Relevant bibliographies by topics / Sleep markers

Academic literature on the topic 'Sleep markers'

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Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Sleep markers"

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Rector, David M., Jennifer L. Schei, Hans P. A. Van Dongen, Gregory Belenky, and James M. Krueger. "Physiological markers of local sleep." European Journal of Neuroscience 29, no. 9 (May 2009): 1771–78. http://dx.doi.org/10.1111/j.1460-9568.2009.06717.x.

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Martínez-Rodríguez, Jose E., and Joan Santamaria. "CSF markers in sleep neurobiology." Clinica Chimica Acta 362, no. 1-2 (December 2005): 12–25. http://dx.doi.org/10.1016/j.cccn.2005.05.014.

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Orr, William C., Joe A. Othman, and O. H. Rundell. "Genetic Markers in Familial Narcolepsy." Sleep 14, no. 3 (May 1991): 270–71. http://dx.doi.org/10.1093/sleep/14.3.270.

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Dennis, Jeff A., Ahmad Alazzeh, Ann Marie Kumfer, Rebecca McDonald-Thomas, and Alan N. Peiris. "The Association of Unreported Sleep Disturbances and Systemic Inflammation: Findings from the 2005-2008 NHANES." Sleep Disorders 2018 (October 9, 2018): 1–8. http://dx.doi.org/10.1155/2018/5987064.

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Background/Objective. Sleep apnea is associated with elevated inflammatory markers. A subgroup of patients never report sleep disturbances to their physician. The inflammatory status of this subgroup is not known. The present study aims to evaluate two inflammatory markers, C-reactive protein (CRP) and red cell distribution width (RDW), in those with unreported sleep disturbances and compares these findings to those with and without reported sleep disorders. We also investigate the utility of RDW as an inflammatory marker in sleep disorders. Methods. Sample includes 9,901 noninstitutionalized, civilian, nonpregnant adults from the 2005-2008 National Health and Nutrition Examination Survey, a nationally representative, cross-sectional U.S. study. Sleep questionnaire and laboratory data were used to compare inflammatory markers (CRP and RDW) in five subgroups of individuals: reporting physician-diagnosed sleep apnea, reporting another physician-diagnosed sleep disorder, reported sleep disturbance to physician with no resulting diagnosis, unreported sleep disturbance (poor sleep quality not reported to physician), and no diagnosed sleep disorder or sleep disturbance. Results. Individuals with unreported sleep disturbance had significantly higher odds of elevated RDW (>13.6%) when compared to those without a sleep disturbance in adjusted models (OR=1.33). Those with unreported sleep disturbance had significantly higher odds of elevated CRP levels (>1 mg/L) than those without sleep disturbances (OR 1.34), although the association was not significant when adjusted for obesity and other controls. Conclusion. Self-identified unreported sleep disturbances are associated with significantly higher odds of elevated RDW than those without sleep disturbances. RDW may serve as a valuable indicator in identifying individuals at higher risk for sleep apnea and other sleep disorders.
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Hirsch, Sophie, Jane Gaultney, and Patricia Crane. "018 The Role of Inflammatory Markers in Sleep in Individuals Post-Myocardial Infarction." Sleep 44, Supplement_2 (May 1, 2021): A9. http://dx.doi.org/10.1093/sleep/zsab072.017.

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Abstract Introduction Despite positive secondary prevention strategies post myocardial infarction (MI), including statin use and lifestyle changes, 32% of the annual MIs are recurrent (MIR). As coronary heart disease is related to atherosclerosis, a chronic inflammatory process, and sleep is associated with cardiovascular disease and innate immunity, understanding the role of sleep and inflammation and MIR is important in developing interventions to improve sleep, reduce inflammation, and delay or prevent MIR. This study aimed to explore the role of sleep quality and inflammatory markers on MIRs. Methods We conducted a secondary analysis of cross-sectional data of individuals (N=156) having at least one or more MIs within the last 3 to 7 years. Using the hypothalamus-pituitary-adrenal axis model (Irwin, 2019), we tested sleep quality (Pittsburgh Sleep Quality Index [PSQI]) predicting MIR, using inflammatory markers (hs C-Reactor Protein [CRP], Interleukin-1ß [IL-1ß] and Tumor Necrosis Factor alpha [TNFα]) as the simultaneous indirect paths. Race, sex and body mass index (BMI) were also examined using moderated mediation. Results The sample ranged in age from 34 to 92 (M = 65.37, SD = 12.13), BMI averaged 31.11 (SD = 7.34), and was comprised of mostly male (57.1%) and White adults (67.9%). PSQI predicted only IL-1ß (ß= .02; p < .01). IL-1ß predicted MIR (ß = .80, p = .05). The direct effect of PSQI to MIR was not significant (p =.12), the indirect path via IL-1ß was. This relationship was not moderated by race, sex, nor BMI. Conclusion IL-1ß is an inflammatory marker elevated after acute MI which does not reflect our selected sample. Inflammation may be an important marker of risk for MIR in those with poor sleep quality. Future studies should examine other markers of inflammation and sleep in those with MIR. Support (if any):
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Mendlewicz, Julien, I. Schweitzer, N. Biddle, G. Szmukler, and B. M. Davies. "SLEEP DEPRIVATION, DIETING, AND DEPRESSION MARKERS." Lancet 329, no. 8524 (January 1987): 105–6. http://dx.doi.org/10.1016/s0140-6736(87)91945-3.

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Mullen, PaulE, and ChristopherR Linsell. "SLEEP DEPRIVATION, DIETING, AND DEPRESSION MARKERS." Lancet 329, no. 8528 (February 1987): 323. http://dx.doi.org/10.1016/s0140-6736(87)92043-5.

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Goel, Namni. "Genetic Markers of Sleep and Sleepiness." Sleep Medicine Clinics 12, no. 3 (September 2017): 289–99. http://dx.doi.org/10.1016/j.jsmc.2017.03.005.

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Wang, Lei, Paul Y. Kim, David E. McCarty, Clifton Frilot, Andrew L. Chesson, Simona Carrubba, and Andrew A. Marino. "EEG recurrence markers and sleep quality." Journal of the Neurological Sciences 331, no. 1-2 (August 2013): 26–30. http://dx.doi.org/10.1016/j.jns.2013.04.019.

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Kaufmann, Christopher, and Katie Stone. "INTERACTIONS BETWEEN SLEEP AND BIOLOGICAL MARKERS OF AGING." Innovation in Aging 6, Supplement_1 (November 1, 2022): 363. http://dx.doi.org/10.1093/geroni/igac059.1435.

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Abstract There is well documented evidence that sleep architecture and circadian patterns change as people age in part due to accumulation of comorbidities, polypharmacy, and other factors inherent to the aging process. While a number of studies have examined sleep in the context of these factors, recent advances in the assessment of biological aging (including epigenetic age, metabolomics, and measurement of inflammatory biomarkers) have made it possible to identify potential mechanisms by which sleep impacts the aging course. In this symposium, we will highlight research that investigates the link between sleep disturbances and biological factors in order to identify whether sleep could be a modifiable risk factor for accelerated aging. Our first presentation will examine the association between insomnia symptoms and short sleep duration with epigenetic age acceleration using data from the Health and Retirement Study. Second, we will describe whether objectively measured sleep characteristics are associated with a number of metabolomic markers of aging. The third presentation will center around the extent to which social disparities in presence of inflammatory biomarkers may be mediated by sleep quality. Finally, we will examine how daytime sleepiness may be associated with longitudinal BMI trajectories. These presentations will, as a whole, highlight the importance of sleep as a contributor to healthy aging and longevity, and inform the development of interventional approaches targeting biological mechanisms to promote successful aging more broadly.
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Dissertations / Theses on the topic "Sleep markers"

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Hollway, Jill Ann. "CORRELATES AND RISK MARKERS FOR SLEEP DISTURBANCE IN CHILDREN WITH AUTISM SPECTRUM DISORDERS." The Ohio State University, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=osu1339639912.

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Lutsey, Pamela L., Faye L. Norby, Rebecca F. Gottesman, Thomas Mosley, Richard F. MacLehose, Naresh M. Punjabi, Eyal Shahar, Clifford R. Jack, and Alvaro Alonso. "Sleep Apnea, Sleep Duration and Brain MRI Markers of Cerebral Vascular Disease and Alzheimer’s Disease: The Atherosclerosis Risk in Communities Study (ARIC)." Public Library of Science, 2016. http://hdl.handle.net/10150/621324.

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Background A growing body of literature has suggested that obstructive sleep apnea (OSA) and habitual short sleep duration are linked to poor cognitive function. Neuroimaging studies may provide insight into this relation. Objective We tested the hypotheses that OSA and habitual short sleep duration, measured at ages 54-73 years, would be associated with adverse brain morphology at ages 67-89 years. Methods Included in this analysis are 312 ARIC study participants who underwent in-home overnight polysomnography in 1996-1998 and brain MRI scans about 15 years later (2012-2013). Sleep apnea was quantified by the apnea-hypopnea index and categorized as moderate/ severe (>= 15.0 events/hour), mild (5.0-14.9 events/hour), or normal (<5.0 events/hour). Habitual sleep duration was categorized, in hours, as <7, 7 to <8, >= 8. MRI outcomes included number of infarcts (total, subcortical, and cortical) and white matter hyperintensity (WMH) and Alzheimer's disease signature region volumes. Multivariable adjusted logistic and linear regression models were used. All models incorporated inverse probability weighting, to adjust for potential selection bias. Results At the time of the sleep study participants were 61.7 (SD: 5.0) years old and 54% female; 19% had moderate/severe sleep apnea. MRI imaging took place 14.8 (SD: 1.0) years later, when participants were 76.5 (SD: 5.2) years old. In multivariable models which accounted for body mass index, neither OSA nor abnormal sleep duration were statistically significantly associated with odds of cerebral infarcts, WMH brain volumes or regional brain volumes. Conclusions In this community-based sample, mid-life OSA and habitually short sleep duration were not associated with later-life cerebral markers of vascular dementia and Alzheimer's disease. However, selection bias may have influenced our results and the modest sample size led to relatively imprecise associations.
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Marcolongo, Ellen. "The Relationships Between Sleep Disturbances, Depression, Inflammatory Markers, and Sexual Trauma in Female Veterans." Scholar Commons, 2014. https://scholarcommons.usf.edu/etd/5266.

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The purpose of this secondary data analysis was to assess for the relationships among sleep disturbances, depressive symptoms, inflammatory markers, and sexual trauma in female veterans. This may contribute to an understanding of the physical and mental health effects of sexual trauma in female veterans. Correlational analyses were conducted to evaluate the strength of these relationships. A reported history of sexual trauma was significantly correlated with longer sleep latencies, poorer sleep efficiency, shorter sleep durations, more daytime dysfunction, and poorer overall sleep quality in female veterans. A reported history of sexual trauma was also significantly correlated with depressive symptoms including anhedonia and a negative affect in female veterans. No significant correlations were noted between inflammatory markers and a reported history of sexual trauma in female veterans. Female veterans with a reported history of sexual trauma had more trouble falling and staying asleep, had more trouble functioning during daytime hours, and had total poorer sleep quality. These veterans also appeared depressed and they found normally pleasurable activities unenjoyable. Disturbed sleep and depressive symptoms may be risk factors in the development of chronic health diseases. By assessing and treating the sleep disturbances and depressive symptoms experienced by sexually traumatized female veterans, nurses may help to prevent the development of costly and deadly chronic diseases
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Blevins, Jennifer Susanne. "The relationship between markers of disease severity in obstructive sleep apnea patients and hemodynamic and respiratory function during graded exercise testing." Diss., Virginia Tech, 2000. http://hdl.handle.net/10919/29947.

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Obstructive sleep apnea (OSA) is estimated to affect 2 to 4 percent of the adult population (Young T 1993, Skomro and Kryger 1999). However, an estimated 80 to 90 percent of adults with moderate to severe OSA may be clinically undiagnosed. Identification of those at risk and their subsequent diagnosis is, obviously, of great concern to clinicians. This investigation included three distinct research aims, which were the following: (1): In order to establish reliability of hemodynamic measures to be used during exercise testing, a study was conducted on the acetylene single-breath cardiac output (Qc) technique in 15 healthy subjects. This was completed in order to establish reliability of exercise Qc and total peripheral resistance (TPR), these responses could then be investigated acutely in the context of evaluating the relation of these measures to markers of disease in OSA patients. (2): The primary research aim was to describe the extent to which graded exercise testing may reveal abnormalities in hemodynamic function in obstructive sleep apnea (OSA) patients, particularly with respect to cardiac output (Qc), mean arterial pressure (MAP), and TPR that may be related to polysomnography (PSG) markers of OSA severity. Cardiorespiratory and hemodynamic responses that were evaluated included the following: peak oxygen consumption (VO2pk), end-tidal carbon dioxide production (PETCO2), end-tidal oxygen pressure (PETO2), heart rate (HR), blood pressure (systolic = SBP and diastolic = DBP), rate pressure product (RPP), TPR and its derivatives including MAP and Qc, in OSA patients. A global biochemical marker of vascular function, 24-hour urinary nitrite/ nitrate elimination was also determined for each patient. (3): The last aim was included in order to provide qualitative information concerning treatment, subjective sleep and daytime function, and physical activity levels of the OSA patients in this investigation as well as to give insights into the special challenges and potential for doing trials involving nCPAP and physical exercise training with OSA patients. Results from this study can be used to improve clinical evaluation procedures as well as to better understand underlying mechanisms relative to the link between cardiovascular disease and OSA
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Alessandria, Maria <1979&gt. "Sleep motor activity in parkinsonian syndromes at onset: a prospective study to determine potential diagnostic and prognostic markers." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2014. http://amsdottorato.unibo.it/6657/.

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Aim of this study is to describe the possible diagnostic value of sleep disturbances in the differential diagnosis of neurodegenerative diseases characterized by parkinsonism at onset. 42 consecutive patients with parkinsonian features and disease duration up to 3 years were included in the BO-ProPark study. Each patient was evaluated twice, at baseline (T0) and 16 months later (T1). Patients were diagnosed as Parkinson disease (PD, 27 patients), PD plus (PD with cognitive impairment/dementia or dysautonomia, 4 patients) and parkinsonian syndrome (PS, 11 patients). All patients underwent a full night video-polysomnography scored by a neurologist blinded to the clinical diagnosis. Sleep efficiency and total sleep time were reduced in all patients; wake after sleep onset was higher in patients with atypical parkinsonisms than in PD patients. No significant differences between groups of patients were detected in other sleep parameters. The mean percentage of epochs with enhanced tonic muscle EMG activity during REM sleep was higher in PD plus and PS than in PD. No difference in phasic muscle EMG activity during REM sleep was seen between the two groups. REM behaviour disorder was more frequent in PD plus and PS than in PD patients. Our data suggest that REM sleep motor control is more frequently impaired at disease onset in patients with PS and PD plus compared to PD patients. The presence of RBD or an enhanced tonic muscle EMG activity in a patient with recent onset parkinsonian features should suggest a diagnosis of atypical parkinsonism, rather than PD. More data are needed to establish the diagnostic value of these features in the differential diagnosis of parkinsonisms. The evaluation of sleep disorders may be a useful tool in the differential diagnosis of parkinsonism at onset.
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Weidenauer, Corina [Verfasser], and Christoph [Akademischer Betreuer] Randler. "Circadian Preference and Amplitude - “Under Consideration of Physiological Markers, Activity and Sleep/Wake Timing as well as References to Attention, Mood and Motivation in Everyday School Life” / Corina Weidenauer ; Akademischer Betreuer: Christoph Randler." Tübingen : Universitätsbibliothek Tübingen, 2020. http://d-nb.info/1203726201/34.

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Pittaras, Elsa. "Marqueurs comportementaux et neurochimiques individuels de la prise de décision chez la souris et effets d'une dette de sommeil." Thesis, Université Paris-Saclay (ComUE), 2016. http://www.theses.fr/2016SACLS122/document.

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La prise de décision est un processus adaptatif essentiel dont l’efficacité dépend de processus exécutifs, motivationnels, émotionnels et donc de l’intégrité de différents circuits cérébraux. Au sein d’une population saine, il existe des variabilités individuelles décisionnelles influencées par des facteurs génétiques, épigénétiques et environnementaux. De plus, de nombreuses pathologies mentales, neurobiologiques et neurodégénératives provoquent des altérations des processus décisionnels. Ainsi, déterminer des traits comportementaux et des substrats neurobiochimiques impliqués dans ce dysfonctionnement représente un intérêt majeur.Nous avons développé, chez la souris, un test de prise de décision, basé sur le test classiquement utilisé chez l’homme (l’Iowa Gambling Task), qui reproduit une situation incertaine, complexe et conflictuelle de choix : le Mouse Gambling Task (MGT). Grâce à une approche différentielle du comportement, nous avons observé des différences spontanées de capacités décisionnelles : certaines souris ont un comportement rigide et évitent toute pénalité (souris safe), d’autres ont un comportement exploratoire quitte à prendre des risques (souris risky), et une majorité des souris a un comportement intermédiaire (souris average). Nous avons ensuite révélé que les souris safe ont un comportement plus anxieux, une activation préfrontale plus faible que les autres groupes à l’issu du MGT, et un taux de sérotonine à l’état basal plus faible au niveau du cortex préfrontal. Les souris risky ont un comportement plus risqué dans plusieurs tests comportementaux et sont moins sensibles à la récompense. De plus, elles présentent un faible taux de sérotonine au niveau du cortex orbitofrontal ainsi qu’un taux de dopamine, noradrénaline et sérotonine plus important au niveau hippocampique.Afin de tester l’effet d’une modification de l’environnement sur les profils décisionnels caractérisés précédemment, nous avons réalisé le MGT sur un groupe de souris soumises soit à une dette aiguë de sommeil (DAS) soit à une dette chronique de sommeil (DCS). Nous avons alors montré qu’une DCS n’a pas d’effet sur les profils décisionnels mais qu’une DAS accentue ces profils décisionnels: les animaux safe deviennent d’autant plus rigides et évitent encore d’avantage les pénalités alors que les animaux risky choisissent systématiquement les options plus risquées, en adoptant un comportement rigide. Ces observations comportementales peuvent s’expliquer par un métabolisme sérotoninergique diminué au niveau du cortex orbitofrontal et augmenté au niveau hippocampique, ainsi que par un taux élevé de dopamine au niveau du striatum dorsal, structure cérébrale clé des processus d’automatisation.Par conséquent, le MGT permet de révéler, chez des souris consanguines saines, les caractéristiques comportementales et neurobiologiques individuelles de stratégies décisionnelles inadaptées pouvant être amplifiées par un stress environnemental. Ce modèle permettra, notamment, de déterminer les facteurs de vulnérabilité au développement de certaines psychopathologies (l’addiction et la dépression, par exemple) dont le manque de sommeil pourrait être un déclencheur ou un amplificateur
Affective abilities that rely on the integrity of several neural circuits. In healthy subjects, inter-individual variability during decision-making exists due to genetic, epigenetic and environmental factors. Moreover, many psychiatric and neurobiological disorders are characterized by poor decision-making processes. Therefore, determining behavioral traits and neurobiological substrates involved in these processes is of major interest to unravel markers that could predict the emergence of neuropathologies.Based on the Iowa Gambling Task in humans, we developed a decision-making task in mice that assesses their ability to choose between several conflicting options under uncertainty. Thanks to a differential approach of mice’s behavior, we show that decision-making skills differed between mice: some mice exhibit a rigid behavior and avoid penalty (safe mice); others maintained exploratory behavior even if they took risks (risky mice); a majority of mice exhibit an intermediate behavior (average mice). We found that a combination of behavioral characteristics related to different psychopathologies in humans were specifically associated with extreme behavior in mice: safe mice exhibited a more anxious behavior, a lower prefrontal activation after the MGT than others subgroups of performance together with a lower basal rate of serotonin in the prefrontal cortex. Risky mice displayed a riskier behavior in various behavioral tasks, were less sensitive to reward, and had a lower basal rate of serotonin in the orbitofrontal cortex as well as a higher basal rate of serotonin, dopamine and noradrenaline in the hippocampus.To investigate the consequences of environmental changes on decision-making individual profiles, we performed the MGT on groups of mice either under Acute Sleep Dept (ASD) or under chronic sleep debt (CSD). We show that CSD didn't play any apparent effect but that ASD emphasized decision-making profiles: safe mice became drastically more rigid and avoided penalty; and risky mice chose systematically riskier options and developed rigid and unefficient decisions. These behavioral data could be explained by a decreased serotonin metabolism in the orbitofrontal cortex, an increase in the hippocampus and a high level of dopamine in the caudate putamen, the key brain area of habits.Therefore, in healthy inbred mice the MGT reveals individual inadapted decision-making strategies which are characterized by behavioral and neurobiological substrates exacerbated by an environmental stress. This paradigm also allows the determination of mice vulnerability to develop psychopathologies (e.g. depression, addiction) for which sleep debt could a trigger or a magnifier
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Franceschini, Christian <1980&gt. "REM sleep behavior disorder nella narcolessia: ricerca di un marker clinico e strumentale." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2009. http://amsdottorato.unibo.it/1839/.

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Kobzová, Lucie. "Návrh na zlepšení marketingového řízení firmy Red bull." Master's thesis, Vysoké učení technické v Brně. Fakulta podnikatelská, 2008. http://www.nusl.cz/ntk/nusl-376774.

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My master’s thesis from marketing area is focused on marketing management of Red Bull Company, which is world leading energy drink producer. My work should improve marketing management of the firm and suggest new ways of marketing communication, the way of addressing consumers and non-consumers of Red Bull energy drink. The results of this master’s thesis will be suggested to the management of Red Bull Company for implementation into praxis.
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Bat-Pitault, Flora. "Marqueurs sommeil et émotionnels du risque de dépression chez les mères et leurs enfants." Thesis, Aix-Marseille, 2015. http://www.theses.fr/2015AIXM5063/document.

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L’objectif de ce travail était de rechercher des marqueurs sommeil et émotionnels du risque de dépression chez les mères et leurs enfants dits « à risque ». Après une description clinique des liens étroits entre sommeil, dépression, émotion et cognition chez les mères durant la grossesse et le post-partum ainsi que chez les enfants et les adolescents, nous présentons une première étude rétrospective du sommeil sur des enfants et adolescents nés de mères déprimées. Elle met en évidence des particularités macro-architecturales du sommeil en lien avec le risque dépressif chez les adolescents à risque. Nous présentons ensuite une étude prospective sur 302 dyades mère-enfant suivies de la naissance aux 36 mois de l’enfant. Notre but a été de décrire d’abord chez les mères, des anomalies du sommeil pendant la grossesse susceptibles d’indiquer un risque de dépression du post-partum et d’avoir un impact sur le développement de l’enfant. Cette large cohorte nous a ensuite permis d’étudier le lien entre altérations précoces du sommeil des enfants et particularités cognitives et émotionnelles à 36 mois. Nous avons également pu décrire chez les enfants à risque, âgés de 6 mois, des altérations macro et micro-architecturales du sommeil pouvant indiquer un risque de psychopathologie ultérieure via une altération de la neuroplasticité tôt au cours du développement ; et chez ces mêmes enfants à 36 mois, un biais négatif de reconnaissance émotionnelle, potentiel facteur de vulnérabilité de psychopathologie ultérieure, notamment dépressive. Le suivi de cette cohorte reste déterminant pour vérifier parmi les enfants à risque ceux qui développeront finalement un épisode dépressif majeur
The main objective of this work was to search for markers in sleep and emotions level of risk of developing major depression in mothers and their children known as "at risk”. After a clinical description of close links between sleep, depression, emotion and cognition in mothers during pregnancy and the postpartum period and in children and adolescents, we conducted a retrospective study of the first children and adolescents sleep mothers with a personal history of depression. This study highlighted the macro-level architectural features related to depressive identifiable risk adolescents at risk. We then conducted a broader prospective study which involved 302 mother-child dyads followed from birth to 36 months of the child. Initially, our goal was to describe in mothers sleep abnormalities during pregnancy can indicate a risk of postpartum depression and more broadly to induce a number of consequences on the development of the child. Secondly this large cohort allowed us to link early alterations of child sleep with cognitive and emotional peculiarities to 36 months. We have also been able to describe in children 6 months to risk of depression, macro and micro-architectural deterioration of sleep may constitute a subsequent psychopathology risk factor via impaired neuroplasticity early in development; and in these same children 36 months through a negative emotional recognition constitutes a subsequent psychopathology vulnerability factor particularly depressed. The monitoring of this longer-term cohort remains crucial to observe children at risk among those who develop other sleep or emotional anomalies and ultimately a major depressive episode
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Books on the topic "Sleep markers"

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Busby, Thomas L., and Patsy Busby Dow, eds. The Markets Never Sleep. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2012. http://dx.doi.org/10.1002/9781119197331.

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Busby, Thomas L. The Markets Never Sleep. New York: John Wiley & Sons, Ltd., 2007.

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Insight, LLC Medtech. U.S. markets for blood gas/electrolyte monitoring, pulmonary function assessment, and sleep apnea management products. Newport Beach, CA: Medtech Insight, 2005.

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Wong, Maria M. Sleep Problems During the Preschool Years and Beyond as a Marker of Risk and Resilience in Substance Use. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676001.003.0008.

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Individuals with alcohol problems have well-described disturbances of sleep, but the development of these disturbances both before and after the onset of problem drinking is poorly understood. This chapter first discusses sleep physiology and its measurement in humans. It then examines the functions of sleep and its role in development. Next, it reviews recent research on the relationship between sleep and alcohol use and related problems. Whereas sleep problems (e.g., difficulties falling or staying asleep) increase the risk of early onset of alcohol use and related problems, sleep rhythmicity promotes resilience to the development of alcohol use disorder and problem substance use. Based on existing research, this chapter proposes a theoretical model of sleep and alcohol use, highlighting the role of self-regulatory processes as mediators of this relationship.
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Dow, Patsy Busby, and Thomas L. Busby. Markets Never Sleep: Global Insights for More Consistent Trading. Wiley & Sons, Incorporated, John, 2010.

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Dow, Patsy Busby, and Thomas L. Busby. Markets Never Sleep: Global Insights for More Consistent Trading. Wiley & Sons, Incorporated, John, 2007.

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Dow, Patsy Busby, and Thomas L. Busby. Markets Never Sleep: Global Insights for More Consistent Trading. Wiley & Sons, Limited, John, 2015.

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The Markets Never Sleep: Global Insights for More Consistent Trading (Wiley Trading). Wiley, 2007.

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The End of Illness. New York, London, Toronto, Sydney, New Delhi: Free Press, 2012.

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Agus, David B. End of Illness. Free Press, 2016.

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Book chapters on the topic "Sleep markers"

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Cofta, S., H. M. Winiarska, A. Płóciniczak, L. Bielawska, A. Brożek, T. Piorunek, T. M. Kostrzewska, and E. Wysocka. "Oxidative Stress Markers and Severity of Obstructive Sleep Apnea." In Advances in Experimental Medicine and Biology, 27–35. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/5584_2019_433.

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Kupfer, D. J., E. Frank, and C. L. Ehlers. "Sleep-Physiological Characteristics as Potential Biological Markers in Affective Disorders." In Neuropsychopharmacology, 196–207. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-74034-3_20.

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Kosacka, M., A. Brzecka, P. Piesiak, A. Korzeniewska, and R. Jankowska. "Soluble Ligand CD40 and Uric Acid as Markers of Atheromatosis in Patients with Obstructive Sleep Apnea." In Advances in Experimental Medicine and Biology, 55–60. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/5584_2014_44.

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Langford, Alexandra, Alana Brekelmans, and Geoffrey Lawrence. "‘I want to sleep at night as well’." In Markets in their Place, 122–40. London: Routledge, 2021. http://dx.doi.org/10.4324/9780429296260-7.

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Korn, Ralf, and Bernd Luderer. "Sleep Well Despite Turbulent Markets? The Immunization Property of the Duration." In Money and Mathematics, 125–27. Wiesbaden: Springer Fachmedien Wiesbaden, 2021. http://dx.doi.org/10.1007/978-3-658-34677-5_33.

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Jones, D., S. Kelwala, S. Dube, E. Jackson, and N. Sitaram. "Cholinergic Rem Sleep Induction Response as a Marker of Endogenous Depression." In Advances in Behavioral Biology, 375–83. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5194-8_35.

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Jauhiainen, Jussi S., and Miriam Tedeschi. "Undocumented Migrants’ Everyday Lives in Finland." In IMISCOE Research Series, 93–130. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-68414-3_4.

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AbstractThe everyday lives of undocumented migrants are littered with challenges, such as constantly having to find new places in which to live and sleep; seeking employment and a livelihood, even in the grey market; contacting families and friends, to overcome feelings of loneliness and despair; maintaining hope for the future, despite living in a country that is rejecting them; and escaping the police. Living without legal permission in a country makes them wear a variety of identities and masks, and continually devise new survival strategies and practices in order to survive and make ends meet.The chapter illustrates how undocumented migrants in Finland manage to find more or less secure accommodation, and how some of them even find jobs despite the law forbidding them to work in Finland. The chapter also explores in detail their everyday social lives, who they turn to when they need something, and their aspirations and hopes for the future. We also pay attention to their migrations to Finland, within Finland, and their potential on-migration from Finland, including return migration.
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Krueger, James M., Ferenc Obál Jr., Joseph W. Harding, John W. Wright, and Lynn Churchill. "Sleep Modulation of the Expression of Plasticity Markers." In Sleep and Brain Plasticity, 363–76. Oxford University Press, 2003. http://dx.doi.org/10.1093/acprof:oso/9780198574002.003.0018.

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Luyster, PhD, Faith S., Lynn M. Baniak, Eileen R. Chasens, Christine A. Feeley, Christopher C. Imes, and Jonna L. Morris. "Sleep Among Working Adults." In The Social Epidemiology of Sleep, 119–38. Oxford University Press, 2019. http://dx.doi.org/10.1093/oso/9780190930448.003.0005.

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This chapter discusses sleep among working-age adults (defined as roughly between 18 and 65 years old). Five markers of adulthood include (a) living alone or owning a home; (b) finding a long-term partner and living together; (c) working in a full-time, permanent job, which can involve different work environments, commuting, and shift work, and possible unemployment; (d) having children; and (d) saving for the future. This chapter discusses these markers as they relate to sleep, including changes in sleep behaviors across the lifespan in association with employment as well as shift work and the impact of cosleeping with a partner.
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Dadisman, Amy K., Noah D. Andrews, Reena Mehra, and Irene L. Katzan. "Sooner Than Later If Not Right After Stroke." In Sleep Disorders, 927–50. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190671099.003.0055.

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The case involves a patient with interrelated sleep symptoms related to central hypersomnia, restless legs syndrome, and sleep disordered breathing (SDB) occurring after stroke. The prevalence of SDB after stroke has been noted to be very high, up to 70%, and consists of a higher contribution of central sleep apnea compared to the general population. Recurrent stroke is associated with a higher prevalence of SDB compared to first-time stroke. Contributions to SDB as related to stroke do not appear to have consistencies in terms of location and size of stroke. Untreated SDB can also contribute to stroke risk, with mechanisms involving increased systemic inflammation, prothrombotic biochemical markers, and dysfunction of cerebral autoregulation. Although there are post-hoc subgroup data in a randomized controlled trial suggesting reduction in stroke outcomes when SDB is treated with positive airway pressure, overall there are few studies that have rigorously examined the impact of treatment. Hypersomnia can be treated with modafinil and restless legs syndrome may become manifest due to sleep fragmentation.
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Conference papers on the topic "Sleep markers"

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Chami, Hassan, Emelia Benjamin, John Keaney, João Fontes, Vasan Ramachandran, Martin Larson, George T. O'Connor, and Daniel Gottlieb. "Markers Of Vascular Inflammation In Sleep-Disordered Breathing." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2477.

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Guzeev, Mikhail, Valentina Simonova, Nikita Kurmazov, and Yur Pastukhov. "EEG MARKERS OF CHRONIC SLEEP RESTRICTION IN RATS." In XVI International interdisciplinary congress "Neuroscience for Medicine and Psychology". LLC MAKS Press, 2020. http://dx.doi.org/10.29003/m1014.sudak.ns2020-16/169-170.

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Deviaene, Margot, Pascal Borzée, Bertien Buyse, Dries Testelmans, Sabine Van Huffel, and Carolina Varon. "Pulse Oximetry Markers for Cardiovascular Disease in Sleep Apnea." In 2019 Computing in Cardiology Conference. Computing in Cardiology, 2019. http://dx.doi.org/10.22489/cinc.2019.205.

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Borker, P. V., B. Macatangay, A. Morris, and S. R. Patel. "Sleep Apnea and Inflammatory Markers in Chronic HIV Infection." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a4688.

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Fox, Nurit, Alan Mulgrew, John Hill, C. Frank Ryan, John A. Fleetham, and Najib Ayas. "Oxidative Stress Markers In Patients With Obstructive Sleep Apnea (OSA)." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a3710.

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Maierean, A. D., D. M. Vulturar, and D. A. Todea. "Inflammation and oxidative stress markers in obstructive sleep apnea patients." In ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.3408.

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Nosetti, Luana M., Valentina Milan, Michela Gaiazzi, Ramona C. Di Maio, Franca Marino, Marco Cosentino, and Luigi Nespoli. "Circadian Rhythms Of Inflammatory Markers In Pediatric Obstructive Sleep Apnea Syndrome." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a5325.

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Martínez García, Miguel Ángel, Antonio Martorell, Eduardo Nagore, Jose Luis Rodriguez-Peralto, Manuel Sanchez de la torre, Luis Hernandez, Maria Josefa Diaz-Atauri, et al. "Sleep-disordered breathing and aggressiveness markers of cutaneous melanoma. A multicentric study." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa342.

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Loh, Vanesa, Yan Yi Cheung, Siew Pang Chan, See Meng Khoo, and Chi-Hang Lee. "Obstructive sleep apnea and cardiovascular risk markers in community individuals- Results from the sleep apnea and vascular evaluation (SAVE) study." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa2080.

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Bouloukaki, Izolde, Charalampos Mermigkis, Stylianos Michelakis, Eleni Mauroudi, Violeta Moniaki, Sophia Schiza, and Nikolaos Tzanakis. "Evaluation of inflammatory markers in a large sample of Obstructive Sleep Apnea Patients." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa2319.

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Reports on the topic "Sleep markers"

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Bunn, Sarah, and Lev Tankelevitch. Sleep and Health. Parliamentary Office of Science and Technology, September 2018. http://dx.doi.org/10.58248/pn585.

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A POSTnote that explains what is known about sleep and sleep disorders, the effects of poor sleep on performance, and on physical and mental health. It also describes the role of sleep in the context of public and occupational health, road safety, education and the consumer technology market.
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