Academic literature on the topic 'Streptococcus pneumonias'
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Journal articles on the topic "Streptococcus pneumonias"
Martynova, A. V., L. A. Balabanova, and O. A. Chulakova. "MULTILOCI SEQUESTERANT STRAINS OF STREPTOCOCCUS PNEUMONIAE ISOLATED FROM ELDERLY PATIENTS WITH COMMUNITY ACQUIRED PNEUMONIA." Bulletin of Siberian Medicine 13, no. 3 (June 28, 2014): 40–45. http://dx.doi.org/10.20538/1682-0363-2014-3-40-45.
Full textSchleupner, Charles J., and David K. Cobb. "A Study of the Etiologies and Treatment of Nosocomial Pneumonia in a Community-Based Teaching Hospital." Infection Control & Hospital Epidemiology 13, no. 9 (September 1992): 515–25. http://dx.doi.org/10.1086/646591.
Full textЧулакова, O. Chulakova, Балабанова, L. Balabanova, Мартынова, A. Martynova, Шепарев, and A. Sheparev. "Molecular-Epidemiological Monitoring of Strains of Streptococcus Pneumoniae Isolated from Elderly Patients with Community-Acquired Pneumonia." Journal of New Medical Technologies 21, no. 3 (September 5, 2014): 69–72. http://dx.doi.org/10.12737/5902.
Full textSzabó, Bálint Gergely, Katalin Szidónia Lénárt, Béla Kádár, Andrea Gombos, Balázs Dezsényi, Judit Szanka, Ilona Bobek, and Gyula Prinz. "A Streptococcus pneumoniae (pneumococcus) -infekciók ezer arca." Orvosi Hetilap 156, no. 44 (November 2015): 1769–77. http://dx.doi.org/10.1556/650.2015.30293.
Full textNikolenko, V. V., A. V. Nikolenko, and M. R. Minikeeva. "Nutritive status changes, studied in hiv-positive patients with pneumonias, caused by Streptococcus pneumoniae." Perm Medical Journal 35, no. 4 (December 15, 2018): 14–19. http://dx.doi.org/10.17816/pmj35414-19.
Full textDoerschuk, C. M., R. K. Winn, H. O. Coxson, and J. M. Harlan. "CD18-dependent and -independent mechanisms of neutrophil emigration in the pulmonary and systemic microcirculation of rabbits." Journal of Immunology 144, no. 6 (March 15, 1990): 2327–33. http://dx.doi.org/10.4049/jimmunol.144.6.2327.
Full textLaitinen, LA, AK Miettinen, E. Kuosma, L. Huhtala, and K. Lehtomaki. "Lung function impairment following mycoplasmal and other acute pneumonias." European Respiratory Journal 5, no. 6 (June 1, 1992): 670–74. http://dx.doi.org/10.1183/09031936.93.05060670.
Full textBriones, Maria Luisa, José Blanquer, David Ferrando, Maria Luisa Blasco, Concepción Gimeno, and Julio Marín. "Assessment of Analysis of Urinary Pneumococcal Antigen by Immunochromatography for Etiologic Diagnosis of Community-Acquired Pneumonia in Adults." Clinical and Vaccine Immunology 13, no. 10 (October 2006): 1092–97. http://dx.doi.org/10.1128/cvi.00090-06.
Full textCirino, Luís Marcelo Inaco, Filumena Maria da Silva Gomes, and Bernardo Nogueira Batista. "The etiology of extensive pleural effusions with troublesome clinical course among children." Sao Paulo Medical Journal 122, no. 6 (December 2004): 269–72. http://dx.doi.org/10.1590/s1516-31802004000600008.
Full textAhn, Danielle, and Alice Prince. "Participation of Necroptosis in the Host Response to Acute Bacterial Pneumonia." Journal of Innate Immunity 9, no. 3 (2017): 262–70. http://dx.doi.org/10.1159/000455100.
Full textDissertations / Theses on the topic "Streptococcus pneumonias"
Yoshioka, Cristina Ryoka Miyao. "Estudo das pneumonias causadas por Streptococcus pneumoniae em crianças internadas na enfermaria de pediatria do Hospital Universitário da Universidade de São Paulo." Universidade de São Paulo, 2009. http://www.teses.usp.br/teses/disponiveis/5/5141/tde-07122009-185246/.
Full textIntroduction: Currently, the annual incidence of the acquired pneumonia in the developing country communities are around 150.7 million cases, among childrens under 5 years of age, and 11 to 20 million (7-13%) of those require hospitalization due to their gravity. In general, the treatments used to be empirical, however, it is important to be noted that Streptococcus pneumoniae is far the major bacterial etiologic agent. It is necessary to keep monitoring the serotypes and the pattern of resistance in order to improve the therapy guidance. Methodology: Retrospective cohort study with inclusion of the 107 childrens with clinical and radiological diagnosis of the pneumonia, and the isolation of Streptococcus pneumoniae in the blood and/or pleural fluid during the period of January 2003 to October 2008. It was performed determination of the minimum inhibitory concentration (MIC) related to the penicillin and other antibiotics. The sensitivity analysis to the penicillin was based on Clinical and Laboratory Standards Institute (CLSI), 2008, recommendations. They were performed serotyping in 96 pneumococcal strains (89.7%) and they were analyzed datas referred to the considered population and their clinical course. Results: About 47.5% of admissions in the ward were caused by pneumonia or bronchopneumonia, and the average positive occurrences in the pneumococcal (blood and / or pleural) culture were 2.5%. It was noted a clear seasonality phenomena of the pneumococcal pneumonia. About 70% of the cases occurred during months of June to October. The median age was 23 months (82.2%<5 years); with predominance of males (58.9%); in the 23,4% of the cases the antibiotic therapy was used during two months prior to the admission; the daycare frequency of the childs less than 2 years were 36.4%; only one case with complete vaccination heptavalent; associated disease was detected in the 44.9% of the cases and the most frequent was wheezing (77.1%); time of fever and respiratory symptoms before admission were 4 days; the need for noninvasive oxygen therapy occurred in 70.1% being 4 days of the average time of the use; the need for mechanical ventilation occurred in 19.6%; the median period of stay were 9 days. In 62% of the cases there were the most frequent complications: empyema (53%) and non-complicated pleural effusion (42%). The childrens with empyema had more necrotizing pneumonia, lung abscess, sepsis, pneumothorax, need for decortication, and even higher mortality (all with p<0.05). The childrens with complications had more days of respiratory symptoms before admission (3x5days), more time of the fever after initiation with antibiotic (1x4, 5days), they need noninvasive oxygen therapy (58,5 x77, 3%) and mechanical ventilation (7 , 3x27, 3%) for more time and remained hospitalized during longer period(5x12 days). Among 107 pneumococcal strains, 100 (93.5%) were susceptible to penicillin and 7 (6.5%) presented intermediate sensitivity. All strains tested were sensitive to rifampicin and vancomycin, and they maintained good sensitivity to clindamycin, chloramphenicol, ceftriaxone, erythromycin and levofloxacin. Five strains were multi-resistant. It was noted that the geometric mean of minimum inhibitory concentrations (GMC) to penicillin were higher in children with complications. The most frequent serotypes were: 14 (36.5%), 1 (16.7%), 5 (14.6%) and 6B (6.3%). The serotype 14 presented the highest GMC for penicillin and it was verified a progressive increase during the years of the study. The coverage of serotypes by the heptavalent vaccine would be cover 53.1% and this less coverage is represented by serotype 1 and 5, which corresponds to 31.3% of the cases. The coverage of serotypes associated with resistance would be 94.2%. The coverage of the 10-valent vaccine would be 86.5% and for 13-valent would be 96.9%. Three cases that carried to died (2.8%) had median age of 18 months, all they male, all they with minimum inhibitory concentration for penicillin <= 1g/mL, all they progressed to empyema and sepsis. Two of them were serotype 5 and one of them was serotype 14. Conclusions: Approximately 2.5% of children were admitted with diagnosis of pneumonia were diagnosed as pneumococcal pneumonia. It was verified a clear seasonality phenomena. They were observed complications in 62% of the cases. The most frequent were: empyema and non-complicated pleural effusion cases. It was confirmed a higher GMC for penicillin in children with complications compared to the children without complications. The most frequent serotypes were 14, 1, 5 and 6B and the serotypes 1 and 5 accounted 31.3%. The coverage of heptavalent vaccine for the isolated serotypes would be 53.1%. The sensitivity to the penicillin of the isolated pneumococcal was 93.5%. Therefore, the therapy option remains being the penicillin.
Bazaz, Rohit. "The effect of Streptococcus pneumoniae pneumonia on atherosclerosis." Thesis, University of Sheffield, 2016. http://etheses.whiterose.ac.uk/16897/.
Full textKoppe, Uwe Moritz Eberhard. "Role of type I interferons in Streptococcus pneumoniae pneumonia." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2012. http://dx.doi.org/10.18452/16532.
Full textStreptococcus pneumoniae is the leading cause of community-acquired pneumonia world-wide. A detailed understanding of the host-pathogen interactions is required in order to foster the development of new therapeutic strategies. Here, I (I) characterized an innate immune recognition pathway that senses pneumococcal infection and triggers the production of type I interferons (IFNs), and (II) examined the role of type I IFNs in pneumococcal pneumonia in mice. Human and murine macrophages, but not alveolar epithelial cells, produced type I IFNs after infection with S. pneumoniae. This induction was dependent on the virulence factor pneumolysin, the phagocytosis of the bacteria, and the acidification of the endosome. Moreover, it appeared to be mediated by a cytosolic DNA-sensing pathway involving the adaptor molecule STING and the transcription factor IRF3. Type I IFNs produced by S. pneumoniae-infected macrophages positively regulated the expression of IFN-stimulated genes (ISGs) and chemokines in macrophages and co-cultured alveolar epithelial cells in vitro and in mouse lungs in vivo. However, in a murine model of pneumococcal pneumonia, type I IFN signaling was detrimental to the host defense. Mice deficient in the type I IFN signaling or double deficient in type I and type II IFN signaling had a significantly reduced bacterial load in the lung and a diminished reduction of body temperature and body weight compared to wild-type mice. The decreased susceptibility of the knockout mice was unlikely to be attributable to alterations in cell recruitment or cytokine/chemokine production. In conclusion, type I IFNs are induced during pneumococcal infection. However, despite their positive effects on the expression of some ISGs and chemokines, they negatively affect the outcome of pneumococcal pneumonia in an in vivo mouse model. Targeting the type I IFN system could potentially be an effective way in enhancing the immune response in patients with S. pneumoniae pneumonia.
McNamee, Lynnelle Ann. "Effects of a primary influenza infection on susceptibility to a secondary Streptococcus pneumoniae infection." Diss., Montana State University, 2006. http://etd.lib.montana.edu/etd/2006/mcnamee/McNameeL1206.pdf.
Full textSilva, Júnior Jailton de Azevedo. "Estudo da colonização nasofaringeana por Streptococcus pneumoniae em crianças com suspeita clínica de pneumonia." reponame:Repositório Institucional da FIOCRUZ, 2011. https://www.arca.fiocruz.br/handle/icict/4220.
Full textMade available in DSpace on 2012-07-19T21:35:50Z (GMT). No. of bitstreams: 1 Jailton de Azevedo Silva Junior Estudo da colonização....pdf: 1101716 bytes, checksum: 049972199c76bb97406fc006c2ba9c27 (MD5) Previous issue date: 2011
Fundação Oswaldo Cruz. Centro de Pesquisas Gonçalo Moniz. Salvador, Bahia, Brasil
Streptococcus pneumoniae constitui um dos mais importantes patógenos bacterianos do trato respiratório, podendo causar infecções invasivas e não invasivas, levando a altas taxas de morbi-mortalidade, particularmente em crianças menores de cinco anos de idade. A bactéria ganha acesso ao hospedeiro através da colonização da nasofaringe, que representa um importante reservatório para a transmissão deste patógeno na comunidade, contribuindo para a disseminação horizontal de S. pneumoniae entre os indivíduos de uma população. No presente estudo, procuramos caracterizar o perfil de colonização nasofaringeana por S. pneumoniae em pacientes menores de cinco anos de idade com suspeita clínica de pneumonia, atendidos na Unidade de Saúde de São Marcos, Bairro de Pau da Lima, Salvador, no ano de 2009. Um total de 205 swabs foram coletados entre as crianças consideradas elegíveis para o estudo. Os isolados de S. pneumoniae foram identificados através de métodos microbiológicos clássicos e a determinação do sorogrupo/sorotipo foi realizada empregando-se a técnica de Multiplex-PCR. A sensibilidade a sete antimicrobianos foi testada através da técnica de microdiluição em caldo, sendo que os isolados com CIM para penicilina ≥ 0,125 μg/mL foram considerados não-susceptíveis. A técnica de PFGE foi realizada para 26 isolados correspondentes aos sorotipos mais frequentes e associados a não-sensibilidade à penicilina (sorotipos 14, 19F e 23F). Um total de 72 (35,1%) crianças foram diagnosticadas com pneumonia, sendo 39 (54,2%) menores de dois anos de idade. A taxa de colonização geral foi de 50,2%, não havendo diferença entre essas taxas quando se considerou o grupo de crianças confirmadas e suspeitas para pneumonia. Crianças na faixa etária de 36 a 47 meses formaram o grupo com maior risco de ter pneumonia bacteriana (OR: 3.17 [1.29-7.88]). Entre os sorotipos encontrados, o sorogrupo 6 (6A/6B) (17,3%) foi predominante, seguido dos sorotipos 14 (15,4%), 19F (10,6%), sorogrupo 15 (15B/15C) (9,6%), 23F (6,7%) e o sorotipo 19A (6,7%). Os demais sorotipos e sorogrupos compreenderam 33,7%. O padrão de sorotipos foi semelhante aqueles encontrados nos casos de meningite pneumocócica na cidade de Salvador. Um total de 41 isolados (39,8%) apresentaram CIM ≥ 0,125 μg/mL para penicilina e a resistência a SMX-TMP foi identificada em 69,2% dois isolados. A tipagem por PFGE identificou 11 padrões eletroforéticos, sendo que a maioria dos isolados do sorotipo 14 estavam relacionados a clones amplamente disseminados entre os casos de doença pneumocócica (“A” e “GK”). Um total de 50,5% dos isolados foram de sorotipos inclusos na vacina decavalente (PCV10) e considerando os isolados não-susceptíveis à penicilina, esta representatividade foi de 90,2%. O estudo ressalta a importância de um contínuo monitoramento do perfil de sorotipos na colonização nasofaringeana por S. pneumoniae, no período pós-vacina e da necessidade de busca de novos métodos de diagnóstico que otimizem a definição da pneumonia.
Streptococcus pneumoniae is one of the most important bacterial pathogens of the respiratory tract, causing invasive and noninvasive infections, leading to high rates of morbidity and mortality, particularly among children under five years old. The bacterium gains access to the host by colonizing the nasopharynx, which represents an important reservoir for transmission of this pathogen in the community, contributing to the horizontal spread of S. pneumoniae among individuals in a population. In this study, we sought to characterize the profile of nasopharyngeal colonization by S. pneumoniae in patients under five years of age with clinical suspicion of pneumonia seeking medical care at the Unidade de Saúde de São Marcos, District of Pau da Lima, Salvador, in 2009. A total of 205 swabs were collected from children eligible for the study. The isolates of S. pneumoniae were identified by classical methods and the determination of the serogroup / serotype was performed using the technique of multiplex-PCR. The sensitivity to seven antibiotics was tested by the microdilution broth method, and strains with MIC for penici≥lli n 0.125 mg/mL were considered non-susceptible. The PFGE technique was performed for 26 strains corresponding to serotypes more frequent and associated with nonsusceptibility to penicillin (serotypes 14, 19F and 23F). A total of 72 (35.1%) children were diagnosed with pneumonia, 39 (54.2%) less than two years old. The overall colonization rate was 50.2%, with no difference between those rates when considering the children's group confirmed and suspected to pneumonia. Children aged 36 to 47 months formed the group with higher risk for bacterial pneumonia (OR: 3.17 [1.29-7.88]). Among the serotypes, serogroup 6 (6A/6B) (17.3%) predominated, followed by serotypes 14 (15.4%), 19F (10.6%), serogroup 15 (15B/15C) (9.6%), 23F (6.7%) and serotype 19A (6.7%). The other serotypes and serogroups comprised 33.7%. The pattern of serotypes was similar to those found in cases of pneumococcal meningitis in Salvador. A total of 41 isolates (39.8%) had MIC ≥ 0.125 mg / mL and resistance to TMP-SMX was identified in 69.2% of isolates. Molecular typing identified 11 electrophoretic patterns, whereas most isolates of serotype 14 was associated with widespread clones among cases of pneumococcal disease ("A" and "GK"). The 10-valent conjugate vaccine (PCV10) implemented in Brazil shows a coverage of 50.5% from serotypes in the population and 90.2% for isolates not susceptible to penicillin. The study underscores the importance of continued monitoring of the prevalence of serotypes in nasopharyngeal colonization by S. pneumoniae, in the post-vaccine era, and the need to search for new methods for diagnosing pneumonia.
Scholtz, Janet. "The serotypes and antimicrobial susceptibility patterns of streptococcus pneumoniae in the Cape Peninsula." Thesis, Cape Technikon, 2000. http://hdl.handle.net/20.500.11838/1464.
Full textStreptococcus pneumoniae (S.pneumoniae) infections are an important cause of morbidity and mortality in adults and children worldwide. Mortality rates are highest amongst the very young and the elderly. Streptococcus pneumoniae is the most common form of community acquired bacterial pneumonia. Other diseases commonly caused by Streptococcus pneumoniae include meningitis, pericarditis, bacteraemia and septicaemia. Penicillin is today still consid3red the drug of choice when treating pneumococcal infections. The emergence of resistant pneumococcal strains has made it necessary to adapt antimicrobial regimens when treating pneumococcal infections. Hansman (1967) reported the first penicillin resistant strain, which was isolated from a woman in Australia in 1967. Since then penicillin and multi-resistant Streptococcus pneumoniae strains have been observed worldwide, including South Africa. Streptococcus pneumoniae infections may be caused by anyone of the 84 serotypes recognized to date. The distribution of serotypes varies, depending on geographical area, age and site of infection. High-level penicillin resistance and multiple resistant Streptococcus pneumoniae strains have been recognised worldwide in a few pneumococcal serotypes. Pneumococcal vaccines have been used since the seventies. These capsular polysaccharide vaccines are generally recommended for at risk population such as the elderly and immunocompromised patients. This vaccine is not effective in children under 2 years old. The current vaccine in South Africa (Pneumovax, MSD) consists of purified capsular polysaccharides of 23 pneumococcal serotypes. Conjugated polysaccharide vaccines have been developed to overcome the problems of efficacy in children < 2 years old. These vaccines consist of a capsular polysaccharide linked to a protein carrier, which makes them immunogenic in infants. Clinical trials of these vaccines are currently under way to demonstrate safety, efficacy and immunogenicity. Knowledge of serotype distribution and antimicrobial susceptibility patterns are important in relation to the treatment of pneumococcal diseases and vaccination programmes.
Abdeldaim, Guma M. K. "PCR detection of Streptococcus pneumoniae and Haemophilus influenzae in pneumonia patients." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl.[distributör], 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-107931.
Full textEkdahl, Karl. "Immunological aspects on pneumococcal infections with special reference to bacteremic pneumococcal infections and recurrent pneumonia /." Lund : Dept. of Infectious Diseases, University of Lund, 1995. http://catalog.hathitrust.org/api/volumes/oclc/39177549.html.
Full textMachado, Lais Del Prá Netto. "Pneumonia bacteriana adquirida na comunidade: avaliação clínico-epidemiológica e padronização de métodos moleculares para detecção de Mycoplasma pneumoniae, Haemophilus influenzae e Streptococcus pneumoniae." reponame:Repositório Institucional da UFSC, 2015. https://repositorio.ufsc.br/xmlui/handle/123456789/158899.
Full textMade available in DSpace on 2016-02-09T03:17:04Z (GMT). No. of bitstreams: 1 336847.pdf: 2261623 bytes, checksum: 5220583fc8a657b02f17b72e42ab63bc (MD5) Previous issue date: 2015
A pneumonia pode ser causada por diversos microrganismos e classificada de forma abrangente, havendo poucos e frágeis estudos clínicos e epidemiológicos sobre pneumonias adquiridas na comunidade (PACs). Os patógenos mais frequentes nas PACs são Streptococcus pneumoniae e Haemophilus influenzae (em pneumonias típicas) e Mycoplasma pneumoniae (em pneumonias atípicas). Assim, o objetivo deste trabalho foi padronizar uma metodologia para detecção molecular dessas bactérias em amostras de orofaringe, avaliar sua prevalência e o perfil clínico-epidemiológico de pacientes com PAC em um hospital na cidade de Blumenau/SC. Para tanto, além da técnica de PCR padronizada in house, foram realizadas culturas de raspado de orofaringe e pesquisa de anticorpos específicos para detecção de M. pneumoniae. A reação de PCR realizada com os iniciadores desenhados neste estudo para M. pneumoniae apresentou sensibilidade 10x maior que a reação mais citada na literatura, e a sensibilidade das reações para S. pneumoniae e H. influenzae foi 0,1ng de DNA/reação. Dos 58 pacientes incluídos no estudo, H. influenzae e S. pneumoniae foram detectados respectivamente em 41,38% e 15,52% das amostras. M. pneumoniae não foi detectado em nenhuma amostra analisada por métodos de cultura, PCR e sorologia com anticorpos específicos IgM. Todavia não se exclui a circulação dessa bactéria na região devido à presença de anticorpos IgG específicos. A maioria dos pacientes com PAC avaliados apresentou idade =65 anos e pelo menos uma comorbidade. A maioria dos pacientes apresentou quatro ou mais sinais e sintomas, destes os mais prevalentes foram dispneia, tosse, secreção purulenta e crepitações. Evidenciou-se, neste estudo, a baixa aderência dos clínicos às Diretrizes Brasileiras para diagnóstico, estratificação e tratamento dos pacientes com suspeita de PAC, pois os exames complementares para diagnóstico e avaliação de risco dos casos e o antibiótico escolhido para grande parte dos pacientes não estava de acordo com a determinação das diretrizes. Sugere-se a replicação desta pesquisa, pois os resultados foram de encontro àqueles apresentados na literatura relacionada ao entendimento de PAC, acompanhamento dos ciclos epidêmicos de M. pneumoniae na região e conhecimento da real prevalência dos patógenos típicos, uma vez que o H. influenzae foi o patógeno mais detectado.
Abstract : Pneumonia can be caused by different microorganisms and classified through comprehensive forms, but there are few and fragile clinical and epidemiological studies of community-acquired pneumonia (CAPs). The most common pathogens in CAPs are Streptococcus pneumoniae and Haemophilus influenzae (in typical pneumonia) and Mycoplasma pneumoniae (in atypical pneumonia). Therefore, the aim of this study was to standardize a methodology for molecular detection of these bacteria in the oropharynx samples, and to evaluate their prevalence and the clinical and epidemiological profile of patients with CAP in a hospital in Blumenau/SC. Thus, besides the standard technique PCR in-house, oropharyngeal swab cultures and search for specific antibodies for detection of M. pneumoniae were performed. The PCR reaction performed with primers designed in this study for M. pneumoniae showed sensitivity greater than 10-fold the most cited in the literature reaction and the sensitivity of the reactions to S. pneumoniae and H. influenzae was 0.1 ng DNA / reaction. Out of the 58 patients included in the study, H. influenzae and S. pneumoniae were detected respectively in 41.38% and 15.52% of the samples. M. pneumoniae was not detected in any sample analyzed by culture methods, PCR and serology with IgM specific antibodies. Nevertheless it is not possible to exclude the circulation of this bacterium in the region due to the presence of specific IgG antibodies. Most CAP patients evaluated were 65 years or older and had at least one comorbidity. Most of the patients had four or more signs and symptoms, the most prevalent ones were dyspnea, cough, purulent secretion and crackles. It is evident in this study the low adherence of the practitioners to Brazilian Guidelines for the diagnosis, stratification and treatment of patients with suspected CAP, as the laboratory tests for diagnosis and case risk assessment and also the antibiotic selected for most patients were not determined according to the guidelines. Replication of this research is indicated for a better understanding of the CAP, support of epidemic cycles of M. pneumoniae in the region and knowledge of the real prevalence of the typical pathogens.
Piroth, Lionel. "Apports d'un modèle de pneumonie expérimentale bactériémique à streptococcus pneumoniae de sensibilité diminuée à la pénicilline dans la prise en charge des pneumonies humaines." Dijon, 2001. http://www.theses.fr/2001DIJOMU02.
Full textBooks on the topic "Streptococcus pneumonias"
Iovino, Federico, ed. Streptococcus pneumoniae. New York, NY: Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9199-0.
Full textNuorti, J. Pekka. Prevention of pneumococcal disease among infants and children: Use of 13-valent pneumococcal conjugate vaccine and 23-valent pneumococcal polysaccharide vaccine : recommendations of the Advisory Committee on Immunization Practices (ACIP). Atlanta, GA: Dept. of Health and Human Services, Centers for Disease Control and Prevention, 2010.
Find full textLife with the pneumococcus: Notes from the bedside, laboratory, and library. Philadelphia: University of Pennsylvania Press, 1985.
Find full textBrenwald, Nigel Peter. Characterisation of a multidrug efflux system in Streptococcus pneumoniae. Birmingham: University of Birmingham, 2000.
Find full textJohnston, Nicole J. Prevalence and characterization of the mechanisms of macrolide, lincosamide, and streptogramin resistance among isolates of Streptococcus pneumoniae and viridans streptococci. Ottawa: National Library of Canada, 1999.
Find full textDoherty, Neil Christopher. A molecular analysis of hyaluronate lyase production in Streptococcus pneumoniae. [s.l.]: typescript, 2000.
Find full textBoost, Maureen Valerie. Carriage and antibiotic resistance of Streptococcus pneumoniae in Hong Kong. [S.l: The Author], 2004.
Find full textFile, Thomas. New insights in the treatment of severe infections in the multiple-drug resistant situation: Proceedings of a satellite symposium to the 11th International Congress on Infectious Diseases, Cancun, Mexico, March 5, 2004. Basel, Switzerland: Karger, 2004.
Find full textI, Tuomanen Elaine, ed. The pneumococcus. Washington, DC: ASM Press, 2004.
Find full textRankin, Barbara Anne. The development of a dot immunobinding assay for the detection of streptococcus pneumoniae in sputum. [s.l: The Author], 1990.
Find full textBook chapters on the topic "Streptococcus pneumonias"
Kashani, John, Richard D. Shih, Thomas H. Cogbill, David H. Jang, Lewis S. Nelson, Mitchell M. Levy, Margaret M. Parker, et al. "Streptococcus pneumoniae." In Encyclopedia of Intensive Care Medicine, 2146–51. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_165.
Full textNolte, Oliver. "Streptococcus pneumoniae." In Lexikon der Infektionskrankheiten des Menschen, 779–83. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-39026-8_1051.
Full textRoy, Bronwen, and Marcel Leroi. "Streptococcus pneumoniae." In PCR for Clinical Microbiology, 201–3. Dordrecht: Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-90-481-9039-3_26.
Full textTuomanen, Elaine. "Streptococcus pneumoniae." In The Prokaryotes, 149–62. New York, NY: Springer US, 2006. http://dx.doi.org/10.1007/0-387-30744-3_4.
Full textBalakrishnan, Indran. "Streptococcus pneumoniae." In Principles and Practice of Clinical Bacteriology, 41–57. Chichester, UK: John Wiley & Sons, Ltd, 2006. http://dx.doi.org/10.1002/9780470017968.ch3.
Full textSutcliffe, Joyce, and Marilyn C. Roberts. "Streptococcus pneumoniae." In Frontiers in Antimicrobial Resistance, 314–29. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555817572.ch23.
Full textNeves, Felipe P. G., and Tatiana C. A. Pinto. "Streptococcus pneumoniae." In Molecular Typing in Bacterial Infections, Volume I, 139–52. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-74018-4_6.
Full textGasc, Anne-Marie, and Sol H. Goodgal. "Streptococcus pneumoniae R6." In Bacterial Genomes, 755–57. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-6369-3_81.
Full textPaton, James C., and David E. Briles. "Streptococcus pneumoniae Vaccines." In New Bacterial Vaccines, 294–310. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0053-7_19.
Full textKashani, John, Richard D. Shih, Thomas H. Cogbill, David H. Jang, Lewis S. Nelson, Mitchell M. Levy, Margaret M. Parker, et al. "Streptococcus pneumoniae Infections." In Encyclopedia of Intensive Care Medicine, 2151. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_2237.
Full textConference papers on the topic "Streptococcus pneumonias"
Zhang, Y., L. K. Sharma, A. J. Losier, E. Ifedigbo, M. Sauler, I. S. Bazan, C. Dela Cruz, and P. Lee. "Role of PINK1 Mediated Mitophagy During Streptococcus Pneumoniae Pneumonia." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a7230.
Full textSanz, F., L. A. Ruiz Iturriaga, M. M. García Clemente, P. P. España Yandiola, L. Serrano Fernández, A. Uranga Echeverria, J. Herrero Huertas, E. Fernández Fabrellas, and PneumoCuore Group. "Streptococcus Pneumoniae Serotype Determine Cardiac Complications During Invasive Pneumococcal Pneumonia." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4228.
Full textCosta, Anna Carolina Dockhorn de Menezes Carvalho, Lucas Dalvi Armond Rezende, Maria Gabriella Bianconi, Maxsuelen Rosa Da Silva Santos, and Gabriel Confalonieri Brtoldi Brtoldi. "O QUE HÁ DE EVIDÊNCIAS SOBRE PNEUMONIA ADQUIRIDA NA COMUNIDADE (PAC) EM CRIANÇAS: UMA REVISÃO DA LITERATURA EM MEIO À PANDEMIA DO COVID-19." In I Congresso Nacional de Microbiologia Clínica On-Line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/1203.
Full textLosier, A. J., L. K. Sharma, Y. Zhang, W. Liu, P. Lee, and C. Dela Cruz. "Streptococcus Pneumoniae Pneumonia Induces Mitochondrial Biogenesis and Antioxidant Responses in Mice." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5595.
Full textHofstra, JJ, EJ Hoorn, AP Vlaar, D. Wouters, MM Levi, S. Zeerleder, and MJ Schultz. "Complement Inhibition with Human C1-Inhibitor Concentrate in Streptococcus pneumoniae Pneumonia in Rats." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3232.
Full textPaternotte, Nienke, Bart Kamies, W. G. Boersma, and W. A. Van Der Reijden. "Adjusted lytA qPCR for improved detection of Streptococcus pneumoniae in community-acquired pneumonia." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa4550.
Full textYang, Minlan, Gianluigi Li Bassi, Anna Motos, Hua Yang, Joaquim Bobi, Andrea Meli, Denise Battaglini, et al. "Corticosteroid therapy combined with antibiotics for severe Streptococcus pneumoniae pneumonia in ventilated piglets." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa4553.
Full textUmer, Erum, Zafar Ahmed, and Syed Ali Arsalan. "Macrolides resitance to streptococcus pneumonia." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa2267.
Full textPereira, Enzzo Cavalcante, CÁSSIO DE SOUSA LEAL, CLARISSE FRANCELINO BASTOS, GILDÊNIO ESTEVAM FREIRE, and EDLAINNY ARAUJO RIBEIRO. "PANORAMA DA MORTALIDADE ASSOCIADA A PNEUMONIA EM REDENÇÃO-PA NO PERÍODO DE 2010 A 2019." In II Congresso Nacional de Microbiologia Clínica On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/ii-conamic/39.
Full textSteck, Patrick, Anja Honecker, Felix Ritzmann, Giovanna Vella, Christian Herr, Markus Bischoff, Timo Speer, Robert Bals, and Christoph Beisswenger. "IL-17RE/IL-17C mediate the recruitment of neutrophils during acute Streptococcus pneumoniae pneumonia." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa2381.
Full textReports on the topic "Streptococcus pneumonias"
Kindy, Mark S. ENU Mutagenic Screen for Susceptibility and Resistance to Streptococcus Pneumoniae. Fort Belvoir, VA: Defense Technical Information Center, November 2005. http://dx.doi.org/10.21236/ada441504.
Full textCrum, N. F., C. P. Barrozo, F. A. Chapman, M. A. Ryan, and K. Russell. An Outbreak of Conjunctivitis Due to a Novel Unencapsulated Streptococcus pneumonia Among Military Trainees. Fort Belvoir, VA: Defense Technical Information Center, October 2004. http://dx.doi.org/10.21236/ada433371.
Full textAlexandrova, Alexandra, Lena Setchanova, Daniela Pencheva, and Ivan Mitov. Molecular Serotyping of Serogroup 6 Streptococcus pneumoniae Isolates Has Shown Emergence of Serotype 6C After the Implementation of 10‑valent Pneumococcal Conjugate Vaccine (PhiD‑CV) in Bulgaria. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, August 2019. http://dx.doi.org/10.7546/crabs.2019.08.14.
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