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Journal articles on the topic 'Stress (Psychology) Immunological aspects'

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Published: 4 June 2021
Last updated: 1 August 2024

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1

Romaniuk, Volodymyr L. "STRESS-REACTIVITY AND STRESS-RESISTANCE AND MENTAL HEALTH OF PERSONALITY." Scientific Notes of Ostroh Academy National University: Psychology Series 1, no. 14 (January 27, 2022): 9–15. http://dx.doi.org/10.25264/2415-7384-2022-14-9-15.

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Human health, as a biopsychosocial phenomenon, substantially depends on the responsiveness of an organism under certain conditions of activity and existence. Regarding a human being, the following types of responsiveness have been determined: biological or species-level responsiveness; group or constitutional responsiveness; individual physiological responsiveness; individual pathological responsiveness; specific responsiveness; non-specific responsiveness. Stress as the general adaptation syndrome and non-specific responsiveness of an organism significantly affects the physical and mental health of a personality by dint of the fight-or-flight or escape mechanisms. Concurrently, the scope of reaction related to the fight or escape is used to characterize stress responsiveness, which is genetically determined and is strictly individual. Thus, the behavioral mechanism of fight or escape preeminently refers to a constituent of unconditioned-reflex and instinctive activities of the human organism. Furthermore, phylogenetic constituents of the fight or escape mechanism are essentially supplemented by ontogenetic experience, becoming firmly established at the level of conditioned-reflex and instrumental activities. As a result, the main adaptation mechanism of stress responsiveness or mechanism of fight or escape can be viewed as a unique functional combination of inherited and acquired responses (unconditioned-reflex and conditioned-reflex reactions), which will considerably determine somatic and psychic norms as well as pathology of an organism. In its turn, stress responsiveness is tightly connected with the stress resistance of an organism. Indeed, stress resistance is an individual capability of an organism to maintain normal working efficiency under the influence of a certain stressor. Stress resistance can be enhanced with the help of learning; the essence of this learning lies in the conscious control of the activities of particular organs and systems and the behavioral activity of an organism as a whole. In psychology, stress resistance serves as the basis for successful social cooperation of a personality, which is predominantly characterized by emotional stability, as well as a low level of anxiety. Hence, stress resistance as a constituent of mental health in systemic relations with stress responsiveness contributes to the stability of homeostasis indicators with the involvement of morphological and functional reserves of an organism. In general, it is expedient to emphasize the following forms of stress responsiveness – low stress responsiveness (hypo stress responsiveness), adequate stress responsiveness (normal stress responsiveness), and increased stress responsiveness (hyper stress responsiveness). At the same time, there is a probable negative influence of both decreased and increased stress responsiveness on the constituents of mental health of a personality. In a holistic way, stress responsiveness and stress resistance of an organism significantly depend on the particularities of the central nervous system and autonomic nervous system, as well as the specifics of temperament in the corresponding systemic relations. In this respect, the two most important aspects of temperament, such as activity and emotionality (including anxiety) are distinguished. The diagnostics of stress resistance in the functional connection with stress responsiveness, along with morphological and functional reserves of an organism can include the following levels: 1) morphological level; 2) physiological level; 3) hematological level; 4) biochemical level; 5) immunological level; 6) level of the emotional sphere; 7) level of the cognitive sphere; 8) behavioral level. Psychophysiological particularities of active leisure and cognitive-behavioral therapy are aimed at the strengthening of stress resistance of an organism, as well as preservation of mental health of a personality under the corresponding conditions of activity and existence.
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2

Magnon, Claire. "Cancer Builds a Noxious Partnership with Psychologic Stress." Cancer Research 84, no. 7 (April 1, 2024): 956–57. http://dx.doi.org/10.1158/0008-5472.can-24-0628.

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Abstract I was recently surprised to hear a medical doctor on a TV show refute the role of stress in cancer, assuming that “the whole population would have cancer if this was the case.” This statement illustrates a long and winding road since Hippocrates suggested the potential relationship between cancer and psychologic disturbances. The 20th and 21st centuries have finally witnessed the evidence of how physical or psychosocial stress situations contribute to the development and progression of cancer, and it is now assumed that psychologic stress does affect multiple aspects of cancer such as angiogenesis, immunologic escape, invasion, and metastasis. The 2010 publication by Sloan and colleagues in Cancer Research achieved a mechanistic step toward the understanding of how physical distress enhances metastasis through perturbation of the tumor immune system and paves the way for future cancer research in psychoneuroimmunology. This Landmark commentary places this publication in the historical context of science, discusses major advances in the field, and asks questions to be answered while drawing perspectives on the key role of the peripheral and central nervous systems in cancer. See related article by Sloan and colleagues, Cancer Res 2010;70:7042–52
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3

Brosschot, Jos F., Guido L. R. Godaert, Robert J. Benschop, Miranda Olff, Rudy E. Ballieux, and Cobi J. Heijnen. "Experimental Stress and Immunological Reactivity." Psychosomatic Medicine 60, no. 3 (1998): 359–61. http://dx.doi.org/10.1097/00006842-199805000-00024.

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4

Leonard, Brian E. "Stress and the Immune System: Immunological Aspects of Depressive Illness." International Review of Psychiatry 2, no. 3-4 (January 1990): 321–30. http://dx.doi.org/10.3109/09540269009026603.

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5

PROHASZKA, Z. "Immunological aspects of heat-shock proteins?the optimum stress of life." Molecular Immunology 41, no. 1 (May 2004): 29–44. http://dx.doi.org/10.1016/j.molimm.2004.02.001.

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6

Jonsdottir, Ingibjörg H., and Anna Sjörs Dahlman. "MECHANISMS IN ENDOCRINOLOGYEndocrine and immunological aspects of burnout: a narrative review." European Journal of Endocrinology 180, no. 3 (March 2019): R147—R158. http://dx.doi.org/10.1530/eje-18-0741.

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Burnout has several different definitions, and attempts have been made to discriminate between burnout as a psychological construct and burnout as a clinical entity. A large body of research has focused on elucidating the biological link between stress exposure and burnout and/or finding a clinically usable biomarker for burnout. The objective of this narrative review is to summarize the main endocrine and immune findings in relation to burnout. The literature has primarily focused on dysregulation of the hypothalamus-pituitary-adrenal (HPA) axis. However, albeit the large body of studies, it cannot be concluded that clear effects are seen on HPA axis function in people with burnout. The HPA axis and anabolic acute reactivity to stress might be affected in clinical burnout. Plausible, effects of chronic stress might rather be seen when measuring responses to acute stress rather than resting state hormonal levels. Studies on other hormones, including thyroid hormones, prolactin and growth hormone in burnout subjects are inconclusive. It is important to note that this field is faced with many methodological challenges, one being the diurnal and pulsatile nature of many of the hormones of interest, including cortisol, which is not always considered. Another challenge is the heterogeneity regarding definitions and measurements of stress and burnout. Existing studies on burnout and immune function are heterogeneous regarding the results and no firm conclusion can be made if clinically relevant immune changes are present in burnout subjects. An overall conclusion is that existing research cannot confirm any homogenous reliable endocrinological or immunological changes related to burnout.
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7

Tomaszewska, Katarzyna, Aleksandra Słodka, Bartłomiej Tarkowski, and Anna Zalewska-Janowska. "Neuro–Immuno–Psychological Aspects of Chronic Urticaria." Journal of Clinical Medicine 12, no. 9 (April 26, 2023): 3134. http://dx.doi.org/10.3390/jcm12093134.

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Urticaria is a condition characterized by the development of itchy wheals (hives), angioedema, or both. The pathophysiology of chronic spontaneous urticaria (CSU) is still poorly understood. It is suggested that there is no dominant and independent mechanism of CSU; however, there are different immunological and non-immunological abnormalities that act simultaneously or/and follow each other resulting in clinical symptoms. The latest hypothesis points out that mast cells (MCs) to be activated via autoantibodies in autoallergic or autoimmune mechanism mediators released from degranulated MCs are responsible for the vasoactive and neurospecific effect in CSU. According to many clinical observations, it is suggested that psychological stress can be both a triggering factor in the onset of CSU and a modulating one in the course of the disease and therapy effectiveness. Of importance, the mechanistic background of the psychological stress response in the skin has not yet been fully elucidated. However, of note, a variety of inflammatory mediators, neuropeptides, and neurotransmitters facilitate this phenomenon. This review presents recent findings on the neuro–immuno–psychological aspects of CSU, highlighting an emerging role of neuro–immune interactions. It also points out the usefulness of psychological tools employment for the baseline diagnosis of perceived stress level and the presence of its symptoms. Furthermore, it proposes the implementation of non-invasive interventions to reduce psychological stress and anxiety. A bio–psycho–social approach including psychological support and patient education seems to be as important as traditional pharmacotherapy for CSU. It facilitates the effective control of active disease and a prolonged remission time in this disease.
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8

Hulette, Christine M., and Roy L. Walford. "IMMUNOLOGICAL ASPECTS OF ALZHEIMER DISEASE: A REVIEW." Alzheimer Disease & Associated Disorders 1, no. 2 (1987): 72–82. http://dx.doi.org/10.1097/00002093-198701020-00002.

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9

Brosschot, J. F., R. J. Benschop, G. L. Godaert, M. Olff, M. De Smet, C. J. Heijnen, and R. E. Ballieux. "Influence of life stress on immunological reactivity to mild psychological stress." Psychosomatic Medicine 56, no. 3 (May 1994): 216–24. http://dx.doi.org/10.1097/00006842-199405000-00007.

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10

Zeitlin, Diane, Steven E. Keller, Samuel C. Shiflett, Steven J. Schleifer, and Jacqueline A. Bartlett. "Immunological Effects of Massage Therapy During Academic Stress." Psychosomatic Medicine 62, no. 1 (January 2000): 83–84. http://dx.doi.org/10.1097/00006842-200001000-00012.

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11

Moirasgenti, Maria, Karolina Doulougeri, Efharis Panagopoulou, and Theodoros Theodoridis. "Psychological stress reduces the immunological benefits of breast milk." Stress and Health 35, no. 5 (November 12, 2019): 681–85. http://dx.doi.org/10.1002/smi.2903.

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12

Eysenck, H. J. "Stress: Conceptual and biological aspects." Personality and Individual Differences 20, no. 6 (June 1996): 810–11. http://dx.doi.org/10.1016/0191-8869(96)83457-x.

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13

Podoprigora, Gennadiy Ignat'evich, Lyudmila Ivanovna Kafarskaya, Nikolay Alekseevich Baynov, and Andrey Nikolaevich Shkoporov. "Bacterial Translocation from Intestine: Microbiological, Immunological and Pathophysiological Aspects." Annals of the Russian academy of medical sciences 70, no. 6 (December 6, 2015): 640–50. http://dx.doi.org/10.15690/vramn564.

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Bacterial translocation (BT) is both pathology and physiology phenomenon. In healthy newborns it accompanies the process of establishing the autochthonous intestinal microbiota and the host microbiome. In immunodeficiency it can be an aethio-pathogenetic link and a manifestation of infection or septic complications. The host colonization resistance to exogenous microbic colonizers is provided by gastrointestinal microbiota in concert with complex constitutional and adaptive defense mechanisms. BT may be result of barrier dysfunction and self-purification mechanisms involving the host myeloid cell phagocytic system and opsonins. Dynamic cell humoral response to microbial molecular patterns that occurs on the mucous membranes initiates receptor signaling pathways and cascade of reactions. Their vector and results are largely determined by cross-reactivity between microbiome and the host genome. Enterocyte barriers interacting with microbiota play leading role in providing adaptive, homeostatic and stress host reactivity. Microcirculatory ischemic tissue alterations and inflammatory reactions increase the intestinal barrier permeability and BT. These processes a well as mechanisms for apoptotic cells and bacteria clearance are justified to be of prospective research interest. The inflammatory and related diseases caused by alteration and dysfunction of the intestinal barrier are reasonably considered as diseases of single origin. Maternal microbiota affects the formation of the innate immune system and the microbiota of the newborn, including intestinal commensal translocation during lactation. Deeper understanding of intestinal barrier mechanisms needs complex microbiological, immunological, pathophysiological, etc. investigations using adequate biomodels, including gnotobiotic animals.
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14

Klyushnik, T. P., A. B. Smulevich, S. A. Zozulya, D. V. Romanov, and V. M. Lobanova. "Clinical and Immunological Aspects of Delusional Disorders in Paranoid Schizophrenia." Psikhiatriya 21, no. 2 (May 6, 2023): 6–16. http://dx.doi.org/10.30629/2618-6667-2023-21-2-6-16.

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Background: insufficient study of heterogeneous delusional disorders in schizophrenia and the role of inflammation in the development of the disease served as the basis for this study. The aim: to establish the role of immune mechanisms in the processes of the interaction of different forms of delusional symptom complexes in schizophrenia. Patients: 60 patients (mean age 38.4 ± 1.11 years) with the diagnosis “paranoid schizophrenia, continuous progressive course” (F20.00, ICD-10) were included in the study. The state of patients was defined by persistent delusional/hallucinatory delusional disorders. Based on the clinical assessment, patients were divided into three groups: 27 patients (group 1) with interpretative delusion, 22 patients (group 2) with delusion of influence based on the phenomena of mental automatism, and 11 examinees (group 3) with mixed forms of delusions (interpretative and delusions of influence with mental automatism). The control group consisted of 17 mentally and somatically healthy people, comparable with the patients by sex and age. Methods: inflammatory and autoimmune markers leukocyte elastase (LE) and α1-proteinase inhibitor (α1-PI) activity, leukocyte inhibitor index (LII) and antibody (aAb) level to S100B and MBP were determined in the blood. Results: in all groups of patients, an increase in the activity of LE and α1-PI was revealed compared with the control (p < 0.05). In group 2, an increase in aAb level to S100B was also detected (p < 0.05). Intra-group differences in LE activity served as the basis for dividing patients into three clusters. Cluster 1 was characterized by moderate activation of the immune system and was represented mainly by patients with interpretative delusions (54.5% of patients in the corresponding clinical group). Clusters 2 and 3 were distinguished by a higher level of immune system activation. A distinctive feature of cluster 3 was low LE activity against the background of high α1-PI activity and elevated level of aAb to S100B. Clusters 2 and 3 were represented mainly by patients with delusion of influence (74.1%). Сonclusion: the study confirmed the involvement of inflammation in the pathophysiology of delusional disorders in paranoid schizophrenia and allowed us to identify the relationship between the psychopathological structure of these disorders and the features of the spectrum of immune markers. The highest level of activation of the immune system, as well as immunological features presumably indicating impaired permeability of the blood-brain barrier, were associated mainly with delusions of influence with the phenomena of mental automatism.
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Panaschatenko, A. S., I. A. Panova, A. I. Malyshkina, E. A. Rokotyanskaya, A. V. Kudryashova, N. Yu Sotnikova, L. V. Kulida, and E. V. Protsenko. "IMMUNOLOGICAL AND PATHOMORPHOLOGICAL ASPECTS OF EARLY AND LATE PREECLAMPSIA." Medical Immunology (Russia) 23, no. 4 (October 19, 2021): 845–52. http://dx.doi.org/10.15789/1563-0625-iap-2292.

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Preeclampsia (PE) is one of the most common complications of pregnancy, and it can be after 20 weeks of gestation. It ends only with a complete dissection of afterbirth. Traditionally, PE is subdivided into the early one, taking place through 34 weeks of pregnancy (EOPE) and the late one, which is after 34 weeks of gestation (LOPE). Clinical manifestations are similar in both cases however, risk factors and the severity of PE are different . It has been established that EOPE is determined by impaired trophoblast invasion and transformation of the spiral arteries of the uterus in early pregnancy, and late onset of PE is associated with oxidative stress of syncytiotrophoblast, which occurs secondarily, with limited gas exchange and insufficient intake of nutrients. Numerous studies have noted a significant contribution of immune responses to the pathogenesis of preeclampsia, however, the state of B-lymphocytes in EOPE and LOPE has not been studied. A comprehensive assessment of the condition of women with early (up to 34 weeks of pregnancy inclusive) and late (after 34 weeks) development of preeclampsia was carried out, taking into account clinical and anamnestic characteristics, the peculiarities of the formation of the structural components of the placenta, as well as determining the nature of differentiation and functional activity of B-lymphocytes. In peripheral venous blood, the content of CD19+, CD20+, CD19+CD27+IgD±, CD19+CD20- CD38+, CD20+CD5+-cells and serum levels of IL-5, IL-9, IL-13 were examined. Morphological examination included gross description, organometry, survey histology, and transmission electron microscopy. In the group of women with early preeclampsia in history, there were more often perinatal losses, premature births and medical abortions, and in the current pregnancy, intrauterine infection, oligohydramnios, placental insufficiency and fetal growth retardation. With late preeclampsia, metabolic syndrome, anemia, and a history of arterial hypertension were more often observed. In the peripheral blood of all women with preeclampsia, there was an increase in the content of CD20+CD5+-cells in comparison with those in uncomplicated pregnancy, more pronounced in the late onset of preeclampsia. Only in women with early preeclampsia blood levels of CD19+CD20- CD38+ and CD19+CD27+IgD±-cells, IL-5, IL-9 and IL-13 increased. Studies of the placenta in early preeclampsia indicated impaired implantation and pathological placentation with the development of primary placental insufficiency, which becomes chronic. In late preeclampsia, the development of placental insufficiency was determined by chronic disorders of maternal and fetal hemocirculation with increased deposition of fibrin and fibrinoid in the basal lamina and in the zones of villous epithelium necrosis. The study showed that the timing of the manifestation of preeclampsia is determined by the action of factors of the clinical history, structural rearrangements in the placenta and immune responses of B-lymphocytes are closely interrelated.
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Fernandes, Eduardo Vignoto, Celio Estanislau, and Emerson José Venancio. "MODERATE INTENSITY PHYSICAL EXERCISE: PSYCHONEUROIMMUNOLOGICAL ASPECTS." Revista Brasileira de Medicina do Esporte 24, no. 5 (September 2018): 395–98. http://dx.doi.org/10.1590/1517-869220182405185533.

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ABSTRACT The literature presents several instances of interaction between the nervous system (NS) and the immune system (IS). These interactions are promoted by several molecules, such as cytokines and hormones, with modulating action for both the NS and IS. In this sense, the two systems may influence each other: changes in behavior may be accompanied by alterations in the IS (e.g., immunosuppression) and immunological disorders, such as infections, may modulate behavior (e.g., anxiety and depression). Considering that chronic stress, in addition to affecting behavior, also modulates the IS and that there is evidence that moderate intensity physical exercise (PE) protects physical and mental health, the objective of this review is to explore the influence of moderate-intensity PE on behavior and immunity. Level of Evidence V; Expert opinion.
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17

Joseph, Stephen, William Yule, and Ruth Williams. "Post-traumatic stress: Attributional aspects." Journal of Traumatic Stress 6, no. 4 (October 1993): 501–13. http://dx.doi.org/10.1002/jts.2490060407.

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18

Krapić, Mia, Inga Kavazović, and Felix M. Wensveen. "Immunological Mechanisms of Sickness Behavior in Viral Infection." Viruses 13, no. 11 (November 8, 2021): 2245. http://dx.doi.org/10.3390/v13112245.

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Sickness behavior is the common denominator for a plethora of changes in normal behavioral routines and systemic metabolism during an infection. Typical symptoms include temperature, muscle weakness, and loss of appetite. Whereas we experience these changes as a pathology, in fact they are a carefully orchestrated response mediated by the immune system. Its purpose is to optimize immune cell functionality against pathogens whilst minimizing viral replication in infected cells. Sickness behavior is controlled at several levels, most notably by the central nervous system, but also by other organs that mediate systemic homeostasis, such as the liver and adipose tissue. Nevertheless, the changes mediated by these organs are ultimately initiated by immune cells, usually through local or systemic secretion of cytokines. The nature of infection determines which cytokine profile is induced by immune cells and therefore which sickness behavior ensues. In context of infection, sickness behavior is typically beneficial. However, inappropriate activation of the immune system may induce adverse aspects of sickness behavior. For example, tissue stress caused by obesity may result in chronic activation of the immune system, leading to lasting changes in systemic metabolism. Concurrently, metabolic disease prevents induction of appropriate sickness behavior following viral infection, thus impairing the normal immune response. In this article, we will revisit recent literature that elucidates both the benefits and the negative aspects of sickness behavior in context of viral infection.
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19

Fernandes, Gilberto A. "Immunological stress in rats induces bodily alterations in saline-treated conspecifics." Physiology & Behavior 69, no. 3 (May 2000): 221–30. http://dx.doi.org/10.1016/s0031-9384(99)00226-7.

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20

Benschop, R. J., G. L. R. Godaert, R. Geenen, J. F. Brosschot, M. B. M. De Smet, M. Olff, C. J. Heijnen, and R. E. Ballieux. "Relationships between cardiovascular and immunological changes in an experimental stress model." Psychological Medicine 25, no. 2 (March 1995): 323–27. http://dx.doi.org/10.1017/s0033291700036229.

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SynopsisTo investigate the relationships between cardiovascular variables (SBP, DBP, and HR) and circulating natural killer (NK) cell numbers, 70 male volunteers were subjected to a rest condition (N = 30) or a stressful laboratory task (N = 40). At baseline, no significant relationships could be demonstrated between the number of NK cells and the cardiovascular variables. Analysis of covariance showed that the stressor induced increases in the number of NK cells, SBP, DBP, and HR. Changes in NK cell numbers were highly correlated to changes in cardiovascular variables in both the task and the no-task group. These results indicate that there is no relationship between the number of circulating NK cells and cardiovascular levels per se, but that changes in these variables, either stress-induced or under rest conditions, are regulated by a common mechanism.
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21

Vedhara, Kav, and Kenneth Nott. "The assessment of the emotional and immunological consequences of examination stress." Journal of Behavioral Medicine 19, no. 5 (October 1996): 467–78. http://dx.doi.org/10.1007/bf01857679.

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22

Naliboff, B. D., D. Benton, G. F. Solomon, J. E. Morley, J. L. Fahey, E. T. Bloom, T. Makinodan, and S. L. Gilmore. "Immunological changes in young and old adults during brief laboratory stress." Psychosomatic Medicine 53, no. 2 (March 1991): 121–32. http://dx.doi.org/10.1097/00006842-199103000-00002.

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23

Vadra, Priti, and S. Sultan Akhtar. "Measuring Social and Family Aspects of Role Stress." Psychological Reports 66, no. 2 (April 1990): 466. http://dx.doi.org/10.2466/pr0.1990.66.2.466.

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24

Spring, Dee. "Ethnocultural Aspects of Posttraumatic Stress Disorder." Art Therapy 16, no. 1 (January 1999): 43–45. http://dx.doi.org/10.1080/07421656.1999.10759351.

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25

Shulgina, S. M., M. P. Rykova, O. V. Kutko, V. A. Shmarov, E. N. Antropov, E. A. Zhirova, E. A. Lysenko, K. D. Orlova, D. D. Vlasova, and S. A. Ponomarev. "Immunological Aspects of Reactivation of Latent Infections in Space Flight and Antarctica." Физиология человека 49, no. 6 (November 1, 2023): 98–116. http://dx.doi.org/10.31857/s0131164623700418.

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The complex of space flight (SF) factors can have a negative impact on the human organism, including the innate and adaptive immunity. One of the negative effects of impaired functional activity of the immune system is a decrease of latent infections immunological control. During short- and long-term SF, reactivation of latent viruses was shown, while the virus was detected in body fluids for some time after returning to Earth. Despite the fact that in most cases reactivation was asymptomatic, there are fears that during long expeditions into deep space, reactivation of latent pathogens can lead to the development of diseases that are dangerous to the crewmembers health. There is a lot of data about the viral pathogens reactivation in SF, but the reactivation of bacterial agents in cosmonauts has received almost no attention. Due to the complexity of conducting research in space, an important role is played by ground-based analog experiments that simulate the space station conditions. One of the most significant analogues of SF is the long-term Antarctic expedition, which makes it possible to obtain unique information about the effect of isolation, increased mental and physiological stress on the latent pathogens reactivation. Currently, latency is increasingly considered as a symbiotic existence of a pathogen and host, during which the human body acquires additional resistance to certain infectious agents. Thus, an important task of the upcoming research is a comprehensive analysis of the personal immunological status in the SF, establishing its relationship with the latent infections reactivation and developing systems for monitoring, preventing and treating the negative consequences of reactivation.
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Śmierciak, Natalia. "Unraveling the Complex Nexus of Chronic Spontaneous Urticaria: Immunological, Infectious and Psychosomatic Dimensions." Psychology & Psychological Research International Journal 8, no. 4 (2023): 1–10. http://dx.doi.org/10.23880/pprij-16000374.

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Chronic Spontaneous Urticaria (CSU) is a complex dermatologic allergic condition characterized by unknown origins, frequent relapses, and resistance to therapy, contributing to a substantial psychosocial burden. Stressful experiences appear to coincide with CSU onset and exacerbation, suggesting a psychosomatic element within a psychoneuroimmunological framework. The condition significantly impairs patients’ quality of life, disrupting daily activities and exhibiting a high prevalence of psychiatric comorbidities. Recent theories propose autoantibodies activating mast cells as a potential key factor in CSU development. Psychological stress is considered both a trigger for CSU onset and a factor influencing the disease course and therapy effectiveness. Although the skin’s response mechanisms to psychological stress are not fully understood, inflammatory mediators, neuropeptides, and neurotransmitters are believed to play a role. Chronic Spontaneous Urticaria (CSU) is estimated to impact 0.5–1% of the global population at any given time, making up approximately two-thirds of all Chronic Urticaria (CU) cases. Among individuals with CSU/CIU, it is suggested that 33–67% experience both itchy welts and deeper skin swelling, while 29–65% solely have itchy welts, and 1–13% exclusively experience deeper skin swelling. Despite extensive research on the clinical aspects and origin of CSU/CIU in the last decade, the comprehensive understanding of the humanistic and economic implications of CSU/CIU remains unclear, especially for those with an inadequate response to initial treatment strategies. Research indicates that infections act as a facilitating factor for CSU initiation and perpetuation, with stress potentially necessary for the expression of the CSU phenotype. Detection and treatment of hidden infections have shown improvements in CSU patients, reinforcing the association between infections and CSU. A comprehensive, multidisciplinary approach that considers biological, psychological, and social aspects is emphasized for effective urticaria treatment. Current research underscores the significant psychological impact of CSU, particularly for individuals with more severe forms or those diagnosed with chronic autoimmune urticarial.
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Al-Shwilly, H. A. J. "Potency of garlic juice supplementation on some physiological and immunological aspects of broilers exposed to heat stress." Iraqi Journal of Veterinary Sciences 31, no. 2 (December 28, 2017): 0. http://dx.doi.org/10.33899/ijvs.2017.145606.

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Bertino, Lucrezia, Fabrizio Guarneri, Serafinella Patrizia Cannavò, Marco Casciaro, Giovanni Pioggia, and Sebastiano Gangemi. "Oxidative Stress and Atopic Dermatitis." Antioxidants 9, no. 3 (February 26, 2020): 196. http://dx.doi.org/10.3390/antiox9030196.

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Atopic dermatitis is a common chronic/chronically relapsing inflammatory skin disease, with increasing worldwide prevalence. Etiopathogenesis is complex and multifactorial, with a mix of genetic, immunological and environmental aspects. Like in other chronic inflammatory diseases, oxidative stress plays an important pathogenetic role. We reviewed in vivo research studies on humans about oxidative stress and atopic dermatitis. Although sometimes contrasting, overall, they suggest that oxidative stress may have a significant role in atopic dermatitis, but our understanding is still incomplete, at least concerning in vivo data, because of limitations of available literature. Research consists of 33 papers published in 28 years, was not always performed on large study populations, represents a limited number of countries and ethnicities—not always in proportion to their size—and is scattered over multiple papers that, in the majority of cases, cannot be pooled and/or compared because many biomarkers were studied, in different tissues and with different methods. Further, larger studies appear warranted and necessary to shed more light on this aspect of atopic dermatitis, which is important not only to improve our understanding of this disease, but also for potential clinical and therapeutic implications.
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Bar-tal, Yoram, Jiska Cohen-mansfield, and Hava Golander. "Which Stress Matters? The Examination of Temporal Aspects of Stress." Journal of Psychology 132, no. 5 (September 1998): 569–76. http://dx.doi.org/10.1080/00223989809599290.

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Nazarovets, Tetiana, and Alona Nastoiashcha. "Neuropsychological aspects of stress manifestation." Personality and environmental issues 3, no. 1 (March 26, 2024): 38–43. http://dx.doi.org/10.31652/2786-6033-2024-3(1)-38-43.

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Modern life, unfortunately, is not devoid of stressful situations. This article explores how stress affects various cognitive functions, making us more vulnerable. Memory, attention, decision-making, inhibition, and abstract thinking are just some aspects of the effects of stress. Studies show that prolonged or repeated stress can weaken the immune system, increasing inflammation and even destroying neurons in the hippocampus responsible for memory. Maturation of the frontal cortex, which is accompanied by an increase in gray matter volume and the number of synapses, can be disrupted by stress, leading to cognitive decline. Recent research on the impact of stressful situations on psychological and physical health is characterized by a wide range of methodological approaches and a variety of sources. Sapolsky R. highlights the impact of stress on memory-related processes, as well as on the formation of certain parts of the brain. In the work of D. Nakazawa, we get acquainted with neural development and the natural processes associated with this development. Thus, the latest research and publications in the field of stress psychology are an important contribution to our understanding of this complex problem and the development of practical recommendations for overcoming the negative effects of stress. Sleep is crucial for restoring energy and consolidating memory, but it also becomes a victim of stress. Lack of sleep and deterioration in its quality make us more vulnerable to the negative effects of stress. Children who have experienced stress risk losing even more neurons than their peers, as the natural neuronal pruning at puberty can be intensified. Chronic stress can lead to neurotic reactions, psychophysiological symptoms, and psychosomatic illnesses. This emphasizes the importance of nervous system rehabilitation and neuropsychological interventions.
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Saadati, Seyed Milad, and Fatemeh Rezazadeh. "Stress, Resilience, and the Immune System: A Health Psychology Analysis." KMAN Counseling and Psychology Nexus 1, no. 1 (2023): 109–17. http://dx.doi.org/10.61838/kman.psychnexus.1.1.14.

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To investigate the relationship between stress resilience and immune system functionality, emphasizing the psychological mechanisms that contribute to immune regulation and the potential for resilience-building interventions to enhance immune responses. This comprehensive review synthesizes existing research from psychological, immunological, and epidemiological studies. It examines the impact of acute and chronic stress on immune function, explores the role of psychological resilience as a mediator, and evaluates the effectiveness of various stress management and resilience-building strategies. Evidence indicates a significant link between psychological resilience and stronger immune function. Individuals with higher resilience levels exhibit better immune responses, likely due to the effective management of stress and its physiological consequences. Additionally, interventions aimed at increasing resilience, such as mindfulness practices, cognitive-behavioral therapy, and lifestyle changes, have shown promise in bolstering immune health. Strengthening psychological resilience holds substantial potential for improving immune system outcomes, suggesting a need for holistic health approaches that incorporate mental, physical, and social well-being components. Future research should focus on identifying specific mechanisms through which resilience affects immune function and developing targeted interventions to enhance both psychological well-being and immune health.
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Huang, Chun-Jung, Heather E. Webb, Ryan S. Garten, Gary H. Kamimori, Ronald K. Evans, and Edmund O. Acevedo. "Stress hormones and immunological responses to a dual challenge in professional firefighters." International Journal of Psychophysiology 75, no. 3 (March 2010): 312–18. http://dx.doi.org/10.1016/j.ijpsycho.2009.12.013.

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Endresen, I. M., B. Ellertsen, C. Endresen, A. M. Hjelmen, R. Matre And, and H. Ursin. "Stress at work and psychological and immunological parameters in a group of Norwegian female bank employees." Work & Stress 5, no. 3 (July 1991): 217–27. http://dx.doi.org/10.1080/02678379108257020.

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Anurogo, Dito. "The Art of Psychoneuroimmunology in Menopause Management." Journal of Biomedical Research & Environmental Sciences 4, no. 6 (June 2023): 972–92. http://dx.doi.org/10.37871/jbres1758.

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Psychoneuroimmunology is a field that explores the intricate connections between the mind, brain, and immune system. This narrative review integrates various aspects of psychoneuroimmunology in the context of menopause. It highlights the hormonal changes during menopause, their impact on psychological well-being and the immune system, as well as the psychological aspects of menopause such as emotional changes, cognitive functioning, and sleep disturbances. The immunological changes during menopause, including alterations in immune cell function and cytokine production, are discussed along with their influence on psychological well-being. This comprehensive review emphasizes the bidirectional communication between the brain and immune system, the effects of hormonal, psychological, and immunological factors on each other, and the role of chronic inflammation in menopause-related psychological symptoms. Additionally, it explores the influence of psychosocial factors like stress and social support on menopausal symptoms, and presents various psychoneuroimmunology interventions such as mind-body techniques, pharmacological options, and lifestyle modifications. The importance of integrating psychoneuroimmunology approaches in menopause research, potential areas for further investigation, and the call for comprehensive strategies to improve menopausal care are also highlighted, underscoring the significance of this interdisciplinary field in understanding and enhancing menopausal health and well-being.
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Bohus, Béla, Jaap M. Koolhaas, Coby J. Heijneri, and Onno de Boer. "Immunological Responses to Social Stress: Dependence on Social Environment and Coping Abilities." Neuropsychobiology 28, no. 1-2 (1993): 95–99. http://dx.doi.org/10.1159/000119008.

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Amalina, Dinda Rizky, Fadila Annisa, Nurhabibah, Patricia Viona Evelin, and Silfi Erisa Hestari. "DESCRIPTIVE STUDY OF ACADEMIC STRESS IN UNP PSYCHOLOGY STUDENTS." In Trend : International Journal of Trends in Global Psychological Science and Education 1, no. 3 (July 5, 2024): 109–13. http://dx.doi.org/10.62260/intrend.v1i3.176.

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Academic stress is a type of stress that originates from the demands of academic activities or the learning process. This stress is perceived by students as a burden that exceeds the limits of ability, which is characterized by various reactions that affect physical, emotional and behavioral aspects. Academic stress affects psychological states, including emotional, cognitive, and behavioral factors that can contribute to illness. This research aims to determine the level of academic stress in psychology students at Padang State University. The research design used was descriptive analytical. The sampling technique used in this research was simple random sampling. The method in this research is to use descriptive qualitative research methods. The population of this research is Padang State University students, while the sample of this research is Padang State University Psychology students. The instrument in this research uses an academic stress questionnaire which is based on 4 aspects according to Robotham (2008), consisting of cognitive, affective, physiological and behavioral aspects. Based on research that has been conducted with the population of this study, namely Padang State University students, while the sample from this research is Padang State University Psychology students. Of the 48 respondents who participated, it was found that 6 respondents were at a low stress level with a percentage of 12.5%, and 37 respondents were at a moderate stress level with a percentage of 77.08%, then 5 respondents were at a high stress level. with a percentage of 10.4%. Based on research conducted on 48 respondents who were psychology students at Padang State University, it was found that the majority of academic stress was at a moderate stress level.
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Hänggi, Yves. "Stress and Emotion Recognition: An Internet Experiment Using Stress Induction." Swiss Journal of Psychology 63, no. 2 (June 2004): 113–25. http://dx.doi.org/10.1024/1421-0185.63.2.113.

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Past findings have lead us to assume that the decoding of emotions in facial expression is impaired in stressful situations. The effect of stress on decoding ability is examined through use of an Internet based experiment in which an episode of stress is induced in one group of participants. A fictitious “EQ online evaluation study” serves as a framework. The main task consisted in recognizing 24 emotions from facial expressions in the Ekman-Friesen-series ( Ekman & Friesen, 1976 ), which were presented for approximately 200 ms. Data collected from 86 participants support the underlying hypotheses that stress negatively affects decoding performance. Results are discussed in terms of considerations that must be taken into account due to the method of collecting data in an Internet based study. Ethical aspects of Internet experiments are also discussed.
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Park, Hyun Jung, Sung Ja Rhie, and Insop Shim. "Regulatory role of cytokines on etiology of depression in animal models: their biological mechanisms and clinical implication with physical exercise." Journal of Exercise Rehabilitation 18, no. 6 (December 27, 2022): 344–49. http://dx.doi.org/10.12965/jer.2244506.253.

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It has been known that chronic psychological or physical stress elicits depressive behaviors (learned helplessness, anhedonia, anxiety, etc.) and also activates to release proinflammatory cytokines in the brain. Especially, postmenopausal women under stress condition exacerbates neuroimmune systems and mood disorder. Repeated restraint stress in the ovariectomized female rats poses an immune challenge which was capable of inducing depressive-like behaviors, promoting exaggerated corticosterone responses and changing the proinflammatory cytokine expression such as interleukin (IL)-1β in the brain. Also, anti-inflammatory cytokines including IL-4 are known to regulate inflammation caused by immune response or stress challenge. Furthermore, some studies reported that physical activity can reduce stress hormones and improve personal immunity. Physical exercise has been shown to be associated with decreased symptoms of depression and anxiety, and with improved physical health, immunological function, and psychological well-being. This paper aims to discuss an overview of how stress shapes neuroimmune response and diverse roles of cytokines in animals models, acting on depressive-like behavioral changes; some beneficial aspects of exercise on stress-related disorders are addressed.
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Dollard, M. F., C. Dormann, C. M. Boyd, H. R. Winefield, and A. H. Winefield. "Unique Aspects of Stress in Human Service Work." Australian Psychologist 38, no. 2 (July 2003): 84–91. http://dx.doi.org/10.1080/00050060310001707087.

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40

Zahra Ali, Syeda, Tahira Mubashar, Soulat Khan, and Tanvir Akhtar. "Psychosocial Aspects of Adaptation of International Students in Pakistan." Pakistan Journal of Psychological Research 36, no. 1 (March 31, 2021): 135–56. http://dx.doi.org/10.33824/pjpr.2021.36.1.09.

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This study examined the psychosocial aspects (acculturation stress, ethnic discrimination, nostalgia, and sentimentality) of socio cultural and psychological adaptation of international students in Pakistan. Sample of 143 international students was taken from different government and private universities of Pakistan. Measures of Acculturative Stress Scale for International Students (Sandhu & Asarbadi, 1994), Brief Perceived Ethnic Discrimination Questionnaire-Community Version (Brondolo et al., 2005), Index of Nostalgia Proneness (Holak, Havlena, & Matveev, 2006), Emotionality Subscale of the HEXACO Personality Inventory (Lee & Ashton, 2009), Brief Psychological Adaptation Scale (Demes & Geeraert, 2014), and Brief Socio-Cultural Adaptation Scale (Demes & Geeraert, 2014) were used. Results showed that ethnic discrimination, acculturation stress, nostalgia, and sentimentality were negatively correlated with psychological and socio-cultural adaptation. Sentimentality moderated the relationship of ethnic discrimination with socio-cultural and psychological adaptation. Moreover, sentimentality also moderated the relationship between nostalgia and psychological adaptation.
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41

Schlebusch, Lourens, and Michael J. Cassidy. "Stress, Social Support and Biopsychosocial Dynamics in HIV-AIDS." South African Journal of Psychology 25, no. 1 (March 1995): 27–30. http://dx.doi.org/10.1177/008124639502500104.

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Research trends in psychosocial aspects of HIV-AIDS are reviewed, exploring the role of psychosocial cofactors in disease progression. This is undertaken within a biopsychosocial model and gives cognisance to the role of psychosocial stress, social support, and emotional adjustment. Research data from a study of biopsychosocial interrelationships in a sample of HIV-positive patients show a significant correlation between social support and emotional adjustment and that social support exerts a mediatory, stress-buffering effect in these patients. Some observations are made on aspects of the social conditions of South Africans with HIV-AIDS.
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Suzuki, Katsuhiko. "Chronic Inflammation as an Immunological Abnormality and Effectiveness of Exercise." Biomolecules 9, no. 6 (June 7, 2019): 223. http://dx.doi.org/10.3390/biom9060223.

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Reduced levels of physical activity in people’s daily lives cause the development of metabolic syndromes or age-related disorders. Chronic inflammation is now understood to be an underlying pathological condition in which inflammatory cells such as neutrophils and monocyte/macrophages infiltrate into fat and other tissues and accumulate when people become obese due to overeating and/or physical inactivity. Pro-inflammatory mediators such as cytokines that are secreted in excess from inflammatory cells will not only lead to the development of arteriosclerosis when they chronically affect blood vessels but also bring tissue degeneration and/or dysfunction to various organs. Chronic inflammation is also involved in sarcopenia that brings hypofunction in the elderly, dementia, osteoporosis, or cancer and negatively affects many chronic diseases and people’s healthy life expectancy. In this paper, outlines of such studies are introduced in terms of homeostatic inflammation, which occurs chronically due to the innate immune system and its abnormalities, while focusing on the efficacy of exercise from aspects of immunology and oxidative stress. The preventative effects of functional food ingredients in combination with exercise are also introduced and described. The challenges and future directions in understanding the role of exercise in the control of chronic inflammation are discussed.
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43

Kedem, Peri, Benjamin Bartoov, Mario Mikulincer, and Tamar Shkolnik. "Psychoneuroimmunology and male infertility: A possible link between stress, coping and male immunological infertility." Psychology & Health 6, no. 3 (July 1992): 159–73. http://dx.doi.org/10.1080/08870449208403181.

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44

Beerda, Bonne, Matthijs B. H. Schilder, Wilbert Bernadina, JAN A. R. A. M. Van Hooff, Hans W. De Vries, and Jan A. Mol. "Chronic Stress in Dogs Subjected to Social and Spatial Restriction. II. Hormonal and Immunological Responses." Physiology & Behavior 66, no. 2 (April 1999): 243–54. http://dx.doi.org/10.1016/s0031-9384(98)00290-x.

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45

Djordjevic, Brankica, Byron Morales-Lange, Charles McLean Press, Jake Olson, Leidy Lagos, Luis Mercado, and Margareth Øverland. "Comparison of Circulating Markers and Mucosal Immune Parameters from Skin and Distal Intestine of Atlantic Salmon in Two Models of Acute Stress." International Journal of Molecular Sciences 22, no. 3 (January 21, 2021): 1028. http://dx.doi.org/10.3390/ijms22031028.

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Ensuring salmon health and welfare is crucial to maximize production in recirculation aquaculture systems. Healthy and robust mucosal surfaces of the skin and intestine are essential to achieve this goal because they are the first immunological defenses and are constantly exposed to multistressor conditions, such as infectious diseases, suboptimal nutrition, and environmental and handling stress. In this work, Atlantic salmon, split from a single cohort, were subjected to acute hypoxia stress or 15-min crowding stress and observed over a 24-h recovery period. Samples were collected from fish at 0, 1, 3, 6, 12 and 24 h post-stress to analyze plasma-circulating markers of endocrine function (cortisol), oxidative stress (glutathione peroxidase) and immune function (interleukin 10 (IL-10), annexin A1). In addition, mucosal barrier function parameters were measured in the skin mucus (Muc-like protein and lysozyme) and distal intestine (simple folds, goblet cell size and goblet cell area). The results showed that both acute stress models induced increases of circulating cortisol in plasma (1 h post-stress), which then returned to baseline values (initial control) at 24 h post-stress. Moreover, the hypoxia stress was mostly related to increased oxidative stress and IL-10 production, whereas the crowding stress was associated with a higher production of Muc-like protein and lysozyme in the skin mucus. Interestingly, in the distal intestine, smaller goblet cells were detected immediately and one hour after post-hypoxia stress, which could be related to rapid release of the cellular content to protect this organ. Finally, the correlation of different markers in the hypoxic stress model showed that the circulating levels of cortisol and IL-10 were directly proportional, while the availability of Muc-like proteins was inversely proportional to the size of the goblet cells. On the other hand, in the crowding stress model, a proportional relationship was established between plasma cortisol levels and skin mucus lysozyme. Our results suggest key differences in energy partitioning between the two acute stress models and support the need for further investigation into the interplay of multistressor conditions and strategies to modulate immunological aspects of mucosal surfaces.
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Hernández-Avalos, I., E. Flores-Gasca, D. Mota-Rojas, A. Casas-Alvarado, A. E. Miranda-Cortés, and A. Domínguez-Oliva. "Neurobiology of anesthetic-surgical stress and induced behavioral changes in dogs and cats: A review." Veterinary World 14, no. 2 (February 11, 2021): 393–404. http://dx.doi.org/10.14202/vetworld.2021.393-404.

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The anesthetic-surgical stress response consists of metabolic, neuroendocrine, hemodynamic, immunological, and behavioral adaptations through chemical mediators such as the adrenocorticotropic hormone, growth hormone, antidiuretic hormone, cortisol, aldosterone, angiotensin II, thyroid-stimulating hormone, thyroxine, triiodothyronine, follicle-stimulating hormone, luteinizing hormone, catecholamines, insulin, interleukin (IL)-1, IL-6, tumor necrosis factor-alpha, and prostaglandin E-2. Behavioral changes include adopting the so-called prayer posture, altered facial expressions, hyporexia or anorexia, drowsiness, sleep disorders, restriction of movement, licking or biting the injured area, and vocalizations. Overall, these changes are essential mechanisms to counteract harmful stimuli. However, if uncontrolled surgical stress persists, recovery time may be prolonged, along with increased susceptibility to infections in the post-operative period. This review discusses the neurobiology and most relevant organic responses to pain and anesthetic-surgical stress in dogs and cats. It highlights the role of stress biomarkers and their influence on autonomous and demeanor aspects and emphasizes the importance of understanding and correlating all factors to provide a more accurate assessment of pain and animal welfare in dogs and cats throughout the surgical process.
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Lees, Mary C., and Richard W. J. Neufeld. "Decision-theoretic aspects of stress arousal and coping propensity." Journal of Personality and Social Psychology 77, no. 1 (1999): 185–208. http://dx.doi.org/10.1037/0022-3514.77.1.185.

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Hochberg, Judith G. "First steps in the acquisition of Spanish stress." Journal of Child Language 15, no. 2 (June 1988): 273–92. http://dx.doi.org/10.1017/s030500090001237x.

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ABSTRACTThis article uses longitudinal data from four Mexican-American children to explore two aspects of the acquisition of Spanish word stress that precede and accompany learning of the stress system itself. First, contrary to Allen & Hawkins' proposed universal ‘trochaic bias’ (Allen 1982, Allen & Hawkins 1977, 1979, 1980), it is shown that children have a ‘neutral start’ in stress learning: they approach the task of stress learning unbiased towards any particular stress type. Secondly, several examples are found in which children's attention to phonetic or semantic aspects of normatively unstressed syllables leads them to shift stress to that syllable.
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Chavajay, Pablo, and Jeffrey Skowronek. "Aspects of Acculturation Stress among International Students Attending a University in the USA." Psychological Reports 103, no. 3 (December 2008): 827–35. http://dx.doi.org/10.2466/pr0.103.3.827-835.

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Acculturation stress reported by 130 international students attending a university in Utah for about 2 yr. was examined. On the Acculturative Stress Scale for International Students, few students reported experiencing acculturation stress, but responses to four open-ended questions indicated many students perceived experience of acculturation stresses related to discrimination, feelings of loneliness, and academic concerns. The contrast of findings for the scale scores and the open-ended questions indicate the complexity of assessing international students' acculturation experiences of living and studying in the USA and suggest the usefulness of complementary methodologies for assessing such experience.
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Robertson, Michael, Paul J. Rushton, Dee Bartrum, and Rebecca Ray. "Group-Based Interpersonal Psychotherapy for Posttraumatic Stress Disorder: Theoretical and Clinical Aspects." International Journal of Group Psychotherapy 54, no. 2 (April 2004): 145–75. http://dx.doi.org/10.1521/ijgp.54.2.145.40384.

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