Journal articles on the topic 'Stress (Physiology)'

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1

Modaresi, Mehrdad, and Mansoureh Emadi. "The Effects of Rosemary Extract on Spermatogenesis and Sexual Hormones of Mice under Heat Stress." Trends Journal of Sciences Research 3, no. 2 (September 7, 2018): 69–74. http://dx.doi.org/10.31586/physiology.0302.02.

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2

Dawson, Todd. "Physiology and Plant Stress." Ecology 70, no. 3 (June 1989): 793. http://dx.doi.org/10.2307/1940233.

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3

Levine, Samara, and Ozgul Muneyyirci-Delale. "Stress-Induced Hyperprolactinemia: Pathophysiology and Clinical Approach." Obstetrics and Gynecology International 2018 (December 3, 2018): 1–6. http://dx.doi.org/10.1155/2018/9253083.

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While prolactin is most well known for its role in lactation and suppression of reproduction, its physiological functions are quite diverse. There are many etiologies of hyperprolactinemia, including physiologic as well as pathologic causes. Physiologic causes include pregnancy, lactation, sleep-associated, nipple stimulation and sexual orgasm, chest wall stimulation, or trauma. Stress is also an important physiologic cause of hyperprolactinemia, and its clinical significance is still being explored. This review will provide an overview of prolactin physiology, the role of stress in prolactin secretion, as well as the general clinical approach to hyperprolactinemia.
4

Zhou, Qi, Shuang Song, Xin Wang, Chao Yan, Chunmei Ma, and Shoukun Dong. "Effects of drought stress on flowering soybean physiology under different soil conditions." Plant, Soil and Environment 68, No. 10 (October 17, 2022): 487–98. http://dx.doi.org/10.17221/237/2022-pse.

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Soybean is highly sensitive to drought stress during its flowering period. Heinong84 (HN84) and Hefeng46 (HF46) were planted in clay loam, silty loam, and sandy clay. We studied the effects of drought stress on the content of membrane lipid peroxides in flowering soybean leaves, the activity of antioxidant enzymes, and the activity of key enzymes of nitrogen metabolism under different soil conditions. Our results showed that soybean had clear physiological responses to drought stress. With increasing drought stress, the malondialdehyde, glutathione reductase, and glutathione peroxidase levels in soybean leaves increased continuously. Superoxide dismutase, peroxidase, glutamine synthase, and glutamate synthase levels increased with drought stress, reaching a maximum under moderate drought stress and then decreased; nitrate reductase activity decreased continuously. Under the condition of sufficient water, the performance of soybean in the three soils is almost the same, but there are differences under drought stress; particularly, soybean grown in clay loam shows the strongest drought resistance. In summary, the physiological state of soybean is easily affected by drought stress, which varies greatly among different cultivars and in different soil types.
5

West, D. W. "STRESS PHYSIOLOGY IN TREES - SALINITY." Acta Horticulturae, no. 175 (March 1986): 321–32. http://dx.doi.org/10.17660/actahortic.1986.175.48.

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6

De Castro, James, Robert D. Hill, Claudio Stasolla, and Ana Badea. "Waterlogging Stress Physiology in Barley." Agronomy 12, no. 4 (March 24, 2022): 780. http://dx.doi.org/10.3390/agronomy12040780.

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Barley (Hordeum vulgare L.) is the most susceptible cereal species to excess moisture stress. Waterlogging-induced hypoxia causes major morphological, physiological, and metabolic changes, some of which are regulated by the action of plant growth regulators and signal molecules including nitric oxide. Recent studies have evidenced the participation of phytoglobins in attenuating hypoxic stress during conditions of excessive moisture through their ability to scavenge nitric oxide and influence the synthesis and response of growth regulators. This review will highlight major cellular changes linked to plant responses to waterlogging stress with emphasis on phytoglobins.
7

Lefcourt, Herbert M. "Understanding the Physiology of Stress." Contemporary Psychology: A Journal of Reviews 40, no. 1 (January 1995): 24–25. http://dx.doi.org/10.1037/003323.

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8

Dakora, F. D., and J. Van Staden. "Foreword Special Issue Stress Physiology." South African Journal of Botany 70, no. 5 (December 2004): v. http://dx.doi.org/10.1016/s0254-6299(15)30186-1.

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9

Chin, G. J. "PHYSIOLOGY: More Stress, Less Inflammation." Science 288, no. 5468 (May 12, 2000): 931c—931. http://dx.doi.org/10.1126/science.288.5468.931c.

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10

Ranawat, Preeti, and Seema Rawat. "Stress response physiology of thermophiles." Archives of Microbiology 199, no. 3 (January 17, 2017): 391–414. http://dx.doi.org/10.1007/s00203-016-1331-4.

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11

Wu, Yanyou. "Plant Physiology under Abiotic Stresses: Deepening the Connotation and Expanding the Denotation." Horticulturae 9, no. 2 (February 7, 2023): 218. http://dx.doi.org/10.3390/horticulturae9020218.

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Abiotic stress factors influence many aspects of plant physiology. The works collected in the Special Issue deepen plant physiology’s connotation (such as plant electrophysiology) under abiotic stress and expand the denotation (such as environmental pollutants as abiotic stress factors). At the same time, the achievements of the selected papers published in the Special Issue also exhibit their potential application value in the production of horticultural plants.
12

Collier, R. J., B. J. Renquist, and Y. Xiao. "A 100-Year Review: Stress physiology including heat stress." Journal of Dairy Science 100, no. 12 (December 2017): 10367–80. http://dx.doi.org/10.3168/jds.2017-13676.

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13

Sharma, Dushyant Kumar. "Physiology of Stress and its Management." Journal of Medicine: Study & Research 1, no. 1 (August 31, 2018): 1–5. http://dx.doi.org/10.24966/msr-5657/100001.

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14

Zimmer, Cedric, H. Arthur Woods, and Lynn B. Martin. "Information theory in vertebrate stress physiology." Trends in Endocrinology & Metabolism 33, no. 1 (January 2022): 8–17. http://dx.doi.org/10.1016/j.tem.2021.10.001.

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15

Syvertsen, J. P. "Aspects of stress physiology of citrus." Acta Horticulturae, no. 1177 (November 2017): 51–58. http://dx.doi.org/10.17660/actahortic.2017.1177.5.

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16

Etherington, J. R., M. G. Hale, and D. M. Orcutt. "The Physiology of Plants Under Stress." Journal of Ecology 76, no. 4 (December 1988): 1247. http://dx.doi.org/10.2307/2260647.

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17

Lay, Ryan. "Perioperative physiology and the stress response." Journal of Operating Department Practitioners 2, no. 5 (July 2, 2014): 235–40. http://dx.doi.org/10.12968/jodp.2014.2.5.235.

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18

Elenkov, Ilia J., and George P. Chrousos. "Stress System – Organization, Physiology and Immunoregulation." Neuroimmunomodulation 13, no. 5-6 (2006): 257–67. http://dx.doi.org/10.1159/000104853.

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19

Bornkamm, R. "The physiology of plants under stress." Agriculture, Ecosystems & Environment 26, no. 2 (July 1989): 157. http://dx.doi.org/10.1016/0167-8809(89)90030-3.

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20

Papadimitriou, Konstantinos, Ángel Alegría, Peter A. Bron, Maria de Angelis, Marco Gobbetti, Michiel Kleerebezem, José A. Lemos, et al. "Stress Physiology of Lactic Acid Bacteria." Microbiology and Molecular Biology Reviews 80, no. 3 (July 27, 2016): 837–90. http://dx.doi.org/10.1128/mmbr.00076-15.

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SUMMARYLactic acid bacteria (LAB) are important starter, commensal, or pathogenic microorganisms. The stress physiology of LAB has been studied in depth for over 2 decades, fueled mostly by the technological implications of LAB robustness in the food industry. Survival of probiotic LAB in the host and the potential relatedness of LAB virulence to their stress resilience have intensified interest in the field. Thus, a wealth of information concerning stress responses exists today for strains as diverse as starter (e.g.,Lactococcus lactis), probiotic (e.g., severalLactobacillusspp.), and pathogenic (e.g.,EnterococcusandStreptococcusspp.) LAB. Here we present the state of the art for LAB stress behavior. We describe the multitude of stresses that LAB are confronted with, and we present the experimental context used to study the stress responses of LAB, focusing on adaptation, habituation, and cross-protection as well as on self-induced multistress resistance in stationary phase, biofilms, and dormancy. We also consider stress responses at the population and single-cell levels. Subsequently, we concentrate on the stress defense mechanisms that have been reported to date, grouping them according to their direct participation in preserving cell energy, defending macromolecules, and protecting the cell envelope. Stress-induced responses of probiotic LAB and commensal/pathogenic LAB are highlighted separately due to the complexity of the peculiar multistress conditions to which these bacteria are subjected in their hosts. Induction of prophages under environmental stresses is then discussed. Finally, we present systems-based strategies to characterize the “stressome” of LAB and to engineer new food-related and probiotic LAB with improved stress tolerance.
21

Schäffer, L., and E. Beinder. "Intrauterine determination of neonatal stress physiology." Journal of Reproductive Immunology 86, no. 2 (November 2010): 102. http://dx.doi.org/10.1016/j.jri.2010.08.044.

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22

Cincotta, Richard P., Maynard G. Hale, David M. Orcutt, and Laura K. Thompson. "The Physiology of Plants under Stress." Journal of Range Management 43, no. 1 (January 1990): 86. http://dx.doi.org/10.2307/3899132.

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23

Ayres, Peter. "The physiology of plants under stress." Physiological and Molecular Plant Pathology 36, no. 4 (April 1990): 361–62. http://dx.doi.org/10.1016/0885-5765(90)90065-6.

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24

Pickering, A. "Book Review: Stress Physiology in Animals." Journal of Fish Biology 56, no. 2 (February 2000): 454–55. http://dx.doi.org/10.1006/jfbi.1999.1190.

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25

Alkadhi, Karim. "Brain Physiology and Pathophysiology in Mental Stress." ISRN Physiology 2013 (June 9, 2013): 1–23. http://dx.doi.org/10.1155/2013/806104.

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Exposure to various forms of stress is a common daily occurrence in the lives of most individuals, with both positive and negative effects on brain function. The impact of stress is strongly influenced by the type and duration of the stressor. In its acute form, stress may be a necessary adaptive mechanism for survival and with only transient changes within the brain. However, severe and/or prolonged stress causes overactivation and dysregulation of the hypothalamic pituitary adrenal (HPA) axis thus inflicting detrimental changes in the brain structure and function. Therefore, chronic stress is often considered a negative modulator of the cognitive functions including the learning and memory processes. Exposure to long-lasting stress diminishes health and increases vulnerability to mental disorders. In addition, stress exacerbates functional changes associated with various brain disorders including Alzheimer’s disease and Parkinson’s disease. The primary purpose of this paper is to provide an overview for neuroscientists who are seeking a concise account of the effects of stress on learning and memory and associated signal transduction mechanisms. This review discusses chronic mental stress and its detrimental effects on various aspects of brain functions including learning and memory, synaptic plasticity, and cognition-related signaling enabled via key signal transduction molecules.
26

Robinson, Alexandra M. "Let's Talk about Stress: History of Stress Research." Review of General Psychology 22, no. 3 (September 2018): 334–42. http://dx.doi.org/10.1037/gpr0000137.

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The reference to stress is ubiquitous in modern society, yet it is a relatively new field of research. The following article provides an overview of the history of stress research and its iterations over the last century. In this article, I provide an overview of the earliest stress research and theories introduced through physiology and medicine and eventually as a concept in psychology. I begin with an exploration of the research of biological stressors 1st explored by experimental physiologist Claude Bernard and eventually adopted as a foundational concept in stress research when Walter Cannon expanded on Bernard's work and identified homeostasis. The contributions of Hans Selye, considered the father of stress research; Sir William Osler; Yerkes and Dodson; and Richard Lazarus are also discussed. Finally, I discuss how, in the new millennium, research on psychological stress has expanded across disciplines ranging from physiology to medicine, chemistry, endocrinology, neurosciences, epidemiology, psychiatry, epigenetics, and psychology, reflecting the complexity of the construct both theoretically and biologically.
27

Musiała, Nikola, Iga Hołyńska-Iwan, and Dorota Olszewska-Słonina. "Cortisol – inspection in the physiology and stress." Diagnostyka Laboratoryjna 54, no. 1 (April 13, 2018): 29–36. http://dx.doi.org/10.5604/01.3001.0013.7553.

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Cortisol, also called “the” stress hormone is a glucocorticoid secreted by the adrenal cortex. This hormone plays a significant role in maintaining homeostasis, according to the body’s total stress. Cortisol interferes with many organs, affects glucose and fatty acids metabolism and neurotransmitter secretion. Predominantly, cortisol influences the carbohydrate metabolism, stimulating gluconeogenesis in the liver and inhibiting glucose utilization in peripheral tissues. As it is an element “fight or flight” it also stimulates central nervous system and enhances blood flow. To some extent cortisol influences also the renal handling of electrolytes, namely: increasing sodium resorption, and renal excretion of potassium, calcium and phosphates. Through its anti-inflammatory and immunosuppressive character this glucocorticoid modulates the immune system functioning. Cortisol has a circadian rhythm following ACTH (adrenocorticotropic hormone) secretion. Increased cortisol levels are observed physiologically during stress and pathologically in Cushing’s syndrome. Chronic hypercortisolism is harmful or the body, and its effects present an extremely wide spectrum, including insulin resistance, obesity, insomnia and even depression. Thus, laboratory diagnosis of cortisol level is important for the diagnosis, monitoring and evaluate the effectiveness of hypercortisolism treatment.
28

Osmond, C. B., M. P. Austin, J. A. Berry, W. D. Billings, J. S. Boyer, J. W. H. Dacey, P. S. Nobel, S. D. Smith, and W. E. Winner. "Stress Physiology and the Distribution of Plants." BioScience 37, no. 1 (January 1987): 38–48. http://dx.doi.org/10.2307/1310176.

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29

Hasanuzzaman, Mirza, and Masayuki Fujita. "Plant Oxidative Stress: Biology, Physiology and Mitigation." Plants 11, no. 9 (April 28, 2022): 1185. http://dx.doi.org/10.3390/plants11091185.

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30

Kim, Leen. "Stress, Sleep Physiology, and Related Insomnia Disorders." Journal of the Korean Medical Association 53, no. 8 (2010): 707. http://dx.doi.org/10.5124/jkma.2010.53.8.707.

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31

Collier, Robert J., Lance H. Baumgard, Rosemarie B. Zimbelman, and Yao Xiao. "Heat stress: physiology of acclimation and adaptation." Animal Frontiers 9, no. 1 (October 29, 2018): 12–19. http://dx.doi.org/10.1093/af/vfy031.

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32

Bray, Elizabeth A. "Physiology of plants under stress: Abiotic factors." Field Crops Research 55, no. 1-2 (January 1998): 192–93. http://dx.doi.org/10.1016/s0378-4290(97)00069-5.

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33

REED, R. "Microbial water stress physiology: Principles and perspectives." Trends in Biotechnology 8 (1990): 365. http://dx.doi.org/10.1016/0167-7799(90)90229-q.

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34

Munns, R. "Comparative physiology of salt and water stress." Plant, Cell & Environment 25, no. 2 (February 2002): 239–50. http://dx.doi.org/10.1046/j.0016-8025.2001.00808.x.

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35

Dickens, Molly J., David J. Delehanty, and L. Michael Romero. "Stress and translocation: alterations in the stress physiology of translocated birds." Proceedings of the Royal Society B: Biological Sciences 276, no. 1664 (March 4, 2009): 2051–56. http://dx.doi.org/10.1098/rspb.2008.1778.

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Translocation and reintroduction have become major conservation actions in attempts to create self-sustaining wild populations of threatened species. However, avian translocations have a high failure rate and causes for failure are poorly understood. While ‘stress’ is often cited as an important factor in translocation failure, empirical evidence of physiological stress is lacking. Here we show that experimental translocation leads to changes in the physiological stress response in chukar partridge, Alectoris chukar . We found that capture alone significantly decreased the acute glucocorticoid (corticosterone, CORT) response, but adding exposure to captivity and transport further altered the stress response axis (the hypothalamic–pituitary–adrenal axis) as evident from a decreased sensitivity of the negative feedback system. Animals that were exposed to the entire translocation procedure, in addition to the reduced acute stress response and disrupted negative feedback, had significantly lower baseline CORT concentrations and significantly reduced body weight. These data indicate that translocation alters stress physiology and that chronic stress is potentially a major factor in translocation failure. Under current practices, the restoration of threatened species through translocation may unwittingly depend on the success of chronically stressed individuals. This conclusion emphasizes the need for understanding and alleviating translocation-induced chronic stress in order to use most effectively this important conservation tool.
36

JACOB, LINI, RV MANJU, ROY STEPHEN, MM VIJI, and BR REGHUNATH. "Effect of abiotic stress factors on growth, physiology and total withanolide production in Withania somnifera (L.) Dunal." Journal of Medicinal and Aromatic Plant Sciences 37, no. 1 (December 31, 2015): 18–21. http://dx.doi.org/10.62029/jmaps.v37i1.jacob.

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An investigation was carried out to study the effects of abiotic stress factors on growth, physiology and total withanolide production in Withania somnifera (L.) Dunal. The abiotic stresses were provided in the form of three levels of light stress (25%,50% and 75% shade) and three levels of water stress (25%,50% and 75% FC) along with control under optimum conditions. Withanolide production was significantly affected by various stress factors. Maimum values for plant height (57.75cm), length of tap root (28.00cm) total dry matter production (28.08g/plant) and specific leaf area(93.40 cm2/g)were recorded in plants grown under 75% shade. The secondary metabolite withanolide was found to be significantly affect by abiotic stress factors and was also recorded maximum (64.75mg/g) in plants grown under 75% shade.
37

Hall, R. W., and K. J. S. Anand. "Physiology of Pain and Stress in the Newborn." NeoReviews 6, no. 2 (February 1, 2005): e61-e68. http://dx.doi.org/10.1542/neo.6-2-e61.

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38

Hall, M. A., A. W. Berry, N. V. J. Harpham, G. Roveda-Hoyos, A. R. Smith, and S. O. El-Abd. "STRESS PHYSIOLOGY IN THE CONTEXT OF PROTECTED CULTIVATION." Acta Horticulturae, no. 323 (February 1993): 379–400. http://dx.doi.org/10.17660/actahortic.1993.323.36.

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39

Léon, Sébastien. "Endocytosis and stress: From mechanisms to cellular physiology." Biology of the Cell 113, no. 11 (October 14, 2021): 439–40. http://dx.doi.org/10.1111/boc.202100072.

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40

Lee, Young-Woo, Suh-Yeon Bea, Sang-Gyu Seo, Ie-Sung Shim, Sun-Hyung Kim, Sang-Gon Kim, Kyu-Young Kang, and Sun-Tae Kim. "Korean plant proteomics: pioneers in plant stress physiology." Journal of Plant Biotechnology 38, no. 2 (June 30, 2011): 151–61. http://dx.doi.org/10.5010/jpb.2011.38.2.151.

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41

Grout, Brian W. W. "Book Review: The Physiology of Plants Under Stress." Outlook on Agriculture 17, no. 2 (June 1988): 86. http://dx.doi.org/10.1177/003072708801700208.

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42

Negrão, A. B., P. A. Deuster, P. W. Gold, A. Singh, and G. P. Chrousos. "Individual reactivity and physiology of the stress response." Biomedicine & Pharmacotherapy 54, no. 3 (April 2000): 122–28. http://dx.doi.org/10.1016/s0753-3322(00)89044-7.

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43

Cobb, Mia, Alan Lill, and Pauleen Bennett. "Canine stress physiology and coping styles in kennels." Journal of Veterinary Behavior 9, no. 6 (November 2014): e11. http://dx.doi.org/10.1016/j.jveb.2014.09.036.

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44

Schüler, Göde, Axel Mithöfer, Ian T. Baldwin, Susanne Berger, Jürgen Ebel, Jonathan G. Santos, Gabriele Herrmann, et al. "Coronalon: a powerful tool in plant stress physiology." FEBS Letters 563, no. 1-3 (March 11, 2004): 17–22. http://dx.doi.org/10.1016/s0014-5793(04)00239-x.

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45

Hulten, Edward A., Marcio Sommer Bittencourt, Brian Ghoshhajra, and Ron Blankstein. "Stress CT perfusion: Coupling coronary anatomy with physiology." Journal of Nuclear Cardiology 19, no. 3 (March 29, 2012): 588–600. http://dx.doi.org/10.1007/s12350-012-9546-5.

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46

Chaturvedi, Palak, Arindam Ghatak, and Wolfram Weckwerth. "Pollen proteomics: from stress physiology to developmental priming." Plant Reproduction 29, no. 1-2 (June 2016): 119–32. http://dx.doi.org/10.1007/s00497-016-0283-9.

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47

Gutierrez, Claude, Tjakko Abee, and Ian R. Booth. "Physiology of the osmotic stress response in microorganisms." International Journal of Food Microbiology 28, no. 2 (December 1995): 233–44. http://dx.doi.org/10.1016/0168-1605(95)00059-3.

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48

Jäger, H. J., U. Hertstein, and A. Fangmeier. "The European Stress Physiology and Climate Experiment—project." European Journal of Agronomy 10, no. 3-4 (April 1999): 155–62. http://dx.doi.org/10.1016/s1161-0301(99)00006-4.

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49

Bhatnagar, M., and A. Bhatnagar. "Physiology ofAnabaena khannae andChlorococcum humicola under fluoride stress." Folia Microbiologica 49, no. 3 (May 2004): 291–96. http://dx.doi.org/10.1007/bf02931045.

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50

Lees, A. M., and J. B. Gaughan. "The physiology of heat stress in beef cattle." Animal - science proceedings 14, no. 2 (April 2023): 409. http://dx.doi.org/10.1016/j.anscip.2023.01.541.

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