Academic literature on the topic 'Streptococcus mutans'
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Journal articles on the topic "Streptococcus mutans"
Grönroos, L., M. Saarela, J. Mättö, U. Tanner-Salo, A. Vuorela, and S. Alaluusua. "Mutacin Production by Streptococcus mutans May Promote Transmission of Bacteria from Mother to Child." Infection and Immunity 66, no. 6 (June 1, 1998): 2595–600. http://dx.doi.org/10.1128/iai.66.6.2595-2600.1998.
Full textKamiya, Regianne Umeko, Tiago Taiete, and Reginaldo Bruno Gonçalves. "Mutacins of Streptococcus mutans." Brazilian Journal of Microbiology 42, no. 4 (December 2011): 1248–58. http://dx.doi.org/10.1590/s1517-83822011000400001.
Full textAl-Ahmad, Ali, Thorsten Mathias Auschill, Gabriele Braun, Elmar Hellwig, and Nicole Birgit Arweiler. "Overestimation of Streptococcus mutans prevalence by nested PCR detection of the 16S rRNA gene." Journal of Medical Microbiology 55, no. 1 (January 1, 2006): 109–13. http://dx.doi.org/10.1099/jmm.0.46280-0.
Full textPetersen, F. C., S. Assev, H. C. van der Mei, H. J. Busscher, and A. A. Scheie. "Functional Variation of the Antigen I/II Surface Protein in Streptococcus mutans and Streptococcus intermedius." Infection and Immunity 70, no. 1 (January 2002): 249–56. http://dx.doi.org/10.1128/iai.70.1.249-256.2002.
Full textCheng, X., S. Redanz, P. Treerat, H. Qin, D. Choi, X. Zhou, X. Xu, J. Merritt, and J. Kreth. "Magnesium-Dependent Promotion of H2O2 Production Increases Ecological Competitiveness of Oral Commensal Streptococci." Journal of Dental Research 99, no. 7 (March 20, 2020): 847–54. http://dx.doi.org/10.1177/0022034520912181.
Full textKreth, Jens, Yongshu Zhang, and Mark C. Herzberg. "Streptococcal Antagonism in Oral Biofilms: Streptococcus sanguinis and Streptococcus gordonii Interference with Streptococcus mutans." Journal of Bacteriology 190, no. 13 (April 25, 2008): 4632–40. http://dx.doi.org/10.1128/jb.00276-08.
Full textChia, Jean-San, Huei-Ting Lien, Po-Ren Hsueh, Pei-Min Chen, Andy Sun, and Jen-Yang Chen. "Induction of Cytokines by Glucosyltransferases of Streptococcus mutans." Clinical and Vaccine Immunology 9, no. 4 (July 2002): 892–97. http://dx.doi.org/10.1128/cdli.9.4.892-897.2002.
Full textWang, Bing-Yan, and Howard K. Kuramitsu. "Interactions between Oral Bacteria: Inhibition of Streptococcus mutans Bacteriocin Production by Streptococcus gordonii." Applied and Environmental Microbiology 71, no. 1 (January 2005): 354–62. http://dx.doi.org/10.1128/aem.71.1.354-362.2005.
Full textTao, L., and J. M. Tanzer. "Novel Sucrose-dependent Adhesion Co-factors in Streptococcus mutans." Journal of Dental Research 81, no. 7 (July 2002): 505–10. http://dx.doi.org/10.1177/154405910208100715.
Full textNilsson, Martin, Michael Givskov, Svante Twetman, and Tim Tolker-Nielsen. "Inactivation of the pgmA Gene in Streptococcus mutans Significantly Decreases Biofilm-Associated Antimicrobial Tolerance." Microorganisms 7, no. 9 (September 3, 2019): 310. http://dx.doi.org/10.3390/microorganisms7090310.
Full textDissertations / Theses on the topic "Streptococcus mutans"
Thevenot, Tracy Lynn. "Aspects of sugar transport via the phosphoenolpyruvate sugar phosphotransferase system of streptococcus mutans /." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/nq23673.pdf.
Full textMonteiro-Oliveira, Marcela Pinto 1982. "Estudo da ação antimicrobiana da terapia fotodinamica sobre lesões de carie produzidas in vitro na dentina de dentes bovinos." [s.n.], 2010. http://repositorio.unicamp.br/jspui/handle/REPOSIP/288089.
Full textDissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba
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Resumo: Durante o processo conhecido como terapia fotodinâmica, a aplicação de fotossensibilizadores associados a uma fonte de luz de comprimento de onda complementar, gera produtos que podem danificar componentes essenciais das células, e causar a morte celular. Dentro desse contexto, a aplicação dessa terapia sobre microrganismos presentes em lesões de cárie é de grande valia, uma vez que poderá reduzir a quantidade de tecido dental a ser removido no tratamento da cárie, diminuir as chances de progressão da doença bem como os riscos de acometimento pulpar do elemento dentário. Assim, o objetivo deste estudo in vitro foi determinar parâmetros para o uso de um diodo emissor de luz (LED) associado ao corante azul de orto toluidina (TBO) na redução da contagem de Streptococcus mutans presentes em lesões de cárie dentinária. Para isto, 72 espécimes de dentina coronária de dentes bovinos foram imersos em cultura contendo Streptococcus mutans para produzir lesões de cárie. Tais espécimes foram divididos aleatoriamente em 6 grupos (n=12): Controle (exposição a NaCl a 0,9% por 5 min); TBO (exposição ao TBO a 0,01% por 5 min); LEDA (exposição ao LED por 4,2 min); LEDB (exposição ao LED por 6,5 min); PDTA (exposição ao corante associado ao LED por 4,2 min) e PDTB (exposição ao corante associado ao LED por 6,5 min). As densidades de energia utilizadas para os tempos de 4,2 e 6,5 min, foram de 166 e 249 J/cm2, respectivamente. Antes e após os tratamentos, amostras de tecido dentinário cariado foram coletadas e analisadas microbiologicamente, por meio da contagem das unidades formadoras de colônia (UFC) de S. mutans. A profundidade das lesões de cárie produzidas pelo modelo microbiológico utilizado foi determinada por meio da microscopia de luz polarizada. Foram utilizados os testes ANOVA/Tukey para comparar os valores de log redução dos grupos (a=5%). Observou-se redução significativa de S. mutans nos grupos em que aplicou-se TBO associado ao LED, com as duas densidades de energia utilizadas. Entretanto, nenhuma diferença significativa foi encontrada para os diferentes tempos de irradiação. Concluiu-se que os parâmetros utilizados no presente estudo, para o emprego do LED associado ao TBO, foram efetivos em reduzir a contagem de S. mutans presentes em lesões de cárie dentinária.
Abstract: Photodynamic therapy (PDT) is a technique that consists in the activation of certain photosensitizers by light in the presence of tissue oxygen, resulting in the production of reactive radicals capable of inducing cell death. In this context, this therapy may become a suitable approach to disinfect the dentin tissue during the caries treatment, and reduce the tissue removal, minimizing the probability of caries progression and pulp involvement. This randomized in vitro study determined parameters for using a light-emitting diode (LED) with toluidine blue O (TBO) for reduction of Streptococcus mutans counts inside dentin caries. Seventy two bovine coronary dentin slabs were immersed in Streptococcus mutans culture for demineralization production. Dentin slabs were allocated to 6 groups (n=12) as follows: Control (treated with 0.9% NaCl solution for 5 min); TBO (treated with 0.1 mg/ml TBO for 5 min); LEDA (submitted to irradiation for 4.2 min); LEDB (submitted to irradiation for 6.5 min); PDTA (treated with TBO plus irradiation for 4.2 min) and PDTB (treated with TBO plus irradiation for 6.5 min). The energy densities used for 4.2 and 6.5 min correspond at 166 and 249 J/cm2, respectively. Before and after treatments, dentin samples were analyzed with regard to S. mutans counts. The caries lesion depth produced by the microbiological model was analyzed by polarized light microscopy. ANOVA/Tukey tests were utilized to compare log reductions among groups (a=5%). Bacterial reduction was observed when dentin was exposed to both TBO and LED at both irradiation times. However, no difference in S. mutans reduction was found between the two energy densities. Concluding, although the use of LED combined with TBO was effective in reducing the Streptococcus mutans counts in carious dentin, this effect may not have clinical significance.
Mestrado
Odontopediatria
Mestre em Odontologia
Vizoto, Natália Leal 1982. "Avaliação da função biológica do sistema de dois componentes SptRS de Streptococcus mutans." [s.n.], 2015. http://repositorio.unicamp.br/jspui/handle/REPOSIP/288675.
Full textTese (doutorado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba
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Resumo: Streptococcus mutans é uma espécie bacteriana comum da microbiota bucal de seres humanos envolvida na patogênese da cárie dentária e em endocardites infecciosas promovidas por bacteremias de origem bucal. Para ser transmitido e ocupar seus nichos ecológicos, S. mutans precisa persistir na saliva e se adaptar fisiologicamente a cada fase da colonização, um processo que provavelmente envolve diversas alterações do seu transcriptoma. Para isto, S. mutans utiliza sistemas reguladores de transcrição de dois componentes (SDC). O SDC SptRS foi identificado através de análises in silico do genoma da cepa S. mutans UA159, como ortólogo do sistema SptSR (Spt de Saliva persistence) de Streptococcus pyogenes. O objetivo deste trabalho foi investigar a participação do sistema SptRS de S. mutans em fenótipos importantes para a colonização bucal. Para isto, mutantes knockout dos genes sptR e sptS, SMU.927 e SMU.928 respectivamente, foram construídos a partir da cepa UA159 (UAsptR- e UAsptS-) e comparados com a cepa parental em análises de morfologia, crescimento planctônico sob diferentes condições nutricionais, persistência em saliva humana, formação de biofilme e autólise a 44oC. Além disto, genes do regulon de SptRS foram pesquisados através de ensaios da Imunoprecipitacão de Cromatina seguido de sequenciamento (ChIP-seq), RT-PCR quantitativo (RT-PCRq) e de Ensaios de Retardamento da Mobilidade Eletroforética (EMSA) com proteína recombinante SptRr de S. mutans. Os mutantes sptR e sptS mostraram crescimento planctônico lento em meios de cultura RPMI e em MQD comparados à cepa parental, além de atividade autolítica reduzida em 22,4 e 53,13%, respectivamente. Não foram observadas, entretanto, alterações significativas na morfogênese, formação de biofilmes in vitro, nem na persistência em saliva humana. Os dados de ChIP-seq e RT-qPCR indicaram que SptRS regula genes envolvidos na resposta de estringência (SMU.926), repressão catabólica (ccpA), metabolismo de múltiplos açúcares (SMU.78, SMU.137, SMU.542, SMU.1734), sistemas fosfoenolpiruvato-fosfotransferase (PTS) (SMU.2047, SMU.114, SMU.115) sistemas de transporte do tipo ABC (SMU.182, SMU.880, SMU.905, SMU.1035, SMU.1095, SMU.1178c, SMU.1939) e biogênese de parede celular (SMU.1091, SMU.2147, SMU.609, SMU.1434c, SMU.22). SptR funcionou como um regulador negativo em 86% (37/43) dos genes testados. Análises de RT-qPCR e EMSA indicaram ainda que SptR regula diretamente o regulador de transcrição CovR (SMU.1924), envolvido na repressão de genes de virulência e formação de biofilmes. Este estudo fornece evidências de que SptRS regula diversas funções de S. mutans importantes para a sobrevivência em condições nutricionalmente escassos, aparentemente coordenando o metabolismo com o crescimento bacteriano e com a expressão de genes de virulência
Abstract: Streptococcus mutans is a common bacterial species of the bucal microbiota of humans involved in the pathogenesis of dental caries and infectious endocarditis promoted by bacteremia of bucal origin. To be transmitted and occupy their ecological niches, S. mutans need to persist in saliva and adapt physiologically to each phase of colonization, a process that probably involves several changes in its transcriptome. To this end, S. mutans uses transcriptional regulatory systems called Two Component System (TCS). The TCS SptRS was identified in an in silico analysis of the genome of S. mutans strain UA159, as an orthologue of the SptRS system (Spt of Saliva persistence) of Streptococcus pyogenes. The aim of this study was to investigate the role of the TCS SptRS in functional traits important for S. mutans to colonize its bucal niches. Thus, knockout mutants of sptR and sptS (SMU.927 and SMU.928, respectively) were obtained in strain UA159 (UAsptR-, UAsptS-) and compared to parental strain regarding morphology, planktonic growth under different nutritional conditions and persistence in human saliva, biofilm formation and autolysis at 44oC. In addition, genes of SptRS regulon were analised by Chromatin Immunoprecipitation followed the sequencing (ChIP-seq), quantitative RT-PCR (RT- qPCR) and Electrophoretic Mobility Shift Assays (EMSA) with S. mutans SptR recombinant protein. Inactivation of sptR/S promotes slow planktonic growth in RPMI and CDM, culture media 22.4 to 53.13% reductions in autolysis respectively, but does not significantly affect morphogenesis. However, mutants do not show significant alterations in biofilm formation or in persistence in human saliva. ChIP-seq and RT-qPCR analyses showed that SptRS regulates genes for stringent response (SMU.926), metabolism of multiple sugars (SMU.78, SMU.137, SMU.542, SMU.1734), catabolite repression (SMU.1591, ccpA), phosphoenolpyruvate phosphotransferase systems (PTS), ABC transport systems (SMU.182, SMU.880, SMU.905, SMU.1035, SMU.1095, SMU.1178c, SMU.1939) and cell wall biogenesis (SMU.22, SMU.609, SMU.1091, SMU.1434c, SMU.2147) SptR worked as a negative regulator of 86% (37/43) of the tested genes. RT-qPCR and EMSA analyses further showed that SptR directly represses expression of the transcriptional regulator CovR (SMU.1924), which is a repressor of genes involved in biofilm formation and virulence. This study provides evidence that SptRS regulates several functions important for S. mutans survival under poor nutritional conditions, apparently coordinating metabolism with bacterial growth and expression of virulence genes
Doutorado
Microbiologia e Imunologia
Doutora em Biologia Buco-Dental
Lewis, Christopher Roger. "Chromosomal deletions in Streptococcus mutans." Thesis, University of Newcastle Upon Tyne, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.297569.
Full textHale, John D. F., and n/a. "Small bacteriocins produced by Streptococcus mutans and Streptococcus sanguis." University of Otago. Department of Microbiology & Immunology, 2006. http://adt.otago.ac.nz./public/adt-NZDU20060905.144149.
Full textGalvão, Lívia Câmara de Carvalho 1985. "Avaliação da atividade antimicrobiana de óleos essenciais contra microrganismos do grupo mutans e determinação da atividade antiproliferativa." [s.n.], 2012. http://repositorio.unicamp.br/jspui/handle/REPOSIP/288527.
Full textDissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba
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Resumo: O objetivo desse trabalho foi avaliar a atividade antimicrobiana, in vitro, de óleos essenciais e frações dos óleos de melhor atividade, contra microrganismos do grupo mutans em estado planctônico. Além disso, os biofilmes de Streptococcus mutans foram submetidos às frações ativas e os óleos de melhor atividade e frações ativas foram avaliados quanto à sua citotoxicidade e caracterizados quimicamente. Para isso, vinte óleos essenciais (OE) foram obtidos por hidrodestilação a partir de plantas pertencentes ao banco de germoplasmas da Coleção de Plantas Medicinais e Aromáticas (CPMA/CPQBA/UNICAMP). Estes OE foram avaliados quanto à sua atividade antimicrobiana por meio dos ensaios: concentrações inibitória (CIM) e bactericida mínima (CBM) contra Streptococcus mutans UA159. Controles positivo (clorexidina 0,12 %) e negativo (propilenoglicol 6,12 % e 25 %) também foram testados...Observação: O resumo, na íntegra, poderá ser visualizado no texto completo da tese digital
Abstract: The aim of this study was to evaluate the in vitro antimicrobial activity of essential oils (EO) and fractions from highest activity EO against planktonic cells of mutans streptococci. Besides, the biofilms formed by this microorganism were submitted to active fractions and the higher activity EO and active fractions were evaluated regarding their citotoxicity and chemically characterized. For this, twenty essentinal oils were obtained from plants of the "Collectio of Medicinal and Aromatic Plants" (CPMA, CPQBA/UNICAMP), germplasm bank by hydrodistillation. These EO were evaluated by antimicrobial assays: minimum inhibitory (MIC) and bactericidal (MBC) concentrations against Streptococcus mutans UA159. Positive (chlorhexidine 0.12%) and negative (propylene glycol 6.12 % and 25%) controls were also tested...Note: The complete abstract is available with the full electronic document
Mestrado
Farmacologia, Anestesiologia e Terapeutica
Mestre em Odontologia
Carneiro, Haline de Lima. "Avaliação de propriedades de superfície da liga Ti-35Nb-7Zr-5Ta submetida à anodização e seus efeitos n adesão bacteriana /." Araraquara, 2014. http://hdl.handle.net/11449/110821.
Full textCo-orientador: Laiza Maria Grassi Fais
Banca: Valfrido Antonio Pereira Filho
Banca: Valentim Adelino Ricardo Barão
Resumo: Este estudo comparou as propriedades de superfície e a adesão Streptococcus mutans na liga Ti-35Nb-7Zr-5Ta e no titânio comercialmente puro (Ti cp) antes e após a anodização. Foram utilizados discos (Ø8mmx2mm; N = 40) divididos em 2 grupos: T (Ti cp), TNZT (Ti-35Nb- 7Zr-5Ta), e subdivididos conforme a realização (A+) ou não (A-, controle) da anodização eletroquímica (300 V, 1 min) em β-glicerofostato de sódio + acetato de cálcio. As propriedades avaliadas foram: topografia de superfície e identificação qualitativa dos elementos químicos (microscópio eletrônico de varredura-MEV/EDS), energia livre de superfície (ELS, mensurada em goniômetro) e rugosidade média (Ra, determinada em rugosímetro). Para avaliação da adesão bacteriana, os discos foram contaminados com Streptococcus mutans (NTCC 25175) para determinação de UFC/mL e do padrão de adesão (MEV). Os valores de Ra e ELS de cada grupo foram comparados (A- vs. A+) por meio do teste Kruskal-Wallis associado ao teste de Dun (α = 0,05). Os valores de Ra (μm) e ELS (mN/m), respectivamente, foram: A-- T=0,97/44,24; TNZT=0,17/36,68; A+ - T=1,21/56,88; TNZT=0,53/53,64, com aumento significante de ambas propriedades (p < 0,05) após a anodização. A análise em MEV/EDS indicou a formação de uma camada multiporosa, com deposição de íons Ca e P nos subgrupos A+. Após a anodização houve aumento na adesão do patógeno apenas na liga TNZT. Conclui-se que a anodização do Ti cp e da liga TNZT alteram as propriedades de superfície com potencial para melhorias na osseointegração, contudo há aumento na adesão de S. mutans na liga TNZT.
Abstract: The aim of this study was to compare the surface properties and the adhesion of Streptococcus mutans on Ti-35Nb-7Zr-5Ta and commercially pure titanium (cp Ti) before and after the anodization. Discs (Ø8mmx2mm, N=40) were divided into 2 groups: T (cp Ti), TNZT (Ti-35Nb-7Zr- 5Ta), and subdivided in untreated (A- , control) or anodic treated (A+) in β- glicerofostato + calcium acetate (300 V, 1min). The evaluated surface properties were: surface topography and qualitative identification of chemical elements (in scanning electron microscope -SEM/EDS), surface free energy (SFE, measured with a goniometer), and the average roughness (Radetermined in profimoleter). The discs were contaminated with Streptococcus mutans (NTCC 25175) for determination of CFU/mL; the surfaces with adhered viable cell were also analyzed with SEM. The values of Ra and ELS were compared (A- vs . A+) by means of Kruskal-Wallis associated Dun test (α= 0.05). The median Ra (μm) and ELS (mN/m), respectively, were: A- T=0.97/44.24; TNZT=0.17/36.68; A+ T=1.21/56.88; TNZT=0.53/ 53.64. All groups showed significantly higher values of Ra and ELS (p < 0.05) after anodizing. The analysis in SEM/EDS indicated the formation of a multiporous layer with deposition of Ca and P ions. Only anodic treated TNZT exhibited an increase in adhesion of S. mutans. It was concluded that the anodic tretament of Ti cp and TNZT change the surface properties with potential improvements for osseointegration, despite the increase in the adhesion of S. mutans on TNZT.
Mestre
Neilands, Jessica. "Acid tolerance of Streptococcus mutans biofilms /." [Malmö, Sweden] : Malmö University, Faculty of Odontology, Department of Oral Biology, 2007. http://www.mah.se/muep.
Full textAlshammari, Abdulaziz. "IN VITRO EFFECT OF STATINS ON STREPTOCOCCUS MUTANS, STREPTOCOCCUS SANGUIS, AND STREPTOCOCCUS SALVARIUS." Master's thesis, Temple University Libraries, 2016. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/368075.
Full textM.S.
Objectives: Cardiovascular disease (CVD), including heart attack, angina, and stroke, is ranked as the number one cause of mortality world wide. High blood cholesterol is linked to CVD and is an important risk factor. Statins – cholesterol lowering drugs- are first choice drugs for reducing the chance of suffering a CVD event. In the USA alone, approximately 32 million individuals take statins. Although randomized control trials of statins have demonstrated their efficacy in preventing CVD, much less information has been reported on their unintended effects. Although not thought of traditionally as antimicrobials, statins have been shown to have antimicrobial effects in vitro. The statins belong to a family of drugs that lower cholesterol levels by inhibiting 3-hydroxy-3-methylglutaryl-CoA reductase, a rate limiting enzyme in the human mevalonate pathway of which cholesterol in the biosynthetic end product. The mevalonate pathway is an important cellular metabolic pathway present in many bacteria. Hence, the aim of this study was to assess the in vitro efficacy of statins against selected strains of oral streptococci, as determined by the minimum inhibitory concentration. A second related objective is to assess the in vitro effect of statins on single species biofilm formation , as determined by binding of the same streptococci to hydroxyapatite pegs. Methods: The effect of statins on S. mutans, S. sanguis, and S. salivarius was determined by finding the minimum inhibitory concentration (MIC) by broth dilution assays. Simvastatin, pravastatin atorvastatin, and rousuvastatin were used in this study. The minimum inhibitory concentration was considered to be the lowest concentration of statin that prevented bacterial growth, i.e. a clear test tube. Experiments were repeated twice for each bacterial species. The effect of simvastatin, atorvastatin, and pravastatin on the ability of S. mutans and S. sanguis to form single species biofilm was assayed using sterile microplates and the MBEC Biofilm Inoculator (Innovatech). Results: Two trials indicated that the MIC of simvastatin against the selected oral bacteria was determined to be 15.6 μg/ml for S. mutans and S. sanguis, and 7.8 μg/ml for S. salivarius. The MIC of rosuvastatin and atorvastatin was determined to be 100 μg/ml against all three streptococci, whereas the MIC of pravastatin was even higher (200 μg/ml) against all three streptococci. Likewise, two trials indicated that statins decreased single species biofilm formation by S. mutans and S. sanguis. For simvastatin, biofilm formation was decreased by concentrations eight fold below the MIC . The results were substantiated by spectrophotometric assay . For atorvastatin and pravastatin, biofilm formation was decreased by concentrations 3-4 fold below the MIC. Conclusions: These experiments demonstrate the in vitro antimicrobial effect of statins on S. mutans, S.sanguis, and S. salivarius. The data indicate that the statins inhibit growth of the test organisms with MIC’s ranging from 7.8-200 μg/ml. Simvastatin has in vitro efficacy against the specific strains of bacteria used in this study at concentrations slightly less than the observed MIC’s of 15.6-7.8 μg/ml . The MIC’s for atorvastatin, pravastatin, and rosuvastatin are much higher than simvastatin, in the range of 100-200 μg/ml . The effects of statins on biofilm parallels the effect on growth of the bacteria.
Temple University--Theses
Branting, Christina. "Studies on S̲t̲r̲e̲p̲t̲o̲c̲o̲c̲c̲u̲s̲ m̲u̲t̲a̲n̲s̲ glucans with special reference to cell adhesion." Stockholm : Kongl. Carolinska Medico Chirurgiska Institutet, 1988. http://catalog.hathitrust.org/api/volumes/oclc/18171129.html.
Full textBooks on the topic "Streptococcus mutans"
Harmandayan, Rita. Transformation of Streptococcus mutans by electroporation. [Toronto: Faculty of Dentistry, University of Toronto], 1990.
Find full textShigeyuki, Hamada, ed. Molecular microbiology and immunobiology of Streptococcus Mutans ; proceedings of an International Conference on "Cellular, Molecular, and Clinical Aspects of Streptococcus Mutans" held in Birmingham, Alabama, USA, on September 18-20, 1985. Amsterdam: Elsevier Science Publishers, 1986.
Find full textHanna, Michael N. Molecular mechanisms involved in the acid tolerance response of Streptococcus mutans. Ottawa: National Library of Canada, 2001.
Find full textTwetman, Svante. Antibacterial effects of human salivary lysozyme with special reference to Streptococcus mutans. Stockholm: Departments of Pedodontics and Oral Microbiology, Karolinska Institutet, 1985.
Find full textBranting, Christina. Studies on S øt ør øe øp øt øo øc øo øc øc øu øs ø m øu øt øa øn øs ø glucans with special reference to cell adhesion. Stockholm: Kongl. Carolinska Medico Chirurgiska Institutet, 1988.
Find full textZarrabian, Shirin. Development of improved slow-release dental varnish formulations effective against streptococcus mutans in vitro. [Toronto: Faculty of Dentistry, University of Toronto], 1992.
Find full textZarrabian, Shirin. Development of improved slow-release dental varnish formulations effective against Streptococcus mutans in vitro. Ottawa: National Library of Canada, 1994.
Find full textWang, Dongsheng. Molecular cloning and nucleotide sequence of a Streptococcus mutans gene encoding biotin carboxyl carrier protein. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1993.
Find full textWang, Dongsheng. Molecular cloning and nucleotide sequence of a streptococcus mutans gene encoding biotin carboxyl carrier protein. [Toronto: Faculty of Dentistry, University of Toronto], 1992.
Find full textNewsholme, Heather Dawn Bickell. Influence of a substituted guar gum on the adshesion of streptococcus mutans to glass and hydroxylapatite. Salford: University of Salford, 1985.
Find full textBook chapters on the topic "Streptococcus mutans"
Nakano, Kazuhiko, Ichiro Nakagawa, Satu Alaluusua, and Takashi Ooshima. "Molecular Typing of Streptococcus mutans." In Molecular Typing in Bacterial Infections, 127–47. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-62703-185-1_9.
Full textLewis, C. R., and R. R. B. Russell. "Chromosomal Deletions in Streptococcus mutans." In Streptococci and the Host, 677–79. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1825-3_158.
Full textLemos, J. A., S. R. Palmer, L. Zeng, Z. T. Wen, J. K. Kajfasz, I. A. Freires, J. Abranches, and L. J. Brady. "The Biology of Streptococcus mutans." In Gram-Positive Pathogens, 435–48. Washington, DC, USA: ASM Press, 2019. http://dx.doi.org/10.1128/9781683670131.ch27.
Full textCurtiss, R. "Genetic Analysis of Streptococcus mutans Virulence." In Current Topics in Microbiology and Immunology, 253–77. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70586-1_14.
Full textLion, Christine, A. Lozniewski, M. Weissenbach, and M. Weber. "“Streptococcus mutans” Group in Human Saliva." In Streptococci and the Host, 173–75. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1825-3_42.
Full textKuramitsu, Howard K. "The Virulence Properties of Streptococcus mutans." In Gram-Positive Pathogens, 340–46. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555816513.ch28.
Full textAjdic, Dragana, and Joseph J. Ferretti. "Regulation of the Galactose Operon of Streptococcus mutans." In Streptococci and the Host, 1015–18. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1825-3_239.
Full textSpatafora, Grace A., and Meagan W. Moore. "Growth of Streptococcus mutans in an iron-limiting medium." In Methods for studying the genetics, molecular biology, physiology, and pathogenesis of the streptococci, 217–21. Dordrecht: Springer Netherlands, 1998. http://dx.doi.org/10.1007/978-94-017-2258-2_24.
Full textHaas, Wolfgang, and Jeffrey A. Banas. "The Glucan Binding Domain of the Streptococcus mutans Glucan Binding Protein." In Streptococci and the Host, 707–8. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1825-3_165.
Full textKing, William F., Tsute Chen, Ruchele Nogueira, Renata Mattos-Graner, and Daniel J. Smith. "Epitopes shared among pioneer oral flora and Streptococcus mutans GbpB." In Interface Oral Health Science 2009, 416–17. Tokyo: Springer Japan, 2010. http://dx.doi.org/10.1007/978-4-431-99644-6_119.
Full textConference papers on the topic "Streptococcus mutans"
Tek, Erhan, and Nizami Duran. "Efficacy of Capsaicin on Cell Adhesion and Invasion of Oral Pathogens." In The 9th International Conference on Advanced Materials and Systems. INCDTP - Leather and Footwear Research Institute (ICPI), Bucharest, Romania, 2022. http://dx.doi.org/10.24264/icams-2022.iii.19.
Full textRago, I., A. Bregnocchi, E. Zanni, A. G. D'Aloia, F. De Angelis, M. Bossu, G. De Bellis, A. Polimeni, D. Uccelletti, and M. S. Sarto. "Antimicrobial activity of graphene nanoplatelets against Streptococcus mutans." In 2015 IEEE 15th International Conference on Nanotechnology (IEEE-NANO). IEEE, 2015. http://dx.doi.org/10.1109/nano.2015.7388945.
Full textBeier, Brooke D., Robert G. Quivey, and Andrew J. Berger. "Confocal Raman Microscopy of Streptococcus Sanguis and Mutans." In Frontiers in Optics. Washington, D.C.: OSA, 2008. http://dx.doi.org/10.1364/fio.2008.fwd6.
Full textBeier, Brooke D., Robert G. Quivey, and Andrew J. Berger. "Confocal Raman Microspectroscopy of Streptococcus sanguis and mutans." In Frontiers in Optics. Washington, D.C.: OSA, 2010. http://dx.doi.org/10.1364/fio.2010.ftuf7.
Full textAstuti, Suryani Dyah, A. Zaidan, Ernie Maduratna Setiawati, and Suhariningsih. "Chlorophyll mediated photodynamic inactivation of blue laser on Streptococcus mutans." In 5TH INTERNATIONAL CONFERENCE AND WORKSHOP ON BASIC AND APPLIED SCIENCES (ICOWOBAS 2015). AIP Publishing LLC, 2016. http://dx.doi.org/10.1063/1.4943353.
Full textBurns, Tracy, Michael Wilson, and G. J. Pearson. "Mechanism of killing of streptococcus mutans by light-activated drugs." In BiOS Europe '95, edited by Benjamin Ehrenberg, Giulio Jori, and Johan Moan. SPIE, 1996. http://dx.doi.org/10.1117/12.230944.
Full textSoekanto, Sri Angky, Saly Salim Alatas, Rima Ristanti, Ferry P. Gultom, and Muhamad Sahlan. "Efficacy of propolis fluoride in inhibiting the formation of Streptoccocus mutans, Streptococcus gordonii, and Streptococcus sanguinis biofilm." In SECOND INTERNATIONAL CONFERENCE OF MATHEMATICS (SICME2019). Author(s), 2019. http://dx.doi.org/10.1063/1.5096712.
Full textChismirina, Santi, Suzanna Sungkar, Ridha Andayani, Sri Rezeki, and Darmawi. "Existence of Streptococcus Mutans and Streptococcus Sobrinus in Oral Cavity as Main Cariogenc Bacteria of Dental Caries." In 1st Aceh International Dental Meeting (AIDEM 2019), Oral Health International Conference On Art, Nature And Material Science Development 2019. Paris, France: Atlantis Press, 2021. http://dx.doi.org/10.2991/ahsr.k.210201.020.
Full textGegiu, Gabriela. "STUDY REGARDING THE EFFECT OF PRUNUS SPINOSA SPECIES ON STREPTOCOCCUS MUTANS." In 2nd International Multidisciplinary Scientific Conference on Social Sciences and Arts SGEM2015. Stef92 Technology, 2015. http://dx.doi.org/10.5593/sgemsocial2015/b11/s2.121.
Full textSui, Lei, Pingting Wang, Rui Li, and Changyi Li. "Antibacterial Effect of Photodynamic Therapy on Prepared Tooth Structure Contaminated with Streptococcus mutans." In 2009 Symposium on Photonics and Optoelectronics. IEEE eXpress Conference Publishing, 2009. http://dx.doi.org/10.1109/sopo.2009.5230128.
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