Relevant bibliographies by topics / STM1

Academic literature on the topic 'STM1'

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Published: 4 June 2021
Last updated: 3 February 2022

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Journal articles on the topic "STM1"

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Ligr, Martin, Iris Velten, Eleonore Fröhlich, Frank Madeo, Matthias Ledig, Kai-Uwe Fröhlich, Dieter H. Wolf, and Wolfgang Hilt. "The Proteasomal Substrate Stm1 Participates in Apoptosis-like Cell Death in Yeast." Molecular Biology of the Cell 12, no. 8 (August 2001): 2422–32. http://dx.doi.org/10.1091/mbc.12.8.2422.

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We have identified the yeast gene STM1 in an overexpression screen for new proteasomal substrates. Stm1 is unstable in wild-type cells and stabilized in cells with defective proteasomal activity and thus a bona fide substrate of the proteasome. It is localized in the perinuclear region and is required for growth in the presence of mutagens. Overexpression in cells with impaired proteasomal degradation leads to cell death accompanied with cytological markers of apoptosis: loss of plasma membrane asymmetry, chromatin condensation, and DNA cleavage. Cells lacking Stm1 display deficiency in the apoptosis-like cell death process induced by treatment with low concentrations of H2O2. We suggest that Stm1 is involved in the control of the apoptosis-like cell death in yeast. Survival is increased when Stm1 is completely missing from the cells or when inhibition of Stm1 synthesis permits proteasomal degradation to decrease its amount in the cell. Conversely, Stm1 accumulation induces cell death. In addition we identified five other genes whose overexpression in proteasomal mutants caused similar apoptotic phenotypes.
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Barlow, Blake R., Lovreet S. Shergill, Mandy D. Bish, and Kevin W. Bradley. "Investigations of the Potential Interactions Between Pre-emergence Residual Herbicides, Variety, and Seed Treatments in Soybean." Weed Technology 32, no. 5 (September 24, 2018): 570–78. http://dx.doi.org/10.1017/wet.2018.44.

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AbstractField experiments were performed in 2016 and 2017 in Missouri to determine whether interactions exist between PRE herbicides and seed treatments in soybean. The experiments consisted of a randomized complete block design with factorial arrangements of varieties, seed treatments, and herbicides. We selected two genetically similar varieties of soybean, one with known tolerance to PPO-inhibiting herbicides and one with known sensitivity. Each variety of seed received three separate seed treatment mixtures (STMs): (1) STM1, imidacloprid plus prothioconazol+penflufen+metalaxyl plus metalaxyl plusBacillus subtilis+B. pumilis, (2) STM2,Pasteuria nishizawaeplus thiamethoxam plus prothioconazol+penflufen+metalaxyl plus metalaxyl plusB. subtilis+B. pumilis, and (3) STM3, fluopyram plus imidacloprid plus prothioconazol+penflufen+metalaxyl plus metalaxyl plusB. subtilis+B. pumilis. Chlorimuron-ethyl+flumioxazin+pyroxasulfone, chlorimuron-ethyl+flumioxazin+metribuzin, and chlorimuron-ethyl+sulfentrazone were applied PRE to each variety and seed treatment combination at 1× and 2× the labeled use rate. Chlorimuron-ethyl+sulfentrazone treatment at the 2× rate resulted in greater injury of 8% and 14% to the sensitive variety than the tolerant in 2016 and 2017, respectively; this was the highest injury observed from any herbicide treatment in either year. In 2017, chlorimuron-ethyl+sulfentrazone resulted in the greatest height reductions in both varieties, but this reduction was more evident in the sensitive (19%) than in the tolerant (6%) variety. Overall, yield differences between the two varieties were not consistent between years, and for both varieties, the sulfentrazone-containing treatments resulted in the highest yield losses. The results of this research indicate that there is a larger interaction between herbicides and varieties than there is between herbicides and seed treatments, or seed treatments and varieties.
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Bachtiar, Endang W., Kuo-Ching Sheng, Theodora Fifis, Anita Gamvrellis, Magdalena Plebanski, Peter J. Coloe, and Peter M. Smooker. "Delivery of a heterologous antigen by a registeredSalmonellavaccine (STM1)." FEMS Microbiology Letters 227, no. 2 (October 2003): 211–17. http://dx.doi.org/10.1016/s0378-1097(03)00683-9.

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Kumar, Chandrakesh, and Rajan Mishra. "Miniaturized Dual Band Meander Antenna For WLAN/STM1 Application." i-manager's Journal on Communication Engineering and Systems 4, no. 3 (July 15, 2015): 20–24. http://dx.doi.org/10.26634/jcs.4.3.3454.

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Balagopal, V., and R. Parker. "Stm1 modulates translation after 80S formation in Saccharomyces cerevisiae." RNA 17, no. 5 (April 1, 2011): 835–42. http://dx.doi.org/10.1261/rna.2677311.

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Balagopal, Vidya, and Roy Parker. "Stm1 Modulates mRNA Decay and Dhh1 Function in Saccharomyces cerevisiae." Genetics 181, no. 1 (November 17, 2008): 93–103. http://dx.doi.org/10.1534/genetics.108.092601.

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Hata, Hiroaki, Hisayuki Mitsui, Hong Liu, Yongli Bai, Clyde L. Denis, Yuki Shimizu, and Akira Sakai. "Dhh1p, a Putative RNA Helicase, Associates with the General Transcription Factors Pop2p and Ccr4p from Saccharomyces cerevisiae." Genetics 148, no. 2 (February 1, 1998): 571–79. http://dx.doi.org/10.1093/genetics/148.2.571.

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Abstract The POP2 (Caf1) protein in Saccharomyces cerevisiae affects a variety of transcriptional processes and is a component of the Ccr4p complex. We have isolated five multicopy suppressor genes of a pop2 deletion mutation: CCR4, DHH1 (a putative RNA helicase), PKC1, STM1, and MPT5 (multicopy suppressor of pop two). Overexpression of either the CCR4 or DHH1 genes effectively suppressed phenotypes associated with pop2 mutant cells; overexpression of PKC1, STM1, or MPT5 genes produced only partial suppression. Disruption of the CCR4 or DHH1 genes resulted in phenotypes similar to those observed for pop2 cells. In addition, overexpression of the DHH1 gene also suppressed the ccr4 mutation, suggesting a close relationship between the POP2, CCR4, and DHH1 genes. Two-hybrid analysis and coimmunoprecipitation experiments revealed that Pop2p and Dhh1p interact physically, and these and other data suggest that Dhh1p is also a component of the Ccr4p complex. Finally, we investigated the genetic interaction between factors associated with POP2 and the PKC1 pathway. The temperature-sensitive growth defect of dhh1 or mpt5 cells was suppressed by overexpression of PKC1, and the defect of mpk1 cells was suppressed by overexpression of MPT5. These results and phenotypic analysis of double mutants from the POP2 and PKC1 pathways suggested that the POP2 and the PKC1 pathways are independent but have some overlapping functions.
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Hayashi, Hikari, Riku Nagai, Taisho Abe, Miki Wada, Koichi Ito, and Nono Takeuchi-Tomita. "Tight interaction of eEF2 in the presence of Stm1 on ribosome." Journal of Biochemistry 163, no. 3 (October 23, 2017): 177–85. http://dx.doi.org/10.1093/jb/mvx070.

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Katayama, T., N. Inoue, and H. Torigoe. "Location of the triplex DNA-binding domain of Saccharomyces cerevisiae Stm1 protein." Nucleic Acids Symposium Series 51, no. 1 (November 1, 2007): 123–24. http://dx.doi.org/10.1093/nass/nrm062.

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Hayashi, N., and S. Murakami. "STM1, a gene which encodes a guanine quadruplex binding protein, interacts with CDC13 in Saccharomyces cerevisiae." Molecular Genetics and Genomics 267, no. 6 (August 2002): 806–13. http://dx.doi.org/10.1007/s00438-002-0712-3.

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Dissertations / Theses on the topic "STM1"

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Ilina, Yulia. "Functions of the yeast protein Stm1 and its involvement in apoptotic cell death." [S.l. : s.n.], 2005.

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Balagopal, Vidya. "STM1 IS A NOVEL REGULATOR OF MESSENGER RNA TRANSLATION AND DEGRADATION IN SACCHAROMYCES CEREVISIAE." Diss., The University of Arizona, 2010. http://hdl.handle.net/10150/145717.

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In eukaryotes, regulation of translation and decay of messenger RNA are critical for fine-tuned control of gene expression. An important point of control is the key transition where mRNAs exit translation and assemble into a non-translating mRNP state that can accumulate in cytoplasmic granules such as P bodies and/or Stress granules. In the budding yeast Saccharomyces cerevisiae , the activators of decapping Dhh1 and Pat1 appear to promote the exit of mRNAs from translation. In my work, summarized below, I describe a new regulator of translation repression and mRNA degradation, Stm1, and its novel mode of action. First, I identified Stm1 as a novel regulator of translation repression and mRNA decay. Stm1 shows several genetic interactions with Pat1 and Dhh1, in a manner consistent with Stm1 promoting the function of Dhh1. This suggests that Stm1 has a role to play in translation repression and/or activation of mRNA decay. stm1 δ strains are defective in the degradation of a subset of mRNAs that include EDC1 and COX17 . These results strongly argue that Stm1 is a novel addition to the mRNA degradation machinery. Second, I have shown that Stm1, a known ribosome-associated protein, can bind and stall 80S ribosomes to repress translation and promote decay. Stm1 is able to repress translation and stall an 80S complex in vitro . Several mutations were identified in the protein, which link the in vitrophenotype to its biological functionin vivo. The analysis of different steps in translation reveals Stm1 functions in a novel manner to inhibit translation after the formation of an 80S complex. Since most of the regulation of translation is thought to happen at the stage of initiation, this study reveals a novel mode of translation regulation. These results also provide a direct and mechanistic link between ribosome function, inhibition of translation and the degradation of messenger RNAs.
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Gatsos, Xenia, and xgatsos@optusnet com au. "The development of live vectored vaccines targeting the alpha-toxin of Clostridium perfringens for the prevention of necrotic enteritis in poultry." RMIT University. Applied Sciences, 2007. http://adt.lib.rmit.edu.au/adt/public/adt-VIT20080212.142403.

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The ƒÑ-toxin of Clostridium perfringens is a toxin involved in numerous diseases of humans and agriculturally important animals. One of these diseases is necrotic enteritis (NE), a sporadic enteric disease which affects avian species world-wide. This study involved the inactivation of alpha-toxin (ƒÑ-toxin) for use as a potential vaccine candidate to combat NE in chickens, and other diseases caused by C. perfringens type A. During the course of this research a number of ƒÑ-toxin recombinant proteins were developed through molecular inactivation of the ƒÑ-toxin gene, plc. Proteins plc316 and plc204 were developed by the deletion of the first three and seven ƒÑ-helices of the N-terminal domain respectively. These deletions resulted in proteins which were unstable in solution, constantly aggregated into insoluble masses and elicited lower overall antibody responses when administered to mice. A third protein, plcInv3 was developed from the deletion of part of the catalytic domain of the ƒÑ-toxin. PlcInv3 was highly soluble and upon immunisation of mice elicited a significant antibody response which was also capable of protecting mice against a live challenge of C. perfringens. The fourth and final protein developed was plc104. The smallest of the recombinant ƒÑ-toxin proteins, it consisted entirely of the C-terminal domain of ƒÑ-toxin. Its small size did not affect its ability to induce a strong antibody response when administered to mice, the antibodies of which were also protective during a challenge with C. perfringens. STM1, an attenuated strain of S. Typhimurium was used in the development of a vectored vaccine for the expression and oral delivery of plcInv3 and plc104 within the mouse host. The proteins were expressed within STM1 from expression plasmids containing the in vivo inducible promoters PhtrA and PpagC. A measurable humoral immune response against ƒÑ-toxin was absent following three oral vaccinations with the vectored vaccines, although, cytokine profiling of splenocytes from vaccinated mice revealed an increase in the number of interleukin-4 (IL-4)secreting cells and the lack of interferon-gamma (IFN-ƒ×) secreting cells. This indicated the stimulation of a T-helper type 2 (TH2) immune response which also lead to partial protection against a live C. perfringens challenge. This study demonstrates the feasibility of using STM1 as a carrier for the in vivo expression of the C. perfringens ƒÑ-toxin recombinant proteins plcInv3 and plc104. It is the first study to express C. perfringens antigens within an attenuated strain of S. Typhimurium, STM1.The partial protection of mice immunised with these vaccines indicates there is potential for this vectored vaccine system to be used in the protection of diseases caused by the ƒÑ-toxin of C. perfringens.
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ABREU, FERNANDA DE MELLO. "TIME-DOMAIN OPTICAL MULTIPLEXING IN STM-16, STM-64 AND STM-256 SYSTEMS." PONTIFÍCIA UNIVERSIDADE CATÓLICA DO RIO DE JANEIRO, 2001. http://www.maxwell.vrac.puc-rio.br/Busca_etds.php?strSecao=resultado&nrSeq=2361@1.

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PONTIFÍCIA UNIVERSIDADE CATÓLICA DO RIO DE JANEIRO
ALCATEL TELECOMUNICAÇÕES
Este trabalho tem como foco o up-grade da taxa de bits em enlaces ópticos através da tecnologia OTDM. Os sistemas analisados contemplam os up-grades das taxas de 2,48 Gbps para 10 Gbps e também da taxa de 10 Gbps para 40 Gbps. Para tal, foram introduzidos módulos de transmissão e recepção, capazes de utilizar arquiteturas quase totalmente ópticas. É avaliado então, através de simulações, o comportamento da arquitetura proposta em infra-estruturas de enlaces já instalados no Brasil, destacando os pontos mais críticos. No que se refere ao up-grade de 10 Gbps para 40 Gbps, foi dado enfoque especial para as penalidades relativas à PMD (Polarization Mode Dispersion).
This work aims at up grading the bit rate of optical links through the OTDM technology. The analyzed up-grades change the bit rate of 2,48 Gbps up to 10 Gbps and also from the bit rate of 10 Gbps up to 40 Gbps. To reach these objectives, transmission and reception modules were introduced, using all optical networks topologies. The performance of the proposed architecture was simulated using a infrastructure of links already installed in Brazil. The most critical issues were pointed out. Concerning the up-grade from 10 Gbps to 40 Gbps, a special focus was given to the penalties due to PMD (Polarization Mode Dispersion).
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Al, Badine Samir. "STMM soumission de travaux en mode messagerie /." Grenoble 2 : ANRT, 1988. http://catalogue.bnf.fr/ark:/12148/cb37611499t.

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Lampe, Birgit [Verfasser]. "Transkranielle Einzelimpulsstimulation (sTMS) bei akustischer Verbgenerierung / Birgit Lampe." Köln : Deutsche Zentralbibliothek für Medizin, 2012. http://d-nb.info/1024715361/34.

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Cardoso, Aline Monticelli 1988. "Estudos sobre a internalização celular da STC1 humana." [s.n.], 2015. http://repositorio.unicamp.br/jspui/handle/REPOSIP/314360.

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Orientador: Jörg Kobarg
Dissertação (mestrado) - Universidade Estadual de Campinas, Instituto de Biologia
Made available in DSpace on 2018-08-27T05:39:09Z (GMT). No. of bitstreams: 1 Cardoso_AlineMonticelli_M.pdf: 19291466 bytes, checksum: 5920430c506c4670ca04da10ad934398 (MD5) Previous issue date: 2015
Resumo: A Stanniocalcina-1 (STC1) humana é uma glicoproteína homóloga a Stanniocalcina (STC) originalmente identificada como um hormônio regulador da homeostase de cálcio em peixes. A STC1 humana secretada atua em diferentes processos fisiológicos incluindo a angiogênese, a hipóxia e, principalmente, a carcinogênese, demonstrando assim uma atividade abrangente. Atualmente não se conhece o receptor da STC1 e pouco se sabe sobre o mecanismo de ação e de entrada nas células dessa proteína. Assim, o objetivo desse trabalho foi investigar um candidato a receptor de membrana dessa proteína, o receptor de transferrina (TfR1), uma proteína transmembrana responsável pela absorção de ferro nas células. Esse receptor é provavelmente expresso por todas as células em diferentes níveis, em destaque em células do sistema hematopoiético, em células em divisão celular e células neoplásicas. Assim, avaliou-se por citometria de fluxo o efeito do tratamento com STC1 em células não transfectadas e células transfectadas superexpressando o receptor de transferrina. Células tratadas com STC1 demonstraram um efeito semelhante ao tratamento com transferrina, um conhecido ligante desse receptor, no qual ambos diminuíram o número de células positivas para a marcação da superfície com transferrina conjugada com fluorocromo (transferrina-Alexa Fluor® 488 - Life Technologies). Em outro conjunto de experimentos de Western Blot foi demonstrado que a STC1 adicionada no sobrenadante das culturas de células é internalizada nas células e detectável no lisado celular, principalmente as células transfectadas para a superexpressão do receptor de transferrina. Complementarmente, em experimentos de localização subcelular por imunofluorescência a STC1 foi detectada em uma forma pontual e espalhada no citoplasma. Em conjunto, todos esses experimentos sugerem que STC1 e transferrina interferem na localização do receptor de transferrina na superfície celular e que possivelmente esse receptor está envolvido em mecanismos de internalização da própria STC1
Abstract: Human Stanniocalcin 1 (STC1) is the mammalian homologue of STC, which was originally identified as a calcium-regulating hormone in bony fishes. The human secreted Stanniocalcin acts on different physiological processes, including angiogenesis, hypoxia and especially carcinogenesis, facts that demonstrate their activity is wide. Currently there are few data on the mechanism of action of this protein or how it enters the cell. Thus, the aim of this study was to investigate transferrin receptor (TfR1) as a candidate to membrane receptor protein of STC1. This receptor is a membrane protein responsible for the iron uptake in cells. This receptor is probably expressed by all cells especially by cells in division and cancer cells, but its expression level may vary. We evaluated by flow cytometry the effect of STC1 treatment in non-transfected cells and cell with TfR1 overexpression. The treatment with STC demonstrated a similar effect to treatment with transferrin, a known ligand for receptor, which decreased the number of positive cells for staining with fluorochrome (transferrin conjugated to Alexa Fluor® 488 - Life Technologies). We also demonstrated by Western Blot that STC1 added to the supernatant of cultures of cells, especially cells that overexpress transferrin receptor, is internalized into the cells and detectable in the cell lysate. Additionally, in subcellular localization experiments by immunofluorescence STC1 was detected in a timely manner and scattered in the cytoplasm. Together all this information suggests that STC1 and transferrin interferes with the localization of the transferrin receptor in the cellular surface and perhaps this receptor is involved in the mechanism of internalization of STC1
Mestrado
Bioquimica
Mestra em Biologia Funcional e Molecular
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Holl, Christian [Verfasser], Markus [Akademischer Betreuer] Morgenstern, and Samir [Akademischer Betreuer] Lounis. "High frequency STM and spin polarized STM on magnetic vortices / Christian Holl ; Markus Morgenstern, Samir Lounis." Aachen : Universitätsbibliothek der RWTH Aachen, 2018. http://d-nb.info/1192217926/34.

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Azevedo, Cristina Maria Lourenço da Cunha Correia de. "Functional analysis of RAR1 and STG1 in disease resistance." Thesis, University of East Anglia, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.399785.

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Troupes, Constantine. "The Role of STIM1 in Hypertrophy-Related Contractile Dysfunction." Diss., Temple University Libraries, 2016. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/403786.

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Biomedical Sciences
Ph.D.
Increases in cardiac afterload caused by disease conditions results in remodeling of heart structure by hypertrophy and alterations in the molecular regulation of contractile performance. These adaptations can be regulated by various Ca2+-dependent signaling processes. STIM1 is an important regulator of Ca2+ signaling in different cell types by sensing endoplasmic reticular Ca2+ levels and coupling to plasma membrane Orai channels. The role of STIM1 in heart is not well understood, given the robust Ca2+ regulatory machinery present within cardiac myocytes. Previous reports indicate that STIM1 may play a role in regulation of cardiac hypertrophy. The goal of this work is to understand how STIM1 can affect contractile Ca2+ regulation in normal and diseased myocytes. We induced cardiac hypertrophy by slow progressive pressure overload in adult cats. Isolated adult feline ventricular myocytes (AFMs) exhibited increased STIM1 expression and activity, which correlated with altered Ca2+ handling. Use of BTP2 to block Orai channels resulted in a reduction of action potential (AP) duration and diastolic spark rate of hypertrophied myocytes, without affecting myocytes from sham-operated animals. Overexpressed STIM1 in cultured AFMs caused persistent Ca2+ influx that resulted in increased diastolic spark rates and prolonged APs, similar to myocytes from banded animals. STIM1 mediated Ca2+ influx could load the sarcoplasmic reticulum and activated CaMKII, which increased spark rates and lead to spontaneous APs. Importantly, STIM1 operated by associating with Orai channels because these effects could be blocked with either BTP2 or with a dominant negative Orai construct. Prolonged Ca2+ entry through this pathway eventually causes cell death. In conclusion, the work presented in this thesis establishes a role for STIM1-Orai in contractile Ca2+ regulation.
Temple University--Theses
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Books on the topic "STM1"

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Hardt, Robert, ed. Six Themes on Variation. Providence, Rhode Island: American Mathematical Society, 2004. http://dx.doi.org/10.1090/stml/026.

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Purwadi, Agung. Studi pembiayaan STM negeri. Jakarta: Pusat Penelitian Kebijakan, Badan Penelitian dan Pengembangan, Departemen Pendidikan Nasional, 2001.

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Katsirikou, Anthi, ed. Open Access to STM Information. Berlin, Boston: DE GRUYTER SAUR, 2011. http://dx.doi.org/10.1515/9783110263749.

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Yamagata, Yoriyuki. Interpretation of STM by CSP. Hyōgo-ken Amagasaki-shi: Sangyō Gijutsu Sōgō Kenkyūjo (Kumikomi Shisutemu Gijutsu Renkei Kenkyūtai), 2012.

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Elizeche, Marco Antonio. El distrito de Stma. Trinidad y el dictador Francia: Semblanzas. Asunción, Paraguay: Marco Antonio Elizeche, 2011.

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Delmonte, Clive. Advances in AFM & STM applied to thenucleic acids. Northampton: Clive Delmonte Publications, 1997.

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Davidson, Fiona. Some effects of added stimuli on pigeon STM. Birmingham: University of Birmingham, 1986.

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STM Study Group on Marketing. Meeting. STM seminar on Spain as a transfer channel of STM information between Europe and Ibero-America: [proceedings of the] STM Study Group on Marketing, thirty-eighth meeting, Barcelona, 25 September 1984. Amsterdam: International Group of Scientific Technical and Medical Publishers, 1985.

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Goetze, Rolf. Population & housing profile, U. S. census STF1, 1990: Boston and its neighborhoods. Amherst, MA: The Center, 1991.

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Miller, Jimmie Andrew. From STM to nanomemory: A transfer of technology feasibility study. [s.l.]: typescript, 1994.

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Book chapters on the topic "STM1"

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Leung, Alexander K. C., Cham Pion Kao, Andrew L. Wong, Alexander K. C. Leung, Thomas Kolter, Ute Schepers, Konrad Sandhoff, et al. "STM." In Encyclopedia of Molecular Mechanisms of Disease, 1996. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_6099.

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Edkins, Stephen. "Spectroscopic-Imaging STM (SI-STM)." In Visualising the Charge and Cooper-Pair Density Waves in Cuprates, 23–49. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-65975-6_2.

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Auffan, Mélanie, Catherine Santaella, Alain Thiéry, Christine Paillès, Jérôme Rose, Wafa Achouak, Antoine Thill, et al. "EC-STM." In Encyclopedia of Nanotechnology, 645. Dordrecht: Springer Netherlands, 2012. http://dx.doi.org/10.1007/978-90-481-9751-4_100203.

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Frischauf, Irene, Marc Fahrner, Isaac Jardín, and Christoph Romanin. "The STIM1: Orai Interaction." In Advances in Experimental Medicine and Biology, 25–46. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-26974-0_2.

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Siegenthaler, H. "STM in Electrochemistry." In Scanning Tunneling Microscopy II, 7–49. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79366-0_2.

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Kuk, Y. "STM on Metals." In Springer Series in Surface Sciences, 17–37. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-79255-7_3.

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Hamers, R. J. "STM on Semiconductors." In Springer Series in Surface Sciences, 83–129. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-79255-7_5.

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van Bentum, P. J. M., and H. van Kempen. "STM on Superconductors." In Springer Series in Surface Sciences, 207–42. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-79255-7_8.

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Hamers, R. J. "STM on Semiconductors." In Springer Series in Surface Sciences, 83–129. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-97343-7_5.

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Siegenthaler, H. "STM in Electrochemistry." In Scanning Tunneling Microscopy II, 7–49. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-97363-5_2.

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Conference papers on the topic "STM1"

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Clayton, G. M., and S. Devasia. "Image-Based Trajectory Estimation for Scanning Tunneling Microscopy." In ASME 2007 International Mechanical Engineering Congress and Exposition. ASMEDC, 2007. http://dx.doi.org/10.1115/imece2007-42262.

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In this article we present an image-based approach to estimate the probe position trajectory in scanning tunneling microscopes (STMs). STMs are key enabling tools in the experimental investigation and manipulation of nano and sub-nano scale phenomena; however, due to an inability to measure the STM-probe position, typical STMs are limited to low bandwidth operations to ensure positioning accuracy. To overcome sensor deficiencies, thus enabling higher bandwidth STMs, an image-based method which produces discrete samples of the STM-tip trajectory has been developed. In this article we explore how the STM calibration sample and the output affect the reconstructability of the STM trajectory.
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Clayton, G. M., and S. Devasia. "Image-Based Control of Dynamic Effects in Scanning Tunneling Microscopes." In ASME 2004 International Mechanical Engineering Congress and Exposition. ASMEDC, 2004. http://dx.doi.org/10.1115/imece2004-59473.

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In this article, we present an image-based control scheme to address the problem of low operating speed in scanning tunneling microscopes (STMs). Low operating speeds are used because dynamic effects cause error in positioning the STM probe over the sample. The STM’s low operating speed limits its capability to investigate fast surface phenomena and its throughput during nanofabrication. The proposed image-based approach to achieve high speed operation exploits the extant imaging capabilities of STMs. In practice, distortions in high-speed images are used to identify and correct errors in the STM probe position. The approach is applied to an STM and experimental results are presented.
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Ravichandran, Kaushik, and Santosh Pande. "F2C2-STM: Flux-Based Feedback-Driven Concurrency Control for STMs." In 2014 IEEE International Parallel & Distributed Processing Symposium (IPDPS). IEEE, 2014. http://dx.doi.org/10.1109/ipdps.2014.99.

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Kestor, Gokcen, Roberto Gioiosa, Tim Harris, Osman S. Unsal, Adrian Cristal, Ibrahim Hur, and Mateo Valero. "STM2: A Parallel STM for High Performance Simultaneous Multithreading Systems." In 2011 International Conference on Parallel Architectures and Compilation Techniques (PACT). IEEE, 2011. http://dx.doi.org/10.1109/pact.2011.54.

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Kim, Jihyun, and Youjip Won. "OpF-STM." In the 2018 International Conference. New York, New York, USA: ACM Press, 2018. http://dx.doi.org/10.1145/3193063.3193076.

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Dolev, Shlomi, Danny Hendler, and Adi Suissa. "CAR-STM." In the twenty-seventh ACM symposium. New York, New York, USA: ACM Press, 2008. http://dx.doi.org/10.1145/1400751.1400769.

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Saha, Bratin, Ali-Reza Adl-Tabatabai, Richard L. Hudson, Chi Cao Minh, and Benjamin Hertzberg. "McRT-STM." In the eleventh ACM SIGPLAN symposium. New York, New York, USA: ACM Press, 2006. http://dx.doi.org/10.1145/1122971.1123001.

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Lee, Sang M., M. Abdelmaksoud, and J. Krim. "Nano-Scale Tribology Study of Organic Adlayer-Metal Interface Using Quartz Crystal Microbalance Combined With Scanning Tunneling Microscopy." In World Tribology Congress III. ASMEDC, 2005. http://dx.doi.org/10.1115/wtc2005-63732.

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A quartz crystal microbalance combined with scanning tunneling microscopy (STM-QCM) was used to investigate the interactions between organic adlayers (C6H6 and C6H5I) on Cu surfaces and a metallic STM tip. STM images of C6H6 covered Cu surface improved when the QCM was simultaneously oscillated during the imaging. In contrast, STM images of C6H5I covered surfaces became noisy when the sample was oscillated. The two systems moreover exhibited frequency changes of opposite signs in response to STM tip contact, indicative of different physical phenomena at the surface. The dependence of the STM image quality and the frequency shift were interpreted in terms of the adsorbate-substrate chemical and physical interactions, and different levels of frictional heating at the interface.
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LOBITZ, J., P. ACKERMAN, and S. WEBER. "STME T/C casting technology." In 28th Joint Propulsion Conference and Exhibit. Reston, Virigina: American Institute of Aeronautics and Astronautics, 1992. http://dx.doi.org/10.2514/6.1992-3280.

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Godett, T. M., R. J. Meijer, R. P. Verhey, C. J. Pearson, and K. Khalili. "STM4-120 Stirling Engine Test Development." In SAE International Congress and Exposition. 400 Commonwealth Drive, Warrendale, PA, United States: SAE International, 1989. http://dx.doi.org/10.4271/890149.

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Reports on the topic "STM1"

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PARSONS ENGINEERING SCIENCE INC DENVER CO. Results of Additional Bioventing Respiration Testing at Sites ST61, ST71, and ST43/55 (Pumphouse III and Valve Pit 3-4). Fort Belvoir, VA: Defense Technical Information Center, July 1996. http://dx.doi.org/10.21236/ada384464.

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Saw-Wai Hla. NSS5/SP-STM2 Joint International Conference. Office of Scientific and Technical Information (OSTI), May 2009. http://dx.doi.org/10.2172/951911.

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SARCOS RESEARCH CORP SALT LAKE CITY UT. STM-Based Hydrophone Sensors. Fort Belvoir, VA: Defense Technical Information Center, July 1991. http://dx.doi.org/10.21236/ada239821.

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SARCOS RESEARCH CORP SALT LAKE CITY UT. STM-Based Hydrophone Sensors. Fort Belvoir, VA: Defense Technical Information Center, May 1991. http://dx.doi.org/10.21236/ada236361.

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Sullivan, T. E., and P. H. Cutler. Laser Interactions in STM and STM-Like Devices: Applications to Infrared and Optical Detection. Fort Belvoir, VA: Defense Technical Information Center, April 1995. http://dx.doi.org/10.21236/ada299797.

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Davis, J. C. STM Studies of Semiconductor Qubit Candidates. Fort Belvoir, VA: Defense Technical Information Center, November 2005. http://dx.doi.org/10.21236/ada455573.

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Hamers, R. J. Methods of Tunneling Spectroscopy With the STM. Fort Belvoir, VA: Defense Technical Information Center, June 1993. http://dx.doi.org/10.21236/ada266507.

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Beaux, Miles Frank, Miguel A. Santiago Cordoba, Stephen Anthony Joyce, and Igor Olegovich Usov. AFM/STM Plutonium capability, research summary and future plans. Office of Scientific and Technical Information (OSTI), June 2016. http://dx.doi.org/10.2172/1259630.

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Sibener, Steven J. AASERT-96 Augmentation Award for STM Studies of Corrosion Reactions. Fort Belvoir, VA: Defense Technical Information Center, October 2000. http://dx.doi.org/10.21236/ada383435.

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Sarid, Dror. Novel Nanostructure Fabrication and Their Characterization by STM and AFM. Fort Belvoir, VA: Defense Technical Information Center, November 2000. http://dx.doi.org/10.21236/ada391137.

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