Relevant bibliographies by topics / Sleep disordered breathing (SDB)

Academic literature on the topic 'Sleep disordered breathing (SDB)'

Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Sleep disordered breathing (SDB)"

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Izci Balserak, Bilgay. "Sleep disordered breathing in pregnancy." Breathe 11, no. 4 (December 2015): 268–77. http://dx.doi.org/10.1183/20734735.009215.

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Key pointsSleep disordered breathing (SDB) is common and the severity increases as pregnancy progresses.Frequent snoring, older age and high pre-pregnancy body mass index (>25 kg⋅m−2) could be reliable indicators for SDB in early pregnancy.SDB screening tools, including questionnaires, used in the nonpregnant population have poor predictive ability in pregnancy.Accumulating evidence suggests that SDB during pregnancy may be associated with increased risk of adverse pregnancy outcomes, including gestational diabetes and pre-eclampsia. However, the results should be interpreted cautiously because several studies failed to adjust for potential maternal confounders and have other study limitations.There are no pregnancy-specific practice guidelines for SDB treatment. Many clinicians and practices follow recommendations for the treatment in the general population. Women with pre-existing SDB might need to be reassessed, particularly after the sixth month of pregnancy, because symptoms can worsen with nasal congestion and weight gain.Educational aimsTo highlight the prevalence and severity of sleep disordered breathing (SDB) in the pregnant population.To inform readers about risk factors for SDB in pregnancy.To explore the impact of SDB on adverse maternal and fetal outcomes, and biological pathways for associated adverse maternal and fetal outcomes.To introduce current management options for SDB in pregnancy, including medical and behavioural approaches.Sleep disordered breathing (SDB) is very common during pregnancy, and is most likely explained by hormonal, physiological and physical changes. Maternal obesity, one of the major risk factors for SDB, together with physiological changes in pregnancy may predispose women to develop SDB. SDB has been associated with poor maternal and fetal outcomes. Thus, early identification, diagnosis and treatment of SDB are important in pregnancy. This article reviews the pregnancy-related changes affecting the severity of SDB, the epidemiology and the risk factors of SDB in pregnancy, the association of SDB with adverse pregnancy outcomes, and screening and management options specific for this population.
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Yang, Yeonmi. "Sleep Disordered Breathing in Children." JOURNAL OF THE KOREAN ACADEMY OF PEDTATRIC DENTISTRY 49, no. 4 (November 30, 2022): 357–67. http://dx.doi.org/10.5933/jkapd.2022.49.4.357.

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Sleep disordered breathing (SDB) is a disease characterized by repeated hypopnea and apnea during sleep due to complete or partial obstruction of upper airway. The prevalence of pediatric SDB is approximately 12 - 15%, and the most common age group is preschool children aged 3 - 5 years. Children show more varied presentations, from snoring and frequent arousals to enuresis and hyperactivity. The main cause of pediatric SDB is obstruction of the upper airway related to enlarged tonsils and adenoids. If SDB is left untreated, it can cause complications such as learning difficulties, cognitive impairment, behavioral problems, cardiovascular disease, metabolic syndrome, and poor growth. Pediatric dentists are in a special position to identify children at risk for SDB. Pediatric dentists recognize clinical features related to SDB, and they should screen for SDB by using the pediatric sleep questionnaire (PSQ), lateral cephalometry radiograph, and portable sleep monitoring test and refer to sleep specialists. As a therapeutic approach, maxillary arch expansion treatment, mandible advancement device, and lingual frenectomy can be performed. Pediatric dentists should recognize that prolonged mouth breathing, lower tongue posture, and ankyloglossia can cause abnormal facial skeletal growth patterns and sleep problems. Pediatric dentists should be able to prevent these problems through early intervention.
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Gokdemir, Yasemin, and Refika Ersu. "Sleep disordered breathing in childhood." European Respiratory Review 25, no. 139 (February 29, 2016): 48–53. http://dx.doi.org/10.1183/16000617.0081-2015.

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Sleep disordered breathing (SDB) in childhood is linked with significant end-organ dysfunction across various systems, particularly with cardiovascular, neurocognitive and metabolic consequences. If we understand the pathophysiology of SDB, diagnose it promptly and treat appropriately, we may be able to prevent morbidity associated with SDB and also save health resources around the world. In this article, we highlight articles on this topic published in medical journals in the past year.
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Young, Terry, Paul E. Peppard, and Shahrad Taheri. "Excess weight and sleep-disordered breathing." Journal of Applied Physiology 99, no. 4 (October 2005): 1592–99. http://dx.doi.org/10.1152/japplphysiol.00587.2005.

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Excess weight is a well-established predictor of sleep-disordered breathing (SDB). Clinical observations and population studies throughout the United States, Europe, Asia, and Australia have consistently shown a graded increase in the prevalence of SDB as body mass index, neck girth, or other measures of body habitus increases. Clinical studies of weight loss and longitudinal population studies provide strong support for a causal association. The role of excess body weight, a modifiable risk factor, with SDB raises many questions relevant to clinical practice and public health. The topic takes on added importance with the alarming rate of weight gain in children as well as adults in industrialized nations. Among adults ages 30–69 yr, averaging over the estimated United States 2003 age, sex, and BMI distributions, we estimate that ∼17% of adults have mild or worse SDB (apnea-hypopnea index ≥ 5) and that 41% of those adults have SDB “attributable” to having a body mass index of ≥25 kg/m2. Similarly, we estimate that ∼5.7% of adults have moderate or worse SDB (apnea-hypopnea index ≥ 15) and that 58% of those adults have SDB attributable to excess weight. Clearly, if the expanding epidemic of obesity seen in the United States continues, the prevalence of SDB will almost certainly increase, along with the proportion of SDB attributable to obesity.
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McMillan, Alison, and Mary J. Morrell. "Sleep disordered breathing at the extremes of age: the elderly." Breathe 12, no. 1 (March 2016): 50–60. http://dx.doi.org/10.1183/20734735.003216.

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Key pointsSleep disordered breathing (SDB) is common and its prevalence increases with age. Despite this high prevalence, SDB is frequently unrecognised and undiagnosed in older people.There is accumulating evidence that SDB in older people is associated with worsening cardio- cerebrovascular, cognitive and functional outcomes.There is now good evidence to support the use of continuous positive airway pressure therapy in older patients with symptomatic SDB.Educational aimsTo highlight the prevalence and presentation of sleep disordered breathing (SDB) in older people.To inform readers about the risk factors for SDB in older people.To explore the impact of SDB in older people.To introduce current evidence based treatment options for SDB in older people.Sleep disordered breathing (SBD) increases in prevalence as we age, most likely due to physiological and physical changes that occur with ageing. Additionally, SDB is associated with comorbidity and its subsequent polypharmacy, which may increase with increasing age. Finally, the increased prevalence of SDB is intrinsically linked to the obesity epidemic. SDB is associated with serious outcomes in younger people and, likewise, older people. Thus, identification, diagnosis and treatment of SDB is important irrelevant of age. This article reviews the age-related changes contributing to SDB, the epidemiology and the risk factors for SDB in older people, the association of SDB with adverse outcomes, and diagnostic and treatment options for this population.
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Carrijo, Fanny Cavalcante, Winicius Arildo Ferreira Araujo, Iorrana Morais de Oliveira, Crystina Alcântara Carvalho, Marcelo Costa Rodrigues, Maria Tereza de Oliveira e. Souza, Glauco Issamu Miyahara, and Grace Kelly Martins Carneiro. "Prevalence of sleep-disordered breathing related to malocclusion in children." Research, Society and Development 10, no. 16 (December 19, 2021): e598101623984. http://dx.doi.org/10.33448/rsd-v10i16.23984.

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Sleep-disordered breathing is characterized by airway dysfunction that can occur in any age, but most prevalent in children, caused by the occurrence of respiratory effort, snoring or even by apnea during sleep. Therefore, the aim of this study was to survey the prevalence of sleep disorders associated with malocclusion in children aged 3 to 12 years in Mineiros, State of Goiás, Brazil. Material and Methods: this is a field research with a sample of 99 children affected by some type of sleep-disordered breathing and malocclusions. Data were collected through a questionnaire about sleep-disordered breathing and a clinical record carried out through intraoral clinical examination. Results: among the 24 children with SDB, 17 had SDB and Malocclusion, which is 70.8% of the children had SDB associated with malocclusion. Of the 75 children without SDB, 11 (14.7%) had malocclusion. Conclusion: No significant differences were found between sleep-disordered breathing and sex-related malocclusions.
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Clark, Christine M., Dale S. DiSalvo, Jansie Prozesky, and Michele M. Carr. "Sleep Disordered Breathing May Signal Laryngomalacia." Open Anesthesiology Journal 11, no. 1 (August 21, 2017): 68–74. http://dx.doi.org/10.2174/1874321801711010068.

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Background: Pediatric anesthesiologists are often confronted with children with sleep-disordered breathing (SDB) presenting for tonsillectomy and/or anesthesia. The typical patient has symptoms of obstructive sleep apnea (OSA) with enlarged tonsils; however, a subset of patients may have underlying laryngomalacia (LM) without tonsillar hypertrophy. Both OSA and LM significantly increase the risk of intra- and postoperative airway obstruction and sensitivity to narcotics. The prevalence of LM may be underestimated, because direct laryngoscopy (DL) is not routinely performed in the diagnostic evaluation of patients with SDB who lack tonsillar hypertrophy. Aim: To identify the prevalence and DL findings in pediatric patients with SDB without tonsillar hypertrophy. Methods: Retrospective chart review of 108 patients with SDB who underwent general anesthesia for adenotonsillectomy (TA) or adenoidectomy with concomitant DL. The following data were collected: demographic information, medical comorbidities, polysomnography results, anesthetic techniques, and postoperative complications. Results: 94.5% of children had DL findings consistent with LM, including a retropositioned epiglottis and short aryepiglottic folds. Postglottic edema was observed in 42.2%, and these patients were significantly more likely to have a diagnosis of gastroesophageal reflux (P=0.023). 57.8% had vocal cord edema. 75.3% of children who received routine postoperative follow-up care experienced complete symptom resolution. Postoperative complications following discharge from hospital occurred in 12.4% of patients, and 15.7% underwent supraglottoplasty for continued SDB symptoms after TA or adenoidectomy. Conclusion: A substantial proportion of patients with SDB who lacked tonsillar hypertrophy had findings consistent with LM, suggesting that the larynx may be the primary site of upper airway obstruction in these patients. This has significant implications in terms of perioperative management. The majority of patients with SDB had symptomatic improvement following TA or adenoidectomy; however, a subset required further surgical intervention with supraglottoplasty.
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Abazi, Yllka, Fabian Cenko, Marianna Cardella, Gjergji Tafa, and Giuseppina Laganà. "Sleep Disordered Breathing: An Epidemiological Study among Albanian Children and Adolescents." International Journal of Environmental Research and Public Health 17, no. 22 (November 19, 2020): 8586. http://dx.doi.org/10.3390/ijerph17228586.

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Sleep Disordered Breathing (SDB) comprises a group of diseases characterized by alterations in the frequency and/or depth of breathing during sleep. The aim of this study was to investigate the frequency of SDB in a group of Albanian children and adolescents and to describe its social, physiological, psychological, sleep-related, and anthropometric risk factors, in relation to the sociodemographic situation. A total of 6087 participants (mean age: 10.42 years, range: 6 to 15 years, 52.3% females and 47.7% males) attending public schools all over Albania took part in the cross-sectional study. On a sample of 6087 questionnaires distributed, 4702 (77.25% of the original sample) were returned and included in the study. High risk status for SDB was assessed using the Paediatric Sleep Questionnaire (PSQ). The prevalence of SDB was 7.9%. No statistically significant difference was found for gender at high risk for SBD. Compared to participants living in urban aeras (7.3%), participants living in rural areas (10.4%) reported significantly higher SDB prevalence rates. No other significant correlations were detected between the high-risk subjects and the age. The prevalence of the subjects at high risk of SBD obese participants (20.8%) was statistically higher than among nonobese ones (6.3%). SDB is highly prevalent in Albanian growing population and further prevalence studies are recommended.
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Mosquera, Ricardo A., Mary Kay Koenig, Rahmat B. Adejumo, Justyna Chevallier, S. Shahrukh Hashmi, Sarah E. Mitchell, Susan E. Pacheco, and Cindy Jon. "Sleep Disordered Breathing in Children with Mitochondrial Disease." Pulmonary Medicine 2014 (2014): 1–8. http://dx.doi.org/10.1155/2014/467576.

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A retrospective chart review study was performed to determine the presence of sleep disordered breathing (SDB) in children with primary mitochondrial disease (MD). The symptoms, sleep-related breathing, and movement abnormalities are described for 18 subjects (ages 1.5 to 18 years, 61% male) with MD who underwent polysomnography in our pediatric sleep center from 2007 to 2012. Of the 18 subjects with MD, the common indications for polysomnography were excessive somnolence or fatigue (61%,N= 11), snoring (44%,N= 8), and sleep movement complaints (17%,N= 3). Polysomnographic measurements showed SDB in 56% (N= 10) (obstructive sleep apnea in 60% (N= 6), hypoxemia in 40% (N= 4), and sleep hypoventilation in 20% (N= 2)). There was a significant association between decreased muscle tone and SDB (P: 0.043) as well as obese and overweight status with SDB (P=0.036). SDB is common in subjects with MD. Early detection of SDB, utilizing polysomnography, should be considered to assist in identification of MD patients who may benefit from sleep-related interventions.
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Garde, Alison J. B., Neil A. Gibson, Martin P. Samuels, and Hazel J. Evans. "Recent advances in paediatric sleep disordered breathing." Breathe 18, no. 3 (September 2022): 220151. http://dx.doi.org/10.1183/20734735.0151-2022.

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This article reviews the latest evidence pertaining to childhood sleep disordered breathing (SDB), which is associated with negative neurobehavioural, cardiovascular and growth outcomes. Polysomnography is the accepted gold standard for diagnosing SDB but is expensive and limited to specialist centres. Simpler tests such as cardiorespiratory polygraphy and pulse oximetry are probably sufficient for diagnosing obstructive sleep apnoea (OSA) in typically developing children, and new data-processing techniques may improve their accuracy. Adenotonsillectomy is the first-line treatment for OSA, with recent evidence showing that intracapsular tonsillectomy results in lower rates of adverse events than traditional techniques. Anti-inflammatory medication and positive airway pressure respiratory support are not always suitable or successful, although weight loss and hypoglossal nerve stimulation may help in select comorbid conditions.Educational aimsTo understand the clinical impact of childhood sleep disordered breathing (SDB).To understand that, while sleep laboratory polysomnography has been the gold standard for diagnosis of SDB, other diagnostic techniques exist with their own benefits and limitations.To recognise that adenotonsillectomy and positive pressure respiratory support are the mainstays of treating childhood SDB, but different approaches may be indicated in certain patient groups.
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Dissertations / Theses on the topic "Sleep disordered breathing (SDB)"

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kuo, Tracy F. "Sleep-disordered breathing (SDB) and neuropsychological function: A community sample study." Diss., The University of Arizona, 2000. http://hdl.handle.net/10150/284215.

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This is a case-control study investigating the relationship of sleep-disordered breathing (SDB) to neuropsychological functioning. Participants were recruited from the Tucson Sleep Heart Health Study (SHHS), which is a population-based study examining cardiovascular consequences of SDB. A sample of 103 men and women, ages 40-75, consisting of 51 controls (CTL group) and 52 persons with SDB (SDB group), matched by age (± 5 years) and sex, was enrolled. CTL subjects had a respiratory disturbance index (RDI), a measure of SDB, ≤ 5 while the SDB subjects had a RDI 20 to 50, inclusive. All participants had an overnight in-home polysomnography (PSG) prior to undergoing a neuropsychological evaluation. Psychological functioning was assessed and the areas of cognitive functioning that were tested included general intelligence, attention and working memory, psycho-visuo-motor efficiency, manual dexterity, and frontal/executive function. The SDB group performed significantly worse on the Stroop Color-Word test, made more errors on the Controlled Oral Word Association test, and overestimated time elapsed. The SDB group also demonstrated a statistical trend ( p ≤ 0.10) for worse performance on Wechsler Adult Intelligence Scale - III Digit Span, Letter-Number Sequencing and Digit Symbol Coding subtests, and the non-dominant hand performance on the Grooved Pegboard. Factor analyses were performed to reduce the number of neuropsychological variables and measures of SDB. Controlling for IQ, multiple regression analyses showed a significant negative association between a "nocturnal hypoxemia" factor and both "manual dexterity" and "semantic memory" factors. The results suggest that persons with moderate SDB, compared to controls, did not report increased depression or other aspects of psychological distress. SDB is, however, associated with subtle neuropsychological decrements in frontal/executive function, psycho-visuo-motor efficiency, and working memory. The performance decrement in tasks of frontal executive function and manual dexterity was primarily associated with nocturnal hypoxemia and not to the frequency of sleep fragmentation.
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Davies, Robert J. O. "Sleep disordered breathing and the cardiovascular system." Thesis, University of Southampton, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.404009.

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Smith, Lindsay Anne. "Sleep-disordered breathing and chronic heart failure." Thesis, University of Edinburgh, 2009. http://hdl.handle.net/1842/29371.

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Methods: Patients with stable symptomatic chronic heart failure were screened for sleep-disordered breathing by home sleep study. Daytime sleepiness was assessed by Epworth Sleepiness Scale and heart failure severity by symptom class, left ventricular ejection fraction and serum N-terminal pro-brain natriuretic peptide concentrations. In a subset of patients, synchronous in-laboratory limited sleep studies and polysomonography, and home limited sleep studies, were performed prospectively. Patients with obstructive sleep apnoea and stable symptomatic chronic heart failure were randomised to nocturnal auto-titrating continuous positive airway pressure or sham for six weeks each in crossover design. Results: In the era of modern therapy, sleep-disordered breathing is common in patients with stable symptomatic chronic heart failure, predominantly obstructive in aetiology, without clear relationship to heart failure severity and is difficult to diagnose because of major overlap in symptomatology. Limited sleep studies compare well diagnostically to polysomnography when tested under identical patient and environmental conditions but less so when tested in the home setting. Auto-titrating continuous positive airway pressure improves daytime sleepiness is patients with obstructive sleep apnoea and chronic heart failure but not other subjective or objective measures of heart failure severity. Conclusions: Sleep-disordered breathing is difficult to detect clinically in patients with chronic heart failure, and as such, the diagnosis is reliant on accurate sleep studies. However, the clinical utility of limited sleep studies in detection and diagnosis of sleep-disordered breathing is restricted by a number of technical and situational factors which are exacerbated in patients with chronic heart failure. The potential therapeutic benefits of continuous positive airway pressure in patients with obstructive sleep apnoea and chronic heart failure are achieved by alleviation of obstructive sleep apnoea rather than by improvement in cardiac function.
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Pirilä-Parkkinen, K. (Kirsi). "Childhood sleep-disordered breathing – dentofacial and pharyngeal characteristics." Doctoral thesis, Oulun yliopisto, 2011. http://urn.fi/urn:isbn:9789514296024.

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Abstract The aim of this work was to examine distinct dentofacial and pharyngeal features in children with sleep-disordered breathing (SDB). A further aim was to test the validity of the conventional methods used in orthodontics for recognition of obstructed airways. Seventy children (36 girls, 34 boys, age range 4.2–11.9 years), who were diagnosed as having SDB during the years 2000–2002, constituted the source of subjects for four separate studies. The study protocol included otorhinolaryngological and orthodontic examinations. Dental impressions and lateral cephalograms were taken. In addition, upper airway was imaged in 36 children in different head postures by magnetic resonance imaging (MRI). Age- and gender-matched healthy children were used as controls. The results showed that children with SDB had increased overjet, smaller overbite, narrower upper and shorter lower dental arches and higher incidence of anterior open bite and distal molar relationship when compared with control children. The severity of the obstruction correlated with larger deviations from normal in the dental characteristics. There were more children with crowding and with anterior open bite with increased severity of the disorder. Children with SDB displayed an increased sagittal jaw relationship, a more vertical growth pattern of the mandible, a longer and thicker soft palate, a lower hyoid bone position, larger craniocervical angles and narrower pharyngeal airway measured at multiple levels, when compared with the controls. Deviation in pharyngeal variables showed highest correlation with the severity of SDB. MRI indicated a smaller than normal oropharyngeal airway in children with SDB. The effect of head posture on pharyngeal airway dimensions differed in children with SDB when compared with the controls. These findings verify that the developmental course of occlusal characteristics and craniofacial structures may be modulated by breathing pattern, and, on the other hand, that certain features may predispose to the development of the disorder. It is important to recognize these signs in order to guide further development in a more favorable direction. The results confirmed that cephalometry is a valid screening tool when assessing nasopharyngeal and retropalatal airway dimensions. Clinical examination of tonsillar size was found reliable when retroglossal airway size was evaluated
Tiivistelmä Väitöskirjatyön tarkoituksena oli selvittää lasten unenaikaisten hengityshäiriöiden yhteyttä kasvojen ja leukojen kasvuun, purennan kehitykseen sekä ylempien hengitysteiden rakenteeseen. Lisäksi tutkittiin, kuinka luotettavasti hammaslääkäreiden käytössä olevin menetelmin pystytään arvioimaan lasten ahtautuneita ilmateitä. Tutkimusaineistossa oli mukana 70 lasta (36 tyttöä, 30 poikaa, ikä 4.2–11.9 vuotta), joilla diagnosoitiin unenaikainen obstruktiivinen hengityshäiriö vuosina 2000–2002. Lapsille tehtiin kliiniset tutkimukset oikojahammaslääkärin ja korva-, nenä- ja kurkkutautien erikoislääkärin toimesta sekä otettiin lateraalikallokuva ja hampaistosta kipsimallijäljennökset. Lisäksi ylempien hengitysteiden magneettitutkimus suoritettiin 36 lapselle eri pään asennoissa. Verrokkiryhmässä oli sama ikä- ja sukupuolijakauma kuin tutkimusryhmässä. Tulokset osoittivat, että unenaikaisista hengityshäiriöistä kärsivillä lapsilla horisontaalinen ylipurenta oli suurentunut, vertikaalinen ylipurenta oli pienentynyt, ylähammaskaari oli kapeampi ja alahammaskaari lyhyempi kuin terveillä verrokeilla. Alaetualueen ahtaus ja etualueen avopurenta olivat yleisempiä niillä lapsilla, joilla oli vaikeampiasteinen hengityshäiriö. Lasten unenaikaiset hengityshäiriöt liittyivät suurentuneeseen leukojen väliseen kokoepäsuhtaan, alaleuan avautuvaan kasvumalliin, pidempään ja paksumpaan pehmeään suulakeen, kieliluun alempaan asentoon, kohonneeseen pään asentoon sekä ahtaampiin nielun ilmatilan mittoihin verrattaessa terveisiin lapsiin. Häiriön vaikeusaste korreloi suurempiin poikkeamiin nielun mitoissa. Lapsilla, joilla todettiin unenaikainen hengityshäiriö, magneettitutkimukset osoittivat nielun olevan ahdas. Pään asennon vaikutukset ilmatien rakenteeseen poikkesivat unenaikaisista hengityshäiriöistä kärsivillä lapsilla. Saadut tulokset osoittivat, että hengitystapa voi vaikuttaa hampaiston, kasvojen ja leukojen rakenteiden kehitykseen. Tietyt kasvojen ja leukojen piirteet puolestaan saattavat altistaa häiriön kehittymiselle. On tärkeää tunnistaa nämä merkit, jotta kehitystä voidaan ohjata suotuisampaan suuntaan. Tutkimukset osoittivat, että kefalometrinen analyysi on luotettava arvioitaessa nenänielun sekä pehmeän suulaen takana olevan ilmatilan kokoa. Nielurisojen kliinisen koon arviointi on luotettava arvioitaessa alemman suunielun ilmatilan kokoa
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Aran, Reza. "Craniofacial morphology and sleep disordered breathing in children." Thesis, University of British Columbia, 2013. http://hdl.handle.net/2429/44379.

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Objective: The aim of this study is to understand how craniofacial morphology and the severity of a malocclusion can contribute to sleep disordered breathing (SDB) symptoms in children when controlled for age, gender and body mass index (BMI). Methods: A total of 301 subjects with complete records were included in this study. Two hundred and thirty-seven were preadolescents, of which 97 were male and 140 were female (mean age 9.9±1.6); 64 were adolescents, of which 24 were male and 40 were female (mean age 13.8±0.9). All the subjects’ parents were asked to complete a SDB questionnaire. Lateral cephalometric images were analyzed to assess the position of the hyoid bone, length of the soft palate, and the maxillary and mandibular dental and skeletal relationship. A clinical examination was performed to determine the Angle classification, Mallampati score, tonsil size (Brodesky), and BMI. Results: Data from 301 children that completed the questionnaires and underwent a cephalometric analysis were evaluated. Subjects were divided into two groups based on their age and each group was further divided based on gender. By comparing preadolescents with adolescents we found that preadolescents presented a significantly higher incidence of hyperactivity, morning headaches, more frequent snoring, and bedwetting. Adolescents exhibited significantly higher daytime sleepiness, difficulty getting up, and impaired daytime function. When comparing female and male subjects, we found that frequent snoring, and morning headaches were more prevalent among females, while daytime sleepiness, and hyperactivity were more common among males. Craniofacial features that have a significant relationship with SDB symptoms are, a lower position of the hyoid bone, retruded mandible, steeper mandibular plane angle, and retroclined lower incisors. There was no statistically significant relationship between Angle classifications, tonsil size, Mallampati score, and BMI with SDB symptoms in this sample. Conclusion: This study suggests that craniofacial morphology, but not severity of malocclusions, could be a potential contributing factor to SDB symptom severity.
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Kaminska, Marta. "Sleep-disordered breathing and fatigue in multiple sclerosis." Thesis, McGill University, 2011. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=103496.

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Fatigue is common and disabling in multiple sclerosis (MS). It is distinct from sleepiness. Both fatigue and sleepiness have been associated with sleep-disordered breathing, particularly obstructive sleep apnea (OSA) in other populations. While fatigue in MS is probably multi-factorial, OSA may play an important role. The main objectives of this project were to evaluate the relationship of OSA to fatigue and sleepiness in MS patients vs. normal controls, and to evaluate predictors of severe fatigue in MS subjects. Stable, ambulatory MS patients without known sleep disorders were recruited from the Montreal Neurological Hospital MS clinic. Normal controls were age and sex frequency-matched. All participants underwent diagnostic overnight polysomnography and a multiple sleep latency test (objective measure of daytime sleepiness). Fatigue was measured with the Fatigue Severity Scale (FSS) and the Multidimensional Fatigue Inventory (MFI), and subjective sleepiness with the Epworth Sleepiness Scale (ESS). Covariates included age, sex, body mass index, MS disability level, depression, pain and restless legs syndrome severity. OSA, defined as an apnea-hypopnea index (AHI) ≥ 15, was found in 36 of 62 (58%) MS subjects and 15 of 32 (47%) control subjects – OR (95% CI) 1.57 (0.67, 3.74). After adjusting for confounders, severe fatigue (FSS > 5) and the MFI-mental fatigue scale (> group median) were associated with AHI and respiratory-related arousals, in MS but not in control subjects. MFI-mental fatigue, in MS subjects, was also associated with intermittent hypoxemia. Subjective and objective sleepiness were not related to OSA in either group. In a multivariate model for MS subjects alone, the significant predictors of severe fatigue were the MS disability level – OR (95% CI) 1.88 (1.18, 3.33) and severe OSA (AHI ≥ 30) – OR (95% CI) 19.55 (2.31, 322.09). In summary, while OSA was not more frequent in MS than in control subjects, it was associated with severe fatigue only in MS subjects. No association was found with sleepiness. Severe fatigue in MS subjects showed a marked association with severe OSA, and a less pronounced association with MS disability level. In that OSA is a treatable condition, these findings open the door to further research that may lead to important changes in management of severely fatigued MS subjects.
La fatigue est fréquente et incapacitante dans la sclérose en plaque (SP). Elle est distincte de la somnolence. La fatigue et la somnolence ont été associées aux troubles respiratoires du sommeil, particulièrement à l'apnée obstructive du sommeil (AOS). La fatigue dans la SP est vraisemblablement multifactorielle, mais l'AOS pourrait y jouer un rôle important. Les principaux objectifs de ce projet étaient d'évaluer le rapport entre l'AOS et la fatigue ainsi que la somnolence chez les patients atteints de SP comparativement au sujets témoins, et d'évaluer les facteurs associés à la fatigue sévère chez les sujets atteints de SP. Des patients atteints de SP stables et ambulatoires sans troubles du sommeil connus ont été recrutés à la clinique de SP de l'Hôpital Neurologique de Montréal. Le groupe de sujets contrôle a été assorti en âge et sexe. Tous les participants ont passés une polysomnographie diagnostique et un test itératif de latence à l'endormissement (mesure objective de la somnolence diurne). La fatigue fut mesurée par la 'Fatigue Severity Scale' (FSS) et le 'Multidimensional Fatigue Inventory' (MFI), et la somnolence subjective avec l'échelle de somnolence Epworth (ESS). Les autres variables étaient l'âge, le sexe, l'indice de masse corporel, le niveau d'incapacité lié à la SP, la dépression, la douleur et la sévérité du syndrome des jambes sans repos. L'AOS, défini par un indice d'apnée-hypopnée (IAH) ≥ 15, a été retrouvée chez 36 des 62 (58%) sujets SP et 5 des 32 (47%) sujets témoins – rapport de cote (RC, IC 95%) 1.57 (0.67, 3.74). Après ajustement pour les facteurs confondants, la fatigue sévère (FSS > 5) et le MFI – fatigue mentale (> médiane du groupe) étaient associés avec l'IAH et les micro-éveils respiratoires, chez les sujets SP mais non chez les sujets contrôle. Le MFI – fatigue mentale, chez les sujets SP, était aussi associé à l'hypoxémie intermittente. Ni la somnolence subjective, ni objective n'étaient associées à l'AOS dans aucun des 2 groupes. Dans un modèle à plusieurs variables pour les sujets SP seulement, les seules variables prédisant la fatigue sévère étaient le niveau d'incapacité – RC (IC 95%) 1.88 (1.18, 3.33) et l'AOS sévère (IAH ≥ 30) – RC (IC 95%) 19.55 (2.31, 322.09). En résumé, quoique l'AOS n'aie pas été plus fréquente dans la SP que chez les sujets témoins, elle était associée à la fatigue sévère chez les sujets SP seulement. Aucune association avec la somnolence n'a été mise en évidence. La fatigue sévère chez les sujets SP était associée de façon marquée à l'AOS sévère, et de façon moins prononcée au niveau d'incapacité. Comme l'AOS est une condition qui se traite, ces résultats ouvrent la porte à des recherches plus poussées qui pourraient ultérieurement changer la prise en charge des patients atteints de SP avec fatigue sévère.
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Johnston, Christopher David. "Sleep-disordered breathing : a cephalometric and clinical study." Thesis, Queen's University Belfast, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.313925.

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Bruyneel, Marie. "Ambulatory diagnostic and monitoring techniques for sleep disordered breathing." Doctoral thesis, Universite Libre de Bruxelles, 2015. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/216824.

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Techniques ambulatoires de diagnostic et de monitoring des troubles respiratoires liés au sommeil.Le syndrome d’apnées obstructives du sommeil (SAOS) est un trouble du sommeil très fréquent, fortement lié à l’obésité, ce qui explique sa prévalence en pleine expansion. En parallèle, la demande d’examens polysomnographiques (PSG) en laboratoire du sommeil, méthode diagnostique de référence, est en croissance. Comme l’accès à cette technique est peu aisé, de nombreux appareils simplifiés d’enregistrement de sommeil ont été récemment développés, mais restent imparfaits (mauvaise évaluation du temps de sommeil, sous-estimation de la sévérité du SAOS, faux négatifs, taux d’échec élevé) et sont d’un apport limité pour le diagnostic du SAOS. La PSG au domicile (PSG-d) est une alternative bien plus informative, permettant d’éviter nombre des désavantages rencontrés par l’usage d’appareils simplifiés. Nous l’avons dès lors étudiée pour le diagnostic du SAOS, au travers d’une étude randomisée comparant la PSG-d vs la PSG hospitalière. En termes d’efficacité diagnostique, les résultats sont excellents, avec un faible taux d’échec d’examens à domicile (4.7 vs 1.5%). Les patients préfèrent être enregistrés dans leur propre environnement où la qualité de leur sommeil est d’ailleurs meilleure. Nous avons ensuite voulu faire le point sur la littérature récente au travers d’un article de revue, en analysant les études prospectives randomisées comparant la PSG-d et au labo du sommeil. Les résultats de ces études concordent pour démontrer que la PSG-d constitue une excellente alternative aux tests réalisés à l’hôpital. Outre le SAOS, l’outil permet le diagnostic d’autres troubles du sommeil, comme les mouvements périodiques des jambes durant le sommeil, les troubles du rythme circadien, Une question restée jusqu’ici sans réponse était l’influence de la localisation du branchement des PSG-d, à l’hôpital ou à domicile. Une étude prospective randomisée nous a permis d’établir que la localisation du branchement des PSG-d n’influençait pas la qualité globale de l’examen, ce qui simplifiera l’utilisation de cet outil à l’avenir. Enfin, nous avons utilisé des techniques de télé monitoring (TM) pour contrôler, en temps réel, la qualité des PSG-d. Dans une première étude pilote, la faisabilité a été confirmée, malgré quelques difficultés techniques. Nous avons voulu appliquer la technique à une population de patients souffrant d’un syndrome coronarien aigu, incapables d’être enregistrés au labo du sommeil. Nous avons étudié la qualité du screening du SAOS par PSG vs polygraphie (PG). Les résultats se sont révélés surprenants :82% de cette population présentait des troubles respiratoires liés au sommeil, principalement centraux. La PSG était nettement plus sensible que la PG, et le TM améliorait la qualité des PSG. Chez les patients traités pour SAOS, nous avons ensuite utilisé un outil de monitoring, l’actigraphie (Act), afin d’observer, dans la vie de tous les jours, les changements de schémas de sommeil et d’activité physique engendrés par la pression positive continue (PPC). Dans un premier travail, rétrospectif, nous avons observé ces paramètres chez des SAOS avant traitement, puis au travers d’une étude prospective multicentrique, nous avons suivi 150 patients avant et après PPC, et observé chez eux une augmentation de temps de sommeil, mais pas de l’activité physique. En conclusion, nous avons démontré dans cette thèse l’intérêt clinique de deux excellents outils ambulatoires, la PSG-d et l’Act, pour la prise en charge du SAOS. Les implications potentielles sont une meilleure accessibilité diagnostique pour le SAOS, une initiation thérapeutique plus précoce et un suivi plus précis des SAOS traités, dans des conditions ambulatoires, plus confortables et plus adéquates pour les patients.
Ambulatory diagnostic and monitoring techniques for sleep disordered breathingSleep disordered breathing (SDB), including obstructive sleep apnea syndrome (OSAS), is directly related to obesity. Significant morbi-mortality is associated with OSAS, explaining the increasing demand for in-hospital polysomnography (PSG), the reference diagnostic method. As this technique is complex and time-consuming, many simplified portable monitoring (PM) devices for home sleep testing have been developed. However, the ability of PM devices to detect OSA remains limited: sleep time is not correctly assessed, OSA severity is underestimated, false negative results occur and the failure rate of the tests is high, up to 30%. Home-PSG (H-PSG) is an interesting alternative, avoiding many of these drawbacks. In the first part of this work, we studied the tool in an original study comparing H-PSG and in-lab PSG. Diagnostic efficacy was good and the failure rate low (4.7 vs 1.5%). Patients slept in their own environment and thus sleep quality was better. We were then interested by reviewing recent literature data regarding prospective randomised trials comparing H-PSG and in-lab PSG. We concluded that H-PSG is an excellent alternative for in-lab PSG, allowing not only OSA detection but also diagnosis of a large panel of other sleep disorders (periodic leg movements during sleep, circadian disorders,). As the best place to perform set-up for H-PSG remained unknown, we studied, in another prospective randomised study, the recording’s quality obtained in both settings. As no difference was observed, lab set up was found to be the simpler option for performing H-PSG. We then tested, in a prospective pilot study, real-time telemonitoring (TM) of H-PSG in order to enhance recording quality. Results were encouraging but we faced some technical problems. In a second study, we applied TM coupled with PSG to detect SDB in acute coronary syndrome, in patients too unstable to come in the sleep lab. We compared also PSG results to polygraphy (PG). Surprisingly, 82% of patients suffered from SDB. PSG was much more sensitive than PG to screen SDB in this population and TM improves recording quality. In the second part of this work, we have used actigraphy (Act) to assess sleep and physical activity in OSA patients in real-life conditions. Firstly, in a retrospective study, we documented these parameters before treatment. In a second multicentre study, we evaluated the changes in sleep schemes and physical activity under continuous positive airway pressure (CPAP) in 150 OSA patients. We observed that sleep time was increased under CPAP, but physical activity was not improved, contrarily to sleepiness and quality of life. In conclusion, we have shown through these works the clinical interest of two excellent ambulatory tools, H-PSG and Act, for OSA management. Potential clinical implications include enhanced healthcare accessibility, earlier treatment initiation and a closer follow-up of treated patients, through ambulatory tools, in a comfortable environment for the patients.
Doctorat en Sciences médicales (Médecine)
info:eu-repo/semantics/nonPublished
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Wang, David. "Sleep disordered breathing in stable methadone maintenance treatment patients /." Connect to thesis, 2006. http://eprints.unimelb.edu.au/archive/00002992.

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Sutton, Amy M. "Executive Function in the Presence of Sleep Disordered Breathing." Digital Archive @ GSU, 2008. http://digitalarchive.gsu.edu/cps_diss/16.

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The purpose of the study was to investigate whether sleep-disordered breathing (SDB) impairs executive functioning in children. Additionally, the study sought to identify the executive functions at risk in SDB and the contribution of daytime sleepiness. SDB represents a spectrum of upper airway conditions that can be mild, such as snoring, or severe, such as obstructive sleep apnea (OSA). Children with these problems may present with excessive sleepiness, failure to thrive, and a variety of cognitive and behavioral dysfunctions including impaired executive functioning. Beebe and Gozal (2002) developed a theoretical model to explain the impact of sleepiness and hypoxia on executive functioning. This model provided a framework to examine links between the medical disorder and the neuropsychological consequences. Twenty-seven children with suspected SDB were tested with polysomnography (PSG) and a neuropsychological battery. Parents completed subjective measures of cognitive function and sleep symptoms. The children were ages 8 to 18 and had no congenital or acquired brain damage. They were matched for age and gender with 21 healthy controls. The executive function protocol included subtests from the Delis-Kaplan Executive Function System (D-KEFS), the digit span subtest from the Wechsler Intelligence Scale for Children (WISC-IV), the Tower of London-II-Drexel University (TOL-II), the Behavioral Rating Inventory of Executive Functioning (BRIEF), and the Conners’ Continuous Performance Test (CPT-II). Statistical analysis was performed using 2 statistical software packages, SAS and NCSS. Regression analysis was used to evaluate all variables. Due to significant group differences in socio-economic status (SES), SES was included as a covariate, along with IQ. No group differences in IQ were found. Significantly less robust executive function in children with SDB was identified in the domains of cognitive flexibility and impulsivity. Additionally, poorer executive planning and overall inattentiveness was also associated with SDB. Level of significance was set at 0.05 and trends (0.05 < p < 0.10) were acknowledged. Other areas of executive function, including working memory, behavioral and emotional inhibition, and processing speed were not associated with SDB. Moreover, academic functioning was significantly lower in children with SDB, although the differences can be shared equally with SDB, SES and IQ.
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Books on the topic "Sleep disordered breathing (SDB)"

1

Hörmann, Karl. Surgery for sleep-disordered breathing. New York: Springer, 2005.

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Kheirandish-Gozal, Leila, and David Gozal, eds. Sleep Disordered Breathing in Children. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-60761-725-9.

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Hörmann, Karl, and Thomas Verse. Surgery for Sleep Disordered Breathing. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-77786-1.

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Thomas, Verse, ed. Surgery for sleep disordered breathing. 2nd ed. Berlin: Springer, 2010.

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Vicini, Claudio, Fabrizio Salamanca, and Giannicola Iannella, eds. Barbed Pharyngoplasty and Sleep Disordered Breathing. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-96169-5.

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Sajgalikova, Katerina, Erik K. St Louis, and Peter Gay. Neuromuscular disorders and sleep. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0030.

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This chapter examines the range of sleep disturbances seen in patients with neuromuscular disorders, particularly emphasizing sleep-related breathing disorders which may be a presenting manifestation of neuromuscular disorders, and which significantly contribute to morbidity and mortality in this patient population. It provides an overview of physiological and pathological alterations in neuromuscular breathing mechanisms and control during sleep. The symptoms and forms of sleep disordered breathing (SDB) seen in specific neuromuscular disorders such as amyotrophic lateral sclerosis, myopathies, and disorders of neuromuscular junction transmission are reviewed. The chapter concludes with a discussion of management strategies for neuromuscular disorder patients with SDB, which is common in such patients, requiring generalists, neurologists, and sleep physicians to work together toward prompt diagnosis and optimal treatment approaches.
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Zimmerman, Molly E., and Mark S. Aloia. Role of positive pressure therapy on sleep disordered breathing and cognition in the elderly. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0037.

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Efforts aimed toward alleviating senescence have intensified as older adults occupy an increasing proportion of the population. Cognitive abilities become compromised with advancing age, with a vast heterogeneity of presentations, ranging from occasional word-finding difficulties to dementia. The role of sleep disordered breathing (SDB) in moderating or mediating age-related cognitive decline is particularly relevant given its potential reversibility in response to positive airway pressure (PAP) therapies. Establishment of SDB as a significant contributor to the development of dementia and cognitive dysfunction among the elderly has immense public health relevance, underscoring the importance of its early identification and treatment. Although several studies have examined the effect of PAP on cognitive function in older adults with SDB, additional prospective randomized clinical trials are needed. This chapter reviews the literature on SDB and cognition among the elderly as well as cognitive changes in response to PAP. Considerations for future research are also discussed.
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Silvestri, Rosalia. Sleep in older adults. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0050.

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Significant and progressive sleep alterations occur in elderly people, including both circadian and ultradian modifications of sleep. Among these, reduced melatonin and a diminished role of environmental zeitgebers impair sleep rhythmicity, with a tendency toward polyphasic sleep and excessive daytime sleepiness (EDS). The loss of slow-wave sleep (SWS) and EDS are significant, along with behavioral and cognitive alterations in patients with dementia. Sleep disordered breathing (SDB) and restless legs syndrome (RLS)/Willis–Ekbom disease may further aggravate the burden of insomnia and sleep fragmentation, thereby favoring multiple nocturnal arousals with sympathetic activation and cardiovascular dysfunction.
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Junna, Mithri R., Bernardo J. Selim, and Timothy I. Morgenthaler. Central sleep apnea and hypoventilation syndromes. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0018.

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Sleep disordered breathing (SDB) may occur in a variety of ways. While obstructive sleep apnea is the most common of these, this chapter reviews the most common types of SDB that occur independently of upper airway obstruction. In many cases, there is concurrent upper airway obstruction and neurological respiratory dysregulation. Thus, along with attempts to correct the underlying etiologies (when present), stabilization of the upper airway is most often combined with flow generators (noninvasive positive pressure ventilation devices) that modulate the inadequate ventilatory pattern. Among these devices, when continuous positive airway pressure (CPAP) alone does not allow correction of SDB, adaptive servo-ventilation (ASV) is increasingly used for non-hypercapnic types of central sleep apnea (CSA), while bilevel PAP in spontaneous-timed mode (BPAP-ST) is more often reserved for hypercapnic CSA/alveolar hypoventilation syndromes. Coordination of care among neurologists, cardiologists, and sleep specialists will often benefit such patients.
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Thomas Verse,Karl H. Rmann. Surgery for Sleep-Disordered Breathing. Springer, 2008.

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Book chapters on the topic "Sleep disordered breathing (SDB)"

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Mitchell, Ron B. "Sleep-Disordered Breathing (SDB) in Children." In Pediatric Otolaryngology for the Clinician, 197–200. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-127-1_25.

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Goldbart, Aviv D., and Leila Kheirandish-Gozal. "Corticosteroids and Leukotriene Modifiers in Pediatric SDB." In Sleep Disordered Breathing in Children, 521–29. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-60761-725-9_39.

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Rosen, Carol L. "Sleep-Disordered Breathing (SDB) in Pediatric Populations." In Respiratory Medicine, 215–50. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-93739-3_11.

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Kreitinger, Kimberly, Matthew Light, Sagar Patel, and Atul Malhotra. "Sleep-Disordered Breathing." In Sleep Medicine and Mental Health, 131–50. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-44447-1_7.

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Suri, Jagdish Chander, and Tejas M. Suri. "Sleep-Disordered Breathing." In ICU Protocols, 139–45. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-0898-1_15.

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Rohde, Kerstin, and Thomas Verse. "Sleep Disordered Breathing." In Surgery for Sleep Disordered Breathing, 1–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-77786-1_1.

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Suri, Jagdish Chander. "Sleep-Disordered Breathing." In ICU Protocols, 101–6. India: Springer India, 2012. http://dx.doi.org/10.1007/978-81-322-0535-7_13.

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Strohl, Kingman P., and George P. Fahed. "Sleep-Disordered Breathing." In Competencies in Sleep Medicine, 211–33. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-9065-4_13.

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Lynch, Gordon S., David G. Harrison, Hanjoong Jo, Charles Searles, Philippe Connes, Christopher E. Kline, C. Castagna, et al. "Sleep-Disordered Breathing." In Encyclopedia of Exercise Medicine in Health and Disease, 794. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_3044.

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Zwillich, Clifford W., Laurel Wiegand, Kevin Gleeson, John L. Stauffer, and David P. White. "Human Sleep-Disordered Breathing." In Clinical Physiology of Sleep, 125–33. New York, NY: Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4614-7599-6_8.

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Conference papers on the topic "Sleep disordered breathing (SDB)"

1

Barry, A., N. Pyne, S. Munns, and G. Nolan. "Is the Epworth Sleepiness Scale an appropriate screening tool for patients with sleep disordered breathing (SDB): A retrospective clinical audit." In Sleep and Breathing 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/23120541.sleepandbreathing-2021.55.

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Kaminska, M., DA Trojan, D. Da Costa, A. Bar-Or, A. Benedetti, Y. Lapierre, DL Arnold, et al. "Sleep-Disordered Breathing (SDB) and Fatigue in Multiple Sclerosis (MS)." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a2128.

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Gadda, Dario, Luana M. Nosetti, Alberto Caprioglio, Luca Levrini, and Luigi Nespoli. "Oral Modifications Due To Mouth Breathing In Children With Sleep-Disordered Breathing (SDB)." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a3715.

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Kotzia, Doxa, Rishi Pabary, Francois Abel, Aidan Laverty, and Martin Samuels. "Sleep disordered breathing (SDB) in patients with Beckwith-Wiedemann syndrome (BWS)." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa4360.

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Kawar, Eyad, Mohamad Mourad, Eric Gartman, Jigme Sethi, and F. D. McCool. "End Expiratory Lung Volume (EELV) And REM Sleep Disordered Breathing (SDB)." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3594.

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Liu, Shuo, Yann Retory, Amelie Sagniez, Sebastien Hardy, Francois Cottin, Gabriel Roisman, and Michel Petitjean. "Sleep disordered breathing (SDB): respiration simulation on a bench integrating polygraph data." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa2255.

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Bondia, Elvira, Jaime Signes-Costa Miñana, Jose Gavara, Santos Ferrer, Vicente Bodi, Paolo Racugno, Maria P. Lopez-Lereu, Jose Vicente Monmeneu, Maria Pilar Bañuls, and Emilio Servera. "Ventricular structural changes in patients with sleep-disordered breathing (SDB) and myocardial infarction." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa414.

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Pabary, Rishi, Doxa Kotzia, Christophe Goubau, Kylie Russo, and Martin Samuels. "Screening for sleep disordered breathing (SDB) with paediatric sleep questionnaire (PSQ) in children with underlying conditions." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa377.

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Fanfulla, Francesco, Eugenia Taurina, Gian Doenico Pinna, Claudio Bruschi, Roberto Maestri, Elena Robbi, Rita Maestroni, Maurizio Pin, Andrea D'Armini, and Maria Teresa La Rovere. "Sleep Disordered Breathing (SDB) and Chronic Thromboembolic Pulmonary Hypertension: the Effects of Pulmonary Endoarterectomy." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa4722.

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Walsh, SA, J. Meehan, HM Hoey, and P. Greally. "Experience of Sleep Disordered Breathing (SDB) in Irish Children with Prader Willi Syndrome (PWS)." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5045.

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