Relevant bibliographies by topics / Sphincter of Oddi

Academic literature on the topic 'Sphincter of Oddi'

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Published: 4 June 2021
Last updated: 29 January 2023

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Journal articles on the topic "Sphincter of Oddi"

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Thune, A., L. Jivegård, H. Pollard, J. Moreau, J. C. Schwartz, and J. Svanvik. "Location of enkephalinase and functional effects of [Leu5] enkephalin and inhibition of enkephalinase in the feline main pancreatic and bile duct sphincters." Clinical Science 82, no. 2 (February 1, 1992): 169–73. http://dx.doi.org/10.1042/cs0820169.

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1. Morphological studies have demonstrated enkephalinergic nerve fibres in proximity to the sphincter of Oddi, and opiates are known to contract this sphincter. In this study, the flow resistances in the common bile duct and main pancreatic duct sphincters were studied simultaneously in anaesthetized cats using a perfusion technique. 2. Naloxone did not affect the activity of these sphincters under basal conditions, indicating that there is no basal enkephalinergic tone. 3. The response to [Leu5]enkephalin (0.015–15 μg/kg), morphine (1 mg/kg) and ketamine (10 mg/kg) was a naloxone-sensitive increased activity in the sphincters with a raised frequency of phasic contractions. The threshold dose for an effect of [Leu5]enkephalin on the sphincter of Oddi was 0.015 μg/kg and a maximal response was observed at 0.75 μg/kg. There were no differences in the response of the main pancreatic duct sphincter and the bile duct sphincter to the different drugs. 4. Immunoautoradiographic studies demonstrated enkephalinase in the spincter of Oddi. 5. Acetorphan (3 mg/kg intravenously), which inhibits endogenous enkephalinase both in the peripheral and the central nervous system when administered parenterally, caused a naloxone-sensitive contraction, whereas thiorphan (3–20 mg/kg), an enkephalinase inhibitor that does not easily penetrate the blood-brain barrier, had no effect on the sphincter of Oddi. 6. These results show that endogenous and exogenous opiates influence the function of the feline sphincter of Oddi and that enkephalins may be involved in the physiological control of this sphincter, although not under basal conditions.
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Lehman, Glen A., and Stuart Sherman. "Hypertensive Pancreatic Sphincter." Canadian Journal of Gastroenterology 12, no. 5 (1998): 333–37. http://dx.doi.org/10.1155/1998/148150.

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Major papilla pancreatic sphincter dysfunction, a variant of sphincter of Oddi dysfunction, causes pancreatitis or pancreatic-type pain. Endoscopic manometry as performed at endoscopic retrograde cholangiography is the most commonly used method to identify sphincter dysfunction. Noninvasive testing, such as secretin-stimulated ultrasound analysis of duct diameter, is less reliable and of relatively low sensitivity. Two-thirds of patients with sphincter of Oddi dysfunction have elevated pancreatic basal sphincter pressure. Patients with suspected or documented sphincter of Oddi dysfunction may respond to biliary sphincterotomy alone, but warrant evaluation of their pancreatic sphincter if symptoms persist after therapy. Whether such pancreatic and biliary sphincters should be treated at the first treatment session is controversial. Pancreatic sphincterotomy is associated with a complication rate very similar to that of biliary sphincterotomy except that the pancreatitis rate is two- to fourfold higher. Prophylactic pancreatic stenting diminishes such pancreatitis by approximately 50%.
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Haubrich, William S. "Oddi of the sphincter of Oddi." Gastroenterology 116, no. 3 (March 1999): 542. http://dx.doi.org/10.1016/s0016-5085(99)70217-9.

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Polunina, T. E. "Sphincter of Oddi dysfunction. Case history." Medical Council, no. 3 (May 12, 2019): 26–33. http://dx.doi.org/10.21518/2079-701x-2019-3-26-33.

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The article presents the definition and prevalence of sphincter of Oddi dysfunction in patients with various disorders of the biliary system. It describes the characteristics of sphincter of Oddi dysfunction with due account of its anatomical structure. Based on Rome IV Criteria (2016), the authors provide modern approaches to the diagnosis and treatment of sphincter of Oddi dysfunction. Special attention is paid to the diagnostic criteria for sphincter of Oddi dysfunction depending on its type. There are two basic types of sphincter of Oddi dysfunction: biliary dysfunction and pancreatitis. The paper presents the algorithms for the examination and treatment of patients with sphincter of Oddi dysfunction depending on the severity level of the disease and objective findings of laboratory and instrumental tests. The article presents a clinical history of a patient with sphincter of Oddi dysfunction. Depending on the characteristics of a clinical course of dysfunction, it was proposed that the patient was treated with a selective antispasmodic drug and a choleretic drug.
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Della Libera, Ermelindo, Rodrigo Azevedo Rodrigues, Ana Paula Rodrigues Guimarães, Gustavo Andrade de Paulo, Stephan Geocze, and Angelo Paulo Ferrari. "Prevalence of sphincter of Oddi dysfunction in patients referred to endoscopic retrograde cholangiopancreatography." Arquivos de Gastroenterologia 44, no. 1 (March 2007): 18–21. http://dx.doi.org/10.1590/s0004-28032007000100005.

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BACKGROUND: Sphincter of Oddi manometry is the gold-standard method for sphincter of Oddi dysfunction. The prevalence of sphincter of Oddi dysfunction among patients referred to endoscopic retrograde cholangiopancreatography is largely unknown. AIM: To evaluate prospectively the prevalence of biliary sphincter of Oddi dysfunction (B-SOD) among Brazilian patients referred to endoscopic retrograde cholangiopancreatography and to study the safety of sphincter of Oddi manometry in this setting. METHODS: Biliary sphincter of Oddi manometry was intended in 110 patients referred to endoscopic retrograde cholangiopancreatography. The number of attempts to obtain deep cannulation with the manometry catheter was recorded and patients were divided into two groups: up to 5 (easy cannulation) and >5 attempts (difficult cannulation). RESULTS: Sphincter of Oddi manometry was successful in 71/110 patients (64.5%). Sphincter of Oddi dysfunction was found in 18/71 patients (25%). Endoscopic retrograde cholangiopancreatography findings were: normal in 16, biliary stones in 39, malignant biliary strictures in 9 and benign biliary strictures in 7. There was no statistical difference in sphincter of Oddi dysfunction prevalence regarding disease, gender or difficulty of cannulation. Only 2/71 patients developed post-procedure mild pancreatitis. CONCLUSIONS: We have found a high prevalence of sphincter of Oddi dysfunction in patients referred to endoscopic retrograde cholangiopancreatography. Gender, nature of disease or difficulty of cannulation did not influence the prevalence of sphincter of Oddi dysfunction among these patients. Sphincter of Oddi manometry is a safe procedure for the evaluation of sphincter of Oddi dysfunction in patients referred to endoscopic retrograde cholangiopancreatography.
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Zhang, Qi, Mao Ye, Wei Su, Yiwen Chen, Yu Lou, Jiaqi Yang, Tao Ma, et al. "Normal sphincter of Oddi." ASVIDE 7 (November 2020): 274. http://dx.doi.org/10.21037/asvide.2020.274.

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Zhang, Qi, Mao Ye, Wei Su, Yiwen Chen, Yu Lou, Jiaqi Yang, Tao Ma, et al. "Lax sphincter of Oddi." ASVIDE 7 (November 2020): 275. http://dx.doi.org/10.21037/asvide.2020.275.

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Funch-Jensen, P., and N. Ebbehøj. "Sphincter of Oddi Motility." Scandinavian Journal of Gastroenterology 31, sup216 (January 1996): 46–51. http://dx.doi.org/10.3109/00365529609094560.

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Lehman, Glen A., and Stuart Sherman. "Sphincter of Oddi dysfunction." International Journal of Pancreatology 20, no. 1 (August 1996): 11–25. http://dx.doi.org/10.1007/bf02787372.

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Boivineau, G., J. M. Gonzalez, M. Gasmi, V. Vitton, and M. Barthet. "Sphincter of Oddi dysfunction." Journal of Visceral Surgery 159, no. 1 (March 2022): S16—S21. http://dx.doi.org/10.1016/j.jviscsurg.2022.01.008.

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Dissertations / Theses on the topic "Sphincter of Oddi"

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Stoner, Edward Alexander. "Developments in the technique of sphincter of Oddi manometry and investigation of sphincter of Oddi dysfunction." Thesis, Queen Mary, University of London, 1999. http://qmro.qmul.ac.uk/xmlui/handle/123456789/1864.

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The hazardous technique of endoscopic manometry precludes the investigation of "normal volunteers" required to advance our knowledge of the physiology and pathophysiology of the sphincter of Oddi, a suitable animal model is required. Large and small animal models have been proposed, as yet no one model has been accepted as being representative of the human sphincter of Oddi. Furthermore no animal model of sphincter of Oddi dysfunction has been developed. In this thesis a porcine animal model of sphincter of Oddi function has been developed. The importance of selecting the appropriate anaesthetic agent, enflurane, has been proven. The effect of cholecystectomy on the porcine sphincter of Oddi is shown to have no overall significant effect on sphincter motility when compared to a sham laparotomy group. However, two of the seven pigs after cholecystectomy showed a paradoxical rise in sphincter basal pressure after cholecystokinin infusion, these animal may represent porcine sphincter of Oddi dysfunction. Although substance P is found throughout the intestinal tract including the sphincter of Oddi of man and pig its action was hitherto unknown. In this thesis exogenous substance P was shown to stimulate the sphincter of Oddi in vivo. In this thesis the first development in sphincter of Oddi manometry catheter design in nearly twenty years is presented. A superior nine 3 lumen catheter has been evaluated in porcine model and subsequently used to assess sphincter of Oddi asymmetry in man. Two retrospective studies are reported in this thesis; an audit of the largest U. K. series, and a study assessing the relationship of sphincter of Oddi motility and duodenal activity. Tachyoddia dissociated from the duodenal migrating motor complex was associated with a raised sphincter of Oddi basal pressure and may be a part of sphincter of Oddi dysfunction.
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DERREVEAUX, PHILIPPE. "Les tumeurs de la region oddienne : a propos de 18 cas." Reims, 1992. http://www.theses.fr/1992REIMM089.

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Dorandeu, Anne. "Tumeurs oddiennes : analyse et facteurs pronostiques ; a propos de 53 cas." Rennes 1, 1994. http://www.theses.fr/1994REN1M080.

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Cassar, Jean-François. "La sphincterotomie endoscopique, résultats à court et long termes." Bordeaux 2, 1988. http://www.theses.fr/1988BOR25375.

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MARILL, JEAN-LUC. "La manometrie biliaire endoscopique : donnees actuelles, introduction d'une nouvelle methode d'etude du sphincter d'oddi." Nantes, 1988. http://www.theses.fr/1988NANT126M.

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DAILLIE, FAVRE MARIANNE. "La sphincterotomie endoscopique dans le traitement des fistules biliaires : a propos de 12 observations." Lyon 1, 1989. http://www.theses.fr/1989LYO1M194.

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Liard, Olivier. "Les tumeurs villeuses oddiennes : à propos d'un cas." Bordeaux 2, 1989. http://www.theses.fr/1989BOR25136.

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Le, Sidaner Anne. "Les pancreatites aigues biliaires : interet de la cholangio-pancreatographie retrograde endoscopique et de la sphincterotomie endoscopique : a propos de 48 cas." Limoges, 1989. http://www.theses.fr/1989LIMOO116.

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Woods, Charmaine Michelle, and charmaine woods@flinders edu au. "EXOGENOUS PURINES INDUCE DIFFERENTIAL RESPONSES IN THE PROXIMAL AND DISTAL REGIONS OF THE SPHINCTER OF ODDI: PARTIAL CHARACTERISATION OF THE PURINERGIC RECEPTOR SUB-TYPES INVOLVED." Flinders University. School of Medicine, 2006. http://catalogue.flinders.edu.au./local/adt/public/adt-SFU20061120.095902.

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The sphincter of Oddi (SO) is a neuromuscular structure located at the junction of the bile and pancreatic ducts with the duodenum. The primary functions of the SO are to regulate the delivery of bile and pancreatic juice into the duodenum, and to prevent reflux of duodenal contents into the biliary and pancreatic systems. Neural, hormonal or functional disturbances of biliary motility can lead to painful and sometimes life threatening clinical conditions, such as SO dysfunction and acute pancreatitis. Clearly understanding the regulation of biliary and duodenal motility patterns is necessary and may provide useful pharmacological sites for drug development to aid in the treatment of these diseases. Spontaneous activity of the SO is regulated by complex interactions between the enteric nervous system, hormones, possibly interstitial cells of Cajal and other bioactive agents, together with modulation via neural reflexes between the duodenum, common bile duct/gallbladder, and stomach. Purines are one group of neurotransmitters/regulatory agents that have been shown to effect gastrointestinal motility, however their functions in the regulation of SO motility have not been elucidated. The studies described in this thesis used in vitro organ bath techniques and in vivo preparations to determine the effects of exogenous purines on possum SO and duodenal motility. The possum SO has been extensively characterized and is an excellent model for motility studies. In vitro, exogenous adenosine was found to decrease spontaneous activity in both the SO and duodenum. In contrast exogenous ATP induced both excitatory and inhibitory responses in the SO and duodenum. Interestingly, the adenosine and ATP-induced effects were predominantly exhibited by the proximal portion of the SO (proximal-SO), with no or little effect observed in the distal portion of the SO (distal-SO). These data support the hypothesis that the SO is comprised of different functional components that can act differently in response to certain stimuli, and highlights the importance of studying each of the SO components. Agonists and antagonists, together with immunohistochemical studies, were used in an attempt to identify the P1 and P2 receptor sub-types responsible for mediating the adenosine- and ATP-induced responses. In the duodenum the adenosine-induced decrease in spontaneous activity was likely to be mediated by A2A and A3 receptors, but the receptors mediating the proximal-SO response could not be identified. In the duodenum ATP induced a complex non-neural response consisting of a P2X1, and P2Y2 and/or P2Y4 mediated immediate inhibition. This was followed by a return to baseline activity or small excitation. The response concluded with a late inhibitory response, likely to be mediated by P2Y1 receptors, but the effects of other P2Y receptors could not be excluded. In contrast, ATP application to the proximal-SO evoked a partially neurally mediated early excitation, likely via P2X receptors, followed by an inhibition of activity, likely via activation of non-neural P2Y2 and/or P2Y4 receptors. In vivo studies with exogenous application of adenosine and ATP to the SO activated neural pathways to produce increased motor activity. Characterisation of these neural pathways found ATP and/or adenosine to activate excitatory cholinergic motor neurons. ATP also activated an inhibitory nicotinic/nitrergic pathway. This is the first comprehensive investigation of the possible involvement of purines in the regulation of SO motility. These studies demonstrate that exogenous purines influence SO and duodenal motility, inducing complex neural and non-neural responses, acting via multiple P1 and P2 receptors. It now remains to be determined if endogenously released purines induce similar responses, together with elucidation and location of the receptor sub-types involved.
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Béraud, Jean-Michel. "Etude de l'apport du scanner et de la sphinctèrotomie endoscopique en urgence dans le diagnostic et le traitement des pancréatites aigües d'origine biliaire certaine ou suceptible de l'être : résultats préliminaires à partir de 20 cas." Saint-Etienne, 1988. http://www.theses.fr/1988STET6065.

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Books on the topic "Sphincter of Oddi"

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Price, Michael Alan. Cholescintigraphy after endoscopic papillotomy in patients with an intact gallbladder. [New Haven: s.n.], 1989.

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Keshav, Satish, and Alexandra Kent. Gall bladder disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0200.

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The gall bladder is a sac which lies underneath the liver and stores and concentrates bile produced by the liver. As food enters the duodenum, it stimulates the release of cholecystokinin, which in turn stimulates the release of bile, which passes via the cystic duct to the common bile duct, which connects to the duodenum at the sphincter of Oddi. Bile is required in digestion, especially for the emulsification and absorption of fat. Biliary disease can take several forms. Cholelithiasis refers to the presence of gallstones in the gall bladder, whereas choledocholithiasis refers to gallstones in the biliary tree. Cholecystitis is inflammation and infection of the gall bladder. Cholangitis is inflammation and infection of the biliary tree. Sphincter of Oddi dysfunction (SOD) is characterized by symptoms of biliary obstruction, with no structural cause. Other forms of biliary disease are gall bladder polyps, primary biliary cholangitis, and primary sclerosing cholangitis.
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Book chapters on the topic "Sphincter of Oddi"

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Fogel, Evan L., Stuart Sherman, and Glen A. Lehman. "Sphincter of Oddi Manometry." In Successful Training in Gastrointestinal Endoscopy, 324–31. Oxford, UK: Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444397772.ch27.

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Moffatt, Dana C., Stuart Sherman, and Evan L. Fogel. "Sphincter of Oddi Dysfunction." In Textbook of Clinical Gastroenterology and Hepatology, 567–72. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781118321386.ch75.

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Fogel, Evan L., and Stuart Sherman. "Sphincter of Oddi Manometry." In ERCP, 156–69. Chichester, UK: John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118769409.ch10.

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Allescher, Hans-Dieter. "Sphincter of Oddi Manometry." In Clinical Hepatology, 519–23. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-93842-2_47.

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Chen, Tseng-Shing. "Sphincter of Oddi Dysfunction." In Biliopancreatic Endoscopy, 213–24. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-4367-3_19.

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Lehman, Glen A. "Sphincter of Oddi Manometry." In Gastrointestinal Motility, 61–70. Boston, MA: Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4803-4_7.

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Johlin, Frederick C. "Sphincter of Oddi and Pancreatic Sphincter Dysmotility." In Gastrointestinal Motility, 183–96. Boston, MA: Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4803-4_19.

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Gutta, Aditya, and Glen Lehman. "Treatment of Sphincter of Oddi Dysfunction." In Gastrointestinal and Pancreatico-Biliary Diseases: Advanced Diagnostic and Therapeutic Endoscopy, 1–17. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-29964-4_76-1.

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Gutta, Aditya, and Glen Lehman. "Treatment of Sphincter of Oddi Dysfunction." In Gastrointestinal and Pancreatico-Biliary Diseases: Advanced Diagnostic and Therapeutic Endoscopy, 1323–39. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-56993-8_76.

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Bauer, W., R. Ascherl, H. Zimmermann, P. Bauerfeind, and G. Blümel. "Experimentelle Untersuchungen zur Funktion des Sphincter Oddi." In 104. Kongreß der Deutschen Gesellschaft für Chirurgie München, 22.–25. April 1987, 379–83. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71781-9_74.

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Conference papers on the topic "Sphincter of Oddi"

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Menon, Shyam, and Ray Mathew. "PTU-10 Botulinum toxin injection in the management of type II sphincter of oddi dysfunction." In Abstracts of the BSG Annual Meeting, 8–12 November 2021. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2021. http://dx.doi.org/10.1136/gutjnl-2021-bsg.83.

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Menon, Shyam, and Ravi Marudanayagam. "PWE-54 Transduodenal sphincteroplasty in the management of refractory pain in type II sphincter of oddi dysfunction." In Abstracts of the BSG Annual Meeting, 8–12 November 2021. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2021. http://dx.doi.org/10.1136/gutjnl-2021-bsg.317.

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Basavaraju, Umesh, Vicky FP Ritchie, and John S. Leeds. "PTU-015 Medium-term outcome of endoscopic sphincterotomy in biliary manometry confirmed sphincter of oddi dysfuncton type 2." In British Society of Gastroenterology, Annual General Meeting, 4–7 June 2018, Abstracts. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2018. http://dx.doi.org/10.1136/gutjnl-2018-bsgabstracts.283.

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Jamal, S., H. Seddik, S. Berrag, K. Loubaris, F. Bouhamou, S. Morabit, A. Aomari, et al. "THE MACRODILATATION OF THE SPHINCTER OF ODDI OR SPHINCTEROPLASTY IN THE TREATMENT OF LARGE STONES OF THE MAIN BILE DUCT." In ESGE Days 2018 accepted abstracts. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1637630.

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Elkoti, I., K. Loubaris, R. Berraida, S. Lmrabti, H. Boutalaka, A. Sair, A. Bendehmane, et al. "THE MACRODILATATION OF THE SPHINCTER OF ODDI OR SPHINCTEROPLASTY IN THE TREATMENT OF LARGE STONES OF THE MAIN BILE DUCT ABOUT A MOROCCAN POPULATION." In ESGE Days. © Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1704449.

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