Dissertations / Theses on the topic 'Swimming – Physiological aspects'
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Beltz, John D. "Physiological response to interval training." Virtual Press, 1987. http://liblink.bsu.edu/uhtbin/catkey/486191.
Full textKerr, Chadwick G. "Hypothermia during Olympic triathlon : influence of body heat storage during the swimming stage." Virtual Press, 1996. http://liblink.bsu.edu/uhtbin/catkey/1014852.
Full textSchool of Physical Education
Flynn, Michael Gerald. "Fat storage in athletes : the metabolic and hormonal responses to swimming and running exercise." Virtual Press, 1987. http://liblink.bsu.edu/uhtbin/catkey/516210.
Full textD'Acquisto, Leonardo J. "Relationship between swimming economy, skill, power, and performance in the breaststroke." Virtual Press, 1987. http://liblink.bsu.edu/uhtbin/catkey/486593.
Full textGao, Jiaping. "The influence of NaHCOb3s ingestion on interval swimming : acid-base balance and performance." Virtual Press, 1987. http://liblink.bsu.edu/uhtbin/catkey/487342.
Full textVitelli, Carol A. "Comparison of heart rate to lactate as related to performance of competitive male swimmers." Virtual Press, 1986. http://liblink.bsu.edu/uhtbin/catkey/445247.
Full textLyttle, Andrew. "Hydrodynamics of the human body during the freestyle tumble turn." University of Western Australia. Dept. of Human Movement and Exercise Science, 2000. http://theses.library.uwa.edu.au/adt-WU2006.0126.
Full textHawley, John A. "The effect of treadmill running and swimming on citrate synthase activity and glycogen levels in the rat." Virtual Press, 1986. http://liblink.bsu.edu/uhtbin/catkey/450972.
Full textUpshaw, Kris. "The effects of stroke rate and stroke length on upper quadrant stroke patterns in competitive swimming." Virtual Press, 1995. http://liblink.bsu.edu/uhtbin/catkey/958784.
Full textSchool of Physical Education
Slawson, Sian. "A novel monitoring system for the training of elite swimmers." Thesis, Loughborough University, 2010. https://dspace.lboro.ac.uk/2134/6292.
Full textSmith, Heather Karen. "The aerobic energy cost of backstroke swimming in elite male athletes /." Thesis, McGill University, 1987. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=66128.
Full textHoyeck, Edward. "The effects of moderate swimming exercise on immune system function in C57 BL/6(B6) mice /." Thesis, McGill University, 2000. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=33288.
Full textHeieis, Mark Rudolf Alois. "Blood volume distribution in and bioenergetics of swimming and diving ducks." Thesis, University of British Columbia, 1987. http://hdl.handle.net/2429/26417.
Full textScience, Faculty of
Zoology, Department of
Graduate
Malone, Kathleen N. "The effect of body position on spinal cord injured swimmers." Virtual Press, 1990. http://liblink.bsu.edu/uhtbin/catkey/722236.
Full textSchool of Physical Education
Hardt, Julie E. "Optimization of the competitive swimming track start based on lower limb asymmetry." University of Western Australia. School of Sport Science, Exercise and Health, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0192.
Full textBattle, Robert A. "Comparison of total and high-density lipoprotein cholesterol in male recreational swimmers and sedentary controls." Thesis, Virginia Polytechnic Institute and State University, 1985. http://hdl.handle.net/10919/104291.
Full textSleeper, Mark D. "The effect of an equi-intensity treadmill running and swimming training protocol on the adipocyte insulin sensitivity and responsiveness in the rat." Virtual Press, 1987. http://liblink.bsu.edu/uhtbin/catkey/482304.
Full textSantos, Gustavo Barbosa dos 1981. "Melatonina reduz o estresse oxidativo e as alterações cardiovasculares induzidas pelo estanozolol em ratos submetidos ao exercicio de natação." [s.n.], 2010. http://repositorio.unicamp.br/jspui/handle/REPOSIP/314571.
Full textDissertação (mestrado) - Universidade Estadual de Campinas, Instituto de Biologia
Made available in DSpace on 2018-08-15T12:08:35Z (GMT). No. of bitstreams: 1 Santos_GustavoBarbosados_M.pdf: 1865766 bytes, checksum: 33bc98691c7f175ef683408a35219198 (MD5) Previous issue date: 2010
Resumo: Esteróides androgênicos anabolizantes (EAA) são indicados clinicamente para promover aumento da síntese protéica após queimaduras, cirurgias, radioterapia, no tratamento contraceptivo, no hipogonadismo, na osteoporose, na sarcopenia relacionada à idade e à pacientes portadores de HIV. Por outro lado, o uso indiscriminado dos EAA, com intuito de aumentar o desenvolvimento muscular, o desempenho físico, a capacidade aeróbia, a tolerância ao treinamento de alta intensidade e até mesmo para fins estéticos, é crescente entre atletas e esportistas recreacionais. O uso abusivo de EAA está relacionado à toxicidade cardíaca e hepática em consequência do aumento do estresse oxidativo. Por outro lado, estudos apontam a melatonina como substância com significativa ação antioxidante, apresentando efeitos benéficos no tratamento de doenças cardíacas. Este trabalho teve por objetivo avaliar os efeitos da melatonina sobre biomarcadores do estresse oxidativo e parâmetros cardiovasculares e hepáticos em ratos adultos sedentários ou treinados com natação e tratados com estanozolol. Os ratos foram distribuídos nos seguintes grupos: sedentário (S), sedentário+estanozolol (SE), sedentário+estanozolol+melatonina (SEM), treinado (T), treinado+estanozolol (TE) e treinado+estanozolol+melatonina (TEM). Realizou-se avaliação eletrocardiográfica no início e ao final do período experimental (6 semanas), sendo, então, determinada a pressão arterial, atividade de enzimas antioxidantes e da fosfatase alcalina e histologia do coração e do fígado. Os resultados mostraram que o estanozolol provocou bradicardia, queda do peso relativo do fígado e aumento da atividade das enzimas superóxido dismutase cardíaca e hepática, catalase cardíaca e fostatase alcalina hepática. Quando associado ao treinamento, estanozolol aumentou a pressão arterial sistólica e diastólica, o peso relativo do coração, desviou o eixo elétrico cardíaco para esquerda e provocou alterações hepatotóxicas. A administração da melatonina nos ratos tratados com EST, por sua vez, impediu o aumento da pressão arterial sistólica e diastólica e da atividade das enzimas catalase cardíaca, fostatase alcalina hepática além de impedir o desvio do eixo elétrico cardíaco causado pela hipertrofia ventricular esquerda induzida pelo estanozolol. Além disso, melatonina reduziu as alterações nos hepatócitos induzidas pelo estanozolol. Concluímos que, em nossas condições experimentais, a melatonina atenuou os efeitos adversos ao sistema cardiovascular e ao fígado causados pelo uso de doses suprafisiológicas de estanozolol
Abstract: Anabolic androgenic steroids (AAS) are nominated for clinical use to promote protein synthesis after burns, surgery, radiotherapy, on contraceptive treatment, osteoporosis, hypogonadism, age-related sarcopenia and HIV patients. However, the indiscriminate use of ASS aiming to stimulate muscular development, physical performance, aerobic capacity, tolerance to high-intensity training and with aesthetic purpose is increasing among athletes and recreational sportsmen. The abusive use of ASS is related to oxidative stress-induced cardiac and hepatic toxicity. Nonetheless, many studies point to melatonin as a substance with antioxidant properties, with beneficial effects on cardiovascular diseases treatment. The purpose of this study was to assess melatonin's effects on oxidative stress biomarkers, cardiovascular and liver parameters in stanozolol-treated trained rats. Rats were divided into the following groups: sedentary (S), sedentary+stanozolol (SS), sedentary+stanozolol+melatonin (SSM), trained (T), trained+stanozolol (TS) and trained+stanozolol+melatonin (TSM). Electrocardiography assessment were performed at the beginning and at the end of experimental period, and then, blood pressure, antioxidant enzymes and phosphatase alkaline activities, heart and liver histology were determined. Stanozolol induced bradycardia, relative liver weight decreased and increased cardiac and liver superoxide dismutase, cardiac catalase and liver phosphatase alkaline activities. Stanazolol plus training induced increased systolic and diastolic blood pressure, relative heart weight, left cardiac axis deviation and toxic liver damage. Melatonin induced decreased systolic and diastolic blood pressure, cardiac catalase and liver phosphatase alkaline activities, and prevented stanazolol-induced left cardiac axis deviation. Furthermore, melatonin decreased stanozolol-induced liver damage. In conclusion, under our experimental conditions, the side effects of supraphysiology doses of stanozolol on liver and cardiovascular system are mitigated by melatonin
Mestrado
Fisiologia
Mestre em Biologia Funcional e Molecular
Neal, Craig M. "Training intensity distribution, physiological adaptation and immune function in endurance athletes." Thesis, University of Stirling, 2011. http://hdl.handle.net/1893/9299.
Full textHale, David. "Changes in hormone excretion in swimmers over the course of a training season." Thesis, 1991. http://hdl.handle.net/10125/9423.
Full textZochowski, Thomas. "Effect of hand cooling on thermal and psychophysical strain and performance during high intensity intermittent training of elite swimmers." Thesis, 2006. http://hdl.handle.net/1828/2097.
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