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Journal articles on the topic 'T-CAD'

Author: Grafiati

Published: 11 September 2023

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1

Uhm, Tae-Kyoung, Ki-Seung Kim, Yu-Deok Seo, and Sung-Kie Youn. "T-spline Finite Element Method for CAD/CAE Integrated Approach." Transactions of the Korean Society of Mechanical Engineers A 33, no. 2 (2009): 127–34. http://dx.doi.org/10.3795/ksme-a.2009.33.2.127.

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2

Netto, Jeffrey, Andrej Teren, Ralph Burkhardt, et al. "Biomarkers for Non-Invasive Stratification of Coronary Artery Disease and Prognostic Impact on Long-Term Survival in Patients with Stable Coronary Heart Disease." Nutrients 14, no. 16 (2022): 3433. http://dx.doi.org/10.3390/nu14163433.

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Knowledge about cardiac and inflammatory biomarkers in patients with stable coronary artery disease (CAD) is limited. To address this, we analyzed 3072 patients (36% female) with a median follow-up of 10 years in the Leipzig LIFE Heart Study with suspected CAD with coronary angiography. Selected biomarkers included troponin T (hsTNT), N-terminal pro B-type natriuretic peptide (NT-proBNP), copeptin, C-reactive protein (hsCRP), and interleukin-6 (IL-6). Patients were stratified by CAD severity: CAD0 (no sclerosis), CAD1 (non-obstructive, i.e., stenosis < 50%), and CAD2 (≥one stenosis ≥ 50%).

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Lloyd, Peter, Colin C. McAndrew, Michael J. McLennan, et al. "Technology CAD at AT&T." Microelectronics Journal 26, no. 2-3 (1995): 79–97. http://dx.doi.org/10.1016/0026-2692(95)98915-e.

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4

Baltrūnienė, Vaida, Ieva Rinkūnaitė, Julius Bogomolovas, et al. "The Role of Cardiac T-Cadherin in the Indicating Heart Failure Severity of Patients with Non-Ischemic Dilated Cardiomyopathy." Medicina 56, no. 1 (2020): 27. http://dx.doi.org/10.3390/medicina56010027.

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Background and objectives: T-cadherin (T-cad) is one of the adiponectin receptors abundantly expressed in the heart and blood vessels. Experimental studies show that T-cad sequesters adiponectin in cardiovascular tissues and is critical for adiponectin-mediated cardio-protection. However, there are no data connecting cardiac T-cad levels with human chronic heart failure (HF). The aim of this study was to assess whether myocardial T-cad concentration is associated with chronic HF severity and whether the T-cad levels in human heart tissue might predict outcomes in patients with non-ischemic dil

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5

Fukuda, Shiro, Shunbun Kita, Kazuya Miyashita, et al. "Identification and Clinical Associations of 3 Forms of Circulating T-cadherin in Human Serum." Journal of Clinical Endocrinology & Metabolism 106, no. 5 (2021): 1333–44. http://dx.doi.org/10.1210/clinem/dgab066.

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Abstract Context T-cadherin (T-cad) is a glycosylphosphatidylinositol (GPI)-anchored cadherin that mediates adiponectin to induce exosome biogenesis and secretion, protect cardiovascular tissues, promote muscle regeneration, and stimulate therapeutic heart protection by transplanted mesenchymal stem cells. CDH13, the gene locus of T-cad, affects plasma adiponectin levels most strongly, in addition to affecting cardiovascular disease risk and glucose homeostasis. Recently, it has been suggested that T-cad exists in human serum, although the details are still unclear. Objective To validate the e

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6

Philippova, Maria, Danila Ivanov, Manjunath B. Joshi, et al. "Identification of Proteins Associating with Glycosylphosphatidylinositol- Anchored T-Cadherin on the Surface of Vascular Endothelial Cells: Role for Grp78/BiP in T-Cadherin-Dependent Cell Survival." Molecular and Cellular Biology 28, no. 12 (2008): 4004–17. http://dx.doi.org/10.1128/mcb.00157-08.

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ABSTRACT There is scant knowledge regarding how cell surface lipid-anchored T-cadherin (T-cad) transmits signals through the plasma membrane to its intracellular targets. This study aimed to identify membrane proteins colocalizing with atypical glycosylphosphatidylinositol (GPI)-anchored T-cad on the surface of endothelial cells and to evaluate their role as signaling adaptors for T-cad. Application of coimmunoprecipitation from endothelial cells expressing c-myc-tagged T-cad and high-performance liquid chromatography revealed putative association of T-cad with the following proteins: glucose-

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Khanna, Roopali, Avinash Bansal, Sudeep Kumar, et al. "Association between Endogenous Sex Hormones and Coronary Artery Disease in Postmenopausal Women." Indian Journal of Cardiovascular Disease in Women - WINCARS 06, no. 03 (2021): 168–73. http://dx.doi.org/10.1055/s-0041-1736250.

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Background Incidence of coronary artery disease (CAD) increases significantly in postmenopausal women, which is assumed to be an imbalance between serum androgen and estrogen levels. However, studies assessing serum sex hormones and CAD are few and have shown conflicting results. Objective To compare serum sex hormone levels and traditional risk factors among postmenopausal women with angiographically proven CAD and without CAD. Method The study included consecutive postmenopausal women undergoing coronary angiography in our institute from May 2016 to June 2017. The clinical and coronary angio

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8

Labay, V. A., and J. Bornemann. "CAD of T-septum waveguide evanescent-mode filters." IEEE Transactions on Microwave Theory and Techniques 41, no. 4 (1993): 731–33. http://dx.doi.org/10.1109/22.231675.

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9

Foster, Jeffrey C. "CAE/CAD at AT&T: An Introduction." AT&T Technical Journal 70, no. 1 (1991): 2–8. http://dx.doi.org/10.1002/j.1538-7305.1991.tb00493.x.

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10

Abu-Amero, Khaled K., Carol A. Wyngaard, and Nduna Dzimiri. "Prevalence and Role of Methylenetetrahydrofolate Reductase 677 C→T and 1298 A→C Polymorphisms in Coronary Artery Disease in Arabs." Archives of Pathology & Laboratory Medicine 127, no. 10 (2003): 1349–52. http://dx.doi.org/10.5858/2003-127-1349-paromr.

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Abstract Context.—Previous studies reported an association of 677 C→T and 1298 A→C methylenetetrahydrofolate reductase (MTHFR) variants with coronary artery disease (CAD). No previous studies concerning the prevalence of these 2 MTHFR variants or their possible association with CAD in Arabs are currently available in the literature. Objective.—To determine the prevalence of MTHFR variants and their potential relevance to CAD among Arabs. Design.—We used polymerase chain reaction and restriction enzyme digestion to determine the prevalence of these 2 MTHFR polymorphisms in 625 healthy blood don

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11

Joshi, Manjunath B., Danila Ivanov, Maria Philippova, Emmanouil Kyriakakis, Paul Erne, and Thérèse J. Resink. "A requirement for thioredoxin in redox-sensitive modulation of T-cadherin expression in endothelial cells." Biochemical Journal 416, no. 2 (2008): 271–80. http://dx.doi.org/10.1042/bj20080765.

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T-cad (T-cadherin), a glycosylphosphatidylinositol-anchored cadherin superfamily member, is expressed widely in the brain and cardiovascular system, and absent, decreased, or even increased, in cancers. Mechanisms controlling T-cad expression are poorly understood. The present study investigated transcriptional regulation of T-cad in ECs (endothelial cells). Conditions of oxidative stress (serum-deprivation or presence of H2O2) elevate T-cad mRNA and protein levels in ECs. Reporter gene analysis, using serially deleted T-cad promoter stretches ranging from −99 to −2304 bp, located the minimal

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12

Guerrero, Julien, Boris Dasen, Agne Frismantiene, et al. "T-cadherin Expressing Cells in the Stromal Vascular Fraction of Human Adipose Tissue: Role in Osteogenesis and Angiogenesis." Stem Cells Translational Medicine 11, no. 2 (2022): 213–29. http://dx.doi.org/10.1093/stcltm/szab021.

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Abstract Cells of the stromal vascular fraction (SVF) of human adipose tissue have the capacity to generate osteogenic grafts with intrinsic vasculogenic properties. However, cultured adipose-derived stromal cells (ASCs), even after minimal monolayer expansion, lose osteogenic capacity in vivo. Communication between endothelial and stromal/mesenchymal cell lineages has been suggested to improve bone formation and vascularization by engineered tissues. Here, we investigated the specific role of a subpopulation of SVF cells positive for T-cadherin (T-cad), a putative endothelial marker. We found

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13

Turner, Michael J., C. Siddiq Abdul-Alim, Richard A. Willis, Terrence L. Fisher, Edith M. Lord, and John G. Frelinger. "T-cell antigen discovery (T-CAD) assay: a novel technique for identifying T cell epitopes." Journal of Immunological Methods 256, no. 1-2 (2001): 107–19. http://dx.doi.org/10.1016/s0022-1759(01)00436-7.

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14

Song, Xingfa, Haidong Wang, Chao Wang, et al. "Association of Sirtuin Gene Polymorphisms with Susceptibility to Coronary Artery Disease in a North Chinese Population." BioMed Research International 2022 (February 18, 2022): 1–8. http://dx.doi.org/10.1155/2022/4294008.

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Aims. Coronary artery disease (CAD) represents the leading cause of death worldwide. Accumulating evidence also suggests that sirtuins (SIRTS) have been associated with CAD. The present study was aimed at investigating the association between 12 gene polymorphisms for SIRTs and the development of CAD in a Chinese population. Materials and Methods. 12 SNPs (rs12778366 ( T > C ), rs3758391 ( T > C ), rs3740051 ( A > G ), rs4746720 ( C > T ), rs7895833 ( G > A ), rs932658 ( A > C ) for SIRT1, rs2015 ( G > T ) for SIRT2, rs28365927 ( G > A ), rs11246020 ( C > T ) for SIR

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Ko, Eun-Ju, In-Jai Kim, Jeong-Yong Lee, et al. "Study of the Association between VEGF Polymorphisms and the Risk of Coronary Artery Disease in Koreans." Journal of Personalized Medicine 12, no. 5 (2022): 761. http://dx.doi.org/10.3390/jpm12050761.

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Coronary artery disease (CAD), a leading cause of death worldwide, has a complex etiology comprising both traditional risk factors (type 2 diabetes, dyslipidemia, arterial hypertension, and cigarette smoking) and genetic factors. Vascular endothelial growth factor (VEGF) notably contributes to angiogenesis and endothelial homeostasis. However, little is known about the relationship between CAD and VEGF polymorphisms in Koreans. The aim of this study is to investigate the associations of 2 VEGF promoter region polymorphisms (−1154G>A [rs1570360], −1498T>C [rs833061]) and 4 VEGF 3′-UTR pol

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Saigusa, Ryosuke, Jenifer Vallejo, Rishab Gulati, et al. "Sex Differences in Coronary Artery Disease and Diabetes Revealed by scRNA-Seq and CITE-Seq of Human CD4+ T Cells." International Journal of Molecular Sciences 23, no. 17 (2022): 9875. http://dx.doi.org/10.3390/ijms23179875.

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Despite the decades-old knowledge that males and people with diabetes mellitus (DM) are at increased risk for coronary artery disease (CAD), the reasons for this association are only partially understood. Among the immune cells involved, recent evidence supports a critical role of T cells as drivers and modifiers of CAD. CD4+ T cells are commonly found in atherosclerotic plaques. We aimed to understand the relationship of CAD with sex and DM by single-cell RNA (scRNA-Seq) and antibody sequencing (CITE-Seq) of CD4+ T cells. Peripheral blood mononuclear cells (PBMCs) of 61 men and women who unde

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Bytyçi, Ibadete, Liliana Alves, Oscar Alves, Carla Lopes, Gani Bajraktari, and Michael Y. Henein. "Left Ventricular Myocardial and Cavity Velocity Disturbances Are Powerful Predictors of Significant Coronary Artery Stenosis." Journal of Clinical Medicine 11, no. 20 (2022): 6185. http://dx.doi.org/10.3390/jcm11206185.

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Background and Aim: Dobutamine stress echocardiography (DSE) is a well-established noninvasive investigation for significant coronary artery disease (CAD). The aim of this study was to evaluate the accuracy of cardiac Doppler parameters in predicting CAD. Methods: We prospectively studied 103 consecutive patients with suspected CAD based on typical symptoms; 59 proved to have CAD, and 44 patients proved to have no-CAD (n = 44). All patients underwent a complete stress Doppler echocardiographic examination. Total isovolumic time (T-IVT) as a marker of cavity dyssynchrony and wall motion score i

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18

MASUDA, Hiroo. "Trends of Simulation Technolgies. T-CAD and Its Application." Journal of Japan Institute of Electronics Packaging 2, no. 3 (1999): 188–93. http://dx.doi.org/10.5104/jiep.2.188.

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19

Boyer-Nancy, B. "Le CAD améliore-t-il les performances en détection ?" Imagerie de la Femme 14, no. 2 (2004): 75–84. http://dx.doi.org/10.1016/s1776-9817(04)94787-6.

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20

Dong, Mengzhen, Shousheng Liu, Mengke Wang, Yifen Wang, Yongning Xin, and Shiying Xuan. "Relationship between AGT rs2493132 polymorphism and the risk of coronary artery disease in patients with NAFLD in the Chinese Han population." Journal of International Medical Research 49, no. 7 (2021): 030006052110192. http://dx.doi.org/10.1177/03000605211019263.

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Objective To investigate the relationship between angiotensin ( AGT) rs2493132 gene polymorphism and the risk of developing non-alcoholic fatty liver disease (NAFLD) and coronary artery disease (CAD) in the Chinese Han population. Methods Polymerase chain reaction was performed to determine AGT genotypes. Anthropometric and clinical data were investigated and statistically analyzed in the clinical laboratory department of Qingdao Municipal Hospital. Results The AGT rs2493132 CT + TT genotype was an important risk factor for CAD in patients with NAFLD and NAFLD + CAD in healthy controls. The AG

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21

Juilliére, Yves, Mirande Candito, Charles E. Adjalla, et al. "Association of MTRR A66G polymorphism (but not of MTHFR C677T and A1298C, MTR A2756G, TCN C776G) with homocysteine and coronary artery disease in the French population." Thrombosis and Haemostasis 94, no. 09 (2005): 510–15. http://dx.doi.org/10.1160/th05-04-0262.

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Summarymethylenetetrahydrofolate reductase polymorphism (MTHFR C677T) is an established determinant of homocysteine plasma level (t-Hcys) while its association with coronary artery disease (CAD) seems to be more limited. In contrast, the association of the substitutions A2756G of methionine synthase (MTR), A66G of methionine synthase reductase (MTRR) and C776G of transcobalamin (TCN) to both t-Hcys and CAD needs to be evaluated further. The objective was to evaluate the association of these polymorphisms with t-Hcys and CAD in a French population. We investigated the individual and combined ef

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Jha, Chandan K., Rashid Mir, Imadeldin Elfaki, et al. "Evaluation of the Association of Omentin 1 rs2274907 A>T and rs2274908 G>A Gene Polymorphisms with Coronary Artery Disease in Indian Population: A Case Control Study." Journal of Personalized Medicine 9, no. 2 (2019): 30. http://dx.doi.org/10.3390/jpm9020030.

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Coronary artery disease (CAD) is a major cause of death all over the world. CAD is caused by atherosclerosis which is induced by the interaction of genetic factors and environmental factors. Traditional environmental risk factors include hyperlipidemia, diabetes mellitus, lack of exercise, obesity, poor diet and others. Genome-wide association studies have revealed the association of certain gene polymorphisms with susceptibility to CAD. Omentin 1 is an adipokine secreted by the visceral adipose tissues and has been reported to have anti-inflammatory, cardioprotective, and enhances insulin sen

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Jha, Chandan K., Rashid Mir, Imadeldin Elfaki, et al. "Reply to Comment: Evaluation of the Association of Omentin 1 rs2274907 A > T and rs2274908 G > A Gene Polymorphisms with Coronary Artery Disease in Indian Population: A Case–Control Study." Journal of Personalized Medicine 10, no. 4 (2020): 194. http://dx.doi.org/10.3390/jpm10040194.

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Coronary artery disease (CAD) is a major cause of death all over the world. CAD is caused by atherosclerosis which is induced by the interaction of genetic factors and environmental factors. Genome-wide association studies have revealed the association of certain gene polymorphisms with susceptibility to CAD. Omentin 1 is an adipokine secreted by the visceral adipose tissues and has been reported to have anti-inflammatory, cardioprotective, and enhances insulin sensitivity. In this study, we examined the role of omentin-1 common single nucleotide polymorphisms (SNPs) (rs2274907 A > T and rs

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Mir, Rashid, Chandan k. Jha, Imadeldin Elfaki, et al. "Incidence of MicroR-4513C/T Gene Variability in Coronary Artery Disease - A case-Control Study." Endocrine, Metabolic & Immune Disorders - Drug Targets 19, no. 8 (2019): 1216–23. http://dx.doi.org/10.2174/1871530319666190417111940.

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Background: Genetic variants in pre-microRNA genes or the 3'UTR of miRNA target genes could influence miRNA-mediated regulation of gene expression and thus contribute to the susceptibility and prognosis of human diseases. Several studies have investigated the association of genetic variants in the seed region of miRNAs with cardiometabolic phenotypes .Therefore the aim of study was to investigate the potential association of miR-4513 rs2168518 C>T gene variability with the risk of developing CAD and its association with different cardiometabolic phenotypes in an Indian cohort to stratify CA

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Malisuwan, S., J. Sivaraks, P. Promkladpanao, and Y. Thamachareon. "CAD Model and Simulation of T-Shaped Microstrip Antenna for LTE 1800MHz Applications." International Journal of Future Computer and Communication 3, no. 5 (2014): 354–57. http://dx.doi.org/10.7763/ijfcc.2014.v3.326.

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Popova, A. A., I. N. Shubin, and R. E. Aliev. "CALS Technologies in Mechanical Engineering Using the Example of Developing Three-Dimensional Models of Typical Products." Vestnik Tambovskogo gosudarstvennogo tehnicheskogo universiteta 26, no. 4 (2020): 637–49. http://dx.doi.org/10.17277/vestnik.2020.04.pp.637-649.

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Three-dimensional models of the body parts were developed using the basic 3D modeling operations (rotation, extrusion, threading, etc.) of the T-FLEX CAD 3D CAD system, which made it possible to significantly simplify the work with assembly 3D models representing a complex structure consisting of a large number of parts. During the trial operation, the advantages of using the T-FLEX CAD 3D CAD system have been proven when working with complex 3D models. The advantages of using the program in the machine-building cluster are shown.

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Zhou, Liting, Lin Xie, Dongchun Zheng, et al. "Genetic Variants of CD40 Gene Are Associated with Coronary Artery Disease and Blood Lipid Levels." BioMed Research International 2016 (2016): 1–8. http://dx.doi.org/10.1155/2016/1693619.

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Objectives. The present study aimed to evaluate the effect ofCD40andCXCR4genes polymorphisms on CAD susceptibility and the blood lipid levels and history of cardiovascular risk factors in a Chinese Han population.Materials and Methods. A total of 583 unrelated patients with CAD and 540 controls were recruited. Two tag SNPs (rs4239702 and rs1535045) at theCD40locus and one tag SNP (rs2228014) at theCXCR4locus were genotyped using the SEQUENOM Mass-ARRAY system.Results. After adjusting the risk factors, the frequency of rs1535045-T allele was also higher in patients than controls. Haplotype anal

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Wang, Haidong, Chao Wang, Xingfa Song, Hai Liu, Yun Zhang, and Pei Jiang. "Association of FKBP5 polymorphisms with patient susceptibility to coronary artery disease comorbid with depression." PeerJ 8 (June 3, 2020): e9286. http://dx.doi.org/10.7717/peerj.9286.

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Background Coronary artery disease (CAD) and depression cause great burden to society and frequently co-occur. The exact mechanisms of this comorbidity are unclear. FK506-binding protein 51 (FKBP51) is correlated with cardiovascular disease and depression. The aim of this study was to determine the role of the seven single nucleotide polymorphisms (SNPs) of FKBP5 that code FKBP51, namely, rs1360780 (C>T), rs2817032 (T>C), rs2817035 (G>A), rs9296158 (G>A), rs9470079 (G>A), rs4713902 (T>C), and rs3800373 (C>T) in a patient’s susceptibility to comorbid CAD and depression. Met

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Ma, Xiaowei, Jia Huang, Difei Lu, et al. "Genetic Variability of the Glucose-Dependent Insulinotropic Peptide Gene Is Involved in the Premature Coronary Artery Disease in a Chinese Population with Type 2 Diabetes." Journal of Diabetes Research 2018 (March 25, 2018): 1–7. http://dx.doi.org/10.1155/2018/6820294.

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Background. Glucose-dependent insulinotropic polypeptide (GIP) is closely related to diabetes and obesity, both of which are confirmed to increase the risk of coronary artery disease (CAD). Our study aimed to investigate whether the polymorphisms in GIP genes could affect the risk of cardiovascular disease in type 2 diabetic patients in the Chinese Han population. Methods. We selected and genotyped two haplotype-tagging single nucleotide polymorphisms (tag-SNPs) (rs2291725 C>T, rs8078510 G>A) of GIP gene based on CHB data in HapMap Phase II database (r2<0.8). The case-control study of

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Elwazir, Mohamed Y., Mohammad H. Hussein, Eman A. Toraih, et al. "Association of Angio-LncRNAs MIAT rs1061540/MALAT1 rs3200401 Molecular Variants with Gensini Score in Coronary Artery Disease Patients Undergoing Angiography." Biomolecules 12, no. 1 (2022): 137. http://dx.doi.org/10.3390/biom12010137.

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Long non-coding RNAs (lncRNAs) have emerged as essential biomolecules with variable diagnostic and/or prognostic utility in several diseases, including coronary artery disease (CAD). We aimed for the first time to investigate the potential association of five angiogenesis-related lncRNAs (PUNISHER, SENCR, MIAT, MALAT1, and GATA6-AS) variants with CAD susceptibility and/or severity. TaqMan Real-Time genotyping for PUNISHER rs12318065A/C, SENCR rs12420823C/T, MIAT rs1061540C/T, MALAT1 rs3200401T/C, and GATA6-AS1 rs73390820A/G were run on the extracted genomic DNA from 100 unrelated patients with

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Liu, Qing-Li, Hiroyuki Kishi, Kenzo Ohtsuka, and Atsushi Muraguchi. "Heat shock protein 70 binds caspase-activated DNase and enhances its activity in TCR-stimulated T cells." Blood 102, no. 5 (2003): 1788–96. http://dx.doi.org/10.1182/blood-2002-11-3499.

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AbstractDNA fragmentation is a hallmark of cells undergoing apoptosis and is mediated mainly by the caspase-activated DNase (CAD or DNA-fragmentation factor 40 [DFF40]), which is activated when released from its inhibitor protein (ICAD or DFF45) upon apoptosis signals. Here we analyzed the effect of heat shock protein 70 (Hsp70) on CAD activity in T-cell receptor (TCR)–induced apoptosis using a T-cell line (TAg-Jurkat). Overexpression of Hsp70 significantly augmented the apoptotic cell death as well as DNA fragmentation in CD3/CD28- or staurosporine-stimulated cells. Following stimulation of c

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JANCIN, BRUCE. "Troponin T Test May Help Spot Risk in Stable CAD." Family Practice News 38, no. 11 (2008): 11. http://dx.doi.org/10.1016/s0300-7073(08)70704-7.

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Hansen, U., and S. B�ttgenbach. "T-CAD for the analysis and verification of processing sequences." Microsystem Technologies 10, no. 3 (2004): 193–98. http://dx.doi.org/10.1007/s00542-003-0348-1.

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Kim, In-Jai, Jeong-Yong Lee, Hyeon-Woo Park, et al. "Association between HOTAIR lncRNA Polymorphisms and Coronary Artery Disease Susceptibility." Journal of Personalized Medicine 11, no. 5 (2021): 375. http://dx.doi.org/10.3390/jpm11050375.

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Coronary artery disease (CAD), one of the most frequent causes of mortality, is the most common type of cardiovascular disease. This condition is characterized by the accumulation of plaques in the coronary artery, leading to blockage of blood flow to the heart. The main symptom of CAD is chest pain caused by blockage of the coronary artery and shortness of breath. HOX transcript antisense RNA gene (HOTAIR) is a long non-coding RNA which is well-known as an oncogene involved in various cancers, such as lung, breast, colorectal, and gastric cancer. We selected six single nucleotide polymorphism

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Mráz, Miloš, Anna Cinkajzlová, Jana Kloučková, et al. "Coronary Artery Disease Is Associated with an Increased Amount of T Lymphocytes in Human Epicardial Adipose Tissue." Mediators of Inflammation 2019 (February 7, 2019): 1–9. http://dx.doi.org/10.1155/2019/4075086.

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Immunocompetent cells including lymphocytes play a key role in the development of adipose tissue inflammation and obesity-related cardiovascular complications. The aim of the study was to explore the relationship between epicardial adipose tissue lymphocytes and coronary artery disease (CAD). To this end, we studied the content and phenotype of lymphocytes in peripheral blood, subcutaneous adipose tissue (SAT), and epicardial adipose tissue (EAT) in subjects with and without CAD undergoing elective cardiac surgery. Eleven subjects without CAD (non-CAD group) and 22 age-, BMI-, and HbA1C-matche

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Epstein, Rachel. "Prime models of computably enumerable degree." Journal of Symbolic Logic 73, no. 4 (2008): 1373–88. http://dx.doi.org/10.2178/jsl/1230396926.

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AbstractWe examine the computably enumerable (c.e.) degrees of prime models of complete atomic decidable (CAD) theories. A structure has degree d if d is the degree of its elementary diagram. We show that if a CAD theory T has a prime model of c.e. degree c, then T has a prime model of strictly lower c.e. degree b, where, in addition, b is low (b′ = 0′), This extends Csima's result that every CAD theory has a low prime model. We also prove a density result for c.e. degrees of prime models. In particular, if c and d are c.e. degrees with d < c and c not low2 (c″ > 0″), then for any CAD th

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Kaur, Kulwinder, and Chandan Kumar Jha. "Identify the Significant Hypostatic or Epistatic Effect Between the Various SNPs of LDLR gene through SNP-SNP Interaction and Haplotype Analysis - A Case Control Study." International Journal of Health Sciences and Research 12, no. 8 (2022): 255–62. http://dx.doi.org/10.52403/ijhsr.20220834.

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The coronary artery disease (CAD) is a leading cause of the morbidity and mortality worldwide and has also become a major public health burden in India in recent decades. The LDLR gene mutations have been reported associated with familial CVD and are one of the main risk factors for developing coronary artery disease (CAD). In the present study using the contingency chi-square test, statistically significant differences were observed between the CAD cases and matched healthy controls in the distribution of the genotypes of four out six SNPs investigated viz., rs688C>T (p=0.0031), rs1529729C

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Гущина, А. Р., та Д. В. Гулякин. "Сравнительный анализ CAD-систем". ТЕНДЕНЦИИ РАЗВИТИЯ НАУКИ И ОБРАЗОВАНИЯ 93, № 9 (2023): 104–6. http://dx.doi.org/10.18411/trnio-01-2023-462.

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В данной статье рассматриваются легкие, средние и тяжѐлые уровни CAD систем. Каждый уровень отличается функциональностью и сложностью решений поставленных задач. Представлено краткое описание и отличительные черты таких CAD-систем, как AutoCAD, КОМПАС, NanoCAD, T-FLEX CAD, SolidWorks, CATIA. Рассмотрены отличия программ по основным функциям. Выяснено, что в наше время рынок полон различных программ, и существует возможность выбрать программу, подходящую по необходимым критериям.

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Wang, Y. Y., R. Su, B. C. Li, et al. "AB0269 THE CHARACTERISTICS OF PERIPHERAL LYMPHOCYTE SUBSETS AND CYTOKINES LEVEL IN RHEUMATOID ARTHRITIS WITH CORONARY ARTERY DISEASE." Annals of the Rheumatic Diseases 79, Suppl 1 (2020): 1433.1–1433. http://dx.doi.org/10.1136/annrheumdis-2020-eular.2754.

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Background:Rheumatoid arthritis (RA) is a systemic autoimmune disease. It is characterized by highly disabling polyarthritis, but extra-articular features are also common and portend a poor prognosis. Compared with the general population, the incidence and mortality of cardiovascular disease in RA are significantly increased. Chronic autoimmune inflammation is the common pathogenesis of RA and coronary heart disease(CAD). We’ve proved that lymphocyte subsets imbalance and high cytokines expression play an important role in the occurrence and development of RA diseases. However, the level of ly

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Daoud, Mohamed S. "Variants of D9N, G188A, N291S, and 93 T/G Genes in patients with Coronary Artery Diseases." Medical Science and Discovery 8, no. 12 (2021): 698–705. http://dx.doi.org/10.36472/msd.v8i12.636.

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Objective: Our work aimed to study the relationship between LPL variants D9N, G188A, N291S, and 93 T/G genes and CAD in Saudi patients. Materials and Methods: We recruited 253 CAD patients, who underwent diagnostic coronary angiography, and 207 control subjects. Several biochemical and behavioral markers were obtained, and different genotypes of LPL variants, D9N, G188E, N291S, and 93 T/G, were detected using The PCR-RFLP method. Results: The current study found D9N genotypes, AA, AG, and GG in 71.14%, 23.72%, and 5.14% in CAD patients, respectively. the AA, AG, and GG control genotypes were f

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Valo, Misa, Annette Wons, Albert Moeller, and Claudius Teupe. "Markers of Myocardial Ischemia in Patients with Obstructive Sleep Apnea and Coronary Artery Disease." Pulmonary Medicine 2015 (2015): 1–6. http://dx.doi.org/10.1155/2015/621450.

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Obstructive sleep apnea (OSA) is characterized by intermittent hypoxia during sleep. We tested the hypothesis that nocturnal myocardial ischemia is detectable by ST segment depression and elevation of high sensitive troponin T (hsTrop T) and B-type natriuretic peptide (NT-proBNP) in patients with OSA and coexisting coronary artery disease (CAD). Twenty-one patients with OSA and CAD and 20 patients with OSA alone underwent in-hospital polysomnography. Blood samples for hsTrop T and NT-proBNP measurements were drawn before and after sleep. ST segment depression was measured at the time of maximu

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Niessner, Alexander, Senta Graf, Mariam Nikfardjam, et al. "Circulating t-PA antigen predicts major adverse coronary events in patients with stable coronary artery disease – a 13-year follow-up." Thrombosis and Haemostasis 90, no. 08 (2003): 344–50. http://dx.doi.org/10.1160/th02-10-0185.

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SummaryThrombus formation after rupture of an atherosclerotic plaque plays a crucial role in coronary artery disease (CAD). A decreased endogenous fibrinolytic system and prothrombotic factors are supposed to influence coronary thrombosis. It was our aim to investigate the predictive value of tissue plasmino-gen activator (t-PA) antigen, von Willebrand Factor, Lipoprotein (a) and anti-cardiolipin antibodies for major adverse coronary events in patients with stable CAD in a prospective cohort study of more than 10 years.We observed 141 patients with angiographically proven CAD for a median foll

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Kebert, Cory B., June E. Eichner, William E. Moore, et al. "Relationship of the 1793G-A and 677C-T Polymorphisms of the 5,10-Methylenetetrahydrofolate Reductase Gene to Coronary Artery Disease." Disease Markers 22, no. 5-6 (2006): 293–301. http://dx.doi.org/10.1155/2006/825431.

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Numerous studies have investigated the relationship between polymorphisms, in particular 677C-T and 1298A-C, of the methylene-tetrahydrofolate reductase (MTHFR) gene and coronary artery disease (CAD) with conflicting results. This study investigates the potential association of two point mutations inMTHFR, 677C-T and 1793G-A, along with other risk factors, with CAD. This is the first hospital-based study to investigate 1793G-A in this context. Genotype analysis was performed on 729 Caucasians and 66 African Americans undergoing coronary angiography using a novel PCR-based assay involving forma

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Jha, Chandan K., Rashid Mir, Shaheena Banu, Imadeldin Elfaki, and Sukh M. S. Chahal. "Heterozygosity in LDLR rs2228671 and rs72658855 Gene is Associated with Increased Risk of Developing Coronary Artery Disease in India –A Case-Control Study." Endocrine, Metabolic & Immune Disorders - Drug Targets 20, no. 3 (2020): 388–99. http://dx.doi.org/10.2174/1871530319666191015164505.

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Objective: Coronary artery disease (CAD) is one of the most common causes of death worldwide. Risk factors of CAD include high LDL-C, low high-density lipoprotein (HDL), hypertension, lack of exercise, genetic factors, etc. Polymorphisms of the LDLR gene have been associated with CAD in previous studies. Methods: The LDLR-rs72658855 C>T genotyping was detected by using allele-specific PCR (ASPCR). The association of rs2228671 and rs72658855 with CAD in a south Indian cohort (200 CAD patients and 200 matched healthy controls was studied. Results: Our findings showed that rs2228671 gene varia

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Kelle, Sebastian, Kelly Schlendorf, Glenn A. Hirsch, et al. "Gadolinium Enhanced MR Coronary Vessel Wall Imaging at 3.0 Tesla." Cardiology Research and Practice 2010 (2010): 1–9. http://dx.doi.org/10.4061/2010/856418.

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Purpose. We evaluated the influence of the time between low-dose gadolinium (Gd) contrast administration and coronary vessel wall enhancement (LGE) detected by 3T magnetic resonance imaging (MRI) in healthy subjects and patients with coronary artery disease (CAD).Materials and Methods.Four healthy subjects (4 men, mean age29 ± 3years and eleven CAD patients (6 women, mean age61±10years) were studied on a commercial 3.0 Tesla (T) whole-body MR imaging system (Achieva 3.0 T; Philips, Best, The Netherlands). T1-weighted inversion-recovery coronary magnetic resonance imaging (MRI) was repeated up

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Lu, Difei, Jia Huang, Xiaowei Ma, et al. "Rs46522 in the Ubiquitin-Conjugating Enzyme E2Z Gene Is Associated with the Risk of Coronary Artery Disease in Individuals of Chinese Han Population with Type 2 Diabetes." Journal of Diabetes Research 2017 (August 3, 2017): 1–5. http://dx.doi.org/10.1155/2017/4501794.

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Aims. We investigated the association between ubiquitin-conjugating enzyme E2Z (UBE2Z) gene SNP rs46522 and the risk of CAD in a Chinese Han population with type 2 diabetes and explored a possible interactive effect with environmental risk factors of CAD. Methods. 665 patients with T2D were enrolled; 390 were CAD patients and 275 were non-CAD patients. Genotype analysis of rs46522 (T>C) was performed using PCR-RFLP. Results. The SNP rs47522 was associated with the risk of CAD supposing recessive inheritance model (TT versus CC+CT, OR′=1.277, 95%CI′ 1.039–1.570, p′=0.020) and codominant mode

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Mir, Rashid, Imadeldin Elfaki, Jamsheed Javid, et al. "Genetic Determinants of Cardiovascular Disease: The Endothelial Nitric Oxide Synthase 3 (eNOS3), Krüppel-Like Factor-14 (KLF-14), Methylenetetrahydrofolate Reductase (MTHFR), MiRNAs27a and Their Association with the Predisposition and Susceptibility to Coronary Artery Disease." Life 12, no. 11 (2022): 1905. http://dx.doi.org/10.3390/life12111905.

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Coronary artery disease (CAD) is an important cause of death worldwide. CAD is caused by genetic and other factors including hypertension, hyperlipidemia, obesity, stress, unhealthy diet, physical inactively, smoking and Type 2 diabetes (T2D). The genome wide association studies (GWASs) have revealed the association of many loci with risk to diseases such as cancers, T2D and CAD. Nitric oxide (NO) is a potent vasodilator and is required for normal vascular health. It is produced in the endothelial cells in a reaction catalyzed by the endothelial NO synthase (eNOS). Methylenetetrahydrofolate re

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Sitompul, Sanggap Indra, Budi Susetyo Pikir, Aryati, Citrawati Dyah Kencono Wungu, Shafira Kurnia Supandi, and Monika Estherlita Sinta. "Analysis of the Effects of IL-6 -572 C/G, CRP -757 A/G, and CRP -717 T/C Gene Polymorphisms; IL-6 Levels; and CRP Levels on Chronic Periodontitis in Coronary Artery Disease in Indonesia." Genes 14, no. 5 (2023): 1073. http://dx.doi.org/10.3390/genes14051073.

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Interleukin 6 (IL-6) and C-Reactive Protein (CRP) play an important role in chronic periodontitis with coronary artery disease (CAD). Genetic factors can affect a person’s risk of CAD, which affects one-third of the population. This study investigated the role of IL-6 -572 C/G, CRP -757 A/G, and CRP -717 T/C gene polymorphisms. IL-6 and CRP levels on the severity of periodontitis in CAD in Indonesia were also evaluated. This case-control study was conducted with mild and moderate–severe chronic periodontitis groups. A path analysis test was conducted with Smart PLS with a 95% confidence interv

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Graf, Senta, Renate Beckmann, Stephan Hornykewycz, et al. "Fibrinolytic Response to Venous Occlusion Compared to Physical Stress Test in Young Patients with Coronary Artery Disease." Thrombosis and Haemostasis 82, S 01 (1999): 80–84. http://dx.doi.org/10.1055/s-0037-1615560.

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Summary Introduction: Venous occlusion (VO) and exercise stress (ES) are stimulators of the fibrinolytic system. Aim of this study was to answer which of both stimulation tests is more useful in patients with symptom-limited coronary artery disease (CAD) to evaluate possible defects in the fibrinolytic system. Methods and results: We investigated 20 patients (M/F = 15/5; mean age = 36.7 years) with angiographically proven CAD for their plasma levels of tissue-type plasminogen activator (t-PA) and plasminogen activator inhibitor-type-1 (PAI-1) at basal conditions as well as after VO and at maxi

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Tung, Robert T. "Significance of New, Isolated T-wave Inversion in Multiple Electrocardiogram Leads with Regadenoson Injection in Patients with Normal Myocardial Perfusion Imaging." Kansas Journal of Medicine 12, no. 3 (2019): 80–82. http://dx.doi.org/10.17161/kjm.v12i3.11797.

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 Introduction The pharmacologic (regadenoson) stress myocardial perfusion imaging (MPI) is used widely in patients who cannot exercise for detecting coronary artery disease (CAD). The interpretation of these studies depends primarily on the imaging results because the sensitivity of electrocardiograms (ECG) in this setting is poor. Prior study showed that effects of regadenoson on ST-segment occurred infrequently and had low sensitivity for detecting CAD. The significance of T-wave inversion in multiple ECG leads without ST-segment depression with regadenoson injection in patients

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