Relevant bibliographies by topics / Status asthmaticus

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Status asthmaticus'

Author: Grafiati
Published: 24 April 2022

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Journal articles on the topic "Status asthmaticus"

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Bell, Thomas. "EXTRACORPOREAL LIFE SUPPORT FOR STATUS ASTHMATICUS." Pediatrics 94, no. 2 (August 1, 1994): 261. http://dx.doi.org/10.1542/peds.94.2.261b.

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Purpose of the Study. This report presents the experience with one case of status asthmaticus who failed to respond to mechanical ventilation and was successfully managed with extracorporeal life support (ECLS) using venovenous bypass. The purpose is to inform the practitioner of an additional therapy, potentially of benefit, in management of asthma complicated by treatment-resistant respiratory failure. Methods. Low volume venovenous bypass with extracorporeal life support resolved severe respiratory failure in a 23-year-old female asthmatic over a 22-hour period after failure of 5 hours of mechanical ventilation. Bypass was initiated remotely by an ECLS team using a portable ECLS circuit before the 180-mile transport to the "nearby" center. Details of the technique are presented. Conclusion and Reviewer's Comments. This may be the first instance where asthma was the primary indication for ECLS; other asthmatics have been so treated, but for other complicating conditions like pneumonia and adult respiratory distress syndrome. This report provides a further alternative therapy, even in somewhat remote areas, for asthma-caused respiratory failure.
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Apter, Andrea, and Paul A. Greenberger. "Status Asthmaticus." Allergy and Asthma Proceedings 11, no. 4 (July 1, 1990): 168–69. http://dx.doi.org/10.2500/108854190778880097.

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Sonenthal, Kathy R., and Paul A. Greenberger. "Status Asthmaticus." Allergy and Asthma Proceedings 14, no. 2 (March 1, 1993): 125–26. http://dx.doi.org/10.2500/108854193778812125.

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Summer, Warren R. "Status Asthmaticus." Chest 87, no. 1 (January 1985): 87S—94S. http://dx.doi.org/10.1378/chest.87.1.87s.

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Summer, Warren R. "Status Asthmaticus." CHEST Journal 87, no. 1_Supplement (January 1, 1985): 87S. http://dx.doi.org/10.1378/chest.87.1_supplement.87s.

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Cohen, Neal H., Howard Eigen, and Thomas E. Shaughnessy. "STATUS ASTHMATICUS." Critical Care Clinics 13, no. 3 (July 1997): 459–76. http://dx.doi.org/10.1016/s0749-0704(05)70324-9.

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Partana, J. S. "Status Asthmaticus." Paediatrica Indonesiana 15, no. 9-10 (May 29, 2017): 269. http://dx.doi.org/10.14238/pi15.9-10.1975.269-72.

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The therapy of status asthmaticus must be rational. Thus it is important to evaluate: 1. the severity and duration of an asthmatic attack. 2. the degree of dehydration. 3. whether infection plays a role. 4. all medication previously administered. 5. any possible complication.Treatment is as follows :Fluid and electrolyte therapy is important not only for the correction of dehydration and electrolyte disturbances but also for preventing inspissation of mucus in the bronchi. The best route of fluid administration is intravenous.Potassium iodide orally administered may be helpful as an expectorant.After hydration and normal acid-base balance have been established, epinephrine may be of benefit.Aminophylline is effective when administered intravenously. It should be used with extreme caution: the dose should not exceed 3 mg per kg of body weight, it should be given slowly and should not be given more frequently than every 8 hours.Corticosteroids should be administered, especially in cases who have received suppressive doses previously.Humidified oxygen administration is of the utmost importance.Antibiotics are recommended when infection is suspected.Management of complications.
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Janson-Bjerklie, Susan. "Status Asthmaticus." American Journal of Nursing 90, no. 9 (September 1990): 52. http://dx.doi.org/10.2307/3463899.

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Janson - Bjerklie, Susan. "Status Asthmaticus." AJN, American Journal of Nursing 90, no. 9 (September 1990): 52–55. http://dx.doi.org/10.1097/00000446-199009000-00023.

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Zwerdling, Robert G. "Status Asthmaticus." Pediatric Annals 15, no. 2 (February 1, 1986): 105–10. http://dx.doi.org/10.3928/0090-4481-19860201-08.

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Dissertations / Theses on the topic "Status asthmaticus"

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Mondoñedo, Jarred R. "Anesthetic delivery system for treatment of status asthmaticus." Thesis, Boston University, 2013. https://hdl.handle.net/2144/21220.

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Thesis (M.Sc.Eng.) PLEASE NOTE: Boston University Libraries did not receive an Authorization To Manage form for this thesis or dissertation. It is therefore not openly accessible, though it may be available by request. If you are the author or principal advisor of this work and would like to request open access for it, please contact us at open-help@bu.edu. Thank you.
Status asthmaticus (SA) is a severe, acute exacerbation of asthma that is refractory to traditional therapies using standard bronchodilators such as β-agonists and corticosteroids. Inhaled volatile anesthetics are currently used as a rescue therapy for SA due to their potent bronchodilator effects. However, it is unclear whether these agents act in vivo via 1) direct action on airway smooth muscle (ASM); 2) systemic re-circulation; or 3) autonomic reflexes from the central nervous system. Treatment with these agents can also lead to negative side effects, notably hypotension and arrhythmias, especially during prolonged pediatric use. The goals of this thesis were to compare direct versus systemic effects of these inhaled anesthetic agents, and to determine whether sufficient bronchodilation can be achieved via direct diffusion from the airway lumen to the ASM. We designed and developed a computer-actuated, ventilator-valve system to control the serial composition of the inspired gas. Using this system, we delivered inhaled anesthetic agents either a) to the anatomic dead space selectively (direct), or b) continuously throughout inspiration (systemic) in three mongrel canines (20-25 kg) with methacholine-induced bronchoconstriction. Measurements of lung resistance (RL), elastance (EL), and anatomic dead space (VD) demonstrated that isoflurane and sevoflurane result in bronchodilation for both delivery regimes. This suggests that the mechanism of action for these agents is at least partly via direct effects. Fluctuations in VD were not directly coupled with those for RL or EL. Furthermore, there may exist a limit to maximal bronchodilation using inhaled anesthetics, with isoflurane being more potent. In summary, this study illustrates the feasibility of using a targeted anesthetic delivery to treat severe, acute bronchoconstriction. Such a delivery system has the potential to define a rapidly translatable treatment paradigm for SA while increasing patient safety.
2031-01-01
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Trenga, Carol A. "Dietary antioxidants and ozone-induced bronchial hyperresponsiveness in asthmatic adults /." Thesis, Connect to this title online; UW restricted, 1997. http://hdl.handle.net/1773/8462.

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Gaddam, Surender. "The impact of asthma self-management education programs on the health outcomes: A meta-analysis (systemic review) of randomized controlled trials." CSUSB ScholarWorks, 2003. https://scholarworks.lib.csusb.edu/etd-project/2312.

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An attempt has been made in this study to critically appraise, systematically review and gather together the results obtained in individual trials and examine the strength of evidence supporting the component for Education for a Partnership in Asthma Care of the National Asthma Education and Prevention Program (NAEPP) to test whether health outcomes are influenced by education and self-management programs.
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Haverkamp, Hans Christian. "Determinants of arterial blood gas status during exercise in habitually active asthmatics." 2004. http://catalog.hathitrust.org/api/volumes/oclc/61749365.html.

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Books on the topic "Status asthmaticus"

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Bogunović, Slobodan-Giša. Status asthmaticus: Novel. Beograd: NOVA219, 2009.

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Status asthmaticus / Status astmatikus. Belgrade: Nezavisna izdanja S. Mašića, 2009.

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Sentissi, Kinza, and Stephanie Yacoubian. Physiologic Airflow Disruption. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0017.

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Airflow disruption can be triggered through multiple mechanisms. The obstruction can stem from within the airway lumen, airway walls, or the tissues surrounding it. This section focuses on airflow disruption initiated by bronchospasm, obstructive lung disease, asthma and status asthmaticus. Bronchospasm presents with increased airway resistance secondary to airway hyperreactivity or anaphylaxis. Asthma and chronic obstructive pulmonary disease (COPD) are obstructive and inflammatory lung pathologies. Airflow disruption in asthma is reversible between exacerbations. The airway obstruction in COPD is not fully reversible. Status asthmaticus is the most severe presentation of asthma and can be life threatening. Poorly controlled obstructive lung disease can result in perioperative complications. Patients should therefore be medically optimized before undergoing operative procedures.
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McIndoe, Andrew. Anaesthetic emergencies. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198719410.003.0036.

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This chapter discusses anaesthetic emergencies. It begins with a description of adult basic life support and advanced life support. It goes on to describe the management of acute problems, including narrow and broad complex tachycardia, severe hypo- or hypertension, severe hypoxia, laryngospasm, air/gas embolism, gastric aspiration, status asthmaticus, pulmonary oedema, failed intubation, the cannot-intubate-cannot-ventilate scenario, malignant hyperthermia anaphylaxis, intra-arterial injection, and unsuccessful reversal of neuromuscular blockade. It concludes with the management of paediatric emergencies, including paediatric advanced life support, ventricular fibrillation or tachycardia, neonatal resuscitation, the collapsed septic child, paediatric major trauma, acute severe asthma, and anaphylaxis, as well as with a discussion of paediatric drug doses and equipment.
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McIndoe, Andrew. Anaesthetic emergencies. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198719410.003.0036_update_001.

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This chapter discusses anaesthetic emergencies. It begins with a description of adult basic life support and advanced life support. It goes on to describe the management of acute problems, including narrow and broad complex tachycardia, severe hypo- or hypertension, severe hypoxia, laryngospasm, air/gas embolism, gastric aspiration, status asthmaticus, pulmonary oedema, failed intubation, the cannot-intubate-cannot-ventilate scenario, malignant hyperthermia anaphylaxis, intra-arterial injection, and unsuccessful reversal of neuromuscular blockade. It concludes with the management of paediatric emergencies, including paediatric advanced life support, ventricular fibrillation or tachycardia, neonatal resuscitation, the collapsed septic child, paediatric major trauma, acute severe asthma, and anaphylaxis, as well as with a discussion of paediatric drug doses and equipment.
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Beydon, Laurent, and Flavie Duc. Inhalational anaesthetic agents in critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0046.

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Inhalational anaesthetic agents have limited applications in the intensive care unit (ICU), as their delivery requires specific equipment, which are not routinely available. Sevoflurane and isoflurane are the two agents eligible for this purpose. They both show good clinical tolerance and versatility, but may raise cerebral blood flow above 1 minimum alveolar concentration. This property makes them unsuitable for sedation in patients suffering from acute brain injury. Sevoflurane is known to be partly metabolized via the cytochrome pathway in inorganic fluoride. This latter accumulates in a dose- and time-dependent manner, especially in a closed circuit with soda lime. However, no clinical renal injury has been proven, despite several studies reporting on sevoflurane in ICUs. A fresh gas flow above 2 L/min is required to limit inorganic fluoride build-up. Halogenates have been proven to allow efficient sedation in ICU patients for up to several days. They may be considered as therapeutic agents especially in refractory status asthmaticus. Insufficient data exist to recommend halogenates to treat status epilepticus. Nitrous oxide, in 50% oxygen, may serve to allow sedation/analgesia for short and moderately procedures. Xenon, an inert gas that discloses anaesthetic properties with extremely fast onset and recovery, and also has no haemodynamic side effects remains confined to the operating theatre. It requires specific anaesthetic machines and is, at present, too expensive to represent a routine inhalational anaesthetic agent.
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Williams, Erin S. Asthmatic for Adenotonsillectomy. Edited by Erin S. Williams, Olutoyin A. Olutoye, Catherine P. Seipel, and Titilopemi A. O. Aina. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190678333.003.0005.

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Asthma is a chronic reversible pulmonary condition. It is the most common respiratory disease as it affects 6 million to 9 million children in the United States. The patient with asthma can experience reversible bronchoconstriction, airway inflammation, airway hyperresponsivness, and increased mucus production. Inflammation is the fundamental abnormality. This dynamic process exists on a spectrum of mild, moderate, or severe. Patients may exhibit expiratory wheezing, obstruction to expiration, and/or inspiration, cough, and respiratory distress. Given the prevalence of asthma and its potential for significant morbidity and mortality, it is important that the anesthesiologist be able to determine the severity of disease and prevent and/or treat bronchospasm.
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Parker, Philip M. The 2007-2012 Outlook for Bronchial Dilator and Anti-Asthmatic Pharmaceutical Preparations in the United States. ICON Group International, Inc., 2006.

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Frew, Anthony. Air pollution. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0341.

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Any public debate about air pollution starts with the premise that air pollution cannot be good for you, so we should have less of it. However, it is much more difficult to determine how much is dangerous, and even more difficult to decide how much we are willing to pay for improvements in measured air pollution. Recent UK estimates suggest that fine particulate pollution causes about 6500 deaths per year, although it is not clear how many years of life are lost as a result. Some deaths may just be brought forward by a few days or weeks, while others may be truly premature. Globally, household pollution from cooking fuels may cause up to two million premature deaths per year in the developing world. The hazards of black smoke air pollution have been known since antiquity. The first descriptions of deaths caused by air pollution are those recorded after the eruption of Vesuvius in ad 79. In modern times, the infamous smogs of the early twentieth century in Belgium and London were clearly shown to trigger deaths in people with chronic bronchitis and heart disease. In mechanistic terms, black smoke and sulphur dioxide generated from industrial processes and domestic coal burning cause airway inflammation, exacerbation of chronic bronchitis, and consequent heart failure. Epidemiological analysis has confirmed that the deaths included both those who were likely to have died soon anyway and those who might well have survived for months or years if the pollution event had not occurred. Clean air legislation has dramatically reduced the levels of these traditional pollutants in the West, although these pollutants are still important in China, and smoke from solid cooking fuel continues to take a heavy toll amongst women in less developed parts of the world. New forms of air pollution have emerged, principally due to the increase in motor vehicle traffic since the 1950s. The combination of fine particulates and ground-level ozone causes ‘summer smogs’ which intensify over cities during summer periods of high barometric pressure. In Los Angeles and Mexico City, ozone concentrations commonly reach levels which are associated with adverse respiratory effects in normal and asthmatic subjects. Ozone directly affects the airways, causing reduced inspiratory capacity. This effect is more marked in patients with asthma and is clinically important, since epidemiological studies have found linear associations between ozone concentrations and admission rates for asthma and related respiratory diseases. Ozone induces an acute neutrophilic inflammatory response in both human and animal airways, together with release of chemokines (e.g. interleukin 8 and growth-related oncogene-alpha). Nitrogen oxides have less direct effect on human airways, but they increase the response to allergen challenge in patients with atopic asthma. Nitrogen oxide exposure also increases the risk of becoming ill after exposure to influenza. Alveolar macrophages are less able to inactivate influenza viruses and this leads to an increased probability of infection after experimental exposure to influenza. In the last two decades, major concerns have been raised about the effects of fine particulates. An association between fine particulate levels and cardiovascular and respiratory mortality and morbidity was first reported in 1993 and has since been confirmed in several other countries. Globally, about 90% of airborne particles are formed naturally, from sea spray, dust storms, volcanoes, and burning grass and forests. Human activity accounts for about 10% of aerosols (in terms of mass). This comes from transport, power stations, and various industrial processes. Diesel exhaust is the principal source of fine particulate pollution in Europe, while sea spray is the principal source in California, and agricultural activity is a major contributor in inland areas of the US. Dust storms are important sources in the Sahara, the Middle East, and parts of China. The mechanism of adverse health effects remains unclear but, unlike the case for ozone and nitrogen oxides, there is no safe threshold for the health effects of particulates. Since the 1990s, tax measures aimed at reducing greenhouse gas emissions have led to a rapid rise in the proportion of new cars with diesel engines. In the UK, this rose from 4% in 1990 to one-third of new cars in 2004 while, in France, over half of new vehicles have diesel engines. Diesel exhaust particles may increase the risk of sensitization to airborne allergens and cause airways inflammation both in vitro and in vivo. Extensive epidemiological work has confirmed that there is an association between increased exposure to environmental fine particulates and death from cardiovascular causes. Various mechanisms have been proposed: cardiac rhythm disturbance seems the most likely at present. It has also been proposed that high numbers of ultrafine particles may cause alveolar inflammation which then exacerbates preexisting cardiac and pulmonary disease. In support of this hypothesis, the metal content of ultrafine particles induces oxidative stress when alveolar macrophages are exposed to particles in vitro. While this is a plausible mechanism, in epidemiological studies it is difficult to separate the effects of ultrafine particles from those of other traffic-related pollutants.
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Book chapters on the topic "Status asthmaticus"

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Larsen, Reinhard, and Thomas Ziegenfuß. "Status asthmaticus." In Pocket Guide Beatmung, 281–93. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-46219-5_26.

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Larsen, Reinhard, and Thomas Ziegenfuß. "Status asthmaticus." In Beatmung, 646–61. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-662-11228-1_23.

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Larsen, Reinhard, and Thomas Ziegenfuß. "Status asthmaticus." In Pocket Guide Beatmung, 283–95. Berlin, Heidelberg: Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-59657-9_26.

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Larsen, Reinhard, and Thomas Ziegenfuß. "Status asthmaticus." In Beatmung, 409–18. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-29662-8_24.

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Pariente, R. "Status Asthmaticus." In Highlights in Asthmology, 431–37. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-70316-4_49.

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Kashani, John, Richard D. Shih, Thomas H. Cogbill, David H. Jang, Lewis S. Nelson, Mitchell M. Levy, Margaret M. Parker, et al. "Status Asthmaticus." In Encyclopedia of Intensive Care Medicine, 2135. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_2227.

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Larsen, Reinhard, and Thomas Ziegenfuß. "Status asthmaticus." In Beatmung, 429–39. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-662-06009-4_23.

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Larsen, Reinhard, and Thomas Ziegenfuß. "Status asthmaticus." In Beatmung, 646–61. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-662-06010-0_23.

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Larsen, Reinhard, and Thomas Ziegenfuß. "Status asthmaticus." In Pocket Guide Beatmung, 273–85. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-53728-2_26.

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Larsen, Reinhard, and Thomas Ziegenfuß. "Status asthmaticus." In Beatmung, 481–92. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-662-54853-0_27.

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Conference papers on the topic "Status asthmaticus"

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El Husseini, I., and J. Sunderram. "Status Asthmaticus Leading to Fatal Herniation." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6632.

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Paskaradevan, J., L. M. Barber, and D. R. Spielberg. "Foreign Body Aspiration Presenting as Status Asthmaticus." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4952.

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Götschke, J., I. Schröder, M. Barnikel, J. Behr, T. Meis, W. v. Wulffen, N. Kneidinger, and K. Milger. "Omalizumab zur Durchbrechung eines Therapie-refraktären Status asthmaticus." In 60. Kongress der Deutschen Gesellschaft für Pneumologie und Beatmungsmedizin e. V. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1678035.

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Littlefield, L., S. Kagunye, L. Jo, and V. Balasubramanian. ""Saved by Sevo" - Sevoflurane in Refractory Adult Status Asthmaticus." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6128.

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Wasio, A., L. L. Sanchez Fernandez, M. N. Saintil, K. Anis, and H. Singh. "A Case of Status Asthmaticus Refractory to Conventional Treatment Strategies." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a4550.

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Emeruwa, I. O., and N. Qadir. "Status Asthmaticus Requiring Venovenous Extracorporeal Membrane Oxygenation in a Pregnant Woman." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a5198.

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Esangbedo, Ivie D., Matthew Wylie, and William Cutrer. "HIGH-FREQUENCY OSCILLATORY VENTILATION AS RESCUE THERAPY FOR NEAR-FATAL STATUS ASTHMATICUS." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a3065.

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George, J., V. Singh, Q. Abdelal, W. Wiese-Rometsch, and R. Loutfi. "Life Threatening Vapor: A Case of Status Asthmaticus Requiring ECMO After Vaping." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a1848.

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Carroll, Christopher L., Laurie Karamessinis, Kathleen Sala, Yen-Shin D. Tang, Philip C. Spinella, and Aaron R. Zucker. "Respiratory Failure In Children With Status Asthmaticus: A Review Of The Virtual PICU Database." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a6279.

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Bickel, S. G., R. L. Morton, A. Truman, A. R. O'Hagan, and N. S. Eid. "Objective Discharge Criteria Results in 48% Lower Readmission Rates for Children with Status Asthmaticus." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a7182.

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