Relevant bibliographies by topics / Starvation

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Starvation'

Author: Grafiati
Published: 4 June 2021
Last updated: 31 July 2024

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Journal articles on the topic "Starvation"

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Soler, C., P. Claquin, M. Goutx, O. Ragueneau, and B. Moriceau. "Impact of nutrient starvation on the biochemical composition of the marine diatom <i>Thalassiosira weissflogii</i>: from the whole cell to the frustule fraction." Biogeosciences Discussions 7, no. 4 (August 13, 2010): 5953–95. http://dx.doi.org/10.5194/bgd-7-5953-2010.

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Abstract. Interactions between carbon and silica in the diatom frustule play an important role in carbon export through their impact on diatom remineralization (carbon degradation and biogenic silica dissolution). To ameliorate model prediction of the fate of Si and organic matter during sedimentation, there is a need to first understand the origin and nature of Si-OC interactions, their impact on diatom remineralization and their variability with environmental conditions. In this study we focus on the impact of nutrient starvations on the formation and nature of these interactions in an ubiquitous diatom, Thalassiosira weissflogii. Fluorescence reveals the strong impact of all starvations on diatom metabolism while Fourier transformed infrared (FTIR) spectroscopy clearly showed that starvations altered the composition of the different diatom fractions. The relative compositions of whole cells were almost not impacted by starvations except Si(OH)4 starvation that slightly increased proteins relative contribution while decreasing carbohydrate. Starvation impacts became obvious looking at the composition of the different part of the diatom. The relative biochemical composition of the organic coating, protecting the frustule from the environment, was strongly affected by starvation. Under nitrate starvation, carbohydrate contribution increased while protein contribution decreased. Inversely, phosphate starvation increased the proportion of proteins and decreased carbohydrates contribution. Starvations also modified the different frustule phases. bSiO2 contribution decreased in the less reactive phase under silicate and phosphate starvation whereas nitrate starvation rather increased carbohydrate and protein pools. Phosphate starvation also led to an important shift of dominance among protein groups between amide I and amide II which compounds are suspected to play a key role in the frustule synthesis and architecture. Nutrient starvations affected the relative biochemical composition of diatom frustule fractions and organic coating which could imply a strong impact on frustule structure and architecture but also on frustule mechanical and chemical resistance.
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Fukatu, Kazuhiko. "Gut starvation." Japanese Journal of SURGICAL METABOLISM and NUTRITION 48, no. 5 (2014): 197–98. http://dx.doi.org/10.11638/jssmn.48.5_197.

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Bobrovnikova, Lidia A., Maria S. Pakholkova, Roman A. Sidorov, and Maria A. Sinetova. "Starch and triacylglycerol accumulation in the cells of the stain Chlorella sp. IPPAS C-1210." Issues of modern algology (Вопросы современной альгологии), no. 2(26) (2021): 1–7. http://dx.doi.org/10.33624/2311-0147-2021-2(26)-1-7.

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Strain Сhlorella sp. IPPAS C-1210 is an effective lipid and triacilglycerols (TAG) producer. The strain could be used eventually in such industries as bioenergetics, food industry and agriculture. The objective of this work was investigation of conditions in which the strain Сhlorella sp. IPPAS C-1210 accumulates the most starch and TAG in cells with a view to optimise its growth and productivity. The following cultivation parameters were investigated in order to figure out their influence on accumulation of starch and TAG: nitrogen- and phosphorous-starvation and cultivation on media with different nitrogen (nitrate, urea) and carbon (carbon dioxide, bicarbonate) sources. Pigments, starch, protein and lipid content in cells were measured. The exclusion of nitrogen or phosphorus source from medium decreased the biomass productivity significantly, caused chlorosis and reduction of protein content. Total lipid content increased slightly after phosphorous starvation and stayed almost constant under nitrogen starvation, however a greater TAG increase was observed during nitrogen starvation. Both nitrogen and phosphorous starvations caused the increase of the amount of reserve carbohydrates: during phosphorous starvation increase was insignificant, whereas the latter almost doubled the amount of reserve carbohydrates. The highest biomass and lipid productivity was observed in cells grown in bicarbonate supplement medium and the highest starch productivity was observed in cells grown in standard BBM-3N medium.
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Kuo, Macus, Helen Chen, Lynn Feun, and Niramol Savaraj. "Targeting the Proline–Glutamine–Asparagine–Arginine Metabolic Axis in Amino Acid Starvation Cancer Therapy." Pharmaceuticals 14, no. 1 (January 18, 2021): 72. http://dx.doi.org/10.3390/ph14010072.

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Proline, glutamine, asparagine, and arginine are conditionally non-essential amino acids that can be produced in our body. However, they are essential for the growth of highly proliferative cells such as cancers. Many cancers express reduced levels of these amino acids and thus require import from the environment. Meanwhile, the biosynthesis of these amino acids is inter-connected but can be intervened individually through the inhibition of key enzymes of the biosynthesis of these amino acids, resulting in amino acid starvation and cell death. Amino acid starvation strategies have been in various stages of clinical applications. Targeting asparagine using asparaginase has been approved for treating acute lymphoblastic leukemia. Targeting glutamine and arginine starvations are in various stages of clinical trials, and targeting proline starvation is in preclinical development. The most important obstacle of these therapies is drug resistance, which is mostly due to reactivation of the key enzymes involved in biosynthesis of the targeted amino acids and reprogramming of compensatory survival pathways via transcriptional, epigenetic, and post-translational mechanisms. Here, we review the interactive regulatory mechanisms that control cellular levels of these amino acids for amino acid starvation therapy and how drug resistance is evolved underlying treatment failure.
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Ahsan, Chowdhury Rafiqul, Farah Shamma, Nazmul Ahsan, and Moutusee Jubaida Islam. "Environmental Factors Regulate the hlyE Gene Expression in Both S. typhi and E. coli in a Similar Way to Display Haemolytic Activity." Bangladesh Medical Research Council Bulletin 42, no. 1 (March 29, 2017): 33–38. http://dx.doi.org/10.3329/bmrcb.v42i1.32001.

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Haemolysin (HlyE) is an essential virulence factor of Salmonella, Escherichia coli and other enteric bacteria. Although, a substantial degree of haemolytic activity is not seen under normal culture conditions in these organisms, however, the non-haemolytic E. coli K-12 showed significant haemolytic activity under stress conditions. To confirm this phenomenon in other enteric bacteria, in this study, the production of haemolysin in Salmonella enterica serovar Typhi under stress conditions, like oxygen and glucose starvations in vitro was investigated during March-December 2015. For this, S. typhi was cultured under oxygen or glucose starvation condition separately and this organism showed high haemolytic activity. The activity was found to be much higher when both the conditions were applied together. Also, the role of the transcription factor SlyA of S. typhi was investigated on induction of haemolytic activity. When E. coli K-12 was transformed with plasmid containing the gene of SlyA, the recombinant bacteria without any starvation condition, also showed similar haemolytic activity that was exhibited by S. typhi grown under oxygen and glucose starvation conditions. All these findings suggest that both environmental factors like oxygen or glucose starvation and overexpression of the transcription factor SlyA have important role in inducing hlyE gene expression in S. typhi.
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Kueper, Janina, Shaul Beyth, Meir Liebergall, Leon Kaplan, and Josh E. Schroeder. "Evidence for the Adverse Effect of Starvation on Bone Quality: A Review of the Literature." International Journal of Endocrinology 2015 (2015): 1–7. http://dx.doi.org/10.1155/2015/628740.

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Malnutrition and starvation’s possible adverse impacts on bone health and bone quality first came into the spotlight after the horrors of the Holocaust and the ghettos of World War II. Famine and food restrictions led to a mean caloric intake of 200–800 calories a day in the ghettos and concentration camps, resulting in catabolysis and starvation of the inhabitants and prisoners. Severely increased risks of fracture, poor bone mineral density, and decreased cortical strength were noted in several case series and descriptive reports addressing the medical issues of these individuals. A severe effect of severely diminished food intake and frequently concomitant calcium- and Vitamin D deficiencies was subsequently proven in both animal models and the most common cause of starvation in developed countries is anorexia nervosa. This review attempts to summarize the literature available on the impact of the metabolic response to Starvation on overall bone health and bone quality.
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Thounthong, P., B. Davat, S. Rael, and P. Sethakul. "Fuel starvation." IEEE Industry Applications Magazine 15, no. 4 (July 2009): 52–59. http://dx.doi.org/10.1109/mias.2009.932604.

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Frise, Charlotte J., and Lucy Mackillop. "Starvation ketoacidosis." Journal of the Intensive Care Society 17, no. 4 (October 25, 2016): 356. http://dx.doi.org/10.1177/1751143716644462.

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Yates, Darran. "Signalling starvation." Nature Reviews Neuroscience 14, no. 10 (August 29, 2013): 670–71. http://dx.doi.org/10.1038/nrn3592.

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Albano, Caterina. "Questioning starvation." Women's Writing 8, no. 2 (July 1, 2001): 313–26. http://dx.doi.org/10.1080/09699080100200172.

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Dissertations / Theses on the topic "Starvation"

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Robertson, Fiona E. "Starvation-survival in Escherichia coli." Thesis, University of Warwick, 1996. http://wrap.warwick.ac.uk/63636/.

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The population dynamics of carbon-starved E. coli K12 cultures was investigated. It was found that less cell lysis occurred when cells were previously grown in low glucose concentrations. Exponential-phase cells grown previously in 0.05% (w/v) glucose had survival rates comparable with their stationary-phase counterparts, suggesting that the rate of growth is more important in determining the outcome of starvation than the phase of batch culture growth. Long-termstarved cells (18-24 months) showed very little protein, DNA and RNA synthesis. Methionine was shown to alter the de novo synthesis protein profiles of longterm- starved cells and growth was seen to occur in the presence of methionine. This suggests that radio-labelling of proteins with 35S-methionine in these cells should be interpreted with care as the cells have been subjected to a nutrient upshift. Radio-labelling of proteins with 3H-leucine did not have the same effect. The ATP content of cells during prolonged incubation was shown to decrease in the first 48 hours incubation, increase until 5-7 days incubation then decrease after 7-8 days. After 13 days a slow, steady increase occurred. The ATP content of cells incubated for 16 days was higher than that of 48 hour-incubated cells. The physiology of long-term-starved cells was investigated with respect to their permeability to routine bacteriological stains ( e.g. DAPI, saffranin, Geimsa) and it was found that very few of these dyes were able to penetrate the cells, indicating that a decrease in cell permeability may be an important factor in survival as is seen in endospores of Bacillus species and swarmer cells of Rhodomicrobium vannielii and Caulobacter crescentus. Resistance of long-term starved cells to heat and biocide challenge was increased in comparison with exponential- and short-term (48 hour) stationary-phase cells and the resistance to biocides was shown to be retained through subsequent generations. Examination of the nucleoids of long-term-starved cells revealed that a more condensed form was present in cultures incubated for over 14 days, suggesting that dehydration of the DNA had occurred, similar to the situation found in endospores of Bacillus species and suggestive of dormancy. Analysis of outer-membrane proteins and lipopolysaccharide of long-term-starved cells showed that alterations occurred to the surface of the cells and it was demonstrated that hydrophobicity changes occurred. Hydrophobicity reached a maximum after 48 hours incubation then subsequently declined between days 2 and 3 which corresponded with an increase in cell numbers. Cell surface hydrophobicity was shown to be a potential method for separating heterogeneous, carbon-starved populations into homogeneous subpopulations. The data suggest that E. coli produces a dormant survival cell type which is morphologically and physiologically distinct from the parent cell.
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Dwivedi, Padmanabh. "Carbohydrate starvation and plant respiration." Thesis, University of Cambridge, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.624182.

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Price-Bishop, G. P., and Phillip R. Scheuerman. "Effects of Starvation on Bacteria." Digital Commons @ East Tennessee State University, 1992. https://dc.etsu.edu/etsu-works/2891.

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Howard, Jane Katherine. "Leptin, starvation and the immune system." Thesis, Imperial College London, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.396338.

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Poursaitidis, I. "Identification of differential nutrient starvation responses." Thesis, University of Liverpool, 2018. http://livrepository.liverpool.ac.uk/3019213/.

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To promote survival and proliferation cancer cells re-programme their metabolism, altering both uptake and utilisation of extracellular nutrients. To examine correspondences between nutrient supply and viability, in order to identify targetable requirements, I individually depleted amino acid nutrients from diploid Human Mammary Epithelial (HME) isogenic cells expressing commonly activated oncogenes. Cystine deprivation was found to induce massive-oxidative stress associated-cell death of HME cells expressing an activated epidermal growth factor receptor (EGFR). Cell death occurred via an iron-dependent mode, known as ferroptosis associated with increased generation of reactive oxygen species and lipid peroxidation. Pharmacological inhibition of EGFR or mitogen-activated protein kinase/extracellular regulated kinase (MAPK/ERK) signalling was found to block ferroptosis and ROS production and was associated with increased expression of glutathione peroxidase 4 (GPX4). Suppression of GPX4 expression in wild-type or gefitinib-treated HME-EGFR cells was sufficient to sensitise cells to ferroptosis. Importantly, MAPK signals were also important in suppressing cell-cell contact and communication that was found to provide an essential line of defence against ferroptosis induction and spread. In this way, microscopic observation of ferroptosis in wild-type HME cells identified a cell death spread phenotype that is consistent with necrosis phenotypes observed in ischemic models. Inhibition of ROS generation and lipid peroxidation effectively blocked the progression of necrosis indicating that counteracting lipid peroxidation might be beneficial for degenerative conditions where lipid peroxidation is evident. Additional therapeutic application of my findings was modelled using non-small lung cancer cell (NSCLC) lines with overactive ERK signalling. These cells were found to be sensitive to ferroptosis following deprivation of cystine in vitro as well as in vivo where in systemic deprivation of cystine was achieved in xenografted mice following administration of a cystine-degrading enzyme. Taken together, my results show that the presence of common oncogenic mutations can render cells sensitive to the depletion of a specific nutrient, and further suggest potentially novel anti-cancer therapies based on the inability of some MAPK-driven cancer cells to overcome oxidative stress following nutrient depletion, as well as therapies to limit the spreading phenotype of ferroptosis in cells associated with other diseases such as Alzheimer’s Disease, or that occur in normal cells in response to ischemic reperfusion and acute kidney injuries.
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PORCILE, GAETANO. "Subaqueous sand dunes and sediment starvation." Doctoral thesis, Università degli studi di Genova, 2019. http://hdl.handle.net/11567/941829.

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Thomas, Beth Elene Armstrong. "Regulation of phosphate starvation response in Arabidopsis." Texas A&M University, 2006. http://hdl.handle.net/1969.1/5029.

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Phosphate is an essential but limited macronutrient for all plants. In response to limited levels of phosphate, plants have developed highly specialized developmental, biochemical, and molecular responses. To further expand our knowledge of the phosphate starvation induced signal transduction pathway in plants, the expression of the phosphate starvation inducible Purple Acid Phosphatase 1 (PAP1) gene was studied in transgenic Arabidopsis. While few components have been identified regulating gene expression under phosphate starvation conditions in plants, one cis regulatory element recognized by the MYB transcriptions factor Phosphate Starvation Response 1 (PHR1) has been identified in many phosphate starvation induced (PSI) genes. PAP1 and many other genes examined during the course of the mutant characterization contain this cis element. Using the GUS reporter gene under control of the PAP1 promoter, a mutant screen was devised for plants showing abnormal PAP1 response to phosphate nutrition. Three mutant lines were identified and subsequently characterized for the phosphate starvation-induced signal-transduction pathway in Arabidopsis. Two mutants, BT1 and BT2, both with dominant mutations, showed increased GUS staining. The mutations in BT1 and BT2 are tightly linked to the transgene and to each other, but complementation analysis suggested that they are in different genes. Characterization of these mutants indicated that the PSI genes PAP1 and At4 (in BT1 roots), and RNS1 (in BT2 leaves) have alternative or additional methods of regulation other than PHR, even though these genes all contain PHR1 binding sites. A third mutant, BT3, had a phenotype similar to the PAP1 null-mutant and did not show PAP1 phosphatase activity under normal soil-grown conditions. Characterization of BT3 indicates that PAP1, RNS1, and AtIPS1 are not exclusively regulated by PHR1. In an attempt to map the BT3 mutant in a Columbia background by crossing with Landsberg erecta (Ler), it was discovered that the Ler ecotype does not show PAP1 phosphatase activity under normal soil-grown conditions. The PAP1 phosphatase regulatory trait, named BT5, was mapped to a 15,562 bp-region area containing only two genes between the GPA1 and ER markers on Chromosome 2.
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Purdon, Scott Drummond. "Starvation survival response of sulphate-reducing bacteria." Thesis, University of Aberdeen, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.340595.

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This thesis aims to investigate how SRB endure long periods of nutrient deprivation in the oligotrophic conditions of the North Sea. The presence of small cells in the marine environment has been extensively documented. These small cells are termed ultramicrobacteria, and are defined as being less than 0.3 μm in diameter. The formation of small cells by SRB was postulated to facilitate penetration of SRB deep within oil reservoirs, during water injection, exacerbating SRB associated problems. These studies revealed that a maximum of 15% of starving SRB populations formed UMB. Cultures starved for up to 6 years did not demonstrate an increase in UMB formation. Cell size studies revealed that SRB demonstrated a maximum 62% cell size decrease during starvation. Total cell counts revealed a constant cell number throughout starvation studies indicating a decrease in cell size by cell dwarfing. Transmission electron microscopy revealed a decrease in cellular content during starvation. This is consistent with a decrease in cell diameter during starvation. There was no difference in cell size decrease when cells were starved in the presence or absence of sulphate. There appeared, however, to be enhanced recoverability of cells starved in the presence of sulphate. SRB were demonstrated to be able to withstand simultaneous periods of sulphate and carbon starvation. This may have serious consequences for the oil industry as sulphate is often limiting in oil reservoirs. This evidence suggests that SRB could endure such conditions and recover when sulphate becomes available. SRB appear to enter a dormant phase shortly after the onset of starvation. Metabolic studies indicated that the entry into starvation was characterised by an initial increase in metabolic activity followed by a sharp decrease in metabolic activity to negligible levels. Metabolic activity could be re-initiated following inoculation into fresh growth medium.
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Bishop, G. P., and Phillip R. Scheuerman. "Physiological Changes in Bacteria During Starvation Stress." Digital Commons @ East Tennessee State University, 1990. https://dc.etsu.edu/etsu-works/2889.

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Bishop, G. P., and Phillip R. Scheuerman. "Physiological Changes in Bacteria During Starvation Stress." Digital Commons @ East Tennessee State University, 1991. https://dc.etsu.edu/etsu-works/2890.

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Books on the topic "Starvation"

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Pronzini, Bill. Starvation camp. London: Hale, 1985.

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Copyright Paperback Collection (Library of Congress), ed. Starvation camp. New York: Berkley Books, 2001.

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Pronzini, Bill. Starvation camp. Waterville, Me: Thorndike Press, 2003.

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Pronzini, Bill. Starvation camp. Cedar Rapids, Iowa: Mystery Scene Books, 1994.

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Gruber, Frank. Fort starvation. Leicester: Linford, 1992.

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Kjelleberg, Staffan, ed. Starvation in Bacteria. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4899-2439-1.

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Staffan, Kjelleberg, ed. Starvation in bacteria. New York: Plenum Press, 1993.

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Hawkins, Nigel. The starvation blockades. Barnsley, South Yorkshire: Leo Cooper, 2002.

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Hammel, Eric M. Guadalcanal: Starvation island. New York: Crown Publishers, 1987.

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Hammel, Eric M. Guadalcanal: Starvation island. Pacifica, Calif: Pacifica Press, 1992.

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Book chapters on the topic "Starvation"

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Weissman, Charles, and Rawhi Hashem. "Starvation." In Surgical Metabolism, 95–129. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39781-4_5.

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Weik, Martin H. "starvation." In Computer Science and Communications Dictionary, 1656. Boston, MA: Springer US, 2000. http://dx.doi.org/10.1007/1-4020-0613-6_18144.

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Weissman, Charles, and Rawhi Hashem. "Starvation." In Surgical Metabolism, 71–96. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1121-9_4.

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Madea, Burkhard, and Elke Doberentz. "Starvation." In Forensic and Legal Medicine, 478–507. Boca Raton: CRC Press, 2023. http://dx.doi.org/10.1201/9781003138754-56.

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Leung, Alexander K. C., Cham Pion Kao, Andrew L. Wong, Alexander K. C. Leung, Thomas Kolter, Ute Schepers, Konrad Sandhoff, et al. "Starvation." In Encyclopedia of Molecular Mechanisms of Disease, 1979. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_6168.

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Lopp, Michael. "Information Starvation." In Managing Humans, 75–79. Berkeley, CA: Apress, 2016. http://dx.doi.org/10.1007/978-1-4842-2158-7_12.

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Lopp, Michael. "Information Starvation." In Managing Humans, 69–73. Berkeley, CA: Apress, 2012. http://dx.doi.org/10.1007/978-1-4302-4315-1_11.

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von der Lippe, George B., and Viktoria M. Reck-Malleczewen. "Starvation (Fames)." In A History of the Münster Anabaptists, 137–52. New York: Palgrave Macmillan US, 2008. http://dx.doi.org/10.1057/9780230612563_8.

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Lopp, Michael. "Information Starvation." In Managing Humans, 75–79. Berkeley, CA: Apress, 2021. http://dx.doi.org/10.1007/978-1-4842-7116-2_12.

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Hanson, A. H. "Towards Starvation?" In Planning and the Politicians, 252–58. London: Routledge, 2022. http://dx.doi.org/10.4324/9781003259787-22.

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Conference papers on the topic "Starvation"

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Posch, Daniel, Christian Kreuzberger, Benjamin Rainer, and Hermann Hellwagner. "Client starvation." In the 1st international conference. New York, New York, USA: ACM Press, 2014. http://dx.doi.org/10.1145/2660129.2660162.

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White, A., M. Meyer, and K. Jones. "HC-130 fuel starvation testing." In 1999 IEEE Aerospace Conference. Proceedings (Cat. No.99TH8403). IEEE, 1999. http://dx.doi.org/10.1109/aero.1999.790220.

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Gerard, Mathias, Jean-Philippe Poirot-Crouvezier, Daniel Hissel, and Marie-Cecile Pe´ra. "Oxygen Starvation Effects on PEMFC Durability." In ASME 2010 8th International Conference on Fuel Cell Science, Engineering and Technology. ASMEDC, 2010. http://dx.doi.org/10.1115/fuelcell2010-33173.

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The problem of oxygen starvation on PEMFC is often observed (partially or totally in a cell). It is caused by water management problems (water droplet in the channels) or air management faults (delay during peak power). In our previous work, the current distribution has been measured along the cell during air starvation and calculated by modeling; the effects of the local temperature and the hygrometry in the MEA have been identified. However the mechanisms of degradation are still not completely understood. Several points have to be investigated like the short and long dated changes in performances and the degradation mechanisms in such a condition. In this paper, the study is completed by two sets of experiments. The first one is carried out with a bi-cell stack developed with specific design to compute the local current density by measuring the local induced magnetic field. Secondly, a long time ageing test is run with a 6 cells stack (220 cm2) during oxygen starvation cycling conditions. Both tests (with characterizations) coupled with the 2D serpentine meshing stack model (taking into account transport phenomena, heat transfer and semi-empirical electrochemical reactions) provide information on the MEA local conditions effects during oxygen starvation. Especially the different reaction mechanisms at the cathode side are explained likewise the consequences on ageing.
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Warrier, Ajit, Jeongki Min, and Injong Rhee. "Mitigating Starvation in Wireless Sensor Networks." In MILCOM 2006. IEEE, 2006. http://dx.doi.org/10.1109/milcom.2006.301993.

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Kai, Cai Hong, and Soung Chang Liew. "Temporal Starvation in CSMA Wireless Networks." In ICC 2011 - 2011 IEEE International Conference on Communications. IEEE, 2011. http://dx.doi.org/10.1109/icc.2011.5963320.

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Poplawski Ma, Laura, Samuel Nelson, Gregory Lauer, and Stephen Zabele. "ASAP: Preventing Starvation in Backpressure Forwarding." In MILCOM 2018 - IEEE Military Communications Conference. IEEE, 2018. http://dx.doi.org/10.1109/milcom.2018.8599753.

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Negreanu, Lorina. "Modeling Non-starvation in Multi-agent Systems." In 2015 20th International Conference on Control Systems and Computer Science (CSCS). IEEE, 2015. http://dx.doi.org/10.1109/cscs.2015.46.

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Ronasi, Keivan, Sathish Gopalakrishnan, and Vincent W. S. Wong. "Flow Starvation Mitigation for Wireless Mesh Networks." In 2009 IEEE Wireless Communications and Networking Conference. IEEE, 2009. http://dx.doi.org/10.1109/wcnc.2009.4917728.

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Park, Jiyeon, Bongjae Kim, and Yookun Cho. "A DCH starvation DoS attack in UMTS." In the 2012 ACM Research in Applied Computation Symposium. New York, New York, USA: ACM Press, 2012. http://dx.doi.org/10.1145/2401603.2401677.

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Wong, Yu-Shiang, Der-Jiunn Deng, Yang-Sheng Chen, and Jiann-Liang Chen. "On Alleviating Starvation in Wireless Sensor Networks." In ICC 2011 - 2011 IEEE International Conference on Communications. IEEE, 2011. http://dx.doi.org/10.1109/icc.2011.5963194.

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Reports on the topic "Starvation"

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Mason, Gerald A. Operation Starvation. Fort Belvoir, VA: Defense Technical Information Center, February 2002. http://dx.doi.org/10.21236/ada420650.

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Costa, Dora. Overweight Grandsons and Grandfathers' Starvation Exposure. Cambridge, MA: National Bureau of Economic Research, October 2022. http://dx.doi.org/10.3386/w30599.

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3

Whitmyer, Emma. North Korean embassy closures point to struggle and starvation. East Asia Forum, December 2023. http://dx.doi.org/10.59425/eabc.1702029648.

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Sinno, Abdulkader. Afghans stave off starvation in the face of economic sanctions. East Asia Forum, September 2023. http://dx.doi.org/10.59425/eabc.1693692037.

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Matin, A., T. Schmidt, and D. Caldwell. Role of starvation genes in the survival of deep subsurface bacterial communities. Final report. Office of Scientific and Technical Information (OSTI), November 1998. http://dx.doi.org/10.2172/674896.

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Gikandi, Levi. COVID-19 and Vulnerable, Hardworking Kenyans: Why it's time for a strong social protection plan. Oxfam, Kenya Red Cross Society, Concern Worldwide, ACTED, IMPACT Initiatives, The Centre for Rights, Education and Awareness (CREAW), Wangu Kanja Foundation, November 2020. http://dx.doi.org/10.21201/2020.6591.

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Seven NGOs, the Kenyan government, the European Union and the Danish and German governments are working together to implement a ’Safety Nets’ programme targeting Kenya’s millions of informal workers. With rising food insecurity and sexual and gender-based-violence, mounting job losses, poor access to water and sanitation, and a lack of formal safety nets, the Kenyan informal sector has suffered the brunt of the COVID-19 pandemic. The Safety Nets programme has revealed that cash transfers which support the most vulnerable people, and are implemented safely, transparently and accountably, have the potential to help vulnerable households stave off starvation, infection and eviction. They can also help reduce the vulnerability of survivors and those at risk of sexual and gender-based violence. The results of this programme demonstrate that nascent Kenyan ‘social protection’ programmes should be 1) immediately extended and expanded to the many vulnerable Kenyans currently not enrolled in any social protection programme; and 2) strengthened long-term to make them more effective, sustainable and accountable.
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Raghothama, Kashchandra G., Avner Silber, and Avraham Levy. Biotechnology approaches to enhance phosphorus acquisition of tomato plants. United States Department of Agriculture, January 2006. http://dx.doi.org/10.32747/2006.7586546.bard.

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Abstract: Phosphorus is one of the least available macronutrient in the soil. The high affinity phosphate transporters are known to be associated with phosphate acquisition under natural conditions. Due to unique interactions of phosphate with soil particles, up to 80% of the applied phosphates may be fixed forcing the farmers to apply 4 to 5 times the fertilizers necessary for crop production. Efficient uptake and utilization of this essential nutrient is essential for sustainability and profitability of agriculture. Many predictions point to utilization/exhaustion of high quality phosphate rocks within this century. This calls for efforts to improve the ability of plants to acquire and utilize limiting sources of phosphate in the rhizosphere. Two important molecular and biochemical components associated with phosphate efficiency are phosphate transporters and phosphatases. This research project is aimed at defining molecular determinants of phosphate acquisition and utilization in addition to generating phosphate uptake efficient plants. The main objectives of the project were; Creation and analysis of transgenic tomato plants over-expressing phosphatases and transporters Characterization of the recently identified members (LePT3 and LePT4) of the Pi transporter family Generate molecular tools to study genetic responses of plants to Pi deficiency During the project period we have successfully identified and characterized a novel phosphate transporter associated with mycorrhizal symbiosis. The expression of this transporter increases with mycorrhizal symbiosis. A thorough characterization of mutant tomato lacking the expression of this gene revealed the biological significance of LePT3 and another novel gene LePT4. In addition we have isolated and characterized several phosphate starvation induced genes from tomato using a combination of differential and subtractive mRNA hybridization techniques. One of the genes, LePS2 belongs to the family of phospho-protein phosphatase. The functionality of the recombinant protein was determined using synthetic phosphor-peptides. Over expression of this gene in tomato resulted in significant changes in growth, delay in flowering and senescence. It is anticipated that phospho-protein phosphatase may have regulatory role in phosphate deficiency responses of plants. In addition a novel phosphate starvation induced glycerol 3-phosphate permease gene family was also characterized. Two doctoral research students are continuing the characterization and functional analysis of these genes. Over expression of high affinity phosphate transporters in tobacco showed increased phosphate content under hydroponic conditions. There is growing evidence suggesting that high affinity phosphate transporters are crucial for phosphate acquisition even under phosphate sufficiency conditions. This project has helped train several postdoctoral fellows and graduate students. Further analysis of transgenic plants expressing phosphatases and transporters will not only reveal the biological function of the targeted genes but also result in phosphate uptake and utilization efficient plants.
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Carter, Becky, and Luke Kelly. Social Inequalities and Famine and Severe Food Insecurity Risk. Institute of Development Studies (IDS), June 2021. http://dx.doi.org/10.19088/k4d.2021.097.

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This rapid review summarises the evidence on the ways in which social inequalities and discrimination affect the risk of famine or severe food insecurity. Looking at the risk at the national and sub-national level, gender and other horizontal inequities can affect a society’s risk of violent conflict and therefore food insecurity, while fragile livelihoods associated with ethnic marginalisation can impact regional food security. At the individual and household level, there is a lack of disaggregated data on people’s social characteristics and famines. There is a broader literature on the impact of systemic discrimination (based on gender, age, disability, sexuality, and ethnic identity) on individuals’ and households’ livelihoods and assets, thereby increasing their vulnerability to food insecurity. A key finding from the literature is the gender gap, with women more at risk of being food insecure than men. Also, some ethnic groups are highly vulnerable particularly in conflict-related famines; starvation is used as a warfare tactic in political and ethnic conflicts. There is evidence of how social inequalities heighten individuals’ risks during food crises and famines, including through exposure to protection threats, while limiting their access to essential services and humanitarian assistance. A broad range of measures seeks to address the multi-dimensional ways in which social inequalities affect vulnerability and resilience to food insecurity.
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Velychko, Zoriana, and Roman Sotnyk. LINGUISTIC PRESENTATION AND TERMINOLOGICAL ASPECTS OF THE HOLODOMOR OF THE 1920s AND 1930s. Ivan Franko National University of Lviv, March 2024. http://dx.doi.org/10.30970/vjo.2024.54-55.12166.

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The article reveals and analyses a wide range of terms for the Holodomor of the 1920s and 1930s in Ukraine. The main objectives of the study are to find out the peculiarities of the linguistic presentation of the Holodomor phenomenon in scientific, popular science, and journalistic discourses, and to reveal semantic differences in the use of various terms for the Holodomor used in different languages. The main methodological bases of the study are linguistic analysis, socio-cultural method, qualitative content analysis, comparative method, etc. The method of retrospection must be used to substantiate the hypothesis. Thus, the reasons for the formation of the semantic contours of the terms “Holodomor”, “Famine”, “Great Famine”, “Terror by Famine”, “Big Hunger”, etc. were clarified. At the same time, the semantic nuances of word use are identified. As a conclusion, the authors substantiate the fundamental importance of using the term “Holodomor-genocide” in scientific circulation as the one that most accurately represents the essence of the historical phenomenon of the Holodomor. Based on the analysis of the documents, the content of the term “genocide” is formulated. It is explained that the Holodomor is genocide of the Ukrainian people, just as the Holocaust is genocide of the Jewish people. The authors prove the anti-Ukrainian orientation of the consistent and deliberate policy of Stalin and his followers against the Ukrainian nation, which culminated in the murder by starvation. These research findings are significant not only for the development of Ukrainian terminology or international terminology. They are also of great importance for modern politics, political science and historiography, and jurisprudence, especially in the context of a new genocide – the Russian Federation’s full-scale war of aggression against Ukraine. Keywords: Holodomor; genocide; Ukraine; Stalin’s terror; terminology.
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Hoy, Sarah, John Vucetich, and Rolf Peterson. Ecological Studies of Wolves on Isle Royale. Michigan Technological University, April 2023. http://dx.doi.org/10.37099/mtu.dc.wolf-annualreports/2022-2023.

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In February 2023, the wolf population was likely composed of 31 wolves. This is a slight increase from last year’s estimate of 28 wolves (Fig. 1). The wolf population includes an East Pack with 11 wolves, a West Pack with five wolves, three groups of at least three wolves each, and several other wolves that are either loners or only loosely affiliated with one of the smaller groups. There is evidence that pups were born into three, possibly four, litters in April 2022. For context, no litters are thought to have been born between 2015 and 2018. However, following the translocation of wolves to Isle Royale between 2018 and 2019, one litter was born in 2019, two litters were born in both 2020 and 2021, and possibly four litters were born in 2022. Thus, the reproductive success of the wolf population has steadily increased over the last five years. The per-capita kill rate, which is an indication of the rate that wolves acquire food, was 0.52 moose per wolf per month. That rate is somewhat lower than last year’s estimate of 0.71, but similar to what is expected given the number of wolves and moose currently on the island. The estimated abundance of moose is 967, which is a 28 percent decline from last year’s estimate of 1,346. Longer-term population trends suggest that the moose population had increased greatly over an eight-year period (2011-2019) but then started to decline rapidly over the last few years. The proportion of the moose population that is newly recruited individuals (i.e., nine-month-old calves in February) was 1.7 percent, which is very low compared to the long-term average. Predation rate was estimated to be 10 percent, which is similar to last year’s estimate and close to the long-term average. While predation and low recruitment were important contributors to the moose decline, starvation also played an important role.
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