Relevant bibliographies by topics / Sleep apnoea syndromes

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Sleep apnoea syndromes'

Author: Grafiati
Published: 4 June 2021
Last updated: 6 February 2022

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Journal articles on the topic "Sleep apnoea syndromes"

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van Houwelingen, K. "The sleep apnoea syndromes." European Heart Journal 20, no. 12 (June 1999): 858–66. http://dx.doi.org/10.1053/euhj.1998.1484.

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Pepperell, Justin C. "Sleep apnoea syndromes and the cardiovascular system." Clinical Medicine 11, no. 3 (June 2011): 275–78. http://dx.doi.org/10.7861/clinmedicine.11-3-275.

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Cielo, Christopher M., and Carole L. Marcus. "Obstructive sleep apnoea in children with craniofacial syndromes." Paediatric Respiratory Reviews 16, no. 3 (June 2015): 189–96. http://dx.doi.org/10.1016/j.prrv.2014.11.003.

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Szymanski, Filip M., Anna E. Platek, and Krzysztof J. Filipiak. "Obstructive Sleep Apnoea, Atrial Fibrillation and Erectile Dysfunction – The OSAFED Syndrome – Is there More than Meets the Eye?" European Journal of Arrhythmia & Electrophysiology 01, no. 01 (2015): 19. http://dx.doi.org/10.17925/ejae.2015.01.01.19.

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Obstructive sleep apnoea, atrial fibrillation and erectile dysfunction (OSAFED) syndrome is a new clinical entity recently introduced into clinical practice. The acronym consists of the first letters of the three clinical entities that comprise the disease, namely obstructive sleep apnoea (OSA), atrial fibrillation (AF) and erectile dysfunction (ED). As with many other clinical syndromes, OSAFED syndrome groups several clinical entities, which seemingly concern various organs and have different symptoms, but are closely associated by sharing risk factors and phenotype, and effecting cardiovascular risk in the same manner. OSA, AF and ED are also highly prevalent in the general population and tend to coexist. OSAFED syndrome was named as such to show how important diagnosis is of all these components in specific groups of patient. It is crucial for clinicians to improve the diagnosis and early treatment of all – OSA, AF and ED – and the incorporation of all these factors into one syndrome might help to facilitate this process.
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Nagarajan, Lakshmi, Peter Walsh, Pauline Gregory, Stephen Stick, Jennifer Maul, and Soumya Ghosh. "Respiratory Pattern Changes in Sleep in Children on Vagal Nerve Stimulation for Refractory Epilepsy." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 30, no. 3 (August 2003): 224–27. http://dx.doi.org/10.1017/s0317167100002638.

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Background:An altered breathing pattern in sleep, over two to three weeks, reported by the parents of a child on Vagal Nerve Stimulation (VNS) therapy for refractory epilepsy, prompted a sleep study in him. His polysomnography (PSG) revealed respiratory irregularity concordant with VNS activation. Dyspnoea is a well recognised and reported side effect of the VNS. However there are only a few studies looking at respiration in sleep with VNS. We therefore undertook PSGs in seven other children on VNS.Methods:Sleep studies were undertaken, in accordance with standard clinical practice. Sleep and apnoeas and hypopneas were scored in accordance with conventional criteria. Respiratory pattern changes in sleep (RPCS) with VNS were looked for.Results:Respiratory pattern changes in sleep were seen during PSG in seven of eight children on VNS for refractory epilepsy. Decreased effort and tidal volume occurred in seven children, concordant with VNS activation. In one child, this was associated with a fall in respiratory rate, in the other six children with an increase. No study showed an apnoea/hypopnoea index in the abnormal range. The RPCS were not associated with significant hypoxia or hypercapnoea.Conclusion:Our results suggest that RPCS occur in most children with VNS. This is not surprising in view of the significant influence vagal afferents have on respiratory control centres. The RPCS did not appear to have a clinical impact in our group. However further investigations are suggested to explore this phenomenon, especially in patients with sleep apnoea syndromes or compromised respiratory function.
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Walker, Matthew C., and Sofia H. Eriksson. "Epilepsy and Sleep Disorders." European Neurological Review 6, no. 1 (2011): 60. http://dx.doi.org/10.17925/enr.2011.06.01.60.

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There is a close association between sleep and epilepsy. In some epilepsy syndromes, seizures occur predominantly (or even exclusively) during sleep or on awakening. Excessive daytime sleepiness is common in patients with epilepsy and may be due not only to medication but also to nocturnal seizures or concomitant sleep disorders. Sleep disorders such as obstructive sleep apnoea can worsen epilepsy, with improvement of seizure control following appropriate treatment of the sleep disorder. Conversely, epilepsy and antiepileptic medication can worsen sleep disorders. Nocturnal epileptic seizures may be difficult to differentiate from parasomnias, in particular non-rapid eye movement parasomnias such as night terrors, sleepwalking and confusional arousals, on history alone since there are semiological similarities between the two disorders. Schemes have been developed to facilitate differential diagnosis, although this remains a challenge even using the gold standard, video-electroencephalography telemetry.
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Shikhmirzaeva, E. K., and A. P. Zilber. "A.43 “Ohmeda” monitors in diagnostics and management of sleep apnoea syndromes." British Journal of Anaesthesia 76 (June 1996): 13–14. http://dx.doi.org/10.1016/s0007-0912(18)30898-5.

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Carneiro-Barrera, Almudena, Francisco J. Amaro-Gahete, Amparo Díaz-Román, Alejandro Guillén-Riquelme, Lucas Jurado-Fasoli, Germán Sáez-Roca, Carlos Martín-Carrasco, Jonatan R. Ruiz, and Gualberto Buela-Casal. "Interdisciplinary Weight Loss and Lifestyle Intervention for Obstructive Sleep Apnoea in Adults: Rationale, Design and Methodology of the INTERAPNEA Study." Nutrients 11, no. 9 (September 15, 2019): 2227. http://dx.doi.org/10.3390/nu11092227.

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Obesity is a major risk factor for obstructive sleep apnoea (OSA), the most common sleep-disordered breathing related to neurocognitive and metabolic syndromes, type II diabetes, and cardiovascular diseases. Although strongly recommended for this condition, there are no studies on the effectiveness of an interdisciplinary weight loss and lifestyle intervention including nutrition, exercise, sleep hygiene, and smoking and alcohol cessation. INTERAPNEA is a randomised controlled trial with a two-arm parallel design aimed at determining the effects of an interdisciplinary tailored weight loss and lifestyle intervention on OSA outcomes. The study will include 84 males aged 18–65 with a body mass index of ≥25 kg/m2 and severe to moderate OSA randomly assigned to usual care (i.e., continuous positive airway pressure), or interdisciplinary weight loss and lifestyle intervention combined with usual care. Outcomes will be measured at baseline, intervention end-point, and six-month post-intervention, including apnoea-hypopnoea index (primary outcome), other neurophysical and cardiorespiratory polysomnographic outcomes, sleep quality, daily functioning and mood, body weight and composition, physical fitness, blood biomarkers, health-related quality of life, and cost-effectiveness. INTERAPNEA may serve to establish a cost-effective treatment not only for the improvement of OSA and its vast and severe comorbidities, but also for a potential remission of this condition.
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Moruzzi, P., S. Sarzi-Braga, M. Rossi, and M. Contini. "Sleep apnoea in ischaemic heart disease: differences between acute and chronic coronary syndromes." Heart 82, no. 3 (September 1, 1999): 343–47. http://dx.doi.org/10.1136/hrt.82.3.343.

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MacLean, Joanna E., David Fitzsimons, Dominic A. Fitzgerald, and Karen A. Waters. "The spectrum of sleep-disordered breathing symptoms and respiratory events in infants with cleft lip and/or palate." Archives of Disease in Childhood 97, no. 12 (October 6, 2012): 1058–63. http://dx.doi.org/10.1136/archdischild-2012-302104.

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ObjectiveTo determine the prevalence of sleep-disordered breathing (SDB) symptoms and respiratory events during sleep in infants with cleft lip and/or palate (CL/P).DesignProspective observational study.SettingCleft palate clinic, tertiary care paediatric hospital, before palate surgery.PatientsConsecutive newborn infants with CL/P.Main outcome measuresDemographics, clinical history, sleep symptoms, facial measurement and polysomnography (PSG; sleep study) data.ResultsFifty infants completed PSG at 2.7±2.3 months; 56% were male, and 30% had a clinical diagnosis of Pierre Robin sequence (PRS) or a syndrome. The majority of infants (75%) were reported to snore frequently or constantly, while 74% were reported to have heavy or loud breathing during sleep. The frequency of parent-reported difficulty with breathing during sleep was 10% for infants with isolated CL/P, 33% for those with syndrome, and 43% for PRS (χ2 16.1, p<0.05). All infants had an Obstructive–Mixed Apnoea–Hypopnoea Index (OMAHI) >1 event/h, and 75% had an OMAHI >3 events/h. Infants with PRS had higher OMAHI (34.3±5.1) than infants with isolated CL/P (7.6±1.2) or infants with syndromes (15.6±5.7, F stat, p<0.001). Multivariate analysis showed that PRS was associated with higher OMAHI (B 0.53±0.22, p=0.022), but the majority of the variance for SDB was unexplained (constant B 1.31±0.55, p=0.024).ConclusionsThe results highlight that infants across the spectrum of CL/P have a high risk of SDB symptoms and obstructive respiratory events before palate surgery. Clinicians should enquire about symptoms of SDB and consider investigation with polysomnography in all infants with CL/P.
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Dissertations / Theses on the topic "Sleep apnoea syndromes"

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Revol, Bruno. "Pharmacoépidémiologie des apnées du sommeil Impact of concomitant medications on obstructive sleep apnoea Drugs and obstructive sleep apnoeas Diagnosis and management of central sleep apnea syndrome Baclofen and sleep apnoea syndrome: analysis of VigiBase® the WHO pharmacovigilance database Gabapentinoids and sleep apnea syndrome: a safety signal from the WHO pharmacovigilance database Valproic acid and sleep apnea: a disproportionality signal from the WHO pharmacovigilance database Ticagrelor and Central Sleep Apnea What is the best treatment strategy for obstructive sleep apnoea-related hypertension? Who may benefit from diuretics in OSA? A propensity score-matched observational study." Thesis, Université Grenoble Alpes, 2020. http://www.theses.fr/2020GRALV026.

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Avant leur mise sur le marché, l'évaluation clinique des médicaments repose sur des essais contrôlés randomisés. Bien qu'ils représentent la méthode de référence, leurs résultats sont nécessairement limités aux patients inclus dans ces essais. De plus, ils sont d’abord conçus pour mesurer l'efficacité des traitements, avant d’évaluer leurs effets indésirables. Concernant le syndrome d'apnées du sommeil (SAS), alors que de nombreux essais médicamenteux ont été menés, la plupart des résultats sont de faible niveau de preuve, voire contradictoires. Outre la durée et les effectifs limités de ces essais, une explication est que le SAS est une pathologie hétérogène en termes de symptômes et de physiopathologie, incluant divers "phénotypes" de patients. Des données de vie réelle sont donc nécessaires pour définir quels médicaments pourraient améliorer le SAS ou les comorbidités associées et quels patients pourraient en bénéficier. Au contraire, les cliniciens doivent être avertis que certains médicaments peuvent induire ou aggraver le SAS.La pharmacoépidémiologie fait désormais partie de toute enquête de pharmacovigilance, car elle permet une approche à la fois descriptive et comparative des notifications spontanées. Des associations entre l'exposition à un ou plusieurs médicaments et l'apparition d'effets indésirables peuvent ainsi être recherchées. Comme pour toutes les études observationnelles, la principale difficulté consiste à contrôler les facteurs de confusion. L'un des modèles couramment utilisés est l'analyse cas/non-cas, qui étudie la disproportionnalité entre le nombre d’effets indésirables rapportés avec le médicament d’intérêt, par rapport aux effets notifiés pour les autres médicaments. Nous avons ainsi montré des associations significatives entre l'utilisation de baclofène, des gabapentinoïdes ou de l'acide valproïque et la survenue de SAS dans la base de pharmacovigilance de l'OMS, suggérant le rôle du système GABAergique dans la pathogenèse des apnées centrales d’origine médicamenteuse. Un signal de disproportionnalité a également été observé pour le ticagrélor, reposant sur un mécanisme d'action différent.Les analyses pharmacoépidémiologiques permettent également d'étudier le bénéfice des médicaments en vie réelle. Le score de propension est utilisé pour minimiser les biais de sélection et recréer des conditions de comparabilité proches de celles des essais randomisés. À l'aide de ces méthodes statistiques, nous avons évalué l'intérêt potentiel de cibler le système rénine-angiotensine pour la prise en charge de l'hypertension artérielle chez les patients atteints d’apnées obstructives, en particulier avec l’utilisation des sartans. Chez ces mêmes patients apnéiques et hypertendus, nos travaux suggèrent que les diurétiques pourraient diminuer la sévérité des apnées, notamment en cas de surpoids ou d’obésité modérée. Des études prospectives sont désormais nécessaires afin de confirmer ces résultats, car les données de vie réelle ne peuvent se substituer aux essais cliniques contrôlés
The clinical evaluation of drugs before approval is based on randomized controlled trials. Although they are considered as the gold standard for testing drugs, their results are necessarily limited to patients included in the trials. Moreover, almost all clinical trials are primarily designed to assess the efficacy of a treatment, so safety is only a secondary concern. Regarding sleep apnea syndrome (SAS), while many drug trials have been conducted, most of the results are weak or even contradictory. In addition to limited trial duration and population size, one explanation is that the sleep apnea population is highly heterogeneous with respect to symptoms and physiological traits linked to disease pathogenesis, giving various patient “phenotypes”. Real-life data are therefore needed to define which drugs could improve SAS or associated comorbidities and who might benefit from them. On the contrary, clinicians need to be aware that some drugs may induce or worsen sleep apnea.Pharmacoepidemiology is now part of any pharmacovigilance survey, as it provides both descriptive and comparative approaches of spontaneous reports. Associations between the exposure to one or more drugs and the occurrence of adverse effects can thus be sought. As for all observational studies, the major difficulty is to control for confounding factors. One of the study designs commonly used, is the case/non-case analysis, which investigates disproportionality between the numbers of adverse drug reactions reported with the drug of interest compared to the number reported with all other drugs. In this way, we showed significant associations between the use of baclofen, gabapentinoids or valproic acid and the reporting of SAS in the WHO drug adverse event database, suggesting a role of the GABAergic system in the pathogenesis of drug-induced central sleep apnea. A disproportionality signal was also found for ticagrelor, based on a different mechanism of action.Pharmacoepidemiological analyses also make it possible to study the benefit of drugs in real-life. Propensity scores are used to minimize selection bias, leading to a comparability between the exposure groups close to that observed in randomized trials. Using these statistical methods, we have investigated the potential value of targeting the renin-angiotensin system for the management of hypertension in obstructive sleep apnea (OSA) patients, especially the use of sartans. For hypertensive apneic patients, our work suggests that diuretics could decrease the severity of OSA, particularly in the overweight or moderately obese. Prospective studies are now needed to confirm these findings, because real-life data cannot be a substitute for controlled clinical trials
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Solin, Peter 1964. "Central sleep apnoea in heart failure : recognition and pathogenesis." Monash University, Dept. of Medicine, 2000. http://arrow.monash.edu.au/hdl/1959.1/8972.

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Shepherd, Kelly. "Gastro-oesophageal reflux in obstructive sleep apnoea : prevalence and mechanisms." University of Western Australia. School of Anatomy and Human Biology, 2009. http://theses.library.uwa.edu.au/adt-WU2009.0085.

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Background. Obstructive Sleep Apnoea (OSA) is associated with an increase in nocturnal gastro-oesophageal reflux (nocturnalGOR) events and symptoms, however the mechanism for this remains undefined. Treatment of OSA with continuous positive airway pressure (CPAP) has been shown to reduce nocturnalGOR in individuals with OSA however the reasons for this reduction are not clear. The combination of OSA and nocturnalGOR could be particularly problematic for individuals who have had a lung transplant in whom Bronchiolitis Obliterans Syndrome (BOS) limits survival. It is thought that GOR plays a role in the development of BOS in these individuals. Methods and Results. Five interrelated studies were undertaken. The first two studies sought to determine and compare the prevalence and risk factors of nocturnalGOR in OSA patients with the general population. To do this, a GOR questionnaire was completed by 2,042 members of the general community as part of the Busselton Health Survey and by 1,116 patients with polysomnography-diagnosed OSA. Risk of OSA in the general population was determined using a standardised sleep questionnaire. 137 of the OSA patients completed the questionnaire before and after treatment with CPAP. The prevalence of nocturnalGOR symptoms reported more than once a week (frequent symptoms) was greater in OSA patients (10.1%) than the general population (5.8%) (p<0.001), in individuals from the general population at high (11.2%) than low risk of OSA (4.5%) (p<0.001) and in patients with severe (14.7%) than mild OSA (5.2%) (p<0.001). Treatment of OSA with CPAP decreased the prevalence of frequent nocturnalGOR from 9.0% to 3.8% (p=0.04). In the general population, high risk of OSA was independently associated with a 2.4-fold increased risk of frequent ABSTRACT vi nocturnalGOR symptoms than low risk. In the OSA group, disease severity was independently associated with nocturnalGOR symptoms, with an adjusted odds ratio of 1.7 for frequent nocturnalGOR symptoms.
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Lai, Yuen-kwan Agnes, and 賴婉君. "Continuous positive airway pressure education on adherence in adults with obstructive sleep apnoea." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/198849.

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Poor adherence to continuous positive airway pressure (CPAP) treatment in patients with obstructive sleep apnoea (OSA) limits its therapeutic effectiveness and has a major impact on clinical outcomes. Effective education programme is important to enhance CPAP use. However, existing education programmes are either manpower or resource demanding and may not be feasible in clinical practice. Moreover, the Self-Efficacy Measure for Sleep Apnoea (SEMSA) has been widely adopted for assessing adherence-related cognitions on CPAP therapy in OSA patients, but it was not available for Chinese. The aims of this thesis are: (i) to perform linguistic and psychometric evaluation of a Chinese version of SEMSA (SEMSA-C); (ii) to examine the efficacy of brief motivational enhancement education programme in addition to standard care versus standard care only on improving adherence to CPAP treatment in patients with OSA. The SEMSA-C was obtained after the standard forward-backward translation process. A randomised controlled trial was then conducted on newly diagnosed OSA patients. Patients in the control group received standard care (SC) comprising advice on the importance of CPAP therapy and its care while those in the intervention group received SC plus motivational enhancement education programme (ME). ME focused to enhance subjects’ knowledge, motivation and self-efficacy to use CPAP, comprising one 45-minute session on the day after CPAP titration and one 10-minute telephone follow-up shortly after commencing CPAP treatment. Epworth Sleepiness Scale (ESS), SEMSA-C, and quality of life were assessed. CPAP usage data were downloaded at the completion of this 3-month study. The primary outcome was the CPAP adherence. Furthermore, 21 patients were randomly sampled at baseline and completed the SEMSA-C at one week. 100 patients (Men : Women, 84 : 16) with OSA indicated for CPAP treatment were recruited, with an average age of 52±10 years, and apnoea hypopnoea index (AHI) of 36.2±22 events/hour. Factor analysis of SEMSA-C identified three factors: risk perception, outcome expectancies and treatment self-efficacy. Their corresponding internal consistency was high with Cronbach’s alpha >0.88, which were larger than all correlations between subscales (Range: 0.14 to 0.58). The correlations between items and their hypothesized subscale (Range: 0.58 to 0.85) were generally higher than the correlations between items and their competing subscales (Range: -0.10 to 0.58). One-week test-retest intra-class correlation ranged from 0.70 to 0.82. CPAP adherence was associated with outcome expectancies and treatment self-efficacy at 3-month assessment. Furthermore, SEMSA-C demonstrated an improvement in self-efficacy (standardised response mean = 0.33, p = .044) but no significant changes were observed in the other two factors, after CPAP use. The 100 patients were followed for 3 months. The interventional effects maintained during the 3-month study period. There were a better CPAP use [higher daily CPAP usage of 2 hours/day (Cohen d = 1.33, p < .001), four-fold the number of subjects using CPAP for ≥ 70% of days with ≥ 4 hours per day (p < 0.001)], and greater improvements in ESS by 2.2 (p = 0.001) and treatment self-efficacy by 0.2 (p = 0.012) in the intervention group, relative to the control group. The traditional Chinese SEMSA-C possesses satisfactory psychometric properties. It is a reliable and responsive instrument to measure perceived risks, outcome expectancies and treatment self-efficacy in Chinese patients with OSA. Moreover, the newly developed brief motivational enhancement education programme in addition to standard care is effective in improving adherence to CPAP treatment, treatment self-efficacy and daytime sleepiness.
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Nursing Studies
Doctoral
Doctor of Nursing
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Ahrens, Anika. "A randomized clinical trial of the treatment of obstructive sleep apnoea using oral appliances." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47849599.

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　　　Obstructive sleep apnoea (OSA) is the most common sleep-related breathing disorder and is associated with a range of adverse physical, social and psychological outcomes that affect quality of life (QoL). Two systematic reviews of the literature (part of this thesis work) found there is conflicting evidence of how different mandibular advancement device (MAD) designs features may affect clinical and subjective OSA outcomes in certain patients. Therefore, a randomized cross-over trial was conducted. Firstly, the correlation between clinical OSA indicators and QOL was explored among patients referred for OSA treatment using MADs. In addition, associations of OSA risk factors, dental status and demographic variables with clinical OSA indicators and QoL indices were determined. Secondly, the efficacy of two different MADs in the treatment of adult OSA patients was assessed and compared. Thirdly, the efficacy of the two MADs in the treatment of adult OSA patients from the subjective perspective of their bed partners was determined. 　　　A consecutive sample of 45 adult OSA patients referred from Queen Mary Hospital Sleep Centre to the Prince Philip Dental Hospital for oral appliance therapy was recruited and treated with a monobloc MAD and a twinblock MADs for a period of 3 months per MAD (cross-over randomised trial). Changes in clinical OSA outcomes were assessed by polysomnography (PSG) and changes in subjective outcomes by the disease-specific Sleep Apnoea Quality of Life Index (SAQLI) questionnaire, the Functional Outcome of Sleep Questionnaire (FOSQ) and Epworth Sleepiness Scale (ESS). Patient compliance, side-effects and MAD preference, as well as MAD treatment impact on the patients’ bed partner was also assessed. At baseline, some clinical OSA indicators, subjective QoL and certain OSA risk factors were significantly correlated (p<0.05). There were significant variations in clinical OSA indicators and subjective QoL indices with respect to certain risk factors (p<0.05), demographic variables (p<0.05) and dental status (p<0.01). 　　　There was a significant difference in favour of the monobloc MAD in terms of improving the apnoea-hypopnoea index (AHI) (p<0.05) and oxygen desaturation index (ODI) (p<0.01). Significantly more patients achieved clinical treatment success with the monobloc compared to the twinblock (p<0.05). Both MADs were efficacious in improving patients’ SAQLI score (p<0.01), FOSQ score (p<0.01) and ESS score (p<0.01). Significantly more patients achieved QoL treatment success with the monobloc (p<0.05) compared to the twinblock. More patients were ‘very satisfied’ with the monobloc treatment (p<0.05) and 63% preferred it to the twinblock. 　　　 No significant difference was found between patients’ and bed partners perceptions of symptom improvement post treatment, however, the monobloc resulted in a significant reduction in bed partners’ daytime sleepiness (p<0.01) and allowed significantly more co-sleeping at night (p<0.05). 　　　 　　　This study concluded that the monobloc is superior in improving subjective QoL and clinical OSA indicators. The monobloc was the preferred MAD and patients were more satisfied with it; bed partners rated this MAD as superior in improving their own daytime sleepiness and co-sleeping.
published_or_final_version
Dentistry
Doctoral
Doctor of Philosophy
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Wakwella, Ajith S. "Processing of snore related sounds for the diagnosis of obstructive sleep apnoea (OSA) /." [St. Lucia, Qld.], 2005. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe18755.pdf.

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Harris, Ricci, and n/a. "Obstructive sleep apnoea syndrome : symptoms and risk factors among Maori and non-Maori adults in Aotearoa." University of Otago. Weelington School of Medicine & Health Sciences, 2003. http://adt.otago.ac.nz./public/adt-NZDU20070507.112047.

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More is becoming known about the importance of sleep to health, with inadequate sleep recognised as a significant public health issue. Sleep clinics have reported disproportionate numbers of Māori and Pacific peoples with more severe obstructive sleep apnoea syndrome (OSAS), raising concerns about accessibility of services and possible differences in prevalence between ethnic groups. Prevalence information on sleep disorders in Aotearoa is needed to assess its public health impact and plan for population health care needs. This thesis presents a national study examining the prevalence of OSAS symptoms and risk factors among Maori and non-Maori adults in Aotearoa. This project is also situated within the wider scope of ethnic inequalities in health between Maori and non-Maori and is concerned with making a positive contribution to Maori health and the elimination of disparities. Kaupapa Maori Research (KMR) is the underlying methodology that drives this study. As such, it assumes a Maori norm, and prioritises Maori needs. A Maori/non-Maori analytical framework is used that distinguishes Maori as tangata whenua, addresses Maori health needs as well as non-Maori, and enables the monitoring of guarantees as outlined by the Treaty of Waitangi. The goals of this thesis were to estimate the prevalence of OSAS symptoms and risk factors among Maori and non-Maori adults in New Zealand, and to examine independent predictive variables for specific OSAS symptoms. An objective was also to contribute to KMR through designing and undertaking a KMR project using a quantitative method, with the development of concepts for use in other areas of research. In April 1999, a short questionnaire was sent to a sample of 10000 New Zealands (5500 of Maori descent and 4500 non-Maori participants to enable research questions to be examined with equal statistical power for both groups. The results demonstrate that the prevalence of OSAS symptoms and risk factors, particularly among non-Maori, are comparable to other international studies, indicating that OSAS is likely to be a common problem among adults in Aotearoa. Furthermore, the results suggest that Maori are significantly more likely to suffer from OSAS than non-Maori, with higher rates of symptoms and risk factors of OSAS among both men and women. As an area of medicine that is under-serviced in Aotearoa, the results provide important information with which to plan for population needs. There are a number of health implications from this study, relating specifically to the diagnosis and management of OSAS in Aotearoa, and to Maori health and the elimination of disparities. These are multi-levelled and include health service implications across the continuum of care, from specialist sleep services to primary care; public health implications that involve preventive measures and broader determinants of health; and KMR principles that can be applied to interventions and health research in general. As a KMR project the implications and recommendations focus on Maori health research in general. As a KMR project the implications and recommendations focus on Maori health gain and addressing disparities in health. This is consistent with Maori health rights, and a population approach that considers health inequalities and the role of wider determinants of health and health services.
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Joffe, David. "Obstructive sleep apnoea the genesis of daytime somnolence and cognitive impairment : arousals, hypoxia and circadian rhythm /." Connect to full text, 1997. http://hdl.handle.net/2123/382.

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Thesis (Ph. D.)--University of Sydney, 1998.
Title from title screen (viewed Apr. 15, 2008). Submitted in fulfilment of the requirements for the degree of Doctor of Philosophy to the Dept. of Respiratory Medicine, Royal North Shore Hospital, Faculty of Medicine. Degree awarded 1998; thesis submitted 1997. Includes bibliography. Also available in print form.
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Desai, Anup. "Obstructive sleep apnoea and driver performance prevalence, correlates, and implications for driver fatigue /." Connect to full text, 2002. http://hdl.handle.net/2123/589.

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Thesis (Ph. D.)--University of Sydney, 2003.
Includes tables and questionnaires. Title from title screen (viewed Apr. 29, 2008). Submitted in fulfilment of the requirements for the degree of Doctor of Philosophy to the Faculty of Medicine. Degree awarded 2003; thesis submitted 2002. Includes bibliography. Also available in print form.
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Baillieul, Sébastien. "Syndrome d'apnées du sommeil et cerveau : une relation bidirectionnelle Continuous positive airway pressure improves gait control in severe obstructive sleep apnoea: A prospective study Hypoxic conditioning and the central nervous system: A new therapeutic opportunity for brain and spinal cord injuries?" Thesis, Université Grenoble Alpes, 2020. https://thares.univ-grenoble-alpes.fr/2020GRALS025.pdf.

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Les contraintes physiologiques cérébrales rendent le cerveau humain vulnérable à l'hypoxie, qu’elle soit environnementale (haute altitude) ou en lien avec une pathologie hypoxémiante. Parmi ces pathologies, et en raison de sa forte prévalence dans la population générale, le syndrome d'apnées obstructives du sommeil (SAOS) est un modèle physiopathologique reconnu des effets délétères de l'hypoxie sur le cerveau. Les épisodes cycliques d'apnées et d'hypopnées survenant au cours du sommeil qui caractérisent le SAOS entraînent une hypoxie intermittente, une fragmentation du sommeil et des fluctuations de la pression intra-thoracique, tous trois facteurs déclenchant des mécanismes intermédiaires contribuant au développement de maladies cardio-métaboliques ainsi que des répercussions cérébrales (troubles cognitifs et accidents vasculaires cérébraux (AVC)). Ce travail de thèse explore la relation bidirectionnelle entre les syndromes d’apnées du sommeil (SAS) et le cerveau. Le premier axe se concentre sur les conséquences neurocognitives du SAOS au travers du contrôle de la marche. Les répercussions neurocognitives du SAOS sont à ce jour bien décrites et des troubles de la marche ont récemment été mis en évidence, avec une relation de type dose-réponse entre la gravité du SAOS et la sévérité des troubles de la marche. Il a ainsi été suggéré que la marche pouvait représenter un marqueur des répercussions cérébrales du SAOS. Les effets du traitement par pression positive continue (PPC) sur le contrôle de la marche ont été investigués au cours de ce travail de Thèse, avec des résultats contrastés. Dans une première étude prospective contrôlée, 8 semaines de traitement par PPC entraînaient une amélioration du contrôle de la marche chez des patients atteints de SAOS sévère (Baillieul et al., 2018, Plos One). Afin de valider ces résultats et d'étudier les corrélats neurophysiologiques du lien entre marche et SAOS, nous avons mené un essai contrôlé randomisé étudiant l'impact de 8 semaines de traitement par PPC comparativement à la Sham-PPC (Baillieul et al., 2020, Soumis). Contrairement à notre hypothèse, nous n'avons constaté aucune amélioration du contrôle de la marche dans le groupe PPC, résultat corroboré par l'absence d'impact de la PPC sur les déterminants du contrôle de la marche. Le deuxième axe est centré sur les répercussions cérébro-vasculaires des SAS. SAS et AVC sont deux pathologies graves et étroitement liées, le SAS pouvant être à la fois cause et conséquence potentielle des AVC. Le présent travail est axé sur l'identification des traits phénotypiques de SAS chez les patients post-AVC, afin d'en améliorer le diagnostic (Baillieul et al., en préparation). Le dépistage du SAS post-AVC est crucial en raison du risque élevé de morbi-mortalité et de conséquences fonctionnelles associées au SAS après AVC, mais il ne peut être effectif sans une identification plus précise des patients à risque de SAS. Le troisième axe a été conçu comme une perspective qui servira au développement du deuxième axe. Dans ce dernier axe, le potentiel de l'imagerie cérébrale et en particulier de l'imagerie par résonance magnétique pour développer des marqueurs de récupération et étudier les mécanismes physiopathologiques des déficiences liées aux AVC sont présentés, au travers de la marche et de son contrôle. Les corrélats neuronaux de la marche comme activité post-AVC sont mis en évidence, en utilisant une approche de type Voxel-based lesion-symptom mapping (Baillieul et al., 2019, Hum. Mov. Sci.). Les marqueurs d'imagerie basés sur l’utilisation du Diffusion tensor imaging pour prédire la récupération de la marche post-AVC sont également présentés (Soulard et al., 2019, Neurology). Ces travaux sur les marqueurs d'imagerie cérébrale de la récupération post-AVC serviront à développer des outils pour les recherches à venir sur les corrélats neuronaux des SAS post-AVC
The human brain is a perfect example of our dependence on oxygen. Brain physiological constraints render it vulnerable to hypoxia, such as encountered in environmental conditions (high altitude exposure) or pathological hypoxemic conditions. Among those pathological conditions, and due to its high prevalence in general population and the various levels of hypoxia resulting of the different degrees of severity of the pathology, obstructive sleep apnoea syndrome (OSAS) is a pathophysiological model of choice to investigate the detrimental effects of hypoxia on the brain. The cyclical, repeated episodes of apnoea and hypopnea during sleep that characterize OSAS result in intermittent hypoxia, sleep fragmentation and fluctuations in intrathoracic pressure, which are stressors that triggers mechanisms contributing to the initiation and progression of life-threatening cardiometabolic diseases, as well as several brain repercussions, such as cognitive impairment and stroke. This Thesis work explores the bidirectional relationship between sleep apnoea syndromes (SAS) and the brain. The first axis is focused on the neurocognitive consequences of OSAS through the lens of gait control. The neurocognitive signature of OSAS has been thoroughly investigated but recently, gait impairments have been highlighted in severe OSAS, with dose-response relationship between OSAS severity and the magnitude of gait impairments. As gait control relies at least partly on frontal lobe functions, it has been suggested that gait could represent a marker of OSAS brain repercussions. We investigated the effects of continuous positive airway pressure (CPAP) treatment on gait control, with contrasting results. In a first prospective controlled study, eight weeks of CPAP improved gait control in severe OSAS patients (Baillieul et al., 2018, Plos One). In order to validate those results and investigate the neurophysiological correlates of the link between gait control and OSAS, we conducted a randomized controlled trial which investigated the impact of an 8-week CPAP treatment compared to sham-CPAP on gait control in severe OSAS patients (Baillieul et al., 2020, Submitted). Contrary to our hypothesis, we found no improvement in gait control in the CPAP group and this result is substantiated by the absence of impact of CPAP on the determinants of gait control, further illustrating the complexity of the OSAS-neurocognitive relationship. The second axis is focused on the cerebrovascular repercussions of SAS. SAS and stroke are both severe intertwined conditions, SAS being both cause and potentially consequence of stroke. The present work is focused on the identification of phenotypic traits of SAS in post-stroke patients, to improve diagnosis of SAS following stroke (Baillieul et al., in preparation). Screening stroke patients for SAS is crucial due to the high risk of morbimortality and functional consequences associated to SAS following stroke but cannot be achieved without a more accurate identification of patients at risk to develop SAS following stroke. The third axis has been conceived as a perspective that will serve the development of the second axis. In this last axis, the potential of brain imagery and in particular magnetic resonance imagery to develop markers of stroke recovery as well as investigate the pathophysiological mechanisms underlying stroke-related deficiencies are presented, with a specific focus on gait and walking activity. The neural correlates of walking activity following stroke are highlighted, using a voxel-based lesion-symptom mapping approach (Baillieul et al., 2019, Hum. Mov. Sci.). Imagery markers of walking recovery following stroke using diffusion tensor imaging are also presented (Soulard et al. 2019, Neurology). This work on brain imagery markers of stroke recovery will further serve the development of investigations focused on the neural correlates of SAS following stroke
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Books on the topic "Sleep apnoea syndromes"

1

Sleep Apnoea. Plymouth: European Respiratory Society, 2010.

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Kiyoshi, Togawa, ed. Sleep apnea and rhonchopathy. Basel: Karger, 1993.

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Pascualy, Ralph A. Snoring and Sleep Apnea: Sleep Well, Feel Better. 3rd ed. New York: Demos Medical Pub., LLC, 2000.

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Understanding snoring and sleep apnea. New Haven, CT: Yale University Press, 2004.

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Pascualy, Ralph A. Snoring and Sleep Apnea. New York: Demos Medical Publishing, 2009.

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Questions and answers about sleep apnea. Sudbury, Mass: Jones and Bartlett, 2009.

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The sleep apnea syndrome, more as an illness. New York: Nova Science Publishers, 2009.

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Handbook of sleep-related breathing disorders. Oxford: Oxford University Press, 1993.

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Yoshida, Kazuya. Sleep apnea syndrome in the stomatognathic system. Hauppauge, N.Y: Nova Science Publisher's, 2009.

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Chilman-Blair, Kim. Medikidz explain sleep apnea. New York: Rosen Central, 2011.

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Book chapters on the topic "Sleep apnoea syndromes"

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De Backer, W. "Obstructive Sleep Apnea-Hypopnea Syndrome." In Sleep Apnea, 90–96. Basel: KARGER, 2006. http://dx.doi.org/10.1159/000093150.

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Caples, S. M., and V. K. Somers. "Central Sleep Apnea, Hypoventilation Syndromes and Periodic Breathing Disorders." In Sleep Apnea, 180–91. Basel: KARGER, 2006. http://dx.doi.org/10.1159/000093167.

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Messiha, Ashraf, Ben Gurney, and Piet Haers. "Obstructive Sleep Apnoea Syndrome." In Orthognathic Surgery, 690–700. Chichester, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781119004370.ch44.

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Basto, Rita Pinto, and Daniel O. Rodenstein. "Upper Airway Imaging in Sleep Apnea Syndrome." In Sleep Apnea, 69–78. Basel: KARGER, 2006. http://dx.doi.org/10.1159/000093148.

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Locke, Dona. "Sleep Apnea Syndrome." In Encyclopedia of Clinical Neuropsychology, 3197–98. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_279.

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Locke, Dona E. C. "Sleep Apnea Syndrome." In Encyclopedia of Clinical Neuropsychology, 2306–7. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_279.

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Locke, Dona. "Sleep Apnea Syndrome." In Encyclopedia of Clinical Neuropsychology, 1. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56782-2_279-2.

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Lavie, Lena, and Peretz Lavie. "Oxidative Stress – The Culprit of Obstructive Sleep Apnea Syndrome." In Sleep Apnea, 97–104. Basel: KARGER, 2006. http://dx.doi.org/10.1159/000093151.

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Riha, Renata L. "Genetics Aspects of the Obstructive Sleep Apnea/Hypopnea Syndrome." In Sleep Apnea, 105–12. Basel: KARGER, 2006. http://dx.doi.org/10.1159/000093152.

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Krieger, J. "Obstructive sleep apnoea-hypopnea syndrome and upper airway resistance syndrome." In Sleep, 357–89. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0217-3_29.

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Conference papers on the topic "Sleep apnoea syndromes"

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Bailey, M., A. White, and B. Gray. "Obstructive sleep apnoea hypopnoea syndrome (OSAHS) service - streamline of workflow by digital solutions." In Sleep and Breathing 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/23120541.sleepandbreathing-2021.19.

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Morsy, Nesreen, Amani Badawi, Magda Ahmad, and Nabil Awadalla. "Predictors of ocular complications in obstructive sleep apnoea syndrome patients." In ERS/ESRS Sleep and Breathing Conference 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/23120541.sleepandbreathing-2017.p17.

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Delaunay, Nathalie, and Miriam Marchon. "Obstructive sleep apnoea syndrome: a screening test for periodical medical examinations?" In ERS/ESRS Sleep and Breathing Conference 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/23120541.sleepandbreathing-2017.p11.

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Podlipnik, Matevz, Irena Sarc, and Kristina Ziherl. "Restless leg syndrome is common in patients with obstructive sleep apnoea." In ERS/ESRS Sleep and Breathing Conference 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/23120541.sleepandbreathing-2017.p20.

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Mylavarapu, Goutham, Ephraim Gutmark, Mihai Mihaescu, and Shanmugam Murugappan. "Simulation of Flow and Structural Dynamics in Human Upper Airways." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19501.

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Obstructive Sleep Apnea and Hypo Apnea Syndrome (OSAHS) is a respiratory disorder characterized by repeated episodes of partial or complete airway obstruction. OSAHS is also associated with decreased quality of life, decreased cardiovascular health, day-time sleepiness, and increased transportation accidents. Its high prevalence with as much as 4% of American population suffering from OSAHS also makes it an important health care issue[1]. Existing surgical treatments suffer from a moderate to high successful failure rates. Broader research on this respiratory disorder for a better understanding of the pathophysiology of human upper airway, to develop better diagnostic methods and treatment modalities is very much needed.
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Şahin Duyar, S., and S. Fırat. "A new treatment option for treatment-emergent central apnea syndrome: a case series." In Sleep and Breathing 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/23120541.sleepandbreathing-2021.7.

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Jing Zhou, Li-yi Jiang, Su-juan Liu, and Xiao-ming Wu. "Diagnosis algorithm of sleep apnea syndrome." In 2012 IEEE-EMBS International Conference on Biomedical and Health Informatics (BHI). IEEE, 2012. http://dx.doi.org/10.1109/bhi.2012.6211717.

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Marotta, Antonella, Giuseppe Fiorentino, Anna Annunziata, Gianfranco Scotto di Frega, Rosa Cauteruccio, Anna Michela Gaeta, Maria Antonietta Mazza, and Maria Cardone. "Overlap: Asthma and sleep apnea syndrome." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa4174.

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Bailey, M., A. White, and B. Gray. "Obstructive sleep apnoea hypopnoea syndrome (OSAHS) service improvement through streamlining of pathway and upskilling of respiratory physiologists." In Sleep and Breathing 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/23120541.sleepandbreathing-2021.8.

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Gauci, Jonathan, Matthew Zammit, Luca Andrea Borg, Ayrton Borg Axisa, Peter Cassar, Matthew Farrugia, Rachel Gambin, Michael Micallef, Nicole Zammit, and Josef Micallef. "Prevalence of obstructive sleep apnoea syndrome in Malta." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa4414.

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Reports on the topic "Sleep apnoea syndromes"

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Cao, Yanmei, Liangbin Xie, Ping Wang, and Rui Chen. The relationship between obstructive sleep apnea hypopnea syndrome and interstitial lung diseases: an uptated meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, July 2021. http://dx.doi.org/10.37766/inplasy2021.7.0085.

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Zhou, Min, Qijun Liang, Qiulan Pei, Fan Xu, and Hang Wen. Chinese Herbs Medicine Huatan Huoxue Prescription for Obstructive Sleep Apnea Hypopnea Syndrome as Complementary Therapy : a protocol for a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2020. http://dx.doi.org/10.37766/inplasy2020.5.0079.

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