Relevant bibliographies by topics / Status epilepticu

Academic literature on the topic 'Status epilepticu'

Author: Grafiati
Published: 9 March 2023
Last updated: 10 March 2023

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Journal articles on the topic "Status epilepticu"

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Livingston, J. H., J. K. Brown, R. A. Harkness, and G. M. McCreanor. "CEREBROSPINAL FLUID NUCLEOTIDE METABOLITES FOLLOWING NON-CONVULSIVE STATUS EPILEPTICU." Developmental Medicine & Child Neurology 31, no. 2 (November 12, 2008): 168–73. http://dx.doi.org/10.1111/j.1469-8749.1989.tb03975.x.

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Di Bonaventura, C., F. Mari, N. Vanacore, J. Fattouch, A. Zarabla, A. Berardelli, M. Manfredi, M. Prencipe, and A. T. Giallonardo. "Status epilepticus in epileptic patients." Seizure 17, no. 6 (September 2008): 535–48. http://dx.doi.org/10.1016/j.seizure.2008.02.002.

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Mandei, Jose M., and Praevilia M. Salendu. "Tatalaksana Status Epileptikus Terkini pada Anak." e-CliniC 11, no. 1 (December 20, 2022): 146–56. http://dx.doi.org/10.35790/ecl.v11i1.44460.

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Absract: Status epilepticus is one of the most common pediatric neurologic emergencies in children with progressive benzodiazepine pharmaco-resistance due to neurotransmitter receptor disturbance. This has led to revisions of definitions and guidelines to emphasize early treatment and faster escalation. The initial management of the stabilization phase is followed by the administration of benzodiazepines as the first line. Second-line medications such as valproate, fosphenytoin, or levetiracetam, or phenobarbital are recommended, and at this point there is no clear evidence that one of these options is better than the other. If seizures persist after second-line drugs, refractory status epilepticus may be established. Treatment of refractory status epilepticus consists of bolus doses and continuous infusion titration with third-line drugs. In conclusion, potential therapeutic approaches for future study may require consideration of interventions that may speed the diagnosis and treatment of status epilepticus. Major advances in the clinical field with new definitions and classifications give the clinicians a better guidance on when to treat, how aggressively to treat, and how to avoid over- or under-treating the condition of status epilepticus. Keywords: status epilepticus; management; children Abstrak: Status epileptikus merupakan salah satu kedaruratan neurologis yang paling umum pada anak dengan farmakoresistensi benzodiazepine progresif karena gangguan reseptor neurotransmiter. Hal ini menyebabkan dilakukannya revisi definisi dan pedoman untuk menekankan pengobatan dini dan eskalasi yang lebih cepat. Tatalaksana awal fase stabilisasi dilanjutkan pemberian benzodiazepine sebagai lini pertama. Pengobatan lini kedua seperti valproate, fosphenytoin, atau levetiracetam, atau fenobarbital direkomendasikan, dan pada titik ini tidak ada bukti yang jelas bahwa salah satu dari opsi ini lebih baik daripada yang lain. Jika kejang berlanjut setelah obat lini kedua, status epileptikus refrakter dapat ditegakkan. Pengobatan status epileptikus refrakter terdiri dari dosis bolus dan titrasi infus kontinu dengan obat lini ketiga. Simpulan studi ini ialah pendekatan terapeutik potensial untuk studi masa depan mungkin memerlukan pertimbangan intervensi yang dapat mempercepat diagnosis dan pengobatan status epileptikus. Kemajuan besar dalam bidang klinis dengan definisi dan klasifikasi baru memberikan panduan yang lebih baik kepada dokter tentang kapan harus mengobati, seberapa agresif untuk mengobati, dan bagaimana menghindari pengobatan yang berlebihan atau kurang dari kondisi stastus epileptikus. Kata kunci: status epileptikus; tatalaksana; anak
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Poursadeghfard, Maryam, Zabihollah Hashemzehi, and Nahid Ashjazadeh. "Status Epilepticus in Adults: A 6-Year Retrospective Study." Galen Medical Journal 3, no. 3 (May 21, 2014): 153–59. http://dx.doi.org/10.31661/gmj.v3i3.200.

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Background: Status epilepticus (SE) is one of the most common neurological emergencies with high mortality and morbidity. This study aims to determine the most common causes and outcomes of SE in adult patients from Fars Province in southern Iran. Materials and Methods: 134 patients with SE (either convulsive or non-convulsive), admitted to Namazi Hospital, were enrolled from January 2006 to February 2012. We designed a questionnaire to collect the patients' demographics and disease characteristics and their outcomes according to Glasgow Outcome Scale (GOS). Statistical analyses were performed using SPSS software version 15. P-value<0.05 was considered statistically significant. Results: Seventy patients were female and 64 were male with mean age of 42.97±19.66 years and 39.42±18.89 years old, respectively. Sixty-two patients (46.26%) had a history of epilepsy, and 72 patients (53.73%) had no history of epilepsy. Antiepileptic drugs (ADEs) withdrawal and cerebral infarction were the most common causes of SE in epileptics and non-epileptics, respectively. One hundred and twenty-three patients (91.8%) had generalized tonic-clonic SE. Thirty patients (22.4%) could return to work and 33 (24.6%) died during hospitalization. There was a significant relationship between mortality and age over 60 years (P<0.001), and mortality and cerebral infarction or anoxia (P= 0.022). Conclusion: ADEs withdrawal in the epileptic patients was the main cause of SE (28.40% of the total population and 62% of the epileptic patients) that can be prevented by patient and family education. This study showed that high mortality and morbidity were significantly related to the increasing age and etiology of SE.
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Gainetdinova, Guzel R., and Tatiana V. Danilova. "Epidemiology, clinic and diagnosis of status epilepticus in adults: A review." Consilium Medicum 24, no. 11 (January 5, 2023): 805–10. http://dx.doi.org/10.26442/20751753.2022.11.201958.

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Status epilepticus is one of the causes of impaired consciousness. Status epilepticus can develop both in patients with epilepsy and in patients without an epileptic history. The most difficult diagnostic task is to identify non-convulsive status epilepticus. The most frequently discussed in the literature issues of the clinical picture, diagnosis of different variants of status epilepticus were analyzed. Particular attention was paid to the peculiarities of status epilepticus in patients of older age groups.
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Momcilovic-Kostadinovic, Dragana, Perisa Simonovic, Dusan Kolar, and Nebojsa Jovic. "Chlorpromazine-induced status epilepticus: A case report." Srpski arhiv za celokupno lekarstvo 141, no. 9-10 (2013): 667–70. http://dx.doi.org/10.2298/sarh1310667m.

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Introduction. It is largely known that some antipsychotic agents could have proconvulsive and proepileptogenic effects in some patients and could induce EEG abnormalities as well. However, the association of status epilepticus with certain antipsychotic drugs has been very rarely reported. Case Report. A case of an 18-year-old adolescent girl, with chlorpromazine therapy started for anxiety-phobic disorder was reported. Her personal history disclosed delayed psychomotor development. Shortly after the introduction of the neuroleptic chlorpromazine therapy in minimal daily dose (37.5 mg), she developed myoclonic status epilepticus, confirmed by the EEG records. Frequent, symmetrical bilateral myoclonic jerks and altered behavior were associated with bilateral epileptiform discharges of polyspikes and spike-wave complexes. This epileptic event lasted 3.5 hours and it was stopped by the parenteral administration of valproate and lorazepam; she was EEG monitored until stable remission. Status epilepticus as initial epileptic event induced by neuroleptic agent was not previously reported in our national literature. Conclusion. Introduction of chlorpromazine to a patient without history of seizures is associated with the evolution of an epileptic activity, including the occurrence of status epilepticus. Clinical evaluation of the risk factors possibly related to chlorpromazine-induced seizure is recommended in individual patients before administering this drug.
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Karantali, Eleni, Symela Chatzikonstantinou, Ioannis Mavroudis, Constantin Trus, and Dimitrios Kazis. "Cognitive Status Epilepticus: Two Case Reports." Medicina 57, no. 8 (August 3, 2021): 799. http://dx.doi.org/10.3390/medicina57080799.

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Cognitive status epilepticus is an uncommon form of focal status epilepticus presenting with a dysfunction of language, thinking or associated higher cortical functions. The absence of ictal manifestations can be misleading and delay a prompt diagnosis. Here we present two patients; one with amnesic and one with aphasic status epilepticus. Through these cases, we aim to highlight the value of EEG performance early in the diagnostic work-up and early antiepileptic drug initiation in cases where an epileptic disorder cannot be excluded.
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Hasan, H., Caner F. Demir, and Hasan S. Cura. "Absence status seen in an adult patient." Journal of Neurosciences in Rural Practice 04, no. 03 (July 2013): 342–44. http://dx.doi.org/10.4103/0976-3147.118809.

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ABSTRACTAbsence status epilepticus (ASE) is a type of nonconvulsive status epilepticus in which continuous or recurrent generalized epileptiform discharges are associated with a varying grade of consciousness impairment. Absence status epilepticus may be obtained during progress of many epileptic syndromes, in several metabolic disturbances and related to use of several drugs. Absence status epilepticus is generally seen in childhood; rarely it can be seen in adulthood. In this paper, the case which has never diagnosed until now in spite of many absence seizures for years, applied for absence seizures to our clinic and diagnosed for juvenile absence epilepsy, has been discussed.
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Assis, Telma M. R., Aroldo Bacellar, Gersonita Costa, and Osvaldo J. M. Nascimento. "Mortality predictors of epilepsy and epileptic seizures among hospitalized elderly." Arquivos de Neuro-Psiquiatria 73, no. 6 (June 2015): 510–15. http://dx.doi.org/10.1590/0004-282x20150043.

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Epilepsy and epileptic seizures are common brain disorders in the elderly and are associated with increased mortality that may be ascribed to the underlying disease or epilepsy-related causes.Objective To describe mortality predictors of epilepsy and epileptic seizures in elderly inpatients.Method Retrospective analysis was performed on hospitalized elderly who had epilepsy or epileptic seizures, from January 2009 to December 2010. One hundred and twenty patients were enrolled.Results The most common etiology was ischemic stroke (37%), followed by neoplasias (13%), hemorrhagic stroke (12%), dementias (11.4%) and metabolic disturbances (5.5%). In a univariate analysis, disease duration (p = 0.04), status epilepticus (p < 0.001) and metabolic etiology (p = 0.005) were associated with mortality. However after adjustment by logistic regression, only status epilepticus remained an independent predictor of death (odds ratio = 13; 95%CI = 2.3 to 72; p = 0.004).Conclusion In this study status epilepticus was an independent risk factor for death during hospitalization.
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Zimmermann, R., V. I. Hülsmeyer, C. Sauter-Louis, and A. Fischer. "Status Epilepticus and Epileptic Seizures in Dogs." Journal of Veterinary Internal Medicine 23, no. 5 (September 2009): 970–76. http://dx.doi.org/10.1111/j.1939-1676.2009.0368.x.

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Dissertations / Theses on the topic "Status epilepticu"

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GIOENI, DANIELA. "THE INFLUENCE OF ANESTHESIA IN THE CENTRAL NERVOUS SYSTEM STUDY." Doctoral thesis, Università degli Studi di Milano, 2020. http://hdl.handle.net/2434/708703.

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Central nervous system is a complex machine; it is constituted by more or less 172 billions of cells divided between neurons and glia. This incredible number of cells, particularly neurons, are constantly connected, working 24 hours a day, never stopping. Their activity is maintained even during particular conditions such as sleep, pathological unconsciousness (coma) or general anesthesia. For all these peculiarities, since ancient times, man has devoted much effort to the study of the most fascinating organ of living beings (mammals in particular). Nowadays, thanks to the advance in medical technology, many tools are available to “look inside the brain”. Magnetic resonance imaging and, particularly, functional magnetic resonance imaging (fMRI) is a modern biomedical imaging method, which allows a non-invasive assessment of brain function. The detection of brain activity is based on the coupling between neuronal activity, energy consumption, and blood flow. Functional connectivity (FC) of brain regions is modulated in various central nervous system diseases, during sleeping and general anesthesia. Anesthesia during MRI procedures is commonly used in preclinical setting and, sometimes, is required also in clinical setting (uncollaborative patients, children, drugs induced-coma etc). The study of the relationship between anesthetics and FC presents a double value: allows to distinguish the alterations induced by anesthesia on FC, avoiding possible confounding elements, and permits an in depth investigation of drugs behaviour. For all these reasons, the main topic of this PhD dissertation is the relationship between anesthesia and central nervous system. We started from theoretical studies in healthy subjects and we arrived to clinical setting, describing the possible application of anesthetics drugs as a treatment of neurologic diseases. The first study included aims to describe the way in which dexmedetomidine and isoflurane modulate FC in guinea pigs. We analysed the characteristic of cortical, subcortical and cortico- subcortical connectivity under both drugs with resting state fMRI. The second study presented partial results of a more complex work concerning FC in rats under 4 different anesthetics protocols. Because in FC studies blood flow represents a crucial element we dedicated a part of the work to the study of haemodynamic alterations through the administration 3 of contrast medium. Dynamic Suceptibility Contrast MRI analysis allows the study of cerebral blood flow and cerebral blood volume of different brain anatomic regions under dexmedetomidine, isoflurane, midazolam-dexmedetomidine and midazolam-isoflurane. Finally, we moved to clinical setting to describe the successful treatment of 3 dogs suffering from idiopathic epilepsy presented in emergency department in a state of super refractory status epilepticus. They were treated successfully with a continuous infusion of dexmedetomidine and ketamine. In conclusion, taken together this thesis gives a little contribute to better understand anesthetics behavior at brain level. We believe that it is important to make a “rational choice” when we decide the anesthetic protocol for neuroimaging procedures (both in clinical and preclinical setting) and this “rational choice” could be make only if we have a widespread literature that describe the highest number of anesthetic protocol and their interaction on central nervous system. Finally, thanks to the continuous improvement of our understanding of anesthetics mechanism of action, especially from a molecular and functional point of view, is it possible to use different anesthetics as a therapy for different neurologic conditions, particularly the one based on neurotransmitters imbalance.
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Inoue, Yushi. "Ring chromosome 20 and nonconvulsive status epilepticus : A new epileptic syndrome." Kyoto University, 1998. http://hdl.handle.net/2433/182253.

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Damasceno, Patrícia Gomes. "O uso da vigabatrina como droga de adição no controle de crises epilépticas neonatais." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/17/17140/tde-25042018-143839/.

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Introdução: A vigabatrina (VGB - Gama-Vinil-GABA) é um fármaco que eleva os níveis de GABA no organismo, por inibição irreversível da GABA transaminase, cuja eficácia foi bem demonstrada no controle dos espasmos epilépticos em lactentes, especialmente na síndrome de West secundária à esclerose tuberosa. Há escassez de estudos clínicos evidenciando um possível papel deste fármaco no controle de crises epilépticas neonatais e pouco se sabe sobre o potencial impacto do seu uso nessa faixa etária, seus possíveis efeitos adversos, ou se sua introdução teria associações positivas com controle mais adequado das crises na evolução e melhor desenvolvimento neuropsicomotor da criança. A VGB foi introduzida em nosso serviço como terapia de adição para o controle de crises neonatais refratárias, há vários anos, instigando nossa impressão sobre a eficácia deste medicamento no período neonatal. Objetivos: Avaliar a efetividade do uso da VGB como adjuvante no controle das crises eletrográficas e eletroclínicas do período neonatal e seus efeitos sobre o padrão do eletroencefalograma (EEG); Avaliar a evolução clínica e eletrográfica das crianças durante seguimento ambulatorial; Pesquisar associação entre \"controle de crises neonatais com introdução de VGB\" e diversas características demográficas, clínicas e evolutivas destes recém nascidos; Quantificar e caracterizar a ocorrência de efeitos adversos precoces e durante o seguimento. Pacientes e métodos: Estudo transversal retrospectivo, envolvendo o levantamento dos prontuários de uma amostra de recém-nascidos que receberam VGB como tratamento para crises neonatais refratárias aos fármacos convencionais e status epilepticus, no período de janeiro de 2007 a março de 2014, no Serviço de Neonatologia e Terapia Intensiva Neonatal do HCFMRP-USP, mantendo seguimento ambulatorial por pelo menos 1 ano. Foram avaliados os dados demográficos, etiologia e semiologia clínico-eletroencefalográfica das crises, esquema terapêutico prescrito, indicação da introdução da VGB, tempo de internação e tempo para atingir o controle das crises, evolução clínica e eletrencefalográfica durante a internação e no seguimento ambulatorial, época da suspensão da VGB, além de seus efeitos adversos. Resultados: De 48 recém-nascidos avaliados, 34 (79,2 %) obtiveram controle de crises eletrográficas e/ou clínicas durante o período neonatal, havendo melhora no padrão eletrográfico após a introdução da VGB em 79%. Quanto aos critérios para sua indicação, 33,3% (16 indivíduos) iniciaram VGB devido a falha terapêutica no controle das crises com fenobarbital e/ou fenitoína; 27,1% (13 recém nascidos), pela presença de estado de mal epilético e, em 12 crianças (25%), por falha terapêutica do midazolam. Ao final do primeiro ano de vida, a atividade de base do EEG mostrou-se desorganizada em 58,1% (18 de 29 pacientes que o realizaram aos 12 meses de vida). No seguimento ambulatorial de 38 pacientes, algum grau de atraso do desenvolvimento neuropsicomotor foi detectado em 20 crianças (52,6%); 19 lactentes (39,5%) mantiveram o uso da VGB em politerapia, tendo 22 crianças (57,9%) evoluído com persistência das crises epilépticas. Já 37,8% (14 pacientes) enquadraram-se em um padrão de encefalopatia epiléptica, que correspondeu à síndrome de West em 13,9% (5 de 36 crianças). Quanto ao EEG realizado em 34 crianças nessa fase, 17,6% (6 casos) demonstraram a presença de hipsarritmia, enquanto anormalidades focais ou multifocais foram detectadas em 50% (17 lactentes). A taxa de óbito ao final do primeiro ano foi de 23,3% (10 de 43 crianças analisadas quanto a este dado). Não foi possível comprovar déficit visual relacionado diretamente ao uso da VGB. A variável \"controle de crises no período neonatal com o uso da VGB\" foi associada aos seguintes desfechos clínicos favoráveis: melhora no padrão eletrográfico (92,1%), proporção menor de crianças evoluindo para síndrome de West e outras encefalopatias epilépticas (71,9% não tiveram tal desfecho); menor frequência de hipsarritmia no EEG (92,9% sem hipsarritmia), maior alcance de desenvolvimento neuropsicomotor normal (56,2% com bom desenvolvimento neurológico), menor índice de óbito neonatal (97,4% vivos nesta fase) e durante os primeiros doze meses de vida (87,9%). Conclusão: Acreditamos que a VGB seja uma opção terapêutica efetiva e com adequada relação custo-benefício, a ser implementada no controle de crises epilépticas neonatais refratárias como fármaco adjuvante aos convencionais. Entretanto, estudos randomizados e controlados são necessários para confirmar sua eficácia quando comparada a outros medicamentos disponíveis para uso nesta população, bem como para avaliar seus possíveis efeitos adversos a longo prazo.
Introduction: Vigabatrin (VGB - Gama-Vinil-GABA) is an antiepileptic drug which increases systemic GABA levels by irreversibly inhibiting GABA transaminase, with well demonstrated efficacy in the control of infantile epileptic spasms, specially related to West syndrome due to tuberous sclerosis. Clinical studies demonstrating a possible role of VGB in the control of neonatal seizures are still very scarce and very little is known on the impact of its use at this early age, as well as on its possible side effects or eventual positive associations from its use with more adequate seizure control or better neuropsychomotor development in the outcome. VGB has been used in our service as an add-on therapy for refractory neonatal seizures arising the impression that this could be an effective antiepileptic medication in the neonatal period. Objectives: To evaluate the use of VGB as an add-on medication regarding its effectiveness for the control of neonatal electrographic and electroclinical seizures, as well as its effects over the EEG pattern; To evaluate clinical and electrographic evolution of the children in follow-up; To estimate VGB efficacy on the control of neonatal seizures in relation to the demographical and clinical characteristics of those newborns; To quantify and characterize the occurrence of early and late side effects of this medication along follow-up. Patients and methods: This is a transverse retrospective study carried out through charts analysis from a sample of newborns who received VGB as add-on medication for seizures and/or status epilepticus refractory to conventional drugs, from January 2007 through March 2014, at the Neonatal Intensive Care Service of HCFMRP-USP, keeping follow-up in our institution for at least 1 year. Demographical and etiological data were analyzed, as well as clinical-electrographical semiology, VGB prescription indication, therapeutic schedule, time to reach seizure control, clinical and electrographical evolution while in hospital and at the follow-up, age at VGB withdrawal, besides adverse effects. Results: Among 48 newborns evaluated, 34 (79.2%) reached control of electrographic and/or clinical seizures during neonatal period, with improvement of the EEG pattern after VGB introduction in 79%. As for drug introduction criteria, 33.3% (16 children) were started on VGB due to therapeutic failure of phenobarbital and/or phenytoin; 27.1% (13 newborns), due to status epilepticus and, in 12 babies (25%), due to therapeutic failure of midazolam. By the end of the first year of life, EEG background activity was disorganized in 58.1% (18 out of 29 children who had EEG registered at 12 month of life). Along the one year follow-up of 38 patients, 20 infants (52.6%) showed some degree of neurodevelopmental delay; 19 children (39.5%) remained on VGB in polytherapy, with seizure persistence in 22 (57.9%). Evolution to an epileptic encephalopathy was found in 14 kids (37.8%), with West Syndrome being characterized in 13.9% (5 out of 36 kids). As for the EEG carried out in 34 children at the follow-up, 17.6% (6 cases) showed hypsarrhythmia while focal or multifocal abnormalities were seen in 50% (17 infants). Up to 12 month of life, the death rate was 23.3% (10 out of 43 children evaluated for such endpoint). Visual deficit directly related to VGB use could not be determined. The variable \"seizure control during the neonatal period after VGB use\" was associated to the following endpoints: improvement of the EEG pattern (92,1% of children with seizure control after VGB), lower proportion of children evolving into West syndrome and other epileptic encephalopathies (71.9% did not show such endpoint), lower frequency of hypsarrhythmia in the EEG (92.9% without hypsarrhythmia), better milestones reached regarding neuropsychomotor development (56.2% with good neurological outcome), lower rate of neonatal death (97.4% alive by the end of neonatal period) and along the first year of life (87.9%). Conclusion: VGB is an effective therapeutic option with adequate cost-benefit relationship which should be implemented for the control of refractory neonatal seizures as add-on therapy to conventional drugs. However, controlled randomized studies are necessary to confirm such efficacy as compared to other drugs available for use in the neonatal period, as well as to evaluate its possible long term side effects.
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Walker, M. C. "The treatment of status epilepticus." Thesis, University College London (University of London), 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.301305.

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Alotaibi, Khalid Nijr. "Convulsive status epilepticus in children." Thesis, University of Nottingham, 2017. http://eprints.nottingham.ac.uk/38829/.

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Convulsive status epilepticus (CSE) is an emergency condition associated with mortality and morbidity. It is commonly treated with antiepileptic drugs (AEDs), but these may cause serious adverse events and even death in children. Research on their effectiveness for CSE, and related adverse events in children remains limited. The primary aim of this research was thus to evaluate the effectiveness and safety of AEDs in treating acute tonic-clonic seizure including convulsive status epilepticus (CSE). Two systematic reviews and meta-analyses were conducted to address these aims. The first evaluated the effectiveness of AEDs in children with acute tonic-clonic seizures including (CSE). The second evaluated the safety of AEDs in this population. The systematic review of AED effectiveness identified 20 studies published between 1946 and April 2015. It showed that buccal midazolam was more effective than rectal diazepam for treating acute tonic-clonic seizures including CSE in children, and was associated with a lower recurrence rate. Lorazepam and diazepam were equally effective in terminating seizures, but for lorazepam, intravenous administration was more effective than the buccal, sublingual or intranasal routes. Intravenous valproate appeared to be more effective than intravenous phenytoin and phenobarbital; however, the difference was not significant. The systematic review of AED safety for children with acute tonic-clonic seizures identified 25 studies, published between 1946 and April 2015. These studies were predominantly randomised controlled trials and of these 19 studies reported more than one adverse event, while 6 reported none. A total of 203 adverse events were documented, most commonly respiratory depression (101 children), mainly after treatment with diazepam (46 children). The rates of respiratory depression with buccal midazolam and rectal diazepam were similar (3.0% and 3.3%, respectively). Compared to intravenous diazepam, intravenous lorazepam was associated with less respiratory depression. No child suffered respiratory depression associated with intravenous valproate treatment, compared to one child with intravenous phenobarbital. When looking at all adverse events, intravenous valproate was significantly safer than intravenous phenobarbital. Respiratory depression was not noted in children who received intravenous levetiracetam; however, all levetiracetam studies identified in this review were cohort and non-comparative. In conclusion, in the treatment of acute tonic-clonic seizures (including CSE), buccal was the best administration route for children admitted to the emergency department. Intravenous lorazepam treatment was associated with less respiratory depression than intravenous diazepam. Where IV access was practicable, intravenous lorazepam was the drug of choice. More randomised control trials are needed to evaluate the effectiveness and safety of AEDs as a second-line treatment.
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Lankinen, M. (Markus). "Ensihoitolääkäritoiminnan vaikutus status epilepticus -potilaiden hoitoon." Master's thesis, University of Oulu, 2018. http://jultika.oulu.fi/Record/nbnfioulu-201806132563.

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Tiivistelmä. Pitkittynyt epilepsiakohtaus on hengenvaarallinen tila, jonka hoito suositusten mukaisesti on kolmiportaista. Kolmiportaisen hoidon toteutumiselle oman haasteensa tuo lääkkeiden heikko säilyvyys, jolloin sairaalan ulkopuolella potilaita hoitavat ensihoitolääkärit eivät voi noudattaa hoitosuosituksia. Tämän tutkimuksen tarkoituksena oli selvittää, miten ensihoitolääkäreiden hoito sairaalan ulkopuolella vaikuttaa potilaiden hoitoon kokonaisuudessa Oulun yliopistollisen sairaalan alueella. Tutkimusryhmään kerättiin vuosilta 2011–2012 ensihoitolääkärin hoitamat potilaat, joiden luokse hätäkeskus oli lähettänyt ensihoitolääkärin pitkittyneen kouristuskohtauksen takia hälytyskoodilla 722B ja ensihoitolääkäri oli saattanut potilaan Oulun yliopistolliseen sairaalaan. Lisäksi huomioitiin muutamat verrokkiryhmästä löytyneet aluesairaalasta lääkärin saattamina yliopistolliseen sairaalaan saapuneet potilaat. Kokonaisuudessaan tutkimusryhmään päätyi 30 lääkärin saattamaa potilasta. Verrokkiryhmä koostui Oulun yliopistollisen sairaalan ensiapupoliklinikalla vuonna 2012 hoidetuista potilaista, joiden diagnoosiksi oli todettu ICD-10 järjestelmän mukaan G41 epileptinen sarjakohtaus. Verrokkiryhmään ei hyväksytty potilaita, jotka olivat tulleet sairaalaan lääkärin saattamina. Verrokkiryhmään päätyi 19 potilasta. Lapset rajattiin kokonaan tutkimuksen ulkopuolelle. Tutkimus on taannehtiva tapaus-verrokkitutkimus. Tärkeimpänä tutkimustuloksena todettiin ensihoitolääkärien hoitamilla potilailla vähemmän uusiutuneita kohtauksia. Lisäksi ensihoitolääkärin intuboimat potilaat saivat todennäköisemmin keuhkokuumeen kuin sairaalassa intuboidut. Sairaalassa intuboiduilla potilailla oli pitempi kokonaishoitoaika kuin ensihoitolääkärin intuboimilla tai ilman intubaatiota hoidetuilla potilailla.
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Othman, Jalal. "Der therapierefraktäre Status Epilepticus : Prädiktoren, Therapie, Prognose /." Berlin : Mensch-und-Buch-Verl, 2004. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=013087340&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.

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Zimmermann, Romina. "Status epilepticus und epileptische Anfälle beim Hund." Diss., lmu, 2010. http://nbn-resolving.de/urn:nbn:de:bvb:19-111701.

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Rußmann, Vera Franziska Agnes. "Untersuchungen in einem Post-Status epilepticus-Modell." Diss., lmu, 2012. http://nbn-resolving.de/urn:nbn:de:bvb:19-148708.

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Towne, Alan R. "Outcomes of Status Epilepticus in the Elderly." VCU Scholars Compass, 2007. http://hdl.handle.net/10156/2076.

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Books on the topic "Status epilepticu"

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Drislane, Frank W., ed. Status Epilepticus. Totowa, NJ: Humana Press, 2005. http://dx.doi.org/10.1385/1592599451.

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Drislane, Frank W., and Peter W. Kaplan MBBS, eds. Status Epilepticus. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-58200-9.

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Stefan, Hermann, and Jürgen Bauer. Status epilepticus. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-76027-3.

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Wang, Xuefeng, and Shichuo Li, eds. Refractory Status Epilepticus. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-5125-8.

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1951-, Kaplan Peter W., and Drislane Frank, eds. Nonconvulsive status epilepticus. New York: Demos Medical Pub., 2009.

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Frank, Drislane, ed. Status epilepticus: A clinical perspective. Totowa, N.J: Humana Press, 2005.

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G, Wasterlain Claude, and Treiman David M, eds. Status epilepticus: Mechanisms and management. Cambridge, Mass: MIT Press, 2006.

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Frank, Drislane, ed. Status epilepticus: A clinical perspective. Totowa, N.J: Humana Press, 2005.

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America, Epilepsy Foundation of, ed. The Legal rights of persons with epilepsy: An overview of legal issues and laws affecting persons with epilepsy. 6th ed. Landover, MD (4351 Garden City Dr., Landover 20785): Epilepsy Foundation of America, 1992.

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Status epilepticus: Its clinical features and treatment in children and adults. Cambridge: Cambridge University Press, 1994.

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Book chapters on the topic "Status epilepticu"

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Stefan, Hermann, and Jürgen Bauer. "Geschichtliche und klassifikatorische Übersicht zum Status epilepticus." In Status epilepticus, 1–6. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-76027-3_1.

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Stefan, Hermann, and Jürgen Bauer. "Pathologisch-anatomische Folgen und pathophysiologische Grundlagen des Status epilepticus." In Status epilepticus, 7–16. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-76027-3_2.

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Stefan, Hermann, and Jürgen Bauer. "Epileptische Syndrome und Status epileptici." In Status epilepticus, 17–19. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-76027-3_3.

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Stefan, Hermann, and Jürgen Bauer. "Diagnostische Untersuchungs-Methoden." In Status epilepticus, 20–29. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-76027-3_4.

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Stefan, Hermann, and Jürgen Bauer. "Symptome des Status epilepticus." In Status epilepticus, 30–141. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-76027-3_5.

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Stefan, Hermann, and Jürgen Bauer. "Allgemeine Aspekte der Diagnose und Therapie des Status epilepticus." In Status epilepticus, 142–59. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-76027-3_6.

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Stefan, Hermann, and Jürgen Bauer. "Differenzierte Folgetherapie nach Versagen von Benzodiazepinen und Phenytoin bei der Behandlung des Grand-mal-Status." In Status epilepticus, 160–66. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-76027-3_7.

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Stefan, Hermann, and Jürgen Bauer. "Übersicht therapierelevanter Antiepileptika zur Statusbehandlung." In Status epilepticus, 167–91. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-76027-3_8.

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Kaplan, Peter W., and Eugen Trinka. "History of Status Epilepticus." In Status Epilepticus, 3–8. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-58200-9_1.

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Fountain, Nathan B., and Suchitra Joshi. "Neuropathology of Generalized Convulsive Status Epilepticus." In Status Epilepticus, 123–30. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-58200-9_10.

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Conference papers on the topic "Status epilepticu"

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Santos, Maria do Carmo Vasconcelos, Mariana Moreira Soares de Sa, Emanuelle Ferreira Barreto, Aline Cursio Moraes, Roberta Kelly Netto Vinte Guimarães, and Antonio Pereira Gomes Neto. "Progressive myoclonic epilepsy: case report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.688.

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Context: Progressive myoclonic epilepsy (PMS) begins in childhood or during adolescence, being a heterogeneous group of symptomatic progressive progressive generalized epilepsy. Composed of cortical myoclonus, multiple epileptic seizures, delayed or regressed neuropsychomotor development and cerebellar manifestations. Genetics is heterogeneous with a similar clinical presentation, which makes etiological definition difficult. Report a clinical case of generalized epilepsy, myoclonus, cerebellar condition and severe mental impairment. Analysis of medical records of a patient at Santa Casa de Belo Horizonte. Case report: MVPP, 17 years old, previously healthy, adopted son, normal neuropsychomotor development, first generalized tonic-clonic seizure at 8 years old, recurrence at 12 years old, being initiated by Valproato and Clobazam. In 2018 there was a worsening of the crises, perceived myoclonus, added Lamotrigine and Oxcarbazepine. EEG with continuous diffuse epileptic activity of subclinical epilepticus status and unchanged skull MRI. In 2019 he started with gait ataxia, balance changes, dysarthria, dysmetria, cognitive decline, loss of functionality and refractoriness to treatment. Valproate reduced and oxcarbazepine suspended. Video- EEG with ictal pattern of generalized wave polyspicle. Deteriorated cerebellar condition with extensive propaedeutic without alterations. There was no feasibility of genetic testing at the time. Methylprednisolone pulse therapy with partial improvement. Unsuccessful attempt to levetiracetam due to psychotic symptom. He presented lowering of the sensorium, bronchoaspiration and orotracheal intubation. He evolved with myoclonic status, adjusted for anti-crisis drugs, midazolam, thiopental, tracheostomy and gastrostomy. He maintained super- refractory status, being opted for callosotomy. He died within weeks of the procedure. Conclusion: The early diagnosis of PMS is a challenge, and its evolution is usually debilitating, with a poor prognosis and scarce specific treatment. Whenever possible, a genetic study is needed to define an etiological diagnosis.
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Tan, Tracie HL, Piero Perucca, Patrick Kwan, Terence J. O’Brien, and Mastura Monif. "019 Differentiating status epilepticus from prolonged psychogenic non-epileptic seizures – can peripheral cell ratios help?" In ANZAN Annual Scientific Meeting 2021 Abstracts. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/bmjno-2021-anzan.19.

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Saragadam, S. D., J. Garza, T. Dang, S. Mahmood, A. Anwar, V. K. Babu, N. Panchagnula, M. K. Sidhu, A. Garcia Fernandez, and L. Oud. "Trends in Status Epilepticus-Related Mortality in the United States, 2010-2019." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a2458.

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U., Saraf U., Asranna A., Menon R. N., Radhakrishnan A., Manju P., Vibina V. P., Cherian A., and Thomas S. V. "Electroclinical Predictors of Cognitive and Seizure Outcome in Children with Epileptic Encephalopathy Due to Electrical Status Epilepticus in Sleep (ESES)." In 20th Joint Annual Conference of Indian Epilepsy Society and Indian Epilepsy Association. Thieme Medical and Scientific Publishers Private Ltd., 2018. http://dx.doi.org/10.1055/s-0039-1694859.

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Shen, Lei, Xinyi Geng, Huichun Luo, Jingying Wang, and Shouyan Wang. "Epileptic States Recognition Using Transfer Learning." In 2019 41st Annual International Conference of the IEEE Engineering in Medicine & Biology Society (EMBC). IEEE, 2019. http://dx.doi.org/10.1109/embc.2019.8857265.

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Deotale, Anuradha, Kalpana Kulkarni, and Nikunj Garia. "Thiopentone the most effective drug in status epilepticus." In 17th Annual Conference of Indian Society of Neuroanaesthesiology and Critical Care. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0038-1667586.

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Yadav, Sidharth, Suvasini Sharma, Bijoy Patra, Rajeev Malhotra, and Virendra Kumar. "Status Epilepticus in Pediatric Patients Severity Score (STEPSS): A Clinical Score to Predict the Outcome of Status Epilepticus in Children." In 20th Joint Annual Conference of Indian Epilepsy Society and Indian Epilepsy Association. Thieme Medical and Scientific Publishers Private Ltd., 2018. http://dx.doi.org/10.1055/s-0039-1694897.

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Iosifescu, S. M., and J. Wassermann. "Personality Changes and Status Epilepticus in an Undomiciled Patient." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6659.

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Murray, M., and A. Rabin. "Baclofen Overdose Associated with Prolonged Super-Refractory Status Epilepticus." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a1701.

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Borges, Karin, Marla Gearing, Bruce H. Wainer, Dayna L. McDermott, Amy B. Smith, and Raymond Dingledine. "OSTEOPONTIN EXPRESSION IN NEURONS AND ASTROGLIA AFTER STATUS EPILEPTICUS." In 3rd International Conference on Osteopontin and SIBLING (Small Integrin-Binding Ligand, N-linked Glycoprotein) Proteins, 2002. TheScientificWorld Ltd, 2002. http://dx.doi.org/10.1100/tsw.2002.250.

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Reports on the topic "Status epilepticu"

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Shujaa, Asaad Suliman, and Qasem Almulihi. The efficacy and safety of ketamine in treating refractory and super-refractory status epilepticus in pediatric and adult populations, A systemic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0011.

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Review question / Objective: This study is to assess the efficacy and safety of ketamine in treating refractory and super-refractory status epilepticus in pediatric and adult populations. Rationale: Refractory status epilepticus (RSE) is either generalized or complex partial status epilepticus (SE) that fails to respond to first and second-line therapies. Super refractory status epilepticus (SRSE) is SE that remains unresponsive despite 24 hours of therapy with general anesthesia [1, 2]. Both RSE and SRSE pose significant challenges for the managing intensivist. There exists a race against time for control of epileptic activity in the RSE/SRSE patient to preserve cortical function and reduce morbidity/mortality. However, despite the best intentions, and not uncommonly, standard frontline antiepileptic drugs (AEDs) fail to control or reduce seizure activity once seizures approach the 30-minute mark. The following review provides an analysis of ketamine in treating RSE/SRSE, focusing on the potential target population, dosing, concerns, and the role of early administration.
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Ballough, G. P., and M. G. Filbert. A Viable Neuroprotection Strategy Following Soman-induced Status Epilepticus. Fort Belvoir, VA: Defense Technical Information Center, December 2003. http://dx.doi.org/10.21236/ada443565.

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McDonough, John H., Kerry E. Van Shura, Megan E. Lyman, Claire G. Eisner, Amelia Mazza, Robert K. Kan, and Tsung-Ming Shih. Evaluation of ADD392124 for the Delayed Treatment of Nerve Agent-Induced Status Epilepticus Seizures. Fort Belvoir, VA: Defense Technical Information Center, September 2011. http://dx.doi.org/10.21236/ada555367.

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ALMULIHI, QASEM, FATIMAH ALMUHANNA, EMAN ALSULTAN, and MOHAMMED ALMUHANNA. Comparison of safety and effectiveness between levetiracetam and phenytoin in treatment Pediatric Status Epilepticus: A meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2021. http://dx.doi.org/10.37766/inplasy2021.10.0082.

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Xin, Wu, and Xue Tao. The efficacy and safety of neuromodulation in refractory epilepsy: a systematic review and network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0042.

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Review question / Objective: To assess the efficacy and safety of different neuromodulation applied to the refractory epilepsy and provide a better choice for clinical practice. Condition being studied: Epilepsy is a frequent neurologic illness defined by bursts of hypersynchronized neural network activity that afflict about 1% of the global population. Unfortunately, roughly 30% of people with drug-resistant epilepsy (DRE) continue to experience seizures despite three anti-seizure drugs. In most cases, resective surgery, as the first-line treatment for DRE, is considered a curative therapy for achieving long-term seizure-free status, but about half of patients are not candidates for surgery due to a variety of factors such as multiple/diffuse/widespread seizure foci, epileptic foci arising from eloquent, primary generalized epilepsy, or patients unwilling to undergo surgery. Neuromodulation, albeit palliative, is an important alternative treatment for these individuals to prevent or decrease ictal episodes, which can affect the nervous system in a variety of ways.
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