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Relevant bibliographies by topics / T cells responses

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Academic literature on the topic 'T cells responses'

Author: Grafiati

Published: 14 March 2023

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Journal articles on the topic "T cells responses"

1

Tadokoro, Carlos E., Guy Shakhar, Shiqian Shen, et al. "Regulatory T cells inhibit stable contacts between CD4+ T cells and dendritic cells in vivo." Journal of Experimental Medicine 203, no. 3 (2006): 505–11. http://dx.doi.org/10.1084/jem.20050783.

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Regulatory T (T reg) cells exert powerful down-modulatory effects on immune responses, but it is not known how they act in vivo. Using intravital two-photon laser scanning microscopy we determined that, in the absence of T reg cells, the locomotion of autoantigen-specific T cells inside lymph nodes is decreased, and the contacts between T cells and antigen-loaded dendritic cells (DCs) are of longer duration. Thus, T reg cells can exert an early effect on immune responses by attenuating the establishment of stable contacts during priming of naive T cells by DCs.

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Krawczyk, Connie M., Hao Shen, and Edward J. Pearce. "Memory CD4 T Cells Enhance Primary CD8 T-Cell Responses." Infection and Immunity 75, no. 7 (2007): 3556–60. http://dx.doi.org/10.1128/iai.00086-07.

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ABSTRACT CD4 T-cell help is required for optimal memory CD8 T-cell responses. We have found that engaging preexisiting CD4 Th1, but not Th2, memory cells at the time of CD8 T-cell priming results in increased CD8 effector responses to both bacterial and viral pathogens. The enhanced responses are characterized by increased numbers of cytokine-producing, antigen-specific cells. These findings suggest that engaging endogenous memory Th1 cells may increase cellular responses in an immunotherapy or vaccination setting.

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Jiang, Xiaodong, Rachael Clark, Luzheng Liu, et al. "CD4+ T cells regulate skin resident memory CD8+ T cells (TRM) (49.8)." Journal of Immunology 188, no. 1_Supplement (2012): 49.8. http://dx.doi.org/10.4049/jimmunol.188.supp.49.8.

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Abstract The requirement of CD4+ T cell help for CD8+ T cell responses has been extensively explored in secondary lymphoid organs using systemic infection models. Whether such help also exists in peripheral tissues like the skin is unknown. We recently reported that skin resident memory CD8+ T cells (TRM) are non-recirculating and are superior to central memory CD8+ T cells (TCM) in protecting against re-infection. Interestingly, we also found that the acute CD8+ T cell migration into infected skin does not require CD4+ T cell help. Further study showed that the acute protection response of CD

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Chauhan, Priyanka, Shuxian Hu, Wen S. Sheng, and James R. Lokensgard. "Regulatory T-Cells Suppress Cytotoxic T Lymphocyte Responses against Microglia." Cells 11, no. 18 (2022): 2826. http://dx.doi.org/10.3390/cells11182826.

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Regulatory T-cells (Tregs) play pivotal roles during infection, cancer, and autoimmunity. In our previous study, we demonstrated a role for the PD-1:PD-L1 pathway in controlling cytolytic responses of CD8+ T lymphocytes against microglial cells presenting viral peptides. In this study, we investigated the role of Tregs in suppressing CD8+ T-cell-mediated cytotoxicity against primary microglial cells. Using in vitro cytotoxicity assays and flow cytometry, we demonstrated a role for Tregs in suppressing antigen-specific cytotoxic T-lymphocyte (CTL) responses against microglia loaded with a model

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Weis-Banke, Stine Emilie, Thomas Landkildehus Lisle, Maria Perez-Penco, et al. "Arginase-2-specific cytotoxic T cells specifically recognize functional regulatory T cells." Journal for ImmunoTherapy of Cancer 10, no. 10 (2022): e005326. http://dx.doi.org/10.1136/jitc-2022-005326.

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BackgroundHigh expression of the metabolic enzyme arginase-2 (ARG2) by cancer cells, regulatory immune cells, or cells of the tumor stroma can reduce the availability of arginine (L-Arg) in the tumor microenvironment (TME). Depletion of L-Arg has detrimental consequences for T cells and leads to T-cell dysfunction and suppression of anticancer immune responses. Previous work from our group has demonstrated the presence of proinflammatory ARG2-specific CD4 T cells that inhibited tumor growth in murine models on activation with ARG2-derived peptides. In this study, we investigated the natural oc

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Wong, Wei. "Activating T cells with asparagine." Science Signaling 14, no. 668 (2021): eabg8244. http://dx.doi.org/10.1126/scisignal.abg8244.

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Olson, Matthew, and Stephen Turner. "CD4 T cells license dendritic cells to enhance polyfunctional anti-viral T helper cell responses (P6094)." Journal of Immunology 190, no. 1_Supplement (2013): 173.3. http://dx.doi.org/10.4049/jimmunol.190.supp.173.3.

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Abstract CD4 T cell help is critical for the generation of optimal B cell and CD8 T cell responses after immunization or infection. Robust and polyfunctional CD4 T cells are also generated after exposure to antigen, however, the factors that govern the development of potent CD4 T cell immunity are less clear. We demonstrate here that augmenting CD4 T cell help by adoptive transfer of increasing numbers of influenza A virus (IAV)-specific T cell receptor transgenic T cells resulted in enhanced polyclonal IAV-specific CD4 T cell responses after infection. Virus-specific CD4 T cells in mice that

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Schilbach, Karin, Hendrik Ziegler, Jan Haarer, Marco Sterk, Hans-Georg Rammensee та Rupert Handgretinger. "Human peripheral Vδ1+ γδ T cells can develop into αβ T cells (P4460)". Journal of Immunology 190, № 1_Supplement (2013): 52.47. http://dx.doi.org/10.4049/jimmunol.190.supp.52.47.

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Abstract The T cell compartment can be divided in two large classes: αβ and γδ T cells. It is believed that they develop in the thymus from a common progenitor and that the choice between αβ and γδ T cell fate is the first lineage decision made by progenitors after they commit to the T-cell lineage. However, here we show that peripheral Vδ1+ γδ T cells in an inflammatory environment can transdifferentiate into αβ T cells. Upon their extrathymic route of differentiation, that resembles well-characterized molecular program of thymic αβ lineage development, Vδ1+ T cells upregulate CD4+ coreceptor

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Plitas, George, and Alexander Y. Rudensky. "Regulatory T Cells in Cancer." Annual Review of Cancer Biology 4, no. 1 (2020): 459–77. http://dx.doi.org/10.1146/annurev-cancerbio-030419-033428.

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The immune system has evolved complex effector mechanisms to protect the host against a diversity of pathogenic organisms and regulatory adaptations that can curtail pathological sequelae of inflammatory events, prevent autoimmunity, and assist in tissue repair. Cancers, by virtue of their local manifestations of tissue dysfunction and destruction, inflammation, and genomic instability, can evoke these protective mechanisms, which support the progression of tumors and prevent their immune eradication. Central to these processes is a subset of CD4+ T cells, known as regulatory T (Treg) cells, t

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Kursar, Mischo, Kerstin Bonhagen, Joachim Fensterle, et al. "Regulatory CD4+CD25+ T Cells Restrict Memory CD8+ T Cell Responses." Journal of Experimental Medicine 196, no. 12 (2002): 1585–92. http://dx.doi.org/10.1084/jem.20011347.

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CD4+ T cell help is important for the generation of CD8+ T cell responses. We used depleting anti-CD4 mAb to analyze the role of CD4+ T cells for memory CD8+ T cell responses after secondary infection of mice with the intracellular bacterium Listeria monocytogenes, or after boost immunization by specific peptide or DNA vaccination. Surprisingly, anti-CD4 mAb treatment during secondary CD8+ T cell responses markedly enlarged the population size of antigen-specific CD8+ T cells. After boost immunization with peptide or DNA, this effect was particularly profound, and antigen-specific CD8+ T cell

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Dissertations / Theses on the topic "T cells responses"

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Crawford, A. "How B cells influence T cell responses." Thesis, University of Edinburgh, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.645118.

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Although studies using B cell deficient mice have been useful in understanding the importance of B cells under different conditions, it is difficult to then dissect exactly how B cells could be regulating T cell responses. By transferring OT-II transgenic T cells into either B cell deficient (μMT) or C57BL/6 mice, expansion and contraction of T cells can be tracked <i>ex vivo. </i>Expansion of OT-II cells is reduced in μMT mice compared to C57BL/6 mice. Thus, B cells can provide costimulatory signals, secrete cytokines and influence the lymphoid microarchitecture. To dissect which B cell facto

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Crawford, Alison. "Role of B cells in influencing T cell responses." Thesis, University of Edinburgh, 2004. http://hdl.handle.net/1842/13483.

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Smith, Trevor Robert Frank. "Modulation of CD4+ T cell responses by CD4+CD25+ regulatory T cells and modified T cell epitopes." Thesis, Imperial College London, 2004. http://hdl.handle.net/10044/1/11317.

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Preciado-Llanes, Lorena. "Inhibition of T-cell responses by microbes and immune cells." Thesis, University of Sheffield, 2013. http://etheses.whiterose.ac.uk/4717/.

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Some antigens can induce B-cell activation and antibody production without 'T cell help' and hence are called T-independent (TI) antigens. Similarly to bacterial capsular polysaccharides, anti- IgDconjugated dextran (α-δ-DEX) is a TI type 2 mimic which stimulates B lymphocytes by crosslinking of numerous B-cell receptor molecules. This thesis demonstrates that α-δ-DEX-activated B-cells directly inhibit TCR-induced CD4+ T-cell proliferation and activation in vitro. Experiments performed with purified cell populations excluded the possibility of α-δ-DEX acting directly on CD4+ T lymphocytes and

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Barroso, Herrera Osquel Miguel. "Manipulation of antigen-specific T cell responses by modified dendritic cells." Thesis, Imperial College London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.405941.

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Li, Xiaoying. "T cell receptor repertoires of immunodominant CD8 T cell responses to Theileria parva." Thesis, University of Edinburgh, 2015. http://hdl.handle.net/1842/19552.

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Previous research has provided evidence that CD8 T cells mediate immunity against infection with Theileria parva. However, the immunity induced by one parasite strain doesn‟t give complete protection against other strains and this is associated with parasite strain specificity of the CD8 T cell responses. There is evidence that such strain specificity is a consequence of the CD8 T cell responses of individual animals being focused on a limited number of immunodominant polymorphic peptide-MHC determinants. Dominant responses to the Tp2 antigen have been demonstrated in animals homozygous for th

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Wangteeraprasert, Apirath. "CD8+ T-cells responses in Dengue virus infection." Thesis, Imperial College London, 2012. http://hdl.handle.net/10044/1/39398.

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Dengue virus, which has four serotypes, has several clinical manifestations including asymptomatic infection, a self-limiting febrile illness termed dengue fever (DF) and a severe form characterized by plasma leakage termed dengue hemorrhagic fever (DHF). The pathogenesis of DHF is not fully understood and many studies have shown that it is more prevalent during secondary infection. In addition to a mechanism termed antibody dependent enhancement (ADE), the role of T-cells in the pathogenesis of dengue also has been investigated. It has been hypothesized that upon secondary infection dengue-sp

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Deng, Jun, and 鄧軍. "Leptin modulates T cells responses in autoimmune arthritis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/208601.

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Leptin, a protein hormone encoded by obese (ob) gene, is mainly produced by adipocytes. Leptin plays an important role in regulating neuroendocrine function and energy metabolism. As a cytokine, leptin is involved in modulating the hematopoiesis and lymphopoiesis. Although leptin has been found to promote T cells activation, it is largely unclear whether and how leptin regulates T cell differentiation and function. Leptin has been associated with disease severity in rheumatoid arthritis (RA). Elevated leptin levels have been detected in the sera and synovial fluid of active RA patients. Th1

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Sutherland, Andrew Peter Robert St Vincents Clinical School UNSW. "BAFF regulation of peripheral T cell responses." Awarded by:University of New South Wales. St Vincents Clinical School, 2005. http://handle.unsw.edu.au/1959.4/22788.

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The activation and effector function of CD4+ T cells are critical points of regulation during an antigen specific T cell response. Dysregulation of these processes can lead to the development of human diseases, encompassing both immunodeficiency and autoimmunity. Members of the TNF superfamily have recently emerged as important regulators of T cell responses, with their overexpression causing autoimmune inflammation in animal models. As overproduction of the novel TNF superfamily ligand BAFF is associated with several autoimmune conditions, we sought to examine the potential role of BAFF as a

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Easterfield, Alistair John. "Foetal modulation of maternal T lymphocyte responses." Thesis, St George's, University of London, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.325057.

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Books on the topic "T cells responses"

1

Tomkins, Paul Thomas. Interferon modulation of T-cell responses to Semliki Forest virus infected murine brain cells. typescript, 1989.

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Dockery, Dee. T cell responses to OspA, a candidate Lyme Disease vaccine. s.n.], 1993.

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Nagorsen, Dirk, and F. M. Marincola, eds. Analyzing T Cell Responses. Springer Netherlands, 2005. http://dx.doi.org/10.1007/1-4020-3623-x.

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Brouwenstijn, Nathalie. Characterization of the T-cell mediated immune response to renal cell carcinoma. University of Leiden], 1998.

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Regulatory T cells: Methods and protocols. Humana Press, 2011.

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Liu, Yang. The costimulatory pathway for T cell response. R.G. Landes, 1994.

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L, Edelson Richard, ed. Antigen and clone-specific immunoregulation. New York Academy of Sciences, 1991.

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Na, Songqing, and Chandrasekar Venkataraman Iyer. Effector CD4+ T cells in health and disease 2007. Transworld Research Network, 2007.

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Alexander, Michael A. Immune-based cancer treatment: The T lymphocyte response. CRC Press, 2011.

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O'Leary, Paula Frances Gerardine. T cell influences in antibody responses to lipopolysaccharides and polysaccharides. University of Birmingham, 2000.

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Book chapters on the topic "T cells responses"

1

Munitic, Ivana, César Evaristo, Hsueh Cheng Sung, and Benedita Rocha. "Transcriptional Regulation during CD8 T-Cell Immune Responses." In Memory T Cells. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-6451-9_2.

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Camus, Matthieu, and Jérôme Galon. "Memory T-Cell Responses and Survival in Human Cancer: Remember to Stay Alive." In Memory T Cells. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-6451-9_13.

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Sieper, Joachim, and Jürgen Braun. "T cell responses in reactive and Lyme arthritis." In T Cells in Arthritis. Birkhäuser Basel, 1998. http://dx.doi.org/10.1007/978-3-0348-8823-3_9.

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Sieper, Joachim, and Jürgen Braun. "T cell responses in reactive and Lyme arthritis." In T Cells in Arthritis. Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-4285-8_9.

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Alderson, Kory L., and William J. Murphy. "Antigen Specific Memory T Cells and Their Putative Need for the Generation of Sustained Anti-Tumor Responses." In Memory T Cells. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-6451-9_12.

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Young, Betty M., Susan Wiechert, Ruth A. Coleman, Prajwal Gurung, and Robert T. Cook. "Polyclonal and Antigen-specific Responses of T Cells and T Cell Subsets." In Alcohol. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-242-7_19.

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Zingoni, Alessandra, Cristina Cerboni, Michele Ardolino, and Angela Santoni. "Modulation of T Cell-Mediated Immune Responses by Natural Killer Cells." In Natural Killer Cells. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-02309-5_17.

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Maltzman, Jonathan S., Angus Thomson, and David M. Rothstein. "T cells and the principles of immune responses." In Transplant Immunology. John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781119072997.ch6.

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Tournilhac, Olivier, and Peter Dreger. "Chronic Lymphocytic Leukaemia." In The EBMT/EHA CAR-T Cell Handbook. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-94353-0_14.

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AbstractAlthough chronic lymphocytic leukaemia (CLL) was one of the first two entities in which CAR-T cells were evaluated, it has not yet arrived in the clinical routine. Since the landmark study by Porter et al. (2011), only six CLL-specific clinical trials have been published, altogether comprising no more than 155 patients (Porter et al. 2015; Gill et al. 2018; Turtle et al. 2017; Gauthier et al. 2020; Siddiqi et al. 2020; Wierda et al. 2020; Frey et al. 2020). All six of these studies investigated CD19-directed CAR-T constructs in heavily pretreated patients, mostly having failed BTKi+/− venetoclax therapy. Despite overall response rates of 60–95%, including MRD clearance in a large proportion of patients, the CR rates appear to be relatively low, and only a few durable responses have been reported in patients achieving a CR (Porter et al. 2015; Frey et al. 2020; Cappell et al. 2020). While toxicity includes 5–20% grade 3 cytokine release syndrome and 5–25% grade 3 neurotoxicity and appears manageable, long-term efficacy remains an unresolved issue. CLL-specific efficacy barriers for CD19 CAR-T cells could include a reduced capacity for sustained T cell expansion in extensively pretreated elderly CLL patients (Lemal and Tournilhac 2019), along with impaired T cell motility, impaired T cell mitochondrial fitness, and T cell exhaustion (Bair and Porter 2019). Concurrent use of ibrutinib might reduce the CRS rate and severity (Gauthier et al. 2020; Gill et al. 2018; Wierda et al. 2020) without impairing CAR-T cell expansion.

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Mage, Rose G., and Claire Rogel-Gaillard. "Immunogenetics in the rabbit." In The genetics and genomics of the rabbit. CABI, 2021. http://dx.doi.org/10.1079/9781780643342.0005.

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Abstract This chapter on immunogenetics in the rabbit focused on some genes with genetic and genomic sequence information including those encoding: soluble circulating immunoglobulin molecules (Igs) and their surface-bound forms on B lymphocytes (BCRs); T-cell receptors on T lymphocyte surfaces, (TCRs); the rabbit Leukocyte Antigen (RLA) complex (proteins on cells that function to present antigen fragments to TCRs); and some cytokine genes that encode key regulators of T- and B-cell responses.

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Conference papers on the topic "T cells responses"

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Pillay, Janesh, Vera M. Kamp, Tjaakje Visser, et al. "Myeloid Derived Suppressor Cells Inhibit T-cell Responses In Critically Ill Patients." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a6148.

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Rastogi, Ichwaku, and Douglas McNeel. "271 B cell primed CD8 T cells generate similar phenotype, function and anti-tumor responses to DC primed CD8 T cells." In SITC 37th Annual Meeting (SITC 2022) Abstracts. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/jitc-2022-sitc2022.0271.

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Wallington, Joshua, Anthony P. Williams, Karl J. Staples, and Tom M. A. Wilkinson. "Cytotoxic responses of mucosal-associated Invariant T cells to NTHi infection." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa2621.

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Kedmi, Ranit, Kai Mesa, and Dan Littman. "Abstract B167: Antigen-presenting cells as coordinators of T-cell responses to gut microbiota." In Abstracts: Fourth CRI-CIMT-EATI-AACR International Cancer Immunotherapy Conference: Translating Science into Survival; September 30 - October 3, 2018; New York, NY. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/2326-6074.cricimteatiaacr18-b167.

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Kamran, Neha, Hikmat Assi, Marianela Candolfi, et al. "Abstract 1085: Glioma-infiltrating myeloid derived suppressor cells inhibit anti-tumor T cell responses." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-1085.

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Lin, Ko Wei, Takeshi Nakajima, Kai Yu Jen, Erika C. Crouch, David L. Perkins, and Patricia W. Finn. "Surfactant Protein D Modulation Of Adaptive Immune Responses: Role Of T Cells." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a5161.

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Gavin, Marc A., Alexander Gragerov, Erik Espling, et al. "Abstract B45: Phosphatidylserine suppresses T cells through GPR174, and co-inhibition of adenosine receptors and GPR174 synergistically enhances T cell responses." In Abstracts: AACR Special Conference on Tumor Immunology and Immunotherapy; November 17-20, 2019; Boston, MA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/2326-6074.tumimm19-b45.

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Kamran, Neha, Youping Li, Mariela Moreno-Ayala, et al. "Abstract 457: Depletion of glioma infiltrating myeloid derived suppressor cells promotes anti-tumor T cell responses." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-457.

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Wang, Zhaoming, and George J. Weiner. "Abstract 1687: T cells are required to maintain anti-CD20-mediated NK cell responses in vitro." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-1687.

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Szylar, G., and J. Brown. "S10 Suppression of macrophage inflammatory responses to streptococcus pneumoniae by regulatory t cells." In British Thoracic Society Winter Meeting 2017, QEII Centre Broad Sanctuary Westminster London SW1P 3EE, 6 to 8 December 2017, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2017. http://dx.doi.org/10.1136/thoraxjnl-2017-210983.16.

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Reports on the topic "T cells responses"

1

Wong, Jr, and K. K. Regulatory T Cells and Host Anti-CML Responses. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada487614.

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Wong, Jr, and K. K. Regulatory T Cells and Host Anti-CML Responses. Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada510759.

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Beg, Amer A. Potentiation of T Lymphocyte Responses by Modulating NF-kB Activity in Dendritic Cells. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada437633.

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Beg, Amer A. Potentiation of T Lymphocyte Responses by Modulating NF - Kappa Beta Activity in Dendritic Cells. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada417929.

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Baszler, Timothy, Igor Savitsky, Christopher Davies, Lauren Staska, and Varda Shkap. Identification of bovine Neospora caninum cytotoxic T-lymphocyte epitopes for development of peptide-based vaccine. United States Department of Agriculture, 2006. http://dx.doi.org/10.32747/2006.7695592.bard.

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The goal of the one-year feasibility study was to identify specific cytotoxic T-lymphocyte (CTL) epitopes to Neosporacaninum in the natural bovine host in order to make progress toward developing an effective peptide-based vaccine against bovine neosporosis. We tested the hypothesis that: N. caninum SRS2 peptides contain immunogenicCTLepitope clusters cross-presented by multiple bovine MHC-I and MHC-IIhaplotypes. The specific objectives were: (1) Map bovine CTLepitopes of N. caninum NcSRS-2 and identify consensus MHC-I and class-II binding motifs; and (2) Determine if subunit immunization with

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Fluhr, Robert, and Maor Bar-Peled. Novel Lectin Controls Wound-responses in Arabidopsis. United States Department of Agriculture, 2012. http://dx.doi.org/10.32747/2012.7697123.bard.

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Innate immune responses in animals and plants involve receptors that recognize microbe-associated molecules. In plants, one set of this defense system is characterized by large families of TIR–nucleotide binding site–leucine-rich repeat (TIR-NBS-LRR) resistance genes. The direct interaction between plant proteins harboring the TIR domain with proteins that transmit and facilitate a signaling pathway has yet to be shown. The Arabidopsis genome encodes TIR-domain containing genes that lack NBS and LRR whose functions are unknown. Here we investigated the functional role of such protein, TLW1 (TI

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Medof, M. E. Augmentation of Antitumor T-Cell Responses by Increasing APC T-Cell C5a/C3a-C5aR/C3aR Interactions. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada585489.

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Palmer, Guy, Varda Shkap, Wendy Brown, and Thea Molad. Control of bovine anaplasmosis: cytokine enhancement of vaccine efficacy. United States Department of Agriculture, 2007. http://dx.doi.org/10.32747/2007.7695879.bard.

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Anaplasmosis an arthropod-born disease of cattle caused by the rickettsia Anaplasma marginale and is an impediment to efficient production of healthy livestock in both Israel and the United States. Currently the only effective vaccines are derived from the blood of infected cattle. The risk of widespread transmission of both known and newly emergent pathogens has prevented licensure of live blood-based vaccines in the U.S. and is a major concern for their continued use in Israel. Consequently development of a safe, effective vaccine is a high priority. In this collaborative project we focused

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Eldar, Avigdor, and Donald L. Evans. Streptococcus iniae Infections in Trout and Tilapia: Host-Pathogen Interactions, the Immune Response Toward the Pathogen and Vaccine Formulation. United States Department of Agriculture, 2000. http://dx.doi.org/10.32747/2000.7575286.bard.

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In Israel and in the U.S., Streptococcus iniae is responsible for considerable losses in various fish species. Poor understanding of its virulence factors and limited know-how-to of vaccine formulation and administration are the main reasons for the limited efficacy of vaccines. Our strategy was that in order to Improve control measures, both aspects should be equally addressed. Our proposal included the following objectives: (i) construction of host-pathogen interaction models; (ii) characterization of virulence factors and immunodominant antigens, with assessment of their relative importance

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Banai, Menachem, and Gary Splitter. Molecular Characterization and Function of Brucella Immunodominant Proteins. United States Department of Agriculture, 1993. http://dx.doi.org/10.32747/1993.7568100.bard.

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The BARD project was a continuation of a previous BARD funded research project. It was aimed at characterization of the 12kDa immunodominant protein and subsequently the cloning and expression of the gene in E. coli. Additional immunodominant proteins were sought among genomic B. abortus expression library clones using T-lymphocyte proliferation assay as a screening method. The 12kDa protein was identified as the L7/L12 ribosomal protein demonstrating in the first time the role a structural protein may play in the development of the host's immunity against the organism. The gene was cloned fro

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