Relevant bibliographies by topics / Sleep apnoea

Academic literature on the topic 'Sleep apnoea'

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Published: 4 June 2021
Last updated: 20 February 2023

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Journal articles on the topic "Sleep apnoea"

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Guilleminault, C., C. Crowe, MA Quera-Salva, L. Miles, and M. Partinen. "Periodic leg movement, sleep fragmentation and central sleep apnoea in two cases: reduction with Clonazepam." European Respiratory Journal 1, no. 8 (August 1, 1988): 762–65. http://dx.doi.org/10.1183/09031936.93.01080762.

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Two subjects presented with periodic leg movement (PLM) syndrome during sleep that was characterized by marked sleep fragmentation and repetitive short central apnoeas. Treatment of PLM using Clonazepam, a benzodiazepine with hypnotic properties, markedly reduced the sleep fragmentation due to PLM and, despite its depressant properties on the central nervous system, controlled the repetitive central apnoeas. These two observations, although rare, give insight into the role of non-ventilatory variables in the development of sleep apnoea. Significant sleep fragmentation should be considered when assessing factors leading to respiratory instability during sleep and/or the pathophysiology of sleep apnoea syndromes.
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Andreas, S., B. von Breska, K. Magnusson, and H. Kreuzer. "Validation of automated sleep stage and apnoea analysis in suspected obstructive sleep apnoea." European Respiratory Journal 6, no. 1 (January 1, 1993): 48–52. http://dx.doi.org/10.1183/09031936.93.06010048.

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Full-night polysomnography is necessary for the diagnosis of obstructive sleep apnoea (OSA). However, analysis of the sleep stages and apnoeas is time-consuming. Computer systems for automated analysis have, thus, been developed to alleviate this task. We investigated 27 consecutive patients referred to our sleep laboratory with suspected OSA. The analysis of sleep stages and apnoeas was performed by visual scoring, according to Rechtschaffen and Kales, and by commercially available automated analysis device. The mean difference between visual scoring and automated analysis was -1, 111, -140, -3, 1 and 27 min, for sleep stages awake, I, II, III, IV and rapid eye movement (REM) respectively. For the apnoea index, the automated analysis rated a lower figure (mean difference 7.h-1, 95% confidence interval 2-12.h-1). The diagnosis of OSA was performed with a sensitivity of 85% and a specificity of 93% by automated analysis. Comparison of two independent handscores showed good agreement, with a mean difference of 6, 4, 3, -7, 1 and -1 min, for sleep stages awake, I, II, III, IV and REM, respectively. In conclusion, the automated analysis underestimates stage I sleep and the apnoea index. Visual scoring is advisable for control of the results. Automated analysis should only be used by those who are able to perform a visual analysis.
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Pokorski, M., and U. Jernajczyk. "Nocturnal Oxygen Enrichment in Sleep Apnoea." Journal of International Medical Research 28, no. 1 (February 2000): 1–8. http://dx.doi.org/10.1177/147323000002800101.

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We hypothesized that a modest oxygen enrichment, rather than 100% oxygen supplementation as used in previous trials, could result in improvement in ventilatory and cardiac symptoms, in patients with obstructive sleep apnoea (OSA), without jeopardizing the chemostimulant ventilatory drive. This hypothesis was tested in five male patients with OSA in a single-blinded trial consisting of one night spent sleeping in control room air (control night), followed by one night spent sleeping while exposed to air with a 9% enriched oxygen content (oxygen-enriched night). Oxygen enrichment resulted in a significant shift in the oxygen saturation profile towards values of ≥ 95% and to decrease desaturation dips throughout the night. The apnoea index decreased from the control night to the oxygen-enriched night from 52.7 ± 10.4 to 38.9 ± 9.3; the decrease being greatest for the longest apnoeas (≥ 30 s). Additionally, the cardiovascular status improved. No signs of depressed chemostimulant drive in the oxygen-enriched night were detected. We conclude that nocturnal oxygen enrichment merits consideration for therapeutic trial in the prevention of long apnoeic and desaturation episodes.
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Jun, Jonathan C., Swati Chopra, and Alan R. Schwartz. "Sleep apnoea." European Respiratory Review 25, no. 139 (February 29, 2016): 12–18. http://dx.doi.org/10.1183/16000617.0077-2015.

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Sleep apnoea is a disorder characterised by repetitive pauses in breathing during sleep caused by airway occlusion (obstructive sleep apnoea) or altered control of breathing (central sleep apnoea). In this Clinical Year in Review, we summarise high-impact research from the past year pertaining to management, diagnosis and cardio-metabolic consequences of sleep apnoea.
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de Chazal, Philip, Conor Heneghan, and Walter T. McNicholas. "Multimodal detection of sleep apnoea using electrocardiogram and oximetry signals." Philosophical Transactions of the Royal Society A: Mathematical, Physical and Engineering Sciences 367, no. 1887 (October 30, 2008): 369–89. http://dx.doi.org/10.1098/rsta.2008.0156.

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A method for the detection of sleep apnoea, suitable for use in the home environment, is presented. The method automatically analyses night-time electrocardiogram (ECG) and oximetry recordings and identifies periods of normal and sleep-disordered breathing (SDB). The SDB is classified into one of six classes: obstructive, mixed and central apnoeas, and obstructive, mixed and central hypopnoeas. It also provides an estimated apnoea, hypopnoea and apnoea–hypopnoea index. The basis of the method is a pattern recognition system that identifies episodes of apnoea by analysing the heart variability, an ECG-derived respiration signal and blood oximetry values. The method has been tested on 183 subjects with a range of apnoea severities who have undergone a full overnight polysomnogram study. The results show that the method separates control subjects from subjects with clinically significant sleep apnoea with a specificity of 83 per cent and sensitivity of 95 per cent. These results demonstrate that home-based screening for sleep apnoea is a viable alternative to hospital-based tests with the added benefit of low cost and minimal waiting times.
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Hanning, C. D. "Sleep apnoea." Anaesthesia 44, no. 9 (September 1989): 786. http://dx.doi.org/10.1111/j.1365-2044.1989.tb09279.x.

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Robinson, Tracey D., and Iven H. Young. "Sleep apnoea." Medicine 32, no. 1 (January 2004): 91–95. http://dx.doi.org/10.1383/medc.32.1.91.28473.

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Wright, John, Trevor A. Sheldon, and Ian Watt. "Sleep apnoea." Lancet 354, no. 9178 (August 1999): 600. http://dx.doi.org/10.1016/s0140-6736(05)77956-3.

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Primhak, R. "SLEEP APNOEA." Archives of Disease in Childhood - Education and Practice 90, no. 4 (December 1, 2005): ep87-ep91. http://dx.doi.org/10.1136/adc.2005.072975.

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Prowse, K., and M. B. Allen. "Sleep apnoea." British Journal of Diseases of the Chest 82 (January 1988): 329–40. http://dx.doi.org/10.1016/0007-0971(88)90085-x.

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Dissertations / Theses on the topic "Sleep apnoea"

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Gunaratnam, Kogulan. "OBSTRUCTIVE SLEEP APNOEA AND PERIODONTITIS." Thesis, Faculty of Dentistry, 2008. http://hdl.handle.net/2123/4057.

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Obstructive sleep apnoea (OSA) and its associated daytime symptoms form a syndrome, obstructive sleep apnoea-hypopnoea syndrome (OSAHS) that affects about 5% of the population worldwide (Young et al 2002a, Pack 2006). OSA is characterized by repeated episodes of upper airway obstruction during sleep, resulting in recurrent hypoxemia and sleep fragmentation (Hensley & Ray 2005). These in turn are associated with neurocognitive disorders, hypertension and cardiovascular complications (Pack 2006). Current therapies for this condition include surgical interventions, oral appliances and continuous positive airways pressure (CPAP). Systemic and local airway inflammation has recently been linked to OSA and is hypothesized to increase the risk of cardiovascular complications (Lavie 2005). While the exact mechanism is not certain, it is believed that the underlying systemic inflammation from OSA is due to the hypoxia/reperfusion injury from intermittent hypoxia that occurs with OSA (Lavie 2005). Specifically, the episodic hypoxia in OSA leads to increased production of reactive oxidative species (ROS) and, via various pathways, in the formation of systemic inflammatory mediators. The resultant inflammatory response is then responsible for the increased cardiovascular morbidity and mortality by potentiating disease in those that already have inflammatory disease or triggering inflammatory diseases in people with existing genetic, behavioural and environmental exposure. Periodontitis involves the supporting structures of the tooth and is a disease caused by specific bacteria that triggers an inflammatory response (Kinane 2001). Tissue damage and destruction, including loss of the connective tissue attachment between the tooth and the jaw, together with resorption of supporting bone, is initiated by the micro-organisms and mediated by the host response. Periodontitis, which is a severe form of periodontal disease, is one of the most common chronic infections in the world. The prevalence of moderate to severe periodontitis across the globe is in the range of 5 to 20 % (Burt 2005). Recent studies have speculated on an association between periodontitis and systemic inflammation in, for example, diabetes (Soskolne & Klinger 2001), rheumatoid arthritis (Mercado et al. 2000) and cardiovascular disease (CVD) (Beck & Offenbacher 2005), but no research has been undertaken on the link between OSA and periodontitis. This review will focus on features of OSA, inflammation and periodontitis to examine if there is a possible link between OSA and periodontitis by means of systemic inflammation.
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Mason, Rebecca Helen. "Vascular complications of obstructive sleep apnoea." Thesis, University of Bristol, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.619138.

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Obstructive sleep apnoea (OSA) is the third commonest respiratory condition after Asthma and COPD and has been increasingly linked to cardiovascular consequences. This thesis examines how OSA might affect different vascular beds; large (the aorta), medium (the carotid artery) and small (retinal and cerebral blood vessels) through five different studies. Each study will be reported as a separate chapter and a final discussion will assess the overall conclusions. Methodology Study one examines the prevalence of OSA in individuals with an abdominal aortic aneurysm and demonstrates the increased prevalence and rate of aneurysm expansion in those with severe OSA. Study two demonstrates the increased prevalence of OSA in individuals with type two diabetes and clinically significant diabetic macular oedema (CS MO). Study three examines the clinical benefit of continuous positive airway pressure (CPAP) in individuals with OSA and CS MO and demonstrates an improvement in visual acuity when CPAP is used for >2.5hrs per night. Study four is a retrospective examination of the effect of snoring on carotid vessel disease and shows no significant difference between the severity of snoring and degree of carotid artery stenosis. Study five describes the effect of minimally symptomatic obstructive sleep apnoea on cerebrovascular disease and shows no association between OSA and small white matter change but, does confirm the association of increasing age and hypertension. Discussion This thesis adds to our understanding of the association of OSA and vascular disease and the potential therapeutic benefits of CPAP in these individuals.
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Papaioannou, Ioannis. "Glucose intolerance in obstructive sleep apnoea." Thesis, Imperial College London, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.516557.

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Davies, David Paul. "Snoring, obstructive sleep apnoea and stroke." Thesis, University of Newcastle Upon Tyne, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.364858.

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McMillan, Alison. "Obstructive sleep apnoea in older people." Thesis, Imperial College London, 2014. http://hdl.handle.net/10044/1/28969.

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Obstructive sleep apnoea (OSA) is common and the prevalence increases with age. When OSA leads to sleep disruption and excessive daytime sleepiness, it is referred to as obstructive sleep apnoea syndrome (OSAS). The aim of this thesis was to investigate the consequences of OSAS in older people (˃ 65 years) and the effect of continuous positive airway pressure (CPAP) therapy. CPAP is the treatment of choice in moderate to severe OSAS in middle aged people. However, there is a paucity of evidence on the therapeutic and economic benefits of CPAP in older people with OSAS. The two studies in this thesis aimed to address this by comparing outcomes in older people with OSAS before and after treatment with CPAP. The first study presented is the thesis is the PREDICT trial; a multicentre randomised controlled trial of CPAP in older people with OSAS. The trial studied the clinical efficacy of CPAP after 3 months, while determining the cost effectiveness of treatment over 12 months. The results of the trial showed that CPAP was an effective treatment for reducing excessive daytime sleepiness by -2.1 (95%CI -3.0 to -1.3); p < 0.001 points as measured by the Epworth sleepiness scale. CPAP also improved quality of life, with a statistically significant increase in the quality adjusted life years calculated with the SF-6D, equating to one week. The CPAP group also accrued marginally lower health care costs over 12 months compared to the group treated with best supportive care alone. Overall the economic benefit of CPAP was linked to the reduced healthcare usage offsetting the cost of the equipment. The second study presented in the thesis was a single centre randomised controlled trial to investigate the impact of CPAP on cognitive function and brain morphology in older people with minimally symptomatic OSAS after 6 months of treatment. In this study I tested the hypothesis that older patients with OSAS have cognitive impairment and corresponding brain changes which would be modifiable with treatment. The results of this study suggested older people with minimally symptomatic OSAS had normal cognitive function but impaired attention and executive function. CPAP treatment improved one aspect of attention, although memory and overall cognitive function were unchanged. The results of the brain MRI scans are not presented, and are in the process of being analysed. In conclusion the data presented in this thesis support the use of CPAP therapy in older people with excessive daytime sleepiness due to OSAS.
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Revol, Bruno. "Pharmacoépidémiologie des apnées du sommeil Impact of concomitant medications on obstructive sleep apnoea Drugs and obstructive sleep apnoeas Diagnosis and management of central sleep apnea syndrome Baclofen and sleep apnoea syndrome: analysis of VigiBase® the WHO pharmacovigilance database Gabapentinoids and sleep apnea syndrome: a safety signal from the WHO pharmacovigilance database Valproic acid and sleep apnea: a disproportionality signal from the WHO pharmacovigilance database Ticagrelor and Central Sleep Apnea What is the best treatment strategy for obstructive sleep apnoea-related hypertension? Who may benefit from diuretics in OSA? A propensity score-matched observational study." Thesis, Université Grenoble Alpes, 2020. http://www.theses.fr/2020GRALV026.

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Avant leur mise sur le marché, l'évaluation clinique des médicaments repose sur des essais contrôlés randomisés. Bien qu'ils représentent la méthode de référence, leurs résultats sont nécessairement limités aux patients inclus dans ces essais. De plus, ils sont d’abord conçus pour mesurer l'efficacité des traitements, avant d’évaluer leurs effets indésirables. Concernant le syndrome d'apnées du sommeil (SAS), alors que de nombreux essais médicamenteux ont été menés, la plupart des résultats sont de faible niveau de preuve, voire contradictoires. Outre la durée et les effectifs limités de ces essais, une explication est que le SAS est une pathologie hétérogène en termes de symptômes et de physiopathologie, incluant divers "phénotypes" de patients. Des données de vie réelle sont donc nécessaires pour définir quels médicaments pourraient améliorer le SAS ou les comorbidités associées et quels patients pourraient en bénéficier. Au contraire, les cliniciens doivent être avertis que certains médicaments peuvent induire ou aggraver le SAS.La pharmacoépidémiologie fait désormais partie de toute enquête de pharmacovigilance, car elle permet une approche à la fois descriptive et comparative des notifications spontanées. Des associations entre l'exposition à un ou plusieurs médicaments et l'apparition d'effets indésirables peuvent ainsi être recherchées. Comme pour toutes les études observationnelles, la principale difficulté consiste à contrôler les facteurs de confusion. L'un des modèles couramment utilisés est l'analyse cas/non-cas, qui étudie la disproportionnalité entre le nombre d’effets indésirables rapportés avec le médicament d’intérêt, par rapport aux effets notifiés pour les autres médicaments. Nous avons ainsi montré des associations significatives entre l'utilisation de baclofène, des gabapentinoïdes ou de l'acide valproïque et la survenue de SAS dans la base de pharmacovigilance de l'OMS, suggérant le rôle du système GABAergique dans la pathogenèse des apnées centrales d’origine médicamenteuse. Un signal de disproportionnalité a également été observé pour le ticagrélor, reposant sur un mécanisme d'action différent.Les analyses pharmacoépidémiologiques permettent également d'étudier le bénéfice des médicaments en vie réelle. Le score de propension est utilisé pour minimiser les biais de sélection et recréer des conditions de comparabilité proches de celles des essais randomisés. À l'aide de ces méthodes statistiques, nous avons évalué l'intérêt potentiel de cibler le système rénine-angiotensine pour la prise en charge de l'hypertension artérielle chez les patients atteints d’apnées obstructives, en particulier avec l’utilisation des sartans. Chez ces mêmes patients apnéiques et hypertendus, nos travaux suggèrent que les diurétiques pourraient diminuer la sévérité des apnées, notamment en cas de surpoids ou d’obésité modérée. Des études prospectives sont désormais nécessaires afin de confirmer ces résultats, car les données de vie réelle ne peuvent se substituer aux essais cliniques contrôlés
The clinical evaluation of drugs before approval is based on randomized controlled trials. Although they are considered as the gold standard for testing drugs, their results are necessarily limited to patients included in the trials. Moreover, almost all clinical trials are primarily designed to assess the efficacy of a treatment, so safety is only a secondary concern. Regarding sleep apnea syndrome (SAS), while many drug trials have been conducted, most of the results are weak or even contradictory. In addition to limited trial duration and population size, one explanation is that the sleep apnea population is highly heterogeneous with respect to symptoms and physiological traits linked to disease pathogenesis, giving various patient “phenotypes”. Real-life data are therefore needed to define which drugs could improve SAS or associated comorbidities and who might benefit from them. On the contrary, clinicians need to be aware that some drugs may induce or worsen sleep apnea.Pharmacoepidemiology is now part of any pharmacovigilance survey, as it provides both descriptive and comparative approaches of spontaneous reports. Associations between the exposure to one or more drugs and the occurrence of adverse effects can thus be sought. As for all observational studies, the major difficulty is to control for confounding factors. One of the study designs commonly used, is the case/non-case analysis, which investigates disproportionality between the numbers of adverse drug reactions reported with the drug of interest compared to the number reported with all other drugs. In this way, we showed significant associations between the use of baclofen, gabapentinoids or valproic acid and the reporting of SAS in the WHO drug adverse event database, suggesting a role of the GABAergic system in the pathogenesis of drug-induced central sleep apnea. A disproportionality signal was also found for ticagrelor, based on a different mechanism of action.Pharmacoepidemiological analyses also make it possible to study the benefit of drugs in real-life. Propensity scores are used to minimize selection bias, leading to a comparability between the exposure groups close to that observed in randomized trials. Using these statistical methods, we have investigated the potential value of targeting the renin-angiotensin system for the management of hypertension in obstructive sleep apnea (OSA) patients, especially the use of sartans. For hypertensive apneic patients, our work suggests that diuretics could decrease the severity of OSA, particularly in the overweight or moderately obese. Prospective studies are now needed to confirm these findings, because real-life data cannot be a substitute for controlled clinical trials
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Riha, Renata Ludmilla. "Genetics of the sleep apnoea/hypopnoea syndrome." Thesis, University of Edinburgh, 2004. http://hdl.handle.net/1842/25122.

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This thesis examined possible candidate genes that might contribute towards the development of OSAHS. The genes of interest included tumour necrosis factor –alpha (potential associations with ageing, hypercytokinaemia in OSAHS, obesity and sleepiness). Apolioprotein E (associations with the development of cerebrovascular disease), the serotonin receptor 2A (modulation of upper airway muscle tone and response to selective serotonin re-uptake inhibitors), beta-2 adrenoreceptor (growth, fat metabolism and blood pressure) and the growth hormone receptor (influence on postnatal bone growth and height including the craniofacial complex). 557 consecutive subjects with a diagnosis of OSAHS were approached at the Scottish Sleep Centre. 104 subjects (all Caucasian) were recruited together with 107 of their siblings as well as an additional 17 unrelated subjects without OSAHS and underwent overnight polysomnography and cephalometry. Blood was taken for DNA analysis. Subjects were classified as having definite OSAHS (n=110), indeterminate status (n=34) or not having OSAHS (n=83) based on their apnoea/hypopnoea frequency and sleepiness as measured by the Epworth Sleepiness Score. DNA was extracted using standard techniques and polymorphisms in the candidate genes were examined using allelic discrimination testing with TaqMan™. The Apolipoprotein E4 polymorphisms were determined using polymerase chain reaction, restriction fragment length polymorphism. DNA from192 random, healthy UK blood donors (assumed not to have OSAHS) was used as an additional control. Differences between subjects with and without OSAHS were as expected: there were over twice as many men in the OSAHS group compared to the non-OSAHS group (p<0.0001) and systolic blood pressure was significantly higher (p = 0.002) in the OSAHS group. Furthermore, the OSAHS group were more obese (p<0.0001) and had a greater neck circumference (p<0.0001) than the non-OSAHS group. Cephalometry revealed that both male and female apnoeics had significantly lower-set hyoid bones than non-apnoeic snorers (p = 0.01 and p = 0.038 respectively). In male subjects with OSAHS, a smaller mandible and lower-set hyoid were the most important characteristics distinguishing siblings with from sibs without OSAHS, independently of age and BMI. However, age, sex, BMI and edentulism were found to influence craniofacial parameters in both groups. For the genetic analyses, the Apo E e4 allele (examined in 73 subjects) was not associated significantly with a diagnosis of OSAHS. In addition, the TNF-a – 308 A allele showed significant association with the OSAHS phenotype when comparing siblings discordant for carriage of this allele. The increased prevalence of some of the minor polymorphisms in the study population with OSAHS suggested there may be abnormalities in metabolism and the regulation of growth, which may directly contribute to its aetiology. These preliminary findings would require exploration in other populations, but are compatible with OSAHS being a polygenic disorder. This thesis highlights that there is much to be done in our search for relevant genetic factors that will lead to a greater understanding of this complex, chronic and very common disease.
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West, Sophie Diana. "Obstructive sleep apnoea and type 2 diabetes." Thesis, University of Leicester, 2007. http://hdl.handle.net/2381/29539.

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Aims: To establish the prevalence of OSA in individuals with type 2 diabetes, and whether treatment with CPAP improves glycaemic control and insulin resistance. Methods and Results: A questionnaire was sent to 1682 men with type 2 diabetes from hospital and primary care databases. Fifty-six percent replied; 57% scored as 'high' and 39% as 'low' risk for OSA; 4% had known OSA. Overnight oximetry in 240 respondents from the 'high' and 'low' risk groups showed 31% and 13% respectively had significant OSA, verified by sleep studies. Exploration and oximetry data to the questionnaire respondent population suggests 23% have OSA. Comparison with a general population showed OSA prevalence to be significantly higher in the diabetes population (p<0.001). Multiple linear regression revealed diabetes was a significant independent OSA predictor after correction for BMI, explaining 8% of OSA variance (p<0.001). There was no correlation of OSA with HbA1c. A double blind randomized controlled trial of CPAP in men with type 2 diabetes and newly diagnosed OSA was performed. Forty-two men attended for baseline investigations and then received either therapeutic or placebo CPAP for 3 months; baseline tests were then repeated. In the therapeutic group, significantly improved subjective and objective sleepiness were noted, however no significant improvement in HbA1c, euglycaemic clamp, adiponectin or HOMA-%S were found. Conclusions: OSA is highly prevalent in men with type 2 diabetes; most individuals are undiagnosed. Diabetes may be a significant independent contributor to OSA risk. CPAP treatment of OSA does not improve insulin resistance or HbA1c in men with type 2 diabetes.
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Filtness, Ashleigh J. "Obstructive sleep apnoea and daytime driver sleepiness." Thesis, Loughborough University, 2011. https://dspace.lboro.ac.uk/2134/8338.

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Driver sleepiness is known to be a major contributor to road traffic incidents (RTIs). An initial literature review identified many studies reporting untreated obstructive sleep apnoea (OSA) sufferers as having impaired driving performance and increased RTI risk. It is consistently reported that treatment with continuous positive air pressure (CPAP) improves driving performance and decreases RTI risk, although most of these studies are conducted less than one year after starting treatment. UK law allows treated OSA patients to continue driving if their doctor states that treatment has been successful. Despite the wealth of publications surrounding OSA and driving, 6 key areas were identified from the literature review as not fully investigated, the: (i) prevalence of undiagnosed OSA in heavy goods vehicle (HGV) drivers in the UK; (ii) impact of sleep restriction on long term CPAP treated OSA compared with healthy controls; (iii) ability of treated OSA participants to identify sleepiness when driving; (iv) impact of one night CPAP withdrawal on driving performance; (v) individual difference in driving performance of long term CPAP treated OSA participants; (vi) choice of countermeasures to driver sleepiness by two groups susceptible to driver sleepiness, OSA and HGV drivers. Key areas (i) and (vi) were assessed using questionnaires. 148 HGV drivers were surveyed to assess OSA symptoms and preference of countermeasures to driver sleepiness. All participants completing the driving simulator study were also surveyed. 9.5% of HGV drivers were found to have symptoms of suspected undiagnosed OSA. Additionally the OSA risk factors were more prevalent for HGV drivers than reported in national statistics reports for the general population. The most effective countermeasures to driver sleepiness (caffeine and a nap) were not the most popular. Being part of a susceptible group (OSA or HGV driver) and prior experience of driver sleepiness did not promote effective choice of countermeasure. Key areas (ii) to (v) were assessed using a driving simulator. Driving simulators present a safe environment to test participants in a scenario where they may experience sleepiness without endangering other road users.
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Robertson, Helen. "POISE : Prediction of imminent sleep apnoea episodes." Thesis, University of Strathclyde, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.510870.

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Books on the topic "Sleep apnoea"

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Sleep Apnoea. Plymouth: European Respiratory Society, 2010.

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Karmakar, Nemai Chandra, Yang Yang, and Abdur Rahim. Microwave Sleep Apnoea Monitoring. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-6901-7.

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Fraser, Andrew K. Obstructive sleep apnoea and allied disorders. Glasgow: Scottish Forum for Public Health Medicine, 1997.

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Rees, Karen. Mechanisms of arousal responses from NREM sleep in patients with obstructive sleep apnoea. Salford: University ofSalford, 1995.

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Sleep with Buteyko: The only way to stop snoring, sleep apnoea and insomnia. Galway, Ireland: Buteyko Books, 2011.

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Nicoll, Deborah J. Prospective evaluation of pulse transit time in the diagnosis and management of the obstructive sleep apnoea/hypopnoea syndrome. Oxford: Oxford Brookes University, 1999.

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Pascualy, Ralph A. Snoring and Sleep Apnea. New York: Demos Medical Publishing, 2009.

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Carr, Michele M. Pediatric obstructive sleep apnea. Alexandria, VA: American Academy Of Otolaryngology--Head and Neck Surgery Foundation, 2007.

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Chilman-Blair, Kim. Medikidz explain sleep apnea. New York: Rosen Central, 2011.

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Kiyoshi, Togawa, ed. Sleep apnea and rhonchopathy. Basel: Karger, 1993.

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Book chapters on the topic "Sleep apnoea"

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Clarke, R. W. "Obstructive Sleep Apnoea." In Pediatric ENT Radiology, 199–205. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-59367-3_14.

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Goh, Daniel Y. T. "Obstructive Sleep Apnoea." In Paediatric Sleep Disorders, 67–77. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-5791-8_8.

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Jokic, Ruzica. "Obstructive Sleep Apnoea." In Sleep Disorders in Psychiatric Patients, 213–38. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-642-54836-9_12.

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Clarke, Raymond W. "Obstructive Sleep Apnoea." In Diseases of the Ear, Nose & Throat in Children, 73–75. Boca Raton: CRC Press, 2022. http://dx.doi.org/10.1201/9780429019128-16.

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Montgomery, Paul, and Lindsay Dianne Shepard. "Sleep Apnoea and Sleep Disorders." In Pathy's Principles and Practice of Geriatric Medicine, 617–27. Chichester, UK: John Wiley & Sons, Ltd, 2012. http://dx.doi.org/10.1002/9781119952930.ch54.

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Dhillon, Ramindar S., and James W. Fairley. "Sleep apnoea in children." In Multiple-choice Questions in Otolaryngology, 184–85. London: Palgrave Macmillan UK, 1989. http://dx.doi.org/10.1007/978-1-349-10805-3_273.

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Messiha, Ashraf, Ben Gurney, and Piet Haers. "Obstructive Sleep Apnoea Syndrome." In Orthognathic Surgery, 690–700. Chichester, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781119004370.ch44.

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Powell, Steven. "Paediatric Obstructive Sleep Apnoea." In Scott-Brown’s Otorhinolaryngology Head and Neck Surgery, 293–309. Eighth edition. | Boca Raton : CRC Press, [2018] | Preceded by Scott-Brown’s otorhinolaryngology, head and neck surgery.: CRC Press, 2018. http://dx.doi.org/10.1201/9780203731017-27.

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"Sleep Apnoea." In Canadian Family Practice Guidelines. New York, NY: Springer Publishing Company, 2019. http://dx.doi.org/10.1891/9780826194985.0144.

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Harrington, Jane. "Sleep apnoea." In Cambridge Handbook of Psychology, Health and Medicine, 883–86. Cambridge University Press, 2001. http://dx.doi.org/10.1017/cbo9780511543579.235.

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Conference papers on the topic "Sleep apnoea"

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Bouloukaki, Izolde, Athanasia Pataka, Paschalis Steiropoulos, Ludger Grote, Marisa Bonsignore, Jan Hedner, Oreste Marrone, et al. "Positional obstructive sleep apnea in the European Sleep Apnoea Database (ESADA) study." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa4335.

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Baia Afonso, M. F., J. Carvalho, E. Nabais, M. Escaleira, R. Staats, P. Pinto, and C. Bárbara. "Positional obstructive sleep apnoea – an individualized phenotype." In Sleep and Breathing 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/23120541.sleepandbreathing-2021.70.

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Neffati, O., H. Zaibi, B. Dhahri, J. Ben Ammar, M. A. Baccar, S. Azzabi, and H. Aouina. "Obstructive sleep apnoea and hypothyroidism." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa2366.

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Trakada, Georgia, Angeliki Konsta, Lemonia Velentza, and Dimitris Dikeos. "Obstructive sleep apnoea and electroconvulsive therapy complications." In ERS/ESRS Sleep and Breathing Conference 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/23120541.sleepandbreathing-2017.p97.

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Grote, Ludger, Holger Hein, Ozen K. Basoglu, Ondrej Ludka, Johan Verbraecken, Athanasia Pataka, Stefan Mihaicuta, et al. "Reference values for mean overnight saturation in sleep apnoea – the European Sleep Apnoea Database (ESADA)." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.4996.

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Chaidas, K., and A. Ashman. "Funding for treatment of adult Obstructive Sleep Apnoea in England." In Sleep and Breathing 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/23120541.sleepandbreathing-2021.33.

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SABIL, AbdelKebir, Guillaume Baffet, Cédric Freycenon, and Jean Pinguet. "Apnoea characterisation using tracheal sounds spectral analysis." In ERS/ESRS Sleep and Breathing Conference 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/23120541.sleepandbreathing-2017.p18.

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Lo Iacono, C. A. M., C. D'Alessandro, E. Amato, and I. Carbone. "Sex differences in obstructive sleep apnoea in aging: a retrospective study." In Sleep and Breathing 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/23120541.sleepandbreathing-2021.10.

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Vukoja, Marija, Dragana Milicic, Aleksandar Bokan, Ilija Andrijevic, and Ivan Kopitovic. "Lung function in patients with obstructive sleep apnoea." In ERS/ESRS Sleep and Breathing Conference 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/23120541.sleepandbreathing-2017.p14.

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Lamprou, K., K. Chaidas, A. Nickol, and J. Stradling. "Discrepancy between patient- and partner- reported sleepiness in patients with obstructive sleep apnoea." In Sleep and Breathing 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/23120541.sleepandbreathing-2021.29.

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Reports on the topic "Sleep apnoea"

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Yu, Zhixiang, Qiuhe Ji, Fu Yi, and Jinxiang Cheng. Association Between Obstructive Sleep Apnoea And T2DM: A Dose-Response Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2020. http://dx.doi.org/10.37766/inplasy2020.11.0027.

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Torres, Claudia Fernandez, and Alvaro Zubizarreta Macho. Mandibular advancement appliances to treat apnea: an update of the most used currently. A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0034.

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Review question / Objective: Mandibular advancement devices used to treat obstructive sleep apnea. Condition being studied: Obstructive sleep apnea is characterized by episodes of a complete (apnea) or partial collapse (hypopnea) of the upper airway with an associated decrease in oxygen saturation or arousal from sleep. This disturbance results in fragmented, nonrestorative sleep. Other symptoms include loud, disruptive snoring, witnessed apneas during sleep, and excessive daytime sleepiness. OSA has significant implications for cardiovascular health, mental illness, quality of life, and driving safety.
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Czerwaty, Katarzyna, Karolina Dżaman, Krystyna Maria Sobczyk, and Katarzyna Irmina Sikrorska. The Overlap Syndrome of Obstructive Sleep Apnea and Chronic Obstructive Pulmonary Disease: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0077.

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Review question / Objective: To provide the essential findings in the field of overlap syndrome of chronic obstructive pulmonary disease and obstructive sleep apnea, including prevalence, possible predictors, association with clinical outcomes, and severity compared to both chronic obstructive pulmonary disease and obstructive sleep apnea patients. Condition being studied: OSA is characterized by complete cessation (apnea) or significant decrease (hy-popnea) in airflow during sleep and recurrent episodes of upper airway collapse cause it during sleep leading to nocturnal oxyhemoglobin desaturations and arousals from rest. The recurrent arousals which occur in OSA lead to neurocognitive consequences, daytime sleepiness, and reduced quality of life. Because of apneas and hypopneas, patients are experiencing hypoxemia and hypercapnia, which result in increasing levels of catecholamine, oxidative stress, and low-grade inflammation that lead to the appearance of cardio-metabolic consequences of OSA. COPD is a chronic inflammatory lung disease defined by persistent, usually pro-gressive AFL (airflow limitation). Changes in lung mechanics lead to the main clini-cal manifestations of dyspnea, cough, and chronic expectoration. Furthermore, patients with COPD often suffer from anxiety and depression also, the risk of OSA and insomnia is higher than those hospitalized for other reasons. Although COPD is twice as rare as asthma but is the cause of death eight times more often.
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Torres-Castro, Rodrigo, Lilian Solis-Navarro, Homero Puppo, Roberto Vera-Uribe, Victoria Alcaraz-Serrano, and Jordi Vilaró. Respiratory muscle training in patients with obstructive sleep apnea: A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, February 2022. http://dx.doi.org/10.37766/inplasy2022.2.0096.

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Review question / Objective: Is respiratory muscle training effective in patients with obstructive sleep apnea? Condition being studied: Effects of respiratory muscle training in patients with obstructive sleep apnea. Information sources: We included the following databases: Pubmed, CENTRAL, Web of Science, CINAHL, EMBASE, Scopus and Scielo. Additionally, the references list of the included studies will be manually reviewed.
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Hazboun, Tawfiq N. Obstructive Sleep Apnea Oral Appliance vs. Auto Titrating Positive Airway Pressure. Fort Belvoir, VA: Defense Technical Information Center, June 2013. http://dx.doi.org/10.21236/ad1012981.

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Sun, Dong, Yanan Zhang, and Di Zhou. The relationship between obstructive sleep apnea and retinal vein occlusion : a Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, February 2022. http://dx.doi.org/10.37766/inplasy2022.2.0068.

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Lu, Jiyuan, Lingdan Xu, Yanduo Yang, Yucheng Meng, Yi Li, Huihui Wang, and Bin Liu. Obstructive sleep apnea and serum total testosterone: a system review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2022. http://dx.doi.org/10.37766/inplasy2022.3.0110.

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Parthasarathy, Sairam, Stefano Guerra, Stuart F. Quan, Michael Grandner, and Patricia L. Haynes. Does a Peer Support Program Improve Satisfaction With Treatment Among Patients With Obstructive Sleep Apnea? Patient-Centered Outcomes Research Institute (PCORI), April 2020. http://dx.doi.org/10.25302/04.2020.ihs.130602505.

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Zhang, Dong, Zhi Xiang Yu, and Fu Yi. Atrial fibrillation And the Severe of Obstructive Sleep Apnea (OSA): A Dose-Response Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2020. http://dx.doi.org/10.37766/inplasy2020.12.0104.

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Nakase-Richardson, Risa, Jessica M. Ketchum, Leah Drasher-Phillips, Daniel J. Schwartz, Karel Calero, Marie N. Dahdah, Kimberley R. Monden, et al. Comparing Ways to Identify Sleep Apnea in People with Traumatic Brain Injury during Inpatient Rehabilitation. Patient-Centered Outcomes Research Institute (PCORI), September 2021. http://dx.doi.org/10.25302/09.2021.cer.151133005.

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