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Books on the topic 'Streptococcus pneumoniae'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Iovino, Federico, ed. Streptococcus pneumoniae. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9199-0.

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2

Brenwald, Nigel Peter. Characterisation of a multidrug efflux system in Streptococcus pneumoniae. University of Birmingham, 2000.

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3

Nuorti, J. Pekka. Prevention of pneumococcal disease among infants and children: Use of 13-valent pneumococcal conjugate vaccine and 23-valent pneumococcal polysaccharide vaccine : recommendations of the Advisory Committee on Immunization Practices (ACIP). Dept. of Health and Human Services, Centers for Disease Control and Prevention, 2010.

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4

Doherty, Neil Christopher. A molecular analysis of hyaluronate lyase production in Streptococcus pneumoniae. typescript, 2000.

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5

Boost, Maureen Valerie. Carriage and antibiotic resistance of Streptococcus pneumoniae in Hong Kong. The Author], 2004.

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6

Kanclerski, Krzysztof. Streptococcus pneumoniae haemolysin (pneumolysin): Production, purification and use as antigen for diagnosis of pneumococcal pneumonia. [s.n.], 1987.

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7

I, Tuomanen Elaine, ed. The pneumococcus. ASM Press, 2004.

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8

Kragsbjerg, Peter. Cytokines in infection: Clinical and experimental studies with special reference to Streptococcus pneumoniae. Uppsala universitet, 1996.

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9

Johnston, Nicole J. Prevalence and characterization of the mechanisms of macrolide, lincosamide, and streptogramin resistance among isolates of Streptococcus pneumoniae and viridans streptococci. National Library of Canada, 1999.

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10

File, Thomas. New insights in the treatment of severe infections in the multiple-drug resistant situation: Proceedings of a satellite symposium to the 11th International Congress on Infectious Diseases, Cancun, Mexico, March 5, 2004. Karger, 2004.

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11

Rankin, Barbara Anne. The development of a dot immunobinding assay for the detection of streptococcus pneumoniae in sputum. The Author], 1990.

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12

Mohammadi, Maryam Khan. Vergleichende Untersuchung der bakteriziden Aktivität von Moxifloxacin und Ciprofloxacin gegenüber Escherichia coli, Staphylococcus aureus, Enterococcus faecalis, Enterococcus faecium und Streptococcus pneumoniae. [s.n.], 1999.

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13

Berlin, Freie Universität, ed. Molekulargenetische Untersuchung des Penicillin-bindenden Proteins (PBP) 1a von Streptococcus pneumoniae: Verwandtschaft von PBP 1a Mosaikgenen in Penicillin-resistenten klinischen Stämmen. [s.n.], 1992.

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14

Streptococcus Pneumoniae. Elsevier, 2015. http://dx.doi.org/10.1016/c2012-0-00722-3.

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15

Gillespie. Streptococcus Pneumoniae. John Wiley & Sons, 2002.

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16

Iovino, Federico. Streptococcus Pneumoniae: Methods and Protocols. Springer New York, 2019.

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17

Lydyard, Peter, Michael Cole, John Holton, et al. Case Studies in Infectious Disease: Streptococcus pneumoniae. Garland Science, 2009. http://dx.doi.org/10.4324/9780203854075.

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18

Tomasz, Alexander, ed. Streptococcus Pneumoniae: Molecular Biology & Mechanisms of Disease. MARY ANN LIEBERT PUBLISHING, 2000.

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19

Brown, Jeremy, Sven Hammerschmidt, and Carlos Orihuela. Streptococcus Pneumoniae: Molecular Mechanisms of Host-Pathogen Interactions. Elsevier Science & Technology Books, 2015.

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20

Ana María Del Carmen Martín Rodríguez. Biología Molecular Del Bacteriófago Cp1 de Streptococcus Pneumoniae. Universidad Complutense de Madrid, Servicio de Publicaciones, 2006.

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21

Parker, James N., and ICON Health Publications. The Official Patient's Sourcebook on Streptococcus Pneumoniae Infections. Icon Health Publications, 2002.

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22

Brown, Jeremy M., Sven Hammerschmidt, and Carlos Orihuela. Streptococcus Pneumoniae: Molecular Mechanisms of Host-Pathogen Interactions. Elsevier Science & Technology Books, 2015.

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23

Austrian, Robert, and Lewis Thomas. Life with the Pneumococcus: Notes from the Bedside, Laboratory, and Library. University of Pennsylvania Press, 2016.

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24

Harrison, Mark. Streptococci and staphylococci. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198765875.003.0013.

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This chapter describes the microbiology of streptococci and staphylococci as it applies to Emergency Medicine, and in particular the Primary FRCEM examination. The chapter outlines the key details of the methods of spread and clinical features of Streptococcus pneumoniae, Streptococcus pyogenes, alpha-haemolytic streptococci, Staphylococcus aureus, MRSA, and Staphylococcus epidermidis. This chapter is laid out exactly following the RCEM syllabus, to allow easy reference and consolidation of learning.

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25

Life with the pneumococcus: Notes from the bedside, laboratory, and library. University of Pennsylvania Press, 1985.

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26

Ortiz, Laura Lagartera. Estructura y Función de Las Fosforilcolín Esterasa de Streptococcus Pneumoniae. Universidad Complutense de Madrid, Servicio de Publicaciones, 2008.

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27

Kröger, Alexa. Einfluss der Desinfektion mit Isopropanol auf die Virulenz von Streptococcus pneumoniae. 1985.

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28

Spratt, Brian G., Donald Morrison, Elaine I. Tuomanen, and Timothy J. Mitchell. Pneumococcus. Wiley & Sons, Limited, John, 2014.

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29

(Editor), Elaine I. Tuomanen, Timothy J. Mitchell (Editor), Donald Morrison (Editor), and Brian G. Spratt (Editor), eds. The Pneumococcus. ASM Press, 2004.

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30

Fernández, Eduardo Díaz. Estudio Mediante Ingeniería de Proteínas de la Autolisina Lyta de Streptococcus Pneumoniae. Universidad Complutense de Madrid, Servicio de Publicaciones, 2005.

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31

The pneumococcus. ASM Press, 2004.

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32

Streptococcus pneumoniae in child care centres: A point prevalence survey of nasopharyngeal carriage, antibiotic resistance and risk factors for resistance. National Library of Canada, 1998.

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33

Albers, Cordula. Einfluss des Zeitpunktes der Gabe von Moxifloxacin und Azithromycin auf die Überlebensfähigkeit humaner respiratorischer Epithelzellen nach Infektion mit Streptococcus Pneumoniae in vitro. 2006.

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34

Kühn, Günter Georg. Untersuchung über den Einfluss von Humanm Vollblut auf Escherichia coli, Streptococcus Paecalis, Klebstella Pneumoniae Staphylococcus Aureus und Pseudomonas Aeruginosa bei Expostion mit Subinhibitorischen Konzentrationen von Penicillinen. 1985.

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35

Informe regional de SIREVA II, 2017. Organización Panamericana de la Salud, 2020. http://dx.doi.org/10.37774/9789275323076.

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La vigilancia pasiva por laboratorio de Streptococcus pneumoniae se realiza en los países de la Región de las Américas desde 1993 bajo la denominación de Sistema Regional de Vacunas (SIREVA), con el apoyo de la Organización Panamericana de la Salud. En 1997, los países de la Región propusieron introducir las pruebas de laboratorio para Haemophilus influenzae, y en el 2000, para Neisseria meningitidis. Así se amplió la vigilancia por laboratorio de las enfermedades bacterianas invasivas con los tres patógenos anteriores, esta vez bajo la denominación de SIREVA II. La red está compuesta por 19 l

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36

Informe regional de SIREVA II, 2018. Pan American Health Organization, 2021. http://dx.doi.org/10.37774/9789275324035.

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En esta nueva edición del Informe regional de SIREVA II correspondiente al 2018 se presenta información sobre los serotipos o serogrupos de los tres agentes asociados con las enfermedades bacterianas invasivas que son objeto de vigilancia pasiva por laboratorio —Streptococcus pneumoniae, Haemophilus influenzae y Neisseria meningitidis—, los cuales se identificaron en muestras obtenidas durante el 2018 de pacientes que presentaron algunas de esas enfermedades. Estos datos se presentan de forma agrupada en cuadros y no han sido objeto de ningún tipo de análisis estadístico. En algunas ocasiones,

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37

García, Yolanda García. Estudio de la Actividad de Amoxicilina/ Clavulanico y Ciprofloxacino in Vitro y en un Modelo Experimental de Neumonía Por Cepas de Streptococcus Pneumoniae de Diferente Serotipo y Sensibilidad a Penicilina. Universidad Complutense de Madrid, Servicio de Publicaciones, 2006.

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38

Rajakrishna, Premil, Stewart Cameron, and Neil Turner. Nephrotic syndrome. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0052.

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Nephrotic syndrome is the constellation of manifestations seen in patients with such severe proteinuria that serum albumin falls below normal levels. Its severity and the risk of complications are graded by the severity of the protein loss. The risks of some complications begin to rise at levels of proteinuria below those conventionally associated with nephrotic syndrome. The main manifestation, oedema, is characterized by avid sodium retention and managed by sodium restriction and diuretics. A pronounced thrombotic tendency is particularly apparent within the first 6 months of diagnosis and i

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39

Informe regional de SIREVA II, 2016. Organización Panamericana de la Salud, 2019. http://dx.doi.org/10.37774/9789275321850.

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[Prefacio, español]. La red SIREVA (Sistema Regional de Vacunas), conocida en toda la Región, completa sus 25 años de existencia y, en todos esos años, viene prestando un trabajo relevante en la vigilancia de laboratorio de enfermedades bacterianas invasivas, especialmente aquellas causadas por el Streptococcus pneumoniae (neumococo), Haemophilus influenzae (Hi) y la Neisseria meningitidis (meningococo), con reconocido nivel de excelencia. La red también caracteriza los respectivos serotipos/serogrupos y la susceptibilidad a los antimicrobianos de las mencionadas bacterias. Desde el 2005, los

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40

Noris, Marina, and Tim Goodship. The patient with haemolytic uraemic syndrome/thrombotic thrombocytopenic purpura. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0174.

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The patient who presents with microangiopathic haemolytic anaemia, thrombocytopenia, and evidence of acute kidney injury presents a diagnostic and management challenge. Haemolytic uraemic syndrome (HUS) and thrombotic thrombocytopenic purpura (TTP) are two of the conditions that frequently present with this triad. They are characterized by low platelet count with normal or near-normal coagulation tests, anaemia, and signs of intravascular red cell fragmentation on blood films, and high LDH levels.HUS associated with shiga-like toxins produced usually by E.coli (typically O157 strains) may occu

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41

Pery, Xavier. Coloring Book - You Will Get Better - Streptococcus Pneumonia. Independently Published, 2021.

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42

Lupton, Joshua. Hospital Acquired Pneumonia. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0023.

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Pneumonia consists of inflammation of the pulmonary parenchyma, typically resulting from a microbial infection. Hospital-acquired pneumonia (HAP) occurs in (typically elderly) patients in long-term care facilities, with regular IV therapy, with immunosuppression, or with a history of recent treatment at a hospital. It is associated with high mortality. The majority HAP patients present with some constellation of cough, fever, sputum production, and pleuritic chest pain. Patients with chronic obstructive pulmonary disease (COPD) and cystic fibrosis are at increased risk for pneumonia. The Infec

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43

Rodriguez-Iturbe, Bernardo, and Mark Haas. Post-infectious glomerulonephritis. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0076.

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Post-infectious glomerulonephritis (GN) defines an inflammatory lesion involving exclusively or predominantly the glomeruli that is a consequence of an infectious disease. There are numerous bacterial, viral, and fungal infections associated with GN. This chapter acts as an overview of the following chapters that discuss only post-streptococcal GN, immunoglobulin A-dominant GN associated with staphylococcal infections, GN associated with bacterial endocarditis, with infected ventriculoatrial shunts (‘shunt nephritis’), and GN associated with deep-seated infections (osteomyelitis, visceral absc

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