Academic literature on the topic 'Sucrose – Physiological effect'
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Journal articles on the topic "Sucrose – Physiological effect"
Philippou, Koumis, James Ronald, Alfredo Sánchez-Villarreal, Amanda M. Davis, and Seth J. Davis. "Physiological and Genetic Dissection of Sucrose Inputs to the Arabidopsis thaliana Circadian System." Genes 10, no. 5 (May 2, 2019): 334. http://dx.doi.org/10.3390/genes10050334.
Full textMartinez-Levy, Ana C., Elisabetta Moneta, Dario Rossi, Arianna Trettel, Marina Peparaio, Eleonora Saggia Civitelli, Gianluca Di Flumeri, Patrizia Cherubino, Fabio Babiloni, and Fiorella Sinesio. "Taste Responses to Chocolate Pudding with Different Sucrose Concentrations through Physiological and Explicit Self-Reported Measures." Foods 10, no. 7 (July 2, 2021): 1527. http://dx.doi.org/10.3390/foods10071527.
Full textTarelkina, Tatiana V., Ludmila L. Novitskaya, Nadezhda N. Nikolaeva, and Veronica De Micco. "Effect of sucrose exposure on the xylem anatomy of three temperate species." IAWA Journal 39, no. 2 (June 13, 2018): 156–76. http://dx.doi.org/10.1163/22941932-20170198.
Full textFerguson, L. V., N. H. Beckett, M. C. French, M. J. Campbell, T. G. Smith, and S. A. Adamo. "Sugar intake interacts with temperature to influence reproduction and immunity in adult Culex pipiens mosquitoes." Canadian Journal of Zoology 97, no. 5 (May 2019): 424–28. http://dx.doi.org/10.1139/cjz-2018-0129.
Full textSathya Devi, V., Obiora O. Chidi, and Denis Coleman. "Dominant effect of ethanol in thermal destabilization of bovine serum albumin in the presence of sucrose." Spectroscopy 23, no. 5-6 (2009): 265–70. http://dx.doi.org/10.1155/2009/423547.
Full textErin, Nindi Novia, Arif Yachya, Alfinda Novi Kristanti, Djarot Sugiarso, and Yosephine Sri Wulan Manuhara. "Effect of Carbon Source Variations on Growth, Physiological Stress, and Saponin Levels of <i>Talinum paniculatum</i> Gaertn. Adventitious Roots." Journal of Tropical Biodiversity and Biotechnology 7, no. 3 (September 23, 2022): 69359. http://dx.doi.org/10.22146/jtbb.69359.
Full textDavis, J. D., and G. P. Smith. "Learning to sham feed: behavioral adjustments to loss of physiological postingestional stimuli." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 259, no. 6 (December 1, 1990): R1228—R1235. http://dx.doi.org/10.1152/ajpregu.1990.259.6.r1228.
Full textGago, Diego, María Ángeles Bernal, Conchi Sánchez, Anxela Aldrey, Beatriz Cuenca, Colin Bruce Christie, and Nieves Vidal. "Effect of Sucrose on Growth and Stress Status of Castanea sativa x C. crenata Shoots Cultured in Liquid Medium." Plants 11, no. 7 (April 1, 2022): 965. http://dx.doi.org/10.3390/plants11070965.
Full textDu, Yanli, Qiang Zhao, Liru Chen, Xingdong Yao, Huijun Zhang, Junjiang Wu, and Futi Xie. "Effect of Drought Stress during Soybean R2–R6 Growth Stages on Sucrose Metabolism in Leaf and Seed." International Journal of Molecular Sciences 21, no. 2 (January 17, 2020): 618. http://dx.doi.org/10.3390/ijms21020618.
Full textHandriani, Irda, Ika Kusuma Nugraheni, and Mariatul Kiptiah. "The effect of sucrose concentration on the transparency of solid soap-based cooking oil." Jurnal Pijar Mipa 17, no. 4 (July 31, 2022): 533–37. http://dx.doi.org/10.29303/jpm.v17i4.3703.
Full textDissertations / Theses on the topic "Sucrose – Physiological effect"
Zacchia, Camillo. "The effects of tryptophan and sucrose on alcohol-induced impairment /." Thesis, McGill University, 1987. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=75345.
Full textStudy II examined the combined effects of sucrose and alcohol in a 3 (100 g sugar, 35 g sugar, 0 g sugar) x 3 (alcohol, placebo, sober) design with 15 subjects per cell. Subjects were tested on tasks similar to those used in Study I at a variety of times following intoxication (i.e., 0.5 hours, 1.5 h, 3.5 h) in order to examine effects when blood glucose peaked as well as at a point when hypoglycemic rebound can occur in some subjects. A strong alcohol x sugar interaction was seen 0.5 h after drinking, with high doses of sugar attenuating intoxication without influencing blood alcohol levels. No hypoglycemia was produced after 3.5 h.
Study III replicated Study II using a simpler design which controlled for the possible effects of aspartame (the placebo sweetener used in Study II). A variety of gastric or central mechanisms, which can account for the finding that sucrose can reduce the intoxicating effects of ethanol, are discussed.
Dalidjan, Mulyani. "Caries inhibitory effect of fluoride co-crystallized sucrose : establishing a field trial /." Title page, contents and summary only, 1995. http://web4.library.adelaide.edu.au/theses/09PH/09phd143.pdf.
Full textWendt, Ellen B. "Comparison of fat free and regular potato chips : taste acceptability and gastrointestinal symptoms in 18-21 year-old female college students." Virtual Press, 2000. http://liblink.bsu.edu/uhtbin/catkey/1178356.
Full textVerplaetse, Terril Lee. "Effects of Prazosin Treatment on Ethanol- and Sucrose-Seeking and Intake in P Rats." Thesis, 2012. http://hdl.handle.net/1805/2970.
Full textBackground: Previous studies show that prazosin, an α1-adrenergic receptor antagonist, decreases alcohol drinking in animal models of alcohol use and dependence and in alcohol-dependent men. These studies extended previous findings by using a paradigm that allows for separate assessment of prazosin on motivation to seek versus consume ethanol or sucrose in selectively bred rats given acute or chronic prazosin treatment. Methods: Alcohol-preferring P rats were trained to complete an operant response that resulted in access to either 2% (Exp. 1) or 1% (Exp.2) sucrose or 10% ethanol. In Experiment 1, a 4-week consummatory testing phase consisted of rats bar-pressing to “pay” a specified amount up front to gain access to unlimited ethanol (or sucrose) for a 20-minute period. A 4-week appetitive testing phase examined how much the rats would bar-press for ethanol in an extinction session when no reinforcer could be obtained. In Experiment 2, during testing, the response requirement was dropped to a 1 and daily session cycles of drug (3 weeks/ 14 sessions from Tues to Fri) or vehicle (2 weeks/ 9 sessions from Tues to Fri) treatment were alternated per drug dose for a total of 3 drug doses (3 cycles) per rat. After each drug cycle, a single non-reinforced extinction session was conducted with no drug ‘on board’ and no reinforcer access. On test days, rats were given IP injections of either vehicle or one of three doses of prazosin (Exp 1: 0.5, 1.0, 1.5 mg/kg; Exp 2: 0.25, 0.5, 1.0 mg/kg; balanced design; -30 min). Results: In Experiment 1, prazosin significantly decreased ethanol-seeking at all doses tested. The highest dose decreased ethanol intake and increased the latency to first lever-press and first lick. Sucrose-seeking and intake were decreased by the same doses of prazosin. In Experiment 2, prazosin significantly decreased reinforcer-seeking at the lowest and highest doses while ethanol intake was not decreased by prazosin. Conversely, sucrose-seeking was decreased at the highest dose of prazosin tested while sucrose consumption was decreased by all doses. Latency to lever-press for sucrose was increased by the lowest dose of prazosin compared to vehicle. Conclusions: These findings extend previous research and indicate that prazosin decreases motivation to seek ethanol and sucrose. The specificity of prazosin on different behaviors and over different reinforcers suggests that these findings are not due to prazosin-induced motor-impairment or malaise. These data suggest that prazosin may work by decreasing the reinforcing properties of reinforcers in general.
Windisch, Kyle Allyson. "ACHIEVING PHARMACOLOGICALLY RELEVANT IV ALCOHOL SELF-ADMINISTRATION IN THE RAT." Thesis, 2012. http://hdl.handle.net/1805/2978.
Full textAlcohol consumption produces a complex array of effects that can be divided into two types: the explicit pharmacological effects of ethanol (which can be quite separate temporally from time of intake) and the more temporally “relevant” effects (primarily olfactory and taste) that bridge the time from intake to the onset of the pharmacological effects. Dissociating these effects is essential to untangling the neurologic underpinnings of alcohol abuse and dependence. Intravenous self-administration of ethanol allows for controlled and precise dosing, bypasses first order absorption kinetics allowing for a faster onset of pharmacologic effects, and eliminates the confounding “non-pharmacological” effects associated with oral consumption. Intravenous self-administration of ethanol has been reliably demonstrated in both mouse and human experimental models; however, consistent intravenous self-administration of pharmacologically relevant levels of ethanol remains elusive in the rat. Previous work has demonstrated reliable elevated intravenous ethanol self administration using a compound reinforcer of oral sucrose and intravenous ethanol. The present study sought to elucidate the role of each component of this reinforcer complex using a multiple schedule study design. Male P rats had free access to both food and water during all intravenous self-administration sessions and all testing was performed in conjunction with the onset of the dark cycle. Once animals achieved stable operant responding on both levers for an orally delivered 1% sucrose solution (1S) on a FR4 schedule, surgery was conducted to implant an indwelling jugular catheter. Animals were habituated to the attachment of infusion apparatus and received twice daily sessions for four days to condition each lever to its associated schedule. Animals were then trained to respond on a multiple FR4-FR4 schedule composed of alternating 2.5 minute components. During one component only oral 1S was presented, while in the second component a compound reinforcer of oral 1S + IV 20% ethanol was presented (25 mg/kg/injection). Both levers were extended into the chamber during the session, with the active lever/schedule alternating as the session progressed across components. Average ethanol intake was 0.47 ± 0.04 g/kg. A significant increase in sucrose only reinforcers and sucrose lever error responding was found suggesting that sucrose not ethanol is responsible for driving overall responding. The current findings suggest that the existing intravenous ethanol self-administration methodology remains aversive in the rat.
Addington, Elizabeth Elaine. "Pre-exercise feedings of glucose, fructose, or sucrose: effects on fuel homeostasis in rats." 1985. http://hdl.handle.net/2097/27384.
Full textSung, Lung-Ying, and 宋龍瑛. "Effects of sucrose pretreatment and cold acclimation on the tolerance to vitrification solution (PVS2) and the changes of physiological properties of cassava." Thesis, 2001. http://ndltd.ncl.edu.tw/handle/95520113600145024506.
Full text國立中興大學
植物學系
89
This study investigates the suitable period of cold acclimation for cryopreservation of cassava (Manihot esculenta Crantz). Measurement of several physiological parameters showed that cassava shoot tips treated with 0.3M sucrose in accompanied with cold acclimation at 15℃ for one to two weeks enhanced the tolerance to vitrification solution (PVS2) used for cryopreservation. Ion leakage and MDA production of cassava shoot tips decreased during one to two weeks acclimation period. The results suggested that lipid peroxidation declined and the structure of membrane acclimated to low temperature. As the time of cold treatment persisted, ion leakage and MDA production rose gradually suggesting the damage of membrane system by prolonged low temperature treatments. System of cellular antioxidation cannot be activated by cold acclimation successfully. With the exception of the activity of SOD increasing continuously, the activities of APX, MDHAR, DHAR and GR did not increase along with the extending period of cold acclimation. As the results from the activities of antioxidative enzymes, antioxidant system of cassava shoot tips did not effectively scavenge the reactive oxygen radicals generated by low temperature treatment including the ability of scavenging H2O2 and generation of ascorbate. This may be one of the reasons that leaded to the decreased tolerance to PVS2 after cold acclimation for more than two weeks. The accumulation of osmoregulatory solutes leaded to the decrease of osmotic potential. The solutes accumulated include sucrose, proline and soluble protein. The time course of sucrose and proline accumulation under low temperature suggested that the most suitable period of cold acclimation treatment is at one to two weeks at 15℃, that match the period of the membrane to adapt low temperature. Sucrose and proline may have the ability to stabilized the structure of proteins and membrane under low temperature but the trends of increase was not steady. Based on the results shown above, treatment of 0.3 M sucrose and cold acclimation one to two weeks at 15℃ can enhance the tolerance to vitrification solution (PVS2) of cryopreservation. The results presented here can be used as a model for pretreatment of cassava cryopreservation.
Books on the topic "Sucrose – Physiological effect"
1928-, Vettorazzi Gaston, and Macdonald Ian 1921-, eds. Sucrose: Nutritional and safety aspects. London: Springer-Verlag, 1988.
Find full textBoutte, Troy Thomas. Methyl glucose and sucrose polyester: Feeding studies and interactions with supercritical carbon dioxide. 1993.
Find full textHollingbery, Patsy Whitmer. Acute effects of dietary caffeine, aspirin and sucrose on urinary mineral excretion in adult women and adolescents. 1986.
Find full textM, Wheeler Raymond, Weigel Russell C, and United States. National Aeronautics and Space Administration., eds. Effect of irradiance, sucrose, and CO₂ concentration on the growth of potato (Solanum tuberosum L.) in vitro. [Washington, DC: National Aeronautics and Space Administration, 1995.
Find full textRippe, James M. Fructose, High Fructose Corn Syrup, Sucrose and Health. Humana, 2014.
Find full textRippe, James M. Fructose, High Fructose Corn Syrup, Sucrose and Health. Humana, 2016.
Find full textRippe, James M. Fructose, High Fructose Corn Syrup, Sucrose and Health. Humana Press, 2014.
Find full textBrick, Scott, and Michael Moss. Salt Sugar Fat: How the Food Giants Hooked Us. Brand: Random House Audio, 2013.
Find full textMoss, Michael. Salt Sugar Fat: How the Food Giants Hooked Us. Ebury Publishing, 2014.
Find full textMoss, Michael. Salt Sugar Fat: How the Food Giants Hooked Us. Penguin Random House, 2013.
Find full textReports on the topic "Sucrose – Physiological effect"
Uni, Zehava, and Peter Ferket. Enhancement of development of broilers and poults by in ovo feeding. United States Department of Agriculture, May 2006. http://dx.doi.org/10.32747/2006.7695878.bard.
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