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Relevant bibliographies by topics / Surveillance immune / Journal articles

Journal articles on the topic 'Surveillance immune'

To see the other types of publications on this topic, follow the link: Surveillance immune.

Author: Grafiati

Published: 25 May 2024

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1

Shastri, Nilabh, Chansu Park, and Jian Guan. "Immune surveillance of immune surveillance." Molecular Immunology 150 (October 2022): 2. http://dx.doi.org/10.1016/j.molimm.2022.05.018.

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2

Swann, Jeremy B., and Mark J. Smyth. "Immune surveillance of tumors." Journal of Clinical Investigation 117, no. 5 (May 1, 2007): 1137–46. http://dx.doi.org/10.1172/jci31405.

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3

Grossman, Zvi, and Ronald B. Herberman. "‘Immune surveillance’ without immunogenicity." Immunology Today 7, no. 5 (May 1986): 128–31. http://dx.doi.org/10.1016/0167-5699(86)90075-7.

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4

Prehn, Richmond T., and Liisa M. Prehn. "The flip side of immune surveillance: immune dependency." Immunological Reviews 222, no. 1 (April 2008): 341–56. http://dx.doi.org/10.1111/j.1600-065x.2008.00609.x.

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5

Kim, Ryungsa, Manabu Emi, and Kazuaki Tanabe. "Cancer immunoediting from immune surveillance to immune escape." Immunology 121, no. 1 (May 2007): 1–14. http://dx.doi.org/10.1111/j.1365-2567.2007.02587.x.

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6

Schaller, Julien, and Judith Agudo. "Metastatic Colonization: Escaping Immune Surveillance." Cancers 12, no. 11 (November 16, 2020): 3385. http://dx.doi.org/10.3390/cancers12113385.

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Cancer immunotherapy has shifted the paradigm in cancer therapy by revitalizing immune responses against tumor cells. Specifically, in primary tumors cancer cells evolve in an immunosuppressive microenvironment, which protects them from immune attack. However, during tumor progression, some cancer cells leave the protective tumor mass, disseminating and seeding secondary organs. These initial disseminated tumor cells (DTCs) should potentially be susceptible to recognition by the immune system in the new host tissues. Although Natural Killer or T cells eliminate some of these DTCs, a fraction escape anti-tumor immunity and survive, thus giving rise to metastatic colonization. How DTCs interact with immune cells and the underpinnings that regulate imperfect immune responses during tumor dissemination remain poorly understood. Uncovering such mechanisms of immune evasion may contribute to the development of immunotherapy specifically targeting DTCs. Here we review current knowledge about systemic and site-specific immune-cancer crosstalk in the early steps of metastasis formation. Moreover, we highlight how conventional cancer therapies can shape the pre-metastatic niche enabling immune escape of newly arrived DTCs.

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7

Lowe, Scott. "Immune Surveillance of Senescent Cells." Innovation in Aging 5, Supplement_1 (December 1, 2021): 246. http://dx.doi.org/10.1093/geroni/igab046.952.

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Abstract Cellular senescence involves a stable cell cycle arrest and a secretory program that modulates the tissue environment. In cancer, senescence acts as a potent barrier to tumorigenesis and, though many cancers evade senescence during the course of tumor evolution, ionizing radiation and conventional chemotherapy can, to varying degrees, induce senescence in tumor cells leading to potent anticancer effects. Conversely, the aberrant accumulation of senescent cells can reduce regenerative capacity and lead to tissue decline, contributing to tissue pathologies associated with age or the debilitating side-effects of cancer therapy. Our laboratory studies mechanisms of cellular senescence with the ultimate goal of developing strategies to modulate senescence for therapeutic benefit. We have focused on how senescent cells trigger immune surveillance to facilitate their own elimination or, when that fails, how synthetic immune cells (i.e. CAR T cells) can be directed to eliminate senescent cells. Recent advances in understanding senescent cell surveillance by the immune system will be discussed.

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8

Ahmad, Aamir. "Tumor microenvironment and immune surveillance." Microenvironment and Microecology Research 4, no. 1 (2022): 6. http://dx.doi.org/10.53388/mmr2022006.

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9

Oh, Julia, and Derya Unutmaz. "Immune cells for microbiota surveillance." Science 366, no. 6464 (October 24, 2019): 419–20. http://dx.doi.org/10.1126/science.aaz4014.

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10

Zanetti, M., and N. R. Mahadevan. "Immune Surveillance from Chromosomal Chaos?" Science 337, no. 6102 (September 27, 2012): 1616–17. http://dx.doi.org/10.1126/science.1228464.

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11

Alderton, Gemma. "Immune surveillance of the brain." Science 366, no. 6472 (December 19, 2019): 1467.18–1469. http://dx.doi.org/10.1126/science.366.6472.1467-r.

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12

Zinkernagel, Rolf, and Paul Klenerman. "Immune surveillance and AIDS progression." Current Biology 7, no. 7 (July 1997): R403—R405. http://dx.doi.org/10.1016/s0960-9822(06)00201-6.

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13

Nagarajan, Niranjana A., and Nilabh Shastri. "Immune surveillance for ERAAP dysfunction." Molecular Immunology 55, no. 2 (September 2013): 120–22. http://dx.doi.org/10.1016/j.molimm.2012.10.006.

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14

Schmid, Dorothee, and Christian Münz. "Immune Surveillance via Self Digestion." Autophagy 3, no. 2 (March 27, 2007): 133–35. http://dx.doi.org/10.4161/auto.3591.

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15

Branch, Pauline, David C. Bicknell, Andrew Rowan, Walter F. Bodmer, and Peter Karran. "Immune surveillance in colorectal carcinoma." Nature Genetics 9, no. 3 (March 1995): 231–32. http://dx.doi.org/10.1038/ng0395-231.

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16

Jenne, Craig N., and Paul Kubes. "Immune surveillance by the liver." Nature Immunology 14, no. 10 (September 18, 2013): 996–1006. http://dx.doi.org/10.1038/ni.2691.

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17

Zanetti, Maurizio. "Chromosomal chaos silences immune surveillance." Science 355, no. 6322 (January 19, 2017): 249–50. http://dx.doi.org/10.1126/science.aam5331.

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18

Ferrone, S. "Melanoma, immune surveillance, and immunotherapy." Journal of Clinical Investigation 93, no. 4 (April 1, 1994): 1351–52. http://dx.doi.org/10.1172/jci117108.

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19

Damhofer, Helene, and Kristian Helin. "EZH2i unlocks PDAC immune surveillance." Nature Cancer 4, no. 6 (June 27, 2023): 781–83. http://dx.doi.org/10.1038/s43018-023-00562-7.

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20

Caruso, Roberta, and Grace Y. Chen. "High fat stems tumor immune surveillance." Cell Reports Medicine 2, no. 12 (December 2021): 100483. http://dx.doi.org/10.1016/j.xcrm.2021.100483.

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21

Oluwadara, Oluwadayo O., Andre Barkhordarian, Luca Giacomelli, Xenia Brant, and Francesco Chiappelli. "Immune surveillance of nasopharyngeal carcinoma (NpC)." Bioinformation 7, no. 5 (October 31, 2011): 271–75. http://dx.doi.org/10.6026/97320630007271.

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22

Jobim, Mariana, and Luiz F. J. Jobim. "Natural killer cells and immune surveillance." Jornal de Pediatria 84, no. 7 (September 29, 2008): 58–67. http://dx.doi.org/10.2223/jped.1780.

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23

Monzavi-Karbassi, Behjatolah, Anastas Pashov, and Thomas Kieber-Emmons. "Tumor-Associated Glycans and Immune Surveillance." Vaccines 1, no. 2 (June 17, 2013): 174–203. http://dx.doi.org/10.3390/vaccines1020174.

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24

Shin, Hwain, Manuela Mally, Marina Kuhn, Cecile Paroz, and Guy R. Cornelis. "Escape from Immune Surveillance byCapnocytophaga canimorsus." Journal of Infectious Diseases 195, no. 3 (February 2007): 375–86. http://dx.doi.org/10.1086/510243.

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25

Gruenbacher, Georg, and Martin Thurnher. "Mevalonate metabolism governs cancer immune surveillance." OncoImmunology 6, no. 10 (July 27, 2017): e1342917. http://dx.doi.org/10.1080/2162402x.2017.1342917.

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26

Eiselein, M. W. Biggs and J. E. "Suppression of immune surveillance in melanoma." Medical Hypotheses 56, no. 6 (June 2001): 648–52. http://dx.doi.org/10.1054/mehy.2000.1211.

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27

Smyth, Mark J., Ilia Voskoboinik, and Joseph A. Trapani. "Immune surveillance of lymphoma in humans?" Blood 105, no. 11 (June 1, 2005): 4159–60. http://dx.doi.org/10.1182/blood-2005-03-1019.

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28

den Hartog, Gerco, Rob van Binnendijk, Anne-Marie Buisman, Guy A. M. Berbers, and Fiona R. M. van der Klis. "Immune surveillance for vaccine-preventable diseases." Expert Review of Vaccines 19, no. 4 (March 29, 2020): 327–39. http://dx.doi.org/10.1080/14760584.2020.1745071.

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29

Agranovich, Alexandra, Tal Vider-Shalit, and Yoram Louzoun. "Optimal viral immune surveillance evasion strategies." Theoretical Population Biology 80, no. 4 (December 2011): 233–43. http://dx.doi.org/10.1016/j.tpb.2011.08.005.

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30

Pawelec, Graham. "MHC-Unrestricted Immune Surveillance of Leukemia1." Cancer Biotherapy 9, no. 3 (January 1994): 265–88. http://dx.doi.org/10.1089/cbr.1994.9.265.

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31

Starck, S. R., S. Cardinaud, and N. Shastri. "Immune surveillance obstructed by viral mRNA." Proceedings of the National Academy of Sciences 105, no. 27 (July 1, 2008): 9135–36. http://dx.doi.org/10.1073/pnas.0804456105.

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32

Mahasa, Khaphetsi Joseph, Rachid Ouifki, Amina Eladdadi, and Lisette de Pillis. "Mathematical model of tumor–immune surveillance." Journal of Theoretical Biology 404 (September 2016): 312–30. http://dx.doi.org/10.1016/j.jtbi.2016.06.012.

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33

Newcombe, D. S. "Immune surveillance, organophosphorus exposure, and lymphomagenesis." Lancet 339, no. 8792 (February 1992): 539–41. http://dx.doi.org/10.1016/0140-6736(92)90349-8.

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34

Chow, Melvyn T., Andreas Möller, and Mark J. Smyth. "Inflammation and immune surveillance in cancer." Seminars in Cancer Biology 22, no. 1 (February 2012): 23–32. http://dx.doi.org/10.1016/j.semcancer.2011.12.004.

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35

Noble, Kim A. "Rheumatoid Arthritis: Immune Surveillance Gone Wild?" Journal of PeriAnesthesia Nursing 26, no. 2 (April 2011): 110–15. http://dx.doi.org/10.1016/j.jopan.2011.01.008.

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36

Kandalaft, Lana E., Gregory T. Motz, Jaikumar Duraiswamy, and George Coukos. "Tumor immune surveillance and ovarian cancer." Cancer and Metastasis Reviews 30, no. 1 (February 8, 2011): 141–51. http://dx.doi.org/10.1007/s10555-011-9289-9.

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37

Boyd, Ashleigh S., and Neil P. Rodrigues. "Stem Cells Cycle toward Immune Surveillance." Immunity 48, no. 2 (February 2018): 187–90. http://dx.doi.org/10.1016/j.immuni.2018.02.006.

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38

Young, Richard A., and Timothy J. Elliott. "Stress proteins, infection, and immune surveillance." Cell 59, no. 1 (October 1989): 5–8. http://dx.doi.org/10.1016/0092-8674(89)90861-1.

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39

Zimmermann, Valerie S., Fabio Benigni, and Anna Mondino. "Immune surveillance and anti-tumor immune responses: an anatomical perspective." Immunology Letters 98, no. 1 (April 2005): 1–8. http://dx.doi.org/10.1016/j.imlet.2004.09.005.

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40

Garcia-Lora, Angel, Ignacio Algarra, and Federico Garrido. "MHC class I antigens, immune surveillance, and tumor immune escape." Journal of Cellular Physiology 195, no. 3 (2003): 346–55. http://dx.doi.org/10.1002/jcp.10290.

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41

Töpfer, Katrin, Stefanie Kempe, Nadja Müller, Marc Schmitz, Michael Bachmann, Marc Cartellieri, Gabriele Schackert, and Achim Temme. "Tumor Evasion from T Cell Surveillance." Journal of Biomedicine and Biotechnology 2011 (2011): 1–19. http://dx.doi.org/10.1155/2011/918471.

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An intact immune system is essential to prevent the development and progression of neoplastic cells in a process termed immune surveillance. During this process the innate and the adaptive immune systems closely cooperate and especially T cells play an important role to detect and eliminate tumor cells. Due to the mechanism of central tolerance the frequency of T cells displaying appropriate arranged tumor-peptide-specific-T-cell receptors is very low and their activation by professional antigen-presenting cells, such as dendritic cells, is frequently hampered by insufficient costimulation resulting in peripheral tolerance. In addition, inhibitory immune circuits can impair an efficient antitumoral response of reactive T cells. It also has been demonstrated that large tumor burden can promote a state of immunosuppression that in turn can facilitate neoplastic progression. Moreover, tumor cells, which mostly are genetically instable, can gain rescue mechanisms which further impair immune surveillance by T cells. Herein, we summarize the data on how tumor cells evade T-cell immune surveillance with the focus on solid tumors and describe approaches to improve anticancer capacity of T cells.

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42

Guan, Jian, Josiah David Peske, Joshua A. Taylor, and Nilabh Shastri. "The nonclassical immune surveillance for ERAAP function." Current Opinion in Immunology 70 (June 2021): 105–11. http://dx.doi.org/10.1016/j.coi.2021.05.008.

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43

Ousman, Shalina S., and Paul Kubes. "Immune surveillance in the central nervous system." Nature Neuroscience 15, no. 8 (July 26, 2012): 1096–101. http://dx.doi.org/10.1038/nn.3161.

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44

Prolo, P., F. Chiappelli, G. Bernard, M. Fiala, A. Ibarra, M. L. Sartori, A. Dovio, and A. Angeli. "Neuroendocrine-Immune Surveillance of Osteosarcoma: Emerging Hypothesis." Journal of Dental Research 82, no. 6 (June 2003): 417–21. http://dx.doi.org/10.1177/154405910308200603.

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45

Moser, B. "Chemokines: role in inflammation and immune surveillance." Annals of the Rheumatic Diseases 63, suppl_2 (November 1, 2004): ii84—ii89. http://dx.doi.org/10.1136/ard.2004.028316.

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46

Schmid, D., and C. Münz. "Immune surveillance of intracellular pathogens via autophagy." Cell Death & Differentiation 12, S2 (October 25, 2005): 1519–27. http://dx.doi.org/10.1038/sj.cdd.4401727.

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47

Kim, Tae Hyun, and Sang Geon Kim. "Role of CXCR6 in Antitumor Immune Surveillance." Gastroenterology 156, no. 6 (May 2019): 1565–68. http://dx.doi.org/10.1053/j.gastro.2019.03.032.

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48

Di Francesco, P., F. Pica, L. Belogi, C. Croce, C. Favalli, E. Tubaro, and E. Garaci. "Influence of cocaine on murine immune surveillance." Pharmacological Research 22 (September 1990): 165. http://dx.doi.org/10.1016/s1043-6618(09)80208-9.

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49

Feinberg, Mark B., and Angela R. McLean. "AIDS: Decline and fall of immune surveillance?" Current Biology 7, no. 3 (March 1997): R136—R140. http://dx.doi.org/10.1016/s0960-9822(97)70072-1.

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50

Wekerle, Hartmut. "Immune reactivity and surveillance in the CNS." Journal of Neuroimmunology 38, no. 1-2 (May 1992): 179. http://dx.doi.org/10.1016/0165-5728(92)90123-3.

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