Relevant bibliographies by topics / Subthalamic nucleu

Academic literature on the topic 'Subthalamic nucleu'

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Published: 11 March 2023

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Journal articles on the topic "Subthalamic nucleu"

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Barlas, Orhan, Ha?met A. Hana?as?, Murat ?mer, H�seyin A. ?ahin, Serra Sencer, and Murat Emre. "Do unilateral ablative lesions of the subthalamic nucleu in parkinsonian patients lead to hemiballism?" Movement Disorders 16, no. 2 (2001): 306–10. http://dx.doi.org/10.1002/mds.1051.

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Nakano, Naoki, Mamoru Taneda, Akira Watanabe, and Amami Kato. "Computed Three-Dimensional Atlas of Subthalamic Nucleus and Its Adjacent Structures for Deep Brain Stimulation in Parkinson's Disease." ISRN Neurology 2012 (January 12, 2012): 1–13. http://dx.doi.org/10.5402/2012/592678.

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Background. Deep brain stimulation (DBS) of the subthalamic nucleus (STN) is one of the standard surgical treatments for advanced Parkinson's disease. However, it has been difficult to accurately localize the stimulated contact area of the electrode in the subthalamic nucleus and its adjacent structures using a two-dimensional atlas. The goal of this study is to verify the real and detailed localization of stimulated contact of the DBS electrode therapeutically inserted into the STN and its adjacent structures using a novel computed three-dimensional atlas built by a personal computer. Method. A three-dimensional atlas of the STN and its adjacent structures (3D-Subthalamus atlas) was elaborated on the basis of sagittal slices from the Schaltenbrand and Wahren stereotactic atlas on a personal computer utilizing a commercial software. The electrode inserted into the STN and its adjacent structures was superimposed on our 3D-Subthalamus atlas based on intraoperative third ventriculography in 11 cases. Findings. Accurate localization of the DBS electrode was identified using the 3D-Subthalamus atlas, and its clinical efficacy of the electrode stimulation was investigated in all 11 cases. Conclusion. This study demonstrates that the 3D-Subthalamus atlas is a useful tool for understanding the morphology of deep brain structures and for the precise anatomical position findings of the stimulated contact of a DBS electrode. The clinical analysis using the 3D atlas supports the contention that the stimulation of structures adjacent to the STN, particularly the zona incerta or the field of Forel H, is as effective as the stimulation of the STN itself for the treatment of advanced Parkinson's disease.
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Etemadifar, Masoud, Seyed-Hossein Abtahi, Seyed-Mojtaba Abtahi, Motahreh Mirdamadi, Sepideh Sajjadi, Aryan Golabbakhsh, Mohammad-Reza Savoj, Mahboobeh Fereidan-Esfahani, Zahra Nasr, and Nasim Tabrizi. "Hemiballismus, Hyperphagia, and Behavioral Changes following Subthalamic Infarct." Case Reports in Medicine 2012 (2012): 1–4. http://dx.doi.org/10.1155/2012/768580.

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The function of subthalamic nucleus (STN) which is a part of the basal ganglia system is not clear, but it is hypothesized that this component might be involved in action selection. Unilateral damage to STN, which can commonly occur due to the small vessel stroke mainly, causes hemiballismus and sometimes hemichorea-hemiballismus. This paper deals with a 60-year-old patient with sudden onset of abnormal movements in his right limbs. He had increased appetite and hyperphagia and also developed mood and behavioral changes (aggressiveness, irritability, anxiety, and sometimes obscene speech). The magnetic resonance imaging revealed infarct area in left subthalamus. In our case, hemiballismus is caused by infarction in left subthalamic area. Occurrence of irritability, anxiety, and some behavioral changes such as aggressiveness and obscene speech can be explained by impairment of STN role in nonmotor behavior and cognitive function as a result of infarct.
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Magalhães, Marcelo José Silva de, Claudiojanes dos Reis, Juliana Rabelo da Silva Sousa, Victória Souza Marques, Tayná Cardoso Gonçalves, Iara Cristina Vieira Ribeiro, Leide Daiana Silveira Cardoso, Victor Caribé Crosland Guimarães, Frederico Gustavo de Souza Marques, and Sarah Dias Pereira. "Subthalamic Nucleus: Neuroanatomical Review." Arquivos Brasileiros de Neurocirurgia: Brazilian Neurosurgery 39, no. 04 (December 18, 2017): 284–88. http://dx.doi.org/10.1055/s-0037-1615268.

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AbstractDiscovered in 1865 by Jules Bernard Luys, the subthalamic nucleus is a set of small nuclei located in the diencephalon, inferior to the thalamus and superior to the substantia nigra, that can be visualized in a posterior coronal section. Histologically, it consists of neurons compactly distributed and filled with a large number of blood vessels and sparse myelinated fibers. This review presents an analysis of this anatomical region, considering what is most recent in the literature. Subthalamic neurons are excitatory and use glutamate as the neurotransmitter. In healthy individuals, these neurons are inhibited by nerve cells located in the side globus pallidus. However, if the fibers that make up the afferent circuit are damaged, the neurons become highly excitable, thus causing motor disturbances that can be classified as hyperkinetic, for example ballism and chorea, or hypokinetic, for example Parkinson disease (PD). The advent of deep brain stimulation has given the subthalamic nucleus great visibility. Studies reveal that the stimulation of this nucleus improves the motor symptoms of PD.
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Velíšková, Jana, Libor Velšek, and Solomon L. Moshé. "Subthalamic nucleus." NeuroReport 7, no. 11 (July 1996): 1786–88. http://dx.doi.org/10.1097/00001756-199607290-00019.

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López-Flores, Gerardo, Juan Miguel-Morales, Juan Teijeiro-Amador, Jerold Vitek, Sahily Perez-Parra, Ramsés Fernández-Melo, Carlos Maragoto, et al. "Anatomic and Neurophysiological Methods for the Targeting and Lesioning of the Subthalamic Nucleus: Cuban Experience and Review." Neurosurgery 52, no. 4 (April 1, 2003): 817–31. http://dx.doi.org/10.1227/01.neu.0000053224.16728.7d.

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Abstract OBJECTIVE To develop a method to place a lesion precisely in the subthalamic nucleus (STN) and evaluate its effectiveness. METHODS A retrospective study of targeting data collected during stereotactic planning to lesion the STN in 31 patients with Parkinson's disease and of results in more than 50 procedures was performed. The targeting method was based on computed tomographic imaging together with semimicroelectrode recording digital processing and electrical stimulation. Two statistical methods were used to correlate initial with final target coordinates and assess the efficacy of the targeting procedure. RESULTS The anatomic target based on computed tomographic imaging data showed electrical activity in the subthalamus in the first pass in 82% of the procedures. In the remaining 18%, the STN was an average of 1.93 mm away from the nearest trajectory that recorded the STN (range, 1.41–2.24 mm). The average number of trajectories per procedure was 7.2; the location of the first trajectory relative to the center of the nucleus determined by electrical and physiological means (P< 0.01, analysis of variance, Student's ttest) was as follows: in the lateral direction, 1.25 ± 1.15 mm; in the anteroposterior direction, 1.53 ± 1.31 mm; and in the vertical direction, 0.67 ± 0.51 mm. The average number of tracts necessary to lesion the STN was two. CONCLUSION The combination of computed tomographic imaging, semimicroelectrode recording, and microstimulation provides an effective method to identify the STN lesion in parkinsonian patients. The method used for anatomic localization and electrophysiological mapping of the subthalamus was found to be effective in reaching the sensorimotor region of the nucleus. We carried out an accurate determination of the subthalamus location and its volume in the lesioning.
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Blandini, Fabio, Giuseppe Conti, Emilia Martignoni, Vittorio Colangelo, Giuseppe Nappi, Renato Di Grezia, and Francesco Orzi. "Modifications of Local Cerebral Metabolic Rates for Glucose and Motor Behavior in Rats with Unilateral Lesion of the Subthalamic Nucleus." Journal of Cerebral Blood Flow & Metabolism 19, no. 2 (February 1999): 149–54. http://dx.doi.org/10.1097/00004647-199902000-00006.

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Inactivation of the subthalamic nucleus (STN) has attracted interest as a therapeutic tool in Parkinson's disease. The functional consequences of the inactivation, however, are uncertain. In this study definition of the pattern of changes of cerebral functional activity associated with lesion of the STN and dopaminergic stimulation, by using the [14C]deoxyglucose method, was sought. Six or 7 days following unilateral lesion of the STN, the animals were divided into two groups: One group (n = 10) was administered apomorphine (1 mg/kg) subcutaneously; the second group (n = 10) received saline. The [14C]deoxyglucose procedure was initiated 10 minutes following the drug or saline injection. The results show that systemic administration of apomorphine to rats with unilateral lesion of the STN causes ipsiversive rotational behavior and asymmetries of glucose utilization of defined brain areas, including the substantia nigra reticulata, globus pallidus, and entopeduncular nucleus. These nuclei are the main targets of the subthalamic excitatory projections. Lesion of the nucleus per se (without challenge with apomorphine) has no significant consequences on glucose utilization. The findings indicate that the STN is involved in the activation of the basal ganglia output nuclei induced by systemic dopaminergic stimulation.
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Young, Geoffrey S., Feng Feng, Hao Shen, and Nan-kuei Chen. "SUSCEPTIBILITY-ENHANCED 3-TESLA T1-WEIGHTED SPOILED GRADIENT ECHO OF THE MIDBRAIN NUCLEI FOR GUIDANCE OF DEEP BRAIN STIMULATION IMPLANTATION." Neurosurgery 65, no. 4 (October 1, 2009): 809–15. http://dx.doi.org/10.1227/01.neu.0000345354.21320.d1.

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Abstract SURGICAL PLANNING FOR deep brain stimulation implantation procedures requires T1-weighted imaging (T1WI) for stereotactic navigation. Because the subthalamic nucleus, the main target for deep brain stimulation, and other midbrain nuclei cannot be visualized on the stereotactic guidance T1WI, additional T2-weighted imaging (T2WI) is generally obtained and registered to the T1WI for surgical targeting. Surgical planning based on the registration of the 2 data sets is subject to error resulting from inconsistent geometric distortions and any subject movement between the 2 scans. In this article, we propose a new method to produce susceptibility-enhanced, contrast-optimized T1-weighted 3-dimensional spoiled gradient recalled acquisition in steady state images with enhanced contrast for midbrain nuclei within the volumetric T1WI data set itself, eliminating the need for additional T2WI. The scan parameters of 3-dimensional spoiled gradient recalled acquisition in steady state are chosen in a way that T1WI can be obtained from conventional magnitude reconstruction and images with improved contrast between midbrain nuclei and surrounding tissues can be produced from the same data by performing susceptibility-weighted imaging reconstruction on a chosen region of interest. In addition, our preliminary experience suggests that the resulting contrast between the midbrain nuclei is superior to the current state-of-the-art fast spin echo T2WI in depicting the subthalamic nucleus as distinct from the substantia nigra pars reticulata and clear depiction of the nucleus ventrointermedius externus of thalamus.
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Sañudo-Peña, M. Clara, and J. Michael Walker. "Role of the Subthalamic Nucleus in Cannabinoid Actions in the Substantia Nigra of the Rat." Journal of Neurophysiology 77, no. 3 (March 1, 1997): 1635–38. http://dx.doi.org/10.1152/jn.1997.77.3.1635.

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Sañudo-Peña, M. Clara and J. Michael Walker. Role of the subthalamic nucleus in cannabinoid actions in the substantia nigra of the rat. J. Neurophysiol. 77: 1635–1638, 1997. The effect of cannabinoids on the excitatory input to the substantia nigra reticulata (SNr) from the subthalamic nucleus was explored. For this purpose a knife cut was performed rostral to the subthalamic nucleus to isolate the subthalamic nucleus and the SNr from the striatum, a major source of cannabinoid receptors to the SNr. The data showed that the cannabinoid agonist WIN55,212-2 blocked the increase in the firing rate of SNr neurons induced by stimulation of the subthalamic nucleus with bicuculline. Furthermore, the cannabinoid antagonist SR141716A antagonized the effect of the cannabinoid agonist. This study showed that cannabinoids regulate not only the striatonigral pathway, as previously reported, but also the subthalamonigral pathway. The opposite influences of these two inputs to the SNr, inhibitory and excitatory respectively, suggest that endogenous cannabinoids play a major role in the physiological regulation of the SNr.
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Moro, Elena, Yu-Yan W. Poon, Andres M. Lozano, Jean A. Saint-Cyr, and Anthony E. Lang. "Subthalamic Nucleus Stimulation." Archives of Neurology 63, no. 9 (September 1, 2006): 1266. http://dx.doi.org/10.1001/archneur.63.9.1266.

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Dissertations / Theses on the topic "Subthalamic nucleu"

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Atherton, Jeremy Francis. "Neurophysiology of the subthalamic nucleus." Thesis, University of Edinburgh, 2001. http://hdl.handle.net/1842/29793.

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Possibly as many as half the neurones in the STN have an axon collateral with branches off from the main axon and reinnervates the nucleus. This suggests that rather than working autonomously as was previously thought, the neurones of the STN can operate together as a network. Computer models of the STN showed that the level of interconnectivity within the STN would be huge, even if each axon collateral only contacted a small number of the total neurones with dendritic fields that overlapped with it. A network model showed that such a system was capable of switch-like behaviour. At low levels of activity the neurones would act autonomously. However, excitatory inputs could increase the degree of non-synchronous correlation between the activity of neurones in the STN leading them all to enter a high activity state. A single cell model was then developed in order to look at how this high activity state could be terminated. An interesting problem arose in the construction of this model; no known kinetics for the voltage-gated sodium and potassium channels could replicate the high frequency (500Hz) firing rates that are obtained by STN neurones. Intracellular recordings were made in vitro to investigate the mechanisms underlying high-frequency firing in the STN. Using a two-pulse protocol the speed of recovery from inactivation was measured giving an estimate of the inactivation characteristics of the ion channels in these neurones. These experiments showed that the neurones have very slow inactivation kinetics suggesting that STN neurones may have a much shortened refractory period, enabling high frequency firing. Such a mode of operation requires a large, fast potassium current. A potential candidate for this current is the Kv3.l potassium channel, which is strongly expressed by STN neurones.
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Clarke, Nicholas Paul. "Neuronal microcircuits of the entopenuncular nucleus and subthalamic nucleus." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.388564.

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Zhang, Yu. "The subthalamic nucleus in health and disease." Thesis, Uppsala universitet, Institutionen för biologisk grundutbildning, 2020. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-410033.

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Parkinson’s disease (PD) is the second most common neurodegenerative disorder in the world. PD is caused by degeneration of the dopaminergic neurons in the substantia nigra pars compacta (SNc). Deep brain stimulation (DBS) is a surgical therapy used in PD to alleviate motor dysfunction by application of high-frequency stimulation through implanted electrodes. STN is an important target of DBS electrodes in PD treatment. However, a series of side effects have been reported upon STN-DBS treatment, and the reliability of the method could be clinically improved. To achieve this, anatomical and functional studies in mice can contribute important knowledge. There are different models to explain the internal STN organization, each of which has experimental evidence. Previous work has shown that the Vesicular glutamate transporter 2 (Vglut2) and Paired-box homeodomain transcription factor 2 (Pitx2) genes are needed for normal development and function of the STN in mice. These genes are expressed throughout the STN and their use as markers for STN neurons has enabled functional studies. To progress, more knowledge of the internal organization of the STN would be useful. Here, three antibodies representing three potential STN markers were tested using immunohistochemistry. PCR analysis was used to genotype Pitx2-Cre transgenic mice that are currently used for functional and behavioral STN studies.
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Xia, Shuang. "The contribution of the subthalamic nucleus to executive functions in rat." Thesis, University of St Andrews, 2014. http://hdl.handle.net/10023/5545.

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Lesions of the subthalamic nucleus (STN) alleviate the cardinal signs of idiopathic as well as MPTP-induced Parkinson's disease in primates. For this reason, the STN is a target for clinical treatment of Parkinson's disease using deep brain stimulation. Despite its small size, the STN plays a vital role in the cortico-basal ganglia-thalamic network. However, the functional features of the STN have yet to be fully uncovered. The research presented in this thesis examines the functions of the STN by measuring behavioural changes resulting from STN lesions in rats performing executive abilities. In the first experiment, a ‘signal change' reaction time task was developed and the performance of humans and rats was compared. The main findings were that although humans and rats used different strategies in the task, the task did challenge the ability to inhibit unwanted responses. In the second and third experiments, the effects of bilateral lesions of the STN on performance of two variants of the ‘signal change' task were examined. Rats with the STN lesions were able to inhibit responses when under stimulus control, but were less able to inhibit responses that were not under stimulus control. In the final experiment, the effects of lesions of the STN on inhibitory control in a nonmotor, cognitive domain were examined. Rats with STN lesions were not impaired on reversal learning, suggesting intact inhibition of previously rewarded responses. The rats with STN lesions did show impairments in selective attention which resulted in an inability to form an attentional set. Together, these findings challenge the conventional view that the STN simply plays a global inhibitory role. Rather, the contribution of the STN to inhibitory control is more complex and neither the motor nor the cognitive effects of the lesions are easily explained simply as a failure of inhibition.
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Temel, Yasin. "The subthalamic nucleus a novel motor-associative-limbic interface /." Maastricht : Maastricht : Universiteit Maastricht ; University Library, Universiteit Maastricht [host], 2007. http://arno.unimaas.nl/show.cgi?fid=7484.

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Wallace, Bradley Andrew. "The neuroprotective effects of subthalamic nucleous (STN) Suppression." Université Joseph Fourier (Grenoble), 2004. http://www.theses.fr/2004GRE10072.

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La maladie de Parkinson est une pathologie neurodégénérative progressive qui affecte environ 0. 1 % de la population âgée de plus de 40 ans. Elle trouve son origine dans la mort des neurones dopaminergiques de la substantia nigra compacta (SNc), avec pour conséquence une déplétion en dopamine dans le striatum. Le traitement de référence à la levodopa, efficace pour diminuer les symptômes, se révèle toutefois incapable de limiter la progression de la maladie. Un nombre important de données récentes identifie un dysfonctionnement de la chaîne de transport d'électrons du complexe I mitochondrial comme élément clé de la physiopathologie de la maladie de Parkinson. Les conséquences de cette déficience se traduisent au niveau cellulaire par une diminution de la production d'énergie (ATP) compromettant le maintien du potentiel de repos membranaire neuronal. La dépolarisation du potentiel de repos membranaire libère progressivement le blocage des récepteurs NMDA par les ions Mg2+, réduisant le seuil d'activation par le glutamate. Il en résulte une augmentation significative de la vulnérabilité des neurones dopaminergiques à l'excitotoxicité par le glutamate. Ce processus est appelé excitotoxicité "indirecte", et contrairement à la classique et plus aigue͏̈ excitotoxicité "directe", il pourrait contribuer à la disparition progressive des neurones de la SNc. De nombreuses structures comme le SNc reçoivent du glumatamate à partir des neurones du noyau subthalamique (NST). Selon le modèle actuel des circuits des ganglions de la base, le NST est hyperactif. Bien qu'il ne soit pas indispensable au déclenchement de l'excitotoxicité indirecte, l'accroissement de l'activité (output) glutamatergique exacerbe l'excitotoxicité [. . . ]
Parkinson's disease (PD) is a progressive neurodegenerative disease affecting 0. 1% of the population over 40 years old. The hallmark of PD pathology is reduced striatal dopamine secondary to the death of dopaminergic neurons in the substantia pars compacta (SNc). The mainstay of treatment, levodopa replacement therapy, is severely limited by side effects and although effective in alleviating symptoms, electron transport chain as a key process in the pathophysiology underlying PD. Complex I defects result cellular energy (ATP) production, imparaing the ability of neurons to maintain their resting membrane potentials (RMP). Depolarization of the RMP relieves the Mg2+blockade of NMDA receptors in a graded fashion, reducing the threshold for activation by glutamate. The result is a significant increase in the vulnerability of SNc dopaminergic neurons to glutamate excitotoxicity. This has been referred to as "indirect" excitotoxicity, that unlike the more acute, classic or "direct" excitotoxicity, may contribute to the demise of SNc neurons on a chronic basis. The subthalamic nucleus (STN) projects glutamate to multiple structures, including the SNc. According to the current model of basal ganglia circuitry, the STN is hyperactive. Althought not essential for indirect excitotoxicity to occur, increased glutamatergic output exacerbates the excitotoxic process [. . . ]
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Gillies, Andrew J. "The role of the subthalamic nucleus in the basal ganglia." Thesis, University of Edinburgh, 1995. http://hdl.handle.net/1842/522.

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The basal ganglia are a collection of interconnected subcortical nuclei which have been implicated inmotor, cognitive and limbic functions. The subthalamic nucleus is the sole excitatory structure within the basal ganglia. Given its central position influencingmany basal ganglia nuclei, it is likely to play an important role in the processing that is performed by the basal ganglia. In this thesis a theoretical analysis of the subthalamic nucleus is presented. In order to explore the multiple facets of processing that may be occurring, models that are designed to capture aspects of the subthalamic nucleus at different levels are developed. These include anatomical, network processing and single neuron multi–compartmental models. Through the integration of the results obtained from these models a new and coherent view of the processing of the subthalamic nucleus is presented. It is predicted that the subthalamic nucleus be considered as a massively connected excitatory network. Two distinct modes of asymptotic behaviour exist in such a network: a low resting state and a high self–sustained state. The single neuron multi– compartmental model demonstrates that the calcium T–type channel is the primary determinant of characteristic neuron behaviour. Such behaviour includes a slowaction potential, initial spike clustering, and a post-response quiescence. The network and single neuron results taken togetherprovide an intrinsicmechanismfor termination of uniform high activity generated by the excitatory network. It is therefore predicted that large regions of the subthalamic nucleus respond uniformly to stimuli, in the form of a pulse of activity with a sharp rise and fall. In addition, the single neuron model indicates that pulses will occur in pairs. It is proposedthat the subthalamic nucleus acts as a “braking mechanism”. It can induce, via intermediate structures, awide-spread pulse of inhibition on basal ganglia target nuclei. Furthermore, the sequence of two pulses can generate a window of disinhibition over the basal ganglia targets. The width of this time window may be under direct striatal control. Variable interpulse duration implies a role for the subthalamic nucleus in temporal processing.
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Fan, Kai Yoon. "GABAergic synaptic transmission, plasticity and integration in the subthalamic nucleus." Thesis, University of Sheffield, 2012. http://etheses.whiterose.ac.uk/3167/.

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Cebi, Idil [Verfasser]. "Preoperative Stratification of Gait Outcome from Subthalamic Nucleus Stimulation / Idil Cebi." Tübingen : Universitätsbibliothek Tübingen, 2021. http://d-nb.info/1226756085/34.

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Çebi, Idil [Verfasser]. "Preoperative Stratification of Gait Outcome from Subthalamic Nucleus Stimulation / Idil Cebi." Tübingen : Universitätsbibliothek Tübingen, 2021. http://d-nb.info/1226756085/34.

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Books on the topic "Subthalamic nucleu"

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Enrico, Marani, ed. The subthalamic nucleus: Development, cytology, topography and connections. Berlin: Springer, 2008.

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Marani, Enrico, Tjitske Heida, Egbert A. J. F. Lakke, and Kamen G. Usunoff. The Subthalamic Nucleus Part I: Development, Cytology, Topography and Connections. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-79460-8.

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Heida, Tjitske, Enrico Marani, and Kamen G. Usunoff. The Subthalamic Nucleus Part II: Modelling and Simulation of Activity. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-79462-2.

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MacMillan, Meeka. Responses of human thalamic and subthalamic nucleus neurons during sequential movements. Ottawa: National Library of Canada, 2002.

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Romas, John. Quantifying decreases in parkinsonian rigidity with surgical intervention in the subthalamic nucleus. Ottawa: National Library of Canada, 2001.

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Enrico Marani,Kamen G. Usunoff,Tjitske Heida. The Subthalamic Nucleus. Springer, 2008.

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The Subthalamic Nucleus. Springer, 2008.

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The Subthalamic Nucleus. Springer, 2008.

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Enrico Marani,Tjitske Heida,Egbert A. J. F. Lakke. The Subthalamic Nucleus. Springer, 2008.

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Cunic, Danny. Functional assessment of the subthalamic nucleus. 2005.

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Book chapters on the topic "Subthalamic nucleu"

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Gill, S. S., and P. Heywood. "Subthalamic Nucleus Lesions." In Progress in Neurological Surgery, 188–95. Basel: KARGER, 2000. http://dx.doi.org/10.1159/000062060.

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Wilson, Charles, and Michael Farries. "Subthalamic Nucleus Cellular Models." In Encyclopedia of Computational Neuroscience, 2909–13. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4614-6675-8_90.

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Wilson, Charles, and Michael Farries. "Subthalamic Nucleus Cellular Models." In Encyclopedia of Computational Neuroscience, 1–5. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-7320-6_90-3.

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Benabid, A. L., A. Koudsie, A. Benazzouz, B. Piallat, N. Van Blerkom, V. Fraix, and P. Pollak. "Subthalamic Nucleus Deep Brain Stimulation." In Progress in Neurological Surgery, 196–226. Basel: KARGER, 2000. http://dx.doi.org/10.1159/000062043.

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Gillies, Andrew, David Willshaw, Jeremy Atherton, and Gordon Arbuthnott. "Functional Interactions within the Subthalamic Nucleus." In Advances in Behavioral Biology, 359–68. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0715-4_36.

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Wilson, Charles, and Michael Farries. "Basal Ganglia: Subthalamic Nucleus Cellular Models." In Encyclopedia of Computational Neuroscience, 1–7. New York, NY: Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4614-7320-6_90-4.

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Benabid, A. L., J. Mitrofanis, S. Chabardes, E. Seigneuret, N. Torres, B. Piallat, A. Benazzouz, et al. "Subthalamic Nucleus Stimulation for Parkinson's Disease." In Textbook of Stereotactic and Functional Neurosurgery, 1603–30. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-69960-6_96.

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Agari, Takashi, and Isao Date. "Stimulation of Subthalamic Nucleus for Parkinson’s Disease." In Deep Brain Stimulation for Neurological Disorders, 73–86. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-08476-3_7.

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Allers, Kelly A., Judith R. Walters, and Deborah S. Kreiss. "Neuronal Firing Patterns in the Subthalamic Nucleus." In Advances in Behavioral Biology, 245–54. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0179-4_25.

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Féger, J., P. Robledo, and N. Renwart. "The Subthalamic Nucleus: New Data, New Questions." In Advances in Behavioral Biology, 99–108. Boston, MA: Springer New York, 1991. http://dx.doi.org/10.1007/978-1-4684-5871-8_11.

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Conference papers on the topic "Subthalamic nucleu"

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Njap, Felix, Jens C. Claussen, Andreas Moser, Ulrich G. Hofmann, Tuan D. Pham, Xiaobo Zhou, Hiroshi Tanaka, et al. "Comparing Realistic Subthalamic Nucleus Neuron Models." In 2011 INTERNATIONAL SYMPOSIUM ON COMPUTATIONAL MODELS FOR LIFE SCIENCES (CMLS-11). AIP, 2011. http://dx.doi.org/10.1063/1.3596632.

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Sun, Kaiqiong, Xuan Shang, and Zhen Chen. "Active contour based subthalamic nucleus segmentation on MRI." In Sixth International Symposium on Multispectral Image Processing and Pattern Recognition, edited by Jianguo Liu, Kunio Doi, Aaron Fenster, and S. C. Chan. SPIE, 2009. http://dx.doi.org/10.1117/12.832909.

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Kim, Jinyoung, Yuval Duchin, Guillermo Sapiro, Jerrold Vitek, and Noam Harel. "Clinical subthalamic nucleus prediction from high-field brain MRI." In 2015 IEEE 12th International Symposium on Biomedical Imaging (ISBI 2015). IEEE, 2015. http://dx.doi.org/10.1109/isbi.2015.7164104.

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Schoeters, Ruben, Thomas Tarnaud, Wout Joseph, Luc Martens, Robrecht Raedt, and Emmeric Tanghe. "Comparison between Direct Electrical and Optogenetic Subthalamic Nucleus Stimulation." In 2018 EMF-Med 1st World Conference on Biomedical Applications of Electromagnetic Fields (EMF-Med). IEEE, 2018. http://dx.doi.org/10.23919/emf-med.2018.8526018.

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Manferlotti, Elena, Matteo Vissani, Alberto Mazzoni, and Arvind Kumar. "Correlated inputs to striatal population drive subthalamic nucleus hyper-synchronization." In 2021 10th International IEEE/EMBS Conference on Neural Engineering (NER). IEEE, 2021. http://dx.doi.org/10.1109/ner49283.2021.9441370.

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Niketeghad, Soroush, Adam O. Hebb, Joshua Nedrud, Sara J. Hanrahan, and Mohammad H. Mahoor. "Motor task event detection using Subthalamic Nucleus Local Field Potentials." In 2015 37th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2015. http://dx.doi.org/10.1109/embc.2015.7319650.

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Kaku, Heet, Musa Ozturk, Ashwin Viswanathan, Joohi Jimenez-Shahed, Sameer Sheth, and Nuri F. Ince. "Grouping Neuronal Spiking Patterns in the Subthalamic Nucleus of Parkinsonian Patients." In 2019 41st Annual International Conference of the IEEE Engineering in Medicine & Biology Society (EMBC). IEEE, 2019. http://dx.doi.org/10.1109/embc.2019.8857418.

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Telkes, I., N. F. Ince, I. Onaran, and A. Abosch. "Localization of subthalamic nucleus borders using macroelectrode local field potential recordings." In 2014 36th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2014. http://dx.doi.org/10.1109/embc.2014.6944160.

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Vieira, Gabriela Albertino, Raquel Medeiros de Souza, Érica Rocha Assunção, Laís Soares Figueiredo, Natália Rafael Perdigão, and Paula Luciana Scalzo. "Effect of DBS on decreasing pain intensity in individuals with PD: a systematic review." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.230.

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Abstract:
Background: Pain is a common non-motor symptom in Parkinson’s disease (PD), causing impairment in the functionality and quality of life. Objectives: To summarize the effects of deep brain stimulation (DBS) on pain intensity in PD. Design: Systematic review. Methods: A search was conducted using the Pubmed, Scielo, Embase, Lilacs, and Cochrane databases. Keywords were: “Parkinson* AND (“DBS” OR “deep brain stimulation”) AND “pain”. Complete available articles that measured pain intensity before and after DBS were selected. Results: Of the 251 studies, 17 met the criteria. The sample included from 14 to 79 patients (n = 532). The time of surgery was 3 to 96 months. The subthalamic nucleus was the main surgical target. Seventeen and 389 individuals were submitted to unilateral and bilateral implantation, respectively. Globus pallidus was used as a surgical target in three studies. The unilateral implant was performed in 12 patients and the bilateral in 37. Different instruments were used to measure the pain intensity. It declined after surgery in all studies. Conclusion: The results show that pain intensity decreased after DBS, and most studies performed bilateral stimulation in the subthalamic nucleus. This information is important in guiding the therapeutic approach in PD patients with pain. However, the different surgical parameters and instruments used to assess pain limit the summarization of results.
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Mamun, K. A., R. Vaidyanathan, M. E. Lutman, J. Stein, X. Liu, T. Aziz, and S. Wang. "Decoding movement and laterality from local field potentials in the subthalamic nucleus." In 5th International IEEE/EMBS Conference on Neural Engineering (NER 2011). IEEE, 2011. http://dx.doi.org/10.1109/ner.2011.5910505.

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