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Journal articles on the topic 'Structural alterations'

Author: Grafiati

Published: 7 September 2022

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1

Sugo, T., Y. Sakata, and M. Matsuda. "Structural Alterations in Hereditary Dysfibrinogens." Current Protein and Peptide Science 3, no. 3 (June 1, 2002): 239–47. http://dx.doi.org/10.2174/1389203023380648.

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2

Agabiti-Rosei, E., M. L. Muiesan, and G. Muiesan. "Regression of Structural Alterations in Hypertension." American Journal of Hypertension 2, no. 2 Pt 2 (February 1, 1989): 70S—76S. http://dx.doi.org/10.1093/ajh/2.2.70s.

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3

Tunç, T., and K. Yıldız. "Structural Alterations in Mechanically Activated Malachite." Acta Physica Polonica A 125, no. 2 (January 2014): 177–79. http://dx.doi.org/10.12693/aphyspola.125.177.

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4

Balek, Vladimir, and Alexandra de Koranyi. "Diagnostics of structural alterations in coal." Fuel 69, no. 12 (December 1990): 1502–6. http://dx.doi.org/10.1016/0016-2361(90)90197-x.

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5

Pourzal, Robin, Ralf Theissmann, Birgit Gleising, Sophie Williams, and Alfons Fischer. "Micro-Structural Alterations in MoM Hip Implants." Materials Science Forum 638-642 (January 2010): 1872–77. http://dx.doi.org/10.4028/www.scientific.net/msf.638-642.1872.

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Since the introduction of CoCrMo alloy metal-on-metal hip replacements have shown a great clinical performance. Metal-on-metal couplings produce a much lower wear rate and volume than e.g. metal-on-polyethylene. However, the particle size is significantly smaller within a nm-range. To evaluate the formation of nano-size wear particles in metal-on-metal hip replacements it is essential to understand the micro-structural changes in the sub-surface region of the CoCrMo alloy. For this study a MoM hip implant was analyzed by means of TEM. The results revealed that the good wear performance of this CoCrMo alloy is linked to a strain induced fcc  hcp phase transformation and in-situ re-crystallization under high shear stresses. The result is a nano-crystalline surface zone of ~200 to 400 nm thickness which undergoes an ongoing process of mechanical intermixing with componants of the interfacial fluid. The incorporation of organic carbon from proteins in between the nano-crystals could be visualised by EFTEM and EDS. This mechanically mixed nc-zone must be the origin of the wear particle detachment. An earlier study by Catelas et. al confirms the hypothesis of the location of wear particle detachment by analyzing the shape and chemical composition of emitted wear particles which exhibits the same size and shape of crystals observed in the nc-zone of the implant analyzed in this study.

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6

Siegel, Norman J., Prasad Devarajan, and Scott Van Why. "Renal Cell Injury: Metabolic and Structural Alterations." Pediatric Research 36, no. 2 (August 1994): 129–36. http://dx.doi.org/10.1203/00006450-199408000-00001.

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7

Chiu, Jane, Hana Farhangkhoee, Bing Ying Xu, Shali Chen, Biju George, and Subrata Chakrabarti. "PARP mediates structural alterations in diabetic cardiomyopathy." Journal of Molecular and Cellular Cardiology 45, no. 3 (September 2008): 385–93. http://dx.doi.org/10.1016/j.yjmcc.2008.06.009.

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8

Chong, Catherine D., Jonathan D. Plasencia, David H. Frakes, and Todd J. Schwedt. "Structural alterations of the brainstem in migraine." NeuroImage: Clinical 13 (2017): 223–27. http://dx.doi.org/10.1016/j.nicl.2016.10.023.

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9

Dyck, P. J., A. C. Lais, C. Giannini, and J. K. Engelstad. "Structural alterations of nerve during cuff compression." Proceedings of the National Academy of Sciences 87, no. 24 (December 1, 1990): 9828–32. http://dx.doi.org/10.1073/pnas.87.24.9828.

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10

FEITOSA, V., S. RECCOPIMENTEL, and A. CARDOSO. "Chromosomal analysis of : Presence of structural alterations." Cell Biology International Reports 14 (September 1990): 81. http://dx.doi.org/10.1016/0309-1651(90)90426-y.

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11

Guerin, A. P., S. J. Marchais, F. Metivier, and G. M. London. "Arterial structural and functional alterations in uraemia." European Journal of Clinical Investigation 35, s3 (December 2005): 85–88. http://dx.doi.org/10.1111/j.1365-2362.2005.01534.x.

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12

Smith, C. D., H. Chebrolu, D. R. Wekstein, F. A. Schmitt, G. A. Jicha, G. Cooper, and W. R. Markesbery. "Brain structural alterations before mild cognitive impairment." Neurology 68, no. 16 (April 16, 2007): 1268–73. http://dx.doi.org/10.1212/01.wnl.0000259542.54830.34.

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13

Aglietti, E. F., J. M. Porto Lopez, and E. Pereira. "Structural alterations in kaolinite by acid treatment." Applied Clay Science 3, no. 2 (May 1988): 155–63. http://dx.doi.org/10.1016/0169-1317(88)90015-4.

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14

Štol, M., I. Cífková, V. Tyrác̆ková, and M. Adam. "Structural alterations of p(HEMA)-collagen implants." Biomaterials 12, no. 5 (July 1991): 454–60. http://dx.doi.org/10.1016/0142-9612(91)90142-w.

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15

Coda, A., R. Bendavid, F. Botto-Micca, M. Bossotti, and A. Bona. "Structural alterations of prosthetic meshes in humans." Hernia 7, no. 1 (March 2003): 29–34. http://dx.doi.org/10.1007/s10029-002-0089-6.

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16

Obaid, Sami, François Rheault, Manon Edde, Guido I. Guberman, Etienne St-Onge, Jasmeen Sidhu, Alain Bouthillier, et al. "Structural Connectivity Alterations in Operculo-Insular Epilepsy." Brain Sciences 11, no. 8 (August 5, 2021): 1041. http://dx.doi.org/10.3390/brainsci11081041.

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Operculo-insular epilepsy (OIE) is an under-recognized condition that can mimic temporal and extratemporal epilepsies. Previous studies have revealed structural connectivity changes in the epileptic network of focal epilepsy. However, most reports use the debated streamline-count to quantify ‘connectivity strength’ and rely on standard tracking algorithms. We propose a sophisticated cutting-edge method that is robust to crossing fibers, optimizes cortical coverage, and assigns an accurate microstructure-reflecting quantitative conectivity marker, namely the COMMIT (Convex Optimization Modeling for Microstructure Informed Tractography)-weight. Using our pipeline, we report the connectivity alterations in OIE. COMMIT-weighted matrices were created in all participants (nine patients with OIE, eight patients with temporal lobe epilepsy (TLE), and 22 healthy controls (HC)). In the OIE group, widespread increases in ‘connectivity strength’ were observed bilaterally. In OIE patients, ‘hyperconnections’ were observed between the insula and the pregenual cingulate gyrus (OIE group vs. HC group) and between insular subregions (OIE vs. TLE). Graph theoretic analyses revealed higher connectivity within insular subregions of OIE patients (OIE vs. TLE). We reveal, for the first time, the structural connectivity distribution in OIE. The observed pattern of connectivity in OIE likely reflects a diffuse epileptic network incorporating insular-connected regions and may represent a structural signature and diagnostic biomarker.

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17

Doucet, Michele, Irene Londoño, Amparo Gómez-Pascual, and Moise Bendayan. "Glomerular Basement Membrane Selective Permeability in Short-term Streptozotocin-induced Diabetic Rats." International Journal of Experimental Diabetes Research 1, no. 1 (2000): 19–30. http://dx.doi.org/10.1155/edr.2000.19.

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In diabetes, the glomerular basement membrane undergoes thickening and structural alterations with loss of glomerular permselectivity properties. However, the onset of the alterations at early phases of diabetes is unclear. Aiming to determine the functional and structural alterations of the glomerular wall in the early stages of diabetes, we have studied the distribution of endogenous circulating albumin and type IV collagen in the glomerular basement membrane, using the immunocytochemical approach. The streptozotocin-injected hyperglycemic rat was our animal model. Renal tissues were examined after 10 days, 2, 4 and 6 months of hyperglycemia. Upon immunogold labelings, changes in the glomerular permeability to endogenous albumin were found altered as early as upon ten days of hyperglycemia. In contrast, no structural modifications were detected at this time point. Indeed, glomerular basement membrane thickening and an altered type IV collagen labeling distribution were only observed after four months of hyperglycemia, suggesting that functional alterations take place early in diabetes prior to any structural modification. In order to evaluate the reversibility of the glomerular alterations, two-month-old diabetic animals were treated with insulin. These animals showed a significant restoring of their glomerular permselectivity. Our results suggest a link between glycemic levels and alteration of glomerular permeability in early stages of diabetes, probably through high levels of glycated serum proteins.

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18

Lee, Robert M. K. W. "Structural alterations of blood vessels in hypertensive rats." Canadian Journal of Physiology and Pharmacology 65, no. 8 (August 1, 1987): 1528–35. http://dx.doi.org/10.1139/y87-241.

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Vascular changes in the mesenteric arteries were examined in three animal models for human essential hypertension. These models are: spontaneously hypertensive rats, which develop hypertension with age; Dahl model of genetic, salt-dependent hypertensive rats; and deoxycorticosterone–salt hypertensive rats. Morphometric measurements of the arterial wall components (e.g., endothelium, media) were carried out in the elastic arteries, muscular arteries, and arteriolar vessels from the mesenteric bed. The observed changes were correlated with the stages of hypertension development and the effect of antihypertension therapy, including sympathectomy. Specific emphasis was made to determine whether the changes observed were primary in nature, and related to the causes of hypertension, or they were secondary adaptive changes. A comparison of the three models showed that common changes in the intima, media, and adventitia were present in the three models. Alterations in the endothelium (e.g., enlargement of subendothelial space, necrotic changes), adventitia (collagen increase), and hypertrophy of the smooth muscle cells are secondary adaptive changes, because these changes occur subsequent to the development of hypertension, and antihypertensive therapy also prevent these changes from taking place. In contrast, hyperplasia of the smooth muscle cells is a primary change, because it occurs prior to the onset of hypertension. Functionally, alteration in the media is probably the most important change, because it can cause hyperreactivity of the arteries in response to stimulation. Damage to the endothelial cells may play a role in the maintenance of hypertension during the later phase. Alteration in adventitia is a passive change, which does not appear to have a major role in hypertension. Sympathectomy studies suggest that primary smooth muscle change may be mediated by the sympathetic nervous system.

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19

Durand, Geneviève, and Nathalie Seta. "Protein Glycosylation and Diseases: Blood and Urinary Oligosaccharides as Markers for Diagnosis and Therapeutic Monitoring." Clinical Chemistry 46, no. 6 (June 1, 2000): 795–805. http://dx.doi.org/10.1093/clinchem/46.6.795.

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Abstract Background: N- and O-oligosaccharide variants on glycoproteins (glycoforms) can lead to alterations in protein activity or function that may manifest themselves as overt disease. Approach: This review summarizes those diseases that are known to be the result of an inherited or acquired glycoprotein oligosaccharide structural alteration and that are diagnosed in blood or urine by chemical characterization of that oligosaccharide alteration. Content: The biochemical synthesis steps and catabolic pathways important in determining glycoprotein function are outlined with emphasis on alterations that lead to modified function. Clinical and biochemical aspects of the diagnosis are described for inherited diseases such as I-cell disease, congenital disorders of glycosylation, leukocyte adhesion deficiency type II, hereditary erythroblastic multinuclearity with a positive acidified serum test, and Wiskott-Aldrich syndrome. We also review the laboratory use of measurements of glycoforms related to acquired diseases such as alcoholism and cancer. Conclusions: Identification of glycoprotein glycoforms is becoming an increasingly important laboratory contribution to the diagnosis and management of human diseases as more diseases are found to result from glycan structural alterations.

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20

Borges, Fabiana Cristina Nascimento, Willian Rafael de Oliveira, and Jonas Kublitski. "Mechanical, structural and tribological properties of superaustenitic stainless steel submitted at solution heat treatment." Matéria (Rio de Janeiro) 20, no. 1 (March 2015): 160–68. http://dx.doi.org/10.1590/s1517-707620150001.0016.

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The superaustenitic stainless steel presents several technological applications, mainly in corrosive environments. The different phase precipitation might alter some of its mechanical properties. Such alterations affect several factors, including the working life of the material under adverse working conditions. In this study, Instrumented Indentation techniques, Tribology and X-ray diffraction (XRD) were used to evaluate alterations in regions close to the surface. The parameters analyzed were: hardness and elastic modulus (instrumented indentation), friction coefficient (tribology) and structural alterations of the unit cell of the identified phases (XRD - Rietveld Refinement). All properties analyzed were compared with those of common austenitic steel. The presence of σ-phase (space group P42mnm) and γ-austenite (space group Fm3m) were detected. Data analyzed indicated that the presence of σ-phase caused small alteration in properties such as hardness in regions close to the surface. In the regions farther from the surface (material bulk) data can be compared to that of conventional austenitic steel.

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21

Mehboob, Riffat. "Role of Epigenetic alterations in the development of cancers." Pakistan BioMedical Journal 5, no. 2 (February 28, 2022): 01. http://dx.doi.org/10.54393/pbmj.v5i2.346.

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Many different factors are involved in the progression of cancers. Genes mutations and chromosomal abnormalities are normally considered main cause of cancers but there are some other reason for the development of cancers. Other cancer causing factors are known as epigenetic alterations [1,2]. Epigentic modification of genome is known as epigenetic alterations, lead toward cancer cells production. Epigentic modification does not cause change in sequences of nucleotide. Similar to genetic alteration epigenetic alteration can’t be ignored [3]. Basically mechanisms behind epigenetic modifications are deregulation of DNA proteins, change in CpG island methylation, change in histone, oncogenes activation and deactivation of tumor suppressor [4]. Epigenetic alterations is directly linked with functional alterations of genome. Alteration in DNA methylation, histone degeneration and functional and structural abnormalities of chromosomes are the major examples of epigenetic modifications [5]. The main function of all epigenetic alterations is to modulate gene expression with same DNA sequences. Means these changes never effect main basal sequence oF DNA [6], which remain same in cell division [7]. Many different types of cancers contains large number of epigenetic alterations, the most important of these are epigenetic alterations that occurs in DNA repair genes. These DNA repair genes drive slow expression of DNA proteins. These abnormalities cause genetic unreliability, which is mainly considered as characteristic of various cancers [8,9].

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22

Hribar, Manja, Dušan Šuput, Saba Battelino, and Andrej Vovk. "Review article: Structural brain alterations in prelingually deaf." NeuroImage 220 (October 2020): 117042. http://dx.doi.org/10.1016/j.neuroimage.2020.117042.

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23

Pilatti, Mauricio Antônio, Deonir Secco, Luiz Antonio Zanão Jr, Araceli Ciotti de Marins, Luciene Kazue Tokura, and Bruna de Villa. "Structural Alterations of Paraná’s Oxisols by Cover Crops." Journal of Agricultural Science 10, no. 9 (August 13, 2018): 180. http://dx.doi.org/10.5539/jas.v10n9p180.

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This work aimed to evaluate the dynamics of physical and hydric attributes of a clayey Latosol cultivated with different cover species. The experimental area was located in the Agronomic Institute of Paraná (IAPAR), in the regional hub of Santa Tereza do Oeste, Paraná, Brazil. The experiment was comprised of seven cover species also called treatments is the course of this work. Three of them were isolated summer species notably Crotalaria juncea, Crotalaria spectabilis, Cajanus cajan (pigeon pea), and the other four treatments winter species cultivated individually or in association including Avena strigosa (Black oat), + (Avena stirgosa + Raphamus sativus (radish), Avena strigosa + Lupinus albus (Lupin bean), and (Avena strigosa + Pisum sativum (pea). The treatments were distributed on a completely random plots of 20 m × 25 m without replication. Soil density, macroporosity, and saturated hydraulic conductivity were measured to follow the changes of the soil structure. Statistical analyses showed that cover crops species did not lead to a significant improvement in soil structural status. Soil density varied between 1.08 and 1.12 Mg m-3, macroporosity from 15.22 and 16.90%, and saturated hydraulic conductivity ranged from 28.83 to 45.07 mm h-1. Soybean grain yield were considered satisfactory in 2016 (mean = 1909.68 kg ha-1) and in 2017 (mean = 3355.30 kg ha-1) most probably due to the good initial structural conditions of the soil, alongside with the good climatic conditions during the two campaigns. Furthermore, the soybean grain yield was positively influenced by Ds which ranged from 1.0 to 1.17 Mg m-3.

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24

Palmar, Maria, Arelis Marcano, and Orlando Castejon. "Fine structural alterations of blood platelets in depression." Biological Psychiatry 42, no. 10 (November 1997): 965–68. http://dx.doi.org/10.1016/s0006-3223(97)00348-x.

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25

Charalambopoulou, G. Ch, Th A. Steriotis, Th Hauss, A. K. Stubos, and N. K. Kanellopoulos. "Structural alterations of fully hydrated human stratum corneum." Physica B: Condensed Matter 350, no. 1-3 (July 2004): E603—E606. http://dx.doi.org/10.1016/j.physb.2004.03.161.

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26

Zweiman, B. "Airway structural alterations selectively associated with severe asthma." Journal of Allergy and Clinical Immunology 112, no. 3 (September 2003): 639. http://dx.doi.org/10.1016/s0091-6749(03)01634-8.

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27

Fellgiebel, Andreas. "Stroke and brain structural alterations in fabry disease." Clinical Therapeutics 29 (2007): S9—S10. http://dx.doi.org/10.1016/s0149-2918(07)80118-4.

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28

Rizzoni, Damiano, Carolina De Ciuceis, Enzo Porteri, Francesco Semeraro, and Enrico Agabiti Rosei. "Structural Alterations in Small Resistance Arteries in Obesity." Basic & Clinical Pharmacology & Toxicology 110, no. 1 (October 6, 2011): 56–62. http://dx.doi.org/10.1111/j.1742-7843.2011.00786.x.

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29

Maleki, Nasim, Lino Becerra, Jennifer Brawn, Marcelo Bigal, Rami Burstein, and David Borsook. "Concurrent functional and structural cortical alterations in migraine." Cephalalgia 32, no. 8 (May 23, 2012): 607–20. http://dx.doi.org/10.1177/0333102412445622.

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30

Foran, Jared RH, Suzanne Steinman, Ilona Barash, Henry G. Chambers, and Richard L. Lieber. "Structural and mechanical alterations in spastic skeletal muscle." Developmental Medicine and Child Neurology 47, no. 10 (September 12, 2005): 713. http://dx.doi.org/10.1017/s0012162205001465.

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31

Afonso, S. G., R. Enriquez de Salamanca, and A. M. Del C. Batlle. "Porphyrin-induced protein structural alterations of heme enzymes." International Journal of Biochemistry & Cell Biology 29, no. 8-9 (August 1997): 1113–21. http://dx.doi.org/10.1016/s1357-2725(97)00045-9.

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32

Mehnert, Jan, and Arne May. "Functional and structural alterations in the migraine cerebellum." Journal of Cerebral Blood Flow & Metabolism 39, no. 4 (July 24, 2017): 730–39. http://dx.doi.org/10.1177/0271678x17722109.

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The cerebellum plays an important role in pain processing but its function in headache and specifically in migraine is not known. We therefore compared 54 migraineurs with pairwise matched healthy controls in a magnetic resonance imaging study on neuronal cerebellar activity in response to nociceptive trigeminal sensation and also investigated possible structural alterations. Headache frequency, disease duration, and the proximity to a migraine attack were used as co-factors. Migraine patients showed functional and structural alterations in the posterior part of the cerebellum, namely crus I and crus II. Gray matter volume changes were seen on the right side whereas functional changes were ipsilateral to the stimulation, on the left side. Neuronal activity in the crus in response to trigeminal pain was modulated by migraine severity and the migraine phase. As the crus is strongly interconnected to higher cognitive areas in the temporal, frontal, and parietal part of the cortex our results suggest an specific cerebellar involvement in migraine. This is further supported by our finding of decreased connectivity from the crus to the thalamus and higher cortical areas in the patients. We therefore suggest an abnormally decreased inhibitory involvement of the migraine cerebellum on gating and nociceptive evaluation.

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33

Kong, J. Y., and S. W. Rabkin. "Palmitate induces structural alterations in nuclei of cardiomyocytes." Tissue and Cell 31, no. 5 (October 1999): 473–79. http://dx.doi.org/10.1054/tice.1999.0062.

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34

Chowdhury, Saheli, Curdin Ragaz, Emma Kreuger, and Franz Narberhaus. "Temperature-controlled Structural Alterations of an RNA Thermometer." Journal of Biological Chemistry 278, no. 48 (September 8, 2003): 47915–21. http://dx.doi.org/10.1074/jbc.m306874200.

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35

Lechleitner, Peter, Anton Theuerl, Franz Kroesslhuber, Gernot Walder, and Alfred Senfter. "Improving the results of resuscitation through structural alterations." Resuscitation 118 (September 2017): e69. http://dx.doi.org/10.1016/j.resuscitation.2017.08.168.

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36

Benayoun, Laurent, Anne Druilhe, Marie-Christine Dombret, Michel Aubier, and Marina Pretolani. "Airway Structural Alterations Selectively Associated with Severe Asthma." American Journal of Respiratory and Critical Care Medicine 167, no. 10 (May 15, 2003): 1360–68. http://dx.doi.org/10.1164/rccm.200209-1030oc.

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37

Sullivan, Edith V. "NEUROPSYCHOLOGICAL AND BRAIN STRUCTURAL ALTERATIONS IN HUMAN ALCOHOLISM." Alcoholism: Clinical & Experimental Research 28, Supplement (August 2004): 81A. http://dx.doi.org/10.1097/00000374-200408002-00448.

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38

Gorshkov, A. N., E. S. Snigirevskaya, and Ya Yu Komissarchik. "Arginine-vasopressin-induced structural alterations in MDCK cells." Cell and Tissue Biology 3, no. 2 (April 2009): 130–42. http://dx.doi.org/10.1134/s1990519x09020047.

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39

Valk, Sofie L., Adriana Di Martino, Michael P. Milham, and Boris C. Bernhardt. "Multicenter mapping of structural network alterations in autism." Human Brain Mapping 36, no. 6 (February 25, 2015): 2364–73. http://dx.doi.org/10.1002/hbm.22776.

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40

Pujol, Jesús, Carles Soriano-Mas, Pino Alonso, Narcís Cardoner, José M. Menchón, Joan Deus, and Julio Vallejo. "Mapping Structural Brain Alterations in Obsessive-Compulsive Disorder." Archives of General Psychiatry 61, no. 7 (July 1, 2004): 720. http://dx.doi.org/10.1001/archpsyc.61.7.720.

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41

Agabiti-Rosei, Enrico, Maria Lorenza Muiesan, and Damiano Rizzoni. "Cardiovascular Structural Alterations in Hypertension: Effect of Treatment." Clinical and Experimental Hypertension 18, no. 3-4 (January 1996): 513–22. http://dx.doi.org/10.3109/10641969609088981.

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42

Schwartz, Alan R., James A. Rowley, David C. Thut, Solbert Permutt, and Philip L. Smith. "Structural Basis for Alterations in Upper Airway Collapsibility." Sleep 19, suppl_10 (December 1996): 184–88. http://dx.doi.org/10.1093/sleep/19.suppl_10.184.

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43

Jiang, Lin, Jingchun Liu, Caihong Wang, Jun Guo, Jingliang Cheng, Tong Han, Peifang Miao, Chen Cao, and Chunshui Yu. "Structural Alterations in Chronic Capsular versus Pontine Stroke." Radiology 285, no. 1 (October 2017): 214–22. http://dx.doi.org/10.1148/radiol.2017161055.

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44

Mittag, Hannelore. "Structural alterations inCandida albicansby caffeine and caffeine salts." Mycoses 37, no. 9-10 (September 1994): 337–41. http://dx.doi.org/10.1111/myc.1994.37.9-10.337.

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45

Foran, Jared RH, Suzanne Steinman, Ilona Barash, Henry G. Chambers, and Richard L. Lieber. "Structural and mechanical alterations in spastic skeletal muscle." Developmental Medicine & Child Neurology 47, no. 10 (February 13, 2007): 713–17. http://dx.doi.org/10.1111/j.1469-8749.2005.tb01063.x.

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46

Lasch, Peter, Tobias Petras, Oliver Ullrich, Jan Backmann, Dieter Naumann, and Tilman Grune. "Hydrogen Peroxide-induced Structural Alterations of RNase A." Journal of Biological Chemistry 276, no. 12 (December 13, 2000): 9492–502. http://dx.doi.org/10.1074/jbc.m008528200.

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47

Randles, Edward G., James R. Thompson, Douglas J. Martin, and Marina Ramirez-Alvarado. "Structural Alterations within Native Amyloidogenic Immunoglobulin Light Chains." Journal of Molecular Biology 389, no. 1 (May 2009): 199–210. http://dx.doi.org/10.1016/j.jmb.2009.04.010.

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48

Østerby, Ruth, Hans-Jacob Bangstad, Gudrun Nyberg, and Susanne Rudberg. "On glomerular structural alterations in type-1 diabetes." Virchows Archiv 438, no. 2 (February 19, 2001): 129–35. http://dx.doi.org/10.1007/s004280000311.

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49

Andrianova, I. A., A. A. Ponomareva, and R. I. Litvinov. "Structural Alterations of Monocytes in Systemic Lupus Erythematosus." BioNanoScience 7, no. 4 (August 12, 2017): 636–39. http://dx.doi.org/10.1007/s12668-017-0441-z.

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50

Weis, Michael. "Functional and Structural Alterations in Cardiac Allograft Vasculopathy." Journal of the American College of Cardiology 71, no. 13 (April 2018): 1457–58. http://dx.doi.org/10.1016/j.jacc.2018.01.063.

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