Relevant bibliographies by topics / Supramaximal exercise and training

Academic literature on the topic 'Supramaximal exercise and training'

Author: Grafiati
Published: 4 June 2021
Last updated: 20 February 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Contents

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Supramaximal exercise and training.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Supramaximal exercise and training"

1

Green, H. J., R. L. Hughson, J. A. Thomson, and M. T. Sharratt. "Supramaximal exercise after training-induced hypervolemia. I. Gas exchange and acid-base balance." Journal of Applied Physiology 62, no. 5 (May 1, 1987): 1944–53. http://dx.doi.org/10.1152/jappl.1987.62.5.1944.

Full text
Abstract:
The effect of an exercise-induced reduction in blood O2-carrying capacity on ventilatory gas exchange and acid-base balance during supramaximal exercise was studied in six males [peak O2 consumption (VO2peak), 3.98 +/- 0.49 l/min]. Three consecutive days of supramaximal exercise resulted in a preexercise reduction of hemoglobin concentration from 15.8 to 14.0 g/dl (P less than 0.05). During exercise (120% VO2peak) performed intermittently (1 min work to 4 min rest); a small but significant (P less than 0.05) increase was found for both O2 consumption (VO2) (l X min) and heart rate (beats/min) on day 2 of the training. On day 3, VO2 (l/min) was reduced 3.2% (P less than 0.05) over day 1 values. No changes were found in CO2 output and minute ventilation during exercise between training days. Similarly, short-term training failed to significantly alter the changes in arterialized blood PCO2, pH, and [HCO-3] observed during exercise. It is concluded that hypervolemia-induced reductions in O2-carrying capacity in the order of 10–11% cause minimal impairment to gas exchange and acid-base balance during supramaximal non-steady-state exercise.
APA, Harvard, Vancouver, ISO, and other styles
2

Cannon, E. W., E. C. Rhodes, A. D. Martin, and K. D. Coutts. "AEROBIC TRAINING AND RECOVERY VO2 KINETICS AFTER SUPRAMAXIMAL EXERCISE." Medicine & Science in Sports & Exercise 30, Supplement (May 1998): 199. http://dx.doi.org/10.1097/00005768-199805001-01131.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Hani, Al Haddad, Paul B. Laursen, Ahmaidi Said, and Buchheit Martin. "Nocturnal Heart Rate Variability Following Supramaximal Intermittent Exercise." International Journal of Sports Physiology and Performance 4, no. 4 (December 2009): 435–47. http://dx.doi.org/10.1123/ijspp.4.4.435.

Full text
Abstract:
Purpose:To assess the effect of supramaximal intermittent exercise on long-term cardiac autonomic activity, inferred from heart rate variability (HRV).Methods:Eleven healthy males performed a series of two consecutive intermittent 15-s runs at 95% VIFT (i.e., speed reached at the end of the 30-15 Intermittent Fitness Test) interspersed with 15 s of active recovery at 45% VIFT until exhaustion. Beat-to-beat intervals were recorded during two consecutive nights (habituation night and 1st night) before, 10 min before and immediately after exercise, as well as 12 h (2nd night) and 36 h (3rd night) after supramaximal intermittent exercise. The HRV indices were calculated from the last 5 min of resting and recovery periods, and the first 10 min of the first estimated slow wave sleep period.Results:Immediate post-supramaximal exercise vagal-related HRV indices were significantly lower than immediate pre-supramaximal exercise values (P < .001). Most vagal-related indices were lower during the 2nd night compared with the 1st night (eg, mean RR intervals, P = .03). Compared with the 2nd night, vagal-related HRV indices were significantly higher during the 3rd night. Variables were not different between the 1st and 3rd nights; however, we noted a tendency (adjusted effect size, aES) for an increased normalized high-frequency component (P = .06 and aES = 0.70) and a tendency toward a decreased low-frequency component (P = .06 and aES = 0.74).Conclusion:Results confirm the strong influence of exercise intensity on short- and long-term post exercise heart rate variability recovery and might help explain the high efficiency of supramaximal training for enhancing indices of cardiorespiratory fitness.
APA, Harvard, Vancouver, ISO, and other styles
4

Green, H. J., J. A. Thomson, and M. E. Houston. "Supramaximal exercise after training-induced hypervolemia. II. Blood/muscle substrates and metabolites." Journal of Applied Physiology 62, no. 5 (May 1, 1987): 1954–61. http://dx.doi.org/10.1152/jappl.1987.62.5.1954.

Full text
Abstract:
Blood and muscle substrates and metabolites were investigated in six healthy males (ranging in age from 19 to 23 yr) during three consecutive days of supramaximal exercise training. Muscle biopsies from the vastus lateralis and arterialized blood samples from a hand vein were extracted before the exercise and at selected times during the intermittent (1 min work to 4 min rest) cycling. The results indicated that blood glucose concentration was significantly depressed (P less than 0.05) on both days 2 and 3 of the training, whereas plasma free fatty acids and blood glycerol, pyruvate, alanine, and lactate were unaffected. At the muscle level, glucose and lactate concentrations were depressed on days 2 and 3, whereas ATP and glycogen were reduced only on the final day of training. No training-induced alterations were noted for muscle glucose 6-phosphate or muscle ADP. These results indicate that the approximate 10–11% reduction in O2-carrying capacity accompanying the short-term training does not directly and negatively influence muscle energy metabolism during the exercise. Rather, the explanation for the altered muscle and blood constituents must be sought from other effects of the training such as impaired carbohydrate repletion.
APA, Harvard, Vancouver, ISO, and other styles
5

Jabbour, Georges, and Horia-Daniel Iancu. "Supramaximal Exercise Training Enhances several Health-Related Outcomes in Obese Adults." Medicine & Science in Sports & Exercise 48 (May 2016): 417. http://dx.doi.org/10.1249/01.mss.0000486255.22195.8b.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Jabbour, Georges, Pascale Mauriege, Denis Joanisse, and Horia-Daniel Iancu. "Effect of supramaximal exercise training on metabolic outcomes in obese adults." Journal of Sports Sciences 35, no. 20 (October 15, 2016): 1975–81. http://dx.doi.org/10.1080/02640414.2016.1243798.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Patterson, Carson, and Christian Raschner. "Supramaximal Eccentric Training for Alpine Ski Racing—Strength Training with the Lifter." Applied Sciences 10, no. 24 (December 10, 2020): 8831. http://dx.doi.org/10.3390/app10248831.

Full text
Abstract:
Eccentric muscular work plays a large role in alpine ski racing. Training with supramaximal eccentric loads (SME) is highly effective to improve eccentric strength but potentially dangerous. Most SME training devices do not allow the athlete to move a barbell freely as they would when performing conventional barbell training. The Intelligent Motion Lifter (IML) allows for safe SME training with a free barbell and no spotters. The IML can be used for free barbell training: a spotter for normal training, eccentric only, concentric only, and squat jumps. It is also a training and testing device for isokinetic and isometric exercise. This commentary addresses the necessity of eccentric training for elite alpine ski racers, the development of the IML and its use in training.
APA, Harvard, Vancouver, ISO, and other styles
8

Bond, Stephanie L., Persephone Greco-Otto, Raymond Sides, Grace P. S. Kwong, Renaud Léguillette, and Warwick M. Bayly. "Assessment of two methods to determine the relative contributions of the aerobic and anaerobic energy systems in racehorses." Journal of Applied Physiology 126, no. 5 (May 1, 2019): 1390–98. http://dx.doi.org/10.1152/japplphysiol.00983.2018.

Full text
Abstract:
A prospective, randomized, controlled study was designed to determine relative aerobic and anaerobic (lactic and alactic) contributions at supramaximal exercise intensities using two different methods. Thoroughbred racehorses ( n = 5) performed a maximal rate of oxygen consumption (V̇o2max) test and three supramaximal treadmill runs (105, 115, and 125% V̇o2max). Blood lactate concentration (BL) was measured at rest, every 15 s during runs, and 2, 5, 10, 20, 30, 40, 50, and 60 min postexercise. In method 1, oxygen demand was calculated for each supramaximal intensity based on the V̇o2max test, and relative aerobic and anaerobic contributions were calculated from measured V̇o2 and the accumulated oxygen deficit. In method 2, aerobic contribution was calculated using the trapezoidal method to determine V̇o2 during exercise. A monoexponential model was fitted to the postexercise V̇o2 curve. Alactic contribution was calculated using the coefficients of this model. Lactate anaerobic contribution was calculated by multiplying the peak to resting change in BL by 3. Linear mixed-effects models were used to examine the effects of exercise intensity and method (as fixed effects) on measured outcomes ( P ≤ 0.05). Relative aerobic and anaerobic contributions were not different between methods ( P = 0.20). Horses’ mean contributions were 81.4, 77.6, and 72.5% (aerobic), and 18.5, 22.3, and 27.4% (anaerobic) at 105, 115, and 125% V̇o2max, respectively. Individual alactic anaerobic energy was not different between supramaximal exercise intensities ( P = 0.43) and was negligible, contributing a mean of 0.11% of the total energy. Relative energy contributions can be calculated using measured V̇o2 and BL in situations where the exercise intensity is unknown. Understanding relative metabolic demands could help develop tailored training programs. NEW & NOTEWORTHY Relative energy contributions of horses can be calculated using measured V̇o2 and BL in situations where the exercise intensity is unknown. Horses’ mean contributions were 81.4, 77.6, and 72.5% (aerobic), and 18.5, 22.3, and 27.4% (anaerobic) at 105, 115, and 125% of V̇o2max, respectively. Individual alactic capacity was unaltered between supramaximal exercise intensities and accounted for a mean contribution of 0.11% of energy use.
APA, Harvard, Vancouver, ISO, and other styles
9

Butcher, Scotty J., Madison T. Yurach, Nichole M. Heynen, Brendan J. Pikaluk, Karla J. Horvey, Ron Clemens, and Darcy D. Marciniuk. "The Physiologic Effects of an Acute Bout of Supramaximal High-Intensity Interval Training Compared with a Continuous Exercise Bout in Patients with COPD." Journal of Respiratory Medicine 2013 (October 24, 2013): 1–6. http://dx.doi.org/10.1155/2013/879695.

Full text
Abstract:
This study compared physiological responses and work performed during a supramaximal high-intensity interval exercise training session (HIIT) and a constant work rate (CWR) exercise session. Fourteen patients with COPD (mean FEV1 53±13% predicted (±SD)) completed an incremental cardiopulmonary exercise test (CPET) and a steep ramp anaerobic test (SRAT) and then two exercise bouts to symptom limitation on separate days, in random order: (1) a CWR trial at 80% of CPET peak work rate (mean 63±15 W) and (2) a HIIT trial using repeats of 30 s at 70% of SRAT peak work rate (mean 112±29 W) followed by 90 s at 20% of CPET peak work rate. Subjects ceased exercise primarily due to dyspnea for both HIIT and CWR (64% vs. 57%, resp.). End-exercise VE, HR, dyspnea, and leg fatigue were similar between the two exercise protocols. Average work rate was lower in HIIT than CWR (32 vs. 63 W, P<0.05); however, subjects performed HIIT longer (542 vs. 202 s, P<0.05) and for greater total work (23.3 vs. 12.0 kJ, P<0.05). The supramaximal HIIT protocol was well tolerated and demonstrated similar maximal physiologic responses to constant work rate exercise, but with greater leg muscle work performed and greater peak exercise intensity.
APA, Harvard, Vancouver, ISO, and other styles
10

Paull, Emily J., and Gary P. Van Guilder. "Remote ischemic preconditioning increases accumulated oxygen deficit in middle-distance runners." Journal of Applied Physiology 126, no. 5 (May 1, 2019): 1193–203. http://dx.doi.org/10.1152/japplphysiol.00585.2018.

Full text
Abstract:
The mediators underlying the putative benefits of remote ischemic preconditioning (IPC) on dynamic whole body exercise performance have not been widely investigated. Our objective was to test the hypothesis that remote IPC improves supramaximal exercise performance in National Collegiate Athletic Association (NCAA) Division I middle-distance runners by increasing accumulated oxygen deficit (AOD), an indicator of glycolytic capacity. A randomized sham-controlled crossover study was employed. Ten NCAA Division I middle-distance athletes [age: 21 ± 1 yr; maximal oxygen uptake (V̇o2max): 65 ± 7 ml·kg−1·min−1] completed three supramaximal running trials (baseline, after mock IPC, and with remote IPC) at 110% V̇o2max to exhaustion. Remote IPC was induced in the right arm with 4 × 5 min cycles of brachial artery ischemia with 5 min of reperfusion. Supramaximal AOD (ml/kg) was calculated as the difference between the theoretical oxygen demand required for the supramaximal running bout (linear regression extrapolated from ~12 × 5 min submaximal running stages) and the actual oxygen demand for these bouts. Remote IPC [122 ± 38 s, 95% confidence interval (CI): 94–150] increased ( P < 0.001) time to exhaustion 22% compared with baseline (99 ± 23 s, 95% CI: 82–116, P = 0.014) and sham (101 ± 30 s, 95% CI: 80–123, P = 0.001). In the presence of IPC, AOD was 47 ± 36 ml/kg (95% CI: 20.8–73.9), a 29% increase compared with baseline (36 ± 28 ml/kg, 95% CI: 16.3–56.9, P = 0.008) and sham (38 ± 32 ml/kg, 95% CI: 16.2–63.0, P = 0.024). Remote IPC considerably improved supramaximal exercise performance in NCAA Division I middle-distance athletes. Greater glycolytic capacity, as estimated by increased AOD, is a potential mediator for these performance improvements. NEW & NOTEWORTHY Our novel findings indicate that ischemic preconditioning enhanced glycolytic exercise capacity, enabling National Collegiate Athletic Association (NCAA) middle-distance track athletes to run ~22 s longer before exhaustion compared with baseline and mock ischemic preconditioning. The increase in “all-out” performance appears to be due to increased accumulated oxygen deficit, an index of better supramaximal capacity. Of note, enhanced exercise performance was demonstrated in a specific group of in-competition NCAA elite athletes that has already undergone substantial training of the glycolytic energy systems.
APA, Harvard, Vancouver, ISO, and other styles
More sources

Dissertations / Theses on the topic "Supramaximal exercise and training"

1

Weber, Clare L., and n/a. "Metabolic Responses to Supramaximal Exercise and Training: A Gender Comparison." Griffith University. School of Physiotherapy and Exercise Science, 2003. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20030407.155407.

Full text
Abstract:
The primary aim of this thesis was to investigate the gender-specific responses to supramaximal cycling and to examine the changes in anaerobic and aerobic metabolism that occur in response to high-intensity interval training (HIT). All subjects in the present experiments were untrained, healthy young adults aged between 18 and 35 yr. Cycle ergometry was used for all experimental test procedures and training programs. The accumulated oxygen (AO2) deficit was used to quantify the production of adenosine triphosphate (ATP) via anaerobic metabolism during supramaximal cycling. In addition, pulmonary oxygen uptake measured at the onset of exercise was described using mathematical modeling to determine the rate response of the aerobic energy system during exercise. The purpose of experiment one was to examine the test-retest reliability of the maximal accumulated oxygen deficit (MAOD) measured at 110% and 120% of peak oxygen uptake for cycling in seven untrained male and seven untrained female subjects. After one familiarization trial, all subjects performed two MAOD tests at a power output corresponding to 110% and two tests at 120% of peak oxygen uptake in random order. MAOD was calculated for each subject as the difference between the estimated AO2 demand and the AO2 uptake measured during the exercise bout. The mean±standard error time to exhaustion (TE) for the group was not significantly different between trial one (226±13 s) and trial two (223±14 s) of the 110% test. Likewise, the difference in the TE between trial one (158±11 s) and trial two (159±10 s) was not significant for the 120% test. The intra-class correlation coefficients for the TE were 0.95 for the 110% test and 0.98 for the 120% test. The mean MAOD value obtained in trial one (2.62±0.17 L) was not significantly different from the mean value obtained in trial two (2.54±0.19 L) for the 110% test. Additionally, the mean values for the two trials did not differ significantly for MAOD (2.64±0.21 L for trial one and 2.63±0.19 L for trial two) in the 120% test. The intra-class correlation coefficients for MAOD were 0.95 for the 110% test and 0.97 for the 120% test. All intra-class correlation coefficients were significant at p < 0.001. When conducted under standardized conditions, the determination of MAOD for cycling was highly repeatable at both 110% and 120% of peak oxygen uptake in untrained male and female subjects. The results observed in experiment one suggest that the MAOD may be used to compare the anaerobic capacity (AC) of men and women and to examine changes in the contribution of the anaerobic energy systems before and after training. Experiment two examined the gender-specific differences in MAOD before and after 4 and 8 wk of HIT. Untrained men (n=7) and women (n=7) cycled at 120% of pre-training peak oxygen uptake to exhaustion (MAOD test) pre-, mid-, and post-training. A post-training timed test was also completed at the MAOD test power output, but this test was stopped at the TE achieved during the pre-training MAOD test. The 14.3±5.2% increase in MAOD observed in males after 4 wk of training was not different from the 14.0±3.0% increase seen in females (p > 0.05). MAOD increased by a further 6.6±1.9% in males and this change was not different from the additional 5.1±2.3% increase observed in females after the final 4 wk of training. Peak oxygen uptake measured during incremental cycling increased significantly (p < 0.01) in male but not in female subjects after 8 wk of training. Moreover, the AO2 uptake was higher in men during the post-training timed test compared to the pre-training MAOD test (p < 0.01). In contrast, the AO2 uptake was unchanged from pre- to post-training in female subjects. The increase in MAOD with training was not different between men and women suggesting an enhanced ability to produce ATP anaerobically in both groups. However, the increase in peak oxygen uptake and AO2 uptake obtained in male subjects following training indicates improved oxidative metabolism in men but not in women. It was concluded that there are basic gender differences that may predispose males and females to specific metabolic adaptations following an 8-wk period of HIT. Increases in AO2 uptake during supramaximal cycling demonstrated in men after training led to the hypothesis that peak oxygen uptake kinetics are speeded in male subjects with short-term HIT. It was suggested that training does not improve peak oxygen uptake kinetics in women as no change in AO2 uptake was found after 8 wk of HIT in female subjects. The purpose of experiment three was to examine peak oxygen uptake kinetics before and after 8 wk of HIT in six men and six women during cycling at 50% (50% test) and 110% (110% test) of pre-training peak oxygen uptake. A single-term exponential equation was used to model the peak oxygen uptake response (after phase I) during the 50% and 110% tests pre- and post-training. In addition, phase II and III of the peak oxygen uptake response during the 110% tests were examined using a two-term equation. The end of the phase I peak oxygen uptake response was identified visually and omitted from the modeling process. The duration of phase I determined during all experimental tests was not different between men and women and did not change with training in either group. Before training, men obtained a phase II peak oxygen uptake time constant (t2) of 29.0±3.3 s during the 50% test which was not different to the t2 of 28.8±2.2 s attained by women. In addition, the t2 determined during the 50% test was unchanged after 8 wk of HIT in both groups. The peak oxygen uptake kinetics examined during the 110% tests before training were well described by a single-term model in all male and female subjects. The t2 determined before training for the 110% test was significantly faster in men than in women. Furthermore, peak oxygen uptake was unchanged in female subjects and the t2 remained unaltered with 8 wk HIT (pre 45.5±2.2; post 44.8±2.3 s). In contrast, male subjects achieved a significantly higher peak oxygen uptake after training and the t2 determined for men during the 110% test was faster after training (36.4±1.6 s) than before training (40.1± 1.9 s). Improved model fits were obtained with the two-term equation compared to the single-term equation in two of the six male subjects during the 110% test post-training. It was found that the onset of the peak oxygen uptake slow component occurred at a mean time of 63.5±2.5 s and the t2 was reduced to 18.4±1.7 s. Using a Wilcoxon Signed Ranks z-test, the t2 described by the single-term equation in the remaining four subjects was determined to be significantly faster after training than before training, thus confirming the results obtained from the original group (n=6) of male subjects. End exercise heart rate (HREE) values obtained during the 50% and 110% tests were not different between men and women. During the 50% test, HREE values were unchanged, whereas HREE was significantly decreased during the 110% test after training in both groups. These data show that HIT might improve oxidative metabolism in men but not in women as reflected by a greater peak oxygen uptake and faster peak oxygen uptake kinetics during supramaximal work rates. We further suggest that the faster peak oxygen uptake kinetics demonstrated in men after training are probably not due to an improvement in cardiac function. Finally, the augmentation of oxidative metabolism during exercise after HIT in men might be dependent on the intensity of the exercise bout at which the peak oxygen uptake response is examined. The findings presented in this thesis suggest that MAOD is a reliable measure in both male and female subjects and can be used to monitor changes in anaerobic ATP production during supramaximal cycling. Moreover, these data suggest that 4 and 8 wk of HIT produce similar changes in anaerobic ATP generation in men and women. Finally, 8 wk of HIT results in the increase of peak oxygen uptake and AO2 uptake as well as the speeding of peak oxygen uptake kinetics during supramaximal cycling in male subjects. There was no evidence to suggest that oxidative metabolism was improved in women after short-term HIT. In conclusion, improvement in supramaximal exercise performances should be examined specifically for changes in the anaerobic and aerobic contributions to energy production. In addition, it is suggested that gender should be of primary consideration when designing exercise-training programs where improvement in both anaerobic and aerobic metabolism is required.
APA, Harvard, Vancouver, ISO, and other styles
2

Weber, Clare L. "Metabolic Responses to Supramaximal Exercise and Training: A Gender Comparison." Thesis, Griffith University, 2003. http://hdl.handle.net/10072/366993.

Full text
Abstract:
The primary aim of this thesis was to investigate the gender-specific responses to supramaximal cycling and to examine the changes in anaerobic and aerobic metabolism that occur in response to high-intensity interval training (HIT). All subjects in the present experiments were untrained, healthy young adults aged between 18 and 35 yr. Cycle ergometry was used for all experimental test procedures and training programs. The accumulated oxygen (AO2) deficit was used to quantify the production of adenosine triphosphate (ATP) via anaerobic metabolism during supramaximal cycling. In addition, pulmonary oxygen uptake measured at the onset of exercise was described using mathematical modeling to determine the rate response of the aerobic energy system during exercise. The purpose of experiment one was to examine the test-retest reliability of the maximal accumulated oxygen deficit (MAOD) measured at 110% and 120% of peak oxygen uptake for cycling in seven untrained male and seven untrained female subjects. After one familiarization trial, all subjects performed two MAOD tests at a power output corresponding to 110% and two tests at 120% of peak oxygen uptake in random order. MAOD was calculated for each subject as the difference between the estimated AO2 demand and the AO2 uptake measured during the exercise bout. The mean±standard error time to exhaustion (TE) for the group was not significantly different between trial one (226±13 s) and trial two (223±14 s) of the 110% test. Likewise, the difference in the TE between trial one (158±11 s) and trial two (159±10 s) was not significant for the 120% test. The intra-class correlation coefficients for the TE were 0.95 for the 110% test and 0.98 for the 120% test. The mean MAOD value obtained in trial one (2.62±0.17 L) was not significantly different from the mean value obtained in trial two (2.54±0.19 L) for the 110% test. Additionally, the mean values for the two trials did not differ significantly for MAOD (2.64±0.21 L for trial one and 2.63±0.19 L for trial two) in the 120% test. The intra-class correlation coefficients for MAOD were 0.95 for the 110% test and 0.97 for the 120% test. All intra-class correlation coefficients were significant at p < 0.001. When conducted under standardized conditions, the determination of MAOD for cycling was highly repeatable at both 110% and 120% of peak oxygen uptake in untrained male and female subjects. The results observed in experiment one suggest that the MAOD may be used to compare the anaerobic capacity (AC) of men and women and to examine changes in the contribution of the anaerobic energy systems before and after training. Experiment two examined the gender-specific differences in MAOD before and after 4 and 8 wk of HIT. Untrained men (n=7) and women (n=7) cycled at 120% of pre-training peak oxygen uptake to exhaustion (MAOD test) pre-, mid-, and post-training. A post-training timed test was also completed at the MAOD test power output, but this test was stopped at the TE achieved during the pre-training MAOD test. The 14.3±5.2% increase in MAOD observed in males after 4 wk of training was not different from the 14.0±3.0% increase seen in females (p > 0.05). MAOD increased by a further 6.6±1.9% in males and this change was not different from the additional 5.1±2.3% increase observed in females after the final 4 wk of training. Peak oxygen uptake measured during incremental cycling increased significantly (p < 0.01) in male but not in female subjects after 8 wk of training. Moreover, the AO2 uptake was higher in men during the post-training timed test compared to the pre-training MAOD test (p < 0.01). In contrast, the AO2 uptake was unchanged from pre- to post-training in female subjects. The increase in MAOD with training was not different between men and women suggesting an enhanced ability to produce ATP anaerobically in both groups. However, the increase in peak oxygen uptake and AO2 uptake obtained in male subjects following training indicates improved oxidative metabolism in men but not in women. It was concluded that there are basic gender differences that may predispose males and females to specific metabolic adaptations following an 8-wk period of HIT. Increases in AO2 uptake during supramaximal cycling demonstrated in men after training led to the hypothesis that peak oxygen uptake kinetics are speeded in male subjects with short-term HIT. It was suggested that training does not improve peak oxygen uptake kinetics in women as no change in AO2 uptake was found after 8 wk of HIT in female subjects. The purpose of experiment three was to examine peak oxygen uptake kinetics before and after 8 wk of HIT in six men and six women during cycling at 50% (50% test) and 110% (110% test) of pre-training peak oxygen uptake. A single-term exponential equation was used to model the peak oxygen uptake response (after phase I) during the 50% and 110% tests pre- and post-training. In addition, phase II and III of the peak oxygen uptake response during the 110% tests were examined using a two-term equation. The end of the phase I peak oxygen uptake response was identified visually and omitted from the modeling process. The duration of phase I determined during all experimental tests was not different between men and women and did not change with training in either group. Before training, men obtained a phase II peak oxygen uptake time constant (t2) of 29.0±3.3 s during the 50% test which was not different to the t2 of 28.8±2.2 s attained by women. In addition, the t2 determined during the 50% test was unchanged after 8 wk of HIT in both groups. The peak oxygen uptake kinetics examined during the 110% tests before training were well described by a single-term model in all male and female subjects. The t2 determined before training for the 110% test was significantly faster in men than in women. Furthermore, peak oxygen uptake was unchanged in female subjects and the t2 remained unaltered with 8 wk HIT (pre 45.5±2.2; post 44.8±2.3 s). In contrast, male subjects achieved a significantly higher peak oxygen uptake after training and the t2 determined for men during the 110% test was faster after training (36.4±1.6 s) than before training (40.1± 1.9 s). Improved model fits were obtained with the two-term equation compared to the single-term equation in two of the six male subjects during the 110% test post-training. It was found that the onset of the peak oxygen uptake slow component occurred at a mean time of 63.5±2.5 s and the t2 was reduced to 18.4±1.7 s. Using a Wilcoxon Signed Ranks z-test, the t2 described by the single-term equation in the remaining four subjects was determined to be significantly faster after training than before training, thus confirming the results obtained from the original group (n=6) of male subjects. End exercise heart rate (HREE) values obtained during the 50% and 110% tests were not different between men and women. During the 50% test, HREE values were unchanged, whereas HREE was significantly decreased during the 110% test after training in both groups. These data show that HIT might improve oxidative metabolism in men but not in women as reflected by a greater peak oxygen uptake and faster peak oxygen uptake kinetics during supramaximal work rates. We further suggest that the faster peak oxygen uptake kinetics demonstrated in men after training are probably not due to an improvement in cardiac function. Finally, the augmentation of oxidative metabolism during exercise after HIT in men might be dependent on the intensity of the exercise bout at which the peak oxygen uptake response is examined. The findings presented in this thesis suggest that MAOD is a reliable measure in both male and female subjects and can be used to monitor changes in anaerobic ATP production during supramaximal cycling. Moreover, these data suggest that 4 and 8 wk of HIT produce similar changes in anaerobic ATP generation in men and women. Finally, 8 wk of HIT results in the increase of peak oxygen uptake and AO2 uptake as well as the speeding of peak oxygen uptake kinetics during supramaximal cycling in male subjects. There was no evidence to suggest that oxidative metabolism was improved in women after short-term HIT. In conclusion, improvement in supramaximal exercise performances should be examined specifically for changes in the anaerobic and aerobic contributions to energy production. In addition, it is suggested that gender should be of primary consideration when designing exercise-training programs where improvement in both anaerobic and aerobic metabolism is required.
Thesis (PhD Doctorate)
Doctor of Philosophy (PhD)
School of Physiotherapy and Exercise Science
Full Text
APA, Harvard, Vancouver, ISO, and other styles
3

Nobbs, Les. "Aetiology of fatigue during maximal and supramaximal exercise." Doctoral thesis, University of Cape Town, 2003. http://hdl.handle.net/11427/3189.

Full text
Abstract:
Includes bibliography.
The aim of this thesis was to investigate the extent of peripheral and central components in the development of fatigue during maximal exercise. Fatigue during maximal and supramaximal excercise has traditionally been modelled from the peripheral context of an inadequate capacity to supply metabolic substrate to the contracting muscles to meet the increased energy demand.
APA, Harvard, Vancouver, ISO, and other styles
4

Ansley, Les. "Aetiology of fatigue during maximal and supramaximal exercise." Diss., University of Cape Town, 2003. http://www.oregonpdf.org.

Full text
Abstract:
Thesis (Ph. D.)--University of Cape Town, 2003.
Includes bibliographical references (leaves 284-287). Also available online (PDF file) by a subscription to the set or by purchasing the individual file.
APA, Harvard, Vancouver, ISO, and other styles
5

Maxwell, Neil S. "Sprint running in man and the effects of performing supramaximal exercise under different conditions of stress." Thesis, University of Strathclyde, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.367034.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Parks, Jason C. "EFFECTS OF A PROPER COOL-DOWN AFTER SUPRAMAXIMAL INTERVAL EXERCISE ON PULSE WAVE REFLECTION, AORTIC STIFFNESS, AND AUTONOMIC MODULATION." Kent State University / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=kent158448596372427.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Leiferman, Jennifer A. "Temporal Specificity in Exercise Training." Thesis, University of North Texas, 1995. https://digital.library.unt.edu/ark:/67531/metadc278652/.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Shamim, Baubak. "Concurrent exercise from training to transcriptome." Phd thesis, Australian Catholic University, 2020. https://acuresearchbank.acu.edu.au/download/4116399d7c75edb34b93aa0f45f835667d00b358764a4a938e474d5b8ff63ca7/2892125/Shamim_2020_Concurrent_exercise_from_training_to_transcriptome.pdf.

Full text
Abstract:
The principle of training specificity dictates that adaptations to exercise training are specific to the mode, frequency, and duration of exercise performed, and result in distinct and divergent skeletal muscle phenotypes. Strength-based training promotes skeletal muscle hypertrophy and maximal force-generating capacity while endurance-based training improves skeletal muscle oxidative capacity and cardiorespiratory fitness. Previous research has suggested the capacity of skeletal muscle to adapt to strength and endurance training when performed simultaneously (i.e., concurrent exercise training) appears to be limited and results in blunted resistance-based adaptations compared to resistance training alone – a phenomenon referred to as the ‘interference effect.’ The molecular basis of skeletal muscle adaptation to exercise training involves the propagation of numerous mechanical and chemical stimuli through signalling cascades that ultimately results in an increase in an array of exercise-induced proteins and increases in maximal enzyme activities. The nature of these alterations is specific to the frequency, intensity, volume, and type of metabolic demands placed upon the muscle during exercise. Given the divergent stimuli associated with endurance- and resistance-based exercise, it has been hypothesised that antagonistic molecular signals may underlie the adaptive interference observed with concurrent training. In order to circumvent this effect, strategies have focused on altering the proximity of training sessions (i.e., same day versus alternate day training) and training variables (i.e., frequency, volume, mode). Additionally, optimising post-exercise nutrition (i.e., dietary protein) has been proposed as a potential variable that may promote anabolic signalling and prevent the interference effect. To determine whether these training strategies in association with a high protein diet (2 g•kg-1•d-1) can attenuate the ‘interference effect,’ 32 recreationally active males (age: 25±5 y; body mass index: 24±3 kgm-2; mean ± standard deviation) performed 12 wk of either isolated resistance (RES; n=10) or endurance (END; n=10) training (3 sessions•wk-1), or concurrent resistance and endurance (CET; n=12) training (6 sessions•wk-1). Maximal strength, maximal aerobic capacity, peak power, body composition, and muscle architecture were assessed throughout the intervention. To explore molecular responses that may underpin any impaired adaptation after concurrent exercise training, satellite cells and myonuclei were assessed by immunohistochemistry from skeletal muscle biopsy samples. In addition, exploratory transcriptomics was performed from a subset of participants from each training condition. The results from the investigations undertaken for this thesis demonstrate that – despite efforts to circumvent the ‘interference effect’ by implementing recommended strategies of alternate day training, minimising exercise volume, and increasing dietary protein intake – maximal anaerobic power development was attenuated following 12 wk of concurrent exercise training. Myofibre hypertrophy increased to the same magnitude in all training modalities without changes to satellite cell content, suggesting that satellite cell content does not limit the magnitude of hypertrophy achieved during concurrent training. Conversely, myonuclear content displayed strong associations with the degree of myofibre hypertrophy. Transcriptome-wide analysis revealed that concurrent exercise training augments gene sets related to plasma membrane structures while suppressing those related to regulation of messenger ribonucleic acid (mRNA) processing and protein degradation, which may contribute to the ‘interference effect’ in myofibre hypertrophy. Additionally, considerable overlap of gene sets enriched for terms related to extracellular matrix remodelling were observed amongst concurrent exercise training and isolated endurance cycle training, which may underlie attenuations in maximal anaerobic power outputs observed following concurrent training. Collectively, these reveal that the current recommendations to maximise muscle hypertrophy with concurrent training do not result in augmented hypertrophic responses compared to single-mode training, and cannot be explained by satellite cell content or inhibition of anabolic gene programs. These findings underpin future investigations of molecular pathways that have not been considered in the context of concurrent training adaptations.
APA, Harvard, Vancouver, ISO, and other styles
9

Hwang, Hyosook. "Exercise training effects on myocardial stunning." Columbus, Ohio : Ohio State University, 2004. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1078942640.

Full text
Abstract:
Thesis (Ph. D.)--Ohio State University, 2004.
Title from first page of PDF file. Document formatted into pages; contains xviii, 132 p.; also includes graphics. Includes abstract and vita. Advisors: George E. Billman and Timothy E. Kirby, Dept. of Educational Services and Research. Includes bibliographical references (p. 124-132).
APA, Harvard, Vancouver, ISO, and other styles
10

Roffey, Darren M. "Exercise intensity, exercise training and energy metabolism in overweight and obese males." Thesis, Queensland University of Technology, 2008. https://eprints.qut.edu.au/17823/1/Darren_Roffey_Thesis.pdf.

Full text
Abstract:
The primary objective of this PhD program was to investigate the impact of training at a constant-load moderate-intensity (FATmax) compared to work-matched high-intensity intervals (HIIT) on the metabolic, physiological and psychosocial health profiles of sedentary overweight and obese men. This study was unique in that it was the first time the effect of exercise intensity had been investigated to examine concurrently the components of whole-body energy metabolism and body composition as measured using gold standard techniques. Based upon the positive alterations in blood lipids, body composition, cardiorespiratory fitness and substrate oxidation, it appears that training at FATmax can positively impact health parameters as well as, or if not better than, high-intensity training. Furthermore, there are ramifications for public health messages and obesity management strategies arising from these findings, primarily attributable to the increased exercise adherence and the reduction in health risks stemming from the significant loss of abdominal visceral adipose tissue after FATmax training.
APA, Harvard, Vancouver, ISO, and other styles
More sources

Books on the topic "Supramaximal exercise and training"

1

Thomas, Tom R. Scientific exercise training. 2nd ed. Dubuque, Iowa: Kendall/Hunt Pub. Co., 1987.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
2

1953-, Kraemer William J., ed. Designing resistance training programs. 3rd ed. Champaign, IL: Human Kinetics, 2004.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
3

Bleue, Scott. The effects of creatine supplementation on the maximal accumulated oxygen deficit and anaerobic capacity during supramaximal exercise. Ottawa: National Library of Canada, 1995.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
4

1948-, Gleeson Michael, and Greenhaff Paul L, eds. Biochemistry of exercise and training. Oxford [England]: Oxford University Press, 1997.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
5

1962-, Ellenbecker Todd S., ed. Strength band training. 2nd ed. Champaign, IL: Human Kinetics, 2011.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
6

Gustavsen, Rolf. Training therapy: Prophylaxis and rehabilitation. 2nd ed. Stuttgart: G. Thieme Verlag, 1993.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
7

Training therapy: Prophylaxis and rehabilitation. New York: Thieme Inc., 1985.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
8

1953-, Kraemer William J., ed. Designing resistance training programs. 2nd ed. Champaign, IL: Human Kinetics, 1997.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
9

Fleck, Steven J. Designing resistance training programs. Champaign, Ill: Human Kinetics Books, 1987.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
10

Corapi, Robert. Superhero training methods. Royal Palm Beach, FL: North Yard Publishing, 2013.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
More sources

Book chapters on the topic "Supramaximal exercise and training"

1

Ferretti, Guido. "Supramaximal Exercise." In Energetics of Muscular Exercise, 157–80. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-05636-4_6.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Proske, Uwe, David L. Morgan, Tamara Hew-Butler, Kevin G. Keenan, Roger M. Enoka, Sebastian Sixt, Josef Niebauer, et al. "Exercise Training." In Encyclopedia of Exercise Medicine in Health and Disease, 324. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_2383.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Bloom, Michael V., and David A. Smith. "Relaxation Exercise Training." In Brief Mental Health Interventions for the Family Physician, 117–21. New York, NY: Springer New York, 2001. http://dx.doi.org/10.1007/978-1-4613-0153-0_14.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Baker, Julien S., Fergal Grace, Lon Kilgore, David J. Smith, Stephen R. Norris, Andrew W. Gardner, Robert Ringseis, et al. "Physical Exercise Training." In Encyclopedia of Exercise Medicine in Health and Disease, 710. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_4584.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Bherer, Louis, and Kristell Pothier. "Physical Activity and Exercise." In Cognitive Training, 319–30. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39292-5_22.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Ebersole, Kyle T., and Ronald E. Pfeiffer. "Athletic Training." In Introduction to Exercise Science, 149–66. Fifth edition. | Milton Park, Abingdon, Oxon ; New York, NY :: Routledge, 2017. http://dx.doi.org/10.4324/9781315177670-7.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Verges, Samuel. "Respiratory Muscle Training." In Exercise and Sports Pulmonology, 143–51. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-05258-4_10.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Keteyian, Steven J., and John R. Schairer. "Exercise Training and Prescription." In Sports Cardiology Essentials, 63–84. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-0-387-92775-6_4.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Boritz, Tali, Shelley McMain, Alexandre Vaz, and Tony Rousmaniere. "Exercise 7. Skills training." In Deliberate practice in dialectical behavior therapy., 89–99. Washington: American Psychological Association, 2023. http://dx.doi.org/10.1037/0000322-009.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Armstrong, Matthew, Rebecca Crouch, and Ioannis Vogiatzis. "Modalities of exercise training." In Pulmonary Rehabilitation, 209–18. Second edition. | Boca Raton : CRC Press, [2020] | Preceded by Pulmonary rehabilitation / Claudio F. Donner, Nicolino Ambrosino, Roger Goldstein. 2005.: CRC Press, 2020. http://dx.doi.org/10.1201/9781351015592-21.

Full text
APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Supramaximal exercise and training"

1

Moraes Frade, Maria Cecília, Thomas Beltrame, Mariana De Oliveira Gois, Ariane Petronilho, Stephanie Nogueira Linares, Silvia Cristina Garcia Moura De Tonello, and Aparecida Maria Catai. "Checking true VO2max values by supramaximal exercise testing: physiological insights." In ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.pa411.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Camata, T. V., J. L. Dantas, T. Abrão, M. A. O. C. Brunetto, A. C. Moraes, and L. R. Altimari. "Fourier and wavelet spectral analysis of EMG signals in supramaximal constant load dynamic exercise." In 2010 32nd Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC 2010). IEEE, 2010. http://dx.doi.org/10.1109/iembs.2010.5626743.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Butcher, Scotty J., Madison Yurach, Brendan J. Pikaluk, Nichole M. Heynen, Robyn L. Chura, and Darcy Marciniuk. "Development Of A Supramaximal Interval Exercise Protocol In Patients With COPD: Effects On Work Performed And Ventilatory Limitations." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a3970.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

"Intelligent Stretching Exercise Training System." In 2nd International conference on Innovative Engineering Technologies. International Institute of Engineers, 2015. http://dx.doi.org/10.15242/iie.e0815039.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Wahyudati, Sri. "Exercise Training after Cardiac Surgery." In The 11th National Congress and The 18th Annual Scientific Meeting of Indonesian Physical Medicine and Rehabilitation Association. SCITEPRESS - Science and Technology Publications, 2019. http://dx.doi.org/10.5220/0009062800760081.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Hermus, James, Cameron Hays, Michal Adamski, Hannah Lider, Jenny Westlund, Austin Scholp, John Webster, and Bjoern Buehring. "Posture monitor for vibration exercise training." In 2015 IEEE Great Lakes Biomedical Conference (GLBC). IEEE, 2015. http://dx.doi.org/10.1109/glbc.2015.7158302.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Schwarzer, M., S. Zeeb, E. Heyne, G. Färber, L. G. Koch, S. L. Britton, and T. Doenst. "High Aerobic Exercise Capacity Predicts Increased Mitochondrial Response to Exercise Training." In 50th Annual Meeting of the German Society for Thoracic and Cardiovascular Surgery (DGTHG). Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1725602.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Barğı, Gülşah, Meral Boşnak Güçlü, Zübeyde Nur Özkurt, and Münci Yağcı. "Upper extremity aerobic exercise training improves exercise capacity in stem cell recipients." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa1441.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Marillier, Mathieu, Anna Borowik, Samarmar Chacaroun, Sébastien Baillieul, Stéphane Doutreleau, Michel Guinot, Bernard Wuyam, et al. "Effect of exercise training on cerebral oxygenation and affects during exercise in obesity." In ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.pa419.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Topasna, Daniela M., and Gregory A. Topasna. "Nonlinear Optics Mathcad Exercise for Undergraduate Students." In Education and Training in Optics and Photonics. Washington, D.C.: OSA, 2007. http://dx.doi.org/10.1364/etop.2007.etd4.

Full text
APA, Harvard, Vancouver, ISO, and other styles

Reports on the topic "Supramaximal exercise and training"

1

Nelson, Matthew A., Dmitry Keselman, and Joseph F. Longo. SMS Software Training Exercise 101. Office of Scientific and Technical Information (OSTI), July 2013. http://dx.doi.org/10.2172/1093943.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

ARMY SAFETY CENTER FORT RUCKER AL. Field Training Exercise Safety Checklist. Fort Belvoir, VA: Defense Technical Information Center, January 1988. http://dx.doi.org/10.21236/ada382899.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

ARMY SAFETY CENTER FORT RUCKER AL. Field Training Exercise Safety Checklist. Fort Belvoir, VA: Defense Technical Information Center, January 1985. http://dx.doi.org/10.21236/ada382993.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Gillilan, Justin. 3rd Quarter 2021 Training Exercise Guide. Office of Scientific and Technical Information (OSTI), August 2021. http://dx.doi.org/10.2172/1812648.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Crane, Peter M., Samuel G. Schiflett, and Randy L. Oser. Roadrunner '98: Training Effectiveness in a Distributed Mission Training Exercise. Fort Belvoir, VA: Defense Technical Information Center, April 2000. http://dx.doi.org/10.21236/ada387746.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Schuld, E. P., and D. F. Cruff. ARGX-87: Accident Response Group Exercise, 1987: A Broken Arrow mini exercise. [Training]. Office of Scientific and Technical Information (OSTI), July 1987. http://dx.doi.org/10.2172/6028768.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Dobranich, P. R., T. W. Widney, P. T. Goolsby, J. D. Nelson, and D. A. Evanko. Exercise manual for the Augmented Computer Exercise for Inspection Training (ACE-IT) software. Office of Scientific and Technical Information (OSTI), September 1997. http://dx.doi.org/10.2172/537399.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Dale, Don, Leisa Davenhall, and Tenisha Highsmith. TA-55 Forensic Support Operations Cross-Training Exercise. Office of Scientific and Technical Information (OSTI), May 2020. http://dx.doi.org/10.2172/1631552.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Gillilan, Justin. RCT Continuing Training 4th Quarter 2021 Exercise Guide. Office of Scientific and Technical Information (OSTI), October 2021. http://dx.doi.org/10.2172/1827535.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Gillilan, Justin. RCT Continuing Training 1st Quarter 2022 Exercise Guide. Office of Scientific and Technical Information (OSTI), January 2022. http://dx.doi.org/10.2172/1838283.

Full text
APA, Harvard, Vancouver, ISO, and other styles
You might also be interested in the extended bibliographies on the topic 'Supramaximal exercise and training' for particular source types:
Journal articles Dissertations / Theses Books
We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!

To the bibliography