Relevant bibliographies by topics / Syndrome de Lance-Adams

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers
  5. Reports

Academic literature on the topic 'Syndrome de Lance-Adams'

Author: Grafiati
Published: 8 February 2025

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Journal articles on the topic "Syndrome de Lance-Adams"

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Shin, Jun-Hwa, Jong Moon Park, A. Ram Kim, Hee Suk Shin, Eun Shin Lee, Min-Kyun Oh, and Chul Ho Yoon. "Lance-Adams Syndrome." Annals of Rehabilitation Medicine 36, no. 4 (2012): 561. http://dx.doi.org/10.5535/arm.2012.36.4.561.

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Lee, Ha Lim, and Ju Kang Lee. "Lance-Adams Syndrome." Annals of Rehabilitation Medicine 35, no. 6 (2011): 939. http://dx.doi.org/10.5535/arm.2011.35.6.939.

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Withanagama, CJ, R. Gamage, and HPMC Caldera. "Lance-Adams Syndrome." Galle Medical Journal 17, no. 1 (May 29, 2012): 53. http://dx.doi.org/10.4038/gmj.v17i1.4363.

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Freund, Brin, Raoul Sutter, and Peter W. Kaplan. "Lance-Adams Syndrome in the Pretargeted Temperature Management Era." Clinical EEG and Neuroscience 48, no. 2 (July 10, 2016): 130–38. http://dx.doi.org/10.1177/1550059416643193.

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Acute posthypoxic myoclonus portends a poor prognosis. Another form of posthypoxic myoclonus, Lance-Adams syndrome, is associated with a better outcome. Differentiating these two entities is important in prognostication and guiding further medical intervention. This can be difficult in the acute setting after hypoxic brain injury but the use of neurophysiologic studies may be helpful. In this article, we present a case of a patient who presented after pulseless electrical activity arrest, underwent targeted temperature management and subsequently developed Lance-Adams syndrome. The neurologic and electroencephalographic findings in Lance-Adams syndrome are discussed with an updated review.
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Guo, Yu, Yan Xiao, Li-Fa Chen, De-Hui Yin, and Ruo-Dan Wang. "Lance Adams syndrome: two cases report and literature review." Journal of International Medical Research 50, no. 2 (February 2022): 030006052110599. http://dx.doi.org/10.1177/03000605211059933.

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Hypoxic myoclonus, also known as Lance Adams syndrome, is a rare syndrome that results from the serious brain damage caused by cerebral hypoxia that often follows cardiopulmonary resuscitation. This current case report describes two patients with post-hypoxic myoclonus, both of whom received cardiopulmonary resuscitation. The neurological symptoms of these two patients were significantly improved by the administration of clonazepam and sodium valproate sustained-release tablets. The report presents a literature review detailing the pathogenesis, diagnosis and treatment of Lance Adams syndrome. The timely diagnosis and treatment of Lance Adams syndrome can significantly improve the quality of life of patients. Valproic acid, clonazepam and other antiepileptic drugs can be used. Whether levetiracetam is effective for cortical myoclonus requires further clinical study.
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Gudivada, KiranK, Cherian Roy, and ManuM K. Varma. "Lance-Adams Syndrome after Cardiac Arrest." Neurology India 70, no. 1 (2022): 166. http://dx.doi.org/10.4103/0028-3886.338676.

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Pokryszko-Dragan, Anna. "Anoxic encephalopathy with myoclonus – Lance-Adams syndrome." Family Medicine & Primary Care Review 4 (2015): 338–40. http://dx.doi.org/10.5114/fmpcr/60413.

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Stubblefield, Kassandra, Salman Zahoor, Hasan Sonmezturk, Kevin Haas, Danielle Mattingly, and Bassel Abou-Khalil. "Perampanel is effective against Lance-Adams syndrome." Epileptic Disorders 23, no. 5 (October 2021): 769–71. http://dx.doi.org/10.1684/epd.2021.1329.

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Waddell, Adam, Ahmed Dirweesh, Fausto Ordonez, Charles Kososky, Leema Reddy Peddareddygari, and Raji P. Grewal. "Lance–Adams syndrome associated with cerebellar pathology." Journal of Community Hospital Internal Medicine Perspectives 7, no. 3 (July 3, 2017): 182–84. http://dx.doi.org/10.1080/20009666.2017.1340730.

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Sanna, Giuseppe D., Antonio Demurtas, Stefano Cossa, Francesca Murgia, Paola Murgia, and Annamaria Ara. "A peculiar case of Lance-Adams syndrome." American Journal of Emergency Medicine 34, no. 2 (February 2016): 342.e5–342.e7. http://dx.doi.org/10.1016/j.ajem.2015.06.033.

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Dissertations / Theses on the topic "Syndrome de Lance-Adams"

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Vellieux, Geoffroy. "Syndrome de Lance-Adams : étude translationnelle de l’homme à l’animal." Electronic Thesis or Diss., Sorbonne université, 2024. http://www.theses.fr/2024SORUS432.

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Le syndrome de Lance-Adams est une pathologie neurologie chronique très handicapante rencontrée chez les survivants d'anoxie cérébrale. Il est caractérisé essentiellement par des myoclonies positives, d'action, multifocales ou généralisées, et des myoclonies négatives. Les mécanismes sous-tendant les myoclonies de cette pathologie sont peu connus. De multiples hypothèses ont été proposées depuis la description initiale de ce syndrome. L'étude multimodale d'une large cohorte de patients avec un syndrome de Lance-Adams a montré que les myoclonies sont générées dans le cortex cérébral, en particulier le cortex moteur (ou sensorimoteur). L'observation de l'histoire clinique de certains patients a par ailleurs permis de proposer, avec succès, une nouvelle approche thérapeutique par électroconvulsivothérapies à une patiente pharmacorésistante. Une revue extensive de la littérature a permis de faire émerger une vue d'ensemble et intégrée de cette pathologie et de mieux délimiter le profil de patients. Enfin, nous avons essayé, par différentes approches, de développer un nouveau modèle murin de myoclonies post-anoxiques, sans succès. Une amélioration de cette préparation expérimentale permettrait la réalisation de multiples explorations, notamment immunohistochimiques et électrophysiologiques, afin de mieux comprendre les mécanismes cellulaires et de réseaux à l'origine des myoclonies du syndrome de Lance-Adams
Lance-Adams syndrome is a post-anoxic disabling chronic neurological disorder. The main clinical features are action-induced multifocal or generalized positive myoclonus, and negative myoclonus. The underlying mechanisms of this disorder are poorly understood. Multiple hypotheses have been proposed since the initial description of this syndrome. The multimodal analysis of a large cohort of patients with Lance-Adams syndrome demonstrated that myoclonus originates in the cerebral cortex, particularly in the motor (or sensorimotor) cortex. Additionally, careful observation of the natural history of patients led to the successful proposal of a new therapeutic approach using electroconvulsive therapy in a pharmacoresistant patient. An extensive literature review provided an integrated overview of this pathology and helped better define the patient profile. Lastly, through various approaches, we attempted to develop a new murine model of post-anoxic myoclonus, without success. An improvement in this experimental preparation would allow for multiple explorations, particularly immunohistochemical and electrophysiological studies, to better understand the cellular and network mechanisms underlying the myoclonus in Lance-Adams syndrome
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Book chapters on the topic "Syndrome de Lance-Adams"

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Shibasaki, H. "Lance–Adams Syndrome☆." In Reference Module in Neuroscience and Biobehavioral Psychology. Elsevier, 2017. http://dx.doi.org/10.1016/b978-0-12-809324-5.00652-0.

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Shibasaki, H. "Lance–Adams Syndrome." In Encyclopedia of Movement Disorders, 116–19. Elsevier, 2010. http://dx.doi.org/10.1016/b978-0-12-374105-9.00040-x.

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Conference papers on the topic "Syndrome de Lance-Adams"

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Anis, K. "Post-hypoxic Myoclonus: A Case of Lance Adams Syndrome." In American Thoracic Society 2023 International Conference, May 19-24, 2023 - Washington, DC. American Thoracic Society, 2023. http://dx.doi.org/10.1164/ajrccm-conference.2023.207.1_meetingabstracts.a3542.

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López, E. Lázaro, MM Jalón Urbina, AI Plano Sánchez, A. Rodríguez Ferreras, C. Carriles Fernández, I. Zapico García, L. Velasco Roces, and L. Gómez de Segura Iriarte. "DI-096 Refractory lance–adams syndrome: pharmacotherapy management and iatrogenic complications." In 22nd EAHP Congress 22–24 March 2017 Cannes, France. British Medical Journal Publishing Group, 2017. http://dx.doi.org/10.1136/ejhpharm-2017-000640.343.

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Primera, G., M. Ingemi, and K. H. Young. "A Case of Lance- Adams Syndrome Following Peri Arrest and Severe Hypoxia From Opioid Overdose: A Case Report." In American Thoracic Society 2023 International Conference, May 19-24, 2023 - Washington, DC. American Thoracic Society, 2023. http://dx.doi.org/10.1164/ajrccm-conference.2023.207.1_meetingabstracts.a5194.

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Reports on the topic "Syndrome de Lance-Adams"

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Shijani, Seyed Mohammad Malakooti, Sina Neshat, Hossein Shayestehyekta, and Milad Gorgani. Lance-Adams syndrome; what we know now. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0025.

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Review question / Objective: 1. In Lance-Adams syndrome, what is the effect of current therapeutic management on improving patients' condition compared with the control group? 2. Are EEG, Brain CT, MRI, and brain SPECT more accurate in diagnosing Lance-Adams syndrome? 3. Does Early diagnosis and treatment influence the quality of life in patients with Lance-Adams syndrome? 4. Are patients with abnormal cortical discharge or cerebellum brain stem and thalamus cortical circuit or neurotransmitter imbalance at higher risk for/of Lance-Adams syndrome compared with patients without these symptoms? Condition being studied: LAS is a group of clinical symptoms; The primary manifestation is action myoclonus which can occur as generalized, focal, or multifocal repeated myoclonic motor movement myoclonus. In some patients, sensory stimuli can trigger myoclonus. Furthermore, negative myoclonus can impair posture and cause falls in the lower extremities.
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