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Dissertations / Theses on the topic 'SurfactantProteins'

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Published: 23 September 2022
Last updated: 28 January 2023

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1

Feilcke, Maria Kroumova. "Surfactantprotein A." Diss., Ludwig-Maximilians-Universität München, 2012. http://nbn-resolving.de/urn:nbn:de:bvb:19-168084.

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2

Paschen, Christian. "Surfactantproteine bei Pulmonaler Alveolarproteinose." Diss., lmu, 2004. http://nbn-resolving.de/urn:nbn:de:bvb:19-30154.

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3

Felber, Joerg. "Keimbesiedelung, Inflammationsreaktion und Surfactantproteine in der Lunge tracheotomierter Kinder." Diss., lmu, 2003. http://nbn-resolving.de/urn:nbn:de:bvb:19-9990.

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4

Kirchberger, Valerie Sara. "Surfactantproteine und ihre Vorstufen bei interstitiellen Lungenerkrankungen im Kindesalter." Diss., lmu, 2009. http://nbn-resolving.de/urn:nbn:de:bvb:19-120317.

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5

Birzele, Jan. "Charakterisierung der Surfactantproteine bei Neugeborenen, Kindern und Erwachsenen mit Alveolarproteinosen." [S.l.] : [s.n.], 2001. http://deposit.ddb.de/cgi-bin/dokserv?idn=964192748.

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6

Tafel, Tim Oliver. "Surfactantproteine B und C bei Patienten mit Cystischer Fibrose und anderen Lungenerkrankungen." Diss., lmu, 2008. http://nbn-resolving.de/urn:nbn:de:bvb:19-81840.

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7

Feilcke, Maria Kroumova [Verfasser], and Matthias [Akademischer Betreuer] Griese. "Surfactantprotein A : Oligomere bei cystischer Fibrose / Maria Kroumova Feilcke. Betreuer: Matthias Griese." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2012. http://d-nb.info/1049393236/34.

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8

Koch, Miriam Isabell [Verfasser]. "Gestörte Prozessierung der hydrophoben Surfactantproteine bei der Idiopathischen Pulmonalen Fibrose / Miriam Isabell Koch." Gießen : Universitätsbibliothek, 2013. http://d-nb.info/1065320574/34.

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9

Brack, Eva. "Einfluss von Surfactantprotein A und D auf die ECP-Degranulation von humanen eosinophilen Granulozyten." Diss., Ludwig-Maximilians-Universität München, 2013. http://nbn-resolving.de/urn:nbn:de:bvb:19-160799.

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Die Rolle, die die hydrophoben Surfactantproteine SP-A und SP-D welche in den oberflächlichen Atemwegsflüssigkeiten zu finden sind spielen lässt sich wie folgt zusammenfassen. Zum einen, eine wichtige immunologische Funktion, indem sie zur Abwehr von Erregern beitragen. Zum anderen, bei der Regulation allergischer Reaktionen, bei welcher der eosinophile Granulozyt mit den von ihm freigesetzten Entzündungsmediatoren als wichtigste Effektorzelle zu nennen ist. Ziel dieser Arbeit war es den Einfluss der Surfactantproteine A und D auf eosinophile Granulozyten in Bezug auf ihre ECP-Degranulation zu untersuchen. In verschiedenen Versuchsreihen wurde die Freisetzung von Eosinophilem kationischem Protein (ECP) ohne (native Zellen) und nach Stimulation der Zellen mittels Fluoreszenzassay gemessen. Als Stimulanzien kamen Ca-Ionophore A23187 als eine potenter, wenn auch unphysiologischer Zellstimulus und die Immunglobuline A und G, sowie das Serum eines Allergikers als physiologische Stimuli zum Einsatz. Die Ergebnisse zeigten, dass durch die Surfactantproteine selbst keine Aktivierung der ECP-Freisetzung aus nativen eosinophilen Zellen ausgelöst werden kann. Die Degranulation stimulierter Zellen hingegen kann durch natürliches SP-A, SP-D und rekombinantes SP-D reduziert werden. Die wirksamen SP-D Konzentrationen lagen zwischen 0,05 und 5000ng/ml, wenn die Zellen mit Ca-Ionophore stimuliert wurden. Wurden IgA und IgG als Stimuli verwendet, konnte die ECP-Degranulation durch SP-A, SP-D und rekombinantes SP-D ebenfalls gehemmt werden, zeigte sich jedoch in keiner Konzentration signifikant. Bei der Stimulation mit Allergikerserum zeigten sowohl SP-A, SP-D als auch rekombinantes SP-D eine hemmende Wirkung. Zusammenfassend zeigen unsere In-vitro-Ergebnisse eine inhibitorische Wirkung von SP-A und D auf die ECP-Degranulation aus stimulierten eosinophilen Granulozyten. Dies ist mit der Hypothese einer protektiven Wirkung dieser Surfactantproteine bei Erkrankung aus dem allergischen Formenkreis vereinbar.
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10

Zemlin, Maren-Verena. "Die Bedeutung von Surfactantprotein A für die Pathogenese und den klinischen Verlauf der Pneumokokkenpneumonie /." Berlin : Mbv, 2008. http://d-nb.info/989978117/04.

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11

Steinecker, Manuela. "Die Surfactantproteine SP-A und SP-D in der Lunge von Kindern mit chronischem Husten." Diss., lmu, 2004. http://nbn-resolving.de/urn:nbn:de:bvb:19-28819.

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12

Bielecki, Eva. "Surfactantproteine A und D im Serum und in Bronchoalveolärer Lavage bei Patienten mit Cystischer Fibrose." Diss., lmu, 2007. http://nbn-resolving.de/urn:nbn:de:bvb:19-69381.

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13

Perino, Julien. "Implication de facteurs lipidiques (DPPG, sulfatide) et protéique (SP-D) dans un modèle d’infection respiratoire par les poxvirus." Grenoble, 2010. http://www.theses.fr/2010GRENV047.

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Après l'éradication de la variole déclarée par l'OMS en 1980 grâce à la vaccination, la réémergence de la variole dans un contexte de menace bioterroriste est possible, ainsi une meilleure compréhension des phénomènes d'infection par les orthopoxvirus est nécessaire. Ainsi ce travail de thèse s'inscrit dans le cadre d'un programme de « développement de contre-mesures médicales contre la variole », par l'étude des mécanismes d'infection et d'immunité. La transmission du virus de la variole ayant lieu par voie respiratoire, il est primordial d'étudier les mécanismes d'entrée du virus dans les poumons et les facteurs protéiques et lipidique impliqués lors de ce mécanisme d'entrée. La fonction d'immunité innée, portée par certains phospholipides présents dans le surfactant pulmonaire, a d'abord été étudiée. La découverte de cette fonction antivirale in vitro pour le DiPalmitoyl PhosphatidylGlycerol (DPPG) a conduit à l'évaluation de son activité in vivo dans un modèle d'infection pulmonaire chez la souris, ce lipide entraînant une protection importante dans un modèle létal. Par ailleurs, l'étude de l'interaction entre le virus de la vaccine et certains lipides présents dans les membranes cellulaires a permis de mettre en évidence un récepteur membranaire secondaire potentiel du virus de la vaccine assurant des fonctions similaires à celles des glycosaminoglycanes : le sulfatide. Enfin, l'étude des protéines de l'immunité innée, spécifiques du surfactant pulmonaire, a permis de mettre à jour l'existence d'une forte interaction entre la protéine SP-D et le virus de la vaccine. Cette interaction entraîne une inhibition de l'infection. Résultat qui reste à être confirmé lors de l'utilisation d'un modèle murin recombinant, KO pour cette protéine. Ces travaux démontrent l'intérêt à consacrer aux facteurs protéiques et lipidiques du surfactant et des membranes cellulaires et suggèrent la possibilité de l'utilisation de ces molécules, modifiées ou non, dans le développement et l'amélioration d'outils thérapeutiques et/ou prophylactiques pour traiter les infections par les poxvirus
Variola virus was declared eradicated in 1980 after a worldwide vaccination campaign. A better understanding of the infection process of orthopoxviruses is nevertheless necessary because of the potential release of variola by bioterrorists. Here we report potential counter-measures against Variola virus that could result from studying mechanisms of viral entry and immunity against Variola virus. The purpose of this work was to study multiple factors in vaccinia virus entry in the lung and thus gain a better understanding of the infectious process that could be used to stop infection by Orthopoxvirus. The innate immune functions displayed by some phospholipids (DiPalmitoyl PhosphatidylGlycerol) in lung surfactant were studied. The discovery of the ability of DPPG to inhibit vaccinia virus infection in cell culture led to the evaluation of its in vivo activity during a lethal vaccinia virus infection. Furthermore, the analysis of the interaction between vaccinia virus and plasma membrane lipids (sulfatide) enabled the definition of a secondary receptor for vaccinia virus in addition to glycosaminoglycans that were characterized previously. Finally, examination of the specific innate immunity provided by proteins in lung surfactant allowed us to highlight interactions between one surfactant protein (Surfactant protein D) and vaccinia virus. These interactions were then characterized as inhibitory interactions for vaccinia virus infection. Our findings underline the importance of lipids and proteins inlung surfactant as well as lipids in the plasma membrane in the Poxvirus infection and suggest that these molecules may be potential new targets for the development of new therapeutic and prophylactic products to efficiently treat poxvirus infection
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14

Steckenbiller, Josef. "Plasmakonzentrationen der Kollektine Mannose-bindendes Lektin (MBL) und Surfactantprotein D (SP-D) bei Patienten mit chronischer Parodontitis." Diss., lmu, 2009. http://nbn-resolving.de/urn:nbn:de:bvb:19-103141.

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15

Zemlin, Maren-Verena [Verfasser]. "Die Bedeutung von Surfactantprotein A für die Pathogenese und den klinischen Verlauf der Pneumokokkenpneumonie / Maren-Verena Zemlin." Berlin : Freie Universität Berlin, 2008. http://d-nb.info/1023049961/34.

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16

Brack, Eva [Verfasser], and Matthias [Akademischer Betreuer] Griese. "Einfluss von Surfactantprotein A und D auf die ECP-Degranulation von humanen eosinophilen Granulozyten / Eva Brack. Betreuer: Matthias Griese." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2013. http://d-nb.info/104214737X/34.

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17

Kindler, Christian [Verfasser], Friedrich [Akademischer Betreuer] Paulsen, and Matthias [Akademischer Betreuer] Ochs. "Nachweis und Funktion der Surfactantproteine A und D im Tränenapparat, der Augenoberfläche und der Tränenflüssigkeit des Menschen / Christian Kindler. Betreuer: Friedrich Paulsen ; Matthias Ochs." Halle, Saale : Universitäts- und Landesbibliothek Sachsen-Anhalt, 2009. http://d-nb.info/1024895866/34.

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18

Diogo, Isabell [Verfasser], and Bernd [Akademischer Betreuer] Müller. "Die Bedeutung der Surfactantproteine SP-A und SP-D bei der Modulation des Entzündungsprozesses im Rahmen des chronischen Asthma bronchiale / Isabell Diogo. Betreuer: Bernd Müller." Marburg : Philipps-Universität Marburg, 2011. http://d-nb.info/1013288548/34.

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19

Lorenz, Elke [Verfasser], and Matthias [Akademischer Betreuer] Griese. "Die hydrophoben Surfactantproteine B und C: biochemische Analyse in bronchoalveolärer Lavage und genetische Analyse bei Neugeborenen und Kindern mit chronischer pulmonaler Symptomatik / Elke Lorenz ; Betreuer: Matthias Griese." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2018. http://d-nb.info/1158496125/34.

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20

Kamalanathan, Ishara Dedunu. "Foam fractionation of surfactant-protein mixtures." Thesis, University of Manchester, 2015. https://www.research.manchester.ac.uk/portal/en/theses/foam-fractionation-of-surfactantprotein-mixtures(a6484b1a-d796-45ff-bc5c-420ef9130363).html.

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Foam fractionation is an adsorptive bubble separation technology that has shown potential as a replacement to the more costly and non-sustainable traditional downstream processing methods such as solvent extraction and chromatography for biological systems. However biological systems mostly tend to be a mixture of surface active species that complicates the foam fractionation separation. In this thesis a detailed experimental study on the application of foam fractionation to separate a well-defined surfactant-protein mixture was performed with emphasis on the competitive adsorption behaviour and transport processes of surfactant-protein mixtures in a foam fractionation process. Surface tension and nuclear magnetic resonance (NMR) measurements showed that nonionic surfactant Triton X−100 maximum surface pressure, surface affinity and diffusivity were a factor of 2.05, 67.0 and 19.6 respectively greater than that of BSA. Thus Triton X−100 dominated the surface adsorption at an air-water surface by diffusing to the surface and adsorbing at the surface faster than BSA. This competitive adsorption behaviour was observed in foam fractionation experiments performed for Triton X−100/BSA mixtures for different feed concentration ratios and air flow rates. The recovery and enrichment of Triton X−100 were found to increase and decrease respectively with increasing air flow rate for all foam fractionation experiments as expected for a single component system. However the recovery and enrichment of BSA were both found to increase with increasing air flow rate for high feed concentrations of Triton X−100.Bubble size measurements of the foamate produced from foam fractionation experiments showed that at steady state conditions the bubbles rising from the liquid pool were stabilised by BSA. However at the top of the column the recovery of Triton X−100 in the foamate (75% to 100%) was always greater than the recovery of BSA (13% to 76%) for all foam fractionation experiments. In addition, for high feed concentrations of both components and at low air flow rates, the enrichment of BSA remained at almost unity for most experiments and only increased when the recovery of Triton X−100 reached 100%. Thus it was concluded that Triton X-100 displaced the adsorbed BSA from the surface. The foam drainage properties of Triton X−100/BSA mixtures were characterised using two methods; forced foam drainage and from pressure profiles of steady state foam fractionation experiments (pressure method). The drainage data from the forced foam drainage was found not to be compatible with experimental foam fractionation results, by indicating that stable foam was not produced during the foam fractionation experiments. However stable foam was repeatedly produced during foam fractionation experiments. The drainage data from the pressure method was found to be in close agreement to experimental foam fractionation experiments. The work in this thesis takes a significant step beyond the literature experimental foam fractionation studies for multicomponent systems. In addition to investigating the effect of foam fractionation process parameters on the separation of mixed systems, the results from the characterisation studies of surface adsorption and foam properties provided insight and deeper understanding of the competitive adsorption behaviour of surfactants and proteins in a foam fractionation process.
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21

Vitasari, Denny. "Adsorption and transport of surfactant/protein onto a foam lamella within a foam fractionation column with reflux." Thesis, University of Manchester, 2014. https://www.research.manchester.ac.uk/portal/en/theses/adsorption-and-transport-of-surfactantprotein-onto-a-foam-lamella-within-a-foam-fractionation-column-with-reflux(8258925c-455b-4df4-8aa7-d8cff0e3b4e0).html.

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Foam fractionation is an economical and environmentally friendly separation method for surface active material using a rising column of foam. The system of foam fractionation column with reflux is selected since such a system can improve the enrichment of the product collected from the top of the column. Due to the reflux, it is assumed that there is more surface active material (surfactant and/or protein) in the Plateau border than that in the foam lamella, so that the Plateau border acts as a surfactant/protein reservoir. The aim of this thesis is to investigate the adsorption and transport of surface active material such as surfactant and/or protein onto the surface of a lamella in a foam fractionation column with reflux using mathematical simulation. There are two steps involved in adsorption of surface active material onto a bubble surface within foam, which are diffusion from the bulk solution into the subsurface, a layer next to the interface, followed by adsorption of that material from the subsurface onto the interface. The diffusion follows the Fick's second law, while the adsorption may follow the Henry, Langmuir or Frumkin isotherms, depending on the properties of the surface active material. The adsorption of mixed protein-surfactant follows the Frumkin isotherm. When there is a competition between protein and surfactant, the protein arrives onto the interface at a later time due to a slower diffusion rate and it displaces the surfactant molecules already on the surface since protein has a higher affinity for that surface than surfactant. The surfactant transport from a Plateau border onto a foam lamella is determined by the interaction of forces applied on the lamella surface, such as film drainage, due to the pressure gradient between the lamella and the Plateau border, the Marangoni effect, due to the gradient of surface tension, and surface viscosity, as a reaction to surface motion. In this thesis, there are two different models of film drainage. One approach uses assumption of a film with a mobile interface and the other model assumes a film with a rigid interface. In the absence of surface viscosity, the Marangoni effect dominates the film drainage resulting in accumulation of surfactant on the surface of the foam lamella in the case of a lamella with a rigid interface. In the case of a film with a mobile interface, the film drainage dominates the Marangoni effect and surfactant is washed away from the surface of the lamella. When the drainage is very fast, such as that which is achieved by a film with a mobile interface, the film could be predicted to attain the thickness of a common black film, well within the residence time in a foam fractionation column, at which point the film stops draining and surfactant starts to accumulate on the lamella surface. The desirable condition in operation of a foam fractionation column however is when the Marangoni effect dominates the film drainage and surfactant accumulates on the surface of a foam lamella such as the one achieved by a film with a rigid interface. In the presence of surface viscosity and the absence of film drainage, the surface viscous forces oppose the Marangoni effect and reduce the amount of surfactant transport onto the foam lamella. A larger surface viscosity results in less surfactant transport onto the foam lamella. In addition, the characteristic time scale required for surfactant transport is shorter with a shorter film length.
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22

Paschen, Christian Peter [Verfasser]. "Surfactantproteine bei pulmonaler Alveolarproteinose / vorgelegt von Christian Peter Paschen." 2004. http://d-nb.info/973328568/34.

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23

Felber, Jörg [Verfasser]. "Keimbesiedelung, Inflammationsreaktion und Surfactantproteine in der Lunge tracheotomierter Kinder / vorgelegt von Joerg Felber." 2003. http://d-nb.info/967819261/34.

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24

Birzele, Jan [Verfasser]. "Charakterisierung der Surfactantproteine bei Neugeborenen, Kindern und Erwachsenen mit Alveolarproteinosen / vorgelegt von Jan Birzele." 2001. http://d-nb.info/964192748/34.

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25

Kirchberger, Valerie Sara [Verfasser]. "Surfactantproteine und ihre Vorstufen bei interstitiellen Lungenerkrankungen im Kindesalter / vorgelegt von Valerie Sara Kirchberger." 2009. http://d-nb.info/1008445339/34.

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26

Tutdibi, Erol [Verfasser]. "Transiente Tachypnoe des Neugeborenen : sind Polymorphismen des Surfactantprotein B auslösend? / vorgelegt von Erol Tutdibi." 2005. http://d-nb.info/978038460/34.

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27

Bagheri, Ariane [Verfasser]. "Die Surfactantkonversion als enzymatischer Prozeß : ist das Surfactantprotein SP-B ein Substrat der Konvertase? / vorgelegt von Ariane Bagheri." 2006. http://d-nb.info/980127971/34.

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28

Bielecki, Eva [Verfasser]. "Surfactantproteine A und D im Serum und in bronchoalveolärer Lavage bei Patienten mit cystischer Fibrose / vorgelegt von Eva Bielecki." 2007. http://d-nb.info/984673520/34.

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29

Steinecker, Manuela [Verfasser]. "Die Surfactantproteine SP-A und SP-D in der Lunge von Kindern mit chronischem Husten / vorgelegt von Manuela Steinecker." 2004. http://d-nb.info/973136936/34.

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30

Tafel, Tim Oliver [Verfasser]. "Surfactantproteine B und C sowie deren Vorstufen bei Patienten mit cystischer Fibrose und anderen Lungenerkrankungen / vorgelegt von Tim Oliver Tafel." 2008. http://d-nb.info/988499487/34.

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31

Steckenbiller, Josef Johann Martin [Verfasser]. "Plasmakonzentrationen der Kollektine-Mannose-bindendes Lektin (MBL) und Surfactantprotein D (SP-D) bei Patienten mit chronischer Parodontitis / vorgelegt von Josef Johann Martin Steckenbiller." 2009. http://d-nb.info/995668868/34.

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