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1

Agbandje-McKenna, Mavis, and Robert McKenna, eds. Structural Virology. Cambridge: Royal Society of Chemistry, 2010. http://dx.doi.org/10.1039/9781849732239.

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Agbandje-McKenna, Mavis, and McKenna Robert. Structural virology. Cambridge: RSC Publishing, 2011.

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3

Uversky, Vladimir N. Flexible viruses: Structural disorder in viral proteins. Hoboken: Wiley, 2012.

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4

J, Gibbs A., Calisher Charles H, and Garcia-Arenal Fernando, eds. Molecular basis of virus evolution. Cambridge [England]: Cambridge University Press, 1995.

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5

Wah, Chiu, Burnett Roger M, and Garcea Robert L, eds. Structural biology of viruses. New York: Oxford University Press, 1997.

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6

Kasteel, Daniella T. J. Structure, morphogenesis and function of tubular structures induced by cowpea mosaic virus. Wageningen: [s.n.], 1999.

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7

McKenna, Robert, Stephen Neidle, David M. J. Lilley, Mavis Agbandje-McKenna, and Roderick E. Hubbard. Structural Virology. Royal Society of Chemistry, The, 2010.

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8

Longhi, Sonia, and Vladimir Uversky. Flexible Viruses: Structural Disorder in Viral Proteins. Wiley & Sons, Incorporated, John, 2011.

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9

Longhi, Sonia, and Vladimir Uversky. Flexible Viruses: Structural Disorder in Viral Proteins. Wiley & Sons, Incorporated, John, 2011.

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10

Longhi, Sonia, and Vladimir Uversky. Flexible Viruses: Structural Disorder in Viral Proteins. Wiley & Sons, Incorporated, John, 2011.

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11

Selected Papers Of Michael G Rossmann With Commentaries The Development Of Structural. World Scientific Publishing Co Pte Ltd, 2014.

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12

Livingston, Schuyler, Benjamin Young, Martin Markowitz, Poonam Mathur, and Bruce L. Gilliam. HIV Virology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0017.

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HIV is a member of the lentivirus subfamily of retroviruses. Two distinct groups of viruses are pathogenic in humans: HIV-1 and HIV-2. Both are transmitted sexually and known to cause immunodeficiency disease. HIV enters the cell through use of the CD4 receptor and chemokine co-receptors, primarily CCR5 and CXCR4. The viral genome is transcribed from RNA to DNA by reverse transcriptase and integrated into the host genome by integrase. The HIV genome encodes 15 proteins, comprising three categories: structural, regulatory, and accessory. After budding from the host cell, the virus matures into its infectious form through cleavage of viral precursor proteins by protease.
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13

Fraenkel-Conrat, Heinz. Comprehensive Virology Volume 13 : Structure and Assembly: Primary, Secondary, Tertiary, and Quaternary Structures. Springer, 2014.

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14

Fraenkel-Conrat, Heinz. Comprehensive Virology Volume 13 : Structure and Assembly: Primary, Secondary, Tertiary, and Quaternary Structures. Springer, 2013.

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Fraenkel-Conrat, Heinz. Comprehensive Virology Volume 13 : Structure and Assembly: Primary, Secondary, Tertiary, and Quaternary Structures. Springer, 2013.

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16

Thu, Ye, and Naiel Nassar. Managing the Patient with Multidrug-Resistant HIV. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0022.

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During approximately the past 15 years, HIV infection has been transformed into a chronic manageable disease primarily due to the effectiveness of antiretroviral therapy. Treatment guidelines emphasize the need for at least two or preferably three fully active medications in the salvage regimens of patients experiencing virologic failure. The new regimen should be started with as little interruption as possible because the structured interruption of treatment in patient with multidrug-resistant HIV infection is associated with greater progression of the disease. The new pharmacokinetic enhancer, cobicistat, is available as a fixed-dose combination product with antiretroviral medication that allows the treatment to be simplified and reduces the pill burden.
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