Relevant bibliographies by topics / Streptococcus pneumoniae

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Streptococcus pneumoniae'

Author: Grafiati
Published: 4 June 2021
Last updated: 31 July 2024

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Journal articles on the topic "Streptococcus pneumoniae"

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Park, Hee Kuk, Sang-Jae Lee, Jang Won Yoon, Jong Wook Shin, Hyoung-Shik Shin, Joong-Ki Kook, Soon Chul Myung, and Wonyong Kim. "Identification of the cpsA gene as a specific marker for the discrimination of Streptococcus pneumoniae from viridans group streptococci." Journal of Medical Microbiology 59, no. 10 (October 1, 2010): 1146–52. http://dx.doi.org/10.1099/jmm.0.017798-0.

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Streptococcus pneumoniae, the aetiological agent of pneumonia and non-gonococcal urethritis, shares a high degree of DNA sequence identity with the viridans group of streptococci, particularly Streptococcus mitis and Streptococcus oralis. Although their clinical and pathological manifestations are different, discrimination between S. pneumoniae and its close viridans cocci relatives is still quite difficult. Suppression subtractive hybridization was performed to identify the genomic differences between S. pneumoniae and S. mitis. Thirty-four resulting S. pneumoniae-specific clones were examined by sequence determination and comparative DNA sequence analysis using blast. S. pneumoniae-specific primers were subsequently designed from one of the clonal DNA sequences containing the cps gene (coding for capsular polysaccharide biosynthesis). The primer specificities were evaluated using 49 viridans streptococci including 26 S. pneumoniae, 54 other streptococci, 14 Lactococcus species, 14 Enterococcus species and three Vagococcus species, and compared with the specificities of previously described autolysin (lytA), pneumolysin (ply), Spn9802 and Spn9828 primers. The newly designed cpsA-specific primer set was highly specific to S. pneumoniae and was even better than the existing primers. These findings may help improve the rapid identification and differentiation of S. pneumoniae from closely related members of the viridans group streptococci.
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Kim, Gyu-Lee, Seung-Han Seon, and Dong-Kwon Rhee. "Pneumonia and Streptococcus pneumoniae vaccine." Archives of Pharmacal Research 40, no. 8 (July 22, 2017): 885–93. http://dx.doi.org/10.1007/s12272-017-0933-y.

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Гатагонова, Tamara Gatagonova, Цаллагова, Olga Tsallagova, Болиева, and Laura Bolieva. "Antibiotic resistance of strains of Streptococcus pneumoniae isolated from hospitalized patients with community-acquired pneumonia in the republic of North Ossetia-Alania." Vladikavkaz Medico-Biological Bulletin 20, no. 30 (November 1, 2014): 105–8. http://dx.doi.org/10.12737/11806.

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An increase of antibiotic resistance of bacteria, in particular, Streptococcus pneumoniae, has been registered recently in most developed countries. This necessitates the study of regional characteristics of etiological structure of causative bacterial agents of community-acquired pneumonia and their sensitivity to antibiotics. The aim of the study was to study the spectrum of bacterial pathogens of community-acquired pneumonia and the sensitivity of Streptococcus pneumoniae to antimicrobial agents in hospitalized patients in the Republic of North Ossetia - Alania. Bacteriological examination of sputum with definition of sensitivity of isolated strains of bacteria to antibiotics was performed in 270 patients with community-acquired pneumonia. According to our data, the main causative agent of community-acquired pneumonia in hospitalized patients in the Republic of North Ossetia-Alania is Streptococcus pneumoniae. III generation cephalosporins, respiratory fluoroquinolones, macrolides, showed high activity against Streptococcus pneumoniae isolated from hospitalized patients. Low activity of natural and semi-synthetic penicillins was shown. The obtained results allow optimizing of antimicrobial therapy of community-acquired pneumonia caused by Streptococcus pneumoniae.
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Limelette, Anne, Thomas Guillard, Marie Laure Boubee, Jean Sébastien Petit, Véronique Vernet-Garnier, Antoine Grillon, Olivier Toubas, and Christophe De Champs. "Necrotizing pneumonia due to Streptococcus pneumoniae." Annales de biologie clinique 73, no. 4 (July 2015): 491–94. http://dx.doi.org/10.1684/abc.2015.1064.

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Palmer, D. L. "Laboratory Diagnosis of Streptococcus pneumoniae Pneumonia." Journal of Infectious Diseases 151, no. 2 (February 1, 1985): 378. http://dx.doi.org/10.1093/infdis/151.2.378.

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Bauer, Torsten, Santiago Ewig, María A. Marcos, Gerhard Schultze-Werninghaus, and Antoni Torres. "STREPTOCOCCUS PNEUMONIAE IN COMMUNITY-AQUIRED PNEUMONIA." Medical Clinics of North America 85, no. 6 (November 2001): 1367–79. http://dx.doi.org/10.1016/s0025-7125(05)70385-0.

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Olarte, Liset, and Mary Anne Jackson. "Streptococcus pneumoniae." Pediatrics in Review 42, no. 7 (July 2021): 349–59. http://dx.doi.org/10.1542/pir.2020-0062.

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Eagle, Kim, and David M. Phillips. "Streptococcus pneumoniae." New England Journal of Medicine 329, no. 7 (August 12, 1993): 477. http://dx.doi.org/10.1056/nejm199308123290706.

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Vaughan, David J., Glenn S. Tillotson, and Keith P. Klugman. "Streptococcus pneumoniae." Infectious Diseases in Clinical Practice 15, no. 2 (March 2007): 92–99. http://dx.doi.org/10.1097/01.idc.0000239722.78078.17.

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SHEFF, BARBARA. "Streptococcus pneumoniae." Nursing 29, no. 3 (March 1999): 17. http://dx.doi.org/10.1097/00152193-199903000-00006.

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Dissertations / Theses on the topic "Streptococcus pneumoniae"

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Bazaz, Rohit. "The effect of Streptococcus pneumoniae pneumonia on atherosclerosis." Thesis, University of Sheffield, 2016. http://etheses.whiterose.ac.uk/16897/.

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Koppe, Uwe Moritz Eberhard. "Role of type I interferons in Streptococcus pneumoniae pneumonia." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2012. http://dx.doi.org/10.18452/16532.

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Streptococcus pneumoniae ist die häufigste Ursache für ambulant erworbene Pneumonien weltweit. Daher müssen die Wirts-Pathogen-Interaktionen erforscht werden, um neue Therapiestrategien zu entwickeln. In dieser Studie habe ich 1. den Typ I Interferon (IFN)-stimulierenden Signalweg des angeborenen Immunsystems in Pneumokokken-infizierten Wirtszellen sowie 2. dessen Bedeutung in der Pneumokokkenpneumonie untersucht. Humane und murine Makrophagen, aber nicht alveolare Epithelzellen, produzierten Typ I IFNs nach Infektion mit S. pneumoniae. Dieses war abhängig vom Virulenzfaktor Pneumolysin und erforderte sowohl die Phagozytose der Bakterien als auch die Ansäuerung der Endosomen. Die Induktion der Typ I IFNs wird durch einen zytosolischen Signalweg vermittelt, welcher wahrscheinlich DNA erkennt und sowohl das Adapterprotein STING als auch den Transkriptionsfaktor IRF3 aktiviert. Typ I IFNs, welche von infizierten Makrophagen gebildet wurden, regulierten die Expression von IFN-stimulierten Genen (ISGs) und Chemokinen in Makrophagen und co-kultivierten alveolaren Epithelzellen in vitro und in Mauslungen in vivo. In einem murinen Pneumoniemodell hatten die Typ I IFNs jedoch einen negativen Effekt für den Wirt. Mäuse mit einem Defekt im Typ I IFN-Rezeptor oder mit einem Knockout im Typ I und Typ II IFN-Rezeptor hatten eine signifikant geringere Bakterienlast in der Lunge und eine verminderte Reduktion der Körpertemperatur und des Körpergewichtes als wild-typ Mäuse. Diese Effekte waren nicht durch Änderungen in der Zellrekrutierung oder durch Änderungen der Zytokin-/Chemokinexpression erklärbar. Zusammenfassend lässt sich feststellen, dass Typ I IFNs durch Pneumokokken induziert werden, aber dass sie trotz einiger positiver Effekte auf die Expression von ISGs einen negativen Gesamteffekt in einem murinen Pneumoniemodell aufweisen. Ein detailliertes Verständnis der Typ I IFN-Antwort während der Pneumokokkeninfektion kann die Entwicklung neuer Therapiestrategien unterstützen.
Streptococcus pneumoniae is the leading cause of community-acquired pneumonia world-wide. A detailed understanding of the host-pathogen interactions is required in order to foster the development of new therapeutic strategies. Here, I (I) characterized an innate immune recognition pathway that senses pneumococcal infection and triggers the production of type I interferons (IFNs), and (II) examined the role of type I IFNs in pneumococcal pneumonia in mice. Human and murine macrophages, but not alveolar epithelial cells, produced type I IFNs after infection with S. pneumoniae. This induction was dependent on the virulence factor pneumolysin, the phagocytosis of the bacteria, and the acidification of the endosome. Moreover, it appeared to be mediated by a cytosolic DNA-sensing pathway involving the adaptor molecule STING and the transcription factor IRF3. Type I IFNs produced by S. pneumoniae-infected macrophages positively regulated the expression of IFN-stimulated genes (ISGs) and chemokines in macrophages and co-cultured alveolar epithelial cells in vitro and in mouse lungs in vivo. However, in a murine model of pneumococcal pneumonia, type I IFN signaling was detrimental to the host defense. Mice deficient in the type I IFN signaling or double deficient in type I and type II IFN signaling had a significantly reduced bacterial load in the lung and a diminished reduction of body temperature and body weight compared to wild-type mice. The decreased susceptibility of the knockout mice was unlikely to be attributable to alterations in cell recruitment or cytokine/chemokine production. In conclusion, type I IFNs are induced during pneumococcal infection. However, despite their positive effects on the expression of some ISGs and chemokines, they negatively affect the outcome of pneumococcal pneumonia in an in vivo mouse model. Targeting the type I IFN system could potentially be an effective way in enhancing the immune response in patients with S. pneumoniae pneumonia.
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Bracht, Dagmar. "Proteomanalyse von Streptococcus pneumoniae." [S.l.] : [s.n.], 2005. http://deposit.ddb.de/cgi-bin/dokserv?idn=977729931.

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McNamee, Lynnelle Ann. "Effects of a primary influenza infection on susceptibility to a secondary Streptococcus pneumoniae infection." Diss., Montana State University, 2006. http://etd.lib.montana.edu/etd/2006/mcnamee/McNameeL1206.pdf.

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Koo, Kun, and 古軍. "Vancomycin tolerance in streptococcus pneumoniae." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2002. http://hub.hku.hk/bib/B31970588.

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Wyres, Kelly L. "Genome evolution in Streptococcus pneumoniae." Thesis, University of Oxford, 2012. http://ora.ox.ac.uk/objects/uuid:985b1fc6-c1a9-41b3-a20a-1735329d962b.

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Streptococcus pneumoniae (the pneumococcus) is a bacterial pathogen responsible for >1.6 million annual deaths globally. Pneumococcal penicillin-resistance is conferred by acquisition of ‘altered’ penicillin-binding protein (pbp) genes. The first penicillin-nonsusceptible pneumococci were identified in the late 1960s. Global pneumococcal penicillin-nonsusceptibility rates rapidly increased in the 1980s/90s. Since 2000, protein-conjugate vaccines, targeting 7, 10 or 13 of the ≥94 different pneumococcal capsule types (serotypes), have been introduced in many countries. Following vaccine implementation there has been a decline in vaccine-type pneumococcal disease and an increase in non-vaccine-type disease. These epidemiological changes result from “serotype replacement” and/or “serotype switching”. The former describes the expansion of non-vaccine-type clones in the absence of vaccine-type pneumococci. The latter describes serotype change following recombination at the capsule polysaccharide synthesis (cps) locus. To fully understand how pneumococci respond to vaccine- and antibiotic-induced selective pressures, we must better understand the evolutionary history of this pathogen. This thesis describes the study of a global collection of 426 pneumococci, dated 1937 - 2007. Serotype, genotype and penicillin-susceptibility data were collected. Nucleotide sequences of three pbp genes (for 389 isolates) and whole-genome sequences (for 96 isolates) were also generated. The data demonstrated the long-term persistence of certain clones within pneumococcal populations, and that pbp and large-fragment (>30 kb) cps ± pbp recombination was occurring prior to both widespread antibiotic use and vaccine implementation. The data highlighted the promiscuous nature of the globally-distributed PMEN1 clone and its contribution to the spread of pneumococcal penicillin-resistance. PMEN1 also donated multiple, large regions (1.7 - 32.3 kb) of its genome to at least two un-related clones. Finally, six “Tn916-like” genetic elements, conferring resistance to non-penicillin antibiotics, were newly identified. These included two of the oldest ever described. These results provided a unique insight into the history of pneumococcal evolution and the importance of genetic recombination.
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Rudmann, Emily. "Parsing the Streptococcus pneumoniae virulome." Thesis, Boston College, 2020. http://hdl.handle.net/2345/bc-ir:108795.

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Thesis advisor: Tim van Opijnen
Streptococcus pneumoniae is a prominent gram-positive commensal and opportunistic pathogen which possesses a large pan-genome. Significant strain-to-strain variability in genomic content drives the use of varied pathways to perform similar processes between strains. Considering this variation, we employ a set of 36 strains, representative of 78% of total pan-genome diversity, with which to perform functional studies. We previously determined the set of genes required by 22 of the 36 strains to maintain successful infection in a host, or the virulome. In this work, we sought to parse from the virulome the genes required specifically for nasopharyngeal adhesion, a crucial step in S. pneumoniae colonization and transmission, and often a precursor to invasive disease, as well as gene requirements for subversion of the macrophage. We performed in vitro attachment Tn-seq in the 22 strains to D562 human nasopharyngeal epithelial cells, identifying thirteen factors that exhibit requirements for adhesion, and preliminarily validated a proposed universal requirement for survival of the macrophage by a killing assay using J774A.1 murine migratory macrophages
Thesis (BS) — Boston College, 2020
Submitted to: Boston College. College of Arts and Sciences
Discipline: A&S Honors
Discipline: Biology
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Koo, Kun. "Vancomycin tolerance in streptococcus pneumoniae." Hong Kong : University of Hong Kong, 2002. http://sunzi.lib.hku.hk/hkuto/record.jsp?B25139216.

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Alghofaili, Fayez Abdullah. "Streptococcus pneumoniae-stress hormone interactions." Thesis, University of Leicester, 2018. http://hdl.handle.net/2381/41267.

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Streptococcus pneumoniae is one of the most important bacterial pathogens of humans causing a wide range of mild to life-threating diseases. It is also a commensal microorganism in the nasopharynx of up to 60% of people. Fundamental aspects of its ability for transition from colonisation to an infectious state as well as how bacterial-host interactions influence this process are largely unknown. In the field of microbial endocrinology, it has been well established in mainly Gram-negative bacteria that stress hormones such as norepinephrine epinephrine and dopamine play an essential role in determining the outcome of bacterial infections. This study successfully established the conditions to investigate S. pneumoniae-stress hormone interactions using modified serum-SAPI media. 13 mutants lacking two-component regulatory system and 4 two-component system fusion reporter strains were created, and examined for their role in S. pneumoniae-stress hormone interactions. This study demonstrated that S. pneumoniae is stress hormone responsive and has mechanisms to recognise and process host stress hormones by a transferrin-iron delivery mechanism, which evidence suggests might be mediated via the TCS09 system since hormone-induced growth and radiolabelled norepinephrine and Fe uptake were reduced in a ΔTCS09 mutant. In addition, the pneumococcal response to stress hormone exposure resulted in a change in cell-cell association from chains into diplococci and cell morphology by reducing cell size and the capsule. Furthermore, the pneumococcal exposure to norepinephrine also increased biofilm formation and significantly altered metabolism. The analysis of in vivo experiments indicated that a stress hormone encounter might trigger translocation from the nasopharynx into the lungs, which may enhance S. pneumoniae in its transition from commensal to pathogen. Therefore, the pneumococcal ability to respond to host stress signals may be key to its capacity to cause life-threatening pneumonia, septicaemia and meningitis.
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Abdeldaim, Guma M. K. "PCR detection of Streptococcus pneumoniae and Haemophilus influenzae in pneumonia patients." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl.[distributör], 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-107931.

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Books on the topic "Streptococcus pneumoniae"

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Iovino, Federico, ed. Streptococcus pneumoniae. New York, NY: Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9199-0.

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Brenwald, Nigel Peter. Characterisation of a multidrug efflux system in Streptococcus pneumoniae. Birmingham: University of Birmingham, 2000.

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Nuorti, J. Pekka. Prevention of pneumococcal disease among infants and children: Use of 13-valent pneumococcal conjugate vaccine and 23-valent pneumococcal polysaccharide vaccine : recommendations of the Advisory Committee on Immunization Practices (ACIP). Atlanta, GA: Dept. of Health and Human Services, Centers for Disease Control and Prevention, 2010.

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Doherty, Neil Christopher. A molecular analysis of hyaluronate lyase production in Streptococcus pneumoniae. [s.l.]: typescript, 2000.

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Boost, Maureen Valerie. Carriage and antibiotic resistance of Streptococcus pneumoniae in Hong Kong. [S.l: The Author], 2004.

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Kanclerski, Krzysztof. Streptococcus pneumoniae haemolysin (pneumolysin): Production, purification and use as antigen for diagnosis of pneumococcal pneumonia. [s.l.]: [s.n.], 1987.

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I, Tuomanen Elaine, ed. The pneumococcus. Washington, DC: ASM Press, 2004.

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Kragsbjerg, Peter. Cytokines in infection: Clinical and experimental studies with special reference to Streptococcus pneumoniae. Uppsala: Uppsala universitet, 1996.

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Johnston, Nicole J. Prevalence and characterization of the mechanisms of macrolide, lincosamide, and streptogramin resistance among isolates of Streptococcus pneumoniae and viridans streptococci. Ottawa: National Library of Canada, 1999.

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File, Thomas. New insights in the treatment of severe infections in the multiple-drug resistant situation: Proceedings of a satellite symposium to the 11th International Congress on Infectious Diseases, Cancun, Mexico, March 5, 2004. Basel, Switzerland: Karger, 2004.

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Book chapters on the topic "Streptococcus pneumoniae"

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Kashani, John, Richard D. Shih, Thomas H. Cogbill, David H. Jang, Lewis S. Nelson, Mitchell M. Levy, Margaret M. Parker, et al. "Streptococcus pneumoniae." In Encyclopedia of Intensive Care Medicine, 2146–51. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_165.

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Nolte, Oliver. "Streptococcus pneumoniae." In Lexikon der Infektionskrankheiten des Menschen, 779–83. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-39026-8_1051.

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Roy, Bronwen, and Marcel Leroi. "Streptococcus pneumoniae." In PCR for Clinical Microbiology, 201–3. Dordrecht: Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-90-481-9039-3_26.

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Tuomanen, Elaine. "Streptococcus pneumoniae." In The Prokaryotes, 149–62. New York, NY: Springer US, 2006. http://dx.doi.org/10.1007/0-387-30744-3_4.

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Balakrishnan, Indran. "Streptococcus pneumoniae." In Principles and Practice of Clinical Bacteriology, 41–57. Chichester, UK: John Wiley & Sons, Ltd, 2006. http://dx.doi.org/10.1002/9780470017968.ch3.

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Sutcliffe, Joyce, and Marilyn C. Roberts. "Streptococcus pneumoniae." In Frontiers in Antimicrobial Resistance, 314–29. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555817572.ch23.

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Neves, Felipe P. G., and Tatiana C. A. Pinto. "Streptococcus pneumoniae." In Molecular Typing in Bacterial Infections, Volume I, 139–52. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-74018-4_6.

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Lydyard, Peter M., Michael F. Cole, John Holton, William L. Irving, Nina Porakishvili, Pradhib Venkatesan, and Katherine N. Ward. "Streptococcus pneumoniae." In Case Studies in Infectious Disease, 321–27. 2nd ed. Boca Raton: CRC Press, 2023. http://dx.doi.org/10.1201/9781003155447-34.

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Gasc, Anne-Marie, and Sol H. Goodgal. "Streptococcus pneumoniae R6." In Bacterial Genomes, 755–57. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-6369-3_81.

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Paton, James C., and David E. Briles. "Streptococcus pneumoniae Vaccines." In New Bacterial Vaccines, 294–310. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0053-7_19.

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Conference papers on the topic "Streptococcus pneumoniae"

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Qadri, A. N., R. Rozzi, C. Burke, N. Jibawi, and R. Sundaram. "Pyogenic Ventriculitis Secondary to Streptococcus Pneumoniae Pneumonia." In American Thoracic Society 2023 International Conference, May 19-24, 2023 - Washington, DC. American Thoracic Society, 2023. http://dx.doi.org/10.1164/ajrccm-conference.2023.207.1_meetingabstracts.a3548.

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Zhang, Y., L. K. Sharma, A. J. Losier, E. Ifedigbo, M. Sauler, I. S. Bazan, C. Dela Cruz, and P. Lee. "Role of PINK1 Mediated Mitophagy During Streptococcus Pneumoniae Pneumonia." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a7230.

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Pinzon Escobar, C., M. Mody, and S. G. Treat. "Purpura Fulminans Secondary to Streptococcus Pneumoniae." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6563.

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Basavaraj, Ashwin, Jose Gomez-Marquez, David Steiger, and Ezra Dweck. "Streptococcus Pneumoniae Bacteremia Following Flexible Bronchoscopy." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a2974.

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Doern, Gary, Sandra Richter, Kristopher Heilmann, Cassie Dohrn, Fathollah Riahi, and Daniel Diekema. "Tracking Resistance In Streptococcus Pneumoniae (TRISP)." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a6083.

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Sanz, F., L. A. Ruiz Iturriaga, M. M. García Clemente, P. P. España Yandiola, L. Serrano Fernández, A. Uranga Echeverria, J. Herrero Huertas, E. Fernández Fabrellas, and PneumoCuore Group. "Streptococcus Pneumoniae Serotype Determine Cardiac Complications During Invasive Pneumococcal Pneumonia." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4228.

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Losier, A. J., L. K. Sharma, Y. Zhang, W. Liu, P. Lee, and C. Dela Cruz. "Streptococcus Pneumoniae Pneumonia Induces Mitochondrial Biogenesis and Antioxidant Responses in Mice." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5595.

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Kjærgaard, Rikke Schmidt, and Ebbe Sloth Andersen. "Seeing Streptococcus pneumoniae, a Common Killer Bacteria." In Electronic Visualisation and the Arts (EVA 2014). BCS Learning & Development, 2014. http://dx.doi.org/10.14236/ewic/eva2014.75.

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Bushueva, T. V., N. A. Roslaya, and E. P. Karpova. "IMMUNOLOGICAL FACTORS OF STREPTOCOCCUS PNEUMONIAE PERSISTENCE IN WORKERS OF COPPER SMELTER." In The 17th «OCCUPATION and HEALTH» Russian National Congress with International Participation (OHRNC-2023). FSBSI «IRIOH», 2023. http://dx.doi.org/10.31089/978-5-6042929-1-4-2023-1-96-100.

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In article we touch upon the problem of immunological factors of persistence of Streptococcus pneumoniae in employees of the copper electrolytic refining enterprise. Materials and methods. A survey of 100 employees of the enterprise for the electrolytic refining of copper was carried out. The main group — 50 smelters of the anode section of the copper smelter, the control group — 50 engineering and technical workers. Smears were taken from the posterior pharyngeal wall to detect S. pneumoniae DNA. Immunological examination included the determination of cellular immunity by flow cytometry, humoral immunity with the determination of immunoglobulins by ELISA. Secretory immunoglobulin A (SigA) was determined in the oral fluid. Results and conclusions. S. pneumoniae was more often found in workers of the main group (p<0.05). Smoking tobacco products increases the chance of developing S. pneumoniae carriage. In workers of the main group, suppression of humoral factors of immunity was revealed; with S. pneumoniae+, the relative number of T‑helpers is reduced and the bactericidal activity of neutrophils is enhanced.
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Rädler, Joachim O., Christoph Polzer, Stefan Ness, Mojtaba Mohseni, Markus Hilleringmann, and Thomas Hellerer. "Nanometer-scale colocalization microscopy of Streptococcus pneumoniae filaments." In Multiphoton Microscopy in the Biomedical Sciences XIX, edited by Ammasi Periasamy, Peter T. So, and Karsten König. SPIE, 2019. http://dx.doi.org/10.1117/12.2509927.

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Reports on the topic "Streptococcus pneumoniae"

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Kindy, Mark S. ENU Mutagenic Screen for Susceptibility and Resistance to Streptococcus Pneumoniae. Fort Belvoir, VA: Defense Technical Information Center, November 2005. http://dx.doi.org/10.21236/ada441504.

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Alexandrova, Alexandra, Lena Setchanova, Daniela Pencheva, and Ivan Mitov. Molecular Serotyping of Serogroup 6 Streptococcus pneumoniae Isolates Has Shown Emergence of Serotype 6C After the Implementation of 10‑valent Pneumococcal Conjugate Vaccine (PhiD‑CV) in Bulgaria. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, August 2019. http://dx.doi.org/10.7546/crabs.2019.08.14.

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Crum, N. F., C. P. Barrozo, F. A. Chapman, M. A. Ryan, and K. Russell. An Outbreak of Conjunctivitis Due to a Novel Unencapsulated Streptococcus pneumonia Among Military Trainees. Fort Belvoir, VA: Defense Technical Information Center, October 2004. http://dx.doi.org/10.21236/ada433371.

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