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Journal articles on the topic 'Systemic lupus erythematosus'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 August 2024

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1

Chinnusamy, Manokaran, Ram Arvind Viswanathan, Sathiyanarayanan Janakiraman, and Roshna Elayidath. "Drug-Induced Lupus Erythematosus Associated with Proton Pump Inhibitor." Journal of Health and Allied Sciences NU 10, no. 03 (September 8, 2020): 132–34. http://dx.doi.org/10.1055/s-0040-1716601.

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AbstractDrug-induced lupus erythematosus is an autoimmune phenomenon where the drug exposure leads to the development of systemic lupus erythematous like clinical features. Drug-induced lupus erythematosus can be divided into systemic lupus erythematous, subacute cutaneous lupus erythematous, and chronic cutaneous lupus erythematous. Here, we report a case of a 29-year-old female presented with systemic lupus erythematous due to chronic use of proton pump inhibitors, which is considered to be very rare.

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2

Garbelini-Lima, Cleide, Gabriela Evangelista de Almeida, Sidharta Quércia Gabdelha, Andrea Cavalcante de Souza, Mara Lúcia Gomes de Souza, and Virginia Vilasboas Figueiras. "Discoid Lupus Erythematosus of the Scalp in a Patient with Systemic Lupus Erythematosus: A Case Report with Complete Hair Regrowth." Journal of the Portuguese Society of Dermatology and Venereology 79, no. 2 (June 26, 2021): 155–58. http://dx.doi.org/10.29021/spdv.79.2.1283.

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Scalp involvement with hair loss is common in systemic lupus erythematosus. Discoid lupus erythematosus may cause scarring alopecia, characterized by well-delimited erythematous plaques with scales, follicular hyperkeratosis and atrophy, which is considered a trichological emergency. Early diagnosis and treatment are necessary in order to prevent permanent hair loss. We describe a 44 years’ old female patient with systemic lupus erythematosus for 4 years, with multiple areas of occipitoparietal alopecia, erythematous plaques, atrophy, scales and some bloody crusts. Trichoscopy, histopathology and direct immunofluorescence led to the diagnosis of discoid lupus erythematosus. After 9 months treatment with thalidomide there was complete hair regrowth.

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3

N, Habib. "Review Article: Systemic Lupus Erythematosus." Open Access Journal of Microbiology & Biotechnology 5, no. 1 (2020): 1–4. http://dx.doi.org/10.23880/oajmb-16000158.

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Systemic lupus erythematosus or SLE is a persistent heterogeneous autoimmune disease that affects multisystem of the body. It is distinguished by acute and chronic inflammation of various tissues and even organs of the body principally the skin and joints. Systemic lupus erythematosus is a multisystem disorder and hence, it can affect any tissues, organs and even systems of the body. There are few categories of lupus for instance, lupus dermatitis or cutaneous lupus erythematosus (CLE) that affects the skin and causes malar rash, discoid lupus erythematosus (DLE) as well as systemic lupus erythematosus that causes damage to single or multiple internal organs. The damage is due to the inflammation that is caused by direct antibody reaction to the body tissues as well the deposition of immune complexes. Glucocorticoids, immunosuppressant, and anti- malarial are the combination therapy used to treat SLE besides providing counseling and awareness. Lupus erythematosus in any form particularly systemic lupus erythematosus (SLE) are prevalent in women compared to men with ratio of 6:1. It has the tendency to affect all ages but most frequently attacks women of aged 20 to 45 years old compared to men. On the other hand, if lupus erythematosus causes damage to internal organs either single or multiple, it is known as systemic lupus erythematosus. The damage is due to the inflammation that is caused by direct antibody reaction to the body tissues as well the deposition of immune complexes.

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4

Franjic, Sinisa. "Systemic Lupus Erythematosus in Gynecology." International Journal of Reproductive Research 1, no. 1 (December 22, 2022): 01–03. http://dx.doi.org/10.58489/2836-2225/005.

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Systemic lupus erythematosus is a chronic autoimmune disease that can affect various organs and parts of the body, especially the skin, joints, blood, kidneys, and central nervous system. Systemic lupus erythematosus is not a contagious disease, it is an autoimmune disease in which the immune system loses the ability to distinguish foreign from the patient's own tissues and cells. The immune system makes mistakes and produces, among other things, autoantibodies that recognize their own cells as foreign and attack them. The result is an autoimmune reaction that causes inflammation. Inflammation means that the affected part of the body becomes warm, red, swollen and sometimes painfully sensitive. If the signs of inflammation are long-lasting, as they may be in the case of systemic lupus erythematosus, tissue damage and its normal function may occur. Therefore, the goal of treatment of systemic lupus erythematosus is to alleviate inflammation. A number of hereditary risk factors along with various environmental factors are thought to be responsible for this impaired immune response. Systemic lupus erythematosus is known to be caused by a variety of factors, including hormonal imbalances during puberty, stress, and environmental factors such as sun exposure, viral infections, and medications.

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5

Chottawornsak, N., P. Rodsaward, S. Suwannachote, M. Rachayon, T. Rattananupong, T. Deekajorndech, P. Asawanonda, D. Chiewchengchol, and P. Rerknimitr. "Skin signs in juvenile- and adult-onset systemic lupus erythematosus: clues to different systemic involvement." Lupus 27, no. 13 (October 18, 2018): 2069–75. http://dx.doi.org/10.1177/0961203318805851.

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Objective We aim to explore the differences of skin signs between juvenile- and adult-onset systemic lupus erythematosus and to identify their associations to the development of systemic involvement. Methods A retrospective chart review of 377 systemic lupus erythematosus patients was performed. Results In total, 171 patients with juvenile systemic lupus erythematosus and 206 with adult systemic lupus erythematosus were studied. All patients were of Southeast Asian descent. The mean duration of follow up was 8.18 ± 6.19 and 9.36 ± 7.68 years for juvenile systemic lupus erythematosus and adult systemic lupus erythematosus, respectively. At diagnosis, most patients presented with acute cutaneous lupus erythematosus, whereas chronic cutaneous lupus erythematosus was twice as common in adult systemic lupus erythematosus ( p < 0.001). The mean Systemic Lupus Erythematosus Disease Activity Index of juvenile systemic lupus erythematosus was significantly higher than that of adult systemic lupus erythematosus (14.29 ± 7.13 vs 11.27 ± 6.53). Multivariate analysis revealed the following associations in juvenile systemic lupus erythematosus: acute cutaneous lupus erythematosus and non-scarring alopecia with increased risk of arthralgia, mucosal ulcers with leukopenia, cutaneous vasculitis with seizure, and finding of granular casts. On the contrary, the associations for adult systemic lupus erythematosus were oral ulcers with arthralgia and cutaneous vasculitis with myositis. Conclusions Cutaneous signs in systemic lupus erythematosus may signal prognostic implication. Interestingly, despite similar cutaneous lesions in systemic lupus erythematosus, different ages of onset are associated with different systemic involvement.

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6

Powers, David B. "Systemic Lupus Erythematosus and Discoid Lupus Erythematosus." Oral and Maxillofacial Surgery Clinics of North America 20, no. 4 (November 2008): 651–62. http://dx.doi.org/10.1016/j.coms.2008.07.001.

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7

Feingold-Link, Mara. "Systemic Lupus Erythematosus." Annals of Internal Medicine 168, no. 6 (March 20, 2018): 452. http://dx.doi.org/10.7326/m17-2553.

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8

Kiriakidou, Marianthi. "Systemic Lupus Erythematosus." Annals of Internal Medicine 159, no. 7 (October 1, 2013): ITC4. http://dx.doi.org/10.7326/0003-4819-159-7-201310010-01004.

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9

Greenberg, Brent, and Margaret Michalska. "Systemic lupus erythematosus." Postgraduate Medicine 106, no. 6 (January 1999): 213–23. http://dx.doi.org/10.3810/pgm.1999.11.779.

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10

Dall'Era, Maria, and John C. Davis. "Systemic lupus erythematosus." Postgraduate Medicine 114, no. 5 (November 2003): 31–40. http://dx.doi.org/10.3810/pgm.2003.11.1528.

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11

Steinberg, Alfred D. "Systemic Lupus Erythematosus." Annals of Internal Medicine 115, no. 7 (October 1, 1991): 548. http://dx.doi.org/10.7326/0003-4819-115-7-548.

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12

Kuper, Barbara Caruso, and Salva Failla. "SYSTEMIC LUPUS ERYTHEMATOSUS." Nursing Clinics of North America 35, no. 1 (March 2000): 253–65. http://dx.doi.org/10.1016/s0029-6465(22)02458-6.

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13

Kiriakidou, Marianthi, and Cathy Lee Ching. "Systemic Lupus Erythematosus." Annals of Internal Medicine 172, no. 11 (June 2, 2020): ITC81—ITC96. http://dx.doi.org/10.7326/aitc202006020.

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14

Hamdani, Muhammad Afzal, Khalid Parvez, Faisal Naseeb, Umair Afzal, Bashiruddin -, Joseph Hope Cal, and Sajjad Hussain. "SYSTEMIC LUPUS ERYTHEMATOSUS;." Professional Medical Journal 24, no. 01 (January 18, 2017): 14–20. http://dx.doi.org/10.29309/tpmj/2017.24.01.479.

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Systemic lupus erythematosus (SLE), is a multifactorial, complex etiologicaldisorder, characterized by inflammation and involvement of multiple organ systems includinglungs. Objective: 1-To evaluate whether high resolution computed tomography (HRCT) helpsin the diagnosis of pulmonary manifestations of SLE. 2-To study the pattern and extent of lunginvolvement using HRCT. Design: A Prospective cross - sectional clinical study. Period: Fouryears, July 2012 to June 2016. Setting: King Khalid University Hospital (KKUH) King SaudUniversity (KSU), Rheumatology division Department of Medicine. Methods: This study included113 patients attending outpatients or admitted as inpatients having respiratory symptomsand diagnosed as SLE according to American College of Rheumatology (ACR) classificationcriteria. Chest X- ray, pulmonary function tests, and HRCT chest were done. Investigationsto detect other organ involvement were done. Pregnant females and patients having otherconnective tissue or occupational diseases were excluded. Results: Of the total 113 patients102 were female and 11 males. Age of patients was 37.1 ±13.0 years. The HRCT abnormalitieswere pleural effusion, pleural thickening, atelectasis, ground glass opacities including nonspecificinterstitial pneumonitis (NSIP) and usual interstitial pneumonitis (UIP), pulmonaryarterial hypertension, pulmonary embolism and hilar lymphadenopathy. Conclusion: Variouspulmonary manifestations are present in a significant number of symptomatic SLE patients anda variety of HRCT patterns can be seen to diagnose and treat them.

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15

Mintz, Gregorio, and Encarnacion Rodriguez-Alvarez. "Systemic Lupus Erythematosus." Rheumatic Disease Clinics of North America 15, no. 2 (May 1989): 255–74. http://dx.doi.org/10.1016/s0889-857x(21)00986-8.

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16

Hochberg, Marc C. "Systemic Lupus Erythematosus." Rheumatic Disease Clinics of North America 16, no. 3 (August 1990): 617–39. http://dx.doi.org/10.1016/s0889-857x(21)00891-7.

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17

Elgendi, Sabah. "Systemic Lupus Erythematosus." International Egyptian Journal of Nursing Sciences and Research 1, no. 2 (January 4, 2021): 31–35. http://dx.doi.org/10.21608/ejnsr.2020.51290.1025.

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18

Park, Sung Hwan. "Systemic Lupus Erythematosus." Journal of the Korean Medical Association 52, no. 7 (2009): 645. http://dx.doi.org/10.5124/jkma.2009.52.7.645.

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19

Wallace, D. J. "Systemic lupus erythematosus." Drugs of Today 38, no. 4 (2002): 259. http://dx.doi.org/10.1358/dot.2002.38.4.765369.

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20

Rahman, Anisur, and David A. Isenberg. "Systemic Lupus Erythematosus." New England Journal of Medicine 358, no. 9 (February 28, 2008): 929–39. http://dx.doi.org/10.1056/nejmra071297.

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21

Tsokos, George C. "Systemic Lupus Erythematosus." New England Journal of Medicine 365, no. 22 (December 2011): 2110–21. http://dx.doi.org/10.1056/nejmra1100359.

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22

Ballou, Stanley P. "Systemic lupus erythematosus." Postgraduate Medicine 81, no. 8 (June 1987): 157–64. http://dx.doi.org/10.1080/00325481.1987.11699868.

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23

Roberts, Donna M., and W. Michael Hughes. "Systemic lupus erythematosus." Postgraduate Medicine 86, no. 8 (December 1989): 191–200. http://dx.doi.org/10.1080/00325481.1989.11704507.

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24

Shaikh, Maliha F., Natasha Jordan, and David P. D'Cruz. "Systemic lupus erythematosus." Clinical Medicine 17, no. 1 (February 2017): 78–83. http://dx.doi.org/10.7861/clinmedicine.17-1-78.

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25

Ravindran, Vinod. "Systemic lupus erythematosus." Indian Journal of Rheumatology 11, no. 3 (2016): 59. http://dx.doi.org/10.4103/0973-3698.187420.

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26

&NA;. "Systemic Lupus Erythematosus." Survey of Anesthesiology 55, no. 2 (April 2011): 107–10. http://dx.doi.org/10.1097/sa.0b013e31820ecb9d.

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27

Petri, Michelle. "Systemic Lupus Erythematosus." JCR: Journal of Clinical Rheumatology 12, no. 1 (February 2006): 37–40. http://dx.doi.org/10.1097/01.rhu.0000200420.67262.04.

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28

El-Magadmi, Masoud, Helena Bodill, Yasmeen Ahmad, Paul N. Durrington, Michael Mackness, Michael Walker, Robert M. Bernstein, and Ian N. Bruce. "Systemic Lupus Erythematosus." Circulation 110, no. 4 (July 27, 2004): 399–404. http://dx.doi.org/10.1161/01.cir.0000136807.78534.50.

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29

Fischer-Betz, R., P. Herzer, and M. Schneider. "Systemic lupus erythematosus." DMW - Deutsche Medizinische Wochenschrift 130, no. 43 (October 2005): 2451–58. http://dx.doi.org/10.1055/s-2005-918589.

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30

Mackillop, Lucy H., Sarah J. Germain, and Catherine Nelson-Piercy. "Systemic lupus erythematosus." BMJ 335, no. 7626 (November 1, 2007): 933–36. http://dx.doi.org/10.1136/bmj.39358.519491.ad.

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31

Block, S. R. "Systemic lupus erythematosus." Annals of the Rheumatic Diseases 44, no. 8 (August 1, 1985): 573. http://dx.doi.org/10.1136/ard.44.8.573-a.

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32

&NA;. "Systemic lupus erythematosus." Current Opinion in Rheumatology 2, no. 5 (October 1990): 841–60. http://dx.doi.org/10.1097/00002281-199002050-00026.

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33

Mills, John A. "Systemic Lupus Erythematosus." New England Journal of Medicine 330, no. 26 (June 30, 1994): 1871–79. http://dx.doi.org/10.1056/nejm199406303302608.

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34

ROONEY, JOAN. "Systemic lupus erythematosus." Nursing 35, no. 11 (November 2005): 54–60. http://dx.doi.org/10.1097/00152193-200511000-00049.

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&NA;. "Systemic lupus erythematosus." Nursing 35, no. 11 (November 2005): 61. http://dx.doi.org/10.1097/00152193-200511000-00050.

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36

Trethewey, Pat. "Systemic Lupus Erythematosus." Dimensions of Critical Care Nursing 23, no. 3 (2004): 111–15. http://dx.doi.org/10.1097/00003465-200405000-00003.

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37

Okamoto, Hiroshi, Ricard Cervera, Tatiana S. Rodriguez-Reyna, Hiroyuki Nishimura, and Taku Yoshio. "Systemic Lupus Erythematosus." Autoimmune Diseases 2012 (2012): 1–2. http://dx.doi.org/10.1155/2012/815753.

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38

Haq, Inam, and David A. Isenberg. "Systemic Lupus Erythematosus." Medicine 30, no. 10 (October 2002): 6–12. http://dx.doi.org/10.1383/medc.30.10.6.28249.

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39

Pearce, Lynne. "Systemic lupus erythematosus." Nursing Standard 30, no. 43 (June 22, 2016): 17. http://dx.doi.org/10.7748/ns.30.43.17.s21.

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40

Johnson, Barbara. "Systemic Lupus Erythematosus." American Journal of Nursing 99, no. 1 (January 1999): 40–41. http://dx.doi.org/10.1097/00000446-199901000-00036.

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41

D'Cruz, David P. "Systemic lupus erythematosus." BMJ 332, no. 7546 (April 13, 2006): 890–94. http://dx.doi.org/10.1136/bmj.332.7546.890.

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42

Von Feldt, Joan M. "Systemic lupus erythematosus." Postgraduate Medicine 97, no. 4 (April 1995): 79–94. http://dx.doi.org/10.1080/00325481.1995.11945982.

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43

Li, Charles K., and David A. Isenberg. "Systemic lupus erythematosus." Medicine 34, no. 11 (November 2006): 445–52. http://dx.doi.org/10.1053/j.mpmed.2006.09.002.

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44

Petri, Michelle. "Systemic Lupus Erythematosus." Obstetrics & Gynecology 136, no. 1 (July 2020): 226. http://dx.doi.org/10.1097/aog.0000000000003942.

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45

Eyrich, Raili, and Birgitta Borulf. "SYSTEMIC LUPUS ERYTHEMATOSUS." Acta Medica Scandinavica 196, no. 1-6 (April 24, 2009): 527–35. http://dx.doi.org/10.1111/j.0954-6820.1974.tb01056.x.

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46

Putterman, Chaim, Roberto Caricchio, Anne Davidson, and Harris Perlman. "Systemic Lupus Erythematosus." Clinical and Developmental Immunology 2012 (2012): 1–2. http://dx.doi.org/10.1155/2012/437282.

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47

Scott, Steven A. "Systemic Lupus Erythematosus." Journal of Pharmacy Practice 12, no. 4 (August 1999): 335–45. http://dx.doi.org/10.1177/089719009901200407.

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Systemic lupus erythematosus (SLE) is a chronic, multiple-organ system inflammatory disorder associated with immune system dysfunction. Autoantibodies are produced that react with self-antigens in cell membranes and nuclear and cytoplasmic constituents to produce tissue damage. Commonly observed clinical manifestations include arthritis, myalgia, fever, cutaneous lesions, cytopenia, and renal, CNS, and cardiopulmonary involvement. Minor manifestations can be managed with relatively nontoxic agents such as nonsteroidal anti-inflammatory drugs, topical corticosteroids, and antimalarials. Severe disease involving the kidneys, CNS, and cardiopulmonary systems requires the aggressive use of more toxic agents such as high-dose corticosteroids, azathioprine, and cyclophosphamide. Other supportive and ancillary therapies are also required to manage the complications frequently associated with SLE.

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48

D'Cruz, David P., Munther A. Khamashta, and Graham RV Hughes. "Systemic lupus erythematosus." Lancet 369, no. 9561 (February 2007): 587–96. http://dx.doi.org/10.1016/s0140-6736(07)60279-7.

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49

Searles, Robert P. "Systemic lupus erythematosus." Postgraduate Medicine 78, no. 6 (November 1985): 93–105. http://dx.doi.org/10.1080/00325481.1985.11699187.

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50

Pisetsky, David S. "Systemic Lupus Erythematosus." Medical Clinics of North America 70, no. 2 (March 1986): 337–53. http://dx.doi.org/10.1016/s0025-7125(16)30957-9.

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