single-Case Experimental Data." In 2020 AERA Annual Meeting. Washington DC: AERA, 2020. http://dx.doi.org/10.3102/1583005.

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Wu, Jianfeng, Yi Su, Eric M. Reiman, Richard J. Caselli, Kewei Chen, Paul M. Thompson, Junwen Wang, and Yalin Wang. "Investigating the Effect of Tau Deposition and Apoe on Hippocampal Morphometry in Alzheimer’s Disease: A Federated Chow Test Model." In 2022 IEEE 19th International Symposium on Biomedical Imaging (ISBI). IEEE, 2022. http://dx.doi.org/10.1109/isbi52829.2022.9761576.

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Wu, Jianfeng, Yi Su, Eric M. Reiman, Richard J. Caselli, Kewei Chen, Paul M. Thompson, Junwen Wang, and Yalin Wang. "Investigating the Effect of Tau Deposition and Apoe on Hippocampal Morphometry in Alzheimer’s Disease: A Federated Chow Test Model." In 2022 IEEE 19th International Symposium on Biomedical Imaging (ISBI). IEEE, 2022. http://dx.doi.org/10.1109/isbi52829.2022.9761576.

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Zardi, Hasni, Yunia Wardi, and Susi Evanita. "Effect of Quality Products, Prices, Locations and Customer Satisfaction to Customer Loyalty Simpang Raya Restaurant Bukittinggi "Salero Nan Tau Raso"." In Proceedings of the 2nd Padang International Conference on Education, Economics, Business and Accounting (PICEEBA-2 2018). Paris, France: Atlantis Press, 2019. http://dx.doi.org/10.2991/piceeba2-18.2019.75.

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Kaloni, P. N., and J. X. Lou. "Electrothermo Convective Instability With Inclined Temperature Gradient." In ASME 2006 International Mechanical Engineering Congress and Exposition. ASMEDC, 2006. http://dx.doi.org/10.1115/imece2006-16357.

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The linear convective stability analysis of a poorly conducting liquid with inclined temperature gradient and under a vertical d. c. electric field is investigated. The liquid is placed between a parallel plate electrode system with the upper electrode heated. The eigenvalue problem is solved by the Chebyshev Tau-QZ method. Consideration is also given to the case when there is no horizontal flow and some interesting results are obtained. For inclined temperature gradient case, it is found that the onset of convection is governed by six parameters RV, RH, Re, Pr, Pe and Se. The effect of varying Re upon critical RV and RH is discussed and some considerations to the occurrence of transverse or longitudinal mode is pointed out.
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Tosatti, Jéssica, Adriana Fontes, Paulo Caramelli, and Karina Gomes. "EFFECTS OF RESVERATROL SUPPLEMENTATION ON THE COGNITIVE FUNCTION OF PATIENTS WITH ALZHEIMER’S DISEASE: A META-ANALYSIS OF RANDOMIZED CONTROLLED TRIALS." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda026.

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Background: Alzheimer’s disease (AD) is characterized by a deposition of β-amyloid peptide and the neurofibrillary tangles of tau protein. Resveratrol is a neuroprotective agent, acting in the prevention of redox impairment, and could reduce neuronal damage in patients with AD. Objectives: Systematic review and meta-analysis about the effects of resveratrol supplementation on cognitive and functional performance in patients with AD. Methods: Databases were searched for primary studies that reported cognitive and functional performance based on ADAS-cog, ADCS-ADL or MMSE instruments in AD patients treated with resveratrol. Primary studies published up to May 2021 and without language and publication date restrictions were included. The measure of effect of the meta-analysis was presented as weighted mean difference (WMD). decrease in ADAS-cog scores [WMD: -3.69 points], and significant increases in ADCS-ADL [WMD: 5.65 points] and MMSE scores [WMD: 2.03 points] in the resveratrol intervention group, when compared to the placebo group. Conclusions: Resveratrol supplementation may result in improving cognitive and functional performance in AD patients.
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Krahforst, Cecilia S., Mark W. Sprague, and Joseph J. Luczkovich. "The impact of vessel noise on oyster toadfish (Opsanus tau) communication." In Fourth International Conference on the Effects of Noise on Aquatic Life. Acoustical Society of America, 2016. http://dx.doi.org/10.1121/2.0000313.

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Reports on the topic "Tau Effect"

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Elmann, Anat, Orly Lazarov, Joel Kashman, and Rivka Ofir. therapeutic potential of a desert plant and its active compounds for Alzheimer's Disease. United States Department of Agriculture, March 2015. http://dx.doi.org/10.32747/2015.7597913.bard.

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We chose to focus our investigations on the effect of the active forms, TTF and AcA, rather than the whole (crude) extract. 1. To establish cultivation program designed to develop lead cultivar/s (which will be selected from the different Af accessions) with the highest yield of the active compounds TTF and/or achillolide A (AcA). These cultivar/s will be the source for the purification of large amounts of the active compounds when needed in the future for functional foods/drug development. This task was completed. 2. To determine the effect of the Af extract, TTF and AcA on neuronal vulnerability to oxidative stress in cultured neurons expressing FAD-linked mutants.Compounds were tested in N2a neuroblastoma cell line. In addition, we have tested the effects of TTF and AcA on signaling events promoted by H₂O₂ in astrocytes and by β-amyloid in neuronal N2a cells. 3. To determine the effect of the Af extract, TTF and AcA on neuropathology (amyloidosis and tau phosphorylation) in cultured neurons expressing FAD-linked mutants. 4. To determine the effect of A¦ extract, AcA and TTF on FAD-linked neuropathology (amyloidosis, tau phosphorylation and inflammation) in transgenic mice. 5. To examine whether A¦ extract, TTF and AcA can reverse behavioral deficits in APPswe/PS1DE9 mice, and affect learning and memory and cognitive performance in these FAD-linked transgenic mice. Background to the topic.Neuroinflammation, oxidative stress, glutamate toxicity and amyloid beta (Ab) toxicity are involved in the pathogenesis of Alzheimer's diseases. We have previously purified from Achilleafragrantissimatwo active compounds: a protective flavonoid named 3,5,4’-trihydroxy-6,7,3’-trimethoxyflavone (TTF, Fl-72/2) and an anti-inflammatory sesquiterpenelactone named achillolide A (AcA). Major conclusions, solutions, achievements. In this study we could show that TTF and AcA protected cultured astrocytes from H₂O₂ –induced cell death via interference with cell signaling events. TTF inhibited SAPK/JNK, ERK1/2, MEK1 and CREBphosphorylation, while AcA inhibited only ERK1/2 and MEK1 phosphorylation. In addition to its protective activities, TTF had also anti-inflammatory activities, and inhibited the LPS-elicited secretion of the proinflammatorycytokinesInterleukin 6 (IL-6) and IL-1b from cultured microglial cells. Moreover, TTF and AcA protected neuronal cells from glutamate and Abcytotoxicity by reducing the glutamate and amyloid beta induced levels of intracellular reactive oxygen species (ROS) and via interference with cell signaling events induced by Ab. These compounds also reduced amyloid precursor protein net processing in vitro and in vivo in a mouse model for Alzheimer’s disease and improvedperformance in the novel object recognition learning and memory task. Conclusion: TTF and AcA are potential candidates to be developed as drugs or food additives to prevent, postpone or ameliorate Alzheimer’s disease. Implications, both scientific and agricultural.The synthesis ofAcA and TTF is very complicated. Thus, the plant itself will be the source for the isolation of these compounds or their precursors for synthesis. Therefore, Achilleafragrantissima could be developed into a new crop with industrial potential for the Arava-Negev area in Israel, and will generate more working places in this region.
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Friedlander, Michael, Clinton Dawes, and Y. (Joel) Kashman. The Interaction between Epiphytes and Seaweeds. United States Department of Agriculture, June 1995. http://dx.doi.org/10.32747/1995.7571355.bard.

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Two Israeli laboratories (IOLR and TAU) cooperated with one American laboratory (USF) in the research of the interaction between epiphytes (Ulva sp.) and the cultivated seaweed (Gracilaria sp.) The main objectives included the following aspects: Structural aspects, effects of different irradiances on growth, sensitivity studies, allelopathic excretions, selective chemicals and integration of studies of epiphytization. The studies were operated in outdoor tanks, indoor growth chambers and in the lab. The main conclusions and their relevance for mariculture are as following: 1. The green algal epiphyte, does penetrate its red algal host. 2. Gracilaria spp. in monoculture released more halogenated hydrocarbons than in biculture with U lactuca, whereas other metabolic parameters did not show a discriminating effect in biculture. 3. Hydrogen peroxide and halogenated hydrocarbons could be a part of the effective excretion compounds in biculture. 4. The presence of mature Gracilaria inhibited the growth of U. lactuca sporelings. 5. G. conferta is most sensitive to epiphytes among Gracilaria species tested. 6. The use of green light can enhance growth in basiphytes but inhibit epiphytes. 7. Effective selectivity has been defined by the use of hydrogen hypochlorite. 8. It may be more profitable in seaweed mariculture to select for epiphyte resistant strains than to search for inhibitors of epiphytization. 9 It is important as well to examine how the basiphyte may be able to prevent penetration. 10. Definition of the effective excretions in biculture has still to be done.
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Menaker, Michael. Effects of the TAU Mutation on Circadian Organization. Fort Belvoir, VA: Defense Technical Information Center, November 1998. http://dx.doi.org/10.21236/ada379434.

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Ricciulli-Marín, Diana. The Fiscal Cost of Conflict: Evidence from La Violencia in Colombia. Banco de la República de Colombia, December 2020. http://dx.doi.org/10.32468/chee.53.

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This paper studies the effect of internal conflict on local fiscal capacity using evidence from Colombia’s political conflict in the mid-20th century, better known as La Violencia. Following a difference-in-differences strategy, I find that internal conflict has negative long-term consequences in local fiscal capacity. More precisely, municipalities affected by La Violencia experienced an average reduction of 10.3% in their tax revenue and a fall of 2.8 percentage points on their ratio of taxes to total revenue. Effects lasted for more than a decade and are only partially explained by a population and economic activity downturn. These results are consistent with previous evidence indicating a negative effect of violence on tax collection efficiency at the local level.
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Frenkel, Jacob, and Assaf Razin. International Effects of Tax Reforms. Cambridge, MA: National Bureau of Economic Research, March 1989. http://dx.doi.org/10.3386/w2873.

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Williams, Roberton. Health Effects in a Model of Second-Best Environmental Taxation or Reconsidering "Reconsidering the Tax-Interaction Effect". Cambridge, MA: National Bureau of Economic Research, December 2000. http://dx.doi.org/10.3386/w8048.

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Desai, Mihir, C. Fritz Foley, and James Hines. Economic Effects of Regional Tax Havens. Cambridge, MA: National Bureau of Economic Research, October 2004. http://dx.doi.org/10.3386/w10806.

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Shaw, Jonathan, Arun Advani, and William Elming. The dynamic effects of tax audits. The IFS, October 2017. http://dx.doi.org/10.1920/wp.ifs.2017.w1724.

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Cogan, John, R. Glenn Hubbard, and Daniel Kessler. The Effect of Tax Preferences on Health Spending. Cambridge, MA: National Bureau of Economic Research, January 2008. http://dx.doi.org/10.3386/w13767.

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Gu, Yuanyuan, and Jhorland Ayala-García. Emigration and Tax Revenue. Banco de la República de Colombia, July 2022. http://dx.doi.org/10.32468/dtseru.312.

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According to the World Migration Report 2020, the number of international migrants increased from 84 million in 1970 to 272 million in 2019, accounting for 3.5% of the world’s population. This paper investigates the aggregated effect of emigration on the tax revenue of sending countries with a focus on developing nations. Using a gravity approach, we construct a time-varying exogenous instrument out of geographic time-invariant dyadic characteristics that allow us to estimate the predicted emigration rate for every country. Then, we follow an instrumental variable approach where we use our predicted emigration rate as an instrument of the observed migration rate. The results show that the predicted emigration rate is a good instrument of the current emigration rate for developing countries, and that there is a positive aggregated effect of emigration on tax revenue of sending countries. The results vary depending on the type of tax: emigration increases goods and services tax revenue, but it decreases income, profit, and capital gains tax revenue.
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