Academic literature on the topic 'Sodium monofluoroacetate'
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Journal articles on the topic "Sodium monofluoroacetate"
Kim, J. B., J. W. Jang, and J. H. Kim. "Reversible leukoencephalopathy in sodium monofluoroacetate intoxication." Neurology 82, no. 13 (March 31, 2014): 1190–91. http://dx.doi.org/10.1212/wnl.0000000000000262.
Full textChi, Chih-Hsien, Tin-Kwang Lin, and Kuan-Wen Chen. "Hemodynamic abnormalities in sodium monofluoroacetate intoxication." Human & Experimental Toxicology 18, no. 6 (June 1999): 351–53. http://dx.doi.org/10.1191/096032799678840200.
Full textParfitt, R. L., C. T. Eason, H. Hoff, and L. K. Heng. "Sodium monofluoroacetate (1080) leaching through soils." Bulletin of Environmental Contamination and Toxicology 55, no. 1 (July 1995): 162–69. http://dx.doi.org/10.1007/bf00212404.
Full textCook, Christian J., Charles T. Eason, Mark Wickstrom, and Chris D. Devine. "Development of antidotes for sodium monofluoroacetate (1080)." Biomarkers 6, no. 1 (January 2001): 72–76. http://dx.doi.org/10.1080/135475001452814.
Full textGOH, CSS, DR HODGSON, SM FEARNSIDE, J. HELLER, and N. MALIKIDES. "Sodium monofluoroacetate (Compound 1080) poisoning in dogs." Australian Veterinary Journal 83, no. 8 (August 2005): 474–79. http://dx.doi.org/10.1111/j.1751-0813.2005.tb13296.x.
Full textEason, Charles. "Sodium monofluoroacetate (1080) risk assessment and risk communication." Toxicology 181-182 (December 2002): 523–30. http://dx.doi.org/10.1016/s0300-483x(02)00474-2.
Full textEason, C. T., C. M. Frampton, R. Henderson, M. D. Thomas, and D. R. Morgan. "Sodium monofluoroacetate and alternative toxins for possum control." New Zealand Journal of Zoology 20, no. 4 (October 1993): 329–34. http://dx.doi.org/10.1080/03014223.1993.10420354.
Full textChi, Chih-Hsien, Kuan-Wen Chen, Shih-Huang Chan, Ming-Ho Wu, and Jeng-Jong Huang. "Clinical Presentation and Prognostic Factors in Sodium Monofluoroacetate Intoxication." Journal of Toxicology: Clinical Toxicology 34, no. 6 (January 1996): 707–12. http://dx.doi.org/10.3109/15563659609013833.
Full textKing, DR, DR King, LE Twigg, LE Twigg, JL Gardner, and JL Gardner. "Tolerance to Sodium Monofluoroacetate in Dasyurids in Western Australia." Wildlife Research 16, no. 2 (1989): 131. http://dx.doi.org/10.1071/wr9890131.
Full textSaunders, Glen, Steven McLeod, and Barry Kay. "Degradation of sodium monofluoroacetate (1080) in buried fox baits." Wildlife Research 27, no. 2 (2000): 129. http://dx.doi.org/10.1071/wr99031.
Full textDissertations / Theses on the topic "Sodium monofluoroacetate"
Nogueira, Vivian de Assun??o. "Intoxica??o experimental por monofluoroacetato de s?dio em bovinos: aspectos cl?nicos e patol?gicos." Universidade Federal Rural do Rio de Janeiro, 2009. https://tede.ufrrj.br/jspui/handle/tede/844.
Full textFunda??o Carlos Chagas Filho de Amparo a Pesquisa do Estado do Rio de Janeiro
Sodium monofluoracetate (MF) was identified, by cromatography, in three of the 12 plants that cause "sudden death" in cattle in Brazil. Among these, Palicourea marcgravii is the most important due to its high toxicity (0.6g/kg), wide distribution, good palatability and cummulative effect. In 1959, D?bereiner & Tokarnia detected in the kidney of cattle poisoned by that plant, hydropic-vacuolar degeneration (HVD) of the distal convoluted uriniferous tubules, a lesion they considered typical for the poisoning. MF inhibits the citrate aconitase enzyme, resulting in blockage of the Krebs Cycle and the production of ATP. The objective of this study was to verify if the ingestion of MF causes the same lesions in the kidney of cattle poisoned by the plants which cause sudden death . This would prove that this substance is responsible for the deaths of animals which ingest these plants. Six cows received orally 0.5 and 1 mg/kg of MF (Sigma Aldrich Co) diluted in 50 mL of distilled water. Clinically the animals presented palpitation, replete jugular vein with positive pulse, abdominal breathing, slight balance loss with sometimes swaying gait, the animals laid down and placed the head on their flank. In the dramatic phase , all the animals fell into lateral decubitus, stretched out the legs, made peddling movements, presented opistotonus, nistagmus, and died. The dramatic phase lasted from 3 to 14 minutes. At postmortem examination, the heart auricles, jugulars and pulmonary veins were moderately ingurgitated. Slight to moderate edema of the subserosa was seen in places of fixation of the gall bladder to the liver, besides slight edema around the duodenum in contact with the pancreas. Histopathology revealed HVD of the epithelial cells of the distal convoluted uriniferous tubules associated with nuclear picnosis in all cows. Coagulation necrosis of individual or groups of hepatocytes and slight hepatic congestion secondary to the venous stasis were also observed. Hydropic-vacuolar degeneration has been observed in cases of poisoning by many substances, which cause acute tubular nephrosis, however not restricted to the distal renal tubules and without nuclear picnosis. This study demonstrates that this peculiar kind of HVD in the kidney can be caused by MF and, in analogy, the compound should be considered responsible for the death of cattle that ingest toxic plants which cause "sudden death" in Brazil. For the first time it has now been proved that MF causes HVD of the distal convoluted uriniferous tubules, what indicates that studies of possible degradation of MF by rumen bacteria could have economic importance, as there die annually at least 600.000 cattle by sudden death causing toxic plants.
Monofluoracetato de s?dio (MF) foi identificado, por cromatografia, em Palicourea marcgravii, Arrabidaea bilabiata e possivelmente em Mascagnia rigida, plantas do grupo das que causam morte s?bita em bovinos no Brasil. Em 1959, D?bereiner & Tokarnia detectaram no rim de bovinos intoxicados por P. marcgravii, uma les?o por eles considerada t?pica para intoxica??o, a degenera??o hidr?pico-vacuolar dos t?bulos urin?feros contorcidos distais (DHV). O modo de a??o do MF baseia-se na inibi??o da enzima citrato aconitase, do que redunda bloqueio do ciclo de Krebs e da produ??o de ATP. O objetivo deste trabalho foi verificar se a ingest?o de MF induz as mesmas les?es observadas no rim de bovinos intoxicados pelas plantas que causam morte s?bita , o que indicaria que essa subst?ncia ? respons?vel pelas mortes dos animais que ingeriram essas plantas. Foram realizados experimentos em que seis vacas receberam, por via oral, 0,5 e 1,0 mg/kg de MF dilu?dos em 50 mL de ?gua destilada. Clinicamente os animais apresentaram taquicardia, jugular repleta com pulso venoso positivo, respira??o abdominal, ligeira perda de equil?brio, por vezes cambaleavam, deitavam e apoiavam a cabe?a no flanco. Na fase dram?tica, todos os animais ca?am em dec?bito lateral, esticavam os membros, faziam movimentos de pedalagem, apresentavam opist?tono, nistagmo, mugidos e morriam. O per?odo da fase dram?tica durou entre 2 e 14 minutos. ? necropsia verificaram-se aur?culas, jugulares, ?zigos e pulmonares moderadamente ingurgitadas. Observaram-se ainda leve a moderado edema da subserosa nos locais de fixa??o da ves?cula biliar no f?gado, al?m de leve edema em torno do intestino delgado (duodeno) em contato com p?ncreas. O exame histopatol?gico revelou, em todos os animais, leve a acentuada DHV das c?lulas epiteliais dos t?bulos urin?feros contorcidos distais associada ? cariopicnose nuclear. Vacuoliza??o e necrose de coagula??o individual ou de grupos de hepat?citos e leve congest?o hep?tica secund?rios ? estase venosa tamb?m foram observados. DHV tem sido observada em casos de envenenamento por outras subst?ncias, por?m esta n?o est? restrita aos t?bulos distais e n?o se observa cariopicnose nuclear. Dessa forma, esse estudo demonstra que a DHV que ocorre no rim de bovinos ? caracter?stica do envenenamento por MF e, por analogia, essa subst?ncia pode ser considerada como um dos fatores importantes, sen?o o mais significativo, implicado no ?bito dos animais que ingerem plantas que causam morte s?bita no Brasil. Comprova-se, pela primeira vez, que o MF induz ? a causa da DHV dos t?bulos uriniferos contorcidos distais, o que indica que estudos que envolvam metaboliza??o de MF por bact?rias ruminais teriam grande aplicabilidade econ?mica, uma vez que pelo menos 600.000 bovinos morrem anualmente intoxicados por plantas do grupo das que causam morte s?bita no Brasil.
Foronda, Natalia, and n/a. "Health risk assessment and health risk management with special reference to sodium monofluoroacetate (1080) for Possum control in New Zealand." University of Otago. Department of Pharmacology & Toxicology, 2007. http://adt.otago.ac.nz./public/adt-NZDU20080131.145423.
Full textGreentree, Carolyn, and n/a. "Experimental evaluation of fox control and the impact of foxes on lambs." University of Canberra. Science &Design, 2000. http://erl.canberra.edu.au./public/adt-AUC20060713.101158.
Full textCunha, Luciana Castro da. "Estudo fitoquímico biomonitorado de Amorimia rigida (Malpighiaceae): 1. Purificação e caracterização do monofluoroacetato de sódio 2. Avaliação bioquímica dos efeitos tóxicos em ratos." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/10/10133/tde-21052013-140600/.
Full textThe first occurrence of sodium monofluoroacetate in plants was reported in Dichapetalum cymosum. Further, this toxic substance was found in other plant species, as Palicourea marcgravii, which causes \"sudden death\". Amorimia rigida, which is well known and frequent in northeastern Brazil, it is among the poisonous plants that have identical lethal effects in cattle to that caused by the ingestion of plants containing sodium fluoroacetate. These intoxication signs are the same that ones presented by experimentally poisoned animals with this toxic. Significant economic losses can be imputed to A. rigida, therefore our aim was to identify its toxic compound and also to establish its biomarkers of effect. The study by cellulose thin layer chromatography using as mobile phase solution of ethanol /ammonium hydroxide/pyridine/water (95:3:1:1, v/v/v/v) and Nile blue as the derivatizing agent - of the aqueous extract of milled dried plant showed the presence of MFA-Na. The fractionation of the hydroalcoholic extract of A. rigida by XAD-2 column chromatography gave four fractions: 100% H2O (F1), H2O: MeOH 80/20 (F2) H2O: MeOH 40/60 (F3) and 100% MeOH (F4); only F1 fraction contained MFA-Na, according to CCDC monitoring. This fraction was subjected to preparative thin layer chromatography, in order to isolate the toxic principle which, in a study by infrared spectroscopy and by comparison with standard MFA-Na, confirmed being a mixture of mono-, di and trifluoroacetate. In the biochemical study, animals intoxicated by MFA-Na and by A. rigida extract showed serum calcium/citrate imbalances; such alterations may be considered biomarkers of MFA-Na exposure effect.
Zuanaze, Rita de Cássia Collicchio [UNESP]. "Perfil hematológico, bioquímico, histopatológico e toxicológico de gatos induzidos experimentalmente com monofluoroacetato de sódio." Universidade Estadual Paulista (UNESP), 2006. http://hdl.handle.net/11449/101302.
Full textCoordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)
Fundação para o Desenvolvimento da UNESP (FUNDUNESP)
O monofluoroacetato de sodio (MFAS) ou composto 1080 e um rodenticida que foi amplamente utilizado para o controle de roedores e predadores domesticos, apos sua descoberta em 1945. Este potente rodenticida age bloqueando o ciclo de Krebs por acao do seu metabolito toxico, o fluorocitrato, e como consequencia, inibe a resposta celular e a producao de ATP. O MFAS foi proibido por lei no Brasil e em diversos paises, mas seu uso indiscriminado continua causando diversos casos de intoxicacoes potencialmente fatais no homem e animais domesticos, principalmente em caes e gatos, representadas por alteracoes neurologicas e cardiacas. Pretendeu-se com este estudo caracterizar o perfil hematologico e bioquimico, as alteracoes histopatologicas e toxicologicas de gatos intoxicados experimentalmente com MFAS, com o objetivo de determinar metodos diagnosticos eficazes. Para tanto, foram utilizadas amostras de sangue de 16 gatos intoxicados experimentalmente com 0,45mg/kg de MFAS, por via oral. Estas amostras foram colhidas por puncao jugular e analisadas quanto ao perfil hematologico, bioquimica serica e analises toxicologicas em cromatografia liquida de alta eficiencia (CLAE), para deteccao e quantificacao do MFAS no soro dos animais. Avaliaram-se tambem as lesoes macro e microscopicas dos animais intoxicados que vieram a obito. Observaram-se leucopenia e trombocitopenia transitorias; hiperglicemia, aumento das enzimas musculares creatinoquinase (CK) e creatinoquinase fracao cardiaca (CK-MB); observaram-se tambem hipocalemia, hipofosfatemia e hipomagnesemia. Os achados macroscopicos e histopatologicos demonstraram lesoes caracteristicas de processos isquemicos e as analises toxicologicas demonstraram um metodo diagnostico simples e eficiente...
Sodium monofluoroacetate (SMFAC) or 1080 compound is a potent rodenticide, largely used since 1945 for rodent and domestic pest control. The toxic effects of SMFAC are caused by fluorocitrato, a toxic metabolite, which has a competitive action with aconitase enzyme, leading to citrate accumulation and interferes in energy production by Krebs cycle blockade. In Brazil, although prohibited by law, there is illegal use, keep causing intoxication in children and domestic animals, specially dogs and cats. The most common intoxication clinical signs are from the cardiac and neurological alterations. In the present study, 16 domestic cats were intoxicated with oral doses of monofluoroacetate (0.45mg/kg). The hematologic and biochemical profiles, and histophatological and blood serum toxicological analysis by high performance liquid chromatography (HPLC) were made to looking for a efficient diagnosis methods. The hematologic profile showed transitory leucopenia and trombocitopenia; the biochemical profile presented hiperglycemia, increase of creatinoquinase enzyme (CK) and creatinoquinase cardiac fraction (CK-MB), hypokalemia and hypophosfatemia were observed. The macrocospic and histopathological findings showed lesions characteristic of degenerative and ischemic processes. The toxicological analysis was shown to be a simple and efficient diagnostic method. SMFAC was detected in 75% of the serum samples analysed, and it was verified an average concentration of 0.32 ìg/mL in them; 4.81% of the serum samples did not show metabolized SMFAC six hours after the induced intoxication of the animals used in this study.
Zuanaze, Rita de Cássia Collicchio. "Perfil hematológico, bioquímico, histopatológico e toxicológico de gatos induzidos experimentalmente com monofluoroacetato de sódio /." Botucatu : [s.n.], 2006. http://hdl.handle.net/11449/101302.
Full textBanca: Noeme Sousa Rocha
Banca: Manoel Lima de Menezes
Banca: Mara Regina Stipp Balarin
Banca: Silvana Lima Gorniak
Resumo: O monofluoroacetato de sodio (MFAS) ou composto 1080 e um rodenticida que foi amplamente utilizado para o controle de roedores e predadores domesticos, apos sua descoberta em 1945. Este potente rodenticida age bloqueando o ciclo de Krebs por acao do seu metabolito toxico, o fluorocitrato, e como consequencia, inibe a resposta celular e a producao de ATP. O MFAS foi proibido por lei no Brasil e em diversos paises, mas seu uso indiscriminado continua causando diversos casos de intoxicacoes potencialmente fatais no homem e animais domesticos, principalmente em caes e gatos, representadas por alteracoes neurologicas e cardiacas. Pretendeu-se com este estudo caracterizar o perfil hematologico e bioquimico, as alteracoes histopatologicas e toxicologicas de gatos intoxicados experimentalmente com MFAS, com o objetivo de determinar metodos diagnosticos eficazes. Para tanto, foram utilizadas amostras de sangue de 16 gatos intoxicados experimentalmente com 0,45mg/kg de MFAS, por via oral. Estas amostras foram colhidas por puncao jugular e analisadas quanto ao perfil hematologico, bioquimica serica e analises toxicologicas em cromatografia liquida de alta eficiencia (CLAE), para deteccao e quantificacao do MFAS no soro dos animais. Avaliaram-se tambem as lesoes macro e microscopicas dos animais intoxicados que vieram a obito. Observaram-se leucopenia e trombocitopenia transitorias; hiperglicemia, aumento das enzimas musculares creatinoquinase (CK) e creatinoquinase fracao cardiaca (CK-MB); observaram-se tambem hipocalemia, hipofosfatemia e hipomagnesemia. Os achados macroscopicos e histopatologicos demonstraram lesoes caracteristicas de processos isquemicos e as analises toxicologicas demonstraram um metodo diagnostico simples e eficiente...(Resumo completo, clicar acesso eletrõnico abaixo)
Abstract: Sodium monofluoroacetate (SMFAC) or 1080 compound is a potent rodenticide, largely used since 1945 for rodent and domestic pest control. The toxic effects of SMFAC are caused by fluorocitrato, a toxic metabolite, which has a competitive action with aconitase enzyme, leading to citrate accumulation and interferes in energy production by Krebs cycle blockade. In Brazil, although prohibited by law, there is illegal use, keep causing intoxication in children and domestic animals, specially dogs and cats. The most common intoxication clinical signs are from the cardiac and neurological alterations. In the present study, 16 domestic cats were intoxicated with oral doses of monofluoroacetate (0.45mg/kg). The hematologic and biochemical profiles, and histophatological and blood serum toxicological analysis by high performance liquid chromatography (HPLC) were made to looking for a efficient diagnosis methods. The hematologic profile showed transitory leucopenia and trombocitopenia; the biochemical profile presented hiperglycemia, increase of creatinoquinase enzyme (CK) and creatinoquinase cardiac fraction (CK-MB), hypokalemia and hypophosfatemia were observed. The macrocospic and histopathological findings showed lesions characteristic of degenerative and ischemic processes. The toxicological analysis was shown to be a simple and efficient diagnostic method. SMFAC was detected in 75% of the serum samples analysed, and it was verified an average concentration of 0.32 ìg/mL in them; 4.81% of the serum samples did not show metabolized SMFAC six hours after the induced intoxication of the animals used in this study.
Doutor
Moraes, Regina Lucia Fonseca de. "Comprovação química e biológica da presença de monofluoroacetato nas folhas de Palicourea marcgravii st. Hil." Universidade de São Paulo, 1993. http://www.teses.usp.br/teses/disponiveis/9/9140/tde-27062008-192851/.
Full textPalicourea marcgravii St. Hil. (Rubiaceae) is one of the most economica1ly important poisonous plants for Brazilian livestock, since it induces not only seizures, cardiac arrythmias, but also \"sudden death\" of large number of the intoxicated animals. ln the present paper, the dissected and grounded leaves of P.marcgravii were extracted with ethanol 95 %, at room temperature, by percolation; the resultant extract was fractionized by partition in ethyl acetate and butanol saturated with water. The obtained residues were administered \"per os\" to rats, trying to look for the presence of the active toxic principIe. The aqueous residue was the only that induced seizures and death to the animals. The presence of monofluoroacetate (MFA) in the aqueous residue was biologica1ly confirmed by the correlation shown in the sodium monofluoroacetate and the aqueous residue dose-response and dose effect curves. MFA was also identified through NMR19F and thin layer chromatography. The obtained results confirmed the presence of MF A in the P.marcgravii leaves; they also suggest that the toxic effects induced by this plant are a consequence of the presence of MFA in its leaves. Further, some hypothesis were also perfomed, in an attempt to better explain the symptoms induced by the plant leaves.
Morgan, David R. "Maximising the effectiveness of aerial 1080 control of possums (Trichosurus vulpecula)." Lincoln University, 2004. http://hdl.handle.net/10182/20.
Full textSerodio, Juliana Job. "Resistência das raças bovinas nelore, curraleiro-pé-duro e pantaneiro a intoxicação experimental por palicourea marcgravii a. St. Hil." Universidade Federal de Goiás, 2013. http://repositorio.bc.ufg.br/tede/handle/tde/2973.
Full textMade available in DSpace on 2014-09-01T14:42:53Z (GMT). No. of bitstreams: 2 Dissertacao Juliana Job Serodio.pdf: 2829653 bytes, checksum: 540f534b828bef881f374e638c97a102 (MD5) license_rdf: 23148 bytes, checksum: 9da0b6dfac957114c6a7714714b86306 (MD5) Previous issue date: 2013-08-29
The study aimed to evaluate bovines from different brazilian breeds that were submitted to experimental intoxication to Palicourea marcgravii A. St. Hill. Six animals from Nelore (G1), Curraleiro Pé-Duro (G2) and Pantaneiro (G3) breeds were used in the groups. The animals were experimental intoxicated with sodium monofluoracetate acid receiving one single dose of the compound in a dosage of 0,5kg/mg that was administered orally. In order to evaluate the animals, several exams were proceeded such as clinical (rectal temperature, heart and pulmonary rate and ruminal motricity),laboratorial (glucose, alcaline phosphatase, gamma glutamyl transferase, aspartase aminitranferase, creatinine, kinase, creatine, urea, bilirubin, total protein, albumin, globulin) and also histopatological examination. The bovines from G1 showed the first clinical signs between 4h28min and 5h55min and the whole group died after. First clinical signs presented by the G2 animals were between 2h45min and 4h05min after the plant was administered culminating in death of all animals. The main clinical signs observed was inapetence, jugular was dilated, tachycardia, tachypnea, paddling movements, vocalization, hypertermia and low ruminal motricity. The hematology and ALP results showed normal parameters in their results. However, GGT, bilirubin, urea and albumin showed relevant alterations that confirmed azotemia in animals that belonged to G1 and G2. Histopatology results showed multifocal citoplasmtic macrovacuolization and nuclei presenting picnose in epithelial cells from the convoluted tubules. The lesions that were found in the bovines kidneys’ were able to prove the efficacy of the intoxication protocol. The lower indexes of death were identified in Curraleiro Pé Duro bovines showing the greater resistency when compared to Nelore and Pantaneiro breeds. The shortest time interval between the plant intake and death suggest that Pantaneiro breed is more sensitive than Nelore and Curraleiro Pé Duro breeds.
O presente estudo teve como objetivo avaliar a resistência entre bovinos de diferentes raças à intoxicação experimental por Palicourea marcgravii A. St Hil. Foram utilizados seis bovinos de cada raça: Nelore(G1), Curraleiro Pé-Duro (G2) e Pantaneiro (G3). Os bovinos foram intoxicados experimentalmente por via oral na dose única de 0,5mg/kg do ácido monofluoracetato de sódio. Exames clínicos (temperatura retal, frequência cardíaca e respiratória e motricidade ruminal) e laboratoriais (glicose, fosfatase alcalina, gama glutamiltransferase, aspartato aminotranferase, creatina quinase, creatinina, ureia, bilirrubinas, proteínas totais, albumina e globulina), além dos exames histopatológicos foram realizados. Os bovinos G1 manifestaram os primeiros sinais clínicos entre 4h28min e 5h55min e todos morreram. Os animais G2 apresentaram os primeiros sinais clínicos entre 3h35min e 8h50min, três indivíduos morreram. Os bovinos G3 manifestaram os primeiros sinais clínicos entre 2h45min e 4h05min após a administração da planta e todos morreram. Os principais sinais observados foram inapetência, jugular distendida, taquicardia, taquipneia, movimentos de pedalagem, mugidos, hipertermia e diminuição da motricidade ruminal. Os resultados hematológicos e a dosagem de ALP foram insignificantes. Alterações de GGT, bilirrubina direta, ureia e albumina sugeriram danos hepáticos somente nos bovinos G1. Elevações de AST e CK indicaram alteração muscular nos três grupos raciais. Aumento dos níveis de creatinina e ureia confirmaram azotemia nos bovinos G1 e G2. Na histopatologia exceto em um bovino do G1, observou-se macrovacuolização citoplasmática multifocal em células epiteliais dos túbulos contorcidos, com picnose nuclear marcada. As lesões identificadas nos rins dos bovinos comprovaram a eficácia do protocolo de intoxicação. O menor índice de morte dos bovinos da raça Curraleiro indica uma maior resistência em comparação aos bovinos Nelore e Pantaneiro. O menor intervalo de tempo entre o fornecimento da planta e morte sugere que os bovinos Pantaneiros são mais sensíveis que os Nelores e Curraleiro Pé-Duro.
Books on the topic "Sodium monofluoroacetate"
Eisler, Ronald. Sodium monofluoroacetate (1080) hazards to fish, wildlife, and invertebrates: A synoptic review. Washington, D.C: U.S. Dept. of the Interior, National Biological Service, 1995.
Find full textEisler, Ronald. Sodium monofluoroacetate (1080) hazards to fish, wildlife, and invertebrates: A synoptic review. Washington, D.C: U.S. Dept. of the Interior, National Biological Service, 1995.
Find full textSodium monofluoroacetate (1080) hazards to fish, wildlife, and invertebrates: A synoptic review. Washington, D.C: U.S. Dept. of the Interior, National Biological Service, 1995.
Find full textBook chapters on the topic "Sodium monofluoroacetate"
"Sodium Monofluoroacetate." In Handbook of Chemical Risk Assessment. CRC Press, 2000. http://dx.doi.org/10.1201/9781420032741.ch26.
Full textParton, Kathy. "Sodium Monofluoroacetate (1080)." In Small Animal Toxicology, 811–16. Elsevier, 2013. http://dx.doi.org/10.1016/b978-1-4557-0717-1.00078-8.
Full textParton, Kathy. "Sodium Monofluoroacetate (1080)." In Small Animal Toxicology, 1055–62. Elsevier, 2006. http://dx.doi.org/10.1016/b0-72-160639-3/50080-0.
Full text