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Books on the topic 'Somatosensory evoked potentials'

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Published: 4 June 2021

Last updated: 31 January 2023

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1

Technologists, American Society of Electroneurodiagnostic. Evoked potentials: Concepts and somatosensory. 2nd ed. Carroll, IA: American Society of Electroneurodiagnostic Technologists, 1998.

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2

Evoked potential primer: Visual, auditory, and somatosensory evoked potentials in clinical diagnosis. Boston: Butterworth, 1985.

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3

Spehlmann, Rainer. Evoked potential primer: Visual, auditory, and somatosensory evoked potentials in clinical diagnosis. Boston: Butterworth, 1985.

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4

Dellon, A. Lee. Somatosensory testing & rehabilitation. Bethesda, MD: American Occupational Therapy Association, 1997.

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5

1950-, Mackenzie Keith, Baran Ernest M, and DeLisa Joel A, eds. Manual of nerve conduction velocity and somatosensory evoked potentials. 2nd ed. New York: Raven Press, 1987.

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6

1931-, Spehlmann Rainer, ed. Spehlmann's evoked potential primer: Visual, auditory, and somatosensory evoked potentials in clinical diagnosis. 2nd ed. Boston: Butterworth-Heinemann, 1994.

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7

I, Hashimoto, and Kakigi Ryusuke, eds. Recent advances in human neurophysiology: Proceedings of the 6th International Evoked Potentials Symposium held in Okazaki, Japan, 21-25 March 1998. Amsterdam: Elsevier, 1998.

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8

Herzwahrnehmung und hirnelektrische Aktivität: Eine Analyse der topographischen Verteilung von herzschlag-synchron evozierten Potentialen (HEP). Frankfurt am Main: P. Lang, 1994.

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9

Baumgartner, Christoph. Clinical electrophysiology of the somatosensory cortex: A combined study using electrocorticography, scalp-EEG, and magnetoencephalography. Wien: Springer-Verlag, 1993.

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10

A, DeLisa Joel, and Lee Hang J, eds. Manual of nerve conduction study and surface anatomy for needle electromyography. 4th ed. Philadelphia: Lippincott Wilkins and Williams, 2005.

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11

Vredeveld, Jan W. Somatosensory evoked potentials (median nerve stimulation) in acute stroke: A prospective study of the SSEP-N20 in relation to the recovery from acute stroke and a discussion of the source of the N20 in relation with CT-scan findings. Lisse: Swets & Zeitlinger, 1985.

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12

1951-, Urban Laszlo, North Atlantic Treaty Organization. Scientific Affairs Division., and NATO Advanced Research Workshop on Cellular Mechanisms of Sensory Processing (1993 : Wye, England), eds. Cellular mechanisms of sensory processing: The somatosensory system. Berlin: Springer-Verlag, 1994.

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13

Furlong, Paul Lawrence. A study of the somatosensory evoked potential in man using brain mapping techniques. Birmingham: AstonUniversity. Department of Vision Sciences, 1990.

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14

Mauguière, François, and Luis Garcia-Larrea. Somatosensory and Pain Evoked Potentials. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0043.

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This chapter discusses the use of somatosensory evoked potentials (SEPs) and pain evoked potentials for diagnostic purposes. The generators of SEPs following upper limb stimulation have been identified through intracranial recordings, permitting the analysis of somatosensory disorders caused by neurological diseases. Laser activation of fibers involved in thermal and pain sensation has extended the applications of evoked potentials to neuropathic pain disorders. Knowledge of the effects of motor programming, paired stimulations, and simultaneous stimulation of adjacent somatic territories has broadened SEP use in movement disorders. The recording of high-frequency cortical oscillations evoked by peripheral nerve stimulation gives access to the functioning of SI area neuronal circuitry. SEPs complement electro-neuro-myography in patients with neuropathies and radiculopathies, spinal cord and hemispheric lesions, and coma. Neuroimaging has overtaken SEPs in detecting and localizing central nervous system lesions, but SEPs still permit assessment of somatosensory and pain disorders that remain unexplained by anatomical investigations.

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15

Nuwer, Marc R. Evoked Potentials. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199341016.003.0009.

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Visual evoked potentials, brainstem auditory evoked potentials, and somatosensory evoked potentials are established clinical tests that are useful for the diagnosis of multiple sclerosis. Motor evoked potentials, cognitive event-related potentials, and vestibular evoked potentials also are used clinically to test additional pathways and functions. These objective, reproducible tools can identify clinically silent lesions, predict clinical deterioration risk, and localize levels of impairment. They differ from magnetic resonance imaging in that they assess function rather than anatomy and thereby fill a complementary role in clinical care. They also are useful in therapeutic trials because they can predict outcomes in parallel with, or earlier than, clinical examinations.

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16

Buchner, Helmut. Evoked potentials. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.003.0015.

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Evoked potentials (EPs) occur in the peripheral and the central nervous system. The low amplitude signals are extracted from noise by averaging multiple time epochs time-locked to a sensory stimulus. The mechanisms of generation, the techniques for stimulation and recording are established. Clinical applications provide robust information to various questions. The importance of EPs is to measure precisely the conduction times within the stimulated sensory system. Visual evoked potentials to a pattern reversal checker board stimulus are commonly used to evaluate the optic nerve. Auditory evoked potentials following ‘click’ stimuli delivered by a headset are most often used to test the auditory nerve and for prognostication in comatose patients. Somatosensory evoked potentials to electrical stimulation of distal nerves evaluate the peripheral nerve and the lemniscal system, and have various indications from demyelinating diseases to the monitoring of operations and prognosis of comatose patients.

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17

BAUMGARTNER, CHRISTOPHER. Clinical Electrophysiology Of The Somatosensory Cortex. Springer, 1993.

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18

Somatosensory Evoked Potentials, Median Nerve Stimulation, in Acute Stroke. Swets & Zeitlinger, 1985.

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19

A, DeLisa Joel, and DeLisa Joel A, eds. Manual of nerve conduction velocity and clinical neurophysiology. 3rd ed. New York: Raven Press, 1994.

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20

Jameson, Leslie. Acute Loss of Intraoperative Evoked Potential Signals. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0069.

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Monitoring of somatosensory and motor evoked potentials has become the standard of care for a large proportion of spine surgeons. Understanding how anesthetic management may affect these evoked potentials is critical to optimizing the ability to detect impending spinal cord or peripheral nerve injury. Similarly, once a nerve injury is detected, knowledge of the various anesthetic and surgical maneuvers possible to avoid permanent injury is essential for the best patient outcomes. This chapter discusses the effects of various anesthetic agents on somatosensory and motor evoked potentials and potential critical interventions that can be made when a nerve injury is identified by this monitoring.

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21

Brock, Mario, Reinhold A. Frowein, and Margareta Klinger. Head Injuries: Prognosis Evoked Potentials Microsurgery Brain Death. Springer London, Limited, 2012.

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22

DeLisa, Joel A., and Hang J. Lee. Manual of Nerve Conduction Study and Surface Anatomy for Needle Electromyography. 4th ed. Lippincott Williams & Wilkins, 2004.

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23

K, Salzman Steven, and Smith Shirley J. 1964-, eds. Neural monitoring: The prevention of intraoperative injury. Clifton, N.J: Humana Press, 1990.

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24

Salzman, Steven K. Neural Monitoring: The Prevention of Intraoperative Injury. Humana Press, 2012.

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25

Head Injuries. Springer Verlag, 1989.

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26

Koht, Antoun, and Tod B. Sloan. Neurophysiologic Monitoring. Edited by David E. Traul and Irene P. Osborn. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190850036.003.0028.

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Intraoperative neurophysiologic monitoring is used for monitoring and mapping of neurological structures during surgery and procedures where the neurological structures are at risk. Among the most commonly used techniques are electrophysiologic techniques, which include spontaneous and evoked electromyography, somatosensory evoked potentials, motor evoked potentials, electroencephalography, and auditory brainstem responses. These methods differ in their responses to anesthesia and in their clinical contribution to monitoring because of differing anatomy. Their use in spinal corrective surgery highlights the role of the anesthesiologist during cases when these techniques are utilized. Optimization of anesthesia, position, and physiology provide better monitoring conditions, enhance signal evaluation, and may lead to better neurological outcome.

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27

Urban, Laszlo. Cellular Mechanisms of Sensory Processing: The Somatosensory System. Springer London, Limited, 2013.

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28

Urban, Laszlo. Cellular Mechanisms of Sensory Processing: The Somatosensory System. Springer London, Limited, 2011.

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29

Tactile Spatial Attention in the Human EEG: Influences of Task Difficulty and Task Relevance. Leipzig, Germany: Leipziger Universitäts-Verlag, 2010.

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30

Peripheral nerve lesions. Berlin: Springer-Verlag, 1990.

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31

Moore, Michael R., and Ehab Farag. Unstable Cervical Spine and Airway Management. Edited by David E. Traul and Irene P. Osborn. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190850036.003.0012.

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In patients with cervical myelopathy, the spinal cord is already compromised to a point at which there is little reserve for surgical maneuvers and the slightest adverse action can result in dramatic consequences. Awake fiberoptic intubation and neurological assessment before induction of anesthesia could be the safest way to avoid waking up the patient before proceeding with surgery in the case of absent motor evoke potentials (MEPs) in spite of increasing the stimulating voltage together with increasing the rate of stimulating pulses. Hypotension is an additional factor, which may lead to irreversible neurologic deficit in a partially compressed but functionally intact spinal cord. Intraoperative neurophysiologic monitoring for cervical myelopathy should include somatosensory evoked potentials, transcranial electric MEPs, and electromyography to provide complementary information and monitor different spinal cord tracts and individual nerve roots.

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32

Brallier, Jess W., and Jonathan S. Gal. Neuroprotection for Spine Surgery. Edited by David L. Reich, Stephan Mayer, and Suzan Uysal. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190280253.003.0020.

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Perioperative neurologic injury related to spine surgery, albeit rare, can result in devastating functional loss. As the number of spine operations has increased, so has the need for strategies designed to avoid and protect against such injury. This chapter reviews the common etiologies of neurologic deficits secondary to spine surgery and the factors that place patients at increased risk for developing these complications. The use of intraoperative neuromonitoring, including somatosensory evoked potentials (SSEPs), electromyography (EMG), and transcranial motor evoked potentials (TcMEPs), to detect surgical trespass of neuronal elements is also reviewed. The authors also summarize the role of physiologic parameter optimization, including mean arterial blood pressure and body temperature, and pharmacologic interventions, should an injury occur. Current practice guidelines for preventing and managing perioperative neurologic injury are described.

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33

Fowle, Adrian J. Clinical neurophysiology of the pelvic floor. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.003.0017.

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This chapter offers a personal view of a service providing pelvic floor studies. It first debunks the myths that put patients and doctors off. A practical approach is outlined to performing the most worthwhile studies from referral to report. Care of the patient and understanding of the anatomy and physiology are emphasized as necessary for the performance of the studies. The most useful studies, examination of the external anal sphincter, and either bulbocavernosus reflex or pudendal somatosensory evoked potentials, are highlighted. Finally, the conditions in which these studies may be useful are discussed, including cauda-equina syndrome, post-partum incontinence, and neurological conditions.

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34

Urban, Laszlo. Cellular Mechanisms of Sensory Processing: The Somatosensory System (Nato a S I Series Series H, Cell Biology). Springer-Verlag Telos, 1994.

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35

Legatt, Alan D., Marc R. Nuwer, and Ronald G. Emerson. Intraoperative Monitoring of Central Neurophysiology. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0034.

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This chapter covers neurophysiological intraoperative monitoring (NIOM). It describes the relevant neurophysiological signals, their anatomical sources, the techniques used to record them, the manner in which they are assessed, and possible causes of intraoperative signal changes. Techniques used include electroencephalography (EEG), electromyography, and auditory, somatosensory, and motor evoked potentials. Some of these techniques can be used to localize and identify areas of cerebral cortex or the corticospinal tract. Recording of the electromyogram generated by reflex activity can be used to evaluate central nervous system function in some circumstances. EEG can be used to assess depth of anesthesia. Signals can be affected by anesthesia, and the chapter discusses various anesthetic agents, their effects on signals, and considerations for anesthetic management during NIOM. Personnel performing NIOM must be knowledgeable about the anatomy and physiology underlying the signals, the technology used to record them, and the factors (including anesthesia) that can affect them.

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36

Chandross, David Maury. The comparison of the role of long interstimulus interval and focussed attention in the enhancement of the P300 in the human somatosensory evoked potential. 1986.

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37

Buetefisch, Cathrin M., and Leonardo G. Cohen. Use-dependent changes in TMS measures. Edited by Charles M. Epstein, Eric M. Wassermann, and Ulf Ziemann. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780198568926.013.0018.

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Adult brains maintain the ability to reorganize throughout life. Cortical reorganization or plasticity includes modification of synaptic efficacy as well as neuronal networks that carry behavioural implications. Transcranial magnetic stimulation (TMS) allows for the study of primary motor cortex reorganization in humans. Motor-evoked potential (MEP) amplitudes change in response to practice. This article gives information about the effect of practice on TMS measures such as motor-evoked potential amplitudes, motor maps, paired-pulse measures, and behavioural measures. These changes may be accompanied by down-regulation of activity in nearby body part representations within the same hemisphere and in homonymous regions of the opposite hemisphere, mediated by interhemispheric interactions. There is evidence pointing towards the influence of practice on a distributed network of cortical representations within regions of cerebral hemispheres. This has lead to the formulation of intervention strategies to enhance the training effects by cortical or somatosensory stimulation in health and disease.

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