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1

Parakh, Neeraj, Ravi S. Math, and Vivek Chaturvedi, eds. Mitral Stenosis. Boca Raton, FL : CRC Press/Taylor & Francis Group, [2018]: CRC Press, 2018. http://dx.doi.org/10.1201/9781315166735.

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2

Abbas, Amr E., ed. Aortic Stenosis. London: Springer London, 2015. http://dx.doi.org/10.1007/978-1-4471-5242-2.

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3

Ibrahim, Ashraf, and Talal Al-Malki. Congenital Esophageal Stenosis. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-10782-6.

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4

Manfrè, Luigi, ed. Spinal Canal Stenosis. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-26270-3.

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5

Postacchini, Franco. Lumbar Spinal Stenosis. Vienna: Springer Vienna, 1989. http://dx.doi.org/10.1007/978-3-7091-9021-0.

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6

Postacchini, Franco. Lumbar spinal stenosis. Wien: Springer-Verlag, 1989.

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7

D, Rittenberg Joshua, ed. Lumbosacral spinal stenosis. Philadelphia, Pa: W.B. Saunders, 2003.

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8

1942-, Andersson Gunnar, and McNeill Thomas W. 1936-, eds. Lumbar spinal stenosis. St. Louis: Mosby Year Book, 1991.

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9

National Institute of Diabetes and Digestive and Kidney Diseases (U.S.), ed. Renal artery stenosis. Bethesda, MD]: U.S. Dept. of Health and Human Services, National Institutes of Health, National Institute of Diabetes and Digestive and Kidney Diseases, 2007.

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10

Denaro, Vincenzo. Stenosis of the cervical spine: Causes, diagnosis, and treatment. Berlin: Springer-Verlag, 1991.

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11

Goudie, R. B. Mitral stenosis and incompetence. Glasgow: Glasgow Royal Infirmary, 1985.

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12

Denaro, Vincenzo. Stenosis of the Cervical Spine. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-76203-1.

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13

Schaller, Bernhard J., ed. Imaging of Carotid Artery Stenosis. Vienna: Springer Vienna, 2007. http://dx.doi.org/10.1007/978-3-211-32509-4.

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14

J, Gould S., Graham J. M, and Malkin Jacqueline, eds. Acquired subglottic stenosis in infants. Ashford, Kent: Headley Brothers, 1988.

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15

Klein, Lloyd W., ed. Coronary Stenosis Morphology: Analysis and Implication. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-6287-0.

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16

W, Klein Lloyd, ed. Coronary stenosis morphology: Analysis and implication. Boston: Kluwer Academic Publishers, 1997.

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17

United States. Agency for Healthcare Research and Quality., ed. Treatment of degenerative lumbar spinal stenosis. [Rockville, Md: Agency for Healthcare Research and Quality, 2001.

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18

Gould, K. Lance. Coronary artery stenosis and reversing atherosclerosis. New York: Chapman & Hall, 1997.

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19

Dixon, Elijah, Charles M. Vollmer, and Gary R. May, eds. Management of Benign Biliary Stenosis and Injury. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-22273-8.

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20

Parakh, Neeraj, Ravi S. Math, and Vivek Chaturvedi. Mitral Stenosis. Taylor & Francis Group, 2020.

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21

Nixon. Spinal Stenosis. Edward Arnold, 1991.

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22

Stanton, Mike. Pyloric stenosis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198759928.003.0032.

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23

Beattie, R. Mark, Anil Dhawan, and John W.L. Puntis. Pyloric stenosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0028.

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Pyloric stenosis 204Infantile hypertrophic pyloric stenosis is a common cause of vomiting in infancy with an incidence of 2 to 3 per 1000 live births. Males are more commonly affected than females and there is a genetic predisposition. First-born males are more commonly affected....
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24

Davey, Patrick, and Jim Newton. Aortic stenosis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0093.

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Aortic stenosis is characterized by thickening and reduced mobility of the aortic valve leaflets and results in restriction to the blood flow from the left ventricle to the aorta, and secondary left ventricular hypertrophy.
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25

Freely Jr, John J., and Michel Sabbagh. Pyloric Stenosis. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0083.

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Pyloric stenosis is one of the most common surgical conditions affecting neonates and young infants. Hypertrophy of the pyloric muscular layers results in gradual gastric outlet obstruction. Persistent episodic projectile vomiting and dehydration as well as hypochloremic, hypokalemic metabolic alkalosis are cardinal features. Definitive treatment is surgical pyloromyotomy, but it is not a surgical emergency. Emergency medical intervention is often required to correct intravascular volume depletion and electrolyte disturbances. Morbidity and mortality should be limited due to advancements in surgical and perioperative care. Morbidity can occur due to poor preoperative resuscitation, anesthetic management difficulties, or postoperative complications. The following manuscript is a review of current evidence-based perioperative care of infants with pyloric stenosis. It reviews the pathophysiology that results in metabolic disturbances and intravascular volume depletion. It focuses on preoperative assessment and correction of electrolyte abnormalities and anesthetic technique including airway management and postoperative analgesia.
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26

Katritsis, Demosthenes G., Bernard J. Gersh, and A. John Camm. Mitral stenosis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199685288.003.0267_update_004.

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27

Katritsis, Demosthenes G., Bernard J. Gersh, and A. John Camm. Aortic stenosis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199685288.003.0325_update_004.

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Diagnosis and risk stratification of patients with aortic stenosis are presented. Indications for surgical therapy and percutaneous valve implantation based on the recommendations of ACC/AHA and ESC are summarized and tabulated.
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28

Spinal stenosis. London: Edward Arnold, 1991.

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29

Parakh, Neeraj, Ravi S. Math, and Vivek Chaturvedi. Mitral Stenosis. Taylor & Francis Group, 2018.

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30

Parakh, Neeraj, Ravi S. Math, and Vivek Chaturvedi. Mitral Stenosis. Taylor & Francis Group, 2018.

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31

Hopp. Spinal Stenosis. Hanley & Belfus, 1987.

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32

Parakh, Neeraj, Ravi S. Math, and Vivek Chaturvedi. Mitral Stenosis. Taylor & Francis Group, 2018.

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33

Parakh, Neeraj, Ravi S. Math, and Vivek Chaturvedi. Mitral Stenosis. Taylor & Francis Group, 2018.

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34

Parakh, Neeraj, Ravi S. Math, and Vivek Chaturvedi. Mitral Stenosis. Taylor & Francis Group, 2018.

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35

Verser, Lloyd. Aortic Stenosis Diagnosis : Spinal Stenosis Symptoms and Diagnosis: Stenosis of the Neck. Independently Published, 2021.

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36

Remme, Clora. Renal Artery Stenosis Diagnosis Gold Standard : Lumbar Spinal Stenosis: Stenosis Artery Diagnosis. Independently Published, 2021.

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37

Ritchie, James, Darren Green, Constantina Chrysochou, and Philip A. Kalra. Renal artery stenosis. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0213.

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Renovascular disease refers to a narrowing of a main or branch renal artery. Consequences include loss of functional renal tissue and renovascular hypertension, with other manifestations depending on the underlying cause. Worldwide the most common cause is atherosclerotic narrowing, with other causal pathologies including fibromuscular disease (FMD) and inflammatory conditions. FMD occurs much more frequently in women than in men, and is associated with smoking but genetic predisposing factors are also suspected. In South East Asia, Takayasu arteritis is an important cause.Takayasu disease often presents in a non-specific syndromic manner with fatigue and malaise. FMD often presents with early-onset hypertension. Atherosclerotic renal artery stenosis is often clinically silent with suspicion raised due to the existence of other cardiovascular pathology with the more dramatic presentations of acute decompensated heart failure or acute kidney injury less common. Clinical criteria can identify patients at risk.
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38

Ritchie, James, Darren Green, Constantina Chrysochou, and Philip A. Kalra. Renal artery stenosis. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0214.

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Modern practice typically utilizes indirect angiography by computed tomography or magnetic resonance imaging as the first-line diagnostic tool for renal artery stenosis, with no established screening tool able to meaningfully impact the pre-test probability of a positive finding. Neither can any current imaging technique reliably predict patient, blood pressure, or renal outcome following renal artery revascularization, although promising developments have been made in recent years.A major mechanism of hypertension in renovascular hypertension is overproduction of renin in response to hypoperfusion. While renin levels can be useful in investigating patients likely to have secondary hypertension, in the setting of renal impairment renin levels are not particularly useful in distinguishing treatable renal artery stenosis from other causes of renal disease.
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39

Ritchie, James, Darren Green, Constantina Chrysochou, and Philip A. Kalra. Renal artery stenosis. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0215.

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In fibromuscular disease (FMD), renal artery occlusion seems to be rare. Balloon angioplasty appears moderately successful in the medium term in controlling hypertension, at least in younger patients. In more complicated circumstances, medical therapy may be preferred. Similar approaches have been used in Takayasu disease but with less information about lasting outcomes.In atherosclerotic renal disease, the risk of renal artery occlusion and loss of renal function seems higher, but so are the complications of invasive management. Randomized clinical studies have not shown better blood pressure control or renal outcomes between medical therapy and percutaneous revascularization. As a consequence, modern management of atherosclerotic renovascular disease is primarily pharmacological, with interventional techniques reserved for selected presentations such as rapidly declining therapy, acute occlusion, or characteristic ‘flash’ pulmonary oedema.Whilst this approach is widely accepted, long-term outcome data are scant and there is ongoing research interest into specific disease phenotypes, refined interventional techniques, and novel treatment strategies aimed at preserving the renal microcirculation.
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40

Morrison, Gavin. Grading subglottic stenosis. Edited by John Phillips and Sally Erskine. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834281.003.0077.

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41

Chaturvedi, Seemant, and Peter Rothwell, eds. Carotid Artery Stenosis. CRC Press, 2005. http://dx.doi.org/10.1201/b14239.

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42

Chaturvedi, Seemant, and Peter M. Rothwell, eds. Carotid Artery Stenosis. CRC Press, 2005. http://dx.doi.org/10.3109/9780203025970.

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43

Gutierrez, Genaro J., and Divya Chirumamilla. Cervical Spinal Stenosis. Edited by Mehul J. Desai. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199350940.003.0006.

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Cervical spinal stenosis is the narrowing of the spinal canal. Degenerative cervical spinal stenosis can occur as a result of disc degeneration, osteophyte formation, and hypertrophy of spinal canal ligaments. Diagnosis is primarily made with clinical history and examination in order to assess for classic myelopathic signs (motor weakness, hyperreflexia, and other specific tests). Radiologic imaging is used to validation the diagnosis and to determine the extent of stenosis. Magnetic resonance imaging is the most useful and noninvasive modality. Cervical spinal stenosis without myelopathy can be managed nonsurgically with strengthening, physical therapy, traction, orthosis, and pain management (cervical epidural steroid injections and selective nerve root blocks). Cervical spondylolisthesis has received insufficient attention in comparison to spondylolisthesis of the lumbar spine. It is primarily considered a surgical condition, yet few publications have been dedicated to the topic.
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44

Nagpal, Ameet, and Brad Wisler. Thoracic Spinal Stenosis. Edited by Mehul J. Desai. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199350940.003.0011.

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Thoracic spinal stenosis is an uncommon pathologic condition of the spine. This chapter reviews its etiology, epidemiology, anatomic features, symptoms, diagnosis, and treatment. Four of the main causes of thoracic spinal stenosis are ossification of the ligamentum flavum, ossification of the posterior longitudinal ligament, thoracic disc herniation, and thoracic spondylosis. Even rarer secondary causes include generalized skeletal disorders, metabolic and endocrine disorders of the spine, neoplastic lesions, and vascular malformations. The chapter presents a brief review of the currently available surgical techniques. An updated review is provided of the literature on non-surgical management of the disease, mainly interventional pain management.
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45

Mazin, David A., and Mehul J. Desai. Lumbar Spinal Stenosis. Edited by Mehul J. Desai. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199350940.003.0016.

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Lumbar spinal stenosis (LSS) is a common, painful spine condition. It generally occurs in older patients who have degenerative changes in the lumbar spine. A careful history will aid in the appropriate diagnosis of LSS, although the physical examination findings may be nonspecific. While exact diagnostic criteria for LSS have not been agreed upon, radiologic and electrodiagnostic studies can help confirm the diagnosis. Treatment generally progresses from conservative to aggressive. High-quality evidence regarding definitive treatment options is lacking, so therapeutic options should be discussed on a case-by-case basis.
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46

Gunnar B.J. Andersson MD PhD and Thomas W. McNeill MD. Lumbar Spinal Stenosis. Mosby International, 1992.

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47

Rothwell, Peter M., and Seemant Chaturvedi. Carotid Artery Stenosis. Taylor & Francis Group, 2019.

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48

Coronary artery stenosis. New York: Elsevier, 1991.

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49

(Editor), Marek Szpalski, and Robert Gunzburg (Editor), eds. Lumbar Spinal Stenosis. Lippincott Williams & Wilkins, 2000.

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50

Ibrahim, Ashraf, and Talal Al-Malki. Congenital Esophageal Stenosis. Springer, 2019.

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