Relevant bibliographies by topics / Sudden infant death syndrome

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
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Academic literature on the topic 'Sudden infant death syndrome'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 August 2024

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Journal articles on the topic "Sudden infant death syndrome"

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Özcanlı Çay, Özlem, and Özlem Kemer Aycan. "Sudden infant death syndrome." Journal of Controversies in Obstetrics & Gynecology and Pediatrics 1, no. 3 (July 30, 2023): 74–77. http://dx.doi.org/10.51271/jcogp-0016.

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Sudden Infant Death Syndrome (SIDS) is a type of sudden and unexpected infant death, a term that encompasses both deaths from SIDS and ultimately all unexpected infant deaths with a determined cause. 1 Between %27 and % 43 of 3500 sudden unexpected infant death cases in the USA annually are due to SIDS. 2, 3 A number of other terms are used in pediatrics to describe sudden and unexpected deaths. Sudden unexpected death of an infant can be used interchangeably with sudden unexpected infant death, and sudden death in youth (VAS) refers to such death in any child 19 years of age or younger. Sudden unexplained early neonatal death is limited to infants who die within the first week of life and is usually congenital. anomaly is caused. Sudden intrauterine unexpected death syndrome refers to stillbirths for which postmortem examination cannot identify a cause, and sudden unexpected death in epilepsy is unexpected death in a person with epilepsy (excluding trauma or suffocation) for which postmortem examination does not reveal an anatomical or toxicological cause.
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KEENS, THOMAS G., and SALLY L. DAVIDSON WARD. "Sudden Infant Death Syndrome." Pediatrics 79, no. 1 (January 1, 1987): 162. http://dx.doi.org/10.1542/peds.79.1.162.

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In Reply.— Dr Stivrins' letter allows us to clarify some important points made by our paper entitled, "Sudden Infant Death Syndrome in Infants Evaluated by Apnea Programs in California" (Pediatrics 1986;77:451-455). The purpose of the study was to investigate the characteristics of infants who died after evaluation by an apnea program for apneic episodes or for statistical increased risk for SIDS. Although this does not represent a complete sampling of the apnea programs in California, it presents data on more than 3,000 infants who were evaluated.
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Koehler, Steve A. "Sudden infant death syndrome deaths." Journal of Forensic Nursing 4, no. 3 (September 2008): 141–42. http://dx.doi.org/10.1097/01263942-200809000-00007.

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Almansoor, Basel Abdulrahman, and Waleed Ahmed Almaiman. "Sudden infant death syndrome." International Journal of Advances in Medicine 10, no. 4 (March 24, 2023): 340–44. http://dx.doi.org/10.18203/2349-3933.ijam20230713.

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The sudden death of infants younger than one year is known as SIDS. It is a major cause of mortality among infants less than one year of age. There is little focus in the literature on sudden infant death syndrome (SIDS). Thus, this review aims to provide new insights on the incidence, risk factors and prevention of SIDS in infants. Following preferred reporting items for systematic review and meta-analysis (PRISMA) guidelines, this review explored eligible articles from PubMed, Google Scholar, and research gate databases. Several keywords were used for the search to obtain all possible related articles. The inclusion criteria were reviews and original articles focused on the current subject and written in English. The eligible articles used to compile this review were published between 2007 and 2021. Three main titles were discussed in this review: incidence, risk factors, and prevention. SIDS is the most common reason for death among infants aged less than one year. There is great variation in the global incidence of SIDS. Factors related to the sleeping environment are the major risk factors for SIDS and should be the focus. Hence, following the recommendations of the American academy of pediatrics can reduce incidence of SIDS.
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Carroll, John L., and Gerald M. Loughlin. "Sudden Infant Death Syndrome." Pediatrics In Review 14, no. 3 (March 1, 1993): 83–93. http://dx.doi.org/10.1542/pir.14.3.83.

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The causes of SIDS are unknown, and there are no tests that predict which infants will die of SIDS. Home monitoring, while it may prevent death in some high-risk infant populations, has not altered the overall incidence of SIDS. Studies have identified several important risk factors, such as prematurity, low birth-weight, inadequate prenatal care, maternal smoking, maternal anemia, and poor socioeconomic conditions. Other recent studies have raised important questions about child care practices, such as infant sleeping position. Still others raise equally important questions about the role of neglect or intentional injury, which now must become the subject of proper scientific investigation. The possibility that underlying metabolic defects may account for a substantial proportion of cases has been raised but requires verification. A common thread is evident: There are risk factors, such as maternal smoking and infant sleeping position, that could be subject to preventive intervention strategies. Approximately 200 new articles are published on the topic of SIDS every year. The pediatrician must have the necessary perspective to incorporate these into the "big picture." Because the main role of the pediatrician involves support, advice, and counseling of parents of present SIDS victims and future siblings, a strong knowledge base is essential. The pediatrician also can serve as an advocate for future children by insisting [See table in the PDF file] on the full and proper investigation of every SIDS case. Although we will never eliminate sudden unexplained death during childhood, we can at least hope, through better diagnosis of treatable conditions and improved preventive intervention strategies, to whittle away this diagnosis of exclusion to its minimum possible incidence.
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Beal, Susan. "Sudden Infant Death Syndrome in Twins." Pediatrics 84, no. 6 (December 1, 1989): 1038–44. http://dx.doi.org/10.1542/peds.84.6.1038.

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The incidence of sudden infant death syndrome is higher among twins than it is among singleton infants. Incidence of sudden infant death syndrome in twins in South Australia, is discussed, as well as some features of these twins and the health of the cotwins. The literature concerning the status of the cotwin is reviewed.
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Moon, R. Y., and L. Y. Fu. "Sudden Infant Death Syndrome." Pediatrics in Review 28, no. 6 (June 1, 2007): 209–14. http://dx.doi.org/10.1542/pir.28-6-209.

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Carroll, J. L., and G. M. Loughlin. "Sudden Infant Death Syndrome." Pediatrics in Review 14, no. 3 (March 1, 1993): 83–93. http://dx.doi.org/10.1542/pir.14-3-83.

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Beal, Susan M. "Sudden infant death syndrome." Medical Journal of Australia 165, no. 4 (August 1996): 179–80. http://dx.doi.org/10.5694/j.1326-5377.1996.tb124917.x.

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Banerjee, Santanu. "Sudden infant death syndrome." Indian Journal of Otolaryngology and Head and Neck Surgery 48, no. 3 (July 1996): 185. http://dx.doi.org/10.1007/bf03048599.

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Dissertations / Theses on the topic "Sudden infant death syndrome"

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Kolikof, Joshua S. "Diagnostic drift in sudden infant death syndrome." Thesis, Boston University, 2013. https://hdl.handle.net/2144/21196.

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Thesis (M.A.) PLEASE NOTE: Boston University Libraries did not receive an Authorization To Manage form for this thesis or dissertation. It is therefore not openly accessible, though it may be available by request. If you are the author or principal advisor of this work and would like to request open access for it, please contact us at open-help@bu.edu. Thank you.
INTRODUCTION: In the years that followed the 1994 Back to Sleep Campaign (BSC), a public health initiative designed to prevent Sudden Infant Death Syndrome (SIDS), the prevalence of SIDS decreased by nearly 50%. However, recent research questions the decline in SIDS with an appreciation of contemporaneous factors which may have contributed to it. There is a growing recognition that other, often indiscernible causes of Sudden Unexpected Infant Deaths (SUID) have increased prevalence rates. Several researchers have addressed the possibility of the effects of a diagnostic drift. OBJECTIVE: To evaluate the impact of certain contemporaneous factors on the decline in the prevalence of Sudden Infant Death Syndrome. METHODS: We examined a historically significant time period surrounding the implementation of the BSC, 1984-2009. It is a time-period that incorporates mortality statistics prior to the BSC, as well as immediately following. We utilized 1984-2009 mortality data obtained from the Center for Disease Control and Prevention and evaluated the following prevalence rates: SIDS, unknown and unspecified causes, Accidental Strangulation and Suffocation in Bed (ASSB), and Neglect. We then amalgamated unknown and unspecified causes, ASSB and Neglect into a single representation of non-SIDS SUID. We then proceeded to perform an analysis on these prevalence rates to determine linear trends. RESULTS: All-cause mortality rate decreased linearly by about .929 per 100,000 per year (p<.0001, β=-.929). SIDS mortality rates also experienced a significant decline of about .951 per 100,000 deaths per year (p<.0001, β=-.951). In contrast, the SUID cohort prevalence increased significantly with a rate of .930 per 100,000 per year (p<.0001, β=.930). DISCUSSION: Over our study period, SIDS declined significantly, but by 2001 it experienced a stagnant decline that was different than that from 1984-2000. From 2001 to 2009, our SUID cohort increased dramatically. It is our conclusion that the potential exists for a possible diagnostic drift from SIDS to these other SUID.
2031-01-01
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Davidson-Olsson, Isis Cherie. "Sudden Infant Death Syndrome : mothers' experiences of parenting." Thesis, University of Hertfordshire, 2013. http://hdl.handle.net/2299/10401.

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Background: The death of a child has been found to have long term consequences for both individual and family functioning. This is particularly true for bereaved siblings who have been found to be at increased risk of developing mental health difficulties in later life. Literature on parental bereavement proposes that the parenting phenomenon, such as replacement child syndrome, subsequent child syndrome and the parenting paradox, which can emerge after the death of a child, may account for this. However, there is very little research on these labels of observed parenting phenomenon and, as a result, any hypothesis offered remains under elaborated. In addition, limited evidence suggests that, due to the sudden, unexpected and unexplained nature of the loss, SIDS parents are more likely to experience a greater degree of distress and adjustment difficulties than other perinatally bereaved populations. Given this, it could be hypothesised that SIDS parents may be likely to experience these parenting phenomena. Despite this, however, SIDS remains a neglected area of research. Aims: As a consequence of this research gap, the study aims to explore mothers’ experiences of parenting in their transition from being a parent unaffected by Sudden Infant Death Syndrome to a parent affected by Sudden Infant Death Syndrome. Methodology: Semi-structured interviews were conducted with seven mothers who had experienced an incident of Sudden Infant Death Syndrome. The interviews were then transcribed and analysed using Interpretative Phenomenological Analysis (IPA). Results: Five master themes emerged from the analysis: ‘Channelling the Parent Within’, a naturally developing and responsive parenting style that is facilitated by internal mechanisms, such as flexibility and confidence; ‘Parenting Outside of Yourself’, a parenting style that develops in the aftermath of a SIDS event, which is characterised by self doubt and a reliance on external mechanisms such as reassurance and restriction; ‘Restoration Through You’, the restorative effect of the subsequent and surviving children, which allows vindication and re-establishes happiness; ‘The Bitter Restoration’, a restoration that encompasses internal knowledge and external evidence of loss, including a disrupted family composition and a continued awareness of existential threat; ‘A Disruptive Appreciation’, the development of a greater appreciation for the subsequent and surviving children that impacts discipline and incorporates indulgence. These, along with the subthemes contributing to them, are presented as a narrative account. Conclusion: The results imply that mothers who have experienced a SIDS event shift into a permissive and anxious style of parenting which is characterised by safety behaviours. A model of parenting in the aftermath of SIDS has been proposed in order to explain the underlying cognitions and processes which drive this behaviour and the factors which serve to maintain it. By doing this it is hoped that, when working with bereaved parents and siblings, clinicians will be better positioned to frame parenting practices and intervene at a cognitive level.
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Howat, William James. "The pulmonary immunopathology of sudden infant death syndrome." Thesis, University of Southampton, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.241962.

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Khorsandi, Mehdi. "Brainstem Gangliosides in Suddden Infant Death Syndrome." Thesis, North Texas State University, 1987. https://digital.library.unt.edu/ark:/67531/metadc504326/.

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Recent studies have shown that the Sudden Infant Death Syndrome (SIDS) is related to abnormal control of respiration (Ischemic degeneration of the brainstem may play an important role in altered respiratory control leading to death). In our studies we have examined brainstem ganglioside compositions in samples derived from SIDS victims and appropriate controls. Gangliosides are acidic glycosphingolipids that contain sialic acid. The high concentration of gangliosides in the central nervous system (CNS) implies that these lipids play an important role in CNS function. Some studies have indicated that gangliosides may function as receptor site determinants or modifiers, and in neural transmission. In our studies we used the Tettamanti, et al methodology to extract gangliosides, and High Performance Thin Layer Chromatography (HPTLC) and laser densitometry techniques for ganglioside analysis. The results of these analyses are being employed to establish lipid profile patterns to determine if there are significant variations in these lipid patterns between SIDS and control groups.
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Mooney, Jennifer Anne. "Statistical analysis of seasonality in sudden infant death syndrome." Thesis, University of Aberdeen, 2002. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU160368.

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SIDS deaths exhibit a seasonal pattern with a winter peak, and the cause of this seasonality is unknown. The seasonal pattern is not symmetrical and it has been thought that the relatively flat winter peak may be due to the existence of more than one underlying population, where each population corresponds to a different cause of seasonality. In this thesis, mixtures of von Mises distributions have been fitted using maximum likelihood estimation to determine whether there is heterogeneity in the UK SIDS data. Various computational problems arise with the fitting procedures and attempts to tackle these for the SIDS data are discussed. A bootstrap likelihood ratio method is used to assess the number of components in the mixture, and its properties are investigated by simulation. Changes in the seasonal pattern since the 'back to sleep' campaign are also examined as any differences might give clues as to what caused the fall in 1992, and what the reasons for the remaining deaths might be. The von Mises distributions are compared with cosinor analysis and skewed regression models to determine the most appropriate method for modelling the seasonality in the data. Mixtures of Weibull and Gamma distributions are used to model the age distribution in SIDS. The motivation for this was to determine whether there are two or more groups of babies whose age-at-death distributions are different and to examine any changes since the 'back to sleep' campaign. Generalised linear models have previously been used to determine whether month of birth is an independent risk factor in addition to month of death and age at death. In this thesis, mixtures of these generalised linear models have been fitted using the EM algorithm to determine whether there are different groups of babies with different risks. Childhood type 1 diabetes mellitus is another condition which exhibits a seasonal pattern in diagnosis. The thesis concludes by considering analysis of these data using the mixture modelling approach.
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Ottaviani, G. M. "Sudden Infant Death Syndrome (SIDS) : cardiac and neuropathological involvement." Doctoral thesis, Università degli Studi di Milano, 2004. http://hdl.handle.net/2434/53288.

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The Sudden Infant Death Syndrome (SIDS) or crib death is defined as the sudden death of an infant under one year of age which remains unexplained after a thorough case investigation, including performance of a complete autopsy, examination of the death scene, and a review of the clinical history. SIDS is the most frequent death-causing syndrome during the first year, striking 1 baby every 700-1000. Since it is a particularly tragic event for a healthy vigorous infant to die suddenly and unexpectedly, it is not surprising that the subject is of great medical and public interest. There is an association of SIDS with sleep, combined with data indicating impaired autonomic function in infants who subsequently die of SIDS, or who suffer apparent life-threatening events (ALTE), in the basis of the central cardio-pulmonary function. The pathology of SIDS includes an extended domain of neonatal pathology, particularly if within the diagnosis of SIDS one wishes to enclose the so-called "borderline" SIDS not definitely separable from the unifying concept of syndrome. SIUD (Sudden Intrauterine Unexplained Death) is the late fetal death before the complete expulsion or retraction of the fetus from the mother. Advances in maternal and fetal care have produced a significant reduction in perinatal mortality, but have not changed the prevalence of SIUD. SIUD represents about one half of the perinatal mortality, with a prevalence of 5-12 per thousand births and its etiology is largely unexplained. The knowledge of the ante and post mortem aspects of SIDS and SIUD is of international public concern, since its prevention would save a great number of potentially productive citizens. On the anatomo-pathological plan, different findings have been reported as possible SIDS substrata: brainstem abnormalities, cardiac conduction system developmental defects, immaturity of the paraganglia, hyper - or hypoplasia of the carotid bodies. Overall, the abnormalities of the autonomous nervous and cardiac conduction systems do represent a plausible substrate for a reflexogenic SIDS of vagal inhibitory nature (dive, feigned death, cardio-auditory reflexes, Ondine syndrome). Vagal cardiorespiratory reflexes, if pathological, could lead to SIDS. From 1987 to 2003, at the Institute of Pathology, University of Milan, many cases of sudden infant and fetal death have been analyzed, since the Institute is the Reference Center for the Lombardy Region for SIDS and late unexplained fetal death (DGR n° 11693 of 20/6/2002). The bill n. 396 "Autopsy regulation on SIDS and unexpected fetal death victims", proposed to the Italian Parliament and recently approved by the Senate of the Italian Republic, designs the Institute of Pathology of the University of Milan as the national reference center "being a recognized institution competent in this issue". Among an even larger amount of cases, a total of 120 SIDS victims, 37 infant controls and 60 late fetal stillborn, after the exclusion of violent causes, were selected for this work. For each analyzed case, the cardiac conduction system was removed in two blocks for paraffin embedding: block 1 (the Sino-atrial node -SAN) and the block 2 (the atrio-ventricular -AV system). The brainstem was divided into three blocks, according to the simplified procedure for the simplified brainstem examination, requiring a much lower number of sections and less work for the histotechnicians. For each brainstem nucleus (in particular, arcuate nucleus and parabrachial/Kölliker-Fuse complex), using serial sections stained with Klüver-Barrera: the neuronal cell body areas, neuronal density, transverse section areas and volume were evaluated. The statistical significance of direct comparison of the means between the two fetus groups (SIUD and stillbirth controls) and two infant groups (SIDS and infant controls) was determined by one-way analysis of variance. The carotid bifurcations, cardiac plexus ganglia and paraganglia were embedded separately and serially cut. In selected cases, immunohistochemistry studies were performed on sections of cardiac conduction system, brainstem, and coronary arteries. The SIDS cases did not show any abnormality of the ordinary myocardium, while the core of the heart, where cardiac rhythm arises and spreads, shows some abnormalities. More than one cardiac conduction change were present in the same infant, but no peculiar cardiac combined anomaly was observed. Areas of resorptive degeneration were detected in 97% of SIDS cases and in 75% of controls. Macrophages were sometimes present adjacent to the small foci of degeneration, acting eventually as scavenger cells. The developmental abnormalities of SIDS include long dendritic spines, marker of neuronal immaturity and hypoplasia of the brainstem nuclei. In SIDS the mono- or bilateral hypoplasia or even agenesis of the arcuate nucleus is particularly frequent. It has been observed and morphometrically studied in 57% of our cases - bilateral in 29/66 cases (44%) and monolateral - always the right side - in 8/66 (12%). We further considered the shape of the cell body, nucleus and nucleolus. Comparative analysis of the serial histological sections obtained from the pons and mesencephalon of SIUD and SIDS cases with those of the two control groups, made it possible to define the morphologic features of the three principal nuclei of the human PB/KF complex: Lateral parabrachial nucleus (lPB), Medial parabrachial nucleus (mPB) and Kölliker-Fuse nucleus (KF). A combined morphologic post mortem study of the cardiac conduction system and brainstem was performed in 42 cases of SIDS and in 12 controls. The Mahaim fibers were observed in 16% of control and in 17% of SIDS cases with arcuate nucleus (ARCn) histologically well developed, and from 50% (severe bilateral hypoplasia) to 71% (monolateral hypoplasia) in SIDS cases with ARCn hypoplasia. The Mahaim fibers were statistically more frequent in SIDS with ARCn hypoplasia than in the cases with well developed ARCn (control and other SIDS cases) (p<0.005). Hyperplasia of the aorticopulmonary paraganglia (APP) was detected in 25% of SIDS cases. The cell clusters and cell diameters were not significantly different from age-matched controls. Two SIDS cases showed intracapsular glomus in the left stellate ganglion and 5 cases presented inflammatory foci in the right stellate ganglion. The observation of frequent anomalies, mostly congenital, of the autonomic nervous system structures, both in SIDS and in SIUD indicates a continuity between these two pathologies. Our research upheld a new approach to SIDS by analogical link with SIUD. Indeed, early SIDS may well depend upon postnatal block of respiratory reflexes for fetus survival, warranted by Kölliker-Fuse nucleus, or upon impaired development of central circuitry for respiratory reflexogenesis. The acronym SIUD-SIDS underlines a possible common morphological substrate. The search shall be continued for a better definition of what is normal or abnormal in the infant and fetal cardiac conduction and autonomic nervous systems.
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Cowgill, Brittany M. "Rest Uneasy: Sudden Infant Death Syndrome in Twentieth-century America." University of Cincinnati / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1439282125.

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Wilson, C. Elizabeth. "The environmental niche of Aboriginal infants, possible implications for sudden infant death syndrome." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/NQ35048.pdf.

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Blair, Peter Sinclair Paul. "Assessing the changing risk factors associated with Sudden Infant Death Syndrome." Thesis, University of Bristol, 1998. http://hdl.handle.net/1983/275cdd77-5f8e-487d-8e8d-3aa62eea16e8.

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Oquendo, Javier. "Elemental Analysis of Brainstem in Victims of Sudden Infant Death Syndrome." Thesis, University of North Texas, 1988. https://digital.library.unt.edu/ark:/67531/metadc500370/.

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A brainstem-related abnormality in respiratory control appears to be one of the most compelling mechanisms for sudden infant death syndrome (SIDS). The elements calcium, copper, iron, potassium, magnesium, sodium, phosphorus, sulfur, and zinc were analyzed by inductively coupled plasma atomic emission spectroscopy in the brainstem of 30 infants who died from SIDS and 10 infants who died from other causes (control). No differences were found between SIDS and control for any element except for more calcium in the SIDS group. A multivariate analysis of the data failed to group the majority of SIDS and control subjects in different clusters. Further research is required to determine the biological significance of the higher calcium found in the SIDS group.,
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Books on the topic "Sudden infant death syndrome"

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Langwith, Jacqueline. Sudden infant death syndrome. Detroit: Greenhaven Press, 2011.

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Sawaguchi, Toshiko, ed. Sudden Infant Death Syndrome. Tokyo: Springer Japan, 2014. http://dx.doi.org/10.1007/978-4-431-54315-2.

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Parks, Peggy J. Sudden infant death syndrome. Detroit: Lucent Books, 2009.

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Oregon Memorial Library for Bereaved Parents., ed. Sudden infant death syndrome. [Bethesda, Md.]: National Institute of Child Health and Human Development, National Institutes of Health, 1997.

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Ottaviani, Giulia. Crib Death - Sudden Infant Death Syndrome (SIDS). Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-08347-6.

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United States. Maternal and Child Health Bureau, ed. Sudden Infant Death Syndrome (SIDS) infant death programs: Centers : National Sudden Infant Death Syndrome/Infant Death Program Support Center, National Center for Cultural Competence, National Sudden Infant Death Syndrome Resource Center. [Rockville, Md.?]: U.S. Dept. of Health & Human Services, Health Resources and Services Administration, Maternal and Child Health Bureau, 2000.

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United States. Maternal and Child Health Bureau., ed. Sudden Infant Death Syndrome (SIDS) infant death programs: Centers : National Sudden Infant Death Syndrome/Infant Death Program Support Center, National Center for Cultural Competence, National Sudden Infant Death Syndrome Resource Center. [Rockville, Md.?]: U.S. Dept. of Health & Human Services, Health Resources and Services Administration, Maternal and Child Health Bureau, 2000.

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D, DeFrain John, ed. Sudden infant death: Enduring theloss. Lexington, Mass: Lexington Books, 1991.

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Luben, Jacquelynn. Cot deaths: Coping with sudden infant death syndrome. London: Bedford Square, 1989.

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University of Surrey. Institute of Public Health., Share, and King's Fund Centre, eds. Postneonatal mortality and sudden infant death syndrome. [U.K.]: King's Fund Centre in collaboration with the University of Surrey, 1992.

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Book chapters on the topic "Sudden infant death syndrome"

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Russell, Charlotte K., Lane E. Volpe, and Helen L. Ball. "Sudden Infant Death Syndrome." In Evolutionary Thinking in Medicine, 61–74. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-29716-3_5.

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Jeffree, Pauline. "Sudden infant death syndrome." In The Practice Nurse, 119–24. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4899-6874-6_12.

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Oyarzún, María Angélica, and Pablo Brockmann Veloso. "Sudden Infant Death Syndrome." In Pediatric Respiratory Diseases, 495–500. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-26961-6_49.

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Kinney, Hannah C., Marco M. Hefti, Richard D. Goldstein, and Robin L. Haynes. "Sudden Infant Death Syndrome." In Developmental Neuropathology, 269–80. Oxford, UK: John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119013112.ch25.

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Leung, Alexander K. C., Cham Pion Kao, Andrew L. Wong, Alexander K. C. Leung, Thomas Kolter, Ute Schepers, Konrad Sandhoff, et al. "Sudden Infant Death Syndrome." In Encyclopedia of Molecular Mechanisms of Disease, 2003–5. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_1689.

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Kohyama, Jun. "Sudden Infant Death Syndrome and the Pedunculopontine Tegmental Nucleus." In Sudden Infant Death Syndrome, 1–13. Tokyo: Springer Japan, 2013. http://dx.doi.org/10.1007/978-4-431-54315-2_1.

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Ozawa, Yuri, Sachio Takashima, Hiroko Nonaka, and Naoki Uga. "Developmental Alteration of Hypocretins (Orexins) in the Brainstem in the Sudden Infant Death Syndrome." In Sudden Infant Death Syndrome, 15–25. Tokyo: Springer Japan, 2013. http://dx.doi.org/10.1007/978-4-431-54315-2_2.

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Franco, P., B. Kugener, S. Scaillet, J. Groswasser, I. Kato, E. Montemitro, I. Kelmanson, et al. "Sudden Infant Death Syndrome from Epidemiology to Pathophysiology: Exploring the Connections." In Sudden Infant Death Syndrome, 27–49. Tokyo: Springer Japan, 2013. http://dx.doi.org/10.1007/978-4-431-54315-2_3.

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Franco, P., J. Groswasser, S. Scaillet, B. Dan, T. Sawaguchi, I. Kato, F. Bat-Pitault, M. J. Challamel, C. Inocente, and J. S. Lin. "Sleep Deprivation in Infants, Children, and Adolescents." In Sudden Infant Death Syndrome, 51–64. Tokyo: Springer Japan, 2013. http://dx.doi.org/10.1007/978-4-431-54315-2_4.

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Franco, P., E. Montemitro, J. Groswasser, S. Scaillet, T. Sawaguchi, I. Kato, I. Kelmanson, B. Dan, A. Raoux, and J. S. Lin. "Sleep Disturbance in Children Resulting from Noise Exposure." In Sudden Infant Death Syndrome, 65–80. Tokyo: Springer Japan, 2013. http://dx.doi.org/10.1007/978-4-431-54315-2_5.

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Conference papers on the topic "Sudden infant death syndrome"

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Wei Lin, Ruikai Zhang, John Brittelli, and Craig Lehmann. "Wireless Infant Monitoring Device for the prevention of sudden infant death syndrome." In 2014 11th International Conference & Expo on Emerging Technologies for a Smarter World (CEWIT). IEEE, 2014. http://dx.doi.org/10.1109/cewit.2014.7021146.

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Ferreira, Andre G., Duarte Fernandes, Sergio Branco, Joao L. Monteiro, Jorge Cabral, Andre P. Catarino, and Ana M. Rocha. "A smart wearable system for sudden infant death syndrome monitoring." In 2016 IEEE International Conference on Industrial Technology (ICIT). IEEE, 2016. http://dx.doi.org/10.1109/icit.2016.7475060.

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Abirami, E., and S. Karthika. "Design of infant monitoring system to reduce the risk of sudden infant death syndrome." In 2017 International Conference on Communication and Signal Processing (ICCSP). IEEE, 2017. http://dx.doi.org/10.1109/iccsp.2017.8286802.

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Kitazono, Yuhki, Chihiro Nakamura, and Shota Nakashima. "Proposal of System to Prevent Sudden Infant Death Syndrome Using Kinect." In International Conference on Industrial Application Engineering 2014. The Institute of Industrial Applications Engineers, 2014. http://dx.doi.org/10.12792/iciae2014.013.

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Alkharabsheh, Abdel Rahman. "An intelligent mobile agents system for sudden infant death syndrome monitoring." In 2018 Advances in Science and Engineering Technology International Conferences (ASET). IEEE, 2018. http://dx.doi.org/10.1109/icaset.2018.8376888.

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Jung, Soonmoon, Jaemin Kim, Youngho Lee, Hyeyeong Song, Yeeun Kang, and Junghwa Hong. "DESIGN OF BABY CUSHION FOR STROLLER USING FINITE ELEMENT SIMULATION." In 2023 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2023. http://dx.doi.org/10.1115/dmd2023-7123.

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Abstract Infants spend most of the day sleeping anywhere. In the past, most parents put their infants to sleep in the prone position, but recently they put their infants to sleep in the prone position because of an issue of sudden infant death syndrome. When infants sleep in a supine position for a long time, occipital bone deformation is occurred because of pressure at the occipital region. To prevent minimize the pressure on the infant’s head and body, through the finite element simulation using the infant model, a baby cushion for the stroller was designed using the central composite design (CCD) method. The maximum contact pressure between the designed cushion and the infant model was 22.8 mmHg and this value is about 50 % lower than the pressure that occurred between the infant model and the general cushion. It can be helpful to reduce the possibility of a baby’s head deformation and other body disorders.
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Nishimura, T. H., and S. Maeda. "Evaluation of EEG in central sleep apnea for sudden infant death syndrome." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1988. http://dx.doi.org/10.1109/iembs.1988.95048.

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Leiter, James C., Donald Bartlettt, Jr., Luxi Xia, and William T. Donnelly. "The Neuropharmacology Of Apnea As It Pertains To Sudden Infant Death Syndrome." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3617.

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Gallo, Xavier Lopez, Santiago Lechon, Stalin Mora, and Diego Vallejo-Huanga. "MARRSIDS: Monitoring Assistant to Reduce the Risk of Sudden Infant Death Syndrome." In 2019 XXII Symposium on Image, Signal Processing and Artificial Vision (STSIVA). IEEE, 2019. http://dx.doi.org/10.1109/stsiva.2019.8730261.

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Rand, Casey M., Elizabeth M. Berry-Kravis, and Debra E. Weese-Mayer. "HTR4 Variation And Sudden Infant Death Syndrome (SIDS): A Case-Control Analysis." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a6553.

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Reports on the topic "Sudden infant death syndrome"

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Markowitz, Sara. The Effectiveness of Cigarette Regulations in Reducing Cases of Sudden Infant Death Syndrome. Cambridge, MA: National Bureau of Economic Research, September 2006. http://dx.doi.org/10.3386/w12527.

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Mueller, Daren, Carl Bradley, Martin Chilvers, Anna Freije, Loren Giesler, Adam Sisson, Damon Smith, Albert Tenuta, and Kiersten Wise. Sudden Death Syndrome. United States: Crop Protection Netework, February 2016. http://dx.doi.org/10.31274/cpn-20190620-028.

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Cianzio, Sylvia R., Gregory D. Gebhart, and Peter Lundeen. Soybean Sudden Death Syndrome Field Screening. Ames: Iowa State University, Digital Repository, 2009. http://dx.doi.org/10.31274/farmprogressreports-180814-2697.

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Robertson, Alison E., Daren S. Mueller, Leonor F. S. Leandro, and Stith N. Wiggs. Effect of Date of Planting on Soybean Sudden Death Syndrome. Ames: Iowa State University, Digital Repository, 2012. http://dx.doi.org/10.31274/farmprogressreports-180814-742.

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Kandel, Yuba R., Daren S. Mueller, Adam J> Sisson, Eric A. Adee, Jason P. Bond, Martin I. Chilvers, Shawn P. Conley, et al. Seed treatment and foliar fungicide impact on Sudden Death Syndrome and soybean yield. United States: Crop Protection Network, December 2019. http://dx.doi.org/10.31274/cpn-20191206-0.

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Cianzio, Sylvia, Greg Gebhart, Peter Lundeen, and Ryan Budnik. Evaluation of Soybean Commercial Varieties for Sudden Death Syndrome in Southern Iowa 2015. Ames: Iowa State University, Digital Repository, 2016. http://dx.doi.org/10.31274/farmprogressreports-180814-49.

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Cianzio, Silvia, Peter Lundeen, Ryan Budnik, and Greg Gebhart. Evaluation of Commercial Varieties in the Northern United States–Soybean Sudden Death Syndrome Commercial Variety Test. Ames: Iowa State University, Digital Repository, 2015. http://dx.doi.org/10.31274/farmprogressreports-180814-2329.

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Scouting for Sudden Death Syndrome in Soybean. United States: Crop Protection Netework, November 2016. http://dx.doi.org/10.31274/cpn-20190620-021.

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