Relevant bibliographies by topics / Stress (Psychology) Post-traumatic stress disorder

Academic literature on the topic 'Stress (Psychology) Post-traumatic stress disorder'

Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Stress (Psychology) Post-traumatic stress disorder"

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Flouri, Eirini. "Post-Traumatic Stress Disorder (PTSD)." Journal of Interpersonal Violence 20, no. 4 (April 2005): 373–79. http://dx.doi.org/10.1177/0886260504267549.

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Newman, Martin. "Post-traumatic stress disorder." Bereavement Care 28, no. 1 (April 2009): 31–33. http://dx.doi.org/10.1080/02682620902746250.

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Nichols, Beverly L., and Ruth Czirr. "24/Post-Traumatic Stress Disorder." Clinical Gerontologist 5, no. 3-4 (November 18, 1986): 417–33. http://dx.doi.org/10.1300/j018v05n03_12.

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Gosselin, Anik. "Review of Post-traumatic stress disorder." Canadian Psychology/Psychologie canadienne 54, no. 2 (2013): 141–42. http://dx.doi.org/10.1037/a0032347.

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McGuire, Brian. "Post-Traumatic Stress Disorder: A Review." Irish Journal of Psychology 11, no. 1 (January 1990): 1–23. http://dx.doi.org/10.1080/03033910.1990.10557784.

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Litz, Brett T., and Lizabeth Roemer. "Post-Traumatic Stress Disorder: An Overview." Clinical Psychology & Psychotherapy 3, no. 3 (September 1996): 153–68. http://dx.doi.org/10.1002/(sici)1099-0879(199609)3:3<153::aid-cpp98>3.0.co;2-5.

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Davidson, J., Vivette Glover, Angela Clow, H. Kudler, K. Meador, and M. Sandler. "Tribulin in post-traumatic stress disorder." Psychological Medicine 18, no. 4 (November 1988): 833–36. http://dx.doi.org/10.1017/s0033291700009764.

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SynopsisTribulin (endogenous monoamine oxidase inhibitor/benzodiazepine receptor binding inhibitor) output was measured in the urine of 18 patients with post-traumatic stress disorder (PTSD) and 13 controls. The level of the two inhibitory activities was highly significantly correlated in the group as a whole. There was no difference between output of either inhibitor in patients and controls. However, when the PTSD group was subdivided according to various psychometric ratings, a pattern of output did emerge. Levels of both inhibitory activities were higher in agitated compared with non-agitated subjects, and lower in extroverts compared with introverts. This finding supports the view that tribulin output is raised in conditions of greater arousal.
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Brende, Joel O., and Richard Goldsmith. "Post-traumatic stress disorder in families." Journal of Contemporary Psychotherapy 21, no. 2 (1991): 115–24. http://dx.doi.org/10.1007/bf00953906.

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Davidson, Jonathan, Susan Roth, and Elana Newman. "Fluoxetine in post-traumatic stress disorder." Journal of Traumatic Stress 4, no. 3 (July 1991): 419–23. http://dx.doi.org/10.1002/jts.2490040309.

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Linning, Lisa M., and Christopher A. Kearney. "Post-Traumatic Stress Disorder in Maltreated Youth." Journal of Interpersonal Violence 19, no. 10 (October 2004): 1087–101. http://dx.doi.org/10.1177/0886260504269097.

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Dissertations / Theses on the topic "Stress (Psychology) Post-traumatic stress disorder"

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Newell, Tracey. "Neurocognition in Post-Traumatic Stress Disorder." Thesis, University of Southampton, 2009. https://eprints.soton.ac.uk/162759/.

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The negative behavioural and emotional symptoms of Post-Traumatic Stress Disorder (PTSD) have been extensively reported in the literature. However, much less is known about the neuropsychological and neurobiological characteristics of the disorder. This thesis consists of two papers, the first being a review which highlights the emerging picture of literature in the field of neuropsychology in PTSD, with particular reference to findings in those cognitive domains of general intellectual functioning, memory, attention and executive function. Given that the findings associated within these domains are mixed, the second paper reports the outcome from a neuropsychological study of cognitive differences that was conducted to contribute to current knowledge in the area of neurocognition and visual memory in PTSD in particular. Trauma exposure, current PTSD, depressive and anxiety symptoms and performance on a range of neuropsychological tests were examined in tertiary care outpatients with PTSD (n=26), individuals who had been exposed to severe trauma but without current PTSD (n=26), and healthy controls (n=26). In addition to previously reported deficits in verbal learning and fluency in PTSD, deficits in visual spatial memory were also found. These observable deficits in visual memory may reflect characteristic features of PTSD, such as reported difficulties in remembering certain aspects of traumatic events and the presence of visual flashbacks. It is uncertain whether these deficits represent a risk factor for PTSD, or a consequence of trauma, as suggested by research in animal models.
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Brown, Kristine L. "The Connection between Post-traumatic Stress Disorder and Suicide Behavior: What Links Post-traumatic Stress Disorder to Suicide?" University of Toledo / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1372598026.

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Isaac, Claire L. "Cognitive function in post-traumatic stress disorder." Thesis, University of Warwick, 2002. http://wrap.warwick.ac.uk/2358/.

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Complaints of poor memory by individuals with posttraumatic stress disorder (PTSD) have engendered research into attention and memory functioning in this disorder. Due to numerous methodological difficulties encountered in research with this group, results have been inconclusive. In Chapter 1 of this thesis the existing literature is reviewed to ascertain whether there is any evidence of a specific pattern of memory disorder associated with PTSD. Studies are reviewed for evidence of cognitive deficits relating to the structures of the limbic system. dysfunction in which has been implicated in PTSD. It is concluded that there is relatively good evidence of deficits related to probable frontal lobe functions. However, there is very little evidence of hippocampal related disorders and no studies have investigated memory functions relating to hypothesised roles of the amygdala in this group. In chapters 2 and 3 experiments are described that aim to investigate cognitive abilities related to amygdala functioning in PTSD. Chapter 2 investigates an hypothesised role of the amygdala in the consolidation of memory for emotional material. The results confirm the possibility of amygdala dysfunction in PTSD by showing that on a test of free recall participants with PTSD forgot emotional word stimuli at a faster rate than control participants, whereas non-emotional stimuli were forgotten at a more normal rate. Chapter 3 investigated a second hypothesised role for the amygdala in the recognition of facial expressions of fear and anger. Results showed that PTSD participants were somewhat impaired in their recognition of these expressions, which contrasted with an enhanced ability, associated with symptoms of hyperarousal, in identifying other negative facial expressions. In Chapter 4, the relevance of neuropsychological research to Clinical Psychology is discussed. It is argued that such research is vital if we are to fully understand the difficulties clients could face on a day-to-day basis.
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Astorga, Delia Marie. "Educating veterans on Post Traumatic Stress Disorder." Thesis, California State University, Long Beach, 2015. http://pqdtopen.proquest.com/#viewpdf?dispub=1571852.

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The purpose of this project was to create program to identify funding sources, and write a grant to fund a support group for veterans who suffer from Post Traumatic Stress Disorder (PTSD) at the Department of Veterans Affairs of, Long Beach. The literature allowed this write to find the main causes of PTSD in this case being exposed to combat, Traumatic Brain Injury (TBO), and the consequences to PTSD (substance abuse, commit suicide, experience family conflicts). This writer also found Cognitive Processing Therapy (CPT), Prolonged Exposure Therapy (PET), and Eye Movement Desensitization and Reprocessing (EMDR) to be effective intervention in treating veterans with PTSD. The proposed program is aimed at providing psychoeducation to veterans and to help improve the lives of our service men and women who suffer from PTSD. The program includes group counseling, and individual counseling for veterans, family counseling. Providing the proper training will help social worker better assess and serve our veterans who return from combat with PTSD. Actual submission and/or funding of the grant were not required for the completion of this project.

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McKinnon, Aimee. "Assessing physiological sensitivity in Post-Traumatic Stress Disorder." Thesis, Cardiff University, 2017. http://orca.cf.ac.uk/110198/.

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People who have endured horrific events often bear predictable psychological harm. The symptoms of this harm can persist to produce a recognised clinical syndrome, Post-Traumatic Stress Disorder (PTSD). The lived reality of PTSD is a condition in which sufferers experience unbearable emotional reactions to traumatic reminders and exist in a persistent state of fear. Although the psychiatric and psychological construct of PTSD has been hotly contested, research and clinical opinion seem to converge around a state of enhanced sensitivity to threat, underpinned by chronic physiological hyper-arousal. This thesis has been concerned with the development of assessment measures that are sensitive to physiological hyper-arousal, including pupillometry and visual contrast sensitivity. In three experiments, a sample of 73 participants recruited from military, addiction and homelessness charity services were assessed for PTSD symptomology with the Clinician Administered PTSD Scale for DSM-V, and the self-report Impact of Event Scale-Revised. During passive viewing of emotive images, individuals with PTSD showed pupil responses that were influenced more by emotive stimuli than controls, and showed a reduced constriction of the pupil to light; revealing altered states of arousal. Due to methodological differences, a task assessing pupil responses to emotive sound clips failed to replicate this result. The assessment of visual contrast sensitivity revealed the heterogeneous nature of PTSD. Multi-dimensional assessment of symptom subscales showed that higher levels of re-experiencing symptoms were related to heightened visual sensitivity, but avoidant symptoms were related to lower sensitivity. Overall, the assessment of psychophysiological responses in PTSD demonstrated the utility of pupillometry for the assessment of PTSD, contributed to the literature on the regulation of the autonomic nervous system in PTSD, and highlighted the diversity of the clinical construct due to opposing effects of the symptom subscales.
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Street, Heidi Griffin. "Self psychology at work in trauma therapy : a project based upon an independent investigation /." View online, 2008. http://hdl.handle.net/10090/5935.

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Amstadter, Ananda Beth Laura L. Vernon Laura L. Burkhart Barry R. "Physiological effects of suppression of neutral and traumatic thoughts in posttraumatic stress disorder." Auburn, Ala, 2008. http://repo.lib.auburn.edu/EtdRoot/2008/SUMMER/Psychology/Dissertation/AMSTADTER_ANA_20.pdf.

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Reid, Louise Marie. "Traumatic brain injury, post-traumatic stress disorder symptom reporting and attentional bias : unravelling the misidentification of post-traumatic stress disorder in people with a traumatic brain injury." Thesis, University of Glasgow, 2009. http://theses.gla.ac.uk/1221/.

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Background: Post-traumatic stress disorder (PTSD) can occur following a traumatic event that has led to moderate to severe traumatic brain injury (TBI) even when there is little or no memory for the event. The incidence of PTSD is higher when diagnosed by self-report questionnaires compared to structured clinical interview. Previous studies suggest PTSD can be misdiagnosed in a significant proportion of cases and the incidence is in fact low. To explore this issue further there is a need to not only understand whether there are differences between cases that do and do not fulfill symptom criteria for PTSD, but also whether some cases have ‘partial PTSD’; that is to say they have PTSD symptoms but do not fulfill the DSM-IV symptom criteria exactly. Aims: The study aims to establish whether an attentional bias to trauma related words exists in people with TBI who report PTSD symptoms and to investigate the relationship between physiological arousal and attentional bias in people with a TBI reporting PTSD symptoms. Method: Forty-one participants with severe-extremely severe TBI were recruited from the community and completed measures of cognitive functioning. Attentional bias was measured using a Stroop task in which trauma, negative, neutral and positive words were administered randomly. Physiological reactivity (heart rate) was recorded and PTSD ‘caseness’ was established using a self-report questionnaire and a clinician-administered structured interview. Results: No significant relationship between PTSD symptom severities and attentional bias to trauma stimuli was apparent. Those with ‘PTSD’ demonstrated significantly slower reaction times to negative words however; this bias was associated with self-report of depression rather than PTSD symptomatology. Heart rate decreased throughout the interview and was not associated with PTSD symptom severities. Conclusions: Greater PTSD symptom reporting was not associated with an attentional bias to trauma words. Heart rate decreased over the course of the interview, independent of PTSD severity and diagnosis. This suggests that ‘partial’ PTSD was not present, and instead those who reported PTSD symptoms were curious about the gap in memory caused by amnesia without the associated fear response.
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Corbo, Vincent. "Interaction of fear and stress: from healthy population samples to post-traumatic stress disorder." Thesis, McGill University, 2011. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=104504.

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Fear and stress are two closely related psychological concepts. At the biological level, activity of the sympathetic nervous system (SNS) measured through galvanic skin response (GSR) is considered as a marker of fear in humans. In parallel, the secretion of cortisol consequent to the activation of the hypothalamic-pituitary-adrenal (HPA) axis has been identified as a reliable marker of stress. However, few human studies have investigated the interaction of endogenous cortisol and GSR in a pavlovian fear-conditioning design. Further, fear-conditioning has been used as a model for Post-Traumatic Stress Disorder (PTSD). This disorder is thought to be a failure to suppress exaggerated fearful reactions acquired at the time of trauma. Cortisol, as the main stress hormone, has been hypothesized as a potential modulator of the fearful reactions observed in PTSD. However, it remains unclear if PTSD is mostly a fear-based disorder or if symptoms may be associated to other factors, such as cortisol and brain structures, that are not part of the fear network.The work presented in this thesis followed two parallel lines. The two first chapters investigated the interaction between cortisol and GSR reactivity in healthy volunteers. We demonstrated that exposing subjects to a fear-conditioning paradigm was not enough to induce a cortisol response. Further, we observed a greater reactivity in women. In our second study, our results showed that an endogenous cortisol rise induced prior to extinction was associated with a faster decrease of the GSR response to the conditioned stimulus. Replicating our first study, we found that women reacted more to the conditioning paradigm compared to men. Lastly, while cortisol secretion was correlated with childhood adversity and anxiety trait, GSR reactivity did not correlate with personality measures.Our second line of investigation targeted civilians exposed to trauma. In our third study, we observed that increased levels of cortisol in response to awakening were associated with resilience to trauma. Furthermore, based on previous work investigating central nervous regulators of the HPA-axis and fear reactivity, our investigation of cortical thickness of individuals recently exposed to trauma confirmed the expected thinner ACC. We also highlighted the association between ventral temporal cortex and frontal pole with symptoms severity. These regions add a cognitive and social dimension to PTSD severity that may share more with stress than fear itself. These two studies argued for a more comprehensive model of PTSD that includes both fear-conditioning and stress reactivity to better account for the wide scope of symptoms.I conclude this thesis by re-examining the current proposed model for interaction between cortisol and peripheral measures of fear. I review the influence of sex as a mediator of fear acquisition, reactivity to stress and extinction of fear. Finally, I extend these findings to our PTSD studies to evaluate the use of pure fear-conditioning as a model for PTSD symptoms emergence and maintenance.
La peur et le stress sont deux concepts psychologiques intimement reliés. Au niveau biologique, l'activité du système nerveux sympathique (SNS), mesuré par la réponse électrodermale (RÉD), est considéré comme un marqueur de la peur chez l'être humain. Parallèlement, la sécrétion de cortisol suite à l'activation de l'axe hypothalamo-hypophyso-adrénergique (HHA) est le marqueur le plus commun du stress. Cependant, peu d'études se sont penchées sur l'interaction entre le cortisol et la RÉD lors d'un conditionnement de peur pavlovien chez l'être humain. De plus, le conditionnement de peur est utilisé comme modèle pour étudier le Trouble de Stress Post-Traumatique (TSPT). Ce trouble est considéré comme un échec de supprimer une réaction de peur exagérée acquise lors du traumatisme. Le cortisol, en tant qu'hormone de stress principale, est considéré comme un agent qui influencerait la force des réactions de peur dans le TSPT. Cependant, il demeure incertain si le TSPT est principalement un trouble relié à la peur ou si sa symptomatologie est relié à d'autres facteurs, tels le cortisol ou des structures neurologiques qui ne sont pas associées au système de la peur.Les travaux de cette thèse suivent deux lignes parallèles. Les deux premiers chapitres présentent les résultats de l'étude de l'interaction entre la peur et le stress chez des participants en santé. Nous illustrons que l'exposition à un conditionnement de peur n'est pas suffisant pour provoquer une réponse de cortisol. De plus, nous avons observé une plus forte réactivité au conditionnement chez les femmes. Les résultats de notre deuxième étude indiquent qu'une augmentation de cortisol endogène est associé à un déclin plus rapide de la réponse au stimulus conditionné lors de l'extinction. Cette étude confirme aussi une plus forte réactivité chez les femmes. Enfin, alors que la sécrétion de cortisol est associée à l'adversité durant l'enfance et l'anxiété, la RÉD n'était pas associée aux traits de personnalité.Parallèlement à ces études, nous avons étudiés des civils exposés à un événement traumatique. Notre troisième étude montre qu'une réponse accrue de cortisol en réaction au réveil est associée à la résilience face à un événement traumatique. De plus, notre étude de l'épaisseur corticale a confirmé que, chez des individus récemment exposés à un événement traumatique, le cortex cingulaire antérieur est correlé négativement à la sévérité des symptômes. Cette étude a aussi mis en lumière deux nouvelles structures, le cortex ventro-temporal et le pôle frontal, qui sont associées à la sévérité des symptômes. Ces deux structures ajoutent une dimension cognitive et sociale à la sévérité du TSPT et sont associés plus fortement au stress qu'à la peur en soi. Elles suggèrent donc un modèle d'étude qui va au-delà du conditionnement de peur et qui intègre l'importance du stress pour mieux décrire la symptômatologie.Je conclue cette thèse en réexaminant le modèle d'interaction entre le stress et les mesures périphériques de la peur. Suivant cela, j'examine le sexe comme médiateur possible dans l'apprentissage de peur, la réactivité au stress et l'extinction de la peur. Enfin, je fais le pont entre les premières études et celles sur le TSPT pour évaluer l'usage du pur conditionnement de peur comme modèle pour décrire l'émergence et le maintient des symptômes.
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Hinkly, David James. "Post-traumatic stress disorder and substance use in military veterans." Thesis, University of Birmingham, 2013. http://etheses.bham.ac.uk//id/eprint/4026/.

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The current literature review evaluated the effectiveness of published treatments for military veterans with co-occurring posttraumatic stress disorder (PTSD) and substance use disorders (SUD). The review begins with a summary of background issues pertinent to the treatment of military veterans with PTSD/SUD. These include: estimates of the prevalence of PTSD/SUD in this population; existing arguments regarding treatment delivery; and the rationale for the review. This is followed by a description of the methods used to select and methodologically evaluate the research literature. Fifteen studies were selected for inclusion in the review and were grouped as follows: psychosocial SUD-only treatment; pharmacological SUD-only treatment; ‘present-focussed’ joint PTSD/SUD treatment; and combined ‘past-’ and ‘present-focussed’ joint PTSD/SUD treatment. The best available evidence for SUD-only treatments was for the effectiveness of disulfiram and naltrexone on alcohol use outcomes, for disulfiram on PTSD outcomes, and for the use of opiate substitution therapy with heroin-dependent veterans. The strongest evidence for present-focussed joint PTSD/SUD treatment was for the effectiveness of the ‘Seeking Safety’ protocol in reducing drug use. Preliminary evidence was found for the effectiveness of combined past- and present-focussed joint PTSD/SUD treatment adopting CBT-orientated approaches. The implications of these findings for further research and treatment delivery are discussed. The current paper presents a qualitative study exploring military veterans’ perspectives on the relationship between their use of alcohol and their experiences of post-traumatic stress. It focuses on participants’ motives for using alcohol and their perceptions of the impact of their use on their subjective experiences of post-traumatic stress. Six male participants were recruited from a treatment centre specialising in the treatment of military veterans diagnosed with PTSD. Each participant was interviewed separately using a semi-structured interview schedule and the resulting transcripts were analysed using Interpretative Phenomenological Analysis (IPA). The paper reports the findings of this analysis, and these are discussed in relation to relevant literature. The analysis suggested that participants had used alcohol to ‘self-medicate’ distressing post-traumatic stress symptoms. Participants’ accounts suggested that alcohol had been partially effective at blocking out, or reducing the intensity of symptoms in the short-term, but that longer-term use had led to an exacerbation in symptoms. Alcohol had also been used by some participants to facilitate dissociative states and to enable engagement in social and work activities. The study’s strengths and limitations, as well as implications for clinical practice and future research, are presented.
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Books on the topic "Stress (Psychology) Post-traumatic stress disorder"

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Post traumatic stress disorder. Hoboken, N.J: Wiley, 2006.

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J, Scott Michael. Counselling for post-traumatic stress disorder. London: Sage Publications, 1995.

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J, Scott Michael. Counselling for post-traumatic stress disorder. 3rd ed. London: SAGE Publications, 2006.

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J, Scott Michael. Counselling for post-traumatic stress disorder. London: SAGE, 1992.

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Understanding combat related post traumatic stress disorder. Jefferson, N.C: McFarland & Co., Publishers, 2012.

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author, Kendall-Tackett Kathleen A., ed. Psychology of trauma 101. New York: Springer Publishing Company, 2015.

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1945-, Rosen Gerald M., and Frueh B. Christopher, eds. Clinician's guide to post traumatic stress disorder. Hoboken, N.J: John Wiley & Sons, 2010.

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Inc, ebrary, ed. Posttraumatic stress disorder: Diagnosis and assessment. Washington, DC: National Academies Press, 2006.

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William, Yule, ed. Post-traumatic stress disorders: Concepts and therapy. Chichester: Wiley, 1999.

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Anxiety disorders: Generalized anxiety disorder, obsessive-compulsive disorder and post-traumatic stress disorder. Malden, Mass: Blackwell Science, 2005.

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Book chapters on the topic "Stress (Psychology) Post-traumatic stress disorder"

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Gros, Daniel F., Peter W. Tuerk, Matthew Yoder, and Ron Acierno. "Post-Traumatic Stress Disorder." In Handbook of Clinical Psychology Competencies, 785–810. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-0-387-09757-2_28.

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Poirier, Joseph G., and Harold V. Hall. "Post-Traumatic Stress Disorder." In Forensic Psychology and Neuropsychology for Criminal and Civil Cases, 403–36. 2nd ed. Boca Raton: CRC Press, 2021. http://dx.doi.org/10.4324/9781003213307-13.

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Kerr, Laura K. "Post-traumatic Stress Disorder (PTSD)." In Encyclopedia of Critical Psychology, 1466–68. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-5583-7_347.

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Foa, Edna B. "Post-Traumatic Stress Disorder: Clinical Features and Treatment." In Perspectives and Promises of Clinical Psychology, 171–75. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4899-3674-5_15.

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Schumm, Jeremiah A., Leanna J. Pittsenbarger, and Caroline A. McClellan. "Post-traumatic Stress Disorder and Chronic Pain Among Military Members and Veterans." In Clinical Health Psychology in Military and Veteran Settings, 219–37. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-12063-3_10.

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Stellman, Jeanne Mager, and Steven D. Stellman. "Chronic disease risks and service-related post-traumatic stress disorder in military veterans." In The Routledge International Handbook of Military Psychology and Mental Health, 451–65. Abingdon, Oxon; New York, NY : Routledge, 2020.: Routledge, 2019. http://dx.doi.org/10.4324/9780429281266-30.

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Figley, Charles R. "Systemic traumatization: Secondary traumatic stress disorder in family therapists." In Integrating family therapy: Handbook of family psychology and systems theory., 571–81. Washington: American Psychological Association, 1995. http://dx.doi.org/10.1037/10172-033.

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Riggs, David S., and Sybil Mallonee. "Barriers to Care for the Complex Presentation of Post-traumatic Stress Disorder and Other Post-combat Psychological Injuries." In Handbook of Military Psychology, 33–44. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-66192-6_3.

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Vermetten, E., D. S. Charney, and J. D. Bremner. "Post-traumatic Stress Disorder." In Contemporary Psychiatry, 1849–91. Berlin, Heidelberg: Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-642-59519-6_120.

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Yule, William, and Patrick Smith. "Post traumatic stress disorder." In Rutter's Child and Adolescent Psychiatry, 806–21. Chichester, UK: John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781118381953.ch59.

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Conference papers on the topic "Stress (Psychology) Post-traumatic stress disorder"

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Kondashevskaya, M., and M. Lapshin. "KEY PATHOGENESIS FACTORS AT MODELING POST-TRAUMATIC STRESS DISORDER." In XVIII INTERNATIONAL INTERDISCIPLINARY CONGRESS NEUROSCIENCE FOR MEDICINE AND PSYCHOLOGY. LCC MAKS Press, 2022. http://dx.doi.org/10.29003/m2793.sudak.ns2022-18/173-174.

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Chernyavtseva, Tatyana. "ALGORITHMS FOR THE DIAGNOSIS OF COMBAT STRESS OF RESCUERS AND POST-TRAUMATIC STRESS DISORDER (PTSD)." In XVII INTERNATIONAL INTERDISCIPLINARY CONGRESS NEUROSCIENCE FOR MEDICINE AND PSYCHOLOGY. LCC MAKS Press, 2021. http://dx.doi.org/10.29003/m2400.sudak.ns2021-17/422-423.

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Wahyuni, Hera, Hamidah, and Nurul Hartini. "The Dynamics of Post-traumatic Stress Disorder among Sexual Violence Victims." In International Conference on Psychology in Health, Educational, Social, and Organizational Settings. SCITEPRESS - Science and Technology Publications, 2018. http://dx.doi.org/10.5220/0008588302830289.

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Belinskaya, Elena, Alexandr Vecherin, and Elena Agadullina. "The relationship of proactive coping and severity of symptoms of post-traumatic stress disorder." In Proceedings of the II International Scientific-Practical Conference "Psychology of Extreme Professions" (ISPCPEP 2019). Paris, France: Atlantis Press, 2019. http://dx.doi.org/10.2991/ispcpep-19.2019.3.

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Jan, Zala, Christian Gostečnik, and Veronika Kralj-Iglič. "Adverse Human Health Outcomes Associated with Psychologi-cal Trauma: A review." In Socratic Lectures 7. University of Lubljana Press, 2022. http://dx.doi.org/10.55295/psl.2022.d7.

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Until 30 years ago it was believed that psychological stress increases cortisol secretion, but later stud-ies gave contradictory results. Decrease in cortisol levels in post-traumatic stress disorder (PTSD) reflects a nonnormative and inadequate response to severe stressors, with its pathophysiology in-volving maladaptation or dysfunction in stress-regulatory systems. To have more insights in re-sponse of human body to physiological stress, inflammatory signals, oxidative stress parameters and other health parameters were measured. As for the cortisol level results, also inflammatory signals, including proinflammatory and anti-inflammatory cytokines and C-reactive protein (CRP), have been reported to increase and decrease in PTSD. Levels of interleukin (IL)-1β, IL-2, IL-4, IL-6, IL-8, IL-10, tumour necrosis factor (TNF)-α, interferon gamma (IFN-γ) and CRP were reported higher and lower in blood samples of individuals with PTSD. Some studies report that dysregulation of the stress axis could have direct effects on brain regions responsible for the regulation of fear and anxiety (such as the prefrontal cortex, insula, amygdala, and hippocampus). Early-life stress, such as child-hood adversity (abuse, neglect, or family disfunction), is a potent risk factor for developing PTSD in response to later trauma, and elevated peripheral markers of inflammation are one of the best-repli-cated findings in children and adults with early-life stress. Those who develop PTSD may have an inability or failure to activate an innate immune response. PTSD can also result in other adverse outcomes, such as heightened oxidative stress (OXS), eating disorders, metabolic disorder, and car-diovascular disease (CVD). Since the results are very contradictory for PTSD and inflammation re-sponse of the human body, further research is important. Small cellular particles that can be isolated from body fluids present potential biomarkers of the clinical status and will be considered in plan-ning the future research. This contribution presents perspectives in assessment of psychological stress by objective parameters. Keywords: Cortisol; Post-traumatic stress disorder; Inflammatory response; Oxidative stress; Cyto-kines; Eating disorders; Metabolic disorder; Cardiovascular disease; Small cellular particles as stress markers, Extracellular vesicles as stress markers
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Corpuz, Abigail. "Adverse Childhood Experiences and complex PTSD: A Theoretical Model Exploring Psychedelic Drugs as a Therapeutic Treatment." In 7th International Conference on Spirituality and Psychology. Tomorrow People Organization, 2022. http://dx.doi.org/10.52987/icsp.2022.001.

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Abstract Complex posttraumatic stress disorder (cPTSD) has several detrimental consequences, including severe anxiety, emotional detachment, mood irregularities, and vivid flashbacks to trauma. In many cases, cPTSD can be linked back to adverse childhood experiences (ACEs). Treatments for cPTSD that involve psychedelic drugs are potentially beneficial, but unfortunately they are understudied in psychology labs due to their classification as a Schedule I substance. Thus, theoretical work is needed to explain potential mechanisms involved in treatment programs. In this new theoretical model, I clarify the mechanistic links between ACEs and cPTSD and then examine why psychedelic drugs may be an ideal therapeutic tool for the treatment of cPTSD. Toxic stress theory posits that exposure to extreme, frequent, and persistent ACEs without the presence of a supportive caretaker chronically activates the stress response system (Jones et al., 2021). Toxic stress results in dysregulation of the limbic-hypothalamic-pituitary-adrenal (LHPA) axis, elevating levels of catecholamines, cortisol, and proinflammatory cytokines (Thermo Fisher Scientific, n.d.). The toxic stress induced by ACEs causes cPTSD due to the persistent exposure to multiple adverse events leading to re-experience of the traumatic events, avoidance behaviors, and paranoia. Psychedelic drugs unlock repressed memories, engaging positively with negative self-concept and dysregulated emotions, which are both characteristic of the Disturbances of Self-Organization symptom cluster of cPTSD. Presentation of this theoretical model would allow for public recognition of the potential benefits of this treatment and further exploration into this topic. Keywords: PTSD, psychedelics, adverse childhood experiences, stress
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Landry, Brian M., Eun Kyoung Choe, Stephen McCutcheon, and Julie A. Kientz. "Post-traumatic stress disorder." In the ACM international conference. New York, New York, USA: ACM Press, 2010. http://dx.doi.org/10.1145/1882992.1883110.

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Costanzo, Heather. "Self-Compassion and Post-Traumatic Growth for Post-Traumatic Stress." In 7th International Conference on Spirituality and Psychology. Tomorrow People Organization, 2022. http://dx.doi.org/10.52987/icsp.2022.008.

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Abstract Self-compassion and Post-Traumatic Growth seem to be inextricably linked. An exploration of the relationship between the two might lend to strategies that potentially could maximize rapid Post-Traumatic Growth after trauma. Consequently, Post-Traumatic Stress, as it is a transitive and transitionary state of the psyche in which the self exists in fragmentation, may benefit from Compassion Focused Therapy as an effective form of early therapy after traumatic experiences. Most centrally, this paper investigates self-compassion’s role in Post- Traumatic Growth focusing on ongoing favorable inner dialogue and self-talk—prospective personal extensions of Compassion Focused Therapy—with habituation likely an important outcome for those who are affected by Post-Traumatic Stress. The discussion encompasses how to utilize self-compassion in one’s personal life and professional practice. Limitations, implications for individuals and professionals, and future research directions are broached. Keywords: Self-compassion, post-traumatic growth, post-traumatic stress
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Drissi, Nidal, Sofia Ouhbi, Mohammed Abdou Janati Idtissi, and Mounir Ghogho. "Mobile Apps for Post Traumatic Stress Disorder." In 2019 41st Annual International Conference of the IEEE Engineering in Medicine & Biology Society (EMBC). IEEE, 2019. http://dx.doi.org/10.1109/embc.2019.8857197.

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Low, Jen YF. "Forgiveness Meditation: Mindful Self-Healing." In 7th International Conference on Spirituality and Psychology. Tomorrow People Organization, 2022. http://dx.doi.org/10.52987/icsp.2022.004.

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Abstract Rising like lotus blooms from bloodied war-torn devastation and muddied destitution of war crimes, divided societies and imperialistic ravages of Western colonialism, the two Indochina nations of Vietnam and Cambodia have shown amazing power of resurgence in less than 50 years. In many regional league tables, Vietnam notably, have even pulled ahead to show amazing achievements in GDP and education. What has happened seems like a distant past today. What are the unique cultural roots of this human resilience and socio-economic dynamism? At an individual level, it is not often that post traumatic stress disorder of abused victims show their mental and emotional suffering. One can only note the behavioral signs which impede normal life and block success. Part of this presentation is to share with the audience the clues to help one recognize such indicative signs with the objective of supporting those who are suffering. There is an effective way to uproot the self-blame, anger and hatred associated with suppressed memories and to overcome the submerged negativities in subconscious minds of the afflicted. Forgiveness meditation is a mindful self-healing way of peaceful living, and when paired as an integral part Insight Meditation, the underlying benefits can empower the healed to progress onward to bigger success. Real-life cases of two personalities who have taken different paths to demonstrate the power of mindful living towards human resilience and effective healing in the midst of bleak uncertainties are shared: A. A Cambodian (multiple) noble peace prize nominee who demonstrated not only to his people, but also the world, to seek the only resource where we can find true peace and genuine understanding of truths... in our own hearts. Like many of his compatriots, his entire family, friends and disciples were massacred. A forest monk and meditation master turned peacemaker at the United Nations, he walked step by step bringing forth the spring of hope in the hearts of the shell shocked survivors. Tens of thousands wept as he chants the timeless metta verses of loving kindness and other traditional spiritual chants lost in the unspeakable sorrows of war and ideological conflicts. B. A postwar Vietnamese case study of a globally successful social entrepreneur, she was left to fend for herself aged 16 years after her entire family was killed by foreign powers. Her social enterprise employed the war destitutes, former prostitutes and the disabled to produce quality handicrafts and furniture made from organic resources. Her voice is recorded here to illustrate her maxim of “one must forgive to move on but the painful lessons must never be forgotten” in order to sustain success. Keywords: forgiveness, meditation, self-healing, mindfulness
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Reports on the topic "Stress (Psychology) Post-traumatic stress disorder"

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Grogin, Phillip W. Post-Traumatic Stress Disorder (PTSD). Office of Scientific and Technical Information (OSTI), July 2016. http://dx.doi.org/10.2172/1296630.

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Thomas, Steven A. Catecholamines in Post-Traumatic Stress Disorder. Fort Belvoir, VA: Defense Technical Information Center, July 2012. http://dx.doi.org/10.21236/ada585061.

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Murray, Mark A. Post Traumatic Stress Disorder: The Facts. Fort Belvoir, VA: Defense Technical Information Center, February 2007. http://dx.doi.org/10.21236/ada467315.

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Seibyl, John. Imaging Neuroinflammation in Post Traumatic Stress Disorder. Fort Belvoir, VA: Defense Technical Information Center, November 2012. http://dx.doi.org/10.21236/ada602887.

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Seibyl, John. SPECT Imaging to Evaluate Post Traumatic Stress Disorder. Fort Belvoir, VA: Defense Technical Information Center, February 2011. http://dx.doi.org/10.21236/ada581405.

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Arnold, Joe E. Emerging Generation of Post-Traumatic Stress Disorder Victims. Fort Belvoir, VA: Defense Technical Information Center, February 2011. http://dx.doi.org/10.21236/ada547398.

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Ogburn, Zeke. Best Practices for Treatment of Post-Traumatic Stress Disorder. Portland State University Library, January 2015. http://dx.doi.org/10.15760/honors.160.

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Tyrka, Audrey R. Biomarkers of Risk for Post-Traumatic Stress Disorder (PTSD). Fort Belvoir, VA: Defense Technical Information Center, May 2010. http://dx.doi.org/10.21236/ada552047.

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Krebs, Erin E. Post-Traumatic Stress Disorder and Pain Comorbidity in Veterans. Fort Belvoir, VA: Defense Technical Information Center, August 2011. http://dx.doi.org/10.21236/ada553762.

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Krebs, Erin E. Post-Traumatic Stress Disorder and Pain Comorbidity in Veterans. Fort Belvoir, VA: Defense Technical Information Center, December 2010. http://dx.doi.org/10.21236/ada554830.

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