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Journal articles on the topic 'Solar keratosis'

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Author: Grafiati

Published: 4 June 2021

Last updated: 20 February 2023

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1

Mark, Brady, Jaxon Dawson, and Dominic Chase. "The Management of Actinic Keratosis and Squamous Cell Carcinoma." Dermatology and Dermatitis 2, no. 1 (February 26, 2018): 01–03. http://dx.doi.org/10.31579/2578-8949/019.

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Background: Actinic keratosis or solar keratosis is a common skin lesion caused by sun damage that progresses to squamous cell carcinoma. It has been suggested that actinic keratosis is in fact SCC in situ. Objective: This literature review was conducted to investigate the differences between actinic keratosis and squamous cell carcinoma and whether actinic keratosis should in fact be managed as squamous cell carcinoma. Methods: A literature review was conducted to assess the differences between actinic keratosis and squamous cell carcinoma. We conducted searches of Pubmed, Cochrane and Medline for articles published between January 1, 2000 and April 30, 2014, using the following search terms: actinic keratosis, solar keratosis, skin cancer, squamous cell carcinoma, dermoscopy, sun exposure, ultra violet radiation, and dysplasia. Studies published in English were selected for inclusion in this review as were additional articles identified from bibliographies. Results: It is difficult to distinguish between both actinic keratosis and squamous cell carcinoma. Perhaps a classification system for actinic keratosis including early in situ SCC type AK1, early in situ SCC type AK2 and in situ SCC type actinic keratosis is needed. Conclusion: Actinic keratosis invades the basement membrane and as such may progress into invasive SCC. Superficially actinic keratoses are not distinguishable from a superficial SCC and as such may go unrecognized or inaccurately diagnosed.

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2

Niu, Dongfeng, Yanhua Bai, Qian Yao, Wei Hou, Lixin Zhou, Xiaozheng Huang, and Chen Zhao. "Expression and Significance of AQP3 in Cutaneous Lesions." Analytical Cellular Pathology 2021 (October 26, 2021): 1–6. http://dx.doi.org/10.1155/2021/7866471.

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Aquaporin 3 (AQP3) is the membrane channel of water and involved in fluid homeostasis. The aim of this study was to reveal the expression and significance of AQP3 in cutaneous lesions. We analyzed AQP3 mRNA levels using RT-PCR in 311 cutaneous lesions and confirmed AQP3 expression in these lesions by immunohistochemistry. AQP3 mRNA was detected in normal epidermis, seborrheic keratosis, solar keratosis, Bowen’s disease, squamous cell carcinoma, eccrine poroma, apocrine carcinoma, and sebaceoma; however, AQP3 mRNA was absent in basal cell carcinoma, nevocellular nevus, or malignant melanoma. By immunohistochemistry, diffuse AQP3 expression was seen in all keratotic lesions including seborrheic keratosis, verruca vulgaris, molluscum contagiosum, solar keratosis, Bowen’s disease, and squamous cell carcinoma. Diffuse AQP3 expression was also present in all extramammary Paget’s disease. No AQP3 staining was obtained in basal cell carcinoma. Positive AQP3 staining was seen in sweat gland tumors including hidradenoma, eccrine poroma, and apocrine carcinoma. Among sebaceous tumors, AQP3 expressed diffusely in all sebaceous hyperplasia and sebaceous adenoma, but not in sebaceous carcinomas. Only focal AQP3 staining was seen in nevocellular nevus and no AQP3 staining in melanoma. Our findings indicate the function of AQP3 maintained in most skin tumors. AQP3 may be used for differential diagnosis in skin tumors.

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3

Ishioka, Priscila, Sílvio Alencar Marques, Amélia Toyomi Hirai, Mariangela E. A. Marques, Sérgio Henrique Hirata, and Sérgio Yamada. "Prevalence of precancerous skin lesions and non-melanoma skin cancer in Japanese-Brazilians in Bauru, São Paulo State, Brazil." Cadernos de Saúde Pública 25, no. 5 (May 2009): 965–71. http://dx.doi.org/10.1590/s0102-311x2009000500003.

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Precancerous lesions and skin cancer are infrequent in Asians, and have received little documentation in the literature. Brazil has the world's largest contingent of Japanese immigrants and their descendants, and 70% live in the State of São Paulo. The prevalence of such skin lesions in Japanese-Brazilians is unknown. This study aimed to assess the prevalence of actinic keratoses and non-melanoma skin cancer in first and second-generation Japanese-Brazilians over 30 years of age, without miscegenation, living in the city of Bauru, São Paulo State, in 2006. Of the 567 Japanese-Brazilians that underwent dermatological examination, actinic keratosis was diagnosed in 76, with a mean age of 68.9 years, and a single case of basal cell carcinoma was detected in a 39-year-old female patient. In Japan, prevalence of actinic keratosis varies from 0.76% to 5%, and the incidence of non-melanoma skin cancer is 1.2 to 5.4/100 thousand. Japanese-Brazilians from Bauru showed a 13.4% prevalence of actinic keratoses and earlier age at onset. Proximity to the Equator and a history of farming contribute to these higher rates. Presence of solar melanosis was associated with a 1.9-fold risk of developing actinic keratosis.

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4

Fu, Wendy, and Clay J. Cockerell. "The Actinic (Solar) Keratosis." Archives of Dermatology 139, no. 1 (January 1, 2003): 66. http://dx.doi.org/10.1001/archderm.139.1.66.

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5

PAPADOGIANNAKIS, E., and V. KONTOS. "IA case of canine squamous cell carcinoma secondary to solar keratosis (actinic carcinoma in situ)." Journal of the Hellenic Veterinary Medical Society 59, no. 1 (March 22, 2018): 64. http://dx.doi.org/10.12681/jhvms.14949.

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We present the case of a squamous cell carcinoma developed in a pre-existing solar keratosis of the trunk in a whitehaired Dogue Argentino. Deep pyoderma along with exfoliative erythroderma were the initial clinical findings. Several months after the diagnosis of solar keratosis, the dog presented with multiple nodules and ulcers on the flank and abdomen and the diagnosis of malignancy was confirmed microscopically.

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6

Hurt, M. A. "The nature of solar (actinic) keratosis." British Journal of Dermatology 156, no. 2 (February 2007): 408–9. http://dx.doi.org/10.1111/j.1365-2133.2006.07689.x.

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7

Ackerman, A. Bernard. "Solar Keratosis Is Squamous Cell Carcinoma." Archives of Dermatology 139, no. 9 (September 1, 2003): 1216. http://dx.doi.org/10.1001/archderm.139.9.1216.

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8

Karadaglić, Đorđije, and Marina Jovanović. "Actinic Keratosis: A New Approach to the Treatment / Aktinična Keratoza: Novi Pristup Lečenju." Serbian Journal of Dermatology and Venerology 1, no. 1 (January 1, 2009): 27–33. http://dx.doi.org/10.2478/v10249-011-0004-1.

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Abstract Actinic keratosis is an intraepidermal proliferation of transformed, atypical keratinocytes, induced by exposure to solar ultraviolet radiation. Many authors believe that it is the earliest form of squamous cell carcinoma. More than 40% of all metastatic squamous cell carcinomas develop from actinic keratosis. The clinical, histological and molecular characteristics of actinic keratosis are those of squamous cell carcinomas. Since it can be extremely hard to distinguish actinic keratosis from some squamous cell carcinomas, treatment can be rather difficult. The best treatment of actinic keratosis is its prevention. The main reason for therapy which is universally accepted, is prevention of squamous cell carcinoma. A number of options are available, but when considering the efficacy, invasive procedures remain the standard treatment. Treatment of individual lesions may prevent further progression of actinic damage present in the surrounding skin

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9

Ackerman, A. B., and J. M. Mones. "Solar (actinic) keratosis is squamous cell carcinoma." British Journal of Dermatology 155, no. 1 (February 20, 2006): 9–22. http://dx.doi.org/10.1111/j.1365-2133.2005.07121.x.

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10

Holmes, Cara, Peter Foley, Michael Freeman, and Alvin H. Chong. "Solar keratosis: Epidemiology, pathogenesis, presentation and treatment." Australasian Journal of Dermatology 48, no. 2 (May 2007): 67–76. http://dx.doi.org/10.1111/j.1440-0960.2007.00339.x.

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11

ACKERMAN, A. "WS095 Solar keratosis is squamous cell carcinoma." Journal of the European Academy of Dermatology and Venereology 9 (September 1997): S98—S99. http://dx.doi.org/10.1016/s0926-9959(97)89254-9.

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12

Millanta, Francesca, Francesca Parisi, Alessandro Poli, Virginia Sorelli, and Francesca Abramo. "Auricular Non-Epithelial Tumors with Solar Elastosis in Cats: A Possible UV-Induced Pathogenesis." Veterinary Sciences 9, no. 2 (January 18, 2022): 34. http://dx.doi.org/10.3390/vetsci9020034.

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The photoinduced etiopathology of actinic keratosis and squamous cell carcinoma in feline species is well known. This etiology has also been reported for non-epithelial cutaneous tumors in other species. To date, no cases of auricular non-epithelial cutaneous neoplasms erased in a contest of actinic keratosis in cats have been reported. The aim of this study was to describe feline auricular non-epithelial cutaneous neoplasms associated with typical UV-induced cutaneous lesions and solar elastosis. The study was conducted on five feline cases diagnosed with auricular non-epithelial cutaneous tumors (two fibrosarcomas, one mixosarcoma, one epithelioid melanoma and one hemangiosarcoma), selected from the Tumor Registry of the Department of Veterinary Sciences of the University of Pisa (1998–2018). Ten and six feline auricular biopsies of normal skin and skin with actinic keratosis, respectively, were used as controls. Orcein stain was used to investigate solar elastosis. Histological changes related to chronic solar irradiation were documented in the skin adjacent to the neoplastic lesions in the five cats. Considering the anatomical localization and the results of histopathology, this study suggests that non-epithelial cutaneous neoplasms may have a UV-induced etiopathogenesis in the feline species.

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13

Watkins, Jean. "Dermatology and the community nurse: actinic (solar) keratosis." British Journal of Community Nursing 15, no. 1 (January 2010): 6–11. http://dx.doi.org/10.12968/bjcn.2010.15.1.45777.

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14

YAMADA, Natsue, Hiroko KOMI, Megumi KONDOU, Mitsuya OKAJIMA, Tetsuo SASAKI, Tetsuo NAGATANI, and Hiroshi NAKAJIMA. "A case of keratoacanthoma complicating multiple solar keratosis." Skin Cancer 14, no. 1 (1999): 129–33. http://dx.doi.org/10.5227/skincancer.14.129.

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15

Cockerell, C. J. "Pathology and pathobiology of the actinic (solar) keratosis." British Journal of Dermatology 149, s66 (November 2003): 34–36. http://dx.doi.org/10.1046/j.0366-077x.2003.05625.x.

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16

FUKAMIZU, HIDEKAZU, KUNIO INOUE, KICHIRO MATSUMOTO, HIDEYO OKAYAMA, and TAKAHIKO MORIGUCHI. "Metastatic Squamous-Cell Carcinomas Derived from Solar Keratosis." Journal of Dermatologic Surgery and Oncology 11, no. 5 (May 1985): 518–22. http://dx.doi.org/10.1111/j.1524-4725.1985.tb01413.x.

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17

Chung, Hye Jin, Kelly L. McGuigan, Katie L. Osley, Kate Zendell, and Jason B. Lee. "Pigmented solar (actinic) keratosis: An underrecognized collision lesion." Journal of the American Academy of Dermatology 68, no. 4 (April 2013): 647–53. http://dx.doi.org/10.1016/j.jaad.2012.10.050.

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18

Adil, Dehhaze, Ahmar-Rass Nour El Imane, Diher Issam, Taybi Othmane, Labbaci Rim, and Daghouri Nada-Imane. "ANATOMOCLINICAL CONFRONTATION BETWEEN MELANOMA, SOLAR LENTIGO AND SEBORRHEIC KERATOSIS: A CASE REPORT." International Journal of Advanced Research 9, no. 10 (October 31, 2021): 728–31. http://dx.doi.org/10.21474/ijar01/13603.

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The primary diagnosis in case of a rapidly developing facipigmented lesion in an elderly patient is Dubreuilhs melanoma . This case report is highlighting the issue of anatomoclinical confrontation between a clinically suspected melanoma and a pathologically confirmed solar lentigo and seborrheic keratosis.

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19

Misago, Noriyuki, Tatsurou Tanaka, and Hiromu Kohda. "Trichilemmal Carcinoma Occurring in a Lesion of Solar Keratosis." Journal of Dermatology 20, no. 6 (June 1993): 358–64. http://dx.doi.org/10.1111/j.1346-8138.1993.tb01298.x.

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20

Kaptanoglu, AF, and L. Kutluay. "Keratoacanthoma developing in previous cryotherapy site for solar keratosis." Journal of the European Academy of Dermatology and Venereology 20, no. 2 (February 2006): 197–98. http://dx.doi.org/10.1111/j.1468-3083.2005.01370.x.

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21

Sim, C. S., S. Slater, and P. H. McKee. "Mutant p53 expression in solar keratosis: an immunohistochemical study." Journal of Cutaneous Pathology 19, no. 4 (August 1992): 302–8. http://dx.doi.org/10.1111/j.1600-0560.1992.tb01366.x.

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22

Roewert-Huber, J., E. Stockfleth, and H. Kerl. "Pathology and pathobiology of actinic (solar) keratosis - an update." British Journal of Dermatology 157 (December 7, 2007): 18–20. http://dx.doi.org/10.1111/j.1365-2133.2007.08267.x.

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23

Buinauskaite, Evelina, Jurgita Makstiene, Jurate Buinauskiene, and Skaidra Valiukeviciene. "Reliability of solar keratosis clinical diagnosis: A prospective study." Australasian Journal of Dermatology 56, no. 2 (March 17, 2014): e49-e52. http://dx.doi.org/10.1111/ajd.12095.

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24

HURWITZ, ROBERT M., and LAWRENCE E. MONGER. "Solar Keratosis: An Evolving Squamous Cell Carcinoma. Benign or Malignant?" Dermatologic Surgery 21, no. 2 (February 1995): 184. http://dx.doi.org/10.1111/j.1524-4725.1995.tb00141.x.

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25

Almuayqil, Saleh Naif, Sameh Abd El-Ghany, and Mohammed Elmogy. "Computer-Aided Diagnosis for Early Signs of Skin Diseases Using Multi Types Feature Fusion Based on a Hybrid Deep Learning Model." Electronics 11, no. 23 (December 2, 2022): 4009. http://dx.doi.org/10.3390/electronics11234009.

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According to medical reports and statistics, skin diseases have millions of victims worldwide. These diseases might affect the health and life of patients and increase the costs of healthcare services. Delays in diagnosing such diseases make it difficult to overcome the consequences of these types of disease. Usually, diagnosis is performed using dermoscopic images, where specialists utilize certain measures to produce the results. This approach to diagnosis faces multiple disadvantages, such as overlapping infectious and inflammatory skin diseases and high levels of visual diversity, obstructing accurate diagnosis. Therefore, this article uses medical image analysis and artificial intelligence to present an automatic diagnosis system of different skin lesion categories using dermoscopic images. The addressed diseases are actinic keratoses (solar keratoses), benign keratosis (BKL), melanocytic nevi (NV), basal cell carcinoma (BCC), dermatofibroma (DF), melanoma (MEL), and vascular skin lesions (VASC). The proposed system consists of four main steps: (i) preprocessing the input raw image data and metadata; (ii) feature extraction using six pre-trained deep learning models (i.e., VGG19, InceptionV3, ResNet50, DenseNet201, and Xception); (iii) features concatenation; and (iv) classification/diagnosis using machine learning techniques. The evaluation results showed an average accuracy, sensitivity, specificity, precision, and disc similarity coefficient (DSC) of around 99.94%, 91.48%, 98.82%, 97.01%, and 94.00%, respectively.

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26

Bujnauskajte, E., S. Rubins, A. Rubins, Y. Makstiene, and Y. Bujnauskiene. "Can the lesions induced with actinic keratosis become an enigma for dermatologists? Results of an open perspective study." Vestnik dermatologii i venerologii 93, no. 2 (April 24, 2017): 53–59. http://dx.doi.org/10.25208/0042-4609-2017-93-2-53-59.

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Different face skin diseases (basal cell carcinoma, actinic keratosis, rosacea, solar elastosis, etc.) could clinically manifest itself as erythematic patches, pimples or plagues. It is very hard to make the clinical exclusion in some cases of these diseases since their characters can partially overlap or certain lesion can mimic another one especially in the cases of skin areas affected with sun. Therefore, the histopathological analysis remains the “golden standard” of the dermatological diagnosis at skin diseases. Our study has shown that certified dermatologists detect actinic keratosis (AK) of face/head skin of I/II levels very well. Verdicts of dermatologists and pathomorfologists are congruent on account of diagnosis in 90,7% cases. Diseases clinically excluded as AK revealed as malignant neoplasms (basal cell carcinoma) in less than 1% of case lesions.

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27

Znamenskaya, L. F., V. V. Chikin, I. A. Kappusheva, and V. V. Kondrasheva. "ISSEMINATED SUPERFICIAL ACTINIC POROKERATOSIS IN PATIENT SUFFERING FROM PLAQUE PSORIASIS: A CASE REPORT." Vestnik dermatologii i venerologii 91, no. 5 (October 24, 2015): 91–96. http://dx.doi.org/10.25208/0042-4609-2015-91-5-91-96.

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Monitoring of disseminated superficial actinic porokeratosis (DSAP) in patient suffering from plaque psoriasis treated by PUVA therapy was presented. DSAP is a rare skin disease developing in the result of keratosis disorder caused by solar radiation and appearing as clearly localized ringed or line plates. As patients with DSAP are sensitive to solar radiation, phototherapy is contraindicated for them. In this regard, subcutaneous injections of Methotrexate 10 mg once a week were prescribed to a patient suffering from psoriasis. Psoriatic rashes were regressed after the second injection, instead of them DSAP rashes remained.

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28

Karkuzhali, Ponnuswamy, Nellainayagam Gomathy, and ParveenBashir Ahamed. "Sebaceous carcinoma of cheek arising in a lesion of solar keratosis." Indian Journal of Dermatology 54, no. 5 (2009): 16. http://dx.doi.org/10.4103/0019-5154.45434.

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29

Heaphy, Michael R., and A. Bernard Ackerman. "The nature of solar keratosis: A critical review in historical perspective." Journal of the American Academy of Dermatology 43, no. 1 (July 2000): 138–50. http://dx.doi.org/10.1067/mjd.2000.107497.

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30

Marks, R. "Who benefits from calling a solar keratosis a squamous cell carcinoma?" British Journal of Dermatology 155, no. 1 (February 20, 2006): 23–26. http://dx.doi.org/10.1111/j.1365-2133.2005.07122.x.

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31

YONEYAMA, Shigeki, Tetsuya SHIOTA, Shinichi FUJISAKA, Minoru TAKATA, Toshiaki SAIDA, Sumio MINEMATSU, Hiroshi TAKEI, and Sueo MIYAKI. "Establishment of a UV-induced solar keratosis model using hairless mice." Skin Cancer 21, no. 2 (2006): 226–31. http://dx.doi.org/10.5227/skincancer.21.226.

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32

Lane, Hilary, Sean O’Loughlin, Frank Powell, Hilary Magee, and Peter A. Dervan. "A Quantitative Immunohistochemical Evaluation of Lentigo Maligna and Pigmented Solar Keratosis." American Journal of Clinical Pathology 100, no. 6 (December 1, 1993): 681–85. http://dx.doi.org/10.1093/ajcp/100.6.681.

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33

Taguchi, M., T. Tsuchida, S. Ikeda, S. Watanabe, and T. Sekiya. "070 Aberrations of p53 gene and ras genes in solar keratosis." Journal of Dermatological Science 15, no. 2 (August 1997): 114. http://dx.doi.org/10.1016/s0923-1811(97)81773-2.

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34

Memon, A. A., J. A. Tomenson, J. Bothwell, and P. S. Friedmann. "Prevalence of solar damage and actinic keratosis in a Merseyside population." British Journal of Dermatology 142, no. 6 (June 2000): 1154–59. http://dx.doi.org/10.1046/j.1365-2133.2000.03541.x.

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35

Marks, R. "Prevalence of solar damage and actinic keratosis in a Merseyside population." British Journal of Dermatology 144, no. 2 (February 2001): 437. http://dx.doi.org/10.1046/j.1365-2133.2001.04055.x.

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36

Gimeno-Carrero, Mónica, María-Jesús Suárez-Fernández, Beatriz Alonso-Martín, Almudena de-Pablo-Cabrera, María-Concepción Garrido-Ruíz, and Enrique Mencía-Gutiérrez. "Conjunctival Tarsal Actinic Keratosis Treated with Interferon Alfa-2b: A Rare Case Report and Literature Review." Case Reports in Ophthalmological Medicine 2021 (January 22, 2021): 1–5. http://dx.doi.org/10.1155/2021/6616021.

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Conjunctival neoplasia is one of the most frequent tumors in the eye. Actinic keratosis (AK) or solar keratosis is a precancerous lesion that is included with other epithelial tumors. This alteration does not break the basal membrane. There is enough evidence of successful outcomes to consider interferon alfa-2b (IFN alfa-2b) as the first choice of treatment for this type of tumors. In addition, side effects are mild and uncommon. We report a case in an 83-year-old woman who was referred to evaluate a leukoplakia in the tarsal conjunctiva of the lower left eyelid that measured 1 cm in diameter. Pathological study revealed AK. After the INF alfa-2b treatment, we observed conjunctival hyperemia, noninfiltrated upper nasal de-epithelization, and inferior nasal bulla. AK with presentation in conjunctiva is rarely described and in tarsal conjunctiva is exceptional. It is the first case published with only tarsal conjunctiva affectation.

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37

El-Mongy, Shaymaa, Nora Darwish, and Mohamed Gab Allah. "EVALUATION OF CERTAIN THERAPEUTIC MODALITIES IN TREATMENT OF SOLAR KERATOSIS AND SOLAR LENTIGINES: A COMPARATIVE STUDY." Mansoura Medical Journal 37, no. 2 (December 1, 2008): 335–55. http://dx.doi.org/10.21608/mjmu.2008.129207.

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38

KOTSUJI, Tomoe, Yoshihiro UMEBAYASHI, Mitsuru IWATA, and Fujio OTSUKA. "A case of malignant trichilemmoma produced in solar keratosis by immunohistochemistry dyeing." Skin Cancer 12, no. 3 (1997): 406–9. http://dx.doi.org/10.5227/skincancer.12.406.

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39

Tsukifuji, R., and H. Shinkai. "071 Expressions of MMP-1, p53, metallothionein and PCNA in solar keratosis." Journal of Dermatological Science 15, no. 2 (August 1997): 114. http://dx.doi.org/10.1016/s0923-1811(97)81771-9.

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40

Ichikawa, Eiko, Shinichi Watanabe, and Fujio Otsuka. "Immunohistochemical Localization of Keratins and Involucrin in Solar Keratosis and Bowenʼs Disease." American Journal of Dermatopathology 17, no. 2 (April 1995): 151–57. http://dx.doi.org/10.1097/00000372-199504000-00007.

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41

Mazzilli, Sara, Reyes Gamo-Villegas, Ana Pampin-Franco, Jose Luis Lopez Estebaran, Fernando Pinedo, Laura Vollono, Monia Di Prete, Elena Campione, and Salvador Gonzalez. "Reflectance Confocal Microscopy of Pigmented Bowen’s Disease: A Case Series of Difficult to Diagnose Lesions." Case Reports in Dermatology 12, no. 2 (May 15, 2020): 98–106. http://dx.doi.org/10.1159/000507916.

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Pigmented Bowen’s disease is a rare variant of in situ squamous skin cell carcinoma. It mainly affects patients between 60 and 70 years of age. Its clinical features include well-demarcated, pigmented plaque arising in photo-exposed areas of the body. The best-characterized feature of the disease by histological examination is the presence of atypical keratinocytes, hyperpigmentation of the epidermis with trans-epidermal elimination of melanin and dermal melanophages. Precise diagnosis is often difficult, both clinically and dermoscopically, as Bowen’s disease is often mistaken with keratinocyte tumors such as solar lentigines, seborrheic keratosis, Bowenoid papulosis, pigmented basal cell carcinoma, pigmented actinic keratosis; or even melanocytic lesions such as melanocytic nevus, pigmented epithelioid melanocytoma, and melanoma. Precise diagnosis often requires biopsy and histopathological examination of the tissue. Reflectance confocal microscopy is a noninvasive technique to diagnose pigmented skin lesions. To date, not much data are available regarding its use in the diagnosis of pigmented Bowen’s disease. Herein, we report a well-represented case series of pigmented Bowen’s disease imaged using dermoscopy and reflectance confocal microscopy.

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42

Snarskaya, Elena S., Zhaber Mukhamed Awad, and L. M. Shnahova. "COMBINED PHOTOINDUCED PATHOLOGY OF THE SKIN AND EYES IN PATIENTS WITH DERMATOHELIOSIS." Russian Journal of Skin and Venereal Diseases 22, no. 1-2 (April 15, 2019): 4–10. http://dx.doi.org/10.17816/dv42929.

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Modern information on the mechanisms of the damaging effect of UV radiation on the skin and the eye is presented, constitutional phototype skin (according to the Fitzpatrick scale) and clinical stages of skin photoaging are described (according to R. Glogau). The range of clinical changes arising on the background of solar elastosis can vary depending on the anatomical site of the lesion and include a number of dermatoses included in the concept of solar elastosis syndromes, resulting in the development of malignant dermatoses. Depending on the type of UV exposure - sporadic short-term or systematic long-term, acute and chronic pathological processes caused by the action of UV may develop. The development of eyelid neoplasms (basal cell, metatypical, squamous cell carcinoma, melanoma), solar keratosis of the conjunctiva, pterygium, cataract, and macular retinal degeneration may result from the systematic UV irradiation of the organs of vision.

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43

Khlebnikova, Albina, Anatoliy Molochkov, Yelena Selezneva, Tatyana Sedova, and L. Belova. "FACTORS ASSOCIATED WITH BASALIOMA IN INHABITANTS OF THE MOSCOW REGION." Problems in oncology 64, no. 5 (May 1, 2018): 633–37. http://dx.doi.org/10.37469/0507-3758-2018-64-5-633-637.

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There were studied the factors associated with basalioma in inhabitants of the Moscow region. 169 patients with solitary and multiple basiolioma and 130 controls were examined. 48 factors were assessed. Among the high-importance factors there were I skin phototype, actinic keratosis, solar cheilitis, actinic elastosis, rhomboid wrinkles, weighed heredity in oncological skin diseases, radiation therapy in the anamnesis. Less important factors were as follows: sunburn in childhood, living in regions with radiation pollution, the presence of occupational hazards. Multiple basalioma more often developed in persons who had various occupational hazards.

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44

Goldenhersh, M. A., R. L. Barnhill, H. M. Rosenbaum, and K. S. Stenn. "Documented evolution of a solar lentigo into a solitary lichen planus-like keratosis." Journal of Cutaneous Pathology 13, no. 4 (August 1986): 308–11. http://dx.doi.org/10.1111/j.1600-0560.1986.tb01527.x.

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Terada, Tadashi, Jun Yamagami, Atsushi Fugimoto, Kyoko Tanaka, and Makoto Sugiura. "Pigmented squamous cell carcinoma of the cheek skin probably arising from solar keratosis." Pathology International 53, no. 7 (July 2003): 468–72. http://dx.doi.org/10.1046/j.1440-1827.2003.01495.x.

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46

Goncharova, Yana, Enas A. S. Attia, Khawla Souid, and Inna V. Vasilenko. "Dermoscopic Features of Facial Pigmented Skin Lesions." ISRN Dermatology 2013 (February 3, 2013): 1–7. http://dx.doi.org/10.1155/2013/546813.

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Abstract:

Four types of facial pigmented skin lesions (FPSLs) constitute diagnostic challenge to dermatologists; early seborrheic keratosis (SK), pigmented actinic keratosis (AK), lentigo maligna (LM), and solar lentigo (SL). A retrospective analysis of dermoscopic images of histopathologically diagnosed clinically-challenging 64 flat FPSLs was conducted to establish the dermoscopic findings corresponding to each of SK, pigmented AK, LM, and SL. Four main dermoscopic features were evaluated: sharp demarcation, pigment pattern, follicular/epidermal pattern, and vascular pattern. In SK, the most specific dermoscopic features are follicular/epidermal pattern (cerebriform pattern; 100% of lesions, milia-like cysts; 50%, and comedo-like openings; 37.50%), and sharp demarcation (54.17%). AK and LM showed a composite characteristic pattern named “strawberry pattern” in 41.18% and 25% of lesions respectively, characterized by a background erythema and red pseudo-network, associated with prominent follicular openings surrounded by a white halo. However, in LM “strawberry pattern” is widely covered by psewdonetwork (87.5%), homogenous structureless pigmentation (75%) and other vascular patterns. In SL, structureless homogenous pigmentation was recognized in all lesions (100%). From the above mentioned data, we developed an algorithm to guide in dermoscopic features of FPSLs.

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Gimeno-Castillo, Javier, Adriano Pablo Martínez Aracil, and Aida Menéndez Parrón. "Blue scales on an “iced” scalp." Our Dermatology Online 13, no. 3 (July 1, 2022): 311–12. http://dx.doi.org/10.7241/ourd.20223.18.

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Abstract:

Although the diagnosis of actinic keratosis (AK) is mainly clinical, dermoscopy is a highly useful and accurate diagnostic technique. The dermoscopic features of AK include the strawberry pattern with white areas, keratotic, follicular openings, a pseudonetwork, and rosettes [1]. An 82-year-old male with a personal history of type 3 autoimmune polyglandular syndrome and hypertension was referred to the dermatology department due to the presence of scaly lesions located on the scalp and forehead. A physical examination revealed asymptomatic, keratotic lesions on the forehead and in the occipital region (Fig. 1a). The patient denied the application of any product except toning shampoo for white hair. Interestingly, the lesions had a blue, superficial scale, although they clinically resembled AK. Dermoscopy evidenced an erythematous background with multiple, arctic-blue, keratotic plugs (Fig. 1b). Moreover, a superficial excision of one of the lesions was performed, evidencing solar elastosis and a chronic dermal infiltrate, as well as basal keratinocytes with loss of polarity, presenting with irregular nuclei (Fig. 2). Thus, the clinical and pathological correlation confirmed the diagnosis of AK. The lesions were treated with cryotherapy leading to their resolution. Figure 1: (a) Clinical presentation of the cutaneous lesions: numerous bluish-gray, scaly lesions in the occipital area; the patient confirmed using toning shampoo for white hair containing Acid Violet 43. (b) Dermoscopy of one of the lesions: an erythematous background, as well as numerous arctic-blue, keratotic plugs; the blue scale compatible with the iceberg sign present in AK neglecta. Figure 2: Solar elastosis and a chronic dermal infiltrate, as well as basal keratinocytes with loss of polarity presenting with irregular nuclei; findings compatible with the clinical suspicion of AK; no basophilic amorphous material present in the stratum corneum (H&E, 20×). Blue AK has been termed AK neglecta while the dermoscopic blue coloration the iceberg sign [2,3]. The latter is described to be caused both by sunscreens containing titanium dioxide [3] or shampoos containing Acid Violet (present in the shampoo that the patient used) [2]. Moreover, this exogenous pigmentation may easily be removed with alcohol [3]. Histologically, a basophilic amorphous material on the stratum corneum is described to be present [2]. A lack of this finding in our case might have been explained due to the 10% formol used to preserve the biopsy, eliminating the pigment. Recognizing this presentation is essential due to the widespread use of toning shampoos and sunscreens among the elder population.

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Oiso, Naoki, Tomohiko Narita, and Akira Kawada. "Imiquimod-induced vitiligo-like depigmentation over multiple solar keratosis in a patient with vitiligo." European Journal of Dermatology 27, no. 1 (January 2017): 77–78. http://dx.doi.org/10.1684/ejd.2016.2891.

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Foley, Peter. "Clinical efficacy of methyl aminolaevulinate photodynamic therapy in basal cell carcinoma and solar keratosis." Australasian Journal of Dermatology 46, s3 (February 2005): S8—S10. http://dx.doi.org/10.1111/j.1440-0960.2004.00117.x.

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Foley, Peter. "Clinical efficacy of methyl aminolaevulinate photodynamic therapy in basal cell carcinoma and solar keratosis." Australasian Journal of Dermatology 46, s2 (March 15, 2005): S8—S10. http://dx.doi.org/10.1111/j.1440-0960.2004.00119.x.

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