Relevant bibliographies by topics / Swine – Fetuses

Academic literature on the topic 'Swine – Fetuses'

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Published: 4 June 2021
Last updated: 19 February 2022

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Journal articles on the topic "Swine – Fetuses"

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Polaček, Vladimir, Biljana Đurđević, Tamaš Petrović, Jasna Prodanov-Radulović, Milena Samojlović, Ivana Vučićević, and Sanja Kovačević-Aleksić. "CLASSICAL SWINE FEVER VIRUS DETECTION IN FETAL SWINE TISSUES BY IMMUNOHISTOCHEMISTRY." Archives of Veterinary Medicine 13, no. 1 (August 10, 2020): 83–100. http://dx.doi.org/10.46784/e-avm.v13i1.235.

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The classical swine fever virus has the ability to cross the placental barrier, resulting in the infection of fetuses, which may consequently lead to persistent infection in piglets. The aim of this study was to report the lesions in fetuses naturally infected with CSFV during late gestation and clarify the nature of infected cells and the distribution of viral antigen in different tissues. A total of twenty-nine fetuses aged 82, 83 and 95 gestational days originating from three naturally CSFV infected sows were examined in this study. In all tested sows and their fetuses CSFV was detected using RT-PCR method. Immunohistochemistry method was used to detect viral antigen and monoclonal antibody WH303 was used on formalin fixed tissue samples of brain, spleen, heart, tonsils, kidney, ileocecal valve and umbilical cord. The most common lesions in the majority of fetuses were hyperemia, petechial haemorrhages in the skin, lymph nodes and kidneys. With the exception of myocardium, CSF viral antigen was detected in all the examined tissues. WH303 positive cells included endothelial cells, monocytes, macrophages and lymphocytes. The largest number of positive cells was found in kidneys in all of the examined fetuses. Reticular cells, macrophages, lymphocytes and endothelial cells in the spleen were also intensely and widely stained in most of the fetuses. These results showed that CSFV antigen can be detected in formalin-fixed, paraffin-embedded fetal tissue specimens originating from naturally CSFV infected sows by using monoclonal antibody WH303. Fetal kidneys proved to be a very useful organ for diagnosis of the CSF virus. Having that in mind, it is assumed that persistently infected piglets may shed a high amount of viral particles through urine. However, further research is needed to confirm this hypothesis.
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INOMATA, Tomoo, Seiya INOUE, Toshio OSHIDA, and Tsunenori NAKAMURA. "Development of External Genitalia in Swine Fetuses." Japanese journal of animal reproduction 35, no. 4 (1989): 228–33. http://dx.doi.org/10.1262/jrd1977.35.228.

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Kim, Hyun S., Han S. Joo, and Martin E. Bergeland. "Serologic, Virologic, and Histopathologic Observations of Encephalomyocarditis Virus Infection in Mummified and Stillborn Pigs." Journal of Veterinary Diagnostic Investigation 1, no. 2 (April 1989): 101–4. http://dx.doi.org/10.1177/104063878900100201.

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Stillborn and mummified swine fetuses from swine farms experiencing reproductive problems were investigated for evidence of infection with encephalomyocarditis (EMC) virus by fetal serology, virus isolation, and histopathologic examination. Fetal sera or thoracic fluids of 478 abnormal fetuses collected during January through December 1987 were tested for the presence of antibody specific to EMC virus. Of 478 samples tested, 175 (36.6%) had EMC virus serum neutralizing antibody titers of 1:64 or greater. The samples positive for EMC virus antibody were obtained from 38 swine farms located in 9 states in the United States. In addition to serologic observations, tissue samples of some abnormal fetuses were examined for the presence of virus and histopathologic lesions. The EMC virus was isolated in 1 case from the fetuses of an aborted litter. The isolate was serologically identical to a reference EMC virus. Nonsuppurative encephalitis and myocarditis were observed in the fetal samples collected from 2 different herds. Thoracic fluid of 1 stillborn pig with lesions was positive for EMC virus antibody at a titer of 1:512. We suggest that a widespread reproductive problem recently experienced in several major swine-producing areas of the United States may have been caused by EMC virus infection.
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Kim, H. S., W. T. Christianson, and H. S. Joo. "Pathogenic properties of encephalomyocarditis virus isolates in swine fetuses." Archives of Virology 109, no. 1-2 (March 1989): 51–57. http://dx.doi.org/10.1007/bf01310517.

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DYCK, G. W., and R. M. McKAY. "INTRAUTERINE ENVIRONMENTAL FACTORS AFFECTING FETAL WEIGHT AT MID-PREGNANCY IN SWINE." Canadian Journal of Animal Science 66, no. 4 (December 1, 1986): 945–50. http://dx.doi.org/10.4141/cjas86-104.

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The relationship between fetal weight (FW) at mid-pregnancy, and uterine weight of the area of placental attachment (UW), fetal membrane weight (FMW), allantoic fluid volume (ALFV), amniotic fluid volume (AMFV), fetal sex (FS), left vs. right uterine horn (UH), number of fetuses per uterine horn (NF), fetal location within the uterine horn (FL), fetal age (FA), dietary intake (DI) and year group of gilts (YG) was determined on 935 fetuses from 93 Lacombe gilts, bred to Yorkshire boars, that were slaughtered at 58–62 d of gestation. A stepwise regression analysis was utilized with 9 of the 11 variables found to have a significant effect and accounted for 66.5% of the variation in fetal weight. Uterine weight of the area of placental attachment (UW) accounted for 47.5% of the variation in fetal weight. The next five variables (FA, AMFV, FS, FMW and ALFV) accounted for a further 17.9% of the variation in fetal weight. The remaining 1.1% reduction in variation in fetal weight occurred with the inclusion of FL, DI and NF. The effects of the UH and YG were not significant. Thus, the parameters associated with the individual fetus are the most important variables influencing fetal weight with the uterine weight of the area of placental attachment being of greatest importance. Key words: Gilts, mid-pregnancy, fetal weight
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Choi, C. S., T. W. Molitor, H. S. Joo, and R. Gunther. "Pathogenicity of a skin isolate of porcine parvovirus in swine fetuses." Veterinary Microbiology 15, no. 1-2 (October 1987): 19–29. http://dx.doi.org/10.1016/0378-1135(87)90125-8.

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Cornell, William D., Muckatira M. Chengappa, Louis A. Stuart, Roxanna L. Maddux, and Robert I. Hail. "Brucella Suis Biovar 3 Infection in a Kentucky Swine Herd." Journal of Veterinary Diagnostic Investigation 1, no. 1 (January 1989): 20–21. http://dx.doi.org/10.1177/104063878900100107.

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Sows from a large far-row-to-finish operation in western Kentucky had late-term abortions. Boars and breeding-age sows were tested serologically for brucellosis, and 83 of 125 were classified as reactors. No brucellae were isolated from the tissues of 6 unbred reactor sows, but Brucella suis biovar 3 was recovered from 5 aborted fetuses. Epidemiological studies failed to determine the source of the infection.
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Kwit, Krzysztof, Małgorzata Pomorska-Mól, and Iwona Markowska-Daniel. "Infectious agents involved in reproduction failure in swine." Medycyna Weterynaryjna 72, no. 6 (2016): 345–51. http://dx.doi.org/10.21521/mw.5523.

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Problems in the reproduction of pigs may be the result of interaction of various factors, both infectious and non-infectious. Among the infectious agents, the greatest economic losses are caused by viral infection of pregnant gilts and sows. In the present study the most important pathogens causing reproductive disorders in pigs, including parvovirus (PPV), porcine reproductive and respiratory syndrome (PRRSV), swine influenza virus (SIV), porcine circovirus type 2 (PCV2), enteroviruses, encephalitis virus (EMCV), Aujeszky's disease virus (ADV), classical swine fever virus (CSFV), Leptospira spp., Brucella suis and Erysipelotrix rhusiopathiae are characterized. So far, three possible ways of natural infection of the embryo or fetus are identified: via placenta, through the cervical canal, and by infection of the egg cell. The consequences of infection of pregnant females depend on the species of the virus, the gestation period, wherein there is an infection and immune status of pregnant females. The most common changes included: embryo death, resorption of embryos, mummification of fetuses, malformations, abortions, birth of dead or very weak piglets. Because of the importance of the reproduction sector for the competitive production of pigs, the monitoring of the health status of breeding stock, including compliance with all biosecurity rules and vaccination schedules, should be strictly respected by veterinarians taking care of pig breeding herds
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Salogni, Cristian, Massimiliano Lazzaro, Enrico Giacomini, Stefano Giovannini, Mariagrazia Zanoni, Matteo Giuliani, Jessica Ruggeri, et al. "Infectious agents identified in aborted swine fetuses in a high-density breeding area." Journal of Veterinary Diagnostic Investigation 28, no. 5 (July 11, 2016): 550–54. http://dx.doi.org/10.1177/1040638716656024.

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Polacek, V., B. Bozic, J. Prodanov-Radulovic, T. Petrovic, I. Vucicevic, Z. Becskei, and S. Kovacevic-Aleksic. "Immunohistochemical Detection of Classical Swine Fever Virus in Fetuses from Naturally-Infected Sows." Journal of Comparative Pathology 156, no. 1 (January 2017): 102. http://dx.doi.org/10.1016/j.jcpa.2016.11.154.

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Dissertations / Theses on the topic "Swine – Fetuses"

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Brown, Larry Dale. "Subchronic bioavailability and disposition of bivalent lead in pregnant swine and fetuses." free to MU campus, to others for purchase, 1998. http://wwwlib.umi.com/cr/mo/fullcit?p9901221.

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Hoyle, Ashley Sabine. "The Role of Supplemental Beef vs. Sugar during Pregnancy on Fetal and Offspring Developmental Programming in Swine." Thesis, North Dakota State University, 2019. https://hdl.handle.net/10365/29794.

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Sugar intake is linked to developmental programming of obesity and diabetes. We hypothesized that supplementing ground beef in place of sugar during pregnancy would reduce fetal and offspring developmental programming. Gestating sows were fed 1 of 4 isocaloric supplements: control, ground beef, granulated sugar, or beef plus sugar. In the fetal study supplements were fed from d 40 to 110 of gestation and in the offspring study from d 40 until weaning. Gene expression differences in fetal liver and muscle were observed for IGF2 (P = 0.04), FBPase (P = 0.03), and IGF2R (P = 0.02). Differences were also seen in offspring back fat (sex by day interaction, P = 0.01), longissimus dorsi muscle area (treatment by sex, P = 0.001), body weight (sex, P = 0.0006; sex by day interaction, P < 0.0001), and plasma insulin concentrations (treatment by sex, P = 0.0002).
North Dakota Beef Commission
Topigs Norsvin
North Dakota. State Board of Agricultural Research and Education
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3

Rigau, Alberto Pérez. "An assessment of the effects of dietary oil supplementation on fetal survival in gilts at 40 days of gestation." Thesis, Virginia Tech, 1993. http://hdl.handle.net/10919/44875.

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Eighty-six crossbred (Duroc x Yorkshire) gilts were used in two trials (50 gilts in Trial 1 and 36 gilts in Trial 2) for an assessment of the effect of supplemental dietary fat during early gestation on fetal survival, fetal development, and fatty acid concentration in gilt plasma and fetal head and body. Three diets contained 4% (w/w) added fat either as coconut, soybean, or fish (menhaden) oils. A fourth diet was used as a control. On d 37 to 45 postbreeding, gilts were slaughtered and numerous fetal and ovarian measurements made. Two sets of four randomly selected fetuses per gilt from Trial 1 were prepared. Blood samples from each gilt were obtained on the day of slaughter for determination of the plasma fatty acid profile. Across both trials, percentage fetal survival did not differ according to treatment, but in Trial 2 fetal survival was higher (P < .06) for gilts fed fish oil, compared with the controls. The fatty acid profile of plasma of gilts and the conceptus tissues were similiar; both were influenced by the fatty acid concentration of the diets. The ratio of n-3/n-6 fatty acids was higher in conceptus tissue than in maternal plasma and the ratio was higher (P < .05) for the fish oil diet compared with the other diets. The relatively high concentration of omega-3 fatty acids in fetal tissues supports the hypothesis that omega-3 fatty acids play a role in the development of the pig conceptus and contributes to improve fetal survival. However, the high percentage fetal survival observed in all the treatments may have masked benefits of supplemental oil.


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4

Wippermann, Wolf. "Diaplazentare Deoxynivalenolintoxikation bei Schweinefeten. Lassen sich am 70. Trächtigkeitstag histomorphologisch und immunhistologisch diagnostisch verwertbare Befunde erheben?" Doctoral thesis, Universitätsbibliothek Leipzig, 2011. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-67849.

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Dean, Sophia Katrina Prince of Wales Clinical School UNSW. "Transplantation of fetal pig islet-like cell clusters as therapy for diabetes." 2007. http://handle.unsw.edu.au/1959.4/40870.

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Fetal pig islet-like cell clusters (ICCs) were transplanted into the thymus or omentum of STZ-induced diabetic pigs immunosuppressed with cyclosporine (CsA) and deoxyspergualin (DSG), as a potential treatment for type 1 diabetes. C-peptide levels in response to glucagon and arginine significantly increased over time using 1 litter of ICCs with highest levels obtained at 100 days post-transplantation. Increasing the number of ICCs to 2 litters was not advantageous. Histology of the graft showed all 4 pancreatic endocrine cells. Normoglycaemia was achieved for transient periods without insulin administration in 4 out of 12 pigs. These results suggest sub-optimal insulin production, possibly due to the adverse effects of CsA on the grafted β cells. The effect of CsA on adult porcine β cells was investigated and adverse effects were shown. Renal toxicity and adverse changes to the haematological parameters did not occur despite high CsA levels although minimal toxicity to the liver was observed. The results indicate that the use of CsA monotherapy in the maintenance phase to prevent rejection of allografted pancreatic β cells may become a subsequent problem over time. As an alternative to chronic immunossuppression, anti-CD3 monoclonal antibody was administered for 10 days in pigs. Using anti-CD3 alone, only 1 out 4 pigs showed cells positive for insulin. The addition of a 5-day CsA course administered the day before transplantation did not promote allograft survival. The use of DSG for 10 days with anti-CD3 promoted graft survival with the histology showing the 4 endocrine cells 3 weeks post-transplantation. An attempt was made to replace any form of immunossuppression by encapsulating fetal pig ICCs in barium alginate, which were able to remain viable when transplanted in NOD/SCID mice. Fibrosis was detected in BALB/c mice transplanted with encapsulated fetal ICCs suggesting that fetal pig ICCs shed antigens that elicit an immune response. Results from this study show that although fetal pig ICCs may be a viable source of insulin-producing cells, the use of CsA to prevent rejection has adverse effects on graft function. Encapsulation as well as transient immunosuppression is worthy of further investigation to prevent rejection of fetal pig ICCs.
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LIN, Yu-chi, and 林鈺齊. "Changes in mRNA profile between embryos and early fetus in swine." Thesis, 2005. http://ndltd.ncl.edu.tw/handle/53503713803221419313.

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碩士
中華大學
資訊工程學系碩士班
93
Pigs are polytocous animals and prolificacy is an economically important trait in swine industry. About one third of embryos on their journey converting into fetus have been lost reportedly before fully completing their uterine implantation, which occurring between day 18 and day 24 of pregnancy in sows. This study was conducted to compare messenger RNA expression profile between tissues of day 18 embryos and day 24 fetus. Three L×Y crossbred gilts aged about 8 months were selected and induced to estrus hormonally. The gilts then were artificially inseminated twice. Surgeries were performed on day 18 (two gilts) or day 24 (one gilt) of pregnancy for collection of embryos or fetus. Total RNA were extracted from 12 samples (6 of each) and separately converted into fluorescent cDNA for the microchip hybridization analysis using probes homologous to human’s sequence. In total 142 genes were expressed specifically, including 102 genes expressed highly in embryos and 40 highly in fetus. The majority of these specific genes expressed at embryonic stage belong to either keratin family or adhesion molecules, suggesting that cellular migration and adhesion of the epithelium are essential to the embryo. On the contrary, most genes expressed predominantly at fetal stage were associated with functions of circulation, immune, or neuron, indicating initiation of vigorous organogenesis. These results may be helpful to deduce the factors that leads to normal fetal development or early fetal death.
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Wippermann, Wolf. "Diaplazentare Deoxynivalenolintoxikation bei Schweinefeten. Lassen sich am 70. Trächtigkeitstag histomorphologisch und immunhistologisch diagnostisch verwertbare Befunde erheben?" Doctoral thesis, 2010. https://ul.qucosa.de/id/qucosa%3A11161.

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Books on the topic "Swine – Fetuses"

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F, Walker Warren, ed. Anatomy and dissection of the fetal pig. 4th ed. New York: W.H. Freeman, 1988.

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Dissection of the fetal pig. Burlington, N.C: Rex Educational Resources Co., 1985.

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Allen, Connie. Fetal pig dissection: A laboratory guide. New York: Wiley, 2003.

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Allen, Connie. Fetal pig dissection: A laboratory guide. 2nd ed. Hoboken, NJ: Wiley, 2006.

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Chiasson, Robert B. Laboratory anatomy of the fetal pig. Dubuque, IA: Wm. C. Brown Publishers, 1995.

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Chiasson, Robert B. Laboratory anatomy of the fetal pig. Dubuque, IA: Wm. C. Brown Publishers, 1997.

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Gilbert, Stephen G. Pictorial anatomy of the fetal pig. 2nd ed. Seattle: University of Washington Press, 1987.

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Donnelly, Patricia J. Laboratory manual for anatomy and physiology, with fetal pig dissections. New York, NY: HarperCollins, 1993.

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Human anatomy & physiology: Laboratory manual, fetal pig dissection. 2nd ed. Boston: McGraw Hill Higher Education, 2004.

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Martin, Terry R. Human anatomy & physiology: Laboratory manual fetal pig dissection. Boston: McGraw Hill, 2001.

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