Relevant bibliographies by topics / Statistics (Vital), France

Academic literature on the topic 'Statistics (Vital), France'

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Published: 26 July 2024
Last updated: 27 July 2024

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Journal articles on the topic "Statistics (Vital), France"

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HOWELL, EMBRY M., and PAUL VERT. "In Reply: International Statistics—Incomparable." Pediatrics 92, no. 4 (October 1, 1993): 637. http://dx.doi.org/10.1542/peds.92.4.637a.

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Dr Sepkowitz makes several correct points about the French vital registration system that leads to incomparability with Michigan vital statistics. • Definitions of fetal deaths are dissimilar. The French registration system ignores fetal deaths at less than 28 weeks gestation. • In France, it is the responsibility of parents to register births. • France has had a unique category of "deaths before registration." However, he is incorrect in suggesting that these differences affected the results reported in our article. It is precisely for these reasons that we did not use data from the French vital registration system. Instead we used data that were carefully collected during a 1-year period in Lorraine's hospitals under the direction of the French Health Ministry that directly controls French hospitals.
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Hodgson, Dennis. "Disciplining Statistics: Demography and Vital Statistics in France and England, 1830–1885." Contemporary Sociology: A Journal of Reviews 37, no. 1 (January 2008): 52–53. http://dx.doi.org/10.1177/009430610803700129.

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Blum, A. "Libby Schweber: Disciplining Statistics. Demography and Vital Statistics in France and England, 1830-1885." European Sociological Review 25, no. 4 (November 25, 2008): 517–18. http://dx.doi.org/10.1093/esr/jcn012.

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Haan, Michael. "Disciplining Statistics: Demography and Vital Statistics in France and England, 1830-1885 (review)." Histoire sociale/Social history 41, no. 81 (2008): 302–4. http://dx.doi.org/10.1353/his.0.0016.

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Mooney, Graham. "Disciplining Statistics: Demography and Vital Statistics in France and England, 1830-1885, by Libby Schweber." Victorian Studies 50, no. 2 (January 2008): 346–48. http://dx.doi.org/10.2979/vic.2008.50.2.346.

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Berlivet, Luc. "Libby Schweber Disciplining statistics: Demography and vital statistics in France and England, 1830-1885 Durham, Duke University Press, 2006, 277 p." Annales. Histoire, Sciences Sociales 65, no. 5 (October 2010): 1286–87. http://dx.doi.org/10.1017/s0395264900037355.

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Connelly, Matthew. "Disciplining Statistics: Demography and Vital Statistics in France and England, 1830–1885. By Libby Schweber. Durham, N.C.: Duke University Press, 2006. Pp. 277. $23.95." American Journal of Sociology 115, no. 1 (July 2009): 267–69. http://dx.doi.org/10.1086/605738.

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Ziade, N., E. Jougla, and J. Coste. "Population-Level Impact of Osteoporotic Fractures on Mortality and Trends Over Time: A Nationwide Analysis of Vital Statistics for France, 1968-2004." American Journal of Epidemiology 172, no. 8 (August 12, 2010): 942–51. http://dx.doi.org/10.1093/aje/kwq215.

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Rusnock, A. "LIBBY SCHWEBER. Disciplining Statistics: Demography and Vital Statistics in France and England, 1830-1885. (Politics, History, and Culture.) Durham, N.C.: Duke University Press. 2006. Pp. 277. Cloth $84.95, paper $23.95." American Historical Review 112, no. 5 (December 1, 2007): 1600–1601. http://dx.doi.org/10.1086/ahr.112.5.1600.

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Hagoel, Lea. "Reviews: Education: Libby Schweber, Disciplining Statistics, Demography and Vital Statistics in France and England, 1830—1885. Durham, NC and London: Duke University Press, 2006, 277 pp., ISBN 0822338149, US$23.95." International Sociology 23, no. 5 (September 2008): 757–60. http://dx.doi.org/10.1177/02685809080230050803.

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Books on the topic "Statistics (Vital), France"

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Boeda, Michel. La France fait ses comptes =: France, the vital statistics. Paris: Economica, 1989.

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2

Vallin, Jacques. Les causes de décès en France de 1925 à 1978. [Paris]: Presses universitaires de France, 1987.

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France, Meslé, ed. Les causes de décès en France de 1925 à 1978. Paris: Institut national d'études démographiques, 1988.

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Vallin, Jacques. Les causes de décès en France de 1925 à 1943. [Paris]: Presses universitaires de France, 1986.

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Vallin, Jacques. Les causes de décès en France de 1925 à 1949. [Paris]: Presses universitaires de France, 1987.

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Vallin, Jacques. Les causes de décès en France de 1925 à 1967. [Paris]: Presses universitaires de France, 1987.

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Vallin, Jacques. Les causes de décès en France de 1925 à 1978. Reclassement par catégories étiologiques et anatomiques. [Paris]: Presses universitaires de France, 1987.

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Christophe, Bergouignan, ed. La population de la France: Évolutions démographiques depuis 1946. Pessac: CUDEP, 2005.

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Geneviève, Gille, and Weill Georges J, eds. Les Archives de l'Ile-de-France: Guide des recherches. [France]: Services d'archives de Paris, de Seine-et-Marne, des Yvelines, de l'Essonne, des Hauts-de-Seine, de Seine-Saint-Denis, du Val-de-Marne et du Val-d'Oise, 1989.

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Adams, Julia, Libby Schweber, and George Steinmetz. Disciplining Statistics: Demography and Vital Statistics in France and England, 1830-1885. Duke University Press, 2006.

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Book chapters on the topic "Statistics (Vital), France"

1

Ouellette, Nadine, France Meslé, Jacques Vallin, and Jean-Marie Robine. "Supercentenarians and Semi-supercentenarians in France." In Demographic Research Monographs, 105–23. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-49970-9_9.

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AbstractThe purpose of this study is twofold. Firstly, it attempts to exhaustively identify cases of French supercentenarians and semi-supercentenarians and to validate their alleged age at death. Secondly, it seeks to uncover careful patterns and trends in probabilities of death and life expectancy at very old ages in France. We use three sets of data with varying degrees of accuracy and coverage: nominative transcripts from the RNIPP (Répertoire national d’identification des personnes physiques), death records from the vital statistics system, and “public” lists of individual supercentenarians. The RNIPP stands out as the most reliable source. Based on all deaths registered in the RNIPP at the alleged ages of 110+ for extinct cohorts born between 1883 and 1901, errors are only few, at least for individuals who were born and died in France. For alleged semi-supercentenarians, age validation on a very large sample shows that errors are extremely rare, suggesting the RNIPP data can be used without any verification until age 108 at the minimum. Moreover, a comparison with “public” lists of individual supercentenarians reveals a single missing occurrence only in the RNIPP transcripts since 1991. While the quality of vital statistics data remains quite deficient at very old ages compared to RNIPP, the analytical results show a significant improvement over time at younger old ages. Our RNIPP-based probabilities of death for females appear to level-off at 0.5 between ages 108 and 111, but data becomes too scarce afterwards to assess the trend. Also, we obtain a quite low life expectancy value of 1.2 years at age 108.
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"France." In Statistical Papers - United Nations (Ser. A), Population and Vital Statistics Report, 433–36. UN, 2013. http://dx.doi.org/10.18356/50c7961e-en.

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"France." In Statistical Papers - United Nations (Ser. A), Population and Vital Statistics Report, 266–67. UN, 2013. http://dx.doi.org/10.18356/3d78a3f0-en.

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Obladen, Michael. "Theirs is the kingdom of heaven." In Oxford Textbook of the Newborn, edited by Michael Obladen, 383–90. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198854807.003.0055.

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For many centuries, one in three newborns did not survive up to their first birthday, and details and causes of their death were unknown. Pioneers of vital statistics were Graunt and Petty in Britain, Süßmilch in Prussia, Berg in Sweden, Quetelet in Belgium, Villermé in France, Bodio in Italy, and Woodbury in the US. Since 1860, halfway reliable records were available, and infant mortality, at that time ranging from 100 to 300 per 1000 live births, was perceived as a humanitarian and political problem. Variables identified as associated with infant mortality included artificial feeding, poor sanitation, parental social and marital status, male sex, ethnicity, prematurity, and others, although their precise mechanisms often remained unclear. Since 1860, a dramatic and sustained decline in infant mortality caused a demographic revolution. Recent infant mortality rates ranged from 2 to 12 per 1000 live births in developed countries. Most of this reduction was achieved through public health measures and improved sanitation. Only since prematurity became the major cause of mortality could medicine claim to have enabled most of this progress.
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Porter, Theodore M. "Statistical Law and Human Freedom." In The Rise of Statistical Thinking, 1820-1900, 159–203. Princeton University Press, 2020. http://dx.doi.org/10.23943/princeton/9780691208428.003.0007.

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This chapter evaluates the criticism of statistics. Already in the early nineteenth century, the statistical approach was attacked on the ground that mere statistical tables cannot demonstrate causality, or that mathematical probability presupposes the occurrence of events wholly by chance. The intent of these early critics was not to suggest the inadequacy of causal laws in social science, but to reject the scientific validity of statistics. The new interpretation of statistics that emerged during the 1860s and 1870s was tied to a view of society in which variation was seen as much more vital. Statistical determinism became untenable precisely when social thinkers who used numbers became unwilling to overlook the diversity of the component individuals in society, and hence denied that regularities in the collective society could justify any particular conclusions about its members. These social discussions on natural science and philosophy bore fruit in the growing interest in the analysis of variation evinced by the late-century mathematical statisticians. To be sure, Francis Galton gave little attention to the debates on human freedom, but Francis Edgeworth was closely familiar with them, and Wilhelm Lexis's important work on dispersion can only be understood in the context of this tradition.
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T. Owolabi, Abiola, Taiwo Abideen Lasisi, and Christianah Folasade Olanrewaju. "Mitigating the Effects of COVID-19 through Vaccination: Evaluating Leading Countries across Continents of the World." In New Topics in Vaccine Development [Working Title]. IntechOpen, 2023. http://dx.doi.org/10.5772/intechopen.113950.

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This research investigates COVID-19 vaccine efficacy across six countries: India, South Africa, France, Australia, the USA, and Brazil, examining their impact on reducing deaths and containing the virus. It analyzes vital epidemiological metrics during pre-vaccination and post-vaccination periods until February 5, 2023. The countries are grouped by their Aridity Index, reflecting climate variations. Employing Pearson correlation, the study explores the relationship between the Aridity Index and vaccination period rates, noting some moderate associations but lacking statistical significance at a 5% level. Comparing case fatality and infection rates before and during vaccination showed no significant differences. However, incidence rates displayed a notable discrepancy at the 5% significance level. The study underscores the need for non-pharmaceutical measures alongside vaccination efforts to mitigate the increase in incidence and infection rates. It emphasizes that while COVID-19 vaccinations play a crucial role, complementary measures remain essential in effectively managing the pandemic. Overall, this research offers critical insights into vaccine efficacy across diverse countries, advocating a continued multi-faceted approach to combat the global health crisis.
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Bandazheuski, Yuri, and Nataliia Dubovaya. "ELEMENTS OF ETIO-PATHOGENESIS OF HYPERHOMOCYSTEINEMIA IN CHILDREN LIVING IN THE REGIONS AFFECTED FROM THE ACCIDENT AT THE CHERNOBYL NUCLEAR POWER PLANT." In The scientific paradigm in the context of technological development and social change. Publishing House “Baltija Publishing”, 2023. http://dx.doi.org/10.30525/978-9934-26-297-5-25.

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The prevalence of hyperhomocysteinemia in the population of children living in the area affected by the accident at the Chernobyl Nuclear Power Plant (ChNPP) requires a deep study of the etio-pathogenesis of metabolic disorders of the sulfur-containing amino acids methionine (Met) and homocysteine (Hcy) under radiation exposure. The purpose of the study was to assess the involvement of genetic (folate cycle polymorphisms – FC) and environmental factors in the occurrence of hyperhomocysteinemia in boys and girls living in the Ivankovsky and Polessky districts of the Kyiv region of Ukraine, near the Chernobyl Exclusion Zone (ChEZ). The methodology of the study is based on the evaluation by statistical methods of the results of genetic and laboratory examinations of 690 children (368 girls and 322 boys), aged 8-17 years, obtained in the course of projects of the European Commission, the Regional Council of Rhone-Alpes (France) and the French public organization "Children Chernobyl". It has been shown that in most cases, the violation of Hcy metabolism and the occurrence of hyperhomocystinemia in children from areas affected by the Chernobyl accident are caused by the association of genotypes with risk alleles of MTHFR:C677T and MTRR:A66G polymorphisms. Given the wide prevalence in the population, combinations of their heterozygous forms are of the greatest importance.The risk allele G of the MTRR:66 polymorphism also has a negative effect on the processes of Hcy methylation when associated with the risk allele G of the MTR:A2756G polymorphism and with compound heterozygosity A/CMTHFR:1298 – C/TMTHFR:677. In the body of boys, compared with the body of girls, combinations of risk alleles for FC polymorphisms are manifested by a more pronounced disturbance of Hcy metabolism. The A/AMTRR:66 genotype promotes Hcy utilization in the transsulfuration cycle, even if only one C allele of the MTHFR:677 polymorphism functions.An external environmental factor in the form of radioactive agents incorporated into vital organs, undermining cellular energy, has a negative impact on the processes of cobalamin methylation. The consequence of this is the occurrence of a state of hyperhomocysteinemia in more than 50 % of boys and girls who do not have risk alleles of MTR:A2756G, MTHFR:C677T and MTRR:A66G polymorphisms in the genome. Conclusions. The main internal cause of hyperhomocysteinemia in children living near the ChEZ is the association of risk alleles for FC genetic polymorphisms.The combined effect of endogenous (genetic mutations of FC) and exogenous (radioactive elements, their decay products, substances formed during the combustion of wood) factors leads to disruption of the Hcymethylation process and the emergence of a state of hyperhomocysteinemia in children living in the territory affected by the Chernobyl accident. This type of metabolic disorder can be considered a distant consequence of the Chernobyl accident. Further research should be aimed at developing measures for the prevention and treatment of hyperhomocysteinemia, as a condition associated with the occurrence of serious pathological processes.
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