Academic literature on the topic 'Subclinical atherosclerosi'
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Journal articles on the topic "Subclinical atherosclerosi"
Drapkina, O. M., O. N. Korneeva, and N. V. Mankova. "Subclinical atherosclerosis: The benefits of calcium antagonists." "Arterial’naya Gipertenziya" ("Arterial Hypertension") 18, no. 2 (April 28, 2012): 118–25. http://dx.doi.org/10.18705/1607-419x-2012-18-2-118-125.
Full textКазакова, М. И., and Н. П. Митьковская. "Subclinical Coronary Atherosclerosis: Significance in Cardiovascular Risk Stratification." Кардиология в Беларуси, no. 4 (September 26, 2022): 482–91. http://dx.doi.org/10.34883/pi.2022.14.4.010.
Full textBoytsov, S. A., V. V. Kukharchuk, Yu A. Karpov, I. V. Sergienko, O. M. Drapkina, A. E. Semenova, and S. Zh Urazalina. "Subclinical atherosclerosis as a risk factor of cardiovascular events." Cardiovascular Therapy and Prevention 11, no. 3 (June 20, 2012): 82–86. http://dx.doi.org/10.15829/1728-8800-2012-3-82-86.
Full textOnalan, Orhan, Adem Adar, Hakan Keles, Goksen Ertugrul, Nurhayat Ozkan, Habibullah Aktas, and Ekrem Karakaya. "Onychomycosis is associated with subclinical atherosclerosis in patients with diabetes." Vasa 44, no. 1 (January 1, 2015): 59–64. http://dx.doi.org/10.1024/0301-1526/a000407.
Full textKousios, Andreas, Panayiotis Kouis, and Andrie G. Panayiotou. "Matrix Metalloproteinases and Subclinical Atherosclerosis in Chronic Kidney Disease: A Systematic Review." International Journal of Nephrology 2016 (2016): 1–11. http://dx.doi.org/10.1155/2016/9498013.
Full textGenkel, Vadim, Ilya Dolgushin, Irina Baturina, Albina Savochkina, Karina Nikushkina, Anna Minasova, Lubov Pykhova, Veronika Sumerkina, Alla Kuznetsova, and Igor Shaposhnik. "Circulating Ageing Neutrophils as a Marker of Asymptomatic Polyvascular Atherosclerosis in Statin-Naïve Patients without Established Cardiovascular Disease." International Journal of Molecular Sciences 23, no. 17 (September 5, 2022): 10195. http://dx.doi.org/10.3390/ijms231710195.
Full textTsiogka, Aikaterini, Stamatios Gregoriou, Alexander Stratigos, Stergios Soulaidopoulos, Natalia Rompoti, Pantelis Panagakis, Marina Papoutsaki, et al. "The Impact of Treatment with IL-17/IL-23 Inhibitors on Subclinical Atherosclerosis in Patients with Plaque Psoriasis and/or Psoriatic Arthritis: A Systematic Review." Biomedicines 11, no. 2 (January 23, 2023): 318. http://dx.doi.org/10.3390/biomedicines11020318.
Full textKatamadze, N. O., L. L. Berstein, and Yu N. Grishkin. "Subclinical atherosclerosis diagnostics as a component of a modern strategy for cardiovascular risk stratification." Cardiovascular Therapy and Prevention 11, no. 2 (April 20, 2012): 76–84. http://dx.doi.org/10.15829/1728-8800-2012-2-76-84.
Full textSimerzin, V. V., O. V. Fatenkov, T. V. Malykhina, I. V. Gagloeva, M. A. Galkina, T. E. Molchanova, and Yu R. Yunusova. "THE VERIFICATION OF SUBCLINICAL CAROTID ATHEROSCLEROSIS IN THE RISK STRATIFICATION AT PRIMARY CARDIOVASCULAR PROPHYLAXIS." Morphological newsletter 25, no. 3 (September 30, 2017): 58–62. http://dx.doi.org/10.20340/mv-mn.17(25).03.58-62.
Full textAhmad, Garg, Dhar, Srivastava, Biswas, P. Barthwal, Shirazi, and Srivastava. "Predictors of atherosclerosis in rheumatoid arthritis." Vasa 41, no. 5 (August 1, 2012): 353–59. http://dx.doi.org/10.1024/0301-1526/a000221.
Full textDissertations / Theses on the topic "Subclinical atherosclerosi"
COGGI, DANIELA. "RELATIONSHIP BETWEEN PLASMA LEVELS OF PCSK9, VASCULAR EVENTS AND MARKERS OF SUBCLINICAL ATHEROSCLEROSIS AND INFLAMMATION." Doctoral thesis, Università degli Studi di Milano, 2021. http://hdl.handle.net/2434/811217.
Full textBackground and purpose: Proprotein convertase subtilisin/kexin type 9 (PCSK9), one of the main regulators of LDL receptor metabolism, has been associated with atherosclerosis development. Several studies have confirmed such association through both lipid and non-lipid pathways. However, the direct relationships between circulating PCSK9 and markers of subclinical and clinical atherosclerosis are still matter of debate. Therefore, we investigated the relationships between plasma PCSK9 levels and some indexes of subclinical (imaging markers) and clinical (vascular events; VEs) atherosclerosis. Another objective was the identification of the independent determinants of PCSK9, with particular attention to lipids and inflammatory biomarkers. Finally, we also assessed the relationship between some imaging markers and four SNPs of the PCSK9 gene, known to be associated with the presence of low levels of LDL-cholesterol. In order to validate the results obtained in this last part, the genetic analyses were replicated in an independent cohort recruited in the United Kingdom (UK). Methods: The study was carried out taking advantage of databases, biobanks and imaging-bank of the IMPROVE study. 3,703 European subjects (54-79 years; 48% men), free of VEs at baseline and defined at high risk for the presence of at least three vascular risk factors, were recruited and followed-up for 36 months. PCSK9 was measured by ELISA and log-transformed prior to analyses. Conventional imaging markers [carotid intima-media thickness (cIMT) and carotid plaque-size], and emerging imaging markers [cIMT change over time, echolucency of the intima-media thickess of common carotid measured in plaque free areas (PF CC-IMTmean), echolucency of the biggest plaque detected in the whole carotid tree, and carotid calcium score (cCS)] were measured on ultrasonographic scans stored in the imaging-bank. In particular, echolucency was measured in terms of grey scale median (GSM) of pixels distribution of a specific region of interest, whereas cCS was calculated as sum of lengths of acoustic shadow cones generated by calcium within carotid plaques. Lipids were measured with enzymatic methods (except for LDL-cholesterol, which was calculated by Friedewald's formula). Among inflammatory markers, high-sensitivity C-reactive protein (hs-CRP) was measured by turbidimetry, whereas white blood cells (WBC) count and the leukocyte formula had already been measured locally. All the IMPROVE study and UK (n=22,179; 48% men) subjects have been genotyped. Results: In the univariate analysis, PCSK9 was positively correlated with total, LDL-, and HDL-cholesterol, and with triglycerides and basophils (all p <0.0001), whereas was negatively correlated with neutrophils and eosinophils (both p=0.04). The positive correlations observed with hs-CRP and WBC count were just close to the statistical significance (p=0.060 and 0.064, respectively). Fibrates or statins therapies (positively; both p <0.0001), as well as male sex and family history of diabetes (negatively; both p <0.05) were the strongest independent predictors of plasma PCSK9 levels. In the unadjusted analysis, a negative correlation was observed between PCSK9 levels and basal cIMT variables (i.e. carotid IMTmean, IMTmax, IMTmean-max, and PF CC-IMTmean), a negative correlation between PCSK9 and cIMT change over time (Fastest-IMTmax-progr) and cCS (all p ≤0.01), whereas a positive trend was observed between PCSK9 and GSM of both PF CC-IMTmean and carotid plaque (both p ≤0.0001). The cCS (positively) and the GSM of PF CC-IMTmean (positively) were significantly (or almost significantly) associated with PCSK9 in several multivariate models (all p ≤0.064). All correlations observed in the univariate analysis between PCSK9 and basal cIMT variables, Fastest-IMTmax-progr and GSM of carotid plaque lost the statistical significance after adjustment for age, sex, latitude, and other potential confounders. During the follow-up [median (interquartile range): 3.01 (2.98; 3.12) years], 215 VEs were recorded: 125 coronary, 73 cerebral and 17 peripheral VEs. Among these, 37 were hard events (i.e. myocardial infarction, sudden death and stroke). In the unadjusted analysis, PCSK9 was positively associated with combined and coronary events (both p <0.01), but not with cerebrovascular events. Also in this case, however, all the associations observed lost the statistical significance after adjustment of the analyses for age, sex, and stratification for latitude. The lack of association with VEs was confirmed also in the model adjusted for all confounding factors considered, and in the analyses focused on hard events. With regard to the role of genetic variants, none of the four SNPs considered was correlated with cIMT (i.e. IMTmean, IMTmax, IMTmean-max) when the analysis was performed in the subjects recruited in the IMPROVE study. The rs11591147 variant, by contrast, was negatively correlated with IMTmax measured in the UK population (p=0.002). By combining the four genetic variants in a score, the relationship with cIMT was not significant in the IMPROVE study, whereas was negative and significant in the UK population (all p <0.01). Conclusions: Plasma PCSK9 levels are not associated with VEs. Regarding markers of subclinical atherosclerosis, PCSK9 levels are associated neither with lesion size, nor with carotid plaque echolucency, but are associated with echolucency of carotid wall thickness and with carotid calcium score. Therefore, further studies are needed to better understand the role of such circulating proprotein in carotid wall thickness echolucency and in carotid calcium score. Fibrates or statins therapies, as well as male sex and family history of diabetes are the strongest independent predictors of PCSK9 levels. The associations, previously observed, between circulating PCSK9 and some lipid and inflammatory markers have been confirmed. The relationship between plasma levels of PCSK9 and other inflammatory markers (neutrophils, basophils and eosinophils) deserves further investigation, as does the association between the four selected PCSK9 variants and cIMT in the UK cohort, as it suggests a possible role of PCSK9 SNPs or gene polymorphisms in atherosclerosis and in its preventive strategies.
Scalzi, Lisabeth Victoria. "Subclinical Atherosclerosis in Systemic Lupus Erythematosus." Case Western Reserve University School of Graduate Studies / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=case1212695307.
Full textMamadu, Hadii M., Antwan Jones, Timir Paul, Pooja Subedi, Sreenivas P. Veeranki, Liang Wang, Hemang P. Panchal, Arsham Alamian, Matthew Budoff, and Ali Alamin. "Does Neighborhood Disadvantage Affect Subclinical Atherosclerosis?" Digital Commons @ East Tennessee State University, 2016. https://dc.etsu.edu/etsu-works/1392.
Full textPalanca, Ana. "Subclinical atherosclerosis in chronic kidney disease and diabetes." Doctoral thesis, Universitat Autònoma de Barcelona, 2020. http://hdl.handle.net/10803/670707.
Full textLa enfermedad cardiovascular es la primera causa de morbilidad y mortalidad mundial. Los individuos con diabetes y enfermedad renal crónica (ERC) presentan un mayor riesgo de eventos cardiovasculares (ECV) con respecto a la población general. En la diabetes, el incremento del riesgo cardiovascular es heterogéneo y se ha relacionado con el grado de afectación renal. Por otra parte, los algoritmos tradicionales para calcular el riesgo cardiovascular no trasladan con suficiente precisión el riesgo futuro de ECV. La evaluación de la aterosclerosis subclínica (AS) mediante ecografía multiterritorial representa una herramienta válida para refinar el riesgo cardiovascular. El propósito de esta tesis fue analizar mediante ecografía multiterritorial la prevalencia, distribución y progresión de AS, así como factores de riesgo asociados, en una amplia cohorte de pacientes, sin enfermedad cardiovascular, con ERC y con y sin diabetes. Posteriormente, se evaluó el valor pronóstico de la AS para determinar la incidencia de ECV en esta población de alto riesgo. Inicialmente, se analizaron los datos de los sujetos con ERC, con diabetes y sin diabetes, de la cohorte del estudio NEFRONA reclutados al inicio del estudio y que asistieron a la visita de control a los 24 meses. Tras realizar un estudio ultrasonográfico carotídeo y femoral tanto en la visita inicial como en la de seguimiento, se evaluó la correlación de factores de riesgo asociados con prevalencia y progresión de placa mediante análisis multivariables. Asimismo, se realizó otro análisis con todos los sujetos del NEFRONA, con y sin diabetes, reclutados inicialmente y a los que se les siguió durante 48 meses. Durante el periodo de seguimiento se registraron los ECV incidentes. Se utilizaron análisis bivariados y análisis de modelo de riesgos competitivos de Fine-Grey para el estudio estadístico. El índice C se estimó para los modelos de riesgo resultantes con mayor potencia. Como resultados, se observó que la proporción de individuos con placa basal fue mayor entre los sujetos con diabetes. Los sujetos con diabetes presentaron con mayor frecuencia la afectación con placa de más de dos territorios vasculares. También se observó una mayor progresión de placa en los individuos con diabetes. Tras realizar el análisis multivariable, se demostró que la presencia de placa basal se asociaba con la edad, el género masculino, el hábito tabáquico y la diálisis en los sujetos sin diabetes mientras que, en los sujetos con diabetes, la presencia de placa basal se asoció tan sólo a la edad y al género masculino. La progresión de placa se asoció a la edad, al número de territorios con placa basal, al hábito tabáquico y a la diálisis en ambos grupos. Se registraron 107 ECV entre los sujetos sin diabetes (19.58 por 1000 años-persona) y 96 entre los sujetos con diabetes (44.44 por 1000 años-persona). El modelo que mejor predijo futuros ECV en individuos sin diabetes contenía las variables: edad, 25-OH vitamina D y número de territorios con placa basal. Entre los participantes con diabetes el modelo más robusto en predecir ECV incidentes contenía tan sólo la variable ‘número de territorios con placa basal’. Para ambos modelos, el índice estadístico C, estimado a los 24 y a los 48 meses, fue superior a 0.70. La AS es más prevalente, conlleva mayor carga y es más progresiva en individuos con ERC y diabetes. En estos sujetos, la diabetes supera otros factores de riesgo descritos. Así mismo, la carga de AS es el predictor más potente de futuros ECV en individuos con diabetes y ERC. La detección precoz de carga AS mediante ultrasonografía multiterritorial podría mejorar la predicción de ECV en esta población.
Cardiovascular disease (CVD) is the leading cause of morbidity and mortality worldwide. Individuals with diabetes and chronic kidney disease (CKD) have remarkably high rates of CVD risk. Moreover, incremental cardiovascular risk in diabetes is heterogeneous and has been often related to concomitant CKD. Typically used risk equations based on traditional cardiovascular risk factors fail to accurately predict cardiovascular risk not only in the general population but also in these subsets of the population. Multi-territorial ultrasonography to assess subclinical atherosclerosis (SA) has emerged as a valid tool to refine cardiovascular risk assessment beyond traditional risk factors. The purpose of this thesis was to analyse the prevalence, distribution, and progression of SA, as well as the associated cardiovascular risk factors in a large cohort of CKD subjects with and without diabetes, free from CVD, using multi-territorial ultrasonography. Subsequently, we further evaluated the prognostic value of SA in determining the incidence of first cardiovascular events (CVE) in this high-risk population. First, we included the data from CKD subjects with and without diabetes and free from previous CVE from the NEFRONA cohort, that were recruited at baseline, and that attended a follow-up visit 24 months later. Participants underwent carotid and femoral ultrasound examinations at baseline and at 24-month follow-up. Risk factors associated with the prevalence and progression of SA were evaluated using multivariate model analyses. In the second hand, we also conducted another analysis including data from the NEFRONA cohort subjects with and without diabetes that were recruited initially and were followed-up for 48 months. During the follow-up period, all CVE were registered. Bivariate analysis and Fine-Gray competing risk models were used to perform the statistical analysis. Concordance Index (C-statistics) was estimated for the strongest resulting risk models. We found that at baseline, the proportion of subjects with plaque at any of the examined territories was higher among diabetic individuals. Diabetic subjects more frequently had more than two vascular territories with plaque. During a 24-month follow-up period, plaque progression occurred in 72.2% individuals with diabetes whereas, among individuals without diabetes, plaque progression occurred in 55.8%. Multivariable analysis indicated that plaque at baseline was significantly associated with age, male gender, smoking, and dialysis in the non-diabetic subjects, while only age and male gender were associated with plaque presence in diabetic subjects. Plaque progression was significantly associated with age, the number of territories with basal plaque, smoking, and renal replacement therapy in both groups. Additionally, during a mean follow-up time of 48 months, CVE rate among participants without diabetes was 19.58 per 1000 person-years and 44.44 per 1000 person-years among participants with diabetes. After competing risk analyses and model selection, those variables that better predicted CVE in individuals without diabetes were the number of territories with plaque, age and serum concentrations of 25-OH vitamin D. Among participants with diabetes, the strongest model predicting incident CVE had only one variable: the number of territories with basal plaque. For both models, the concordance (C) index score was greater than 0.7 at both 24 and 48 months. We concluded that SA is more prevalent, carries a higher plaque burden, and is more progressive in CKD subjects with diabetes than in CKD subjects without diabetes. In these individuals, diabetes outweighs other risk factors associated with the presence of SA. SA is the strongest predictor of future CVE in diabetic individuals with CKD. Early detection of the SA burden by multi-territorial vascular ultrasound could improve CVE prediction in this population.
Xu, Lin, and 徐琳. "Subclinical atherosclerosis, cardiovascular risk factors and metabolicsyndrome in older Chinese people." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B4451430X.
Full textDahlén, Elsa. "Markers of subclinical atherosclerosis and arterial stiffness in type 2 diabetes." Doctoral thesis, Linköpings universitet, Allmänmedicin, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-71404.
Full textMamudu, Hadii M., Antwan Jones, Timir Paul, Pooja Subedi, Liang Wang, Arsham Alamian, Ali E. Alamin, Gerald Blackwell, and Matthew Budoff. "Geographic and Individual Correlates of Subclinical Atherosclerosis in Asymptomatic Rural Appalachian Population." Digital Commons @ East Tennessee State University, 2017. https://dc.etsu.edu/etsu-works/1377.
Full textPeressini, Marisa. "Detection Method of Subclinical Atherosclerosis of the Carotid Artery with a Hemodynamics Modeling Approach." DigitalCommons@CalPoly, 2018. https://digitalcommons.calpoly.edu/theses/1876.
Full textMirjafari, Hoda. "The prevalence and determinants of subclinical atherosclerosis in an early inflammatory polyarthritis inception cohort." Thesis, University of Manchester, 2011. https://www.research.manchester.ac.uk/portal/en/theses/the-prevalence-and-determinants-of-subclinical-atherosclerosis-in-an-early-inflammatory-polyarthritis-inception-cohort(071e4a81-5c0e-459a-8210-0c84b53f06a5).html.
Full textMamudu, Hadii M., Arsham Alamian, Timir Paul, Pooja Subedi, Liang Wang, Antwan Jones, Ali E. Alamin, David Stewart, Gerald Blackwell, and Matthew Budoff. "Diabetes, Subclinical Atherosclerosis and Multiple Cardiovascular Risk Factors in Hard-to-Reach Asymptomatic Patients." Digital Commons @ East Tennessee State University, 2018. https://dc.etsu.edu/etsu-works/2778.
Full textBooks on the topic "Subclinical atherosclerosi"
A, Bianco Jesus, ed. Subclinical atherosclerosis: Assessing the risks. London: Taylor & Francis, 2006.
Find full textVerfasser, Kenawy Sanaa A., and El Sisi, Rehab W. Verfasser, eds. Behçet's disease and subclinical atherosclerosis: An overlooked association. Saarbrücken: LAP LAMBERT Academic Publishing, 2013.
Find full textJain, Neelesh K. The role of endothelial cell shape and nuclear factor-[kappa]B in subclinical human atherosclerosis. Ottawa: National Library of Canada, 2003.
Find full textBianco. Subclinical Atherosclerosis: Assessing the Risks. Taylor & Francis Group, 2005.
Find full textSubclinical Atherosclerosis: Assessing the Risks. Informa Healthcare, 2005.
Find full textBook chapters on the topic "Subclinical atherosclerosi"
Zhu, Daming, Allen J. Taylor, and Todd C. Villines. "Monitoring of Subclinical Atherosclerotic Disease." In Asymptomatic Atherosclerosis, 549–67. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-179-0_41.
Full textPanayiotou, Andrie G., Debra Ann Hoppensteadt, Andrew Nicolaides, and Jawed Fareed. "Novel Biomarkers and Subclinical Atherosclerosis." In Ultrasound and Carotid Bifurcation Atherosclerosis, 461–86. London: Springer London, 2011. http://dx.doi.org/10.1007/978-1-84882-688-5_27.
Full textTarantini, Giuseppe, Paolo Buja, and Michela Facchin. "Subclinical Atherosclerosis and Primary Prevention." In Clinical Applications of Cardiac CT, 15–28. Milano: Springer Milan, 2012. http://dx.doi.org/10.1007/978-88-470-2522-6_3.
Full textYuan, Chun, Hideki Ota, Xihai Zhao, and Tom Hatsukami. "The Role of MRI in Examining Subclinical Carotid Plaque." In Asymptomatic Atherosclerosis, 363–73. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-179-0_27.
Full textHeffernan, Kevin S. "Exercise Stress Testing in Asymptomatic Individuals and Its Relation to Subclinical Atherosclerotic Cardiovascular Disease." In Asymptomatic Atherosclerosis, 197–210. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-179-0_14.
Full textKiechl, Stefan, Philipp Werner, Michael Knoflach, and Johann Willeit. "Subclinical Atherosclerosis, Markers of Inflammation, and Oxidative Stress." In Ultrasound and Carotid Bifurcation Atherosclerosis, 487–509. London: Springer London, 2011. http://dx.doi.org/10.1007/978-1-84882-688-5_28.
Full textErbel, Raimund, Nils Lehmann, Andreas Stang, Sofia Churzidse, Susanne Moebus, and Karl-Heinz Jöckel. "Blood Pressure and Atherosclerosis: Subclinical Arteriosclerosis as an Early Sign of Organ Damage." In Updates in Hypertension and Cardiovascular Protection, 363–81. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-75310-2_26.
Full text"Assessing Anti-Atherosclerotic Strategies on Subclinical Atherosclerosis." In Subclinical Atherosclerosis, 137. CRC Press, 2005. http://dx.doi.org/10.1201/b14635-17.
Full textNicholls, Stephen. "The impact of anti-atherosclerotic strategies on subclinical atherosclerosis." In Subclinical Atherosclerosis, 139–52. CRC Press, 2005. http://dx.doi.org/10.1201/b14635-18.
Full textSchoenhagen, Paul. "Relationship between arterial remodeling and subclinical atherosclerosis." In Subclinical Atherosclerosis, 65–73. CRC Press, 2005. http://dx.doi.org/10.1201/b14635-10.
Full textConference papers on the topic "Subclinical atherosclerosi"
Ismail, M., A. Mosallam, M. Abdelaziz, and A. Moshrif. "AB0726 Subclinical atherosclerosis in patients with ankylosing spondylitis." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.4502.
Full textHansen, Laura, Manu Platt, Roy L. Sutliff, and Rudolph L. Gleason. "The Mechanical and Structural Effects of HIV Proteins on Murine Carotid Arteries." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53693.
Full textPolanco Alonso, Dinora, María Teresa Martín Carpi, Victoria Gil Gómez, Pablo Cubero Marín, Marta Forner Vicente, Ana Lilian Simón Robles, Clara Viñado Mañés, et al. "Progression of subclinical atherosclerosis in obstructive sleep apnoea (OSA)." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa2075.
Full textTomeckova, Ivana, Miriam Kozarova, Darina Petrasova, Ivana Paranicova, Zuzana Malachovska, Ivan Tkac, Pavol Joppa, and Ruzena Tkacova. "Determinants of subclinical atherosclerosis in patients with obstructive sleep apnoea." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa2374.
Full textMarín, José M., M. Teresa Martín Carpi, Santiago J. Carrizo, Marta Andrés, Victoria Gil, Marta Forner, José P. Cubero, Clara Viñado, Dinora Polanco, and Salvador Bello. "Subclinical atherosclerosis in obstructive sleep apnea (OSA): The EPIOSA study." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa3364.
Full textTuzcu, G., A. U. Uslu, A. Tuzcu, R. Aydogan Baykara, A. Omma, and A. Kucuk. "AB0199 Endocan levels and subclinical atherosclerosis in patients with rheumatoid arthritis." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.4656.
Full textLederer, David J., Steven M. Kawut, Craig Johnson, Paul Enright, Gary W. Hunninghake, Ganesh Raghu, Eric A. Hoffman, et al. "Subclinical Parenchymal Lung Disease And Atherosclerosis In The MESA Lung Study." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1104.
Full textKarageorgas, Theofanis, Dimitrios Ioakeimidis, and Clio P. Mavragani. "07.10 Psychological comorbidities associated with subclinical atherosclerosis in primary sjögren’s syndrome." In 37th European Workshop for Rheumatology Research 2–4 March 2017 Athens, Greece. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2016-211054.10.
Full textGiannelou, Maira, Andrianos Nezos, Kyriaki Maselou, Nikolaos Drakoulis, Dimitrios Ioakeimidis, Michael Koutsilieris, Haralampos M. Moutsopoulos, and Clio P. Mavragani. "07.11 Contribution of mthfr gene variants in lupus related subclinical atherosclerosis." In 37th European Workshop for Rheumatology Research 2–4 March 2017 Athens, Greece. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2016-211054.11.
Full textHansen, Laura, Manu O. Platt, Roy L. Sutliff, and Rudolph L. Gleason. "The Mechanical and Structural Changes in Murine Arteries due to the Antiretroviral Drug Azidothymidine (AZT)." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80799.
Full textReports on the topic "Subclinical atherosclerosi"
Tsiogka, Aikaterini, Stamatios Gregoriou, Alexander Stratigos, Stergios Soulaidopoulos, Natalia Rompoti, Pantelis Panagakis, Marina Papoutsaki, et al. The impact of treatment with IL-17/IL-23 inhibitors on subclinical atherosclerosis in patients with plaque psoriasis and/or psoriatic arthritis: a systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2022. http://dx.doi.org/10.37766/inplasy2022.12.0102.
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