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Author: Grafiati

Published: 26 October 2023

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1

Anorexia and its impact upon cachexia in cancer disease: A study with emphasis on the multifactorial pathogenesis and consequences of impaired feeding behavior. Amsterdam: Rodopi, 1985.

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2

Kim, Ŭng-gwŏn. Abellino kangmak iyŏngyangchŭng ŭi pyŏngin palgyŏn mit chʻiryopŏp kaebal =: A study for the identification of pathogenesis and treatment of Avellino corneal dystrophy. [Seoul]: Pogŏn Pokchibu, 2007.

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3

H, Thiers Bruce, and Dobson Richard L, eds. Pathogenesis of skin disease. New York: Churchill Livingstone, 1986.

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4

Hanneke, Schuitemaker, and Miedema F, eds. AIDS pathogenesis. Dordrecht: Kluwer Academic, 2000.

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5

Snape, William J., ed. Pathogenesis of Functional Bowel Disease. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5694-3.

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6

Murray, Dawson Ted, ed. Parkinson's disease: Genetics and pathogenesis. New York: Informa Healthcare, 2007.

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7

J, Snape William, ed. Pathogenesis of functional bowel disease. New York: Plenum Medical Book Co., 1989.

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8

Plant pathogenesis and disease control. Boca Raton, FL: Lewis Publishers, 1994.

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9

Crohn's disease: Treatment and pathogenesis. Boca Raton, Fla: CRC Press, 1987.

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10

N, Cadwallader Jack, ed. Crohn's disease: Etiology, pathogenesis & interventions. New York: Nova Science Publishers, 2008.

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11

The pathogenesis of infectious disease. 3rd ed. London: Academic Press, 1987.

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12

Dumitrescu, Alexandrina L., ed. Etiology and Pathogenesis of Periodontal Disease. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-03010-9.

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13

Beamish, Robert E., Vincenzo Panagia, and Naranjan S. Dhalla, eds. Pathogenesis of Stress-Induced Heart Disease. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4613-2589-5.

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14

D, Cooper Allen, ed. Pathogenesis and therapy of gallstone disease. Philadelphia: Saunders, 1991.

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15

Varga, John, C. P. Denton, and Fredrick M. Wigley. Scleroderma: From pathogenesis to comprehensive management. New York: Springer, 2012.

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16

Fitzgerald, Rebecca C., ed. Pre-Invasive Disease: Pathogenesis and Clinical Management. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6694-0.

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17

Chadwick, Derek, and Jamie A. Goode, eds. Chronic Obstructive Pulmonary Disease: Pathogenesis to Treatment. Chichester, UK: John Wiley & Sons, Ltd, 2000. http://dx.doi.org/10.1002/0470868678.

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18

Fitzgerald, Rebecca C. Pre-invasive disease: Pathogenesis and clinical management. New York: Springer, 2011.

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19

C, Pak Charles Y., ed. Renal stone disease: Pathogenesis, prevention, and treatment. Boston: Nijhoff, 1987.

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20

1953-, Kelly John M., ed. Molecular mechanisms of pathogenesis in Chagas disease. Georgetown, Tex: Landes Bioscience / Eurekah.com, 2003.

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21

O, Castell Donald, Wu Wallace C, and Ott David J. 1946-, eds. Gastro-esophageal reflux disease: Pathogenesis, diagnosis, therapy. Mount Kisco, N.Y: Futura, 1985.

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22

W, Tang J., and Andrews J, eds. Infectious disease: Pathogenesis, prevention, and case studies. Chichester, West Sussex: Wiley-Blackwell, 2009.

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23

Derek, Chadwick, and Goode Jamie, eds. Chronic obstructive pulmonary disease: Pathogenesis to treatment. Chichester: Wiley, 2001.

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24

1953-, Kelly John M., ed. Molecular mechanisms of pathogenesis in Chagas disease. Georgetown, Tex: Landes Bioscience / Eurekah.com, 2003.

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25

B, Fick Robert, ed. Pseudomonas aeruginosa, the opportunist: Pathogenesis and disease. Boca Raton: CRC Press, 1993.

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26

1928-, Day Peter R., Jellis G. J, and British Society for Plant Pathology., eds. Genetics and plant pathogenesis. Oxford [Oxfordshire]: Blackwell Scientific Publications, 1987.

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27

Dorpe, Jo Van. Pathogenesis of Alzheimer's Disease: A Study in Transgenic Mice (Acta Biomedica Lovaniensia, 230). Leuven Univ Pr, 2001.

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28

Tournoy, Jos. Physiological Study of Presenilins & Baces, Two Proteases Involved in the Pathogenesis of Alzheimer's Disease. Leuven University Press, 2006.

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29

Morrison, Karen. Prevention of neurological disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0347.

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Neurological disease is very common. It is estimated that one-third of consultations with general practitioners involve neurological complaints, and neurological disorders are present in one-third of patients admitted to hospital. In considering how to reduce the incidence of neurological disease, one must take into account the feasibility of prevention, and the overall morbidity caused by the disease. In stroke, which is very common, interventions which reduce incidence by a small percentage have the potential to have a large impact on a population basis. A disorder such as migraine, while not life-limiting, accounts for significant morbidity and time off work (one study suggests that there are the equivalent of 112 million bedridden days per year due to migraine alone), so, again, interventions that reduce the frequency of episodes even by a small percentage can have great overall impact. This chapter discusses the major categories of neurological disease based on pathogenesis, and current and future approaches to prevention.

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30

Gu, Wenduo, Yao Xie, and Qingbo Xu. Animal models to study pathophysiology of the vasculature. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0005.

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Animal models are designed to be preliminary tools for a better understanding of the pathogenesis, improvement in diagnosis, prevention, and therapy of vascular diseases in humans. Animal models are easily manageable, as compounding effects of dietary and environmental factors can be controlled experimentally. Blood vessel samples can be taken for detailed experimental and biomolecular examination. A thorough understanding of the animal models used is necessary and complete analysis must be validated so that the data can be extrapolated to humans. There are several species that are used for studying vascular pathophysiology, including mice, rats, rabbits, and pigs. Attracted by the well-defined genetic systems, a number of investigators have begun to use the mouse as an experimental system for arteriosclerosis research. Because vascular disorder is a complicated disease, which includes spontaneous (native) atherosclerosis, transplant arteriosclerosis, vein graft atherosclerosis, and angioplasty-induced restenosis, several models for studying all types of vascular disease have recently been established. Using these animal models, much knowledge concerning the pathogenesis of the disease and therapeutic intervention has been gained. This chapter will not attempt to cover all aspects of animal models, but will rather focus on the major progress in understanding the pathophysiology of the vasculature, the (dis)advantages of a variety of models, and how specific models can be appropriately chosen for different purposes of study.

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31

Chiou, Pinwen Peter. A molecular study of viral proteins in the pathogenesis of infectious hematopoietic necrosis virus. 1996.

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32

Odds, Frank C. Pathogenesis of fungal disease. Edited by Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum, and Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0008.

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The pathogenesis of fungal disease involves an interplay between fungal virulence factors and host immune responses. Most fungal pathogens are opportunists that preferentially invade hosts with immune defects, but the fact that relative pathogenicity varies between fungal species (and even between different strains within a species) is evidence that fungi have evolved multiple, different molecular virulence factors. Experiments in which genes encoding putative virulence attributes are specifically disrupted and the resulting mutants are tested for virulence in a range of vertebrate and invertebrate hosts have identified or confirmed many gene products as significant for the pathogenesis of various types of fungal disease. These include factors determining fungal shape in vivo, biofilm formation, and a plethora of surface components, including adhesins and hydrolytic enzymes. This chapter provides an overview of fungal virulence attributes.

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33

Pletnikov, Mikhail V., Guo-Li Ming, and Christopher A. Ross. Animal and Cellular Models of Psychotic Disorders. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0015.

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Animal and cell models are experimental systems developed to study particular aspects of a disease, as no model can accurately reflect all features of the disease. In this critical review we mention some of the nongenetic models but focus on genetic mouse models, evaluate their advantages and limitations, and comment on potential new prospects for the field. The ability to reprogram somatic cells from patients and unaffected donors to induced pluripotent stem cells (iPSCs) has the potential to substantially enhance our knowledge of normal cellular development and disease pathogenesis. The use of cell and animal models will help elucidate basic cellular and molecular mechanisms of pathogenesis, which will enable the development of targeted therapeutic approaches.

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34

J, Genco Robert, ed. Molecular pathogenesis of periodontal disease. Washington, D.C: ASM Press, 1994.

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35

Mims' Pathogenesis of Infectious Disease. Elsevier, 1995. http://dx.doi.org/10.1016/c2009-0-04278-0.

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36

Mims' Pathogenesis of Infectious Disease. Elsevier, 2015. http://dx.doi.org/10.1016/c2011-0-07888-2.

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37

McLachlan, Sandra M., and Basil Rapoport. Graves' Disease: Pathogenesis and Treatment. Springer London, Limited, 2012.

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38

Jr, William J. Snape. Pathogenesis of Functional Bowel Disease. Springer, 2012.

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39

Oku, Hachiro. Plant Pathogenesis and Disease Control. Taylor & Francis Group, 2020.

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40

Mims, Cedric A., John Stephen, and Anthony A. Nash. Mims' Pathogenesis of Infectious Disease. Elsevier Science & Technology Books, 2000.

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41

Mims' Pathogenesis of Infectious Disease. 5th ed. Academic Press, 2001.

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42

Koka, Prasad S. Stem Cells in Disease Pathogenesis. Nova Science Publishers, Incorporated, 2021.

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43

Mims, Cedric A., Nigel J. Dimmock, John Stephen, and Anthony A. Nash. Mims' Pathogenesis of Infectious Disease. Elsevier Science & Technology Books, 2013.

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44

Stephen, John, Cedric A. Mims, and Anthony Nash. Mims' Pathogenesis of Infectious Disease. 5th ed. Academic Press, 2001.

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45

Oku, Hachiro. Plant Pathogenesis and Disease Control. Taylor & Francis Group, 2020.

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46

Mims' Pathogenesis of Infectious Disease. Elsevier Science & Technology Books, 2015.

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47

Jr, William J. Snape. Pathogenesis of Functional Bowel Disease. Springer, 2013.

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48

Dawson, Ted M. Parkinson's Disease: Genetics and Pathogenesis. Taylor & Francis Group, 2007.

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49

Koka, Prasad S. Stem Cells in Disease Pathogenesis. Nova Science Publishers, Incorporated, 2021.

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50

Nash, Anthony A., Robert G. Dalziel, and J. Ross Fitzgerald. Mims' Pathogenesis of Infectious Disease. Elsevier Science & Technology Books, 2015.

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