Relevant bibliographies by topics / Syringomyelia

Academic literature on the topic 'Syringomyelia'

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Published: 4 June 2021
Last updated: 4 March 2023

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Journal articles on the topic "Syringomyelia"

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Fenchel, M., F. Roser, T. Nägele, U. Ernemann, and M. Horger. "Syringomyelia – Syringomyelie." RöFo - Fortschritte auf dem Gebiet der Röntgenstrahlen und der bildgebenden Verfahren 184, no. 03 (February 17, 2012): 191–95. http://dx.doi.org/10.1055/s-0031-1274771.

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Arévalo-Sáenz, Alejandra, and Manuel Pedrosa Sánchez. "Dorsal hemangioblastoma with holocord syringomyelia: case report." Neurohirurgija - The Serbian Journal of Neurosurgery 2, no. 1 (December 31, 2022): 13–16. http://dx.doi.org/10.55005/v2i1.4.

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Introduction: Intramedullary hemangioblastomas are usually accompanied by syringomyelia. However, a holocord syringomyelia is rare. The most common cause of syringomyelia continues to be Chiari disease, and only 10 cases of hemangioblastomas with holocord syringomyelia reported so far. Case report: We present a case of a 35-year-old patient with a two-month history of cervicobrachialgia at the C7-C8 root level, previously preceded by pain at the D1-D2 level. Cervico-dorso-lumbar MRI revealed a medullar tumor with hyper-uptake mural nodule at the conus medullaris level accompanied by an extensive syringomyelic cavity from C5 to L1 compatible with medullary hemangioblastoma. The patient underwent surgery for tumor resection with subsequent resolution of her painful symptoms. Conclusion: It is important to note that the surgery is aimed at treating the origin of this syringomyelia and not the syringomyelia itself. Although the majority of patients with holocord syringomyelia have Chiari as its cause, the possibility of focal spinal intramedullary tumors as being responsible for syringomyelia should not be forgotten.
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Akgun, Bekir, Ahmet Cemil Ergun, Ibrahim Hanifi Ozercan, and Selman Kok. "Intradural Extramedullary Epidermoid Cyst at the Conus Medullaris Level with Thoracic Syringomyelia: A Case Report." Acta Medica (Hradec Kralove, Czech Republic) 62, no. 1 (2019): 39–42. http://dx.doi.org/10.14712/18059694.2019.45.

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Spinal epidermoid cysts are benign tumors. Syringomyelia secondary to intramedullary tumors are frequently observed. However, the association between syringomyelia and spinal intradural extramedullary epidermoid cyst in the conus medullaris region is extremely rare. We present the case of a 3-year-old male who was admitted with paraparesis and urinary retention. Magnetic resonance imaging (MRI) of the spine demonstrated intradural extramedullary lesion, compatible with epidermoid cyst, that at the conus medullaris level and a large syringomyelia extending from T4 to L1 vertebrae. Total microsurgical excision of the cyst was performed. No additional drainage was carried out for the syringomyelic cavity. Histopathological examination verified the diagnosis of the epidermoid cyst. Total excision of the cyst and disappearance of the syringomyelia were observed on MRI at 15 days postoperatively. We have clarified the etiology, clinical, histopathological and radiological features, differential diagnosis, and treatment modalities of spinal epidermoid cysts. In addition, we have discussed the possible mechanisms of syringomyelia formation in spinal intradural lesions.
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Kokmen, E., W. R. Marsh, and H. L. Baker. "Magnetic Resonance Imaging in Syringomyelia." Neurosurgery 17, no. 2 (August 1, 1985): 267–70. http://dx.doi.org/10.1227/00006123-198508000-00003.

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Abstract Myelography and myelography assisted with computed tomography have been the most commonly used radiographic methods in the study of syringomyelia. These studies have never been entirely reliable in demonstrating the syrinx cavity and its relationship to other intracranial structures. During the 1st year of operation of the magnetic resonance imaging facility, the syringomyelic cavity was demonstrated in 15 patients who all had typical clinical signs and symptoms associated with syringomyelia. Nine cases were syringomyelia with Chiari malformation. One case showed additional hydrocephalus. Four cases were idiopathic, and 1 case was remotely posttraumatic. Magnetic resonance imaging, although it is in its infancy, already promises to be the most important radiographic technique for syringomyelia because it provides an anatomically truthful visualization of the sagittal plane of the cervical cord and can demonstrate the syrinx cavity and its relationship with the cerebellar tonsils, the 4th ventricle, and other related structures.
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Nagahiro, Shinji, Yasuhiko Matsukado, Jun-ichi Kuratsu, Yoshiki Saito, and Seishi Takamura. "Syringomyelia and Syringobulbia Associated with an Ependymoma of the Cauda Equina Involving the Conus Medullaris: Case Report." Neurosurgery 18, no. 3 (March 1, 1986): 357–60. http://dx.doi.org/10.1227/00006123-198603000-00020.

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Abstract A patient with syringomyelia and syringobulbia secondary to an asymptomatic ependymoma of the cauda equina involving the conus medullaris is described. Delayed metrizamide computed tomography myelography was decisive for making the diagnosis of a highly extended syrinx associated with the tumor. Syringomyelic symptoms improved remarkably following the removal of the tumor, and the syrinx was not visualized in a postoperative study. The pathogenesis of syringomyelia associated with a caudally located spinal cord tumor is discussed.
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Batzdorf, Ulrich, Larry T. Khoo, and David L. McArthur. "OBSERVATIONS ON SPINE DEFORMITY AND SYRINGOMYELIA." Neurosurgery 61, no. 2 (August 1, 2007): 370–78. http://dx.doi.org/10.1227/01.neu.0000279971.87437.1f.

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Abstract OBJECTIVE Spine deformities, notably scoliosis, are known to occur in conjunction with syringomyelia. This study aims to analyze the effect of laminectomies performed in the course of treatment of syringomyelia. It examines the incidence, severity, and type of spine deformity as it relates to the extent and location of laminectomies performed. METHODS Records of 169 patients were analyzed for evidence of spinal deformity on imaging studies for the extent of the syringomyelic cavities and for previous surgical procedures on the spine. This analysis included patients with syringomyelia related to Chiari malformation, as well as patients with primary spinal pathology. RESULTS Spinal deformities were encountered in 41% of Chiari-syringomyelia patients who had not undergone previous surgery and in 57% of such patients who underwent reoperation. Scoliosis, the most common type of deformity encountered, was likely to be mild in patients who had not undergone previous surgery and severe in reoperated patients. Spine deformity was significantly more common in those patients who had more extensive bone removal. CONCLUSION Complete laminectomy should be avoided whenever possible in patients with syringomyelia because local denervation of the axial musculature, added to loss of medial anterior horn cells from syringomyelia, favors the development of spine deformities. This is particularly true of laminectomies performed at the junctional areas of the spine, i.e., cervical-thoracic and thoracolumbar. Hemilaminectomy usually suffices for shunt placement; instrumented stabilization should be considered in patients undergoing full laminectomy, especially those considered to be at high risk of developing deformity.
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Derevyanko, Kh P., and Sh M. Safin. "Chiari malformation type I and syringomyelia as keys to understanding CSF circulation (literature review)." Vestnik nevrologii, psihiatrii i nejrohirurgii (Bulletin of Neurology, Psychiatry and Neurosurgery), no. 8 (August 16, 2022): 627–35. http://dx.doi.org/10.33920/med-01-2208-07.

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Pathophysiology of syringomyelia has not been understood completely. The basic «piston» theory is that the downward movement of the cerebellar tonsils that occurs during the cardiac cycle acts like a «piston» on the partially closed spinal subarachnoid space and results in the formation of a syringomyelitic cavity. However, this still needs to be confirmed by additional experimental data. The main pathogenetic question is how the cerebrospinal fluid enters and remains in the syringomyelic cavity, where the pressure is higher than in the subarachnoid space. Based on the literature data on the pathogenesis of Chiari I malformation with syringomyelia, we modeled and presented our vision of the pathogenesis. Our assumption about the formation of syringomyelia in Chiari I malformation is that there may be a CSF channel between the fourth ventricle and the syrinx. This channel takes on the function of a one-way valve when slightly squeezed and with cyclic movements of the cerebellar tonsils. Decompression of the tonsils turns off the one-way valve, collapsing the syrinx. Perhaps this assumption will serve as a working hypothesis for further study of the pathophysiology of Chiari malformation type I and syringomyelia.
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Batzdorf, Ulrich. "Chiari I malformation with syringomyelia." Journal of Neurosurgery 68, no. 5 (May 1988): 726–30. http://dx.doi.org/10.3171/jns.1988.68.5.0726.

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✓ Five patients with a Chiari I-syringomyelia complex of adult onset were evaluated by magnetic resonance (MR) imaging. All patients underwent suboccipital craniotomy with upper cervical (C-1 and part of C-2) laminectomy, arachnoid retraction, and duraplasty. Postoperative MR studies of four patients disclosed collapse of the syringomyelic cavity, even when the cavity extended into the thoracic region. This appeared to be a progressive process taking place over several weeks. Operative complications are noted and physiological implications are discussed.
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Tarianyk, Kateryna, Natalia Lytvynenko, Tetiana Purdenko, and Viktoriia Hladka. "DIAGNOSTIC ERRORS DURING INTRAMEDULLAR PROCESSES." Wiadomości Lekarskie 72, no. 1 (January 2019): 129–32. http://dx.doi.org/10.36740/wlek201901125.

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Introduction: The article describes a clinical case of a malignant tumor of the brain and spinal cord with metastasis, which remained undetected for many years and was treated as syringomyelia. Long-term exhausting examinations of the brain and spinal cord, dynamic follow-up of medical specialists, and repeated surgical interventions on the spine helped to differentiate this process and make the correct diagnosis. The aim: The objectives of the present paper are to analyze the existing classifications of syringomyelia; to examine its etiology, pathogenesis, diagnostic approaches and treatment tactics; to present a clinical case of a malignant tumor of the brain and spinal cord with metastasis, which was misdiagnosed as syringomyelia. Materials and methods: The authors analyzed the existing classifications of syringomyelia and studied its etiology, pathogenesis, diagnostic approaches and treatment tactics using the method of content analysis, analyticalб comparative and contrastive methods. Clinical case: The described clinical case is a variant of the course of syringomyelia, associated with the spinal cord tumor, namely, in particular, anaplastic ependymoma. For a long time, the process remained undetected, despite the long-term examination and drainage of the syringomyelic cavity. Analyzing this case, it is highly important to pay attention to careful history collection, examination of the patient, analysis of the occurrence of certain disorders in order to conduct the neuroimaging examinations in time. Conclusions: Favorable outcome of the disease is directly related to the diagnosis at early stages, especially in young people, the clinical variant of the process, progression of the course, the degree of involvement of various parts of the nervous system and extraneural formations, the severity of the lesion. The treatment tactics and the ability to restore the functions will depend on all these factors.
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Oldfield, Edward H. "Syringomyelia." Journal of Neurosurgery: Spine 95, no. 1 (July 2001): 153–55. http://dx.doi.org/10.3171/spi.2001.95.1.0153.

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Dissertations / Theses on the topic "Syringomyelia"

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Stoodley, Marcus A. "Pathophysiology of Syringomyelia /." Title page, contents and abstract only, 1996. http://web4.library.adelaide.edu.au/theses/09PH/09phs882.pdf.

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Yang, Liqun. "Excitotoxic model of posttraumatic syringomyelia in the rat." Title page, contents and abstract only, 1999. http://web4.library.adelaide.edu.au/theses/09MS/09msy22.pdf.

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Bibliography: leaves 112-127. Study using an animal model (Sprague-Dawley rats) to elucidate the role of EAAs and spinal subarachnoid blockade in posttraumatic syringomelia. Results support the proposal that in posttraumatic spinal cord injury, primary injury and exitotoxic cell death, occuring secondary to elevated levels of EAAs, contribute to a pathologic process leading to the formation of spinal cavities, and a subarachnoid block by arachnoiditis is one of the pathogenic factors most responsible for initiating extension of the cavity.
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Lee, Gabriel Y. F. "Origin of macrophages in rat syringomyelia : an investigative study using rat radiation bone marrow chimeras /." Title page, table of contents and abstract only, 2001. http://web4.library.adelaide.edu.au/theses/09MS/09msl478.pdf.

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Brodbelt, Andrew Robert Prince of Wales Medical Research Institute Faculty of Medicine UNSW. "Investigations in post-traumatic syringomyelia." Awarded by:University of New South Wales. Prince of Wales Medical Research Institute, 2003. http://handle.unsw.edu.au/1959.4/19317.

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Object. More than a quarter of spinal cord injured patients will develop an enlarging cystic cavity, or syrinx, within the spinal cord up to 30 years following the initial trauma. Enlarging syrinxes can cause progressive loss of function in patients who often already have a disability. Less than half the affected patients can be expected to improve following treatment. A lack of understanding of the pathophysiology of the disease is hampering attempts to improve on this poor prognosis. Investigation into the mechanisms of fluid flow and the role of ischaemia in this type of syringomyelia may help understand the causative mechanisms and lead to improved treatment outcomes. Materials and methods. Temporal and dose profiling of an animal model of post-traumatic syringomyelia was performed in Sprague Dawley rats. The cerebrospinal fluid tracer, horseradish peroxidase, was used for detailed analysis of the routes of fluid flow into the syrinx. The effects of variations in subarachnoid space compliance on syrinx formation and fluid flow were examined using shunt insertion, pseudomeningocele formation, and animals without subarachnoid adhesions. Local spinal cord blood flow was investigated with Doppler flowmetry, and the intracellular levels of ATP and adenosine phosphates were measured with a bioluminescence technique. Results. A reliable animal model that mimicked human syrinx pathology was refined. Excitotoxic injury selectively damaged neurones, and induced syrinx formation in a dose and time dependent fashion. Cerebrospinal fluid flows from the subarachnoid space along perivascular spaces into the cord and syrinx. Flow occurred along the perivascular spaces of the central branches of the anterior spinal artery, and was maximal at the level of the syrinx. Improving compliance by shunt insertion reduced syrinx size but preferential fluid movement into the syrinx was maintained. If arachnoid adhesions were absent, this preferential flow ceased and syrinxes were smaller. Pseudomeningocele formation proved technically difficult and was unhelpful in changing cord compliance. Spinal cord blood flow was decreased following syrinx formation. Early reductions occurred in ATP and ADP levels, and the intracellular ATP/ADP ratio remained below 0.2 at all time points following syrinx induction. Conclusions. CSF moves into the syrinx and spinal cord along perivascular spaces. Arachnoid adhesions may act to decrease localized subarachnoid space compliance and encourage fluid flow from the subarachnoid space along perivascular spaces and into the syrinx. Increasing distal subarachnoid space compliance does not prevent this preferential flow but may reduce the amount of flow. Fluid accumulation within the syrinx causes ischaemia through pressure effects on the surrounding spinal cord tissue, and may further syrinx enlargement by apoptosis. The results support and are consistent with a local arterial pulsation dependent pumping mechanism of cerebrospinal fluid flow.
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Hechler, Ashley C. "Identifying and Treating Neuropathic Pain in Dogs with Syringomyelia." The Ohio State University, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1554712901697936.

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Liao, Jinxin Clinical School Prince of Wales Hospital Faculty of Medicine UNSW. "Investigation of endogenous stem cells and reactive astrocytes in post-traumatic syringomyelia." Awarded by:University of New South Wales. Clinical School - Prince of Wales Hospital, 2007. http://handle.unsw.edu.au/1959.4/40674.

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Introduction: Around a quarter of patients with spinal cord injury develop post traumatic syringomyelia (PTS), causing progressive neurological deficits. Current surgical treatment is unsatisfactory. Endogenous stem cell therapy, aiming at replacing lost tissue and repairing damaged ones by endogenous progenitors, may offer hope. Investigation into the reaction of endogenous progenitors in PTS may extend our knowledge about stem cell biology and help to develop a new treatment option for PTS. Endogenous stem cells were found to differentiate into astrocytes. Reactive astrocytes and gliosis are shown to have an important role in spinal cord injury, such as protecting neurons, limiting inflammation and regulating local environment to suit progenitors. We hypothesize that reactive astrocytes may play an important protective and potential therapeutic roles in PTS. The aim of this thesis is to study proliferation, differentition and location of endogenous progenitors and their roles in PTS. Materials and methods: Excitotoxic injury model of PTS was performed in adult Wistar rats. Proliferating cells were marked by either exogenous mitotic marker bromodeoxyuridine or endogenous mitotic marker Ki67.lmmunofluorescence techniques targeting mitotic markers were used to trace the proliferating cells. Immunofluorescent double staining techniques were used to phenotype the proliferating cells. Results: A large number of endogenous progenitors appear in PTS from 24 hours to at least 8 weeks post injury (PI). They proliferate much faster in PTS than in the control animals. Although less endogenous progenitors are observed after 4 weeks PI, their number is still much higher than that in the control animals. Immediately after injury, progenitors exist mainly in the white matter, but the majority of them shift their position closer to the lesion within 2 days. In the chronic stage, the majority of stem cells are located in and around the lesion site. Endogenous progenitors differentiate into astrocytes but not oligodendrocytes or neurons within 8 weeks. Astrocytes respond to injury by upgrading GFAP (1 day PI), becoming hypertrophic (7 days PI) and forming glial scar (2 weeks PI) in PTS. The development of a glial scar corresponds with the stage of cyst stability or reduction in size. Conclusions: Endogenous progenitors exist in PTS and they respond to injury by proliferating and shifting their position towards the lesion. These studies are important in understanding the endogenous stem cell response to PTS and lay the groundwork for future studies examining stem cell therapy for the condition. Endogenous progenitors in the PTS model differentiate into astrocytes, which help to form the glial scar lining the syrinx. Reactive gliosis may play an important role to seclude the injury site from healthy tissue, prevent a cascading wave of uncontrolled tissue damage and restrict the syrinx enlargement.
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Elliott, Novak Samuel Jon. "Mathematical modelling and analysis of cerebrospinal mechanics : an investigation into the parthogenesis of syringomyelia." Thesis, University of Warwick, 2009. http://wrap.warwick.ac.uk/2751/.

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Syringomyelia is a disease in which fluid-filled cavities, called syrinxes, form in the spinal cord causing progressive loss of sensory and motor functions. Invasive monitoring of pressure waves in the spinal subarachnoid space implicates a hydrodynamic origin. Poor treatment outcomes have led to myriad hypotheses for its pathogenesis, which unfortunately are often based on small numbers of patients due to the relative rarity of the disease. However, only recently have models begun to appear based on the principles of mechanics. One such model is the mathematically rigorous work of Carpenter and colleagues. They suggest that a pressure wave due to a cough or sneeze could form a shock-like elastic jump, which when incident at a stenosis, such as a hindbrain tonsil, would generate a transient region of high pressure within the spinal cord and lead to fluid accumulation. The salient physiological parameters of this model are reviewed from the literature and the assumptions and predictions re-evaluated from a mechanical standpoint. It is found that, while the spinal geometry does allow for elastic jumps to occur, their effects are likely to be weak and subsumed by the small amounts of damping that have been measured in the subarachnoid space. The analysis presented here does not support the elastic-jump hypothesis for syrinx formation. Furthermore, the site of maximum transpial pressure dierential due to a cough-induced pulse is most likely to be at the site of pulse origin|not, as supposed, at a distant reflection site. This suggests that there must be some other localising factor more critical to providing the necessary conditions for syrinx formation. Two coaxial tube models are developed that incorporate Darcy's law separately in the pial membrane and the spinal cord tissue. It is shown that permeability plays opposing roles in the spinal cord and pia for wave attenuation; the propagation of a pressure wave is aided by a less-permeable pia but a more-permeable spinal cord. This may have implications in a syringomyelic cord. To understand the dynamic interaction of the fluid and solid components of the spinal cord tissue Biot's theory of poroelasticity is employed. It is concluded that physiological frequencies are probably too low for poroelastic dissipation to be of signicance in such a soft and weak material as the spinal cord. Accumulating evidence in the last decade from animal studies implicates arterial pulsations in syrinx formation. In particular, Bilston and colleagues suggested that a phase difference between the pressure pulse in the spinal subarachnoid space and the perivascular spaces, due to a pathologically disturbed blood supply, could result in a net in flux of cerebrospinal fluid into the spinal cord. A lumped-parameter model is developed of the cerebrospinal system to investigate this conjecture. It is found that although this phase-lag mechanism may operate, it requires the spinal cord to have an intrinsic storage capacity due to the collapsibility of the contained venous reservoir. If this storage requirement is met then the results presented here suggest that, on mechanical grounds, a syringo-subarachnoid shunt may be a better surgical treatment option than a subarachnoid bypass for post-traumatic syringomyelia.
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Knowler, S. P. "Characterisation of chiari-like malformation and secondary syringomyelia in selected toy dog breeds using magnetic resonance imaging." Thesis, University of Surrey, 2017. http://epubs.surrey.ac.uk/844721/.

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Chiari-like Malformation (CM) and secondary Syringomyelia (SM) is a complex, debilitating abnormality which compromises the normal cerebrospinal fluid movement of the central nervous system culminating in the development of fluid-containing cavities within the spinal cord and associated with behavioural signs of pain and neurological deficits. The prevalence of asymptomatic CM dogs suggest that cerebellar indentation and impaction may be normal anatomical variations and unsuitable as a definition of CM. Magnetic Resonance Imaging (MRI) remains the definitive means of diagnosing CM/SM and a morphometric technique of quantifying CM and SM on mid-sagittal MRI has been successfully applied and validated in previous studies to a cohort of Griffon Bruxellois (GB) dogs with and without CM and a mixed breed GB family crossed with a mesaticephalic breed (Australian Terrier). Using a refined technique which took account of recent research findings, morphometries using a triangulation of circles, lines and angles were used to ‘map’ MRIs of the whole brain and cervical region in order to quantify the severity of the CM and SM phenotype in the Cavalier King Charles (CKCS). A further morphometric analysis was undertaken to explore brachycephaly and miniaturization as risk factors for CM and SM by comparing their impact in the CKCS, Affenpinscher and Chihuahua breeds. The collective framework of lines and angles generated a unique ‘signature’ for the dog, characterised by “concertina” type flexures demonstrating the combined nature of segregated traits towards the severity in the phenotype. Compared to controls, CKCS with CM pain are characterised by increased brachycephaly and airorhynchy, while significant traits for SM in the three dog breeds included those reported for the GB, suggesting a common aetiology. The characterisation of the CM phenotype provides the possibility of a diagnostic tool for veterinarians and means to assist breeders with mate selection to reduce symptomatic prevalence of CM/SM.
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Knuppel, Julie M. "Correlation of Neurologic Status As Evaluated By Neurologic Examination And Brainstem Auditory Evoked Response Test With Computed Tomographic And Radiographic Morphometric Analysis Of The Caudal Skull In Cavalier King Charles Spaniels." The Ohio State University, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=osu1243866074.

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Личко, Володимир Станіславович, Владимир Станиславович Личко, Volodymyr Stanislavovych Lychko, and Т. В. Іванова. "Сколіотична деформація хребта у хворих на сирингомієлію з дебютом у дитячому віці." Thesis, Сумський державний університет, 2016. http://essuir.sumdu.edu.ua/handle/123456789/47435.

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Сколіоз, що виникає в дитячо-юнацькому віці, є поширеною патологією, яка може зустрічатися як самостійне захворювання або поєднуватися з патологією спинного мозку. Одним з найважливіших питань при наявності сколіозу у дітей є визначення його природи як ідіопатичного захворювання або пов'язаного з патологією спинного мозку. Важливою і недостатньо вивченою є проблема визначення показань для нейровізуального дослідження спинного мозку при наявності сколіозу. Це необхідно для ранньої діагностики сирингомієлії в дитячо-юнацькому віці і вибору тактики лікування. Метою дослідження було встановлення особливостей клінічних проявів сирингомієлії з її початком у дитячому віці з оцінкою ступеня сколіотичної деформації хребта.
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Books on the topic "Syringomyelia"

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Klekamp, Jörg, and Madjid Samii. Syringomyelia. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-642-56023-1.

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Flint, Graham, and Clare Rusbridge, eds. Syringomyelia. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-13706-8.

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Tamaki, Norihiko, Ulrich Batzdorf, and Tatsuya Nagashima, eds. Syringomyelia. Tokyo: Springer Japan, 2001. http://dx.doi.org/10.1007/978-4-431-67893-9.

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National Institutes of Health (U.S.) and National Institute of Neurological Disorders and Stroke (U.S.). Office of Scientific and Health Reports, eds. Syringomyelia. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1994.

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Anson, John A. Syringomyelia and the Chiari malformations. Edited by AANS Publications Committee. Park Ridge, Ill: American Association of Neurological Surgeons, 1997.

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1928-, Batzdorf Ulrich, ed. Syringomyelia: Current concepts in diagnosis and treatment. Baltimore: Williams & Wilkins, 1991.

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Parker, James N., and Philip M. Parker. The official patient's sourcebook on syringomyelia. Edited by Icon Group International Inc. San Diego, Calif: Icon Health Publications, 2002.

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Deutsche Gesellschaft für Neurochirurgie. Tagung. Stabilizing craniocervical operations: Calcium antagonists in SAH : current legal issues. Berlin: Springer-Verlag, 1990.

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1850-, Hinsdale Guy. Syringomyelia. Franklin Classics, 2018.

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1850-, Hinsdale Guy. Syringomyelia. Franklin Classics Trade Press, 2018.

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Book chapters on the topic "Syringomyelia"

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Klekamp, Jörg, and Madjid Samii. "Introduction." In Syringomyelia, 1–16. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-642-56023-1_1.

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Klekamp, Jörg, and Madjid Samii. "The Pathophysiology of Syringomyelia." In Syringomyelia, 17–25. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-642-56023-1_2.

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Klekamp, Jörg, and Madjid Samii. "Syringomyelia Associated with Diseases at the Craniocervical Junction." In Syringomyelia, 27–109. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-642-56023-1_3.

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Klekamp, Jörg, and Madjid Samii. "Syringomyelia Associated with Diseases of the Spinal Canal." In Syringomyelia, 111–92. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-642-56023-1_4.

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Batzdorf, Ulrich. "Historical Aspects." In Syringomyelia, 1–9. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-13706-8_1.

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Klekamp, Jörg. "Hindbrain-Related Syringomyelia." In Syringomyelia, 141–66. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-13706-8_10.

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Flint, Graham. "Post-traumatic and Post-inflammatory Syringomyelia." In Syringomyelia, 167–84. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-13706-8_11.

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Roy, Anil. "Idiopathic Syringomyelia." In Syringomyelia, 185–92. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-13706-8_12.

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Oakes, Jerry, and Dominic Thompson. "Paediatric Perspectives." In Syringomyelia, 193–207. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-13706-8_13.

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Rusbridge, Clare. "Veterinary Aspects." In Syringomyelia, 209–30. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-13706-8_14.

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Conference papers on the topic "Syringomyelia"

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Azar, M., and C. D. Onofrei. "Prednisone, Syringomyelia and Dyspnea." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a6746.

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Kulinich, Daniel, Ariana Chow, Shivam Rana, Brendan Duong, Aditya Kondajji, Ansley Unterberger, Anjali Pradhan, Farinaz Ghodrati, and Isaac Yang. "The Chiari-I Malformation Associated Syringomyelia." In Special Virtual Symposium of the North American Skull Base Society. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1725432.

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CAVALCANTI, FERNANDA TAVARES DE MELO, SANDRA REJANE CABRAL BATISTA, ANA KARLA GUEDES DE MELO, ALESSANDRA DE SOUSA BRAZ, ANNA LUÍSA MARINHO DE ANDRADE, and EUTÍLIA ANDRADE MEDEIROS FREIRE. "SYRINGOMYELIA AS CAUSE OF CHARCOT'S ARTHROPATHY: A CASE REPORT." In 36º Congresso Brasileiro de Reumatologia. São Paulo: Editora Blucher, 2019. http://dx.doi.org/10.5151/sbr2019-264.

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Marin, Carolina Maria, Gustavo Carvalho Costa, Emilia Correa Souto, Icaro França Navarro Pinto, Igor Braga Farias, Bruno de Mattos Lombardi Badia, Roberta Ismael Lacerda Machado, Paulo Victor Sgobbi Souza, Wladimir Bocca Vieira de Rezende Pinto, and Acary Souza Bulle Oliveira. "Charcot arthropathy in the elbow caused by hydrosiringomyelia." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.148.

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Introduction: Syringomyelia is a chronic disease of the spinal cord that leads to damage to nerve fibers in the spinothalamic tract. The changes in these structures responsible for the thermal and painful sensitivity lead to an abnormal innervation of the joints, which can lead to neuropathic arthropathy, called Charcot arthropathy. Syringomyelia is the main cause of Charcot arthropathy in the upper limbs, and the most involving joints are the shoulder and elbow. It is a rare condition and its recognition allows for early diagnosis and proper management. Case report: A 50-year-old female patient, with a previous history of spinal cord trauma, who has been in a wheelchair since then, started with edema in the left upper limb, mainly in the elbow, evolved with local ulcer and presence of serosanguinolent secretion, weakness and paresthesia in the left hand. Magnetic resonance imaging of the elbow showed signs of neuroarthropathy and the study of the thoracic cervical spine showed enlargement of the cerebrospinal fluid space and tapering of the spinal cord. Conclusions: Charcot’s arthropathy should be considered as a differential diagnosis of pain, edema and limitation of joint movements. When present in the upper limbs, an investigation should be carried out to exclude syringomyelia.
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Elliott, Novak S. J., Anthony D. Lucey, Duncan A. Lockerby, and Andrew R. Brodbelt. "Syringomyelia and the Fluid-Structure Interactions of a Cerebrospinal Waveguide." In ASME 2014 Pressure Vessels and Piping Conference. American Society of Mechanical Engineers, 2014. http://dx.doi.org/10.1115/pvp2014-29095.

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In the disease syringomyelia, fluid-filled cavities, called syrinxes, form in the spinal cord. The expansion of these pathological pressure vessels compresses the surrounding nerve fibers and blood supply, which is associated with neurological damage. We investigate the spinal wave-propagation characteristics, principally to serve as a reference for more anatomically-detailed models. The spinal cord is modeled as an elastic cylinder, which becomes an annulus containing inviscid fluid when a syrinx is included. This is surrounded by an annulus of inviscid fluid, representing the cerebrospinal fluid occupying the subarachnoid space, with an outer rigid boundary approximating the dura mater. The axisymmetric harmonic motion is solved as an eigenvalue problem. We present dispersion diagrams and describe the physical mechanism of each wave mode. We identify potentially damaging syrinx fluid motions and tissue stress concentrations from the eigenvectors. Finally, we determine the dependence of each wave mode on syrinx radius and cord tissue compressibility.
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Cerovac, Anis, Almira Huremović, and Harun Brkić. "Neurosurgical therapy of the Chiari type I malformation with syringomyelia." In NEURI 2015, 5th Student Congress of Neuroscience. Gyrus JournalStudent Society for Neuroscience, School of Medicine, University of Zagreb, 2015. http://dx.doi.org/10.17486/gyr.3.2234.

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Martin, Bryn A., Wojciech Kalata, John N. Oshinski, Francis Loth, and Thomas J. Royston. "Construction and Validation of a Complaint Model of the Cerebrospinal Fluid System With Fluid Filled Syrinx." In ASME 2004 International Mechanical Engineering Congress and Exposition. ASMEDC, 2004. http://dx.doi.org/10.1115/imece2004-62033.

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A simplified model of the cerebrospinal fluid (CSF) system with compliant fluid filled syrinx has been constructed, tested, and verified to closely mimic the in-vivo flow conditions observed through MRI imaging of the pathological CSF system with syringomyelia. The model is subjected to a MRI derived CSF flow waveform from a patient with Syringomyelia through use of a computer controlled pulsatile pump. Model geometry, flow waveform, and spinal cord compliance are obtained at three axial locations along the system through MRI image processing techniques. MRI testing was conducted with the syrinx open and closed to the external environment. Results indicate that the internal and external flow waveforms were in opposite directions when the syrinx was closed and in unison when the syrinx was open. The observed flow waveform and compliance measurements closely mimicked the in-vivo case when the syrinx is open to the external environment.
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Kalata, Wojciech, Bryn Martin, John Oshinski, and Francis Loth. "Hydrodynamics of Cerebrospinal Fluid in Spinal Canal With Chiari Malformation and Syringomyelia." In ASME 2004 International Mechanical Engineering Congress and Exposition. ASMEDC, 2004. http://dx.doi.org/10.1115/imece2004-61918.

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Magnetic resonance imaging (MRI) is has great potential as a tool for diagnosis of neurological diseases such as Chiari malformation (CM) and syringomyelia (SM). Its extended capability to obtain in-vivo velocities of blood or cerebrospinal fluid (CSF) allowed engineers to perform studies with engineering analysis applications such as computational fluid dynamics and physical modeling. Recently, a new MR technique called balanced steady-state free procession (bSSFP) cine imaging was developed for analysis of geometrical changes during the cardiac cycle due to compliance [1]. This study used MRI utilities to investigate hydrodynamic environment of CSF in the sub-cranial subarachnoid space (SAS). A model was constructed to simulate fluid dynamics of CSF in SAS. This model will allow investigation of detailed pressure and velocity using laser Doppler anemometry and pressure transducers. The MRI data from patient, healthy volunteers, the flow model were compared.
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Sparks, C., A. Gorney, K. Williams, B. Lascelles, and N. Olby. "Investigation of Sensory Thresholds in Cavalier King Charles Spaniels with and without Chiari-like Malformations and Syringomyelia." In Pain in Animals Workshop 2017: Abstracts. Schattauer GmbH, 2018. http://dx.doi.org/10.1055/s-0038-1660891.

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Liu, Yifei, Bryn A. Martin, Thomas J. Royston, and Francis Loth. "A Fluid Structure Interaction Simulation of the Cerebrospinal Fluid, Spinal Cord, and Spinal Stenosis Present in Syringomyelia." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19433.

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Syringomyelia (SM) is a neurological disease in which a fluid-filled cystic cavity, or syrinx, forms in the spinal cord (SC) resulting in progressive loss of sensory, motor functions, and/or pain in the patient. It has been hypothesized that abnormal cerebrospinal fluid (CSF) pressure distribution and absorption in the subarachnoid space (SAS), resulting from a CSF flow blockage (stenosis), could be a key etiological factor for syrinx pathogenesis. In particular, the magnitude of the abrupt SAS pressure waves produced during coughing has been correlated with headache and pain in the patient. To better understand the influence of coughing on the spinal SAS, four axisymmetric fluid-structure interaction (FSI) in silico models representative of various conditions associated with SM were constructed. Each of the models was subjected to a cough-like CSF pressure pulse. The CSF flow stenosis was shown to attenuate and decelerate the CSF wave propagation in the SAS. The spinal SAS distensibility was also shown to have significant influence on the wave propagation. The in silico pressure results were found to be in agreement with a set of similar in vitro experiments [1].
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