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Books on the topic 'Spinal motion'

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Published: 4 June 2021
Last updated: 20 February 2023

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1

Insight, LLC Medtech. U.S. markets for spinal motion preserving devices. Newport Beach, CA: Medtech Insight, 2005.

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2

Occhiogrosso, Peter. Inside Spinal Tap. New York: Arbor House, 1985.

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3

Inside Spinal Tap. London: Abacus, 1992.

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4

This is Spinal Tap. Milwaukee, WI: Limelight Editions, 2010.

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5

Michael, McKean, and French Karl, eds. This is Spinal Tap: The official companion. London: Bloomsbury, 2000.

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6

1937-, Taylor Jim, ed. Rick Hansen: Man in motion. Vancouver: Douglas & McIntyre, 1987.

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7

Copyright Paperback Collection (Library of Congress), ed. The spiral path. New York: Berkley Books, 2002.

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8

Putney, Mary Jo. The spiral path. Thorndike, Me: Center Point, 2002.

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9

1953-, Cope Timothy C., ed. Motor neurobiology of the spinal cord. Boca Raton: CRC Press, 2001.

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10

How brain-like is the spinal cord?: Interacting cell assemblies in the nervous system. Berlin: Springer-Verlag, 1988.

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11

1916-, Lin C. C., ed. Spiral structure in galaxies: A density wave theory. Cambridge, Mass: MIT Press, 1996.

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12

"Down a spiral staircase, never-ending": Motion as design in the writing of Czeslaw Milosz. New York: P. Lang, 1990.

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13

Pierrot-Deseilligny, Emmanuel. The circuitry of the human spinal cord: Neuroplasticity and corticospinal mechanisms. Cambridge: Cambridge University Press, 2012.

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14

Nezlin, M. V. Rossby vortices, spiral structures, solitons: Astrophysics and plasma physics in shallow water experiments. Berlin: Springer-Verlag, 1993.

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15

Spectroscopic techniques and hindered molecular motion. Boca Raton: CRC Press, 2012.

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16

Budzynski, Leslie. An inter- and intra-examiner reliability study of motion palpation of the costovertebral joints (T2-T5) in axial rotation in the seated position. [Bournemouth, Eng.]: Anglo-European College of Chiropractic, 1987.

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17

Tap, Spinal, Nigel Tufnel, David St Hubbins, Michael Mckean, and Christopher Guest. Spinal Tap. HarperCollins Publishers, 2011.

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18

European markets for spinal motion-preserving devices. Newport Beach, CA: Medtech Insight, 2006.

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19

The Official "Spinal Tap" Companion. Bloomsbury Publishing PLC, 2001.

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20

Spinal Arthroplasty with DVD: The Preservation of Motion. Saunders, 2007.

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21

French, Karl. This is Spinal Tap: Official Companion. Bloomsbury USA, 2000.

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22

Motion Preservation, An Issue of Neurosurgery Clinics (The Clinics: Surgery). Saunders, 2005.

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23

Boehm, Heinrich, and Y. Raja Rampersaud. Treatment: spinal surgery. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198734444.003.0023.

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Despite remarkable progress in understanding the pathological processes and alleviating symptoms by TNFα‎ blocking medication, the mechanism that converts flexible tissue into bone still cannot be completely prevented or reversed. In axial spondyloarthritis, components of motion segments, such as zygoapophyseal joints, intervertebral discs, and spinal ligaments, can ossify in varying sequence, extent, and location between the ilium and occiput. Throughout this process, the spinal column is vulnerable to kyphotic deformity due to gravity, body weight, muscle action, and life’s flexion-based activities. Areas with low fusion tendency, such as atlanto-axial joints, and post-traumatically weakened spots of formerly ankylosed vertebral block (Andersson’s lesion) can endanger the spinal cord by instability, dislocation, and compression, from what is typically minor trauma or simple repetitive, but otherwise normal, stresses. Once functionally significant deformity or presence of instability and associated symptoms are established, conservative treatment options are lacking and surgical consideration is required.
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24

Muir, John Kenneth. Behind the Screen: This Is Spinal Tap (Behind the Screen). Emmis Books, 2006.

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25

Taylor, Jim, and Rick Hansen. Rick Hansen : Man in Motion. Douglas & McIntyre, 1999.

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26

J, Yue James, ed. Motion preservation surgery of the spine: Advanced techniques and controversies. Philadelphia, PA: Saunders/Elsevier, 2008.

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27

Nonfusion Techniques for the Spine: Motion Preservation & Balance. Quality Medical Publishing, 2005.

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28

H, Maxwell James, Griffith Steven L. 1960-, and Welch William C, eds. Nonfusion techniques for the spine: Motion preservation & balance. St. Louis, Mo: Quality Medical Pub., 2006.

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29

This Is Spinal Tap (Cultographies). Wallflower Press, 2007.

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30

Fife, Leland Kent. The reliability of the Dynavec(TM) LVD: A tool for the assessment of three dimensional lumbar spinal motion. 1991.

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31

The reliability of the Dynavec[superscript TM] LVD: A tool for the assessment of three dimensional lumbar spinal motion. 1991.

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32

Schmid, Stefan, Dennis E. Anderson, Babak Bazrgari, and Lennart Scheys, eds. Using Motion Analysis Techniques and Musculoskeletal Modeling of the Spine to Better Understand Spinal Disorders and Evaluate Treatment Effects. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88976-038-1.

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33

Scepanovic, Darja. Is there a relationship between height, trunk length and leg length and sagittal spinal range of motion in normal subjects?. UEL, 1994.

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34

Mehta, Priyesh, David J. Cormier, Julie Ann Aueron, and Jaspal R. Singh. Core Strengthening. Edited by Mehul J. Desai. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199350940.003.0025.

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Core strengthening remains a vital component of maintaining spine mechanics and assists in the prevention of injury. A complex group of muscles make up the core and function together to instill spine stability and assist with segmental motion. Understanding the key muscles and how they function is essential to developing a core strengthening program for patients with spinal pathology. This chapter summarizes the anatomy and biomechanics of the core involved in maintaining spine stability. It discusses electromyography and imaging data to help understand how these muscles function during specific exercises. Examples of core strengthening exercises are provided.
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35

Gherscovici, Ezequiel, Eli Baron, and Alexander Vaccaro. Regional injuries of the cervical spine: sports-related anatomical, pathophysiological, and clinical perspectives. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199533909.003.0023.

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Cervical spine injuries occur infrequently on the athletic field (Dietz and Lillegard 1999). Nevertheless, sporting events have been reported as the fourth most common cause of spinal cord injury (behind motor vehicle collisions, assaults, and falls) (NSCISC 2006). The possibility of catastrophic cervical spine injury exists with involvement in sports, where it can be defined as ‘structural distortion of the cervical spinal column associated with actual or potential damage to the spinal cord’. This may result in irreversible neurological injury to the athlete (...
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36

Mason, Peggy. Spinal Cord. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190237493.003.0004.

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The spinothalamic and lemniscal pathways carry somatosensory information from the periphery into the brain while the corticospinal pathway carries motor commands from the brain to motoneurons of the spinal cord. Following these pathways through the spinal cord allows the student to infer lesion location from symptoms. To exemplify the clinical importance of sympathetic outputs from thoracic segments, Horner syndrome is described. Similarly, the common problems caused by spinal cord injury on sacral parasympathetic functions are stressed. The contributions of specific spinal segments to breathing, hand and foot dexterity, and micturition are emphasized. Working through the logic of the symptoms caused by spinal hemisection (Brown-Séquard syndrome), pyramidal stroke, and syringomyelia provides the student with a clear framework for understanding spinal function in the clinical context.
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37

Monani, Umrao R., and Darryl C. De Vivo. Spinal Muscular Atrophy. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0033.

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Spinal muscular atrophy (SMA) is a common, inherited, pediatric motor neuron disorder caused by insufficient SMN protein. As of yet, there is no good treatment for the disease. SMA has an incidence of ~1 in 10,000 newborns carrier frequency of 1 in 50, making it the most common inherited cause of infant mortality. Patients with severe SMA, or Werdnig-Hoffman disease, typically manifest weakness during the first 6 months of life. Such patients are so debilitated that they never sit independently, frequently succumbing to the disease before age 2 years. A much milder form of SMA, Kugelberg-Welander disease, with onset after 18 months of age, often during childhood and characterized by prolonged ambulation and a normal life expectancy, was described in 1956. In 1995 mutations in a novel gene, Survival of Motor Neuron 1 (SMN1), were determined to be the specific cause of SMA.
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38

Speed, Cathy. Badminton injuries. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199533909.003.0054.

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The sport of badminton originated in Gloucestershire, England, in the 1860s and has evolved to become a game played by millions across the world. Badminton is the world’s fastest racket sport with shuttles reaching 200 mph in top international competitions. When played at high level, this fast and powerful game demands high levels of strength, agility, endurance, and tactical awareness. Most of the injuries seen are related to microtraumatic overuse, and are associated with high impact, lunging, rotating, and spinal extension. The mechanics of the sport present a good example of the influence of any impairment or dysfunction within the kinetic chain on other links along the same kinetic pathway. For example, restriction in the thoracic spine can lead to increased demands on the shoulder and arm when attempts are made to achieve adequate range of motion to play an overhead shot....
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39

Casha, Steve, and Philippe Mercier. Normal anatomy and physiology of the spinal cord and peripheral nerves. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0220.

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The spinal cord and peripheral nerves carry motor and autonomic efferents, as well as sensory afferents connecting the cerebrum with the body. Efferent and afferent fibres form predictable tracts within the spinal cord, forming spinal nerves as they exit the spinal canal. Peripheral nerves are often formed from complicated plexuses of spinal nerves in the cervical, lumbar, and sacral spine. Dermatomes are formed from spinal nerves that innervate specific areas of skin, while myotomes innervate a specific set of muscles. The detailed anatomy of these structures are discussed. Knowledge of the anatomy of these structures is relevant to many clinical situations encountered in the intensive care unit especially with caring for neurological, neurosurgical, orthopaedic, and trauma patients.
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40

Burghes, Arthur H. M., and Vicki L. McGovern. Spinal Muscular Atrophy. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0034.

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Spinal muscular atrophies affect the lower motor neuron. The most common SMA maps to 5q is an autosomal recessive disorder. SMA is caused by loss or mutation of the SMN1 gene and retention of the SMN2 gene, and these genes lie in a complex area of the genome. Mild missense alleles of SMN1 work to complement SMN2 to give function and therapeutics that restore SMN levels are in clinical testing. Modifiers that lie outside the SMN gene locus and influence severity clearly exist, but what they are remains unknown as do the critical genes affected by SMN deficiency.
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41

Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson, and Nathaniel M. Robbins. A 54-Year-Old Male with Right-Hand Weakness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0002.

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Early in its course, amyotrophic lateral sclerosis (ALS) is mistaken for a number of other neuromuscular problems, including spinal disease, multifocal motor neuropathy, and even carpal tunnel syndrome (CTS) when the weakness is distal and focal. In our patient CTS or cervical spine disease was considered. MRI scan of the appropriate spinal level is important to rule out spinal disease. Nerve conduction studies (NCS) and electromyography (EMG) help to exclude other possibilities and point to the diagnosis of ALS. Later in the clinical course, the clinical picture is pathognomonic with upper and lower motor neuron signs. The differential diagnosis of focal weakness is discussed, as is recognition of the more typical ALS clinical syndrome and familial ALS. NCS and EMG findings in ALS are discussed.
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42

Gutierrez, Genaro J., and Divya Chirumamilla. Cervical Spinal Stenosis. Edited by Mehul J. Desai. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199350940.003.0006.

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Cervical spinal stenosis is the narrowing of the spinal canal. Degenerative cervical spinal stenosis can occur as a result of disc degeneration, osteophyte formation, and hypertrophy of spinal canal ligaments. Diagnosis is primarily made with clinical history and examination in order to assess for classic myelopathic signs (motor weakness, hyperreflexia, and other specific tests). Radiologic imaging is used to validation the diagnosis and to determine the extent of stenosis. Magnetic resonance imaging is the most useful and noninvasive modality. Cervical spinal stenosis without myelopathy can be managed nonsurgically with strengthening, physical therapy, traction, orthosis, and pain management (cervical epidural steroid injections and selective nerve root blocks). Cervical spondylolisthesis has received insufficient attention in comparison to spondylolisthesis of the lumbar spine. It is primarily considered a surgical condition, yet few publications have been dedicated to the topic.
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43

Leung, General. Motion compensation in MRI using variable density spiral trajectories. 2004.

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44

Bates, David. Spinal cord disorders. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0650.

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Non-traumatic spinal cord disease may be caused by compression due to tumour, infection or haematoma, inflammation, infection or post-infection, metabolic disturbances, infarction, and degeneration. The diagnosis is often made easier by the clinical assessment: the patient’s age, the speed of onset of the disease, severity of the deficits, the pattern of motor and sensory involvement, and presence of pain and sphincter symptoms are all important in making an assessment of the site and likely nature of the spinal disease.Investigations are obligatory to confirm a diagnosis and to direct therapy. MRI is the most useful investigation. It has largely replaced myelography which should now only be considered in patients with indwelling cardiac pacing wires. Additional investigations including examination of the cerebrospinal fluid, evoked potentials, and specific blood tests may be required and the value of plain X-rays, CT scan, and, in some instances, angiography should not be overlooked.The remainder of this chapter will consider specific disorders, identifying pathology, clinical presentation, investigation, and management. Acute and chronic conditions are considered separately and those affecting the cauda equina, spinal root, and sphincters are considered in Chapter 29.
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45

Stamenova, M., and S. Sanvito. Atomistic spin-dynamics. Edited by A. V. Narlikar and Y. Y. Fu. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780199533046.013.7.

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This article reviews recent advances towards the development of a truly atomistic time-dependent theory for spin-dynamics. The focus is on the s-d tight-binding model [where conduction electrons (s) are exchange-coupled to a number of classical spins (d)], including electrostatic corrections at the Hartree level, as the underlying electronic structure theory. In particular, the article considers one-dimensional (1D) magnetic atomic wires and their electronic structure, described by means of the s-d model. The discussion begins with an overview of the model spin Hamiltonian, followed by molecular-dynamics simulations of spin-wave dispersion in a s-d monoatomic chain and spin impurities in a non-magnetic chain. The current-induced motion in a magnetic domain wall (DW) is also explored, along with how an electric current can affect the magnetization landscape of a magnetic nano-object. The article concludes with an assessment of spin-motive force, and especially whether a driven magnetization dynamics can generate an electrical signal.
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46

Eriksson, Olle, Anders Bergman, Lars Bergqvist, and Johan Hellsvik. Atomistic Spin Dynamics. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198788669.001.0001.

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The purpose of this book is to provide a theoretical foundation and an understanding of atomistic spin-dynamics, and to give examples of where the atomistic Landau-Lifshitz-Gilbert equation can and should be used. The contents involve a description of density functional theory both from a fundamental viewpoint as well as a practical one, with several examples of how this theory can be used for the evaluation of ground state properties like spin and orbital moments, magnetic form-factors, magnetic anisotropy, Heisenberg exchange parameters, and the Gilbert damping parameter. This book also outlines how interatomic exchange interactions are relevant for the effective field used in the temporal evolution of atomistic spins. The equation of motion for atomistic spin-dynamics is derived starting from the quantum mechanical equation of motion of the spin-operator. It is shown that this lead to the atomistic Landau-Lifshitz-Gilbert equation, provided a Born-Oppenheimer-like approximation is made, where the motion of atomic spins is considered slower than that of the electrons. It is also described how finite temperature effects may enter the theory of atomistic spin-dynamics, via Langevin dynamics. Details of the practical implementation of the resulting stochastic differential equation are provided, and several examples illustrating the accuracy and importance of this method are given. Examples are given of how atomistic spin-dynamics reproduce experimental data of magnon dispersion of bulk and thin-film systems, the damping parameter, the formation of skyrmionic states, all-thermal switching motion, and ultrafast magnetization measurements.
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47

Peralta, Feyce. High or Total Spinal/Epidural. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0044.

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High or total spinal/epidural blockade occurs due to excess spread of local anesthetic within the neuraxial space. While this is an infrequent complication, it can cause respiratory and hemodynamic instability in obstetric patients. If high/total spinal/epidural occurs prior to delivery, such derangements may lead to fetal intolerance and need for emergency delivery. Clinicians should suspect risk for high block when patients lose upper extremity motor function and complain of dysphonia or dyspnea. Intubation and respiratory and hemodynamic support along with adequate sedation should be given until the block recedes. Preventative measures include strict epidural catheter aspiration practice and incremental epidural dosing strategies.
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48

Rossby Vortices, Spiral Structures, Solitons. Springer, 2012.

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49

Rinaldi, Simon. Congenital neurological disorders. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0225.

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This chapter covers four congenital neurological disorders which may be encountered in adult medicine: cerebral palsy, Chiari malformations, spina bifida, and tethered cord syndromes. Cerebral palsy is a disturbance of motor function arising from damage to the developing fetal or infant brain. It usually refers to a disorder resulting from a non-progressive insult which occurred at less than 3 years of age. Chiari malformations are congenital abnormalities of the anatomy and structural relationships of the cerebellum, the brainstem, and the foramen magnum. Dysraphism is a failure of opposition of anatomical structures which are normally fused. Spinal dysraphism is synonymous with spina bifida, a failure of embryological fusion of the neural tube. In all types, the vertebral arch fails to completely form. The tethered cord syndromes involve a restriction of the normal cephalad migration of the conus during life. This can occur both with and without spina bifida.
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50

Cope, Timothy C. Motor Neurobiology of the Spinal Cord. Taylor & Francis Group, 2001.

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