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Relevant bibliographies by topics / Sodium Ion Cells

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Journal articles

Academic literature on the topic 'Sodium Ion Cells'

Author: Grafiati

Published: 6 September 2023

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Journal articles on the topic "Sodium Ion Cells"

1

Ban, Yue, Benjamin E. Smith, and Michael R. Markham. "A highly polarized excitable cell separates sodium channels from sodium-activated potassium channels by more than a millimeter." Journal of Neurophysiology 114, no. 1 (2015): 520–30. http://dx.doi.org/10.1152/jn.00475.2014.

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The bioelectrical properties and resulting metabolic demands of electrogenic cells are determined by their morphology and the subcellular localization of ion channels. The electric organ cells (electrocytes) of the electric fish Eigenmannia virescens generate action potentials (APs) with Na+ currents >10 μA and repolarize the AP with Na+-activated K+ (KNa) channels. To better understand the role of morphology and ion channel localization in determining the metabolic cost of electrocyte APs, we used two-photon three-dimensional imaging to determine the fine cellular morphology and immunohist

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2

Li, Xianji, Andrew L. Hector, John R. Owen, and S. Imran U. Shah. "Evaluation of nanocrystalline Sn3N4derived from ammonolysis of Sn(NEt2)4as a negative electrode material for Li-ion and Na-ion batteries." Journal of Materials Chemistry A 4, no. 14 (2016): 5081–87. http://dx.doi.org/10.1039/c5ta08287k.

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Bulk nanocrystalline Sn<sub>3</sub>N<sub>4</sub>powders were synthesised by a two step ammonolysis route. These provided good capacities in sodium and lithium cells, and good stability in sodium cells.

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3

Sabzpoushan, S. H., and A. Faghani Ghodrat. "Role of Sodium Channel on Cardiac Action Potential." Engineering, Technology & Applied Science Research 2, no. 3 (2012): 232–36. http://dx.doi.org/10.48084/etasr.174.

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Sudden cardiac death is a major cause of death worldwide. In most cases, it's caused by abnormal action potential propagation that leads to cardiac arrhythmia. The aim of this article is to study the abnormal action potential propagation through sodium ion concentration variations. We use a new electrophysiological model that is both detailed and computationally efficient. This efficient model is based on the partial differential equation method. The central finite difference method is used for numerical solving of the two-dimensional (2D) wave propagation equation. Simulations are implemented

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4

Niu, Yu‐Bin, Ya‐Xia Yin, and Yu‐Guo Guo. "Nonaqueous Sodium‐Ion Full Cells: Status, Strategies, and Prospects." Small 15, no. 32 (2019): 1900233. http://dx.doi.org/10.1002/smll.201900233.

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5

Stanton, B. A., and B. Kaissling. "Regulation of renal ion transport and cell growth by sodium." American Journal of Physiology-Renal Physiology 257, no. 1 (1989): F1—F10. http://dx.doi.org/10.1152/ajprenal.1989.257.1.f1.

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Intracellular sodium has been implicated in a variety of cellular processes including regulation of Na+-K+-ATPase activity, mitogen-induced cell growth, and proliferation and stimulation of Na+-K+-ATPase by aldosterone. In renal epithelial cells a rise in sodium uptake across the apical membrane increases intracellular sodium concentration, which in turn stimulates the turnover rate of Na+-K+-ATPase and thereby enhances sodium efflux across the basolateral membrane. A prolonged increase in sodium uptake causes dramatic hypertrophy and hyperplasia and a rise in the quantity of Na+-K+-ATPase in

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6

Karasawa, Akira, Haijiao Liu, Matthias Quick, Wayne A. Hendrickson, and Qun Liu. "Crystallographic Characterization of Sodium Ions in a Bacterial Leucine/Sodium Symporter." Crystals 13, no. 2 (2023): 183. http://dx.doi.org/10.3390/cryst13020183.

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Na+ is the most abundant ion in living organisms and plays essential roles in regulating nutrient uptake, muscle contraction, and neurotransmission. The identification of Na+ in protein structures is crucial for gaining a deeper understanding of protein function in a physiological context. LeuT, a bacterial homolog of the neurotransmitter:sodium symporter family, uses the Na+ gradient to power the uptake of amino acids into cells and has been used as a paradigm for the study of Na+-dependent transport systems. We have devised a low-energy multi-crystal approach for characterizing low-Z (Z ≤ 20

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7

Berkowitz, L. R., and E. P. Orringer. "Passive sodium and potassium movements in sickle erythrocytes." American Journal of Physiology-Cell Physiology 249, no. 3 (1985): C208—C214. http://dx.doi.org/10.1152/ajpcell.1985.249.3.c208.

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Deoxygenation causes an increase in passive Na and K movements across the membrane of the sickle erythrocyte. Some investigators find that these ion movements are accompanied by cell dehydration, while others find no evidence for cell water loss with sickling. Because gelation of hemoglobin S would be enhanced by cell water loss, we reinvestigated Na and K movements in sickle cells to define further the role that ion movements might play in the pathogenesis of sickling. With deoxygenation, we found that sickle cells gained Na and lost K without losing cell water. These net ion movements were n

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8

Peters, Jens, Alexandra Peña Cruz, and Marcel Weil. "Exploring the Economic Potential of Sodium-Ion Batteries." Batteries 5, no. 1 (2019): 10. http://dx.doi.org/10.3390/batteries5010010.

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Sodium-ion batteries (SIBs) are a recent development being promoted repeatedly as an economically promising alternative to lithium-ion batteries (LIBs). However, only one detailed study about material costs has yet been published for this battery type. This paper presents the first detailed economic assessment of 18,650-type SIB cells with a layered oxide cathode and a hard carbon anode, based on existing datasheets for pre-commercial battery cells. The results are compared with those of competing LIB cells, that is, with lithium-nickel-manganese-cobalt-oxide cathodes (NMC) and with lithium-ir

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9

Cragg, Peter J. "Artificial Transmembrane Channels for Sodium and Potassium." Science Progress 85, no. 3 (2002): 219–41. http://dx.doi.org/10.3184/003685002783238780.

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Transport of alkali metals, particularly sodium and potassium, across cell membranes is an essential function performed by special proteins that enable cells to regulate inter- and extracellular ion concentrations with exceptional selectivity. The importance of these channel-forming proteins has led to researchers emulating of their structural features: an ion-specific filter and conduction at rates up to 108 ions per second. Synthetic helical and cyclic polypeptides form channels, however, the specificity of ion transport is often low. Ion-specific macrocycles have been used as filters from w

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10

Van Mil, H. G. J. "Analysis of a Model Describing the Dynamics of Intracellular Ion Composition in Biological Cells." International Journal of Bifurcation and Chaos 08, no. 05 (1998): 1043–47. http://dx.doi.org/10.1142/s0218127498000851.

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An electrophysiological model describing the dynamics of the intracellular ion concentration and the membrane potential (Vm) in biological cells is presented. The model links passive ion fluxes through channels of sodium, potassium and chloride to active ion fluxes generated by the sodium potassium pump. To model the interaction of Vm to the ionic fluxes Kirchhoff current law is used. Only one Vm-dependent permeability as represented by an inwardly rectifying potassium channel (IKR) is incorporated. It is shown that the resulting system of ordinary differential equations is degenerate. Decompo

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