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Journal articles on the topic 'Susceptibilité au stress'

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Published: 20 July 2024

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1

Tiret, Laurence. "Gene-environment interaction: a central concept in multifactorial diseases." Proceedings of the Nutrition Society 61, no. 4 (November 2002): 457–63. http://dx.doi.org/10.1079/pns2002178.

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RésuméA la différence des mutations génétiques rares mais séveères qui causent les maladies monogéniques, les facteurs génétiques qui modulent la susceptibilité individuelle aux maladies multifactorielles (maladies cardiovasculaires, cancer, diabeète) sont des formes fréquentes, fonctionnellement différentes, des ge`nes (polymorphismes) qui ont généralement un effet modeste au niveau individuel, mais en raison de leur grande fréquence dans la population, peuvent être associés à un risque attribuable élevé. Les facteurs environnementaux peuvent révéler ou faciliter l'expression phénotypique de ces ge`nes de susceptibilité. En effet, dans le cas des maladies communes, les effets génétiques peuvent etre considérablement amplifiés en présence de facteurs déclenchants. On sait maintenant que la plupart des geènes de susceptibilité aux maladies communes n'ont pas un rôle étiologique primaire dans la prédisposition a` la maladie mais agissent plutot comme des modificateurs dans la réponse a` des facteurs exoge`nes tels que le stress, l'environnement, la maladie, la prise de médicament, le régime alimentaire. Une meilleure caractérisation des interactions entre facteurs génétiques et environnementaux constitue un élément clé dans la compréhension de la pathogéneèse des maladies multifactorielles. Cet article présente trois exemples d'interaction gène-environnement dans le domaine des maladies coronariennes.

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2

Benajiba, N., J. De Leiris, B. Lyan, A. Derouiche, N. Mokhtar, and H. Aguenaou. "Effet de l’huile d’argan sur la contractilité de l’aorte : susceptibilité au stress oxydatif." Oléagineux, Corps gras, Lipides 13, no. 1 (January 2006): 76–80. http://dx.doi.org/10.1051/ocl.2006.0076.

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3

Chakroun Feki, Nozha, Patrice Therond, Pierre Jouannet, and Jacques Auger. "Composition lipidique des spermatozoides humains et susceptibilité au stress oxydant avant et après migration dans le mucus cervical." Andrologie 13, no. 4 (December 2003): 381–92. http://dx.doi.org/10.1007/bf03035205.

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4

Auxéméry, Y. "L’état de stress post-traumatique comme conséquence de l’interaction entre une susceptibilité génétique individuelle, un évènement traumatogène et un contexte social." L'Encéphale 38, no. 5 (October 2012): 373–80. http://dx.doi.org/10.1016/j.encep.2011.12.003.

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5

Pospichal, A., D. Pokorova, T. Vesely, and V. Piackova. "Susceptibility of the topmouth gudgeon (Pseudorasbora parva) to CyHV-3 under no-stress and stress conditions." Veterinární Medicína 63, No. 5 (May 29, 2018): 229–39. http://dx.doi.org/10.17221/88/2017-vetmed.

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Cyprinid herpesvirus 3 (CyHV-3), also known as koi herpesvirus, is the causative agent of the highly contagious koi herpesvirus disease, which is restricted to koi and common carp and causes significant losses in both fish stock. Some experimental investigations have shown that other cyprinid or non-cyprinid species may be asymptomatically susceptible to this virus and might play roles as potential carriers of CyHV-3 or might contribute to persistence of this virus in environment. Therefore, it seems important to verify not only the susceptibility of other cyprinid or non-cyprinid species, but also their ability to transmit CyHV-3 infection to susceptible species. Our previous investigation of the susceptibility of the topmouth gudgeon (Pseudorasbora parva) did not reveal the presence of CyHV-3 DNA in the tissues of this species after cohabitation with infected koi. Consequently, we changed the experimental conditions and applied two stress factors (removal of skin mucus and scaring) which would presumably mimic the stress most commonly encountered in the wild. Both experiments (without and with stress factors) consisted of primary and secondary challenges. In both the no-stress and stress experiments, the first challenge was focused only on testing the susceptibility of the topmouth gudgeon to the virus. With the secondary challenge, we investigated potential viral transmission from the topmouth gudgeon to healthy naive koi after exposure to stress factors. All fish (dead, surviving and sacrificed) were tested for the presence of CyHV-3 DNA using nested PCR (no-stress experiment) and real-time PCR (stress experiment). After the primary challenge of the no-stress experiment, PCR did not reveal the presence of CyHV-3 DNA in any specimen of cohabitated topmouth gudgeon, but all specimens of dead koi were CyHV-3 DNA-positive. PCR of fish tissues subjected to the secondary challenge did not show the transfer of virus to naive fish. After exposure to stress (removal of skin mucus), qPCR revealed four out of five samples (80%) of topmouth gudgeon to be positive for CyHV-3 DNA. Two out of five samples (40%) of topmouth gudgeon treated by scaring were found to be positive for the presence of viral DNA. Real-time PCR after the secondary challenge did not reveal any viral DNA positivity in specimens of topmouth gudgeon from groups previously exposed to stress. The stress experiments show that removal of skin mucus might potentially lead to susceptibility of topmouth gudgeon to CyHV-3 infection, but the transmission of the virus to koi carp was not observed.

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6

Paré, William P. "Prior stress and susceptibility to stress ulcer." Physiology & Behavior 36, no. 6 (January 1986): 1155–59. http://dx.doi.org/10.1016/0031-9384(86)90493-2.

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7

Salim, Samina, Hesong Liu, and Fatin Atrooz. "Early life stress, stress-resilience/susceptibility and oxidative stress." Free Radical Biology and Medicine 120 (May 2018): S165. http://dx.doi.org/10.1016/j.freeradbiomed.2018.04.542.

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8

Majlingová, A., M. Závacká, and D. Kliment. "An assessment of hucava mountain stream catchment susceptibility to flooding." Journal of Forest Science 58, No. 12 (December 11, 2012): 553–59. http://dx.doi.org/10.17221/75/2011-jfs.

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The paper deals with an assessment of the Hucava mountain stream catchment susceptibility to flooding. The model catchment of the Hucava mountain stream is located in the Polana Protected Landscape Area – Biosphere Reserve, situated in the central part of Slovakia. The assessment of overall susceptibility of the model area to flooding is based on the multi-criteria evaluation of environmental factors, which crucially affect the hydrological cycle. These are represented by the geology, soil types, slope, forest type group, degree of ecological stability, exploitation of non-forest landscape and potential runoff in the model area. The methodology is based on the synthesis and subsequent processing of data in the GIS environment. The result is represented by the specification of categories (degrees of  flood risk)  to classify the model area to five degrees of overall susceptibility to flood as well as by the creation of maps representing the spatial distribution of different categories in the Hucava catchment.  

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9

Ebner, Karl, and Nicolas Singewald. "Individual differences in stress susceptibility and stress inhibitory mechanisms." Current Opinion in Behavioral Sciences 14 (April 2017): 54–64. http://dx.doi.org/10.1016/j.cobeha.2016.11.016.

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10

Pohl, Tobias, and Hanna Hörnberg. "SOCIAL STRESS DURING ADOLESCENCE: NEUROIMMUNOLOGICAL SIGNATURES OF STRESS SUSCEPTIBILITY." IBRO Neuroscience Reports 15 (October 2023): S47. http://dx.doi.org/10.1016/j.ibneur.2023.08.2148.

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11

Nsangou, A. S., D. A. Mbah, C. L. Tawah, T. K. Manchang, G. S. Bah, Y. Manjeli, C. A. Njehoya, Y. Mfopit, and C. Nguetoum. "Amélioration génétique bovine par voie de croisement et de sélection en Afrique Tropicale : Expériences du Cameroun." Journal of the Cameroon Academy of Sciences 17, no. 1 (November 2, 2021): 19–41. http://dx.doi.org/10.4314/jcas.v17i1.2.

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Ce travail passe en revue les résultats des travaux de recherches sur l’amélioration génétique des zébus locaux (Goudali, White et Red Fulani) par voie de sélection et de croisement avec les races exotiques (Holstein, Montbéliarde, Brahman américain, Jersiais, Tarentaise, Limousine et Charolaise). Ces travaux ont été conduits depuis 1952 dans les Centres de Recherches Zootechniques de Wakwa et Bambui par l’Institut de Recherches Zootechniques (IRZ, aujourd’hui connue sous le nom de l’Institut de Recherche Agricole pour le Développement) du Cameroun, en système d’élevage semi-intensif dans les zones des hautes savanes guinéennes et des hauts plateaux de l’Ouest Cameroun. Les données proviennent des rapports d’activité et de consultations, des publications scientifiques et des entrevues. L’objectif est de ressortir les résultats saillants utiles pour les décideurs politiques, les chercheurs et les éleveurs. Les principaux résultats montrent que le potentiel laitier des croisés est généralement supérieur (>4 litres) à celui des races locales (>3,5 litres). Les génotypes ayant moins de 75% de sang exotique sont les plus performants en lait et viande. Les causes de mortalité par ordre d’importance sont nutritionnelles (36,8%), accidentelles (29,6%), pathologiques (21,1%), parasitaires (7,7%) et reproductives (4,8%). Par rapport aux autres produits de croisement, les croisés Holsteins ont été plus lourds à la naissance (34,75 kg) avec une vitesse de croissance plus élevée (0,46 kg), une production laitière plus élevée (5,47 ± 0,17 litres), plus précoces (40,87 mois) et moins susceptibles au stress Nuenvironnemental. Pour la filière bovin-à-viande, les performances de viande de la race synthétique Wakwa sont légèrement supérieures à celles des zébus Goudali sélectionnés dans le cheptel local. Cependant, le taux de susceptibilité à la dermatophilose est estimé à 71,0%, 34,4% et 5,0% chez le pur, ½ et ¼ sang Brahman, respectivement. La sélection massale du zébu Goudali (variété Ngaoundéré) et la race synthétique Wakwa a permis d’obtenir des progrès génétiques significatifs, soient, respectivement, 8,60 kg et 20,40 kg. Cependant, les corrélations génétiques négatives entre les effets directs et maternels montrent que le progrès génétique serait davantage plus important si la sélection était concentrée sur les performances directes et maternelles. Les expériences d’insémination artificielle menées à la SODEPA et à « Tadu Dairy Cooperative Society»montrent que l’industrie laitière est une entreprise rentable avec un rapport bénéfice-coût de 4,21. This work reviews the results of research on the genetic improvement of local zebus (Gudali, White and Red Fulani) through selection and crossbreeding with exotic cattle breeds (Holstein, Montbeliard, American Brahman, Jersey, Tarentaise, Limousine and Charolais). This work was started since 1952 by Institute of Animal Research (IRZ, now known as the Institute of Agricultural Research for Development) at the Wakwa and Bambui Research Centers, under semi-intensive management conditions in the high Guinea savannah areas of the Adamawa Plateau and the highlands of West Cameroon. Data were obtained from activity and consultation reports, scientific publications and interviews. The objective is to highlight salient findings that are useful for policy makers, researchers and livestock producers. The main results show that the milk potential of crosses is generally higher (>4 liters) than that of local breeds (>3.5 liters). Genotypes with less than 75% exotic blood had the best milk and meat production performance. The reported causes of mortality in order of importance are nutritional (36.8%), accidental (29.6%), pathological (21.1%), parasitic (7.7%) and reproductive (4.8%). Compared to the others crosses, Holstein crosses were found to be relatively heavier at birth (34.75 kg) with a higher growth rate (0.46 kg), higher daily milk production (5.47 ± 0.17 liters), earlier maturing (40.87 months), but more adapted to the breeding conditions and less susceptible to environmental stress. For the beef industry, the meat performance of the synthetic Wakwa breed is slightly better than that of the Gudali zebus selected from the local herd. However, the susceptibility rate to dermatophilosis was estimated at 71.0%, 34.4% and 5.0% in pure, ½ and ¼ blood Brahman, respectively. Individual selection of the Gudali zebu (Ngaoundéré subtype) and the synthetic Wakwa breed has resulted in significant genetic progress, 8.60 kg and 20.40 kg, respectively. However, the negative genetic correlations between direct and maternal effects showed that genetic progress would be more important if selection was concentrated on direct and maternal performance. Artificial insemination experiments conducted at SODEPA and Tadu Dairy Cooperative Society showed that the dairy industry is a profitable enterprise with a benefit-cost ratio of 4.21.

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12

Hines, Pamela J. "Early life stress in depression susceptibility." Science 356, no. 6343 (June 15, 2017): 1134.6–1135. http://dx.doi.org/10.1126/science.356.6343.1134-f.

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13

Fanjul, V. Rodriguez, I. Sanchez-Perez, and R. Perona Abellon. "MKP1 regulates susceptibility to genotoxic stress." European Journal of Cancer Supplements 6, no. 9 (July 2008): 19. http://dx.doi.org/10.1016/s1359-6349(08)71249-7.

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14

Gradinski-Vrbanac, B., Z. Stojevič, S. Milinkovič-Tur, T. Balenovič, J. Piršljin, and M. Zdeler-Tuk. "In vitro susceptibility of duck, chicken, and pig erythrocyte lipids to peroxidation." Veterinární Medicína 47, No. 10 - 11 (March 30, 2012): 303–8. http://dx.doi.org/10.17221/5839-vetmed.

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The susceptibility of erythrocyte lipid to in vitro peroxidation as measured by TBARS (thiobarbituric acid-reactive substances) and concentration of reduced glutathione (GSH) in whole blood and erythrocyte suspension before and after incubation with hydrogen peroxide was assessed in ducks, chickens and pigs. A high susceptibility of erythrocytes to peroxidation in vitro was observed in all animals tested, but this susceptibility was of different intensity. Pig erythrocytes exhibited the lowest resistance to oxidative stress in vitro as compared with that in ducks (P < 0.01) and chickens (P < 0.02). A high level of GSH in the erythrocytes of ducks and chickens offers higher resistance to oxidative stress in comparison with that in the pig erythrocytes.

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15

Solovey, L. B., and L. Y. Belenkova. "STUDY OF STRESS SUSCEPTIBILITY OF CONDITIONALLY HEALTHY STUDENTS AND STUDENTS WITH DISABILITIES." Educational Psychology in Polycultural Space 62, no. 2 (2023): 47–55. http://dx.doi.org/10.24888/2073-8439-2023-62-2-47-55.

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The article discusses the peculiarities of stress susceptibility of conditionally healthy young people and young people with disabilities. The relevance of the study is due to the fact that in the modern world, most people experience stress, and this negatively affects the life of people in general. Young people experience stress due to the fact that the social situation of the development of adolescence itself is a stressful factor. Disabled people and young people with disabilities may be more susceptible to stress, as they are in even more unfavorable conditions in contrast to conditionally healthy people. However, we assume that the stress susceptibility of persons with disabilities may not differ much from the stress susceptibility of conditionally healthy young people, since stress susceptibility may depend on various factors, in particular on the internal picture of the disease, on the relationship that a person develops with social reality. The study was conducted on the basis of the Moscow State University of Humanities and Economics, a university where many students with disabilities and disabilities study. The study was conducted during the pandemic. The following methods were used as a research tool: assessment of neuropsychic stress (author T.A. Nemchin), identification of the level of stress susceptibility (author E.A. Tarasov), PSM-25 psychological stress scale (author Lemur-Tessier Fillion). During the study, the hypothesis was confirmed, no significant differences in the stress susceptibility of students with disabilities and conditionally healthy students were found.

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16

S, Alam. "Impact of Nutrition on Covid-19 Susceptibility." Bioequivalence & Bioavailability International Journal 7, no. 1 (January 4, 2023): 1–3. http://dx.doi.org/10.23880/beba-16000184.

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COVID-19 pandemic era had shown a notable threat to humans. Initially, in Wuhan COVID-19 arise and promptly extent throughout China to world-wide. The ability effect of COVID-19 inversely the effect of nutrients at the epidemiology is worth of have a look at thinking about the positive and terrible institutions of nutrients with morbidity and mortality. A healthy nutritional diet is important for the recovery of COVID-19 patient such as pulmonary distress, cardiac distress, or critically ill. Excessive quantities of HFD, subtle CHO, sugars, little ranges of fiber, fat and also antioxidants. HFD intake inhibits B and T lymphocyte feature probably in the adaptive immune system through enhancing a oxidative stress. A systemic research analysis was conducted that took place between 26 August and 11 September 2021. About 88.1% were aware of the potential relation between nutritional status and immune functions. 90.6% of them believed that healthy eating is considered important during Covid-19 pandemic. Proper diet along with physical activity is necessary to keep yourself healthy during Covid-19 pandemic. 33.8% accepted the fact that their daily diet fulfills their basic nutritional requirements, while 27.5% of them denied. There are many evidence that healthy dietary intake will either prevent disease or speed up treatment.

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Haak, Jodie L., Garry R. Buettner, Douglas R. Spitz, and Kevin C. Kregel. "Aging augments mitochondrial susceptibility to heat stress." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 296, no. 3 (March 2009): R812—R820. http://dx.doi.org/10.1152/ajpregu.90708.2008.

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The pathophysiology of aging is accompanied by a decline in tolerance to environmental stress. While mitochondria are primary suspects in the etiology of aging, little is known about their ability to tolerate perturbations to homeostasis in older organisms. To investigate the role of mitochondria in the increased susceptibility to heat stress that accompanies aging, young and old Fischer 344 rats underwent a heat stress protocol known to elicit exaggerated cellular damage with aging. At either 2 or 24 h after heat stress, livers were removed from animals, and hepatic mitochondria were isolated. Electron microscopy revealed extensive morphological damage to mitochondria from young and, to a greater extent, old rats after heat stress. There was also a significant loss of cytochrome c from old, but not young, mitochondria and a persistent increase in 4-hydroxynonenal-modified proteins in old vs. young mitochondria exposed to heat stress. Electron paramagnetic resonance measurements of superoxide indicate greater superoxide production from mitochondria of old compared with young animals and suggest that mitochondrial integrity was altered during heat stress. The mitochondrial stress response, which functions to correct stress-induced damage to mitochondrial proteins, was also blunted in old rats. Delayed and reduced levels of heat shock protein 60 (Hsp60), the main inducible mitochondrial stress protein, were observed in old compared with young mitochondria after heat stress. Additionally, the amount of Hsp10 protein increased in young, but not old, rat liver mitochondria after hyperthermic challenge. Taken together, these data suggest that mitochondria in old animals are more vulnerable to incurring and less able to repair oxidative damage that occurs in response to a physiologically relevant heat stress.

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18

Onaka, Kentaro, Masami Mayuzumi, and Junichi Tani. "ICONE15-10500 Stress Corrosion Cracking Susceptibility of Solution Annealed Stainless Steels with Different Hot Working Directions in High Temperature Water." Proceedings of the International Conference on Nuclear Engineering (ICONE) 2007.15 (2007): _ICONE1510. http://dx.doi.org/10.1299/jsmeicone.2007.15._icone1510_267.

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19

Eisen, Aaron M., Gregory N. Bratman, and Hector A. Olvera-Alvarez. "Susceptibility to stress and nature exposure: Unveiling differential susceptibility to physical environments; a randomized controlled trial." PLOS ONE 19, no. 4 (April 17, 2024): e0301473. http://dx.doi.org/10.1371/journal.pone.0301473.

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Background Emerging epidemiological evidence indicates nature exposure could be associated with greater health benefits among groups in lower versus higher socioeconomic positions. One possible mechanism underpinning this evidence is described by our framework: (susceptibility) adults in low socioeconomic positions face higher exposure to persistent psychosocial stressors in early life, inducing a pro-inflammatory phenotype as a lifelong susceptibility to stress; (differential susceptibility) susceptible adults are more sensitive to the health risks of adverse (stress-promoting) environments, but also to the health benefits of protective (stress-buffering) environments. Objective Experimental investigation of a pro-inflammatory phenotype as a mechanism facilitating greater stress recovery from nature exposure. Methods We determined differences in stress recovery (via heart rate variability) caused by exposure to a nature or office virtual reality environment (10 min) after an acute stressor among 64 healthy college-age males with varying levels of susceptibility (socioeconomic status, early life stress, and a pro-inflammatory state [inflammatory reactivity and glucocorticoid resistance to an in vitro bacterial challenge]). Results Findings for inflammatory reactivity and glucocorticoid resistance were modest but consistently trended towards better recovery in the nature condition. Differences in recovery were not observed for socioeconomic status or early life stress. Discussion Among healthy college-age males, we observed expected trends according to their differential susceptibility when assessed as inflammatory reactivity and glucocorticoid resistance, suggesting these biological correlates of susceptibility could be more proximal indicators than self-reported assessments of socioeconomic status and early life stress. If future research in more diverse populations aligns with these trends, this could support an alternative conceptualization of susceptibility as increased environmental sensitivity, reflecting heightened responses to adverse, but also protective environments. With this knowledge, future investigators could examine how individual differences in environmental sensitivity could provide an opportunity for those who are the most susceptible to experience the greatest health benefits from nature exposure.

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Athayde, N. B., O. A. Dalla Costa, R. O. Roça, A. L. Guidoni, C. B. Ludtke, E. Oba, R. K. Takahira, and G. J. M. M. Lima. "Stress susceptibility in pigs supplemented with ractopamine1." Journal of Animal Science 91, no. 9 (September 1, 2013): 4180–87. http://dx.doi.org/10.2527/jas.2011-5014.

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21

Saul, Alison N., Tatiana M. Oberyszyn, Christine Daugherty, Donna Kusewitt, Susie Jones, Scott Jewell, William B. Malarkey, Amy Lehman, Stanley Lemeshow, and Firdaus S. Dhabhar. "Chronic Stress and Susceptibility to Skin Cancer." JNCI: Journal of the National Cancer Institute 97, no. 23 (December 7, 2005): 1760–67. http://dx.doi.org/10.1093/jnci/dji401.

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22

Sananbenesi, Farahnaz, and Andre Fischer. "Remodeling the susceptibility to stress-induced depression." Nature Medicine 21, no. 10 (October 2015): 1125–26. http://dx.doi.org/10.1038/nm.3970.

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Lockette, Warren, Neil Shepard, Andrew Lyos, Tom Boismier, and Ann Mers. "Altered Coriolis Stress Susceptibility in Essential Hypertension." American Journal of Hypertension 4, no. 8 (August 1991): 645–50. http://dx.doi.org/10.1093/ajh/4.8.645.

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Lloret, Aníbal Pérez. "Identifying Patients With Erythrocyte Oxidative Stress Susceptibility." American Journal of Clinical Pathology 100, no. 3 (September 1, 1993): 364.2–364. http://dx.doi.org/10.1093/ajcp/100.3.364a.

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Dhabhar, F. S. "Stress, anxiety, & susceptibility to skin cancer." Brain, Behavior, and Immunity 24 (August 2010): S68—S69. http://dx.doi.org/10.1016/j.bbi.2010.07.228.

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26

Friedman, A. "Jump-starting natural resilience reverses stress susceptibility." Science 346, no. 6209 (October 30, 2014): 555. http://dx.doi.org/10.1126/science.1260781.

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Murphy, E. A., J. M. Davis, M. D. Carmichael, J. D. Gangemi, A. Ghaffar, and E. P. Mayer. "Exercise stress increases susceptibility to influenza infection." Brain, Behavior, and Immunity 22, no. 8 (November 2008): 1152–55. http://dx.doi.org/10.1016/j.bbi.2008.06.004.

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Dai, Guohong, Xiangjun Xing, Yun Shen, and Xiaohua Deng. "Stress-controlled dynamic susceptibility in FeGa stripes." Journal of Applied Physics 123, no. 24 (June 28, 2018): 243902. http://dx.doi.org/10.1063/1.5030382.

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Ogihara, Tohru, Makoto Kitagawa, Masayuki Miki, Hiroshi Tamai, Hiroshi Yasuda, Ryozo Okamoto, and Makoto Mino. "Susceptibility of Neonatal Lipoproteins to Oxidative Stress." Pediatric Research 29, no. 1 (January 1991): 39–45. http://dx.doi.org/10.1203/00006450-199101000-00008.

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Elenkov, Ilia J., and George P. Chrousos. "Stress, cytokine patterns and susceptibility to disease." Best Practice & Research Clinical Endocrinology & Metabolism 13, no. 4 (December 1999): 583–95. http://dx.doi.org/10.1053/beem.1999.0045.

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Tian Rui, Zhang, Larosa Amanda, Di Raddo Marie-Eve, Wong Vanessa, Wong Alice Shui Ming, and Tak Pan Wong. "Hippocampal Negative Memory Engrams and Stress Susceptibility." Biological Psychiatry 87, no. 9 (May 2020): S31. http://dx.doi.org/10.1016/j.biopsych.2020.02.102.

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32

Papa Gobbi, Rodrigo, Gladis Magnarelli, and María Gabriela Rovedatti. "Susceptibility of placental mitochondria to oxidative stress." Birth Defects Research 110, no. 16 (September 19, 2018): 1228–32. http://dx.doi.org/10.1002/bdr2.1377.

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33

Goodwin, Frederick K. "Behavioral Stress Reactivity Related to Arthritis Susceptibility?" JAMA: The Journal of the American Medical Association 267, no. 7 (February 19, 1992): 910. http://dx.doi.org/10.1001/jama.1992.03480070026007.

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34

Ferreira, Jorge. "Role of mitochondrial fission in stress susceptibility." Lab Animal 53, no. 1 (January 2024): 8. http://dx.doi.org/10.1038/s41684-023-01311-0.

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35

Neufeld, RichardW J. "Sources of susceptibility to stress in psychopathology and stress management procedures." Biological Psychology 20, no. 3 (May 1985): 217–18. http://dx.doi.org/10.1016/0301-0511(85)90098-5.

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Chen, Lin, Huisheng Yang, Yanjing Su, and Lijie Qiao. "Stress corrosion cracking of A517 steel in marine environments." Anti-Corrosion Methods and Materials 68, no. 5 (September 9, 2021): 413–21. http://dx.doi.org/10.1108/acmm-07-2021-2505.

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Purpose The purpose of this study is to develop the existence and mechanism of stress corrosion cracking (SCC) for A517 steel in marine environments. Design/methodology/approach Slow strain rate test (SSRT) and constant load tests were used to investigate the SCC susceptibility of A517 steel. In addition, the additive stresses caused by the corrosion film and hydrogen entering into steel were applied to reveal the fundamental mechanism of the SCC. Findings The SCC susceptibility increased due to anodic dissolution and additive stress caused by the corrosion-produced film under anode polarization. Furthermore, the SCC susceptibility increased with increasing cathodic polarization, which is due to the increased additional stress caused by hydrogen entering into the steel. However, when the cathode polarization further increased, the additional stress remained due to the constant hydrogen content, thus the SCC susceptibility did not vary. Moreover, the SCC susceptibility of A517 steel under an alternate immersion environment (AIE) was lower than that under a full immersion environment and the steel under the AIE with 0.5 W/D had the lowest SCC susceptibility. Originality/value The stress corrosion behaviors of A517 in marine environments under various conditions were systematically analyzed.

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Desai, Iqbal M., HardikKamlesh Bhavsar, Sachin M. Darji, and JitendrakumarS Parmar. "Bacteriological analysis including antimicrobial susceptibility pattern of blood stream infections in tertiary care hospital." Annals of Pathology and Laboratory Medicine 4, no. 3 (May 28, 2017): A332—A337. http://dx.doi.org/10.21276/apalm.1203.

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Reshetnikov, Vasiliy V., and Natalia P. Bondar. "The Role of Stress-Induced Changes of Homer1 Expression in Stress Susceptibility." Biochemistry (Moscow) 86, no. 6 (June 2021): 613–26. http://dx.doi.org/10.1134/s0006297921060018.

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Abstract Stress negatively affects processes of synaptic plasticity and is a major risk factor of various psychopathologies such as depression and anxiety. HOMER1 is an important component of the postsynaptic density: constitutively expressed long isoforms HOMER1b and HOMER1c bind to group I metabotropic glutamate receptors MGLUR1 (GRM1) and MGLUR5 and to other effector proteins, thereby forming a postsynaptic protein scaffold. Activation of the GLUR1–HOMER1b,c and/or GLUR5–HOMER1b,c complex regulates activity of the NMDA and AMPA receptors and Ca2+ homeostasis, thus modulating various types of synaptic plasticity. Dominant negative transcript Homer1a is formed as a result of activity-induced alternative termination of transcription. Expression of this truncated isoform in response to neuronal activation impairs interactions of HOMER1b,c with adaptor proteins, triggers ligand-independent signal transduction through MGLUR1 and/or MGLUR5, leads to suppression of the AMPA- and NMDA-mediated signal transmission, and thereby launches remodeling of the postsynaptic protein scaffold and inhibits long-term potentiation. The studies on animal models confirm that the HOMER1a-dependent remodeling most likely plays an important part in the stress susceptibility, whereas HOMER1a itself can be regarded as a neuroprotector. In this review article, we consider the effects of different stressors in various animal models on HOMER1 expression as well as impact of different HOMER1 variants on human behavior as well as structural and functional characteristics of the brain.

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SHIOKAWA, Makoto, Shigero TOSHIMA, and Eiichi SATO. "Effect of stress and aging on stress corrosion susceptibility of 2017 alloy." Journal of Japan Institute of Light Metals 36, no. 7 (1986): 395–401. http://dx.doi.org/10.2464/jilm.36.395.

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UCHIDA, Hitoshi, Shozo INOUE, Hideshi TOKUHIRA, and Keiji KOTERAZAWA. "Susceptibility to Stress Corrosion Cracking of SUS304 Steel under Different Stress Conditions." Journal of the Society of Materials Science, Japan 43, no. 490 (1994): 874–80. http://dx.doi.org/10.2472/jsms.43.874.

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Zhang, Cheng, Yanjing Su, Lijie Qiao, and Wuyang Chu. "Study on the role of tarnishing film in stress-corrosion cracking of brass in Mattsson's solution." Journal of Materials Research 25, no. 5 (May 2010): 991–98. http://dx.doi.org/10.1557/jmr.2010.0120.

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In this paper, the influence of tarnishing film-induced stress and tarnishing film-induced brittle cracking on stress-corrosion cracking (SCC) of brass in Mattsson’s solution are investigated using hydrogen charging. Results showed that the SCC susceptibility of brass in Mattsson’s solution increased with the increase of tarnishing film-induced tensile stress. Also, the film-induced brittle cracking showed little effect on SCC susceptibility. From the results obtained, an improved SCC mechanism is proposed to explain the role of the tarnishing film-induced stress and the film-induced brittle cracking in SCC of brass in Mattsson’s solution. It seems that the film-induced brittle cracking is responsible for crack initiation of ductile brass. Also, the SCC susceptibility of brass in Mattsson’s solution was controlled by the growth rate of tarnishing film. Hydrogen enhanced the SCC susceptibility, which can be ascribed to the fact that hydrogen facilitates the tarnishing film growth.

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42

Wilkinson, Courtney S., Harrison L. Blount, Marek Schwendt, and Lori A. Knackstedt. "Brain Monoamine Dysfunction in Response to Predator Scent Stress Accompanies Stress-Susceptibility in Female Rats." Biomolecules 13, no. 7 (June 29, 2023): 1055. http://dx.doi.org/10.3390/biom13071055.

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Post-traumatic stress disorder (PTSD) is prevalent in women; however, preclinical research on PTSD has predominantly been conducted in male animals. Using a predator scent stress (PSS) rodent model of PTSD, we sought to determine if stress-susceptible female rats show altered monoamine concentrations in brain regions associated with PTSD: the medial prefrontal cortex (mPFC), nucleus accumbens (NAc), and dorsal (dHIPP) and ventral (vHIPP) hippocampus. Female Sprague–Dawley rats were exposed to a single, 10-min PSS exposure and tested for persistent anhedonia, fear, and anxiety-like behavior over four weeks. Rats were phenotyped as stress-susceptible based on sucrose consumption in the sucrose preference task and time spent in the open arms of the elevated plus maze. Brain tissue was collected, and norepinephrine, dopamine, serotonin, and their metabolites were quantified using high-performance liquid chromatography. Stress-susceptibility in female rats was associated with increased dopamine and serotonin turnover in the mPFC. Susceptibility was also associated with elevated dopamine turnover in the NAc and increased norepinephrine in the vHIPP. Our findings suggest that stress-susceptibility after a single stress exposure is associated with long-term effects on monoamine function in female rats. These data suggest interventions that decrease monoamine turnover, such as MAOIs, may be effective in the treatment of PTSD in women.

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Sun, Xiaoguang, Huaiyun Cui, Zhong Li, Renyang He, Zhiyong Liu, and Lin Lu. "Effect of Service Environmental Parameters on Electrochemical Corrosion Behavior of L80 Casing Steel." Materials 14, no. 19 (September 25, 2021): 5575. http://dx.doi.org/10.3390/ma14195575.

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The corrosion behavior of L80 casing steel was studied in a simulating annulus environment using the electrochemical measurement method, immersion test, and tensile test under a high-temperature and high-pressure H2S/CO2 environment. The partial pressure of CO2 (PCO2), the partial pressure of H2S (PH2S), water content, and preloading stress remarkably affected the corrosion behavior of L80 steel. The influence of PCO2 on stress corrosion cracking (SCC) susceptibility has an inflection point of approximately 1.1 MPa. The SCC susceptibility reaches the maximum when the PCO2 is about 1.1 MPa. The SCC susceptibility has a positive correlation to PH2S and water content. The higher water content of the corrosion medium can increase the electrical conductivity of the corrosion medium and promote the corrosion of L80 steel, which can improve the diffusion of hydrogen into steel and promote the SCC of L80 steel. Preloading stress can promote local corrosion, thereby promoting SCC of steel under stress. The dislocation emergence point caused by preloading stress can accelerate the diffusion of hydrogen into steel and increase SCC susceptibility.

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Martyniuk, І. А., and K. О. Mamchur. "Psychological determinations of stress susceptibility of future psychologists." Humanitarian studios: pedagogics, psychology, philosophy 10, no. 3 (October 23, 2019): 70–77. http://dx.doi.org/10.31548/hspedagog2019.03.070.

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Wood, Susan, Samantha Bouknight, Brittany Pope, and Evelynn Harrington. "Role of Neuroinflammation in Susceptibility to Social Stress." Biological Psychiatry 89, no. 9 (May 2021): S48. http://dx.doi.org/10.1016/j.biopsych.2021.02.136.

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Numa, Chisato, Hirotaka Nagai, Midori Nagai, Tomomi Yamashita, Yusuke Kawashima, Nobuhiko Ohno, Yosky Kataoka, Yuko Mimori-Kiyosue, Taro Kato, and Tomoyuki Furuyashiki. "Roles of synaptic mitochondrial regulations for stress susceptibility." Proceedings for Annual Meeting of The Japanese Pharmacological Society 95 (2022): 1—SS—13. http://dx.doi.org/10.1254/jpssuppl.95.0_1-ss-13.

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De Micheli, Lorenzo, Silvia Maria Leite Agostinho, Giordano Trabanelli, and Fabrizio Zucchi. "Susceptibility to Stress Corrosion Cracking of 254SMO SS." Materials Research 5, no. 1 (March 2002): 63–69. http://dx.doi.org/10.1590/s1516-14392002000100011.

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KONO, Akiko, and Hiroko KURIYAMA. "The relationship between miso preference and stress susceptibility." Japanese Journal of Sensory Evaluation 12, no. 1-2 (2008): 30–33. http://dx.doi.org/10.9763/jjsse.12.30.

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Belsky, Jay, and Michael Pluess. "Beyond diathesis stress: Differential susceptibility to environmental influences." Psychological Bulletin 135, no. 6 (2009): 885–908. http://dx.doi.org/10.1037/a0017376.

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Chattarji, Sumantra, and Rajnish P. Rao. "Blood–brain biomarkers for stress susceptibility: Fig. 1." Proceedings of the National Academy of Sciences 111, no. 37 (September 3, 2014): 13253–54. http://dx.doi.org/10.1073/pnas.1414663111.

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