Journal articles on the topic 'SOD1G93A mouse model'
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Kirby, Alastair J., Thomas Palmer, Richard J. Mead, Ronaldo M. Ichiyama, and Samit Chakrabarty. "Caudal–Rostral Progression of Alpha Motoneuron Degeneration in the SOD1G93A Mouse Model of Amyotrophic Lateral Sclerosis." Antioxidants 11, no. 5 (May 17, 2022): 983. http://dx.doi.org/10.3390/antiox11050983.
Full textBonifacino, Tiziana, Claudia Rebosio, Francesca Provenzano, Carola Torazza, Matilde Balbi, Marco Milanese, Luca Raiteri, Cesare Usai, Ernesto Fedele, and Giambattista Bonanno. "Enhanced Function and Overexpression of Metabotropic Glutamate Receptors 1 and 5 in the Spinal Cord of the SOD1G93A Mouse Model of Amyotrophic Lateral Sclerosis during Disease Progression." International Journal of Molecular Sciences 20, no. 18 (September 13, 2019): 4552. http://dx.doi.org/10.3390/ijms20184552.
Full textMartin, Elodie, William Cazenave, Anne-Emilie Allain, Daniel Cattaert, and Pascal Branchereau. "Implication of 5-HT in the Dysregulation of Chloride Homeostasis in Prenatal Spinal Motoneurons from the G93A Mouse Model of Amyotrophic Lateral Sclerosis." International Journal of Molecular Sciences 21, no. 3 (February 7, 2020): 1107. http://dx.doi.org/10.3390/ijms21031107.
Full textRudnick, Noam D., Christopher J. Griffey, Paolo Guarnieri, Valeria Gerbino, Xueyong Wang, Jason A. Piersaint, Juan Carlos Tapia, Mark M. Rich, and Tom Maniatis. "Distinct roles for motor neuron autophagy early and late in the SOD1G93A mouse model of ALS." Proceedings of the National Academy of Sciences 114, no. 39 (September 13, 2017): E8294—E8303. http://dx.doi.org/10.1073/pnas.1704294114.
Full textChiu, Isaac M., Adam Chen, Yi Zheng, Bela Kosaras, Stefanos A. Tsiftsoglou, Timothy K. Vartanian, Robert H. Brown, and Michael C. Carroll. "T lymphocytes potentiate endogenous neuroprotective inflammation in a mouse model of ALS." Proceedings of the National Academy of Sciences 105, no. 46 (November 7, 2008): 17913–18. http://dx.doi.org/10.1073/pnas.0804610105.
Full textTankersley, Clarke G., Christine Haenggeli, and Jeffery D. Rothstein. "Respiratory impairment in a mouse model of amyotrophic lateral sclerosis." Journal of Applied Physiology 102, no. 3 (March 2007): 926–32. http://dx.doi.org/10.1152/japplphysiol.00193.2006.
Full textMoreno-Martinez, Laura, Miriam de la Torre, María J. Muñoz, Pilar Zaragoza, José Aguilera, Ana C. Calvo, and Rosario Osta. "Neuroprotective Fragment C of Tetanus Toxin Modulates IL-6 in an ALS Mouse Model." Toxins 12, no. 5 (May 17, 2020): 330. http://dx.doi.org/10.3390/toxins12050330.
Full textRojas, Pilar, Ana I. Ramírez, Manuel Cadena, José A. Fernández-Albarral, Elena Salobrar-García, Inés López-Cuenca, Irene Santos-García, et al. "Retinal Ganglion Cell Loss and Microglial Activation in a SOD1G93A Mouse Model of Amyotrophic Lateral Sclerosis." International Journal of Molecular Sciences 22, no. 4 (February 7, 2021): 1663. http://dx.doi.org/10.3390/ijms22041663.
Full textMallozzi, Cinzia, Alida Spalloni, Patrizia Longone, and Maria Rosaria Domenici. "Activation of Phosphotyrosine-Mediated Signaling Pathways in the Cortex and Spinal Cord of SOD1G93A, a Mouse Model of Familial Amyotrophic Lateral Sclerosis." Neural Plasticity 2018 (August 5, 2018): 1–10. http://dx.doi.org/10.1155/2018/2430193.
Full textRei, Nádia, Cláudia A. Valente, Sandra H. Vaz, Miguel Farinha-Ferreira, Joaquim A. Ribeiro, and Ana M. Sebastião. "Changes in adenosine receptors and neurotrophic factors in the SOD1G93A mouse model of amyotrophic lateral sclerosis: Modulation by chronic caffeine." PLOS ONE 17, no. 12 (December 14, 2022): e0272104. http://dx.doi.org/10.1371/journal.pone.0272104.
Full textPost, Julia, Anja Schaffrath, Ian Gering, Sonja Hartwig, Stefan Lehr, N. Jon Shah, Karl-Josef Langen, Dieter Willbold, Janine Kutzsche, and Antje Willuweit. "Oral Treatment with RD2RD2 Impedes Development of Motoric Phenotype and Delays Symptom Onset in SOD1G93A Transgenic Mice." International Journal of Molecular Sciences 22, no. 13 (June 30, 2021): 7066. http://dx.doi.org/10.3390/ijms22137066.
Full textBette, Michael, Eileen Cors, Carolin Kresse, and Burkhard Schütz. "Therapeutic Treatment of Superoxide Dismutase 1 (G93A) Amyotrophic Lateral Sclerosis Model Mice with Medical Ozone Decelerates Trigeminal Motor Neuron Degeneration, Attenuates Microglial Proliferation, and Preserves Monocyte Levels in Mesenteric Lymph Nodes." International Journal of Molecular Sciences 23, no. 6 (March 21, 2022): 3403. http://dx.doi.org/10.3390/ijms23063403.
Full textCrivello, Martin, Saidhbhe L. O'Riordan, Ina Woods, Sarah Cannon, Luise Halang, Karen S. Coughlan, Marion C. Hogg, Sebastian A. Lewandowski, and Jochen H. M. Prehn. "Pleiotropic activity of systemically delivered angiogenin in the SOD1G93A mouse model." Neuropharmacology 133 (May 2018): 503–11. http://dx.doi.org/10.1016/j.neuropharm.2018.02.022.
Full textBame, Monica, Robert E. Grier, Richard Needleman, and William S. A. Brusilow. "Amino Acids as biomarkers in the SOD1G93A mouse model of ALS." Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease 1842, no. 1 (January 2014): 79–87. http://dx.doi.org/10.1016/j.bbadis.2013.10.004.
Full textMarini, Cecilia, Vanessa Cossu, Mandeep Kumar, Marco Milanese, Katia Cortese, Silvia Bruno, Grazia Bellese, et al. "The Role of Endoplasmic Reticulum in the Differential Endurance against Redox Stress in Cortical and Spinal Astrocytes from the Newborn SOD1G93A Mouse Model of Amyotrophic Lateral Sclerosis." Antioxidants 10, no. 9 (August 30, 2021): 1392. http://dx.doi.org/10.3390/antiox10091392.
Full textDobrowolny, Gabriella, Cristina Giacinti, Laura Pelosi, Carmine Nicoletti, Nadine Winn, Laura Barberi, Mario Molinaro, Nadia Rosenthal, and Antonio Musarò. "Muscle expression of a local Igf-1 isoform protects motor neurons in an ALS mouse model." Journal of Cell Biology 168, no. 2 (January 17, 2005): 193–99. http://dx.doi.org/10.1083/jcb.200407021.
Full textVarghese, Merina, Wei Zhao, Kyle J. Trageser, and Giulio M. Pasinetti. "Peroxisome Proliferator Activator Receptor Gamma Coactivator-1α Overexpression in Amyotrophic Lateral Sclerosis: A Tale of Two Transgenics." Biomolecules 10, no. 5 (May 13, 2020): 760. http://dx.doi.org/10.3390/biom10050760.
Full textKoschnitzky, J. E., K. A. Quinlan, T. J. Lukas, E. Kajtaz, E. J. Kocevar, W. F. Mayers, T. Siddique, and C. J. Heckman. "Effect of fluoxetine on disease progression in a mouse model of ALS." Journal of Neurophysiology 111, no. 11 (June 1, 2014): 2164–76. http://dx.doi.org/10.1152/jn.00425.2013.
Full textTaes, Ines, Mieke Timmers, Nicole Hersmus, André Bento-Abreu, Ludo Van Den Bosch, Philip Van Damme, Johan Auwerx, and Wim Robberecht. "Hdac6 deletion delays disease progression in the SOD1G93A mouse model of ALS." Human Molecular Genetics 22, no. 9 (January 30, 2013): 1783–90. http://dx.doi.org/10.1093/hmg/ddt028.
Full textDibaj, Payam, Eike D. Schomburg, and Heinz Steffens. "Contractile characteristics of gastrocnemius-soleus muscle in the SOD1G93A ALS mouse model." Neurological Research 37, no. 8 (April 28, 2015): 693–702. http://dx.doi.org/10.1179/1743132815y.0000000039.
Full textZeldich, Ella, Ci-Di Chen, Emma Boden, Bryce Howat, Jason S. Nasse, Dean Zeldich, Anthony G. Lambert, et al. "Klotho Is Neuroprotective in the Superoxide Dismutase (SOD1G93A) Mouse Model of ALS." Journal of Molecular Neuroscience 69, no. 2 (June 27, 2019): 264–85. http://dx.doi.org/10.1007/s12031-019-01356-2.
Full textSchuster, J. E., R. Fu, T. Siddique, and C. J. Heckman. "Effect of prolonged riluzole exposure on cultured motoneurons in a mouse model of ALS." Journal of Neurophysiology 107, no. 1 (January 2012): 484–92. http://dx.doi.org/10.1152/jn.00714.2011.
Full textTan, Honglin, Mina Chen, Dejiang Pang, Xiaoqiang Xia, Chongyangzi Du, Wanchun Yang, Yiyuan Cui, et al. "LanCL1 promotes motor neuron survival and extends the lifespan of amyotrophic lateral sclerosis mice." Cell Death & Differentiation 27, no. 4 (September 30, 2019): 1369–82. http://dx.doi.org/10.1038/s41418-019-0422-6.
Full textMéndez-López, Iago, Francisco J. Sancho-Bielsa, Tobias Engel, Antonio G. G. García, and Juan Fernando Padín. "Progressive Mitochondrial SOD1G93A Accumulation Causes Severe Structural, Metabolic and Functional Aberrations through OPA1 Down-Regulation in a Mouse Model of Amyotrophic Lateral Sclerosis." International Journal of Molecular Sciences 22, no. 15 (July 30, 2021): 8194. http://dx.doi.org/10.3390/ijms22158194.
Full textValdebenito-Maturana, Braulio, Esteban Arancibia, Gonzalo Riadi, Juan Carlos Tapia, and Mónica Carrasco. "Locus-specific analysis of Transposable Elements during the progression of ALS in the SOD1G93A mouse model." PLOS ONE 16, no. 10 (October 6, 2021): e0258291. http://dx.doi.org/10.1371/journal.pone.0258291.
Full textOgbu, Destiny, Yongguo Zhang, Katerina Claud, Yinglin Xia, and Jun Sun. "Target Metabolites to Slow Down Progression of Amyotrophic Lateral Sclerosis in Mice." Metabolites 12, no. 12 (December 12, 2022): 1253. http://dx.doi.org/10.3390/metabo12121253.
Full textPambo-Pambo, Arnaud, Jacques Durand, and Jean-Patrick Gueritaud. "Early Excitability Changes in Lumbar Motoneurons of Transgenic SOD1G85R and SOD1G93A-Low Mice." Journal of Neurophysiology 102, no. 6 (December 2009): 3627–42. http://dx.doi.org/10.1152/jn.00482.2009.
Full textSpalloni, Alida, Viviana Greco, Giulia Ciriminna, Victor Corasolla Carregari, Federica Marini, Luisa Pieroni, Nicola B. Mercuri, Andrea Urbani, and Patrizia Longone. "Impact of Pharmacological Inhibition of Hydrogen Sulphide Production in the SOD1G93A-ALS Mouse Model." International Journal of Molecular Sciences 20, no. 10 (May 24, 2019): 2550. http://dx.doi.org/10.3390/ijms20102550.
Full textOLIVÁN, Sara, Ana Cristina CALVO, Amaya RANDO, María Jesús MUÑOZ, Pilar ZARAGOZA, and Rosario OSTA. "Comparative study of behavioural tests in the SOD1G93A mouse model of amyotrophic lateral sclerosis." Experimental Animals 64, no. 2 (2015): 147–53. http://dx.doi.org/10.1538/expanim.14-0077.
Full textDutta, Kallol, Priyanka Patel, and Jean-Pierre Julien. "Protective effects of Withania somnifera extract in SOD1G93A mouse model of amyotrophic lateral sclerosis." Experimental Neurology 309 (November 2018): 193–204. http://dx.doi.org/10.1016/j.expneurol.2018.08.008.
Full textSchomburg, Eike D., Heinz Steffens, Jana Zschüntzsch, Payam Dibaj, and Bernhard U. Keller. "Fatigability of spinal reflex transmission in a mouse model (SOD1G93A ) of amyotrophic lateral sclerosis." Muscle & Nerve 43, no. 2 (January 19, 2011): 230–36. http://dx.doi.org/10.1002/mus.21835.
Full textClark, Courtney M., Rosemary M. Clark, Joshua A. Hoyle, Jyoti A. Chuckowree, Catriona A. McLean, and Tracey C. Dickson. "Differential NPY-Y1 Receptor Density in the Motor Cortex of ALS Patients and Familial Model of ALS." Brain Sciences 11, no. 8 (July 23, 2021): 969. http://dx.doi.org/10.3390/brainsci11080969.
Full textPascali, Giancarlo, Daniele Panetta, Mariarosaria De Simone, Silvia Burchielli, Valentina Lucchesi, Elena Sanguinetti, Simone Zanoni, et al. "Preliminary Investigation of a Novel 18F Radiopharmaceutical for Imaging CB2 Receptors in a SOD Mouse Model." Australian Journal of Chemistry 74, no. 6 (2021): 443. http://dx.doi.org/10.1071/ch20247.
Full textViolatto, Martina Bruna, Laura Pasetto, Elisabetta Casarin, Camilla Tondello, Elisa Schiavon, Laura Talamini, Gloria Marchini, et al. "Development of a Nanoparticle-Based Approach for the Blood–Brain Barrier Passage in a Murine Model of Amyotrophic Lateral Sclerosis." Cells 11, no. 24 (December 10, 2022): 4003. http://dx.doi.org/10.3390/cells11244003.
Full textNovoselov, Sergey S., Wendy J. Mustill, Anna L. Gray, James R. Dick, Naheed Kanuga, Bernadett Kalmar, Linda Greensmith, and Michael E. Cheetham. "Molecular Chaperone Mediated Late-Stage Neuroprotection in the SOD1G93A Mouse Model of Amyotrophic Lateral Sclerosis." PLoS ONE 8, no. 8 (August 30, 2013): e73944. http://dx.doi.org/10.1371/journal.pone.0073944.
Full textSeki, Soju, Toru Yamamoto, Kiara Quinn, Igor Spigelman, Antonios Pantazis, Riccardo Olcese, Martina Wiedau-Pazos, Scott H. Chandler, and Sharmila Venugopal. "Circuit-Specific Early Impairment of Proprioceptive Sensory Neurons in the SOD1G93A Mouse Model for ALS." Journal of Neuroscience 39, no. 44 (September 17, 2019): 8798–815. http://dx.doi.org/10.1523/jneurosci.1214-19.2019.
Full textBoërio, Delphine, Bernadett Kalmar, Linda Greensmith, and Hugh Bostock. "Excitability properties of mouse motor axons in the mutant SOD1G93A model of amyotrophic lateral sclerosis." Muscle & Nerve 41, no. 6 (January 21, 2010): 774–84. http://dx.doi.org/10.1002/mus.21579.
Full textFabbrizio, Paola, Savina Apolloni, Andrea Bianchi, Illari Salvatori, Cristiana Valle, Chiara Lanzuolo, Caterina Bendotti, Giovanni Nardo, and Cinzia Volonté. "P2X7 activation enhances skeletal muscle metabolism and regeneration in SOD1G93A mouse model of amyotrophic lateral sclerosis." Brain Pathology 30, no. 2 (August 18, 2019): 272–82. http://dx.doi.org/10.1111/bpa.12774.
Full textBryson, J. Barney, Carl Hobbs, Michael J. Parsons, Karen D. Bosch, Amelie Pandraud, Frank S. Walsh, Patrick Doherty, and Linda Greensmith. "Amyloid precursor protein (APP) contributes to pathology in the SOD1G93A mouse model of amyotrophic lateral sclerosis." Human Molecular Genetics 21, no. 17 (June 7, 2012): 3871–82. http://dx.doi.org/10.1093/hmg/dds215.
Full textLepore, Angelo C., John O'Donnell, Andrew S. Kim, Timothy Williams, Alicia Tuteja, Mahendra S. Rao, Linda L. Kelley, James T. Campanelli, and Nicholas J. Maragakis. "Human Glial-Restricted Progenitor Transplantation into Cervical Spinal Cord of the SOD1G93A Mouse Model of ALS." PLoS ONE 6, no. 10 (October 5, 2011): e25968. http://dx.doi.org/10.1371/journal.pone.0025968.
Full textHegedus, J., C. T. Putman, and T. Gordon. "Time course of preferential motor unit loss in the SOD1G93A mouse model of amyotrophic lateral sclerosis." Neurobiology of Disease 28, no. 2 (November 2007): 154–64. http://dx.doi.org/10.1016/j.nbd.2007.07.003.
Full textSábado, J., A. Casanovas, H. Rodrigo, G. Arqué, and J. E. Esquerda. "Adverse effects of a SOD1-peptide immunotherapy on SOD1G93A mouse slow model of amyotrophic lateral sclerosis." Neuroscience 310 (December 2015): 38–50. http://dx.doi.org/10.1016/j.neuroscience.2015.09.027.
Full textBianchi, F., C. Rossi, L. Muzio, N. Riva, C. Butera, M. Cursi, S. Amadio, G. Comi, A. Quattrini, and U. Del Carro. "54. Early neurophysiological signs of disease in SOD1G93A mouse model: A stimulated single-fiber electromyography study." Clinical Neurophysiology 127, no. 12 (December 2016): e336. http://dx.doi.org/10.1016/j.clinph.2016.10.066.
Full textKang, Jihong, and Serge Rivest. "MyD88-deficient bone marrow cells accelerate onset and reduce survival in a mouse model of amyotrophic lateral sclerosis." Journal of Cell Biology 179, no. 6 (December 17, 2007): 1219–30. http://dx.doi.org/10.1083/jcb.200705046.
Full textVallarola, Antonio, Massimo Tortarolo, Roberta De Gioia, Luisa Iamele, Hugo de Jonge, Giovanni de Nola, Enrica Bovio, et al. "A Novel HGF/SF Receptor (MET) Agonist Transiently Delays the Disease Progression in an Amyotrophic Lateral Sclerosis Mouse Model by Promoting Neuronal Survival and Dampening the Immune Dysregulation." International Journal of Molecular Sciences 21, no. 22 (November 12, 2020): 8542. http://dx.doi.org/10.3390/ijms21228542.
Full textFlis, Damian Jozef, Katarzyna Dzik, Jan Jacek Kaczor, Karol Cieminski, Malgorzata Halon-Golabek, Jedrzej Antosiewicz, Mariusz Roman Wieckowski, and Wieslaw Ziolkowski. "Swim Training Modulates Mouse Skeletal Muscle Energy Metabolism and Ameliorates Reduction in Grip Strength in a Mouse Model of Amyotrophic Lateral Sclerosis." International Journal of Molecular Sciences 20, no. 2 (January 9, 2019): 233. http://dx.doi.org/10.3390/ijms20020233.
Full textMead, Richard J., Ellen J. Bennett, Aneurin J. Kennerley, Paul Sharp, Claire Sunyach, Paul Kasher, Jason Berwick, et al. "Optimised and Rapid Pre-clinical Screening in the SOD1G93A Transgenic Mouse Model of Amyotrophic Lateral Sclerosis (ALS)." PLoS ONE 6, no. 8 (August 18, 2011): e23244. http://dx.doi.org/10.1371/journal.pone.0023244.
Full textHuang, G., X. Lee, Y. Bian, Z. Shao, G. Sheng, R. B. Pepinsky, and S. Mi. "Death receptor 6 (DR6) antagonist antibody is neuroprotective in the mouse SOD1G93A model of amyotrophic lateral sclerosis." Cell Death & Disease 4, no. 10 (October 2013): e841-e841. http://dx.doi.org/10.1038/cddis.2013.378.
Full textNoh, Min Young, Kyung Ah Cho, Heejaung Kim, Sung-Min Kim, and Seung Hyun Kim. "Erythropoietin modulates the immune-inflammatory response of a SOD1G93A transgenic mouse model of amyotrophic lateral sclerosis (ALS)." Neuroscience Letters 574 (June 2014): 53–58. http://dx.doi.org/10.1016/j.neulet.2014.05.001.
Full textRei, N., D. M. Rombo, M. F. Ferreira, Y. Baqi, C. E. Müller, J. A. Ribeiro, A. M. Sebastião, and S. H. Vaz. "Hippocampal synaptic dysfunction in the SOD1G93A mouse model of Amyotrophic Lateral Sclerosis: Reversal by adenosine A2AR blockade." Neuropharmacology 171 (July 2020): 108106. http://dx.doi.org/10.1016/j.neuropharm.2020.108106.
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