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Journal articles on the topic 'Sodium monofluoroacetate'

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Published: 4 June 2021

Last updated: 19 February 2022

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1

Kim, J. B., J. W. Jang, and J. H. Kim. "Reversible leukoencephalopathy in sodium monofluoroacetate intoxication." Neurology 82, no. 13 (March 31, 2014): 1190–91. http://dx.doi.org/10.1212/wnl.0000000000000262.

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2

Chi, Chih-Hsien, Tin-Kwang Lin, and Kuan-Wen Chen. "Hemodynamic abnormalities in sodium monofluoroacetate intoxication." Human & Experimental Toxicology 18, no. 6 (June 1999): 351–53. http://dx.doi.org/10.1191/096032799678840200.

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Hypotension is one of the most important predictors of mortality in sodium monofluoroacetate (SMFA) intoxication. This paper reports the hemodynamic response in one fatal and another survival case of SMFA intoxication. Despite correction of hypovolemia and with inotropic support, the patients remained in shock. Hemodynamic observations have provided evidence that shock after SMFA intoxication is due to diminished systemic vascular resistance and increased cardiac output. This is the first report in which such an invasive hemodynamic investigation has been recorded in a clinical case of SMFA intoxication.

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3

Parfitt, R. L., C. T. Eason, H. Hoff, and L. K. Heng. "Sodium monofluoroacetate (1080) leaching through soils." Bulletin of Environmental Contamination and Toxicology 55, no. 1 (July 1995): 162–69. http://dx.doi.org/10.1007/bf00212404.

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4

Cook, Christian J., Charles T. Eason, Mark Wickstrom, and Chris D. Devine. "Development of antidotes for sodium monofluoroacetate (1080)." Biomarkers 6, no. 1 (January 2001): 72–76. http://dx.doi.org/10.1080/135475001452814.

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5

GOH, CSS, DR HODGSON, SM FEARNSIDE, J. HELLER, and N. MALIKIDES. "Sodium monofluoroacetate (Compound 1080) poisoning in dogs." Australian Veterinary Journal 83, no. 8 (August 2005): 474–79. http://dx.doi.org/10.1111/j.1751-0813.2005.tb13296.x.

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6

Eason, Charles. "Sodium monofluoroacetate (1080) risk assessment and risk communication." Toxicology 181-182 (December 2002): 523–30. http://dx.doi.org/10.1016/s0300-483x(02)00474-2.

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7

Eason, C. T., C. M. Frampton, R. Henderson, M. D. Thomas, and D. R. Morgan. "Sodium monofluoroacetate and alternative toxins for possum control." New Zealand Journal of Zoology 20, no. 4 (October 1993): 329–34. http://dx.doi.org/10.1080/03014223.1993.10420354.

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8

Chi, Chih-Hsien, Kuan-Wen Chen, Shih-Huang Chan, Ming-Ho Wu, and Jeng-Jong Huang. "Clinical Presentation and Prognostic Factors in Sodium Monofluoroacetate Intoxication." Journal of Toxicology: Clinical Toxicology 34, no. 6 (January 1996): 707–12. http://dx.doi.org/10.3109/15563659609013833.

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9

King, DR, DR King, LE Twigg, LE Twigg, JL Gardner, and JL Gardner. "Tolerance to Sodium Monofluoroacetate in Dasyurids in Western Australia." Wildlife Research 16, no. 2 (1989): 131. http://dx.doi.org/10.1071/wr9890131.

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The tolerances to sodium fluoroacetate (1080) were estimated for Dasyurus geoffroii (LD*50, ca. 7.5 mg 1080 kg-1), D. hallucatus (ca. 7.5 mg kg-1), Antechinus flavipes (ca. 11.0 mg kg-1) and Phascogale calura (ca. 17.5 mg kg-1) from Western Australia and comparisons were made with D. viverrinus (ca. 1.5 mg kg-1) and A. flavipes (ca. 3.5 mg kg-1) from south-eastern Australia. The species from Western Australia have had evolutionary exposure to naturally occurring fluoroacetate and were more tolerant to the toxin than dasyurids from south-eastern Australia, Presumably, they have acquired this tolerance through feeding on prey which had fed on plants containing fluoroacetate.

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10

Saunders, Glen, Steven McLeod, and Barry Kay. "Degradation of sodium monofluoroacetate (1080) in buried fox baits." Wildlife Research 27, no. 2 (2000): 129. http://dx.doi.org/10.1071/wr99031.

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The degradation of 1080 in buried Foxoff® baits was investigated. Baits were exposed to 5 different treatments: shelf-stored controls, prevailing weather, no rainfall, average weekly rainfall and twice average weekly rainfall. Curvilinear regression models were used to model the rates of decay of 1080. These predicted that under normal rainfall conditions, baits would become non-lethal to working dogs at 2.2 weeks and foxes at 2.8 weeks. Management implications of these results are discussed.

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11

Booth, L. H., S. C. Ogilvie, G. R. Wright, and C. T. Eason. "Degradation of Sodium Monofluoroacetate (1080) and Fluorocitrate in Water." Bulletin of Environmental Contamination and Toxicology 62, no. 1 (January 1, 1999): 34–39. http://dx.doi.org/10.1007/s001289900838.

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12

Eastland, Warren G., and Samuel L. Beasom. "Acute toxicity of sodium monofluoroacetate to the striped skunk." Bulletin of Environmental Contamination and Toxicology 38, no. 6 (June 1987): 934–36. http://dx.doi.org/10.1007/bf01609075.

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13

Bowen, L. H., D. R. Morgan, and C. T. Eason. "Persistence of sodium monofluoroacetate (1080) in baits under simulated rainfall." New Zealand Journal of Agricultural Research 38, no. 4 (December 1995): 529–31. http://dx.doi.org/10.1080/00288233.1995.9513155.

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14

Twigg, L. E., and L. V. Socha. "Defluorination of sodium monofluoroacetate by soil microorganisms from central Australia." Soil Biology and Biochemistry 33, no. 2 (February 2001): 227–34. http://dx.doi.org/10.1016/s0038-0717(00)00134-6.

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15

Eason, CT, and CM Frampton. "Acute Toxicity of Sodium Monofluoroacetate (1080) Baits to Feral Cats." Wildlife Research 18, no. 4 (1991): 445. http://dx.doi.org/10.1071/wr9910445.

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Feral cat populations have a devastating effect on many native wildlife species. As part of a programme to evaluate improved predator control, feral cats were observed after they had ingested a polymer bait loaded with sodium monofluoroacetate (1080) 0.4-1.6 mg/g bait, equivalent to doses of 0.1-1.3 mg/kg body weight. Deaths occurred with 0.6 mg/g bait and all higher doses. With the highest dose all animals died within 24 h. An approximate oral LD50 of 0.28 mg/kg (0.07-0.49) and LD90 of 0.35 mg/kg (0.14-0.56) were calculated (with 95% confidence limits). Main symptoms were disorientation and lethargy, followed by death. A dose of 2 mg/g bait is recommended as a humane and lethal dose of 1080 for feral cats.

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16

Demarchi, A. C. C. O., M. L. Menezes, A. Mercadante, and I. Vassillief. "Determination of the sodium monofluoroacetate in serum by gas chromatography." Chromatographia 54, no. 5-6 (September 2001): 402–4. http://dx.doi.org/10.1007/bf02492692.

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17

Eason, C. T. "Cholecalciferol as an alternative to sodium monofluoroacetate (1080) for poisoning possums." Proceedings of the New Zealand Weed and Pest Control Conference 44 (January 8, 1991): 35–37. http://dx.doi.org/10.30843/nzpp.1991.44.10799.

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18

Eason, C. T., R. Gooneratne, G. R. Wright, R. Pierce, and C. M. Frampton. "The fate of sodium monofluoroacetate (1080) in water, mammals and invertebrates." Proceedings of the New Zealand Plant Protection Conference 46 (January 8, 1993): 297–301. http://dx.doi.org/10.30843/nzpp.1993.46.11144.

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19

Wong, DH, WE Kirkpatrick, JE Kinnear, and DR King. "Defluorination of Sodium Monofluoroacetate (1080) by Microorganisms Found in Bait Materials." Wildlife Research 18, no. 5 (1991): 539. http://dx.doi.org/10.1071/wr9910539.

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A range of bait materials (kangaroo and horse meat, beff, crackle, egg and oats) was used during pest control programmes that utilized sodium monofluoroacetate (1080 poison). These materials were assayed for the presence of microorganisms (fungi and bacteria) and the ability of these microorganisms to defluorinate 1080 was investigated. In general, the defluorinating activity was low when 1080 was the sole carbon source, but in the presence of an alternative carbon source (e.g. peptone-meat extract) the defluorinating ability of many organisms was greatly enhanced. The extent of defluorination varied among the different types of organisms associated with the baits. Microorganisms isolated from oats and kangaroo meat showed the highest defluorinating activity, whereas those associated with crackle and egg had the lowest. Of the isolates tested only Fusarium oxysporum exhibited high defluorinating ability in soil, defluorinating approx equal to 72% of the available 1080 (20mM) in 15 d at 27 deg C. The implications of these findings for predator/pest control are discussed.

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20

Eason, C. T., R. Gooneratnel, H. Fitzgerald, G. Wright, and C. Frampton. "Persistence of Sodium Monofluoroacetate in Livestock Animals and Risk to Humans." Human & Experimental Toxicology 13, no. 2 (February 1994): 119–22. http://dx.doi.org/10.1177/096032719401300210.

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1 Sodium monofluoroacetate (1080), a vertebrate pesticide widely used in New Zealand, was administered orally to sheep and goats at a dose level of 0.1 mg kg-1 body weight to assess risk to humans of secondary poisoning from meat. Blood, muscle, liver, and kidney were analysed for 1080 residues. 2 The plasma elimination half-life was 10.8 h in sheep and 5.4 h in goats. Concentrations of 1080 in muscle (0.042 ?g g-1), kidney (0.057 ?g g-1), and liver (0.021 ?g g-1) were substantially lower than those in plasma (0.098 ?g m-1) at 2.5 h after dosing. 3 Only traces of 1080 (<0.002 to 0.008 ?g g-1) were found in sheep tissues after 96 hours. 4 Livestock are normally excluded from areas where 1080 is being used for pest control, reducing the risk of secondary poisoning. Even with accidental exposure to a sublethal dose 1080 would not persist in tissues for more than a few days because it is cleared rapidly from the body. Therefore the occurrence of 1080 in meat intended for human consumption is highly unlikely.

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21

Hornshaw, T. C., R. K. Ringer, R. J. Aulerich, and H. H. Casper. "Toxicity of sodium monofluoroacetate (Compound 1080) to mink and European ferrets." Environmental Toxicology and Chemistry 5, no. 2 (February 1986): 213–23. http://dx.doi.org/10.1002/etc.5620050213.

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22

Ataria, J. M., M. Wickstrom, D. Arthur, and C. T. Eason. "Biochemical and histopathological changes induced by sodium monofluoroacetate (1080) in mallard ducks." New Zealand Plant Protection 53 (August 1, 2000): 293–98. http://dx.doi.org/10.30843/nzpp.2000.53.3698.

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Public concern over inadvertent kills of native birds following largescale aerial poisoning operations for possum has highlighted deficiencies in information concerning the effects of 1080 on nontarget species In this study biochemical and histopathological changes were measured in adult male mallard ducks (Anas platyrynchos) dosed orally with 1080 The toxin was quickly absorbed into the blood and distributed to the cardiac tissue The time taken to reach maximum 1080 concentrations in these tissues corresponded closely with the onset of clinical signs of toxicosis Tissue and serum citrate accumulation is a direct result of 1080induced inhibition of the Krebs Cycle enzyme aconitate hydratase Histopathological lesions indicated that skeletal muscle was a target organ for 1080induced damage in birds Dosedependent increases in serum creatinine kinase (CK) and aspartate aminotransferase (AST) were consistent with extensive muscle necrosis Skeletal muscle may be a unique avianspecific target organ for 1080

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23

Ozawa, Hideaki, and Tadashi Tsukioka. "Gas chromatographic determination of sodium monofluoroacetate in water by derivatization with dicyclohexylcarbodiimide." Analytical Chemistry 59, no. 24 (December 15, 1987): 2914–17. http://dx.doi.org/10.1021/ac00151a016.

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24

Foronda, Natalia M., Jefferson Fowles, Nerida Smith, Michael Taylor, and Wayne Temple. "A benchmark dose analysis for sodium monofluoroacetate (1080) using dichotomous toxicity data." Regulatory Toxicology and Pharmacology 47, no. 1 (February 2007): 84–89. http://dx.doi.org/10.1016/j.yrtph.2006.08.002.

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25

Wong, D. H., W. E. Kirkpatrick, D. R. King, and J. E. Kinnear. "Defluorination of sodium monofluoroacetate (1080) by microorganisms isolated from western Australian soils." Soil Biology and Biochemistry 24, no. 9 (September 1992): 833–38. http://dx.doi.org/10.1016/0038-0717(92)90002-f.

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26

Gooneratne, S. R., C. T. Eason, C. J. Dickson, H. Fitzgerald, and G. Wright. "Persistence of sodium monofluoroacetate in rabbits and risk to non-target species." Human & Experimental Toxicology 14, no. 2 (February 1995): 212–16. http://dx.doi.org/10.1177/096032719501400210.

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1 Sodium monofluoroacetate (1080), a vertebrate pesti cide used in New Zealand, was administered orally to rabbits at two dose levels (sub-lethal and lethal) to determine how long 1080 would persist in plasma, liver, kidney, and muscle so that the risk of consump tion of meat from lethally or sub-lethally poisoned rab bits by non-target species could be assessed. 2 The plasma elimination half-life in rabbits receiving a sub-lethal dose was 1.1 h. Retention of 1080 in tissue was greater in rabbits dosed with a lethal dose than in those that received a sub-lethal dose. Irrespective of the dose level, concentration of 1080 in muscle, kidney, and liver was substantially lower than in the plasma. 3 Poisoning of dogs is possible because of their extreme susceptibility to 1080. Poisoning of birds is less likely. The risk of secondary poisoning is reduced as the con centration of 1080 declines in putrefying carcasses.

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27

Wong, D. H., C. F. Runham, L. C. DenHollander, and J. E. Kinnear. "A preliminary report on a bacteriological assay for compound 1080 (sodium monofluoroacetate)." Letters in Applied Microbiology 12, no. 5 (May 1991): 161–63. http://dx.doi.org/10.1111/j.1472-765x.1991.tb00530.x.

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28

Hugghins, Ernest J., Howard H. Casper, and C. David Ward. "Tissue Fluoroacetate Residues in Prairie Dogs Dosed with Low-Level Sodium Monofluoroacetate." Journal of AOAC INTERNATIONAL 71, no. 3 (May 1, 1988): 579–81. http://dx.doi.org/10.1093/jaoac/71.3.579.

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Abstract A total of 83 black-tailed prairie dogs (Cynomys ludovicianus) from South Dakota were subjected to low-level treatment with sodium monofluoroacetate (Compound 1080) in the laboratory (0.01-0.30 mg 1080/kg). The acute oral median lethal dose (LD50) of 1080 administered by oral gavage was established at 0.173 mg/kg. To assay fluoroacetate residues, 8 kinds of tissue from each of 10 prairie dogs dead of low-level 1080 poisoning were analyzed by capillary gas chromatography-mass spectrometry. Of the total of 79 tissues analyzed, 73 contained <100 ppb fluoroacetate, and 67 contained <50 ppb fluoroacetate. To test the effect of secondary poisoning on nontarget species, 8 European ferrets (Mustela furo) were fed ground whole carcasses of prairie dogs dead of low-level 1080 poisoning, with no observable ill effects on the ferrets

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29

Tietjen, H., F. Deines, and W. Stephensen. "Sodium monofluoroacetate (1080): A study of residues in arctic fox muscle tissue." Bulletin of Environmental Contamination and Toxicology 40, no. 5 (May 1988): 707–10. http://dx.doi.org/10.1007/bf01697519.

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30

Eason, CT. "The Acute Toxicity of Cholecalciferol to the European Rabbit, Oryctolagus Cuniculus." Wildlife Research 20, no. 2 (1993): 173. http://dx.doi.org/10.1071/wr9930173.

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The toxicity of cholecalciferol in the rabbit was evaluated as part of an assessment of alternative toxins to sodium monofluoroacetate (1080) and pindone for large-scale rabbit control in the South Island of New Zealand. After a dose-ranging study spanning the known LD50 values for other mammals (up to 400 mg kg-1) an acute toxicity study established an LD50 of 9mg kg-1 and LD95 of 18mg kg-1 for the rabbit, which proved very susceptible to cholecalciferol.

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31

Ozawa, Hideaki, and Tadashi Tsukioka. "Determination of sodium monofluoroacetate in soil and biological samples as the dichloroanilide derivative." Journal of Chromatography A 473 (January 1989): 251–59. http://dx.doi.org/10.1016/s0021-9673(00)91306-2.

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32

Lu, Qin, Peter Wu, and Greg E. Collins. "Contactless conductivity detection of sodium monofluoroacetate in fruit juices on a CE microchip." ELECTROPHORESIS 28, no. 19 (October 2007): 3485–91. http://dx.doi.org/10.1002/elps.200600723.

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33

Obrien, PH. "The Toxicity of Sodium Monofluoroacetate (Compound 1080) to Captive Feral Pigs, Sus-Scrofa." Wildlife Research 15, no. 2 (1988): 163. http://dx.doi.org/10.1071/wr9880163.

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The toxicity of sodium monofluoroacetate (1080) to captive feral pigs, Sus scrofa, was assessed over a range of doses (1.50-21.3 mg 1080 kg-1 bodyweight) administered orally in wheat bait to 80 animals. Calculated LD*50 was 4.11 mg 1080 kg-1 (95% fiducial limits: 3.02-5.34 mg kg-1) and LD*9O was 11.25 mg kg-1 (8.05-21.69 mg kg-1). The incidence (98%) and frequency (mean � SD = l3.6 � 6.7 overall) of vomiting were high. Frequency of vomiting was unrelated to log*10 dose (r= -0.015; N= 80; NS), although time until vomiting began (r= -0.558; N=77; P<O.05) and time until death (r= -0.391; N=47; P<0.05) had a significant negative association with log*10 dose. Median latency was 49 min (range 10-350 min; N=77) and median time until death was 244 min (range 131-7200 min; N=47). Sex and bodyweight had no effect on frequency of vomiting, time until death or prognosis. Feral pigs yere much less sensitive to 1080 under these test conditions than those in earlier studies. Because the present experiment seems likely to resemble field situations, these results warrant careful examination of field intake and mortality of feral pigs during 1080 poisoning.

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34

Obrien, PH, BS Lukins, and JA Beck. "Bait Type Influences the Toxicity of Sodium Monofluoroacetate (Compound 1080) to Feral Pigs." Wildlife Research 15, no. 4 (1988): 451. http://dx.doi.org/10.1071/wr9880451.

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The toxicity of sodium monofluoroacetate (1080) to captive feral pigs was compared in wheat and pellet bait. Mortality following 4.34 mg 1080 kg-1 was significantly higher among pigs receiving 1080 in wheat bait (60%, 24/40) than in pellet bait (28%, 11/40, X*2=7.31, 1 d.f., P<O.05). There were no significant differences between pigs receiving each bait type in terms of time until vomiting began, frequency, mass of vomitus produced, or in time until death. The amount and concentration of 1080 in vomitus and the proportion of 1080 dose ejected were unrelated to bait type. Surviving pigs produced vomitus with a greater 1080 concentration but smaller mass than those that died. Bait type is an important determinant of the toxicity of 1080 to captive feral pigs and should be closely evaluated before specific bait types are used in the field.

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35

Adaska, John M., Guillermo Rimoldi, Patricia C. Blanchard, John Champagne, Robert H. Poppenga, and Michelle Mostrom. "Multiple episodes of 1080 (sodium monofluoroacetate) intoxication in a California calf-raising operation." Journal of Veterinary Diagnostic Investigation 30, no. 5 (July 5, 2018): 747–51. http://dx.doi.org/10.1177/1040638718781435.

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Over a 1-y period, a California calf-raising operation experienced 4 separate episodes of sudden death in 4–6-mo-old steers. Each episode occurred in 1–3 contiguous pens on 1 of 3 properties owned by the operation, but eventually all 3 properties were involved. In each episode, animals appeared normal at the evening feeding but at the subsequent morning feeding were found dead or dying. Remaining live calves had a stiff gait and were often dribbling urine, but did not show respiratory signs until they were down and agonal. At postmortem examination, calves consistently had moderate-to-large numbers of ecchymotic and suffusive hemorrhages on the epicardial surface and moderate-to-large amounts of fluid in the pericardial sac. Pulmonary edema and/or moderate amounts of watery fluid in the thoracic and abdominal cavities were present in a smaller percentage. On histologic examination, the myocardium had variable myofiber degeneration characterized by hypereosinophilia and fragmentation with mild interstitial infiltrates. Testing of heart and liver samples for monensin found levels lower than in previous cases of monensin toxicity. Rumen content was negative for oleandrin and grayanotoxins. Sodium monofluoroacetate (trade name: 1080) was consistently detected at ⩾10 ppb in kidney and liver, and was concluded to be the cause of the intoxication.

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36

Aulerich, R. J., R. K. Ringer, and J. Safronoff. "Primary and secondary toxicity of warfarin, sodium monofluoroacetate, and methyl parathion in mink." Archives of Environmental Contamination and Toxicology 16, no. 3 (May 1987): 357–66. http://dx.doi.org/10.1007/bf01054954.

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37

Twigg, L. E., D. R. King, and A. J. Bradley. "The effect of sodium monofluoroacetate on plasma testosterone concentration in Tiliqua rugosa (gray)." Comparative Biochemistry and Physiology Part C: Comparative Pharmacology 91, no. 2 (January 1988): 343–47. http://dx.doi.org/10.1016/0742-8413(88)90040-0.

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38

Crosbie, SF, FJ Laas, MER Godfrey, JM Williams, and DS Moore. "A Field Assessment of the Anticoagulant Brodifacoum Against Rabbits, Oryctolagus-Cuniculus." Wildlife Research 13, no. 2 (1986): 189. http://dx.doi.org/10.1071/wr9860189.

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The effectiveness of 100 p.p.m. brodifacoum for controlling an island population of rabbits was assessed and compared with that of 200 p.p.m. 1080 (sodium monofluoroacetate). Both compounds were applied in an apple-based, raspberry-flavoured jam bait after pre-feeding with the same non-toxic bait. Brodifacoum and 1080 both reduced rabbit populations by 85% on average, compared with an average 'natural' decline of 34% on untreated sites; these estimates of poisoning mortality may be considered conservative, however, because of post-poisoning migration onto poisoned sites.

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39

Ross, J. G., G. J. Hickling, and D. R. Morgan. "Use of subacute and chronic toxicants to control sodium monofluoroacetate (1080) bait shy possums." Proceedings of the New Zealand Plant Protection Conference 50 (August 1, 1997): 397–400. http://dx.doi.org/10.30843/nzpp.1997.50.11318.

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40

Srinivasan, MS, A. Suren, J. Wech, and J. Schmidt. "Investigating the fate of sodium monofluoroacetate during rain events using modelling and field studies." New Zealand Journal of Marine and Freshwater Research 46, no. 2 (June 2012): 167–78. http://dx.doi.org/10.1080/00288330.2011.619200.

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41

Eason, C. T. "A 90-Day Toxicological Evaluation of Compound 1080 (Sodium Monofluoroacetate) in Sprague-Dawley Rats." Toxicological Sciences 69, no. 2 (October 1, 2002): 439–47. http://dx.doi.org/10.1093/toxsci/69.2.439.

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42

Ogilvie, S. C., M. D. Thomas, G. A. Morriss, D. R. Morgan, and C. T. Eason. "Investigation of sodium monofluoroacetate (1080) bait shyness in wild brushtail possum (Trichosurus vulpecula) populations." International Journal of Pest Management 46, no. 1 (January 2000): 77–80. http://dx.doi.org/10.1080/096708700227615.

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43

Obrien, PH, and BS Lukins. "Factors Influencing the Intake of Sodium Monofluoroacetate (Compound-1080) by Free-Ranging Feral Pigs." Wildlife Research 15, no. 3 (1988): 285. http://dx.doi.org/10.1071/wr9880285.

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Post-mortem investigations of 207 feral pigs poisoned in the field were conducted to assess the influence on 1080 dose of bait type, site, sex, bodyweight and distance from bait stations. The stomachs of most (126/207) pigs contained only bait material., Bait type significantly affected intake and 1080 dose, with pellet intake greater than cereal baits. Sex and bodyweight also influenced intake: females ingested significantly larger 1080 doses than males; and larger pigs tended to consume more bait in absolute terms but lower doses of 1080 (mg kg-1). Average intake of 1080 varied widely at different locations. Pigs that died close to bait stations had higher 1080 doses but smaller proportions of bait in their stomach contents than those that died away from bait stations.

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44

Kimball, Bruce A., and Elizabeth A. Mishalanie. "Gas chromatographic determination of sodium monofluoroacetate as the free acid in an aqueous solvent." Journal of Chromatography A 634, no. 2 (March 1993): 289–96. http://dx.doi.org/10.1016/0021-9673(93)83016-l.

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45

Guan, Fuyu, Huifang Wu, and Yi Luo. "Sensitive and selective method for the determination of sodium monofluoroacetate by capillary zone electrophoresis." Journal of Chromatography A 719, no. 2 (January 1996): 421–26. http://dx.doi.org/10.1016/0021-9673(95)00752-0.

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46

Martin, Gary R., and Laurie E. Twigg. "Sensitivity to sodium fluoroacetate (1080) of native animals from north-western Australia." Wildlife Research 29, no. 1 (2002): 75. http://dx.doi.org/10.1071/wr00117.

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The sensitivity to sodium monofluoroacetate (1080) of 9 species of native animals from north-western Australia was assessed using the increasing dose procedure to determine the Approximate Lethal Dose for each species. Granivorous birds from this region (e.g. ducks, corellas) were generally more sensitive to 1080 than their counterparts from southern Australia, and would be theoretically at risk from primary poisoning during 1080 grainbased baiting programs. However, the tolerance to 1080 of birds of prey from these areas is sufficient that these species face little risk of secondary poisoning during pest-control programs aimed at rodents or rabbits. The risk of primary poisoning to raptors from meat baits containing 6 mg 1080 per bait or less also appears to be low. The coexistence of brown falcons and barn owls with fluoroacetate-bearing vegetation over parts of their range has probably contributed to their development of tolerance to fluoroacetate.

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Warburton, B. "Control of Bennett's and Tammar Wallabies in New Zealand Using Compound 1080 Gel on Foliage Baits." Wildlife Research 17, no. 5 (1990): 541. http://dx.doi.org/10.1071/wr9900541.

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Trials were carried out against Bennett's wallabies (Macropus rufogriseus rufogriseus) and tammar wallabies (M. eugenii) in areas of indigenous forest where browsing is preventing or damaging regeneration. Compound 1080 (sodium monofluoroacetate) in a carbopol gel was spread on the foliage of palatable plants. Numbers of Bennett's wallabies were reduced by 91% and tammar wallabies by 87%. These kill estimates compare favourably with those achieved using aerially sown baits. The costs per hectare of gel poisoning were estimated to be as little as 40% of those for aerial baiting, but the costs of using gel will increase as the control areas become larger, more rugged, and less easily traversed on foot.

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Ogilvie, S. C., M. D. Thomas, H. Fitzgerald, and D. R. Morgan. "Sodium monofluoroacetate (1080) bait-shyness in a wild brushtail possum (Trichosurus vulpecula) population." Proceedings of the New Zealand Plant Protection Conference 49 (August 1, 1996): 143–46. http://dx.doi.org/10.30843/nzpp.1996.49.11445.

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Pessoa, Danielle A. N., Rosane M. T. Medeiros, and Franklin Riet-Correa. "Techniques for prevention and control of poisoning by sodium monofluoroacetate (MFA)-containing plants in ruminants." Pesquisa Veterinária Brasileira 39, no. 10 (October 2019): 771–79. http://dx.doi.org/10.1590/1678-5150-pvb-6373.

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ABSTRACT: Monofluoroacetate (MFA)-containing plants cause sudden death associated with exercise in ruminants, and are responsible for significant losses in Brazilian livestock, estimated at 500,000 bovine deaths annually. Most of the times, the control and treatment of this type of poisoning are not efficient, because disease evolution is superacute, usually causing the death of the animal. Due to the difficulty in controlling this intoxication, several studies have suggested alternatives to prevent it, mainly by making animals resistant to the MFA present in these plants or by avoiding their consumption. This literature review addresses the techniques used experimentally to control the poisoning of ruminants by plants containing MFA. The first studies carried out in Brazil demonstrated that goats and sheep that continuously receive non-toxic doses of plant containing MFA show greater resistance to poisoning than untreated animals, and that this resistance can be transmitted by ruminal fluid transfaunation, suggesting that poisoning occurs due to the presence of bacteria that hydrolyze MFA in the rumen. Based on this hypothesis, several MFA-hydrolyzing bacteria were isolated (Enterococcus faecalis, Bacillus sp., Paenibacillus sp., Burkholderia sp., Cupriavidus sp., Staphylococcus sp., Ancylobacter sp., Ralstonia sp., Stenotrophomonas sp., Pigmentiphaga kullae, and Ancylobacter dichloromethanicus). When some of these bacteria were administered intraruminally, they provided the animal with a different level of protection against poisoning. However, it was observed that protection is gradually lost when the bacterium administration is interrupted. Consequently, to obtain more efficient protection, these bacteria should be administered continuously, probably in the form of probiotics. In another assay, MFA was administered to sheep at non-toxic doses to test the hypothesis that this substance could induce the multiplication of bacteria that hydrolyze it in the rumen. There was no increase in resistance to poisoning after administration of MFA; however, no signs of poisoning were observed when animals received trifluoroacetate and no clinical signs were verified when they were challenged with toxic doses of MFA; in contrast, all control animals presented clinical signs. These results suggest that trifluoroacetate induces the proliferation of MFA-degrading bacteria, and can be used in intoxication prophylaxis. The conditioned food aversion technique, using lithium chloride, has been successfully used experimentally to prevent ruminants from ingesting plants that contain MFA. Another alternative tested was the spraying of Amorimia septentrionalis with the endophytic bacterium Herbaspirillum seropedicae, which degrades MFA, resulting in decreased concentration of this compound in the plants. In conclusion, several experimental techniques have been proved efficient in the control and prophylaxis of MFA-containing plant poisoning; however, none of these techniques are available commercially. Further experiments, mainly in the field, should be carried out to adapt some of these techniques to the conditions of extensive breeding in the numerous areas where MFA-containing plants occur.

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Meenken, D., and L. H. Booth. "The risk to dogs of poisoning from sodium monofluoroacetate (1080) residues in possum (Trichosurus vulpecula)." New Zealand Journal of Agricultural Research 40, no. 4 (January 1997): 573–76. http://dx.doi.org/10.1080/00288233.1997.9513280.

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